Relevant bibliographies by topics / Inflammasom

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Inflammasom'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 February 2022

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Journal articles on the topic "Inflammasom"

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Uchima, Miki, and Eicke Latz. "Inflammasom Signaling und chronische Entzündungsreaktionen." Trillium Immunologie 5, no. 1 (2021): 54–60. http://dx.doi.org/10.47184/ti.2021.01.06.

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Inflammasome sind Multiproteinkomplexe, die typischerweise aus drei Proteinentitäten – einem Sensor, einem Adaptor und Caspase 1 – bestehen. Sie werden als Antwort auf die Erkennung von Pathogen-assoziierten molekularen Strukturen (PAMPs) oder Gefahren-assoziierten molekularen Strukturen (DAMPs) gebildet. Eine Schlüsselrolle im Inflammasom-Signalweg spielt dabei das Zymogen Pro-Caspase 1, das zunächst selbst durch Autoprozessierung aktiviert werden muss. Aktive Caspase 1 prozessiert die Vorstufen der pro-inflammatorischen Interleukine (IL) IL-1beta (IL-1β) und IL-18, die daraufhin sekretiert w
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Aringer, M., C. Hedrich, and A. K. Tausche. "Inflammasom und Gicht." Zeitschrift für Rheumatologie 75, no. 6 (2016): 537–41. http://dx.doi.org/10.1007/s00393-016-0131-1.

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Cordero, Mario D., and Jesús Ruiz-Cabello. "Inflamm-ageing or inflammasom-ageing as independent events." Aging 12, no. 18 (2020): 17759–60. http://dx.doi.org/10.18632/aging.104108.

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Hofmann, S. R., N. Bruck, G. Hahn, A. Rösen-Wolff, and C. M. Hedrich. "Systemisch inflammatorische Erkrankungen als Differenzialdiagnose in der Neugeborenenperiode – Inflammasom-vermittelte Erkrankungen." Arthritis und Rheuma 38, no. 02 (2018): 125–31. http://dx.doi.org/10.1055/s-0038-1649292.

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ZusammenfassungAutoinflammatorische Erkrankungen sind durch systemische oder organbezogene Entzündung gekennzeichnet, welche zumindest initial von fehlregulierten Mechanismen des angeborenen Immunsystems ausgehen. Einige monogene autoinflammatorische Erkrankungen manifestieren in der Perinatalperiode, sodass sie Differenzialdiagnosen zu Infektionen des Neugeborenen darstellen. Im vorliegenden Artikel werden Einblicke in die klinische Präsentation und die molekulare Pathophysiologie Inflammasom-vermittelter monogener Erkrankungen mit Manifestation im Neugeborenenalter gegeben.
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Manigold, Tobias. "Ursachen, Mechanismen und mögliche therapeutische Ziele der Gicht." Therapeutische Umschau 73, no. 3 (2016): 147–52. http://dx.doi.org/10.1024/0040-5930/a000771.

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Zusammenfassung. Die Gicht ist die häufigste Arthritis weltweit mit zunehmender Prävalenz in den entwickelten Ländern und mit enormen sozioökonomischen Konsequenzen. Das Wissen über die Pathomechanismen, welche zur Arthritis führen und diese einschränken, hat in den letzten Jahren stark zugenommen. Entsprechend zeichnen sich auch neue Therapieansätze und Therapeutika ab. In dieser Übersichtsarbeit werden neben einigen Aspekten der Klinik, Diagnose und Therapie der Gicht, detailliert die intrazellulären Mechanismen, welche zur NLRP3 Inflammasom Aktivierung und IL-1beta Sekretion führen, behande
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Inoue, Makoto, and Mari L. Shinohara. "NLRP3 Inflammasome and MS/EAE." Autoimmune Diseases 2013 (2013): 1–8. http://dx.doi.org/10.1155/2013/859145.

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Inflammasomes are cytosolic sensors that detect pathogens and danger signals in the innate immune system. The NLRP3 inflammasome is currently the most fully characterized inflammasome and is known to detect a wide array of microbes and endogenous damage-associated molecules. Possible involvement of the NLRP3 inflammasome (or inflammasomes) in the development of multiple sclerosis (MS) was suggested in a number of studies. Recent studies showed that the NLRP3 inflammasome exacerbates experimental autoimmune encephalomyelitis (EAE), an animal model of MS, although EAE can also develop without th
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Wagatsuma, Kohei, and Hiroshi Nakase. "Contradictory Effects of NLRP3 Inflammasome Regulatory Mechanisms in Colitis." International Journal of Molecular Sciences 21, no. 21 (2020): 8145. http://dx.doi.org/10.3390/ijms21218145.

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The inflammasome is an intracellular molecular complex, which is mainly involved in innate immunity. Inflammasomes are formed in response to danger signals, associated with infection and injury, and mainly regulate the secretion of interleukin-1β and interleukin-18. Inflammasome dysregulation is known to be associated with various diseases and conditions, and its regulatory mechanisms have become of great interest in recent years. In the colon, inflammasomes have been reported to be associated with autophagy and the microbiota, and their dysregulation contributes to colitis and. However, the d
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Spalinger, Marianne R., Marlene Schwarzfischer, and Michael Scharl. "The Role of Protein Tyrosine Phosphatases in Inflammasome Activation." International Journal of Molecular Sciences 21, no. 15 (2020): 5481. http://dx.doi.org/10.3390/ijms21155481.

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Inflammasomes are multi-protein complexes that mediate the activation and secretion of the inflammatory cytokines IL-1β and IL-18. More than half a decade ago, it has been shown that the inflammasome adaptor molecule, ASC requires tyrosine phosphorylation to allow effective inflammasome assembly and sustained IL-1β/IL-18 release. This finding provided evidence that the tyrosine phosphorylation status of inflammasome components affects inflammasome assembly and that inflammasomes are subjected to regulation via kinases and phosphatases. In the subsequent years, it was reported that activation o
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Forn-Cuní, Gabriel, Annemarie H. Meijer, and Monica Varela. "Zebrafish in Inflammasome Research." Cells 8, no. 8 (2019): 901. http://dx.doi.org/10.3390/cells8080901.

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Inflammasomes are cytosolic multiprotein complexes that regulate inflammatory responses to danger stimuli and infection, and their dysregulation is associated with an increasing number of autoinflammatory diseases. In recent years, zebrafish models of human pathologies to study inflammasome function in vivo have started to emerge. Here, we discuss inflammasome research in zebrafish in light of current knowledge about mammalian inflammasomes. We summarize the evolutionary conservation of inflammasome components between zebrafish and mammals, highlighting the similarities and possible divergence
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Yi, Young-Su. "Caspase-11 Non-Canonical Inflammasome: Emerging Activator and Regulator of Infection-Mediated Inflammatory Responses." International Journal of Molecular Sciences 21, no. 8 (2020): 2736. http://dx.doi.org/10.3390/ijms21082736.

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Inflammation is a body’s protective mechanism to eliminate invading pathogens and cellular damaging signals. The inflammatory response consists of two main consecutive steps—a priming step preparing the inflammatory responses and a triggering step boosting the inflammatory responses. The main feature of the triggering step is the activation of the inflammasome, an intracellular multiprotein complex facilitating the inflammatory responses. The regulatory roles of ‘canonical’ inflammasomes in the inflammatory responses and diseases have been largely investigated, so far. New types of inflammasom
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Dissertations / Theses on the topic "Inflammasom"

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Prager, Philipp, Margrit Hollborn, Anja Steffen, Peter Wiedemann, Leon Kohen, and Andreas Bringmann. "P2Y1 receptor signaling contributes to high salt-induced priming of the NLRP3 inflammasome in retinal pigment epithelial cells." Universitätsbibliothek Leipzig, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-216158.

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Background: Systemic hypertension is a risk factor of age-related macular degeneration (AMD), a chronic inflammatory disease. Acute hypertension is caused by increased extracellular osmolarity after intake of dietary salt (NaCl). We determined in cultured human retinal pigment epithelial (RPE) cells whether high extracellular NaCl alters the gene expression of inflammasome-associated proteins, and whether autocrine/paracrine purinergic (P2) receptor signaling contributes to the NaCl-induced NLRP3 gene expression. Methodology/Principal Findings: Hyperosmolarity was induced by the addition of 10
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Bscheider, Michael. "Inflammasom-Aktivierung durch RNA-Viren und Pilze." Diss., lmu, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-149967.

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Meixenberger, Karolin [Verfasser]. "Mechanismen der Inflammasom-Aktivierung durch Listeria monocytogenes / Karolin Meixenberger." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2010. http://d-nb.info/1024365433/34.

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Göß, Christine. "Zytosolische DNA aktiviert das AIM2-Inflammasom in Keratinozyten bei Psoriasis." Diss., lmu, 2013. http://nbn-resolving.de/urn:nbn:de:bvb:19-155286.

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Bscheider, Michael [Verfasser], and Gunther [Akademischer Betreuer] Hartmann. "Inflammasom-Aktivierung durch RNA-Viren und Pilze / Michael Bscheider. Betreuer: Gunther Hartmann." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2012. http://d-nb.info/1028325541/34.

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Wacker, Christian Friedrich [Verfasser]. "Das Nlrp3-Inflammasom verstärkt die diabetische Nephropathie in ortsständigen Zellen der Niere / Christian Friedrich Wacker." Magdeburg : Universitätsbibliothek, 2017. http://d-nb.info/1141230542/34.

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Göß, Christine [Verfasser], and Jürgen [Akademischer Betreuer] Schauber. "Zytosolische DNA aktiviert das AIM2-Inflammasom in Keratinozyten bei Psoriasis / Christine Göß. Betreuer: Jürgen Schauber." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2013. http://d-nb.info/1033504572/34.

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Bertram, Imke [Verfasser]. "Untersuchungen zum Inflammasom bei proinflammatorischem Zellstress des Muskels in vitro und in vivo / Imke Bertram." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2021. http://d-nb.info/123834545X/34.

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Sasu, Phillip Brenya [Verfasser]. "Einfluss von Influenza A-Viren auf die Inflammasom-abhängige Zytokinproduktion in der nachfolgenden Pneumokokkeninfektion / Phillip Brenya Sasu." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2015. http://d-nb.info/1075493110/34.

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Matschinski, Kathrin [Verfasser]. "Inflammasom - Aktivierung in murinen Makrophagen nach Infektion mit Burkholderia pseudomallei und deren Bedeutung für die Virulenz / Kathrin Matschinski." Greifswald : Universitätsbibliothek Greifswald, 2014. http://d-nb.info/1046647792/34.

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Books on the topic "Inflammasom"

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De Nardo, Christine M., and Eicke Latz, eds. The Inflammasome. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-523-1.

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Couillin, Isabelle, Virginie Pétrilli, and Fabio Martinon, eds. The Inflammasomes. Springer Basel, 2011. http://dx.doi.org/10.1007/978-3-0348-0148-5.

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Couillin, Isabelle. The Inflammasomes. Springer Basel AG, 2011.

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Dahle, Gro. Inflammasjon: Historier. Cappelen, 1995.

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Nardo, Christine De, and Eicke Latz. The inflammasome: Methods and protocols. Humana Press, 2013.

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Backert, Steffen, ed. Inflammasome Signaling and Bacterial Infections. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-41171-2.

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Cordero, Mario D., and Elísabet Alcocer-Gómez, eds. Inflammasomes: Clinical and Therapeutic Implications. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-89390-7.

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Amer, Amal O., ed. The inflammasom and its role in infections. Frontiers Media SA, 2011. http://dx.doi.org/10.3389/978-2-88919-002-7.

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Couillin, Isabelle, Virginie Pétrilli, and Fabio Martinon. The Inflammasomes. Springer, 2013.

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Backert, Steffen. Inflammasome Signaling and Bacterial Infections. Springer, 2018.

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Book chapters on the topic "Inflammasom"

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Gong, Tao, and Rongbin Zhou. "Inflammasome." In Encyclopedia of Molecular Pharmacology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-21573-6_10029-1.

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Inoue, Makoto, Masashi Kanayama, and Mari L. Shinohara. "Inflammasomes." In Encyclopedia of Inflammatory Diseases. Springer Basel, 2013. http://dx.doi.org/10.1007/978-3-0348-0620-6_77-1.

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Inoue, Makoto, Masashi Kanayama, and Mari L. Shinohara. "Inflammasomes." In Compendium of Inflammatory Diseases. Springer Basel, 2016. http://dx.doi.org/10.1007/978-3-7643-8550-7_77.

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Pétrilli, Virginie, and Fabio Martinon. "Molecular Definition of Inflammasomes." In The Inflammasomes. Springer Basel, 2011. http://dx.doi.org/10.1007/978-3-0348-0148-5_1.

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Apetoh, Lionel. "Cancer, Inflammasomes, and Adjuvanticity." In The Inflammasomes. Springer Basel, 2011. http://dx.doi.org/10.1007/978-3-0348-0148-5_10.

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Ryffel, Bernhard, Dieudonnée Togbe, and Isabelle Couillin. "Inflammasome and Lung Pathologies." In The Inflammasomes. Springer Basel, 2011. http://dx.doi.org/10.1007/978-3-0348-0148-5_11.

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Gattorno, Marco, and Anna Rubartelli. "Mechanisms of NLRP3 Inflammasome Activation in CAPS Patients." In The Inflammasomes. Springer Basel, 2011. http://dx.doi.org/10.1007/978-3-0348-0148-5_12.

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Hernandez, Juan-Carlos, Cherilyn M. Sirois, and Eicke Latz. "Activation and Regulation of the NLRP3 Inflammasome." In The Inflammasomes. Springer Basel, 2011. http://dx.doi.org/10.1007/978-3-0348-0148-5_13.

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Labbé, Katherine, and Maya Saleh. "Pyroptosis: A Caspase-1-Dependent Programmed Cell Death and a Barrier to Infection." In The Inflammasomes. Springer Basel, 2011. http://dx.doi.org/10.1007/978-3-0348-0148-5_2.

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Ulland, Tyler K., and Fayyaz S. Sutterwala. "Activation of the Inflammasome by Bacterial Pathogens." In The Inflammasomes. Springer Basel, 2011. http://dx.doi.org/10.1007/978-3-0348-0148-5_3.

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Conference papers on the topic "Inflammasom"

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Reichel, C., L. Mittmann, J. Schaubächer, et al. "Das NLRP3-Inflammasom steuert die Rekrutierung von neutrophilen Granulozyten zu HNSCC." In Abstract- und Posterband – 89. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Forschung heute – Zukunft morgen. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1640140.

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Sendler, M., C. van den Brandt, FU Weiss, et al. "Die Inflammasom Aktivierung in phagozytierenden Makrophagen reguliert die systemische Immunantwort und den Schweregrad in der akuten Pankreatitis." In Viszeralmedizin 2017. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1604895.

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Praktiknjo, M., S. Monteiro, J. Grandt, et al. "Distinkte Inflammasom-Aktivierung bei kompensierter und rekompensierter Leberzirrhose ist mit der Entwicklung von akut-auf-chronischem Leberversagen assoziiert." In Viszeralmedizin 2019. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1695368.

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Cox Jr, Ruan, Amanda Hodgkins, Prasanna Tamarapu Parthasarathy, et al. "Resolvin Inhibits The Cryopyrin/Nalp3 Inflammasome." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1428.

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Exline, Matthew C., Anasuya Sarkar, Beth Y. Besecker, Jennifer L. Hollyfield, Srabani Mitra, and Mark D. Wewers. "Inflammasome Constituents Released Via Microvesicles During Sepsis." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1378.

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SHAIK, RAHAMTHULLA, Narasaiah Kolliputi, and Aaron B. Waxman. "Activation Of Inflammasome In Pulmonary Arterial Hypertension." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4865.

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Shin, Jin Na, Elmoataz A. H. Abdel Fattah, Soyoung Ko, and N. T. Eissa. "Altered Proteastasis In Macrophages Induces Inflammasome Activation." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1966.

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Reichel, C., L. Mittmann, J. Schaubächer, et al. "The NLRP3 inflammasome regulates neutrophil trafficking to HNSCC." In Abstract- und Posterband – 89. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Forschung heute – Zukunft morgen. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1640141.

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Graustein, A., W. R. Berrington, K. J. Buckingham, et al. "The NLRP3 Inflammasome in the Cystic Fibrosis Macrophage." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2660.

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Sorrentino, Rosalinda, Michela Terlizzi, Pasquale Imitazione, et al. "Role of the inflammasome in idiopathic pulmonary fibrosis." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa2027.

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Reports on the topic "Inflammasom"

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Testa, Joseph R. Role of the Inflammasome in Asbestos-Induced Mesothelioma Formation. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada570536.

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Schmitz, Karl R. Regulation of the Inflammasome, a Modulator of Caspase-Mediated Cytokine Production. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada488588.

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Schmitz, Karl. Regulation of the Inflammasome, a Modulator of Caspase-1 Mediated Cytokine Production. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada517127.

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Schmitz, Karl R. Regulation of the Inflammasome, a Modulator of Caspase-1 Mediated Cytokine Production. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada509759.

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