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Books on the topic 'Infarcted Myocardium'

Author: Grafiati

Published: 3 June 2025

Last updated: 19 July 2025

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1

G, Reves J., and Society of Cardiovascular Anesthesiologists, eds. Acute revascularization of the infarcted heart. Grune & Stratton, 1987.

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2

H, Schmutzler, Rutsch W. 1941-, and Dougherty F. C. 1946-, eds. Limitation of infarct size. Springer-Verlag, 1989.

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3

1931-, Frohlich Edward D., ed. Preventive aspects of coronary heart disease. Davis, 1990.

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4

Sproule, Laurie Kathleen. Carboxypeptidase N and U in myocardial infarct and angina patients. s.n.], 1992.

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5

H, Mattingly Patrick, Lohr Kathleen N. 1941-, and Institute of Medicine (U.S.). Division of Health Care Services., eds. Acute myocardial infarction: Setting priorities for effectiveness research : report of a study by a committee of the Institute of Medicine, Division of Health Care Services. National Academy Press, 1990.

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6

Reeves, François. Prévenir l'infarctus ou y survivre: Les voies du coeur. Éditions du CHU Sainte-Justine, 2007.

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7

1954-, Topol Eric J., ed. Acute coronary syndromes. 3rd ed. Marcel Dekker, 2005.

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8

Veldkamp, Rolf Frederik. Continuous digital 12-lead ST-segment monitoring in Acute Myocardial Infarction =: Continue digitale 12-afleidingen ST-segment bewaking tijdens het acute myocard infarct : proefschrift. The Author, 1995.

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9

Inc, NetLibrary, ed. Acute coronary syndromes. Marcel Dekker, 1998.

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10

Levin, Rhoda F. Vivre avec un cardiaque: Les voies de la serenite et de l'espoir. Editions de l'Homme, 1992.

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11

Meltzer, Lawrence E. Intensive coronary care: A manual for nurses. 4th ed. Prentice-Hall, 1990.

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12

Naughton, John. Exercise testing: Physiological, biomechanical, and clinical principles. Futura Pub. Co., 1988.

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13

(Editor), Loren J. Field, and Kai C. Wollert (Editor), eds. Rebuilding the Infarcted Heart. Informa Healthcare, 2007.

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14

Salvatori, Daniela, Harsha D. Devalla, and Robert Passier. Cells to repair the infarcted myocardium. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0030.

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The adult mammalian heart has poor regenerative capacity. Loss of functional cardiomyocytes following myocardial infarction leads to the replacement of functional muscle by scar tissue. This has a detrimental effect on cardiac function and may lead to heart failure. Potential regeneration of severe cardiac damage would require replacement of dead and damaged cardiomyocytes by transplantation, recruitment of endogenous progenitor cells, or induction of cardiomyocyte proliferation. For more than a decade, clinical trials to ameliorate the injured heart have been under way. However, after evaluat

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15

Li, Elaine Y. H. Immunological reaction in cell transplantation into the infarcted myocardium. 2004.

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16

Menasché, Philippe. Stem Cell Therapy Post-AMI. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199544769.003.0010.

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• Experimental studies suggest that bone marrow-derived stem cells can improve function of infarcted myocardium• This benefit seems to involve paracrine signalling and limitation of left ventricular remodelling rather than true regeneration of cardiomyocytes from donor cells• These experimental findings have been translated in the clinical setting into significant, although moderate, improvements in cardiac function and LV remodelling but the extent to which these benefits impact on event-free long term survival remains to be determined• Optimisation of this therapeutic strategy will require a

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17

Schmutzler, Horst, Wolfgang Rutsch, and Frank C. Dougherty. Limitation of Infarct Size. Springer, 1988.

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18

Schmutzler, Horst, Wolfgang Rutsch, and Frank C. Dougherty. Limitation of Infarct Size. Springer London, Limited, 2012.

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19

Kaufmann, W., and K. J. Zülch. Brain and Heart Infarct. Springer, 2011.

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20

Pepine, Carl J. Acute Myocardial Infarction. F a Davis Co, 1989.

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21

Kaufmann, W., and K. J. Zülch. Brain and Heart Infarct II. Springer, 2011.

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22

Hossmann, K. A., W. Kaufmann, K. J. Zülch, and V. Hossmann. Brain and Heart Infarct II. Springer London, Limited, 2012.

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23

Heidbuchel, Hein, Mattias Duytschaever, and Haran Burri. Mapping the infarct scar. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198766377.003.0049.

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24

Tuan Peng Chua Andrew Coats. Clinician's Manual On Managing The Post-Myocardial Infarct Patient. Science Press Ltd, 1998.

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25

Coats, Andrew J. S., and Tuan Peng Chua. Clinician's Manual on Managing the Post-myocardial Infarct Patient. Science Press, 1998.

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26

Tcheng, James E. Primary Angioplasty in Acute Myocardial Infarction (Contemporary Cardiology). Humana Press, 2002.

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27

Silent myocardial ischemia and infarction. Dekker, 1986.

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28

Silent myocardial ischemia and infarction. 3rd ed. Dekker, 1993.

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29

Silent myocardial ischemia and infarction. 2nd ed. Dekker, 1989.

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30

Lear, Martha Weinman. Heartsounds: The Story of a Love and Loss. Open Road Integrated Media, Inc., 2014.

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31

Lear, Martha Weinman. Heartsounds: The Story of a Love and Loss. Open Road Integrated Media, Inc., 2014.

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32

Hausenloy, Derek, and Derek Yellon, eds. Coronary No-Reflow and Microvascular Obstruction. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199544769.003.0005.

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• Following an AMI, the restoration of TIMI III coronary blood flow using thrombolytic therapy or primary percutaneous coronary intervention does not guarantee actual myocardial perfusion• In 40–60% of reperfused AMI cases, myocardial perfusion is impeded at the level of the capillaries due to microvascular obstruction (MVO)- a phenomenon termed coronary no-reflow• The presence of coronary no-reflow can be detected as impaired myocardial perfusion using non-invasive imaging modalities such as nuclear myocardial perfusion scanning, myocardial contrast echocardiography or contrast-enhanced cardi

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33

Kaufmann, W., K. J. Zülch, and Konstantin-A. Hossmann. Brain and Heart Infarct: Proceedings of the Third Cologne Symposium, June 16-19 1976. Springer London, Limited, 2012.

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34

Acute Myocardial Infarction: Setting Priorities for Effectiveness Research. National Academies Press, 1990.

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35

Reffelmann, Thorsten, and Robert Kloner. Adjunctive Reperfusion Therapy Post-AMI. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199544769.003.0009.

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• Reperfusion of the occluded coronary artery in an ST-segment-elevation myocardial infarction is the most effective approach for reducing infarct size, preserving left ventricular ejection fraction, lowering the incidence and severity of congestive heart failure and improving prognosis• Hence, several pharmacologic agents intended to improve target vessel patency as an adjunct to thrombolysis or primary percutaneous coronary intervention have been shown to be beneficial in patients with reperfusion therapy for acute myocardial infarction, namely antiplatelet and anticoagulation agents• Animal

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36

Acute Myocardial Infarction: Setting Priorities for Effectiveness Research (Publication Iom). National Academies Press, 1990.

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37

Downey, James, and Michael Cohen. Endogenous Mechanisms of Cardioprotection. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199544769.003.0008.

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• Ischaemic preconditioning is the most powerful endogenous mechanism for limiting myocardial infarct size in the experimental setting. Its clinical application is limited to scenarios in which the index episode of ischaemia and reperfusion can be anticipated such as in the setting of cardiac surgery• Ischaemic postconditioning represents an endogenous cardioprotective strategy which is applied at the onset of myocardial reperfusion, thereby allowing its use as an adjunct to reperfusion in patients presenting with an acute myocardial infarction• Both ischaemic preconditioning and postcondition

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38

Heusch, Gerd, ed. Pathophysiology And Ratioal Pharmacotherapy Of Myocardial Ischemia. Springer, 1990.

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39

Lear, Martha Weinman. Heartsounds: The Story of a Love and Loss. Open Road Integrated Media, Inc., 2014.

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40

Dawson, Dana, and Keith Fox. Anti-Platelet and Anti-Thrombotic Therapy Post-AMI. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199544769.003.0004.

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• Acute coronary syndromes (ACS) encompass a spectrum of presentations which include unstable angina, non-ST-elevation myocardial infarction (NSTEMI or NSTE-ACS), and ST-elevation myocardial infarction (STEMI or STE-ACS)• Anti-platelet and anti-thrombotic agents are administered as ancillary therapy to myocardial reperfusion in patients presenting with an acute coronary syndrome, to maintain the patency of the infarct-related coronary artery• More specific and potent inhibitors of platelet activation and of the coagulation cascade are emerging with the aim being to further improve clinical out

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41

Honda, Michelle. Reverse Heart Disease Naturally: Cures for high cholesterol, hypertension, arteriosclerosis, blood clots, aneurysms, myocardial infarcts and more. Hatherleigh Press, 2017.

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42

Heartsounds: The Story of a Love and Loss. Open Road Integrated Media, Inc., 2014.

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43

Heartsounds: The Story of a Love and Loss. Open Road Integrated Media, Inc., 2014.

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44

Zaret, Barry L. Nuclear Cardiology. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0001.

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Nuclear cardiology is generally considered a clinical phenomenon of the past four decades. However, the field has its roots in earlier times. This chapter focuses on these historical roots as they have evolved into the present era. The initial application of radioisotopes to cardiac studies occurred in the mid-1920s. Ventricular function was evaluated in the 1960s and 1970s by first pass and equilibrium techniques. Myocardial stress perfusion imaging was first performed using potassium-43 and exercise in 1973. Stress imaging rapidly evolved thereafter with new tracers (thallium-201 and technet

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45

Evidence-Based Management of the Acute Coronary Syndrome. Hanley & Belfus, 2001.

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46

AlJaroudi, Wael. Risk Assessment in Acute Coronary Syndromes. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0013.

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Acute coronary syndromes (ACS) include unstable angina pectoris (UAP), non-ST elevation (NSTEMI), and ST elevation acute myocardial infarction (STEMI). Each year, more than 2 million people are hospitalized with ACS in the United States. The initial treatment has evolved over the last few decades from conservative management to early reperfusion therapy. Medical therapy has also significantly changed with the use of newer more potent antiplatelet agents, beta-blockers, angiotensin converting enzyme inhibitors, statins, and anti-anginal drugs, which have resulted in improvement of patient care

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47

Opie, Lionel. Optimal Medical Therapy Post-AMI. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199544769.003.0006.

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• The management of an acute myocardial infarction can be divided into four phases: (a) The initial acute ischaemia causes severe prolonged chest pain when the patient is rushed to a Coronary or Intensive Care Unit; (b) Within the next few hours as ischaemia changes into infarction, the aim at this step is to restore blood flow in the occluded artery by thrombolysis or by percutaneous coronary intervention (PCI); (c) Next, the infarct is established and the left ventricle undergoes early remodeling; (d) Finally, follows the post-AMI post-hospital phase when continued left ventricular remodelin

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48

Connaughton, Mark. Evidence-Based Coronary Care. Churchill Livingstone, 2001.

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49

Frank, Arthur W. At the Will of the Body: Reflections on Illness. Houghton Mifflin Company, 1992.

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50

Meltzer, Lawrence E. Intensive Coronary Care. 4th ed. Appleton and Lange, 1989.

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