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Books on the topic 'Inflammation of the Bone'

Author: Grafiati

Published: 7 June 2025

Last updated: 16 July 2025

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1

L, Barrett Stephen. Heel pain: Healing the heel. Authorhouse, 2009.

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2

Clausen, Björn E., and Jon D. Laman, eds. Inflammation. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6786-5.

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3

Górski, Andrzej, Hubert Krotkiewski, and Michał Zimecki, eds. Inflammation. Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9702-9.

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4

Wang, Yong-Xiao, ed. Inflammation. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-55254-0.

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5

Johnston, Tony. Bone by bone by bone. Roaring Brook Press, 2007.

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6

O'Connell, Carol. Bone by bone. Berkley Books, 2009.

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7

O'Connell, Carol. Bone by bone. G.P. Putnam's Sons, 2008.

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8

O'Connell, Carol. Bone by bone. Wheeler Pub., 2009.

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9

Matthiessen, Peter. Bone by Bone. Random House, 1999.

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10

O'Connell, Carol. Bone by Bone. Penguin USA, Inc., 2008.

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11

Pierangelo, Geppetti, and Holzer, Peter, Mag. rer. nat. Dr. phil., eds. Neurogenic inflammation. CRC Press, 1996.

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12

Winyard, Paul G., and Derek A. Willoughby. Inflammation Protocols. Humana Press, 2003. http://dx.doi.org/10.1385/1592593747.

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13

Miyasaka, Masayuki, and Kiyoshi Takatsu, eds. Chronic Inflammation. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-56068-5.

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14

Zierhut, Manfred, Carlos Pavesio, Shigeaki Ohno, Fernando Orefice, and Narsing A. Rao, eds. Intraocular Inflammation. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-540-75387-2.

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15

(Editor), Slobodan Vukicevic, and Kuber T. Sampath (Editor), eds. Bone Morphogenetic Proteins: Regeneration of Bone and Beyond (Progress in Inflammation Research). Birkhäuser Basel, 2005.

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16

Lories, Rik J., and Georg Schett. Pathology: bone. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0010.

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Axial spondyloarthritis is associated with different types of skeletal damage. Inflammation at the affected sites is linked with both loss of trabecular bone and new bone formation on the cortical side, potentially leading to joint or spine ankylosis. Both aspects of the disease can result in a significant burden for the patient. Bone loss is directly linked to proinflammatory cytokines and activation of osteoclasts. Control of inflammation is therefore the best strategy to prevent loss of bone. The nature of the new bone formation process is less defined. A prominent role for developmental si

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17

Broth, Bone, and Andrew Mills. Bone Broth: Bone Broth Diet - Lose Weight, Fight Inflammation, and Improve Your Health with Delicious Bone Broth Recipes. Independently Published, 2016.

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18

Broth, Bone, and Andrew Mills. Bone Broth: Bone Broth Diet 2 - Lose Weight, Fight Inflammation, and Improve Your Health with Delicious Bone Broth Recipes. Independently Published, 2016.

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19

Broth, Bone, and Andrew Mills. Bone Broth: Bone Broth Diet Box Set - Lose Weight, Fight Inflammation, and Improve Your Health with Delicious Bone Broth Recipes. Independently Published, 2016.

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20

(Editor), Slobodan Vukicevic, and Kuber T. Sampath (Editor), eds. Bone Morphogenetic Proteins: From Laboratory to Clinical Practice (Progress in Inflammation Research). Birkhäuser Basel, 2002.

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21

James, Danielle. The Best Paleo Bone Broth Recipes: Beat Inflammation Be Healthy Improve Overall Wellbeing. CreateSpace Independent Publishing Platform, 2016.

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22

Klingenberg, Roland, and Ulf Müller-Ladner. Mechanisms of inflammation. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0270.

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This chapter provides a brief summary of the immune pathogenesis of atherosclerosis, highlighting shared features with inflammatory pathways in rheumatoid arthritis (RA) described in detail in Chapter 25.4. RA constitutes a prototype autoimmune disease primarily affecting the joints but also the heart and vessels associated with increased cardiovascular mortality. Recent years have produced a wealth of novel insights into the diversity of immune cell types which either propagate or dampen inflammation in atherogenesis. Expansion of this inherent anti-inflammatory component carried by regulator

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23

Török, M. Estée, Fiona J. Cooke, and Ed Moran. Bone and joint infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199671328.003.0022.

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This chapter provides an overview of inflammations of the joint space and bones, such as arthritis and bursitis, including osteomyelitis and bone destruction and formation of sequestra. The chapter also includes prosthetic joint infections such as hip and knee replacements. It also describes diabetic foot infections, defined as any inframalleolar infection in a patient with diabetes mellitus. Infections include paronychia, cellulitis, myositis, abscesses, necrotizing fasciitis, septic arthritis, tendonitis, and osteomyelitis.

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24

Doré-Savard, Louis, Nicolas Beaudet, and Philippe Sarret. Mechanisms of bone cancer pain. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0037.

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The landmark paper discussed in this chapter focuses on pain arising from malignancy of the bone, which, whether primary or originating from a distant site, is the cause for a majority of cancer pain syndromes. Bone is an innervated organ that can relay nociceptive signals triggered by nerve damage, acidosis, inflammation, and hypoxia. The understanding of the physiopathology of skeletal pain has leaped significantly forwards over the last 15 years. The development of animal models that allowed for the visualization of bone microenvironment modifications by the tumour played an important role

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25

Lories, Rik. Mechanisms of bone destruction and proliferation in psoriatic arthritis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0008.

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Psoriatic arthritis is a chronic inflammatory joint disease that can affect both the peripheral and axial skeleton. The clinical presentation of psoriatic arthritis is very heterogeneous and different subforms have been described. Structural damage to the joint is a feared complication of psoriatic arthritis. The severity of joint inflammation and subsequent damage can range from mild to extreme. Over the last decade, insights into the molecular and cellular mechanisms that underlie the skeletal changes in psoriatic arthritis have gradually increased although translational validation of concep

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26

Heel pain: Healing the heel. Authorhouse, 2009.

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27

Avram, Morrell M. Management of Comorbidities in Kidney Disease in the 21st Century: Anemia, Bone Disease, Malnutrition, and Inflammation. Wiley & Sons, Incorporated, John, 2008.

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28

Avram, M. M. Management of Comorbidities in Kidney Disease in the 21st Century: Anemia, Bone Disease, Malnutrition and Inflammation. Blackwell Publishing Limited, 2003.

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29

Sprague, Stuart M., and James M. Pullman. Spectrum of bone pathologies in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0122.

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Histologic bone abnormalities begin very early in the course of chronic kidney disease. The KDIGO guidelines recommend that bone disease in patients with chronic kidney disease should be diagnosed on the basis of bone biopsy examination, with bone histomorphometry. They have also proposed a new classification system (TMV), using three key features of bone histology—turnover, mineralization, and volume—to describe bone disease in these patients. However, bone biopsy is still rarely performed today, as it involves an invasive procedure and highly specialized laboratory techniques. High-turnover

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30

Hood, Wharton P., and Robert Howard Hutton. On Bone-Setting, and Its Relation to the Treatment of Joints Crippled by Injury, Rheumatism, Inflammation, Etc. Franklin Classics Trade Press, 2018.

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31

Hood, Wharton P., and Robert Howard Hutton. On Bone-Setting, and Its Relation to the Treatment of Joints Crippled by Injury, Rheumatism, Inflammation, Etc. Creative Media Partners, LLC, 2018.

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32

Mossor, Gwenn. Vitamin e's Truth : Lower Cholesterol, Reduce Inflammation, Protect Your Liver and Promote Bone Health: Vitamin e for Skin Lightening. Independently Published, 2021.

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33

Raza, Karim, Caroline Cardy, and Elizabeth Justice. Rheumatoid arthritis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0267.

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Rheumatoid arthritis is a chronic systemic inflammatory disorder. It is characterized by inflammation of the synovium with consequent cartilage and bone destruction. Extra-articular manifestations frequently occur.

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34

Sieper, Joachim. Ankylosing spondylitis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0113.

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Ankylosing spondylitis (AS) is a chronic inflammatory disease predominantly of the sacroiliac joint (SIJ) and the spine. It starts normally in the second decade of life and has a slight male predominance. The prevalence is between 0.2 and 0.8% and is strongly dependent on the prevalence of HLA B27 in a given population. For the diagnosis of AS, the presence of radiographic sacroiliitis is mandatory. However, radiographs do not detect active inflammation but only structural bony damage. Most recently new classification criteria for axial spondyloarthritis (SpA) have been developed by the Assess

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35

Sieper, Joachim. Axial spondyloarthropathies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0113_update_003.

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Axial spondyloarthritis (axSpA) is a chronic inflammatory disease predominantly of the sacroiliac joint (SIJ) and the spine. It starts normally in the second decade of life and has a slight male predominance. The prevalence is between 0.2% and 0.8% and is strongly dependent on the prevalence of HLA-B27 in a given population. AxSpA can be split in patients with radiographic axSpA (also termed ankylosing spondylitis (AS)) and in patients with non-radiographic axSpA (nr-axSpA). For the diagnosis of AS, the presence of radiographic sacroiliitis is mandatory. However, radiographs do not detect acti

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36

Lafeber, Floris P. J. G., Nick J. Besselink, and Simon C. Mastbergen. Synovium and capsule. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0006.

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Synovium is an integrated tissue of the diarthrodial joints that interacts with all the other joint tissues and specifically is important in nourishment and lubrication of the articular cartilage, removal of waste products, and immunological surveillance. Chronic as well as recurrent low-grade synovial inflammation definitely contributes to progression and symptoms of certain patients with osteoarthritis. Low-grade inflammation may even be causative in the disease. The challenge is that osteoarthritis is a heterogeneous disorder with inflammation not only of the synovial tissue but with its me

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37

Cohen, Stacey Lynn. The interleukin-10 knockout mouse: A model for studying bone metabolism during intestinal inflammation and the effects of flaxseed oil as a nutritional intervention. 2004.

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38

Harrison, Mark. Wound healing. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198765875.003.0057.

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This chapter describes the pathology of wound healing as it applies to Emergency Medicine, and in particular the Primary FRCEM examination. The chapter outlines the key details of haemostasis, inflammation, reconstruction, epithelialization, and maturation, and the specific tissues affected, including skin, tendon, peripheral nerve, bone, myocardium, and brain. This chapter is laid out exactly following the RCEM syllabus, to allow easy reference and consolidation of learning.

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39

Lewis, Elias. Diclofenac: Ultimate Guide to Reduces Inflammation, Treat Aches Pains, Problems with Joints, Muscles and Bones. Independently Published, 2022.

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40

McGonagle, Dennis, and Iris Eshed. MRI. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0018.

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The features of psoriatic arthritis (PsA) are disparate—from peripheral synovitis to axial inflammation, from bone destruction to bone formation or both, and nail disease. Fat suppression magnetic resonance imaging (MRI) has had a major impact on understanding PsA. MRI suggests a unifying anatomical basis for PsA with the common denominator of disease localization to entheses and adjacent bone and other sites of high biomechanical stress. MRI has also shown that entheseal changes are not uncommon in generalized osteoarthritis and occasionally in normals, making careful clinical correlation ess

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41

Gartman, Karen. Dexamethasone: One Medicine for the Treatment of Blood or Bone Marrow Problems, Inflammation, Severe Allergies, Arthritis, Kidney Problems, Asthma, Flare-Ups of Multiple Sclerosis, Skin Conditions, and Adrenal Problems. Independently Published, 2019.

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42

Peterson, Susan, and Staci Reintjes. Otitis Externa, Otitis Media, and Mastoiditis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0011.

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Otitis Externa is an infection of external auditory canal. Infection typically occurs via penetration of the epithelial barrier. Patients typically present with inflammation of the auricle, external auditory canal, or outer tympanic membrane. First-line therapy includes topical acidic agents and antibiotic drops. Oral antibiotics should be considered for recurrent infections, those resistant to topical therapy, severe disease, extension beyond the external auditory canal, diabetics, or immunocompromised patients. Otitis Media is an infection of the middle ear. Patients typically present with o

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43

Mo Ahn, Joong, Yusuf Menda, and Georges Y. El-Khoury. Imaging. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.0010.

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♦ Each modality of imaging—digital radiography, multidetector computed tomography (MDCT), magnetic resonance imaging (MRI), ultrasound, and nuclear medicine studies—has its own advantages and disadvantages♦ Conventional radiography is the best for initial evaluation of a musculoskeletal problem♦ MDCT rapid survey of multiple trauma patients is easily performed using the new high speed computed tomography scanners♦ MRI is the imaging modality of choice for internal derangement of the knee and other soft tissue injuries♦ Radionuclide bone imaging is most suitable for screening the whole skeleton

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44

Keenan, Robert T., Sneha Pai, and Naomi Schlesinger. Imaging of gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0043.

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Gout is a systemic metabolic disease. The enzyme urate oxidase (uricase) that catalyses the oxidation of uric acid to the more soluble compound allantoin is inactive in humans. This may lead to hyperuricaemia. Hyperuricaemia is often present for many years prior to clinical signs of gout. Acute attacks occur as a result of an inflammatory response to monosodium urate (MSU) crystal deposition leading to intense pain and inflammation in the affected joints. Uncontrolled hyperuricaemia and resultant gout can evolve into a destructive arthritis. Imaging may be helpful in the diagnosis of gout as w

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45

Youatt, William. The Horse: Together With A General History Of The Horse And An Essay On The Ass And The Mule. Kessinger Publishing, LLC, 2007.

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46

Youatt, William. The Horse: With a Treatise of Draught. Adamant Media Corporation, 2001.

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47

Vodopivec, Ivana, and Tracey A. Cho. Neurobiology of Transverse Myelitis and Infectious Myelopathies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0153.

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Infectious agents cause spinal cord pathology by three different mechanisms: direct invasion/infection of neural tissues (i.e., infective myelitis), secondary inflammation and tissue bystander damage with or without autoimmune pathogenesis (parainfectious myelitis), or involvement of extra-axial structures (including the pia-arachnoid, the dura, the epidural space, or the adjacent spinal bones or intervertebral discs), resulting in compressive or ischemic myelopathy. This chapter describes the pathogenesis and treatment of these disorders.

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48

Kelleher, Clare. Diabetic Foot Infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0043.

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Diabetic foot infections (DFI) are diagnosed by two or more classic findings of inflammation (redness, swelling, warmth, and tenderness) or purulent drainage within an existing diabetic foot wound. Wounds without clinical evidence of soft tissue or bone infection often do not require antibiotic therapy. When infection is present, empiric antibiotic regimens must be based on the available clinical and local epidemiologic data, but definitive therapy should be based on cultures of infected tissues or clinical response. Consideration of methicillin-resistant Staphylococcus aureus (MRSA) coverage

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49

Raghavan, Sri. Infection in the Cancer Patient. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0054.

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Cancer patients have increased susceptibility to a variety of both common and atypical infections due to the steady increase in outpatient chemotherapy regimens, these patients are presenting more often to the emergency department when acutely ill. Already immunocompromised, patients’ chemotherapy regimens lead to neutropenia, deficits in cellular and humoral immunity, and disruption of mucosal barriers that predisposing them to severe disease presentations with high morbidity and mortality rates. There are different subsets of oncologic patients predisposed to specific infections. One of the

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50

van Gaalen, Floris, Désirée van der Heijde, and Maxime Dougados. Diagnosis and classification of axial spondyloarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0003.

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Axial spondyloarthritis (axSpA) is a potentially disabling chronic inflammatory disease affecting the spine and sacroiliac (SI) joints. Lead symptoms are chronic back pain and stiffness. The disease is called radiographic axSpA or ankylosing spondylitis (AS) when, on plain radiographs, bone changes consistent with sacroiliitis are present. When no evidence of sacroiliitis is seen on radiographs, it is called non-radiographic axSpA. In such cases, diagnosis is made based on evidence of active inflammation of SI joints on magnetic resonance imaging (MRI) and clinical and laboratory features, or

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