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Relevant bibliographies by topics / Infertility, Male - Genetic aspects

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Academic literature on the topic 'Infertility, Male - Genetic aspects'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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Journal articles on the topic "Infertility, Male - Genetic aspects"

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Druzhinina, N. K., and E. А. Korovyakova. "MALE INFERTILITY. GENETIC ASPECTS." Bulletin "Biomedicine and sociology" 3, no. 4 (December 30, 2018): 49–51. http://dx.doi.org/10.26787/nydha-2618-8783-2018-3-4-49-51.

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Witczak, Bartosz, Justyna E. Klusek, and Jolanta Klusek. "Genetic aspects of male infertility." Medical Studies 4 (2014): 276–79. http://dx.doi.org/10.5114/ms.2014.47928.

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Namiki, Mikio. "Genetic Aspects of Male Infertility." World Journal of Surgery 24, no. 10 (October 2000): 1176–79. http://dx.doi.org/10.1007/s002680010198.

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Pizzol, Damiano, Alessandro Bertoldo, and Carlo Foresta. "Male infertility: biomolecular aspects." Biomolecular Concepts 5, no. 6 (December 1, 2014): 449–56. http://dx.doi.org/10.1515/bmc-2014-0031.

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AbstractMale infertility is a problem that faces increasing interest, and the continuous development of assisted reproduction techniques solicits attempts to identify a precise diagnosis, in particular for idiopathic infertile couples and those undergoing assisted reproductive technique cycles. To date, diagnosis of male infertility is commonly based on standard semen analysis, but in many cases, this is not enough to detect any sperm abnormality. A better understanding of biomolecular issues and mechanism of damaged spermatogenesis and the refinement of the molecular techniques for sperm evaluation and selection are important advances that can lead to the optimization of diagnostic and therapeutic management of male and couple infertility. Faced with a growing number of new proposed techniques and diagnostic tests, it is fundamental to know which tests are already routinely used in the clinical practice and those that are likely to be used in the near future. This review focuses on the main molecular diagnostic techniques for male infertility and on newly developed methods that will probably be part of routine sperm analysis in the near future.

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Cinar, C., C. Beyazyurek, G. Ozgon, S. Ozkan, B. Ismailoglu, O. Oner, F. Fiorentino, and S. Kahraman. "4.002 Genetic aspects of male infertility in assisted reproduction." Reproductive BioMedicine Online 16 (January 2008): s38. http://dx.doi.org/10.1016/s1472-6483(10)61385-5.

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Heidary, Zohreh, Kioomars Saliminejad, Majid Zaki-Dizaji, and Hamid Reza Khorram Khorshid. "Genetic aspects of idiopathic asthenozoospermia as a cause of male infertility." Human Fertility 23, no. 2 (September 9, 2018): 83–92. http://dx.doi.org/10.1080/14647273.2018.1504325.

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7

Vicdan, Arzu, Kubilay Vicdan, Serdar Günalp, Aykut Kence, Cem Akarsu, Ahmet Zeki Işık, and Eran Sözen. "Genetic aspects of human male infertility: the frequency of chromosomal abnormalities and Y chromosome microdeletions in severe male factor infertility." European Journal of Obstetrics & Gynecology and Reproductive Biology 117, no. 1 (November 2004): 49–54. http://dx.doi.org/10.1016/j.ejogrb.2003.07.006.

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Yong, EL, Q. Wang, TG Tut, FJ Ghadessy, and SC Ng. "Male infertility and the androgen receptor: molecular, clinical and therapeutic aspects." Reproductive Medicine Review 6, no. 2 (July 1997): 113–31. http://dx.doi.org/10.1017/s0962279900001459.

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Idiopathic male infertility has previously been diagnosed imprecisely, and has been treated using regimes that are not based on a clear understanding of the underlying pathophysiology; however, this is gradually changing, and a more rational approach is being adopted. Testosterone and its metabolite, DHT, is allimportant for the maintenance of sperm production and this has led us to examine the AR for causes of male infertility. Some, but not all, androgen-binding studies have indicated that in a certain proportion of cases of male infertility, defective androgen binding occurs. The cloning of the AR gene allowed for a more rigorous examination of the molecular pathogenesis which turned out to be both subtle and heterogeneous. Genetic screening of a large group of men with defective spermatogenesis has indicated that up to 30% of infertile males could have variations in the androgenicity of their AR caused by polymorphisms in the length of the polyglutamine tract. Substitutions of the AR in the LBD and the DBD can also lead to reduced AR function and male infertility. In this regard, it is interesting to note that depressed spermatogenesis and prostate cancer represent opposite ends of the spectrum of AR action (Figure 6). Although empirical treatment of AR mutants in some cases has been shown to restore normal AR function and to improve spermatogenesis, a fully rational basis of treatment has to be based on an understanding of the crystallographic structure of the AR LBD. A full understanding could lead to the construction and the administration of ‘designer’ androgen analogues to treat male infertility caused by mutations of the AR gene.

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Mikhaylenko, D. S., I. Yu Sobol, E. A. Efremov, O. I. Apolikhin, A. S. Tanas, B. Ya Alekseev, and M. V. Nemtsova. "Genetic forms of male infertility: main characteristics and practical aspects of laboratory diagnostics." Experimental and Сlinical Urology 12, no. 1 (March 20, 2020): 96–104. http://dx.doi.org/10.29188/2222-8543-2020-12-1-96-104.

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Puzuka, Agrita, Baiba Alksere, Linda Gailite, and Juris Erenpreiss. "Idiopathic Infertility as a Feature of Genome Instability." Life 11, no. 7 (June 29, 2021): 628. http://dx.doi.org/10.3390/life11070628.

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Genome instability may play a role in severe cases of male infertility, with disrupted spermatogenesis being just one manifestation of decreased general health and increased morbidity. Here, we review the data on the association of male infertility with genetic, epigenetic, and environmental alterations, the causes and consequences, and the methods for assessment of genome instability. Male infertility research has provided evidence that spermatogenic defects are often not limited to testicular dysfunction. An increased incidence of urogenital disorders and several types of cancer, as well as overall reduced health (manifested by decreased life expectancy and increased morbidity) have been reported in infertile men. The pathophysiological link between decreased life expectancy and male infertility supports the notion of male infertility being a systemic rather than an isolated condition. It is driven by the accumulation of DNA strand breaks and premature cellular senescence. We have presented extensive data supporting the notion that genome instability can lead to severe male infertility termed “idiopathic oligo-astheno-teratozoospermia.” We have detailed that genome instability in men with oligo-astheno-teratozoospermia (OAT) might depend on several genetic and epigenetic factors such as chromosomal heterogeneity, aneuploidy, micronucleation, dynamic mutations, RT, PIWI/piRNA regulatory pathway, pathogenic allelic variants in repair system genes, DNA methylation, environmental aspects, and lifestyle factors.

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Dissertations / Theses on the topic "Infertility, Male - Genetic aspects"

1

Wong, Yee-man Elaine, and 王怡雯. "Identification and characterization of VCY2 interacting proteins." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B31228008.

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Harris, Tanya Penina. "Genetic analysis of male infertility." Thesis, University of Canterbury. Biological Sciences, 2004. http://hdl.handle.net/10092/6545.

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Approximately one in twenty men has impaired spermatogenesis due to mutation of genes involved in the establishment or maintenance of fertility. Our understanding of male infertility is complicated by the variable phenotypes produced by similar genetic changes, largely due to the practise of screening a single fertility gene in isolation. This thesis aimed to increase our understanding of the role of synergistic mutations in relation to differences in semen quality. Each sample was analysed for mutation in: CAG trinucleotide repeat variation in the X-linked androgen receptor (AR) gene, micro deletion within the three Y chromosome azoospermic factor (AZF) regions, and CAG trinucleotide repeat variation and exonuclease domain mutation in the nuclear polymerase gamma (POLγ gene. These genes have been associated with reduced semen quality in past research. Each gene region was amplified by polymerase chain reaction (PCR), followed by sequencing. Suspected AZF micro-deletions were confirmed by Southern blot hybridisation. Associations with semen quality were evaluated using either a t-test or Gtest for independence at α=0.05. Yq AZF micro-deletions were observed in 6.6% (14/211) of men with poor semen quality but not in normozoospermic samples (0/104); P<0.001). Micro-deletion frequency was greatest in azoospermic and severely oligoasthenozoospermic individuals (15% and 11.5%, respectively). AR CAG repeat length ranged from 9-38 CAG repeats in the normozoospermic population (n=98) and 13-31 CAG repeats in men with poor semen quality (n= 119). Variation in AR CAG trinucleotide repeat number was not significantly related to poor semen quality (P>0.05). Variation in POLγ CAG repeat number was not significantly different between normozoospermic men (n=93) and men with poor semen quality (n= 182); P>0.05. No nucleotide changes were observed in any of the three POLγ exonuclease motifs (n=83 normozoospermic and 191 non-normozoospermic motif, 61 and 65 motif II, and 60 and 64 motif III). Although most gene regions did not show an association with poor semen quality on their own, there was a general trend towards greater severity of impaired spermatogenesis with the presence of both Yq micro-deletion and mitochondrial DNA substitutions or moderately expanded AR CAG repeats. These results support the idea that male infertility is a complex process, due to many factors, some of which act dominantly and others act in concert.

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Lloyd, Michael. "Aspects of the social organisation of "male infertility"." Thesis, University of Canterbury. Sociology, 1994. http://hdl.handle.net/10092/6527.

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This dissertation revolves around three main elements: 'male infertility'; existing social science research on infertility; and ethno methodology. The substantive topic 'male infertility' is enclosed in quotation marks for two reasons. First, following the overall form of ethno methodological inquiry, the aim is to explicate how the sense and order of 'male infertility' is constituted through available socially organised procedures; hence, the quotation marks are used to 'bracket' the phenomenon and focus on the methods that make it available. Second, 'male infertility' is a convenient shorthand topic label, a general organising concept, as opposed to a precise label for a tightly defined phenomenon. While this study's approach makes it very different to existing sociological studies of infertility, the difference is not to the extent of isolation - a strong attempt is made to engage with prior studies. Often this engagement takes a critical form, the general argument being that sociological studies which approach phenomena for the way they 'bear the marks' of societal structures, will ignore the incarnate orderliness of social action - that is, the way social action is readily explicable to members, in and as it occurs, using the resources at-hand, with 'no time-out'. Ethno methodology suggests that this ready explicability is based upon taken-for-granted, socially organised sense-assembly practices - thus, this study's argument that the content, the intelligibility of 'male infertility is interdependent with the social scenes and embedded socially organised procedures, with and within which 'male infertility' is found. Form and content stand or fall together. Consistent with this viewpoint, four empirical analyses of the social organisation of 'male infertility' are offered. The specific topics discussed are: the conversational disclosure of infertility; the language of reproduction; humour and infertility; and high rates of non-response by men in studies of infertility. In general, the empirical analyses are 'indifferent' to the topic of study, that is, there is no overriding aim of offering practical correctives or broader socio-political critique. However, in at least one empirical chapter a more critical stand is taken, and, in the concluding chapter, it is argued that an ethno methodological descriptivist approach can have socio-political implications. Overall, the study supports the growing trend for ethno methodological insights to be utilised in the study of substantive topics; and, since the dissertation is a form of writing 'anew', it can be considered to minimally change 'male infertility' as a form of life.

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Dumargne, Marie-Charlotte. "Genetic and epigenetic factors associated with human male infertility." Thesis, Paris 6, 2016. http://www.theses.fr/2016PA066068.

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La spermatogenèse est un processus complexe qui dépend de la coopération de nombreux gènes. Son produit final le spermatozoïde, est un sujet d’étude idéal car il renferme à la fois des indices d’événements passés ainsi que des informations qui seront transmises à l'ovocyte lors de la fécondation. L'identification de nouveaux acteurs de la spermatogenèse, des modifications spécifiques de l'ADN du sperme ou la présence de transcrits spécifiques pourraient servir comme biomarqueurs dans le diagnostic de l’infertilité. Cette thèse avait pour but d’analyser le génome, le transcriptome et l’épigénome de spermatozoïdes dans le contexte de l'infertilité masculine. Nous avons identifié de nouvelles causes génétiques et confirmé la présence d'anomalies de méthylation dans le sperme d'hommes infertiles. Nous avons découvert 20 mutations dans le gène SOX8, chez des patients atteints de trouble du développement sexuel ou d'infertilité masculine ou féminine, qui apparaît comme un régulateur du développement et de la fonction gonadique. Par séquençage d’exome, une mutation dans le gène ATAD2 modeleur de la chromatine spécifique de la lignée germinale mâle fut également identifiée. Par RNA-seq et MeDIP-chIP du sperme d’hommes fertiles et infertiles, nous avons caractérisé la signature transcriptionnelle du sperme. La majorité des ARNs spermatiques humain est remarquablement conservée chez les mammifères placentaires suggérant des fonctions ancestrales importantes. Enfin, nos données transcriptomiques et épigénétiques tendent à indiquer qu’une expression et une régulation adéquates des gènes impliqués dans le remodelage de la chromatine constituent un facteur clé pour la fertilité masculine
Spermatogenesis is a complex process which depends on the cooperation of many genes. The end-product, the spermatozoon, is an ideal subject for study since it carries both clues of the past events and information which will be transmitted to the oocyte at fertilization. The identification of main actors of spermatogenesis, specific modifications of sperm DNAs or sperm specific isoforms could improve our understanding of a such complex mechanism and could serve as a determination of biomarkers or diagnostic tools for fertility. The aim of the project was to go further three omes: genome, epigenome and transcriptome of mature human sperm in the context of male infertility. We identified new genetic causes of male infertility and confirmed the presence of methylation abnormalities in sperm cells of infertile men. Firstly, SOX8 gene was found mutated in a cohort of 20 patients with disorder of sex development and male or female infertility. Similarly, to NR5A1, SOX8 appears to be a novel regulator of gonadal development and function. Then by exome-sequencing, we identified a homozygous nonsense mutation in the male germline-specific chromatin modeler ATAD2. Furthermore, RNA-seq and MeDIP-chIP of sperm from fertile and infertile men along with bioinformatics analyzes of the generated data, enabled us to characterize more deeply the normal sperm transcriptional signature. We also found that the majority of human sperm RNAs are remarkably preserved in placental mammals suggesting crucial ancestral functions. Finally, proper expression and regulation of chromatin remodelers seem to be critical for male fertility, as revealed by both the transcriptomic and the epigenetic data

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Okutman, Özlem. "Genetics of male infertility : genes implicated in non-obstructive azoospermia and severe oligozoospermia." Thesis, Strasbourg, 2015. http://www.theses.fr/2015STRAJ049/document.

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Parmi les couples avec un projet parental, le facteur masculin d’infertilité est responsable d’environ 20%. Malgré de longues années d’activités d’assistance médicale à la procréation, un nombre important de cas reste idiopathiques. Considérant le nombre élevé des gènes potentiellement impliqués dans la gamétogenèse, il est fort probable que la majorité des formes ‘idiopathiques’ sont d’origine génétique. Dans l'étude présente, nous avons d’identifier deux nouveaux gènes impliqués dans une infertilité masculine. Nos données suggèrent que la mutation dans TEX15 puisse corréler avec une diminution du nombre de spermatozoïdes au fil du temps. Un test diagnostique identifiant la mutation chez un patient pourrait fournir une indication d’organiser au plus tôt une cryopréservation du sperme. On a aussi identifié MAGEB4 liées à l’X comme un nouveau gène impliqué dans une infertilité masculine héritée. Cette étude fournit le premier indice sur la fonction physiologique d'une protéine MAGE
Among couples with a desire for a child, male factor is responsible approximately 20%. Despite long years of assisted reproductive activities, a significant number of cases remain idiopathic. Considering the high predicted number of genes involved in male gametogenesis, it is likely that most ‘idiopathic’ forms may have a genetic origin. In the present study, we have defined two new genes implicated in male infertility. Our data suggested that a nonsense mutation in TEX15 correlates with a decrease in sperm count over time. A diagnostic test identifying the mutation in man could provide an indication of spermatogenic failure and prompt patients to undertake sperm cryopreservation at an early age. We also identified MAGEB4 as a new X-linked gene involved in an inherited male infertility. This study provides the first clue on the physiological function of a MAGE protein

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Lee, Kyeong Hee. "Cytogenetic and histological aspects of male infertility in the Japanese quail (Coturnix coturnix japonica) /." The Ohio State University, 1990. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487681148541197.

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Fouche, Anna Aletta. "Genetic predisposition to DTT-induced DNA decondensation." Diss., University of Pretoria, 2006. http://upetd.up.ac.za/thesis/available/etd-05102007-130900.

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Khan, Sadia Jihan. "Mitochondrial ND Genes: Relevance of Codon Usage to Semen Quality in Men." Thesis, University of Canterbury. Biological Sciences, 2006. http://hdl.handle.net/10092/1434.

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Studies have discovered higher frequencies of single nucleotide polymorphisms (SNPs) in different mitochondrial genes are associated with subnormozoospermia. However, the frequencies of SNPs in ND1 and ND2 are not unknown. The present research was aimed to determine the frequencies of SNPs in ND1 and ND2 genes of the mitochondrial genome in fertile and subfertile men and whether changes in codon usage was associated with fertility phenotypes. Total genomic DNA from 157 semen samples was extracted using the proteinase K/SDS digestion procedure, followed by phenol/chloroform purification and ethanol precipitation. ND1 and ND2 genes were amplified respectively from 80 and 92 DNA samples from different fertility groups. Each PCR product was sequenced to identify mutations. Codon change resulting from a nucleotide substitution was determined by comparison with a reference mtDNA sequence obtained from the NCBI database. The frequency of codon usage in the reference mtDNA was determined by the computer program MEGA version 2.1. Eleven synonymous nucleotide substitutions and two non-synonymous substitutions were found in this study. Four SNPs were previously characterized; all SNPs were homoplasmic. None of the SNPs were likely to affect the function of the proteins on the basis of the hydrophobicity plots or secondary structure predictions. Sixty two percent of synonymous mutations were found to change from a high to a low relative codon usage values; 37% of synonymous mutations changed from a low to a high relative usage value. Chi-square (χ²) test (χ²= 0.067 with 1 d.f.) showed that there was no significant difference at the 5% level between these changes. Thus, change in codon usage was not related to semen quality in men. Further, there were no statistically significant differences in the observed frequencies of SNPs of fertile and subfertile men. However, the sample size was small and this study was only focused on a single NZ Caucasian population. Further study including larger and more diverse population samples may provide further insight into the functional importance of codon usage and its relevance to fertility

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Ghasemi, Behrooz. "Evaluation of Morphological and Other Aspects of male Infertility in Testicilar Neoplasia using Lectin Histochemistry and Semen Analysis." Thesis, University of Manchester, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.492890.

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It has been estimated that approximately 15% of couples attempting to attain pregnancy are unable to do so. A female factor is the primary aetiology in nearly 40% of these couples and in another 30% the factors are purely male. A combmation of male and female factors has been implicated for the remaining 30% of cases. Infertility is a common problem among men with testicular cancer. Such men are especially affected by infertility and they often decide to undergo sperm banking (the collection and freezing of sperm) before beginning chemo/radiotherapy.

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Ellis, P. J. I. "Microarray expression profiling of mouse postnatal testis development and genetic models of infertility using a novel male gonadal geneset." Thesis, University of Cambridge, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598827.

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Microarray technology holds great promise for allowing the global analysis of the testicular transcriptome. The aim of this project is to demonstrate the feasibility and utility of the microarray approach to the study of the spermatogenesis. This thesis details the construction, validation and characterisation of two subtracted cDNA libraries, and their use in microarray analysis of several different models of murine-spermatogenesis. These models consist of the postnatal first wave of spermatogenesis, a series of germ-cell depleted mutant models, a series of models with deletions of the Y chromosome long arm (Yq) and the TM4 juvenile Sertoli cell line under stimulation with follicle stimulating hormone (FSH) and dihydrotestosterone (DHT). Analysis of the first wave has been used to develop a framework of expression patterns associated with different germ cell types within which to analyse other models, allowing better characterisation of the spermatogenic arrest in the germ cell depleted models. Candidate genes (including Xmr, mgclh and the unknown EST AK0059221) have been discovered with regard to the morphological abnormalities exhibited by the spermatozoa in the Yq deletion models. Microarray analysis also leads to a better understanding of the nature of the TM4 cell line, specifically how well it can be said to demonstrate the range of responses of normal Sertoli cells. Selected gene profiles of interest have been validated in this laboratory and others using established techniques of real-time RT-PCR and Northern blotting. The success of microarray technology in analysing these models demonstrates its utility for the study of spermatogenesis, and the utility of the constructed libraries in further microarray experiments. A fuller understanding of genetic events during spermatogenesis may enable therapeutic intervention in cases of male infertility, or yield insights leading to development of novel contraceptives, as well as casting light on the fundamental processes of mitosis, meiosis, fate commitment, cell differentiation and apoptosis.

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Books on the topic "Infertility, Male - Genetic aspects"

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R, Barratt C. L., and Institut national de la santé et de la recherche médicale (France), eds. Genetics of human male fertility: Proceeding [sic] of the International Symposium on "Genetics of Human Male Fertility," held in Collioure, France, 4-6 September 1997. Sèvres: EDK, 1997.

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Ashok, Agarwal, and SpringerLink (Online service), eds. Sperm Chromatin: Biological and Clinical Applications in Male Infertility and Assisted Reproduction. New York, NY: Springer Science+Business Media, LLC, 2011.

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McElreavey, Ken, ed. The Genetic Basis of Male Infertility. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-540-48461-5.

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Male infertility--men talking. London: Routledge, 1993.

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When a husband is infertile: Options for the Christian couple. Grand Rapids, Mich: Baker Books, 1994.

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Snarey, John R. How husbands cope when pregnancy fails: A longitudinal study of infertility and psychosocial generativity. Wellesley, Mass: Wellesley College. Center for Research on Women, 1986.

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Rand, Victoria. Healing gourmet, eat to boost fertility. New York: McGraw-Hill, 2006.

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Not yet pregnant: Infertile couples in contemporary America. New Brunswick [N.J.]: Rutgers University Press, 1991.

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Infertility journeys: Finding your happy ending. Encinitas, CA: Duck Hill Press, 2011.

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Male reproductive cancers: Epidemiology, pathology and genetics. New York: Springer, 2010.

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Book chapters on the topic "Infertility, Male - Genetic aspects"

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Prokai, David, and Orhan Bukulmez. "Genetic Aspects of Male Infertility." In Male Infertility, 147–64. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-32300-4_11.

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Bukulmez, Orhan. "Genetic Aspects of Male Infertility." In Male Infertility, 171–89. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-3335-4_18.

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Bukulmez, Orhan. "Genetic Aspects of Male Infertility." In Male Infertility for the Clinician, 61–94. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7852-2_4.

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Oates, Robert D. "Genetic Issues with Male Fertility." In Male Infertility, 39–45. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-193-6_5.

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Mahon, Joseph Thomas, and Nicholas N. Tadros. "Endocrine Genetic Defects." In Genetics of Male Infertility, 301–12. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-37972-8_19.

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Miyaoka, Ricardo, and Sandro C. Esteves. "Genetic Basis of Unexplained Male Infertility." In Unexplained Infertility, 57–70. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2140-9_8.

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Fakhro, Khalid A., Amal Robay, Juan L. Rodriguez-Flores, and Ronald G. Crystal. "Genetic Evaluation of Male Infertility." In Genetics of Male Infertility, 95–118. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-37972-8_6.

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Tannour-Louet, Mounia, and Dolores J. Lamb. "Genetic Variants in Male Infertility." In Biennial Review of Infertility, 113–27. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-392-3_8.

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Krausz, Csilla. "Genetic Testing of Male Infertility." In Reproductive Endocrinology and Infertility, 431–44. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-1436-1_27.

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Krausz, Csilla. "Genetic Analysis in Male Infertility." In Endocrinology, 1–17. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-29456-8_17-1.

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