Log in

Relevant bibliographies by topics / Inflammation and metabolic syndrome / Journal articles

To see the other types of publications on this topic, follow the link: Inflammation and metabolic syndrome.

Journal articles on the topic 'Inflammation and metabolic syndrome'

Author: Grafiati

Published: 7 June 2025

Last updated: 2 August 2025

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Inflammation and metabolic syndrome.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Lang, Florian, and Christos Stournaras. "Serum and glucocorticoid inducible kinase, metabolic syndrome, inflammation, and tumor growth." HORMONES 12, no. 2 (2013): 160–71. http://dx.doi.org/10.14310/horm.2002.1401.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Weiss, T. W., and M. Rohla. "Metabolic syndrome, inflammation and atherothrombosis." Hämostaseologie 33, no. 04 (2013): 283–94. http://dx.doi.org/10.5482/hamo-13-07-0035.

Full text

Abstract:

SummaryExtensive research of the past decades altered our traditional concept about the genesis of atherosclerosis fundamentally. Today, the crucial role of inflammation in the formation and progression of atherosclerotic plaques is indisputable. Patients at high risk for developing cardiovascular disease, owing to poor diet, obesity and low physical activity have been shown to exhibit a particular inflammatory pattern.Therefore, the present review highlights the crosslink between the metabolic syndrome (MetS), adipose tissue, adipokines and selected inflammatory cytokines in the context of at

APA, Harvard, Vancouver, ISO, and other styles

3

You, Tongjian, and Barbara J. Nicklas. "Inflammation and the Metabolic Syndrome." Medicine & Science in Sports & Exercise 36, Supplement (2004): S1. http://dx.doi.org/10.1249/00005768-200405001-00003.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Sutherland, Julie P., Benjamin McKinley, and Robert H. Eckel. "The Metabolic Syndrome and Inflammation." Metabolic Syndrome and Related Disorders 2, no. 2 (2004): 82–104. http://dx.doi.org/10.1089/met.2004.2.82.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

You, Tongjian, and Barbara J. Nicklas. "Inflammation and the Metabolic Syndrome." Medicine & Science in Sports & Exercise 36, Supplement (2004): S1. http://dx.doi.org/10.1097/00005768-200405001-00003.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Paoletti, Rodolfo, Chiara Bolego, Andrea Poli, and Andrea Cignarella. "Metabolic syndrome, inflammation and atherosclerosis." Vascular Health and Risk Management 2, no. 2 (2006): 145–52. http://dx.doi.org/10.2147/vhrm.2006.2.2.145.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Welty, Francine K., Abdulhamied Alfaddagh, and Tarec K. Elajami. "Targeting inflammation in metabolic syndrome." Translational Research 167, no. 1 (2016): 257–80. http://dx.doi.org/10.1016/j.trsl.2015.06.017.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Lavie, Carl J., and Richard V. Milani. "Metabolic syndrome, inflammation, and exercise." American Journal of Cardiology 93, no. 10 (2004): 1334. http://dx.doi.org/10.1016/j.amjcard.2004.03.018.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Sharma, Praveen. "Inflammation and the Metabolic Syndrome." Indian Journal of Clinical Biochemistry 26, no. 4 (2011): 317–18. http://dx.doi.org/10.1007/s12291-011-0175-6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Wu, Yong, Yunzhou Dong, Shengzhong Duan, Donghui Zhu, and Lin Deng. "Metabolic Syndrome, Inflammation, and Cancer." Mediators of Inflammation 2017 (2017): 1–2. http://dx.doi.org/10.1155/2017/8259356.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

Pablo, I. Altieri. "The Metabolic Syndrome in Hispanics – The Role of Inflammation." Global Journal of Obesity, Diabetes and Metabolic Syndrome 2, no. 1 (2015): 012–17. https://doi.org/10.17352/2455-8583.000009.

Full text

Abstract:

We report clinical and molecular mechanisms relating the pro-inflammatory and anti-inflammatory process in the development of the components of the metabolic syndrome, emphasizing the cardiovascular problems developed in these groups of patients, especially the Hispanic population. Namely, the incidence, component characteristics and complications of the metabolic syndrome in island Puerto Ricans are described and evidence is presented supporting the fact that the metabolic syndrome may be milder in Puerto Rico than in the mainland United States because it is characterized by less aggressive c

APA, Harvard, Vancouver, ISO, and other styles

12

Grundy, Scott M. "Inflammation, Metabolic Syndrome, and Diet Responsiveness." Circulation 108, no. 2 (2003): 126–28. http://dx.doi.org/10.1161/01.cir.0000082641.20034.6a.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Patel, Hinal, and V. H. Patel. "Inflammation and Metabolic Syndrome: An Overview." Current Research in Nutrition and Food Science Journal 3, no. 3 (2015): 263–68. http://dx.doi.org/10.12944/crnfsj.3.3.10.

Full text

Abstract:

Inflammation is a reaction to a disrupted tissue homeostasis. Basically it is a tissue-destroying process that involves the recruitment of blood-derived products. Rapidly destroy or isolate the underlying source of the disturbance, removes damaged tissue and restore tissue homeostasis is the primary function of inflammation. Inflammatory and chronic metabolic alterations that together are termed metabolic syndrome. The risk of developing serious pathological conditions such as cardiovascular disease (CVD), type 2 diabetes mellitus (T2DM) and non-alcoholic fatty liver disease (NAFLD) is signifi

APA, Harvard, Vancouver, ISO, and other styles

14

Das, U. N. "Obesity, metabolic syndrome X, and inflammation." Nutrition 18, no. 5 (2002): 430–32. http://dx.doi.org/10.1016/s0899-9007(01)00747-x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Cole, Christopher, Satsuki Fukushima, John Fraser, Bronwyn Pearse, Adrian Barnett, and Peter Pohlner. "Inflammation, Metabolic Syndrome and Coronary Surgery." Heart, Lung and Circulation 20, no. 4 (2011): 274. http://dx.doi.org/10.1016/j.hlc.2010.11.053.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Yudkin, J. "Inflammation, Obesity, and the Metabolic Syndrome." Hormone and Metabolic Research 39, no. 10 (2007): 707–9. http://dx.doi.org/10.1055/s-2007-985898.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Klöting, Nora, and Matthias Blüher. "Adipocyte dysfunction, inflammation and metabolic syndrome." Reviews in Endocrine and Metabolic Disorders 15, no. 4 (2014): 277–87. http://dx.doi.org/10.1007/s11154-014-9301-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

18

Grundy, Scott M. "Inflammation, Hypertension, and the Metabolic Syndrome." JAMA 290, no. 22 (2003): 3000. http://dx.doi.org/10.1001/jama.290.22.3000.

Full text

APA, Harvard, Vancouver, ISO, and other styles

19

Abhijeet, M. R. Ramya, and Rangaswamy. "The Study of Association of Serum Ferritin Levels in Metabolic Syndrome." International Journal of Pharmaceutical and Clinical Research 16, no. 5 (2024): 687–92. https://doi.org/10.5281/zenodo.11409450.

Full text

Abstract:

<strong>Background and Objectives: </strong>Metabolic syndrome is a collection of metabolic disorders that raises the risk of diabetes and cardiovascular disease. Obesity, especially abdominal obesity, is linked to resistance to insulin’s effects on the use of fatty acids and peripheral glucose, frequently resulting in type 2 diabetes mellitus. In cardio metabolic disorders like hypertension, type 2 diabetes, and dyslipidemia, which are considered to be inflammatory diseases, evidence suggests an increase in serum ferritin. Since metabolic syndrome is thought to occur due to several

APA, Harvard, Vancouver, ISO, and other styles

20

Kazak, Filiz, Gul Fatma Yarim, Elvan Anadol, and Ayris Salt. "Hesperidin alleviates inflammation in the metabolic syndrome model." Veterinarski arhiv 94, no. 1 (2024): 67–76. http://dx.doi.org/10.24099/vet.arhiv.2025.

Full text

Abstract:

In metabolic syndrome, activated inflammatory signaling pathways trigger the release of proinflammatory cytokines. Nowadays, the use of natural bioactive compounds is trending as an alternative method for the treatment and management of metabolic syndrome. This study aimed to assess the potential effects of hesperidin in the metabolic syndrome model by analyzing the proinflammatory and anti-inflammatory cytokines in serum and liver. Rats were divided into 4 groups: Control (Rats were fed a standard chow diet and water ad libitum), hesperidin [Rats were fed hesperidin supplemented standard chow

APA, Harvard, Vancouver, ISO, and other styles

21

Pieńczykowska, Kamila, Anna Bryl, and Małgorzata Mrugacz. "Link Between Metabolic Syndrome, Inflammation, and Eye Diseases." International Journal of Molecular Sciences 26, no. 5 (2025): 2174. https://doi.org/10.3390/ijms26052174.

Full text

Abstract:

Metabolic syndrome (MetS)—a cluster of conditions including obesity, hypertension, dyslipidemia, and insulin resistance—is increasingly recognized as a key risk factor for the development of various eye diseases. The metabolic dysfunctions associated with this syndrome contribute to vascular and neurodegenerative damage within the eye, influencing disease onset and progression. Understanding these links highlights the importance of early diagnosis and management of metabolic syndrome to prevent vision loss and improve ocular health outcomes. This review explores the intricate interplay between

APA, Harvard, Vancouver, ISO, and other styles

22

Yokanan, Naftali, Noor Chozin Iin, Rahayu Sartono Teguh, Djajalaksana Susanthy, Saifur Rohman M., and Setyo Yudhanto Hendy. "Pulmonary Fibrosis with Increasing of Interleukin-6 in Sprague Dawley Rats with Metabolic Syndrome." INTERNATIONAL JOURNAL OF MEDICAL SCIENCE AND CLINICAL RESEARCH STUDIES 03, no. 12 (2023): 3162–69. https://doi.org/10.5281/zenodo.10423347.

Full text

Abstract:

<strong>Background</strong><strong>:</strong> Pulmonary fibrosis is the result of chronic inflammation causing decreased lung function. Severe lung damage and worsening respiratory symptoms can caused by metabolic syndrome. In metabolic syndrome, obesity, dyslipidemia, hypertension, and insulin occurs, also chronic inflammation and increased interleukin-6. Research on rats with high-fat high-fructose diet caused metabolic syndrome that triggered chronic inflammatory formed pulmonary fibrosis, which decreased lung function. The study result aim to increase awareness of pulmonary fibrosis d

APA, Harvard, Vancouver, ISO, and other styles

23

Dzięgielewska-Gęsiak, Sylwia, and Małgorzata Muc-Wierzgoń. "Inflammation and Oxidative Stress in Frailty and Metabolic Syndromes—Two Sides of the Same Coin." Metabolites 13, no. 4 (2023): 475. http://dx.doi.org/10.3390/metabo13040475.

Full text

Abstract:

In developed countries, aging is often seen as typical, but it is made complicated by many disorders and co-morbidities. Insulin resistance seems to be an underlying pathomechanism in frailty and metabolic syndromes. The decline in insulin sensitivity leads to changes in the oxidant–antioxidant balance and an accelerated inflammatory response, especially by adipocytes and macrophages in adipose tissue, as well as muscle mass density. Thus, in the pathophysiology of syndemic disorders—the metabolic syndrome and frailty syndrome—an extremely important role may be played by increased oxidative st

APA, Harvard, Vancouver, ISO, and other styles

24

Devaraj, Sridevi, Rajeev Goyal, and Ishwarlal Jialal. "Inflammation, Oxidative Stress, and the Metabolic Syndrome." US Endocrinology 04, no. 02 (2008): 32. http://dx.doi.org/10.17925/use.2008.04.2.32.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

Kern, Elizabeth. "Metabolic Syndrome and Systemic Inflammation in COPD." COPD: Journal of Chronic Obstructive Pulmonary Disease 8, no. 6 (2011): 395–96. http://dx.doi.org/10.3109/15412555.2011.636554.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Cuda, Cristina. "The role of inflammation in metabolic syndrome." SURG Journal 1, no. 2 (2008): 82–90. http://dx.doi.org/10.21083/surg.v1i2.406.

Full text

Abstract:

The metabolic consequences of obesity have made this highly prevalent condition one of the most common risk factors for type 2 diabetes, hypertension and atherosclerosis. Simultaneous occurrence of these conditions can be explained through the manifestations of metabolic syndrome [MetS]. Clinical indication of MetS is characterized by a clustering of risk factors for complex chronic diseases which all feature metabolic deterioration as a common component. Diagnosis of MetS can be made if a patient exhibits three of the identified risk factors, some of which include: elevated waist circumferenc

APA, Harvard, Vancouver, ISO, and other styles

27

Rethorst, Chad D., Ira Bernstein, and Madhukar H. Trivedi. "Inflammation, Obesity, and Metabolic Syndrome in Depression." Journal of Clinical Psychiatry 75, no. 12 (2014): e1428-e1432. http://dx.doi.org/10.4088/jcp.14m09009.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

Ahluwalia, N., V. A. Andreeva, E. Kesse-Guyot, and S. Hercberg. "Dietary patterns, inflammation and the metabolic syndrome." Diabetes & Metabolism 39, no. 2 (2013): 99–110. http://dx.doi.org/10.1016/j.diabet.2012.08.007.

Full text

APA, Harvard, Vancouver, ISO, and other styles

29

Wu, Yong, Yunzhou Dong, Shengzhong Duan, Donghui Zhu, and Lin Deng. "Corrigendum to “Metabolic Syndrome, Inflammation, and Cancer”." Mediators of Inflammation 2017 (2017): 1. http://dx.doi.org/10.1155/2017/6598540.

Full text

APA, Harvard, Vancouver, ISO, and other styles

30

Kraja, Aldi T., Daniel I. Chasman, Kari E. North, et al. "Pleiotropic genes for metabolic syndrome and inflammation." Molecular Genetics and Metabolism 112, no. 4 (2014): 317–38. http://dx.doi.org/10.1016/j.ymgme.2014.04.007.

Full text

APA, Harvard, Vancouver, ISO, and other styles

31

Phinney, Stephen D. "Fatty acids, inflammation, and the metabolic syndrome." American Journal of Clinical Nutrition 82, no. 6 (2005): 1151–52. http://dx.doi.org/10.1093/ajcn/82.6.1151.

Full text

APA, Harvard, Vancouver, ISO, and other styles

32

Guerrero-Romero, Fernando, and Martha Rodríguez-Morán. "Hypomagnesemia, oxidative stress, inflammation, and metabolic syndrome." Diabetes/Metabolism Research and Reviews 22, no. 6 (2006): 471–76. http://dx.doi.org/10.1002/dmrr.644.

Full text

APA, Harvard, Vancouver, ISO, and other styles

33

Golbidi, Saeid, Azam Mesdaghinia, and Ismail Laher. "Exercise in the Metabolic Syndrome." Oxidative Medicine and Cellular Longevity 2012 (2012): 1–13. http://dx.doi.org/10.1155/2012/349710.

Full text

Abstract:

The metabolic syndrome is a clustering of obesity, diabetes, hyperlipidemia, and hypertension that is occurring in increasing frequency across the global population. Although there is some controversy about its diagnostic criteria, oxidative stress, which is defined as imbalance between the production and inactivation of reactive oxygen species, has a major pathophysiological role in all the components of this disease. Oxidative stress and consequent inflammation induce insulin resistance, which likely links the various components of this disease. We briefly review the role of oxidative stress

APA, Harvard, Vancouver, ISO, and other styles

34

Kidyaeva, Alla V., Anastasiya V. Eichelberg, Natalia A. Shnayder, and Regina F. Nasyrova. "Correlation of Hematologic Coefficients of Inflammation and Metabolic Syndrome: Pilot Study." Personalized Psychiatry and Neurology 5, no. 2 (2025): 31–36. https://doi.org/10.52667/2712-9179-2025-5-2-31-36.

Full text

Abstract:

Antipsychotic therapy often causes side effects, one of which is metabolic syndrome. This condition increases the risk of cardiovascular disease and increases mortality in patients with schizophrenia. Currently, to improve the safety of antipsychotic therapy, the possibility of introducing into clinical practice the monitoring of hematologic inflammation coefficients as predictors of the development of metabolic syndrome is being considered. Objective: was to determine the presence of correlation between hematologic inflammation coefficients and metabolic syndrome in patients with schizophreni

APA, Harvard, Vancouver, ISO, and other styles

35

Navruzova, U.O., N.K Khamidova, and Sh.A. Yusupov. "Features of Periodontitis in Metabolic Disorders." Pharmaceutical and Chemical Journal 6, no. 6 (2019): 108–13. https://doi.org/10.5281/zenodo.13950915.

Full text

Abstract:

The review of literature on features of a current of parodontit at a metabolic disorder is provided in article. Thus, inflammation as one of the most ancient problems of medicine and also the related questions of reactivity and change of various components of a homeostasis including and a complex of metabolic changes, not only did not lose the value, but as the changed structure of incidence shows, acquires today still big relevance.

APA, Harvard, Vancouver, ISO, and other styles

36

Daminov, Abdurasul Takhirovich, Muxlisa Qo'chqorova, Arslonbek Axadov, Sitora Ismoilova, and Lobar Tursunboyeva. "GLUCOCORTICOIDS AND DIABETES." Educational Research in Universal Sciences (ERUS) 4, no. 3 (2025): 106–19. https://doi.org/10.5281/zenodo.14871716.

Full text

Abstract:

Glucocorticoids (GCs), stress hormones produced by the adrenal cortex, are involved in many physiological and metabolic processes, including glucose homeostasis and inflammation. Excess GC signaling results in Cushing‘s syndrome and possibly metabolic syndrome. Diabetes, central adipose tissue, and hyperlipidemia are components of both syndromes. Here, we discuss the mechanisms of GC action, clinical syndromes of GC excess, modulation of glucose homeostasis by GCs, and future GC signaling-based therapies for diabetes.

APA, Harvard, Vancouver, ISO, and other styles

37

Yen, Chi-Hua, Po-Sheng Chang, Ching-Ju Chiu, Yu-Yun Huang та Ping-Ting Lin. "β-Carotene Status Is Associated with Inflammation and Two Components of Metabolic Syndrome in Patients with and without Osteoarthritis". Nutrients 13, № 7 (2021): 2280. http://dx.doi.org/10.3390/nu13072280.

Full text

Abstract:

This study was conducted to investigate the β-carotene status in osteoarthritis (OA) patients and examine its relationships with the risk of inflammation and metabolic syndrome. OA patients were stratified by obesity based on body fat percentage (obese OA, n = 44; non-obese OA, n = 56), and sixty-nine subjects without OA or obesity were assigned as a non-obese control group. β-carotene, metabolic parameters, and inflammation status were assessed. Obese OA patients exhibited a significantly higher rate of metabolic syndrome (p = 0.02), abdominal obesity (p < 0.01), and lower β-carotene statu

APA, Harvard, Vancouver, ISO, and other styles

38

Meiliana, Anna, Nurrani Mustika Dewi, and Andi Wijaya. "Adipose Tissue, Inflammation (Meta-inflammation) and Obesity Management." Indonesian Biomedical Journal 7, no. 3 (2015): 129. http://dx.doi.org/10.18585/inabj.v7i3.185.

Full text

Abstract:

BACKGROUND: Obesity-induced inflammation contributes to the development of type 2 diabetes, metabolic syndrome, and cardiovascular disease.CONTENT:The last decade has seen a sharp increase in our appreciation for the macrophage as a critical regulator of metabolic status in obesity. Activation of adipose tissue (AT) macrophages within fat depots is coupled with the development of obesity-induced proinflammatory state and insulin resistance (IR). The activation of classically activated M1 macrophages at the expense of anti-inflammatory M2 macrophages has been causally linked to the development

APA, Harvard, Vancouver, ISO, and other styles

39

Masenga, Sepiso K., Lombe S. Kabwe, Martin Chakulya, and Annet Kirabo. "Mechanisms of Oxidative Stress in Metabolic Syndrome." International Journal of Molecular Sciences 24, no. 9 (2023): 7898. http://dx.doi.org/10.3390/ijms24097898.

Full text

Abstract:

Metabolic syndrome is a cluster of conditions associated with the risk of diabetes mellitus type 2 and cardiovascular diseases (CVDs). Metabolic syndrome is closely related to obesity. Increased adiposity promotes inflammation and oxidative stress, which are precursors of various complications involving metabolic syndrome components, namely insulin resistance, hypertension, and hyperlipidemia. An increasing number of studies confirm the importance of oxidative stress and chronic inflammation in the etiology of metabolic syndrome. However, few studies have reviewed the mechanisms underlying the

APA, Harvard, Vancouver, ISO, and other styles

40

Nasyrova, R. F., A. V. Kidyaeva, and N. A. Shnayder. "Hematological Predictors of Antipsychotic-induced Metabolic Syndrome in a Female Patient with Schizophrenia: Case Report." Personalized Psychiatry and Neurology 4, no. 2 (2024): 39–46. http://dx.doi.org/10.52667/2712-9179-2024-4-2-39-46.

Full text

Abstract:

Schizophrenia is a chronic mental disorder. It is treated with antipsychotics, which have a high risk of adverse reactions. One of these adverse reactions is metabolic syndrome, which increases the risk of cardiovascular diseases and the mortality rate of patients with schizophrenia. Various studies have shown an association between hematological parameters and metabolic syndrome. In this regard, the use of hematological predictors as a diagnostic tool can help identify risks and timely correct antipsychotic therapy for preventing metabolic syndrome. One of the most promising predictors are he

APA, Harvard, Vancouver, ISO, and other styles

41

Samad, Fahumiya, and Wolfram Ruf. "Inflammation, obesity, and thrombosis." Blood 122, no. 20 (2013): 3415–22. http://dx.doi.org/10.1182/blood-2013-05-427708.

Full text

Abstract:

Abstract Clinical and epidemiological studies support a connection between obesity and thrombosis, involving elevated expression of the prothrombotic molecules plasminogen activator inhibitor-1 and tissue factor (TF) and increased platelet activation. Cardiovascular diseases and metabolic syndrome–associated disorders, including obesity, insulin resistance, type 2 diabetes, and hepatic steatosis, involve inflammation elicited by infiltration and activation of immune cells, particularly macrophages, into adipose tissue. Although TF has been clearly linked to a procoagulant state in obesity, eme

APA, Harvard, Vancouver, ISO, and other styles

42

Russo, Marc. "A Review of Etiological Biomarkers for Fibromyalgia and Their Therapeutic Implications." Pain Physician Journal 27, no. 8 (2024): 495–506. https://doi.org/10.36076/ppj.2024.7.495.

Full text

Abstract:

BACKGROUND: Fibromyalgia is a complex condition that has long puzzled the medical community. Hypotheses to explain the chronic widespread pain associated with the disease have evolved significantly over the years. However, research efforts to identify disease-specific biomarkers and develop effective treatments have been largely unsuccessful. OBJECTIVES: The goals of this study were to review potential etiological biomarkers for fibromyalgia, focusing on micro-inflammation and metabolic syndrome, and to discuss the clinical implications of the review findings. STUDY DESIGN: A narrative review.

APA, Harvard, Vancouver, ISO, and other styles

43

Divella, Rosa, Gennaro Gadaleta Caldarola, and Antonio Mazzocca. "Chronic Inflammation in Obesity and Cancer Cachexia." Journal of Clinical Medicine 11, no. 8 (2022): 2191. http://dx.doi.org/10.3390/jcm11082191.

Full text

Abstract:

Chronic inflammation has long been linked to obesity and related conditions such as type 2 diabetes and metabolic syndrome. According to current research, the increased risk of cancer in people with certain metabolic diseases may be due to chronic inflammation. Adipocytokines, which are pro-inflammatory cytokines secreted in excess, are elevated in many chronic metabolic diseases. Cytokines and inflammatory mediators, which are not directly linked to DNA, are important in tumorigenesis. Cachexia, a type of metabolic syndrome linked to the disease, is associated with a dysregulation of metaboli

APA, Harvard, Vancouver, ISO, and other styles

44

Jani, Ylber, Atila Rexhepi, Bekim Pocesta, et al. "Role of Inflammation on the Control of the Arterial Hypertension among Patients with Metabolic Syndrome." Clinical Cardiology and Cardiovascular Interventions 3, no. 3 (2021): 01–05. http://dx.doi.org/10.31579/2641-0419/035.

Full text

Abstract:

BACKGROUND: It is widely accepted that hypertension is a pro-inflammatory disease and that the immune system plays a vital role in mediating hypertensive outcomes, end organ damages . and modulation of hypertensive pathology [2]. Patient with MetS usually present increased levels of C-reaktive protein (CRP) wich is a prototypic marker of inflammation [5], however the data of the influence of incresed levels of CRP on the control of the aterial hypertension in patients with MetS, are scarce. OBJECTIVE: We sought to determine the incresed CRP levels influence on the control of the aterial hypert

APA, Harvard, Vancouver, ISO, and other styles

45

Jani, Ylber. "Role of Inflammation on the Control of the Arterial Hypertension among Patients with Metabolic Syndrome." Clinical Cardiology and Cardiovascular Interventions 3, no. 3 (2020): 01–05. http://dx.doi.org/10.31579/2641-0419/044.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

Syauqy, Ahmad, Chien-Yeh Hsu, Hsiao-Hsien Rau, Adi Lukas Kurniawan, and Jane C.-J. Chao. "Association of Sleep Duration and Insomnia Symptoms with Components of Metabolic Syndrome and Inflammation in Middle-Aged and Older Adults with Metabolic Syndrome in Taiwan." Nutrients 11, no. 8 (2019): 1848. http://dx.doi.org/10.3390/nu11081848.

Full text

Abstract:

The study determined the association of sleep duration and insomnia symptoms with the components of metabolic syndrome and inflammation in middle-aged and older adults with metabolic syndrome in Taiwan. This cross-sectional study used the database compiled in Taiwan between 2004–2013. A total of 26,016 volunteers aged 35 years and above were selected. Metabolic syndrome was defined according to the International Diabetes Federation. Compared with regular sleep duration (6–8 h/day), short (<6 h/day) or long sleep duration (>8 h/day) and insomnia symptoms significantly increased the odds r

APA, Harvard, Vancouver, ISO, and other styles

47

Montecucco, Fabrizio, Maria Bertolotto, Nicolas Vuilleumier, et al. "Acipimox reduces circulating levels of insulin and associated neutrophilic inflammation in metabolic syndrome." American Journal of Physiology-Endocrinology and Metabolism 300, no. 4 (2011): E681—E690. http://dx.doi.org/10.1152/ajpendo.00527.2010.

Full text

Abstract:

Metabolic syndrome is a proatherosclerotic condition clustering cardiovascular risk factors, including glucose and lipid profile alterations. The pathophysiological mechanisms favoring atherosclerotic inflammation in the metabolic syndrome remain elusive. Here, we investigated the potential role of the antilipolytic drug acipimox on neutrophil- and monocyte-mediated inflammation in the metabolic syndrome. Acipimox (500 mg) was orally administered to metabolic syndrome patients ( n = 11) or healthy controls ( n = 8). Serum and plasma was collected before acipimox administration ( time 0) as wel

APA, Harvard, Vancouver, ISO, and other styles

48

Tarantino, Giovanni, Maurizio Marra, Franco Contaldo, and Fabrizio Pasanisi. "Basal metabolic rate in morbidly obese patients with non-alcoholic fatty liver disease." Clinical & Investigative Medicine 31, no. 1 (2008): 24. http://dx.doi.org/10.25011/cim.v31i1.3138.

Full text

Abstract:

Background. Non-Alcoholic Fatty Liver Disease occurs mainly in severly obese patients and its relationship to Metabolic Syndrome is increasingly recognized. The aim of this study was to determine energy production-utilization by measuring the Basal Metabolic Rate in severely obese patients, characterized by NAFLD, with or without Metabolic Syndrome. Then, the role of systemic inflammation was assessed. Patients and methods. Twenty severly obese men with Metabolic Syndrome were compared with a well-matched cohort of patients without Metabolic Syndrome. All showed hepatic steatosis at UltraSonog

APA, Harvard, Vancouver, ISO, and other styles

49

ter Horst, Rob, Inge C. L. van den Munckhof, Kiki Schraa, et al. "Sex-Specific Regulation of Inflammation and Metabolic Syndrome in Obesity." Arteriosclerosis, Thrombosis, and Vascular Biology 40, no. 7 (2020): 1787–800. http://dx.doi.org/10.1161/atvbaha.120.314508.

Full text

Abstract:

Objective: Metabolic dysregulation and inflammation are important consequences of obesity and impact susceptibility to cardiovascular disease. Anti-inflammatory therapy in cardiovascular disease is being developed under the assumption that inflammatory pathways are identical in women and men, but it is not known if this is indeed the case. In this study, we assessed the sex-specific relation between inflammation and metabolic dysregulation in obesity. Approach and Results: Three hundred two individuals were included, half with a BMI 27 to 30 kg/m 2 and half with a BMI>30 kg/m 2 , 45% were w

APA, Harvard, Vancouver, ISO, and other styles

50

Fernandez-García, Jose, Fernando Cardona, and Francisco Tinahones. "Inflammation, Oxidative Stress and Metabolic Syndrome: Dietary Modulation." Current Vascular Pharmacology 11, no. 6 (2014): 906–19. http://dx.doi.org/10.2174/15701611113116660175.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!