Academic literature on the topic 'Inflammatory breast cancer (IBC)'

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Journal articles on the topic "Inflammatory breast cancer (IBC)"

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Lynce, Filipa. "Abstract ED4-3: Inflammatory Breast Cancer." Cancer Research 83, no. 5_Supplement (2023): ED4–3—ED4–3. http://dx.doi.org/10.1158/1538-7445.sabcs22-ed4-3.

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Abstract Inflammatory Breast Cancer (IBC) has an incidence of 1-5% among all breast cancers diagnosed within the USA, yet it accounts for a disproportionately high rate of mortality, leading to up to 10% of all breast cancer related deaths. Although IBC can present with any receptor status in terms of estrogen, progesterone, and human epidermal growth factor 2 (HER2), there is a higher proportion of HER2 positive cases compared to non-IBC. Given the distinct difference in outcome, it is important to differentiate IBC from more indolent locally advanced non-IBC with secondary inflammatory featu
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Dushkin, Holly, and Massimo Cristofanilli. "Inflammatory Breast Cancer." Journal of the National Comprehensive Cancer Network 9, no. 2 (2011): 233–41. http://dx.doi.org/10.6004/jnccn.2011.0018.

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Inflammatory breast cancer (IBC) represents the most virulent form of breast cancer, characterized by involvement of the skin and rapid progression of the disease. Management involves careful coordination of multidisciplinary modalities, including imaging, systemic chemotherapy, surgery, and radiation therapy. The use of neoadjuvant chemotherapy has contributed significantly to improvement in overall survival since the first descriptions of this entity, and has made the role of locoregional therapy, including surgery and radiation, critical to continued improvements in this disease. This artic
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Chang, S., A. U. Buzdar, and S. D. Hursting. "Inflammatory breast cancer and body mass index." Journal of Clinical Oncology 16, no. 12 (1998): 3731–35. http://dx.doi.org/10.1200/jco.1998.16.12.3731.

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PURPOSE No studies have investigated the etiology of inflammatory breast cancer (IBC), the most lethal form of breast cancer. Because high body mass index (BMI) is associated with decreased risk of premenopausal breast cancer but increased risk of postmenopausal breast cancer, we evaluated whether high BMI was a risk factor for IBC. PATIENTS AND METHODS In a case-comparison study, we matched by ethnicity and registration date 68 IBC patients treated at The University of Texas M.D. Anderson Cancer Center from 1985 to 1996 with 143 patients with non-IBC and 134 patients with cancer at sites othe
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Wang, Xiaoping, Takashi Semba, Lan Thi Hanh Phi, et al. "Targeting Signaling Pathways in Inflammatory Breast Cancer." Cancers 12, no. 9 (2020): 2479. http://dx.doi.org/10.3390/cancers12092479.

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Inflammatory breast cancer (IBC), although rare, is the most aggressive type of breast cancer. Only 2–4% of breast cancer cases are classified as IBC, but—owing to its high rate of metastasis and poor prognosis—8% to 10% of breast cancer-related mortality occur in patients with IBC. Currently, IBC-specific targeted therapies are not available, and there is a critical need for novel therapies derived via understanding novel targets. In this review, we summarize the biological functions of critical signaling pathways in the progression of IBC and the preclinical and clinical studies of targeting
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Marzogi, Alaa Abdulrahman, Reem Mohammed Noor Kalakattawi, Athal Kamal Filemban, and Amani Mohammad Abbad Alwadei. "A Case Study of an Inflammatory Breast Cancer in Men-lobular Subtype." Journal of Pioneering Medical Sciences 14, no. 1 (2025): 8–14. https://doi.org/10.47310/jpms2025140102.

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Inflammatory breast cancer (IBC) is a rare and aggressive form of breast cancer, accounting for 1–5% of all breast malignancies. Male breast cancer is particularly uncommon, comprising less than 1% of all cases and IBC in males presents even greater diagnostic and therapeutic challenges. This case study highlights a 49-year-old Sudanese male diagnosed with inflammatory breast cancer of the lobular subtype. The patient initially presented with a palpable right breast mass and nipple discharge, without characteristic skin changes often associated with IBC. Diagnostic imaging and biopsy confirmed
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Mohamed, Hossam Taha, Valérie Untereiner, Gianfelice Cinque, et al. "Infrared Microspectroscopy and Imaging Analysis of Inflammatory and Non-Inflammatory Breast Cancer Cells and Their GAG Secretome." Molecules 25, no. 18 (2020): 4300. http://dx.doi.org/10.3390/molecules25184300.

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Glycosaminoglycans (GAGs)/proteoglycans (PGs) play a pivotal role in the metastasis of inflammatory breast cancer (IBC). They represent biomarkers and targets in diagnosis and treatment of different cancers including breast cancer. Thus, GAGs/PGs could represent potential prognostic/diagnostic biomarkers for IBC. In the present study, non-IBC MDA-MB-231, MCF7, SKBR3 cells and IBC SUM149 cells, as well as their GAG secretome were analyzed. The latter was measured in toto as dried drops with high-throughput (HT) Fourier Transform InfraRed (FTIR) spectroscopy and imaging. FTIR imaging was also em
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Baker, Jennifer L., John Hegde, Carlie K. Thompson, Minna K. Lee, and Maggie L. DiNome. "Locoregional Management of Inflammatory Breast Cancer." Current Breast Cancer Reports 12, no. 4 (2020): 326–35. http://dx.doi.org/10.1007/s12609-020-00389-6.

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Abstract Purpose of Review Inflammatory breast cancer (IBC) is a biologically aggressive subtype with a high risk for rapid local progression and early distant metastasis. We review the updated data for optimal locoregional management of IBC, including areas of active controversy. Recent Findings Advancements in tri-modality therapies have improved survival among IBC patients in recent years; however, the risk of locoregional and distant recurrence remains high, particularly in triple-negative IBC. Data to support de-escalation of surgery or radiotherapy is limited, and the recommended treatme
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Zhou, Kaiwen, Ying Lin, Mengmeng Zhang, Nan Shao, and Zhen Shan. "Genomic and transcriptomic profiling of inflammatory breast cancers and non-inflammatory breast cancers in Asian women." Journal of Clinical Oncology 41, no. 16_suppl (2023): e12546-e12546. http://dx.doi.org/10.1200/jco.2023.41.16_suppl.e12546.

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e12546 Background: Inflammatory breast cancer (IBC) is the most aggressive form of breast cancer. Despite its rare incidence, it accounts for 10% of breast cancer-caused mortality. Previous studies based on NGS have focused on western populations and found no common genomic and transcriptomic difference that could distinguish IBC from non-IBC. Methods: We performed WES on 15 IBC samples and their paired normal blood to identify somatic genomic alterations, copy number variants and large structural variants. RNA-seq was performed on 16 IBC samples to examine the RNA expression, pathway enrichme
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Wang, Zheng, Hui Wang, Xinyuan Ding, Xiaosong Chen, and Kunwei Shen. "A large-cohort retrospective study of metastatic patterns and prognostic outcomes between inflammatory and non-inflammatory breast cancer." Therapeutic Advances in Medical Oncology 12 (January 2020): 175883592093267. http://dx.doi.org/10.1177/1758835920932674.

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Background and aims: Breast cancer-related death is attributable mainly to metastasis. Inflammatory breast cancer (IBC) is an infrequent subtype of breast cancer that shows a relatively high rate of metastasis. In this study, we aimed to compare the metastatic patterns and prognostic outcomes of IBC and non-inflammatory breast cancer (non-IBC). Methods: We extracted data between 2010 and 2014 from the Surveillance, Epidemiology and End Results (SEER) database. The Chi-square test and Fisher’s exact test were used to compare the categorical parameters among different groups. Logistic regression
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Howard, F. "Abstract ED11-01: Is inflammatory breast cancer a genomically different type of breast cancer?" Cancer Research 84, no. 9_Supplement (2024): ED11–01—ED11–01. http://dx.doi.org/10.1158/1538-7445.sabcs23-ed11-01.

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Abstract Inflammatory Breast Cancer (IBC) is a unique and aggressive disease, constituting 2 - 4% of breast cancer diagnoses yet responsible for up to 10% of breast cancer related deaths. IBC remains a clinical diagnosis, without highly specific molecular markers, and the genomic drivers of the disease are incompletely characterized. The overrepresentation of human epidermal growth factor 2 amplification and triple-negative disease within IBC have confounded comparisons between IBC and non-IBC. Nonetheless, some consistent genomic observations have emerged that may begin to explain the charact
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Dissertations / Theses on the topic "Inflammatory breast cancer (IBC)"

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Tomlinson, James S. "Molecular basis of the metastatic phenotype of inflammatory breast cancer." Diss., Restricted to subscribing institutions, 2009. http://proquest.umi.com/pqdweb?did=1872078261&sid=1&Fmt=2&clientId=1564&RQT=309&VName=PQD.

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Kanao, Shotaro. "Differentiating benign and malignant inflammatory breast lesions: Value of T2 weighted and diffusion weighted MR images." Kyoto University, 2019. http://hdl.handle.net/2433/236592.

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Aguiar, Marco AntÃnio Nasser. "Expression of nuclear factor Kappa B (NF-KB) and Interleukin-18 (IL-18) in inflammatory breast cancer and locally advanced breast cancer." Universidade Federal do CearÃ, 2015. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=15328.

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CoordenaÃÃo de AperfeÃoamento de Pessoal de NÃvel Superior<br>Inflammatory Breast Cancer (IBC) is the most aggressive form of locally advanced breast cancer. It is more common in young women and unfavorably, in most of the times, evolves quickly. Presents with typical signs of inflammation such as hyperemia and hyperthermia its pathogenesis and evolution has been associated with possible participation of inflammatory mediators such as cytokines (TNF-&#945; e IL-1&#946;), enzymes (cyclooxygenase-2 [COX-2] and nitric oxide synthase [iNOS]), as well as, transcription factors (Nuclear Factor kappa
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Birnbaum-Weitzman, Orit. "CBSM Effects on Sickness Behavior and Pro-Inflammatory Cytokine Mechanisms in Breast Cancer Survivors." Scholarly Repository, 2009. http://scholarlyrepository.miami.edu/oa_dissertations/297.

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The concept of sickness behavior offers a framework to view both the neurovegetative and psychological symptoms that accompany illness as a common entity that results from increased inflammatory activation. Despite the prevalence of sickness behavior in medical populations, to our knowledge this study provides the first attempt to develop a standardized measure to assess sickness behavior using standard self-report questionnaires commonly used with cancer patients. The set of items included in the measure match theoretical conceptualizations of sickness behavior and target symptoms that compri
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Hanna, Mirette. "Study on the associations between physical activity, inflammatory markers and two breast cancer risk factors." Doctoral thesis, Université Laval, 2015. http://hdl.handle.net/20.500.11794/27543.

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L'activité physique peut diminuer le risque de cancer du sein par un effet anti-inflammatoire. Nous avons examiné: 1. L'association de l'activité physique avec l'expression protéinique des marqueurs pro-inflammatoires (IL-6, TNF-α, CRP, COX-2, leptine, SAA1, IL-8 et STAT3) et anti-inflammatoires (TGF-β, IL-10 et lactoferrine) dans le tissu mammaire normal; 2. L'association de différents niveaux d'expression protéinique des marqueurs inflammatoires avec deux facteurs de risque de cancer du sein: la densité mammaire et l’involution lobulaire liée à l'âge; 3. L'association de l'activité physique
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Al, Murri Afra M. S. "An investigation of the relationship between the inflammatory response and outcome in patients with breast cancer." Thesis, University of Glasgow, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.443211.

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Diaz-Cruz, Edgar S. "Interrelationships between aromatase and cyclooxygenase-2 and their role in the autocrine and paracrine mechanisms in breast cancer." Connect to resource, 2005. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1117117648.

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Massó, Vallés Daniel. "Inhibiting Myc and the Myc dependent inflammatory response as cancer therapies." Doctoral thesis, Universitat Autònoma de Barcelona, 2017. http://hdl.handle.net/10803/458137.

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Aquesta tesi s’ha realitzat al Laboratori de Modelització de Teràpies Anti-tumorals en Ratolí dirigit per la Dra. Laura Soucek al Programa de Recerca Preclínica del Vall d’Hebron Institut d’Oncologia (VHIO), i comprèn dos projectes diferenciats centrats en dues de les funcions oncogèniques de Myc. Pel que fa al primer projecte, hem validat farmacològicament la inhibició dels mastòcits com a estratègia terapèutica contra l’adenocarcinoma ductal de pàncrees (PDAC). Per a fer-ho, hem utilitzat ibrutinib, un inhibidor de la tirosina quinasa de Bruton (BTK) en models de ratolí de PDAC, el tumor de
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Cheng, Huiwen. "Effects of N-acetyl-L-cysteine and Nitric Oxide-releasing Nonsteroidal Anti-Inflammatory Drugs on Breast Cancer and Melanoma Cell Adhesion." Ohio University / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1384948906.

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Ahmed, Khairiya O. "Relationship between altered myoepithelial phenotype and the inflammatory cell infiltrate in progression of DCIS." Thesis, Queen Mary, University of London, 2015. http://qmro.qmul.ac.uk/xmlui/handle/123456789/7867.

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Changes in the microenvironment have been implicated in the transition of pre-invasive ductal carcinoma in-situ (DCIS) to invasive breast cancer. Normal myoepithelial cells have a tumour suppressor phenotype but they are altered in DCIS and ultimately lost with transition to invasive cancer. A consistent change in DCIS is up-regulation of the integrin αvβ6 in myoepithelial cells. Preliminary observations identified a correlation between myopeithelial αvβ6 and an increased peri-ductal inflammatory infiltrate. The hypothesis of this study is that the altered myoepithelial phenotype influences th
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Books on the topic "Inflammatory breast cancer (IBC)"

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de la Garza-Salazar, Jaime G., Abelardo Meneses-Garcia, and Claudia Arce-Salinas, eds. Inflammatory Breast Cancer. Springer London, 2013. http://dx.doi.org/10.1007/978-0-85729-991-8.

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Ueno, Naoto T., and Massimo Cristofanilli, eds. Inflammatory Breast Cancer: An Update. Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-3907-9.

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Ueno, Naoto T. Inflammatory Breast Cancer: An Update. 2nd ed. Springer Netherlands, 2012.

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Inflammatory Breast Cancer. Anshan, 2010.

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Jaime G. de la Garza-Salazar, Abelardo Meneses-Garcia, and Claudia Arce-Salinas. Inflammatory Breast Cancer. Springer, 2012.

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Jaime G. de la Garza-Salazar, Abelardo Meneses-Garcia, and Claudia Arce-Salinas. Inflammatory Breast Cancer. Springer London, Limited, 2016.

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Jaime G. de la Garza-Salazar, Abelardo Meneses-Garcia, and Claudia Arce-Salinas. Inflammatory Breast Cancer. Springer, 2012.

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Swain, S. M. Inflammatory Breast Cancer (Breast Disease) (Breast Disease). IOS Press, 2006.

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Ueno, Naoto T., and Tamara Fink. Inflammatory Breast Cancer: An Update. Springer, 2012.

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Ueno, Naoto T., and Tamara Fink. Inflammatory Breast Cancer: An Update. Springer, 2014.

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Book chapters on the topic "Inflammatory breast cancer (IBC)"

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Ueno, Naoto T., and Massimo Cristofanilli. "Introduction: Why IBC Now?" In Inflammatory Breast Cancer: An Update. Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-3907-9_1.

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Wong, Ching Yee Oliver, and Dafang Wu. "Case 70: Inflammatory Breast Cancer (IBC)." In Phenotypic Oncology PET. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-09737-9_70.

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Yang, Wei Tse. "Imaging for the Diagnosis and Staging of IBC." In Inflammatory Breast Cancer: An Update. Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-3907-9_6.

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Barsky, Sanford H., and Fredika M. Robertson. "Pathology: Histomorphometrical Features of IBC – Angiogenesis, Lymphangiogenesis, and Tumor Emboli." In Inflammatory Breast Cancer: An Update. Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-3907-9_5.

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Vermeulen, Peter B., Gert Van den Eynden, Pascal Finetti, et al. "Angiogenesis and Lymphangiogenesis in IBC: Insights from a Genome-Wide Gene Expression Profiling Study." In Inflammatory Breast Cancer: An Update. Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-3907-9_18.

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Fleming, R. Y. Declan, and S. Eva Singletary. "Inflammatory Breast Cancer." In Breast Cancer. Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4612-2146-3_23.

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Guler, Nilufer. "Inflammatory Breast Cancer." In Breast Cancer. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-96947-3_11.

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Alvarez, Ricardo Hugo, Shaheenah Dawood, and Massimo Cristofanilli. "Inflammatory Breast Cancer." In Breast Disease. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1145-5_15.

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Robertson, Fredika M., and Massimo Cristofanilli. "Inflammatory Breast Cancer." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27841-9_6667-2.

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Gooch, Jessica C., and Freya Schnabel. "Inflammatory Breast Cancer." In Clinical Algorithms in General Surgery. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-98497-1_28.

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Conference papers on the topic "Inflammatory breast cancer (IBC)"

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El-Sayed, Shrouk Khalaf, Salwa Sabet, Aya Ali El-Sharkawy, et al. "Enhancing Acurate Diagnosis of Inflammatory Breast Cancer Based on Polar Fox Optimization." In 2024 International Conference on Smart-Digital-Green Technologies and Artificial Intelligence Sciences (CSDGAIS). IEEE, 2024. https://doi.org/10.1109/csdgais64098.2024.11064786.

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Fernandez, Sandra V., Zhaomei Mu, Lucy Aburto, et al. "Abstract 5342: FC-IBC-02: A newin vitro-in vivomodel of inflammatory breast cancer (IBC)." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5342.

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Fernandez, Sandra V., Fredika M. Robertson, Sankar Addya, Zhaomei Mu, and Massimo Cristofanilli. "Abstract 4988: Clues for targeted therapies in inflammatory breast cancer (IBC)." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4988.

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Hall, CS, BG Debeb, W. Xu, et al. "Novel inflammatory breast cancer cell line, MDA-IBC-1 expresses and secretes WISP3, a putative tumor suppressor in inflammatory breast cancer." In CTRC-AACR San Antonio Breast Cancer Symposium: 2008 Abstracts. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-1051.

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Caceres, Sara, Laura Peña, Maria J. Illera, et al. "Abstract 651: Male mouse model of canine and human inflammatory breast cancer (IBC)." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-651.

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Dawood, S., N. Ueno, V. Valero, et al. "Brain Metastases in Women with Inflammatory Breast Cancer (IBC): Incidence, Treatment and Outcome." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-2102.

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Kaufman, B., T. Bachelot, Y. Wu, et al. "Lapatinib monotherapy in HER2+ relapsed or refractory inflammatory breast cancer (IBC): quality of life (QOL) assessment." In CTRC-AACR San Antonio Breast Cancer Symposium: 2008 Abstracts. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-4097.

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Batty, Nicolas, Pedro Cano, Pam Alizadeh, et al. "Abstract 3812: Innate immunity in inflammatory breast cancer (IBC) patients: Clues from HLA profiling." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3812.

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Yeh, E., A. Rives, H. Guo, et al. "Abstract P4-02-08: MRI changes in breast skin following preoperative therapy for inflammatory breast cancer (IBC)." In Abstracts: Thirty-Eighth Annual CTRC-AACR San Antonio Breast Cancer Symposium; December 8-12, 2015; San Antonio, TX. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.sabcs15-p4-02-08.

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Iwase, T., K. Harano, H. Masuda, et al. "Abstract P5-05-04: Myc as a poor prognostic marker for ER+ inflammatory breast cancer (IBC): Quantitative estrogen receptor (ER) expression analysis and gene expression analysis in ER+ IBC vs non-IBC." In Abstracts: 2018 San Antonio Breast Cancer Symposium; December 4-8, 2018; San Antonio, Texas. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-p5-05-04.

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Reports on the topic "Inflammatory breast cancer (IBC)"

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Merajver, Sofia. Genetic Determinants of Inflammatory Breast Cancer. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada397618.

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Diergaarde, Brenda. Inflammatory Markers and Breast Cancer Risk. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada491235.

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Merajver, Sofia D. Genetic Determinants of Inflammatory Breast Cancer. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada429666.

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Diergaarde, Brenda, Eleanor Feingold, and Robert Ferrell. Inflammatory Markers and Breast Cancer Risk. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada542413.

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Merajver, Sofia D. Genetic Determinants of Inflammatory Breast Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada411488.

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Merajver, Sofia D. Genetic Determinants of Inflammatory Breast Cancer. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada425765.

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Diergaarde, Brenda, Eleanor Feingold, Russell Tracy, and Robert Ferrell. Inflammatory Markers and Breast Cancer Risk. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada559252.

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Diergaarde, Brenda. Inflammatory Markers and Breast Cancer Risk. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada559254.

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Allinen, Minna, and Kornelia Polyak. Characterization of IBC-1, a Novel Putative Breast Cancer Prognostic and Survival Factor. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada427828.

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Brown, Thomas R. Novel Serum Inflammatory Biomarkers for Early Detection of Breast Cancer. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada488851.

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