Log in

Relevant bibliographies by topics / Inflammasom / Books

To see the other types of publications on this topic, follow the link: Inflammasom.

Books on the topic 'Inflammasom'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 19 books for your research on the topic 'Inflammasom.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

De Nardo, Christine M., and Eicke Latz, eds. The Inflammasome. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-523-1.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Couillin, Isabelle, Virginie Pétrilli, and Fabio Martinon, eds. The Inflammasomes. Springer Basel, 2011. http://dx.doi.org/10.1007/978-3-0348-0148-5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Couillin, Isabelle. The Inflammasomes. Springer Basel AG, 2011.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

Dahle, Gro. Inflammasjon: Historier. Cappelen, 1995.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

5

Nardo, Christine De, and Eicke Latz. The inflammasome: Methods and protocols. Humana Press, 2013.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

6

Backert, Steffen, ed. Inflammasome Signaling and Bacterial Infections. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-41171-2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Cordero, Mario D., and Elísabet Alcocer-Gómez, eds. Inflammasomes: Clinical and Therapeutic Implications. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-89390-7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Amer, Amal O., ed. The inflammasom and its role in infections. Frontiers Media SA, 2011. http://dx.doi.org/10.3389/978-2-88919-002-7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Couillin, Isabelle, Virginie Pétrilli, and Fabio Martinon. The Inflammasomes. Springer, 2013.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

Backert, Steffen. Inflammasome Signaling and Bacterial Infections. Springer, 2018.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

11

DNA Sensors and Inflammasomes. Elsevier, 2019. http://dx.doi.org/10.1016/s0076-6879(19)x0011-7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

12

Inflammasomes: Clinical and Therapeutic Implications. Springer, 2019.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

13

Cordero, Mario D., and Elísabet Alcocer-Gómez. Inflammasomes: Clinical and Therapeutic Implications. Springer, 2018.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

14

Mason, Angie. Inflammasomes: Mechanism of Action, Regulation and Role in Disease. Nova Science Publishers, Incorporated, 2016.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

15

Dalbeth, Nicola. Epidemiology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198748311.003.0003.

Full text

Abstract:

The aetiopathogenesis of gout is initiated by urate overproduction and uric acid under-excretion, leading to hyperuricaemia. Foods such as seafood, red meat, beer, and sugar-sweetened beverages contribute to overproduction. Under-excretion is mediated by renal and gut uric acid transporters such as SLC2A9, ABCG2, and URAT1. In hyperurcaemia, there is formation of monosodium urate (MSU) crystals in joints, with acute gouty arthritis mediated by the innate immune system occurring in response to these crystals. Factors such as urate concentration, proteins present in synovial fluid, temperature,

APA, Harvard, Vancouver, ISO, and other styles

16

Frenkel, Joost, and Hans R. Waterham. Mevalonate Kinase Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0039.

Full text

Abstract:

Mevalonate kinase deficiency (MKD) is an autosomal recessive inborn error of isoprenoid biosynthesis, a pathway yielding sterols and nonsterol isoprenoids.In patients, the enzyme activity of mevalonate kinase is severely reduced due to mutations in the encoding gene, MVK. The substrate, mevalonate, accumulates and is elevated in blood and urine. Shortage of certain downstream products of the pathway, nonsterol isoprenoids, leads to dysregulation of the innate immune system, activation of inflammasomes, and interleukin (IL)-1 mediated inflammation.Symptoms start in early childhood with recurren

APA, Harvard, Vancouver, ISO, and other styles

17

Wiersinga, W. Joost, and Tom van der Poll. The host response to infection in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0303.

Full text

Abstract:

Infection continues to be a leading cause of intensive care unit death. The host response to infection can be seen as a pattern recognition receptor (PRR)-mediated dysregulation of the immune system following pathogen invasion in which a careful balance between inflammatory and anti-inflammatory responses is vital. A measured and rapid response to microbial invasion is essential to health. The same immunological and coagulation systems that protect against localized infection can act to our disadvantage when these systems are activated systemically during generalized microbial infection. Toll-

APA, Harvard, Vancouver, ISO, and other styles

18

Abhishek, Abhishek, and Michael Doherty. Pathophysiology of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0049.

Full text

Abstract:

Calcium pyrophosphate (CPP) dihydrate crystals form extracellularly. Their formation requires sufficient extracellular inorganic pyrophosphate (ePPi), calcium, and pro-nucleating factors. As inorganic pyrophosphate (PPi) cannot cross cell membranes passively due to its large size, ePPi results either from hydrolysis of extracellular ATP by the enzyme ectonucleotide pyrophosphatase/phosphodiesterase 1 (also known as plasma cell membrane glycoprotein 1) or from the transcellular transport of PPi by ANKH. ePPi is hydrolyzed to phosphate (Pi) by tissue non-specific alkaline phosphatase. The level

APA, Harvard, Vancouver, ISO, and other styles

19

Voll, Reinhard E., and Barbara M. Bröker. Innate vs acquired immunity. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0048.

Full text

Abstract:

The innate and the adaptive immune system efficiently cooperate to protect us from infections. The ancient innate immune system, dating back to the first multicellular organisms, utilizes phagocytic cells, soluble antimicrobial peptides, and the complement system for an immediate line of defence against pathogens. Using a limited number of germline-encoded pattern recognition receptors including the Toll-like, RIG-1-like, and NOD-like receptors, the innate immune system recognizes so-called pathogen-associated molecular patterns (PAMPs). PAMPs are specific for groups of related microorganisms

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!