Relevant bibliographies by topics / Inflammation / Dissertations / Theses
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Dissertations / Theses on the topic 'Inflammation'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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1

Lawrence, Clifford M. "Anthralin inflammation." Thesis, University of Newcastle Upon Tyne, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.390318.

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2

Warke, T. J. "Childhood airways inflammation." Thesis, Queen's University Belfast, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268984.

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3

Jatta, Ken. "Inflammation in Atherosclerosis." Doctoral thesis, Örebro : Universitetsbiblioteket, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-478.

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4

Gilroy, Derek William. "Cyclooxygenase 2 inflammation." Thesis, Queen Mary, University of London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265077.

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5

Limb, G. A. "Lymphokines in inflammation." Thesis, Brunel University, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.373086.

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6

Peters, Caren Lorraine. "Hypoxia in inflammation." Thesis, University of Bath, 2003. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.426151.

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7

Sanchez, Moral Lidia. "Role of ZEB1 in adenoma formation, inflammation and inflammation-driven carcinoma." Doctoral thesis, Universitat de Barcelona, 2019. http://hdl.handle.net/10803/667993.

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Multiple studies have highlighted the role of ZEB1 as critical regulator of tumor progression through the regulation of different hallmarks of cancer beyond the induction of EMT. The general aim of this dissertation is to characterize new potential mechanisms that regulate oncogenic transformation in colorectal carcinoma. The specific objectives of this dissertation are: 1. To identify new roles and targets of the transcription factor ZEB1 as a mediator of Wnt-induced intestinal adenoma formation. 2. To characterize ZEB1’s mechanism of action as a transcriptional regulator in inflammation
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8

Levick, Scott P. "Inflammation and cardiovascular remodelling /." [St. Lucia, Qld.], 2005. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19090.pdf.

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9

Blomgran, Parmis. "Inflammation and tendon healing." Doctoral thesis, Linköpings universitet, Avdelningen för Kirurgi, Ortopedi och Onkologi, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-142349.

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Tendons heal through three different overlapping phases; the inflammatory, proliferative and remodeling phase. Many studies have investigated what factors influence healing of tendons. However, little was known about inflammation and the immune cells present during Achilles tendon healing by the time this thesis started. We developed a flow cytometry method for our rat model of tendon healing, which enabled us to study different leukocyte subpopulations during Achilles tendon healing. The general aim of this thesis was to understand more about inflammation and the immune cell populations prese
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10

Eustace, Andrew David. "Syndecan 3 and inflammation." Thesis, University of Bristol, 2016. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.720843.

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11

Kamath, S. V. "Inflammation in paediatric asthma." Thesis, Queen's University Belfast, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269034.

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12

Mihaylova, Dessislava Dimitrova. "Submicron Particles and Inflammation." Thesis, Norges teknisk-naturvitenskapelige universitet, Institutt for bioteknologi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-18590.

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Iron nanoparticles occur naturally in the environment, but their exposure increases dramatically due to the field of nanotechnology and –medicine. It is poorly understood how the intracellular cooperative mechanisms of submicron particles and microorganisms function on mammalian immune system. In this study, superparamagnetic iron oxide (SPIO) submicron particles will be used to benefit the research within environmental diseases, addressing the biocompatibility of these particles. The size-dependent effects in the immune system of two carboxyl coated SPIO particles with stated sizes 100 nm and
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13

Higgins, Lisa Mary. "Regulation of intestinal inflammation." Thesis, Queen Mary, University of London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322813.

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14

Farrell, Adrian J. "Mediators of synovial inflammation." Thesis, University of Bristol, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.284326.

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15

Kaplan, Jennifer Melissa. "Immunomodulation During Systemic Inflammation." University of Cincinnati / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1186158205.

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16

Scotte, Michel. "Régénération hépatique et inflammation." Rouen, 1997. http://www.theses.fr/1997ROUES042.

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Une des plus étonnantes propriétés du foie est sa capacité à réguler sa croissance et sa taille. Ce phénomène de régénération hépatique (RH) est sous l'influence de facteurs de croissance qui agissent sur le foie de manière autocrine, paracrine ou endocrine. La réponse inflammatoire (RI), réponse homéostatique de l'organisme à différentes agressions, est sous la dépendance de cytokines qui induisent les modifications hépatiques de la synthèse protéique. Des interactions fortes existent entre ces deux phénomènes puisque l'hépatectomie induit une RI et que la RI semble avoir une influence sur la
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17

Meng, Luxi. "Adipocytes, Macrophages and Inflammation." Thesis, The University of Sydney, 2013. http://hdl.handle.net/2123/10183.

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While there is a very strong correlation between obesity and the development of insulin resistance and Type II diabetes, the molecular mechanisms involved are still unknown. However, one clue is that in both human subjects and animal models, obese adipose tissue is infiltrated with M1 macrophages and this creates a systemic low-grade, chronic inflammatory state that could be responsible for the negative consequences of obesity. A better understanding of the relationship between the adipocytes and macrophages under the inflammatory milieu may inform intervention strategies. In this study, the
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18

Sobowale, Oluwaseun. "Intracerebral haemorrhage and inflammation." Thesis, University of Manchester, 2018. https://www.research.manchester.ac.uk/portal/en/theses/intracerebral-haemorrhage-and-inflammation(7139560f-bd3c-4ff0-b628-f86ffc6477d2).html.

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Intracerebral haemorrhage (ICH) is a significant healthcare concern worldwide. Following ICH, primary injury occurs due to physical injury to neurones and glia as a result of mass effect from the haematoma. Secondary mechanisms of injury include haematoma expansion, toxic effects of the products of coagulation and blood breakdown products and sterile inflammation. Perihaematomal oedema can exacerbate mass effect in the acute and sub-acute phase of ICH. At present, the pathophysiology behind the secondary mechanisms of injury following ICH is not fully understood and this has led to inability t
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19

Zanoli, Luca Maria. "Inflammation and arterial stiffness." Doctoral thesis, Università di Catania, 2012. http://hdl.handle.net/10761/1088.

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Arterial stiffness is one of the earliest detectable manifestations of adverse structural and functional changes within the vessel wall. It is well known that increased large artery stiffness independently predicts the cardiovascular risk in a variety of populations. The identification of populations at higher risk of increased arterial stiffness and the knowledge of the mechanisms involved in arterial stiffening may help to identificate pharmacological and other treatments to reduce the arterial stiffness and improve the outcome of the patients. Recently, new advances have been proposed about
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20

Mäki-Petäjä, Kaisa Maria. "Inflammation, arterial stiffness and endothelial dysfunction : rheumatoid arthritis, a model of systemic inflammation." Thesis, University of Cambridge, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612095.

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21

Zhao, J. "The roles of myeloid derived cells in retinal inflammation and inflammation-mediated retinal angiogenesis." Thesis, Queen's University Belfast, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.677966.

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Intraocular inflammation encompasses a diverse group of diseases which account for up to 20% of severe vision loss, particularly following pathological neovascularisation in the retina. Nowadays, the use of corticosteroids is still the main armamentarium to manage these devastating conditions. Clearly safer, more target-specific and effective drugs are needed. Experimental autoimmune uveoretinitis (EAU) is an animal model for human intraocular inflammation. It is well known that myeloid derived cells substantially contribute to the pathological activities in this model. The aims of my PhD proj
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22

Chapman, Katie. "Peripheral inflammation after experimental stroke." Thesis, University of Manchester, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.518434.

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23

Downey, D. G. "Airways inflammation in cystic fibrosis." Thesis, Queen's University Belfast, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269047.

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24

Liu, Jia Clinical School Prince of Wales Hospital Faculty of Medicine UNSW. "Nitric oxide in airway inflammation." Publisher:University of New South Wales. Clinical School - Prince of Wales Hospital, 2009. http://handle.unsw.edu.au/1959.4/43678.

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Exhaled breath condensate (EBC) is a non-invasive method of investigating airway inflammation associated with nitric oxide (NO) and the metabolites nitrite/nitrates (NOx) in diseases such as chronic obstructive pulmonary disease (COPD), but some of the variables affecting the results are unknown. It was hypothesised that 1) EBC would be influenced by lung volumes and the type of EBC collection device; 2) fractional exhaled NO (FENO) and EBC NOx in COPD patients would be altered by smoking and glucocorticosteroids (GCS); 3) cigarette smoke could contribute to the EBC NOx concentration while it
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25

Bathoorn, Derk. "COPD exacerbations, inflammation and treatment." [S.l. : Groningen : s.n. ; University Library of Groningen] [Host], 2007. http://irs.ub.rug.nl/ppn/304982296.

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26

Parums, D. V. "Studies on inflammation in atherosclerosis." Thesis, University of Cambridge, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.235059.

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A spectrum of chronic inflammation is commonly seen in association with advanced human atherosclerosis. This local complication of advanced atherosclerosis is termed '<i>chronic periaortitis</i>'. This may be seen sub-clinically in necropsy samples or may present clinically, in more severe cases, as the conditions previously termed 'idiopathic retroperitoneal fibrosis', 'inflammatory aneurysm' or 'peri-aneurysmal retroperitoneal fibrosis'. The inflammatory cells consist of lymphocytes and plasma cells. Thinning or breaching of the media is common to all forms. A histological survey of necropsy
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27

Warland, David Anthony. "Inflammation, the microcirculation and microalbuminuria." Thesis, Northumbria University, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.416355.

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28

Iannitti, Tommaso. "Obesity, inflammation and pathological pain." Thesis, Glasgow Caledonian University, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555680.

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Obesity has become a concern of epidemic proportion and is associated with an increased incidence in chronic pain but the underlying mechanism is not known. Obesity is associated with a low-grade inflammation of white adipose tissue due to a dysregulation in cytokines and adipokines, some of which, including turnor necrosis factor-α and interleukin-1β, have been linked to inflammatory pain. Other adipokines such as adiponectin, display anti- inflammatory properties but their role in modulating inflammatory pain has not been investigated. The aim of this project was to characterise inflammatory
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29

Stevenson, Diane J. "P2X7, inflammation and gastrointestinal disease." Thesis, University of Nottingham, 2008. http://eprints.nottingham.ac.uk/28897/.

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The inflammatory bowel diseases, ulcerative colitis and Crohn's disease are characterised by spontaneously relapsing and remitting inflammation, associated with increased mucosal levels of the inflammatory cytokine, interleukin-1 (IL-1)β. IL-1β processing and release is mediated by ATP stimulation of the purine receptor, P2X7. P2X7 is a membrane ion channel highly expressed in immune cells. Signal transduction occurs via rapid cation exchange, plasma membrane depolarisation and increased intracellular calcium. Additionally, prolonged or repeated P2X7 stimulation leads to formation of a non-sel
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30

Marsden, Paul Anthony. "Cough, asthma and airways inflammation." Thesis, University of Manchester, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.516825.

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Rationale Cough is an important symptom in asthma. In addition, cough is both troublesome to patients and may be important in the development of more severe disease. Moreover, it is a key symptom in the diagnosis of asthma, according to international guidelines. Little is known regarding the mechanism of cough in asthma. Until now, subjective measures of cough have been used in mechanistic and therapeutic studies relating to both cough in asthma and chronic cough. There is no published literature documenting cough rates in asthma or the relationship between cough rates and both subjective meas
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31

Daugherty, Alan. "Lipoproteins, inflammation, and vascular disease." Thesis, University of Bath, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.425851.

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The major focus of the studies described in this thesis is the interaction of lipoproteins and inflammation in vascular diseases. Early studies describe the role of postprandial lipoproteins in the development of lipid-laden macro phages that are a hallmark of atherosclerotic lesions. These studies paved the way for subsequent studies on the role of lipoprotein modification in atherosclerosis. These included the discovery that LDL present in atherosclerotic lesions had properties that were consistent with oxidative modification. To determine whether this modification was a cause or consequence
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32

Poxon, Valerie Anne. "Gastrointestinal inflammation and mucus biosynthesis." Thesis, Birmingham City University, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334499.

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33

Wilson, Susan Jane. "Mucosal inflammation in allergic rhinitis." Thesis, University of Southampton, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295233.

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34

McNulty, Clare. "Ageing, inflammation and cardiovascular function." Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/6541/.

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Age is associated with the development of multi-system function loss including the musculoskeletal, immune and cardiovascular system, as well as body composition changes. These age-related alterations lead to frailty development and disease progression, reducing quality of life. A major lifestyle change occurring in later years is reduced physical activity levels. This thesis sought to examine the associations between physical activity and multi-system function loss in a cohort of elderly individuals, and to better understand the neural mechanisms underpinning the circulatory responses to exer
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35

De, Pablo Paola. "The epidemiology of musculoskeletal inflammation." Thesis, University of Birmingham, 2013. http://etheses.bham.ac.uk//id/eprint/4161/.

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Rheumatoid arthritis (RA) is a chronic inflammatory disease characterized by progressive joint destruction often resulting in functional disability and increased mortality. With appropriate therapies, it is possible to delay or even prevent evolution of patients into RA and/or induce remission. Thus the identification of individuals not only early in the disease course but at risk of developing RA is important. Following on this concept, we investigated the presence of systemic autoimmunity among individuals at risk of RA based on environmental exposures, and conducted a meta-analyses showing
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36

Brügel, Mathias [Verfasser]. "Biomarker der Inflammation / Mathias Brügel." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2021. http://d-nb.info/1227840144/34.

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37

Holm, Angelika. "Aquaporins in Infection and Inflammation." Doctoral thesis, Linköpings universitet, Avdelningen för mikrobiologi och molekylär medicin, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-127500.

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The ability of eukaryotic cells to change their shape and to migrate directionally is highly dependent on active volume regulation in cells building up tissues as well as in individual cells. Transmembrane fluxes of water via specialized water channels, called aquaporins (AQPs), facilitate the changes of volume and shape, which additionally require a complex interplay between the plasma membrane and the cytoskeleton. AQPs have been shown to be involved in the development of inflammatory processes and diseases. The aims of the studies underlying this thesis were to further elucidate the express
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38

Mertens, Kathrin. "Zinc in inflammation and sepsis." Thesis, University of Aberdeen, 2014. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=204050.

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Sepsis is the major cause of mortality on intensive care units (ICU) with ~36,000 deaths annually in the UK. Sepsis is a systemic, dysregulated activation of the innate immune system in response to an infection characterised by excessive inflammatory mediator production and oxidative stress. Mitochondrial dysfunction is implicated in sepsis-­‐induced organ dysfunction and death. Zinc is an essential micronutrient with a multitude of biological functions, including anti-­‐inflammatory and antioxidant properties. A relationship has been established between zinc deficiency and severity of sepsis,
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39

Barroso, Joana Barbara de Bessa. ""Obesity and inflammation: associated polymorphisms"." Master's thesis, Faculdade de Medicina da Universidade do Porto, 2008. http://hdl.handle.net/10216/23729.

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40

Cheong, Poh Yue. "Novel imaging targets in inflammation." Thesis, Imperial College London, 2014. http://hdl.handle.net/10044/1/42989.

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Over the last two decades, research into the imaging of inflammation has been thoroughly investigated as it presents a promising therapeutic target for many diseases, including neurodegenerative and cardiovascular diseases. There are only a few radiopharmaceuticals which are currently in general use for imaging inflammation, e.g. 18F-FDG, nuclear labelled autologous white blood cells, 67Ga-citrate etc. However, none is specific for inflammation thus there is a need to investigate novel imaging agents for distinguishing sterile inflammation from infection. Formyl peptide receptors (FPRs) were o
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41

de, Sá Pereira Inês Tavares Pinto. "Developmental response to brain inflammation." Thesis, University of Oxford, 2017. https://ora.ox.ac.uk/objects/uuid:3c1a3270-4eff-42bb-866c-716a9ad30a96.

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Perinatal inflammation contributes to neurodevelopmental diseases, and animal models have revealed that the inflammatory response within the central nervous system is age dependent. It remains unclear what intrinsic and/or extrinsic factors are responsible for this variation. Here, my aim was to discover the mechanisms responsible for the age-dependent changes in the inflammatory response of the brain by injecting interleukin-1 (IL-1&beta;) into the brain of mice at postnatal day (P)7, P14, P21 or into adult mice. A "window of susceptibility" was found at P14, which was associated with marked
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42

Wardlaw, Andrew. "Studies on asthma and inflammation." Thesis, Imperial College London, 1988. http://hdl.handle.net/10044/1/47294.

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43

Warland, D. A. "Inflammation, the microcirculation & microalbuminuria." Thesis, Exeter and Plymouth Peninsula Medical School, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701316.

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44

Charteris, David Graham. "T lymphocytes in intraocular inflammation." Thesis, University of Edinburgh, 1994. http://hdl.handle.net/1842/19619.

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Pathological and immunohistochemical analyses of two forms of human intraocular inflammatory disease, multifocal posterior uveitis and Behcet's disease, were carried out. Studies of post-mortem eyes from a patient with multifocal posterior uveitis revealed a focal lymphocytic infiltrate in the choroid, extending through Bruch's membrane to the retina. Endothelial cells of choroidal vessels at the inflammatory foci had the morphological appearance of "high endothelial venules" known to be involved in the selective trafficking of lymphocytes. Immunohistochemical analysis of the lymphocytic infil
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45

Moran, Gordon William. "Enteroendocrine peptides in intestinal inflammation." Thesis, University of Manchester, 2011. https://www.research.manchester.ac.uk/portal/en/theses/enteroendocrine-peptides-in-intestinal-inflammation(340b8b63-3b70-4ddd-bbce-4c9ab3e593cc).html.

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Introduction: Appetite is often impaired in patients with gastrointestinal inflammation. Up to 75% of hospitalised Crohn's disease (CD) patients are malnourished. Recent animal research has suggested that immune mediated upregulation of enteroendocrine cell (EEC) activity plays a mechanistic role in the appetite and feeding disturbance observed during gut inflammation. The role of EEC in producing factors regulating satiety and intestinal growth is well recognised but work on their use as therapeutic targets or agents in inflammatory bowel disease (IBD) is still in its infancy. EEC peptide dyn
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46

Kim, Sangmi Sandler Robert Samuel. "Obesity, inflammation and colorectal neoplasia." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2007. http://dc.lib.unc.edu/u?/etd,2164.

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Thesis (Ph. D.)--University of North Carolina at Chapel Hill, 2007.<br>Title from electronic title page (viewed Feb. 26, 2009). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Department of Epidemiology, School of Public Health." Discipline: Epidemiology; Department/School: Public Health.
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47

Vallejo, Abigail. "Inhibition of SAA-mediated inflammation." Thesis, University of Sydney, 2020. https://hdl.handle.net/2123/23011.

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The acute phase protein serum amyloid A (SAA) is a biomarker for acute inflammation, however there is growing evidence to suggest that this protein has bioactivity that contributes to the inflammatory process in disease states such as atherosclerosis whereby endothelial dysfunction is initiated. The data generated in this thesis shows that SAA can elicit the production of pro-inflammatory cytokines such as tumour necrosis factor (TNF), tissue factor (TF), interleukin-6 from human endothelial cell lines. The downstream molecular pathways of SAA is a well-characterised series of pro-inflammatory
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48

Barroso, Joana Barbara de Bessa. ""Obesity and inflammation: associated polymorphisms"." Dissertação, Faculdade de Medicina da Universidade do Porto, 2008. http://hdl.handle.net/10216/23729.

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49

Weidmann, Rolf Günter. "Endothel und Regulation der Inflammation." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2005. http://dx.doi.org/10.18452/15424.

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Die durch Lipopolysaccharid (LPS) induzierte frühe Immunantwort ist ein wesentlicher Mechanismus der Infektabwehr durch die angeborene Immunität. Bei starker LPS-Exposition kann es andererseits zur Ausbildung eines septischen Syndroms kommen. Der endothelialen Sekretion von Interleukin-8 (IL-8/CXCL8), das als Chemokin die Migration neutrophiler Granulozyten vermittelt, kommt dabei herausragende Bedeutung zu. Zielsetzung dieser Arbeit war es, die Relevanz der Rho-Proteine RhoA, Rac1 und Cdc42 für die LPS-induzierte intrazelluläre Signaltransduktion mittels Überexpression inaktiver Mutanten dies
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50

Chen, Shao Ru. "Andrographolide analogues inhibit acute inflammation." Thesis, University of Macau, 2018. http://umaclib3.umac.mo/record=b3953265.

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