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Dissertations / Theses on the topic 'Inflammatory breast cancer (IBC)'

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Published: 3 June 2025
Last updated: 31 July 2025

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1

Tomlinson, James S. "Molecular basis of the metastatic phenotype of inflammatory breast cancer." Diss., Restricted to subscribing institutions, 2009. http://proquest.umi.com/pqdweb?did=1872078261&sid=1&Fmt=2&clientId=1564&RQT=309&VName=PQD.

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2

Kanao, Shotaro. "Differentiating benign and malignant inflammatory breast lesions: Value of T2 weighted and diffusion weighted MR images." Kyoto University, 2019. http://hdl.handle.net/2433/236592.

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3

Aguiar, Marco AntÃnio Nasser. "Expression of nuclear factor Kappa B (NF-KB) and Interleukin-18 (IL-18) in inflammatory breast cancer and locally advanced breast cancer." Universidade Federal do CearÃ, 2015. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=15328.

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CoordenaÃÃo de AperfeÃoamento de Pessoal de NÃvel Superior<br>Inflammatory Breast Cancer (IBC) is the most aggressive form of locally advanced breast cancer. It is more common in young women and unfavorably, in most of the times, evolves quickly. Presents with typical signs of inflammation such as hyperemia and hyperthermia its pathogenesis and evolution has been associated with possible participation of inflammatory mediators such as cytokines (TNF-&#945; e IL-1&#946;), enzymes (cyclooxygenase-2 [COX-2] and nitric oxide synthase [iNOS]), as well as, transcription factors (Nuclear Factor kappa
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4

Birnbaum-Weitzman, Orit. "CBSM Effects on Sickness Behavior and Pro-Inflammatory Cytokine Mechanisms in Breast Cancer Survivors." Scholarly Repository, 2009. http://scholarlyrepository.miami.edu/oa_dissertations/297.

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The concept of sickness behavior offers a framework to view both the neurovegetative and psychological symptoms that accompany illness as a common entity that results from increased inflammatory activation. Despite the prevalence of sickness behavior in medical populations, to our knowledge this study provides the first attempt to develop a standardized measure to assess sickness behavior using standard self-report questionnaires commonly used with cancer patients. The set of items included in the measure match theoretical conceptualizations of sickness behavior and target symptoms that compri
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5

Hanna, Mirette. "Study on the associations between physical activity, inflammatory markers and two breast cancer risk factors." Doctoral thesis, Université Laval, 2015. http://hdl.handle.net/20.500.11794/27543.

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L'activité physique peut diminuer le risque de cancer du sein par un effet anti-inflammatoire. Nous avons examiné: 1. L'association de l'activité physique avec l'expression protéinique des marqueurs pro-inflammatoires (IL-6, TNF-α, CRP, COX-2, leptine, SAA1, IL-8 et STAT3) et anti-inflammatoires (TGF-β, IL-10 et lactoferrine) dans le tissu mammaire normal; 2. L'association de différents niveaux d'expression protéinique des marqueurs inflammatoires avec deux facteurs de risque de cancer du sein: la densité mammaire et l’involution lobulaire liée à l'âge; 3. L'association de l'activité physique
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6

Al, Murri Afra M. S. "An investigation of the relationship between the inflammatory response and outcome in patients with breast cancer." Thesis, University of Glasgow, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.443211.

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7

Diaz-Cruz, Edgar S. "Interrelationships between aromatase and cyclooxygenase-2 and their role in the autocrine and paracrine mechanisms in breast cancer." Connect to resource, 2005. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1117117648.

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8

Massó, Vallés Daniel. "Inhibiting Myc and the Myc dependent inflammatory response as cancer therapies." Doctoral thesis, Universitat Autònoma de Barcelona, 2017. http://hdl.handle.net/10803/458137.

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Aquesta tesi s’ha realitzat al Laboratori de Modelització de Teràpies Anti-tumorals en Ratolí dirigit per la Dra. Laura Soucek al Programa de Recerca Preclínica del Vall d’Hebron Institut d’Oncologia (VHIO), i comprèn dos projectes diferenciats centrats en dues de les funcions oncogèniques de Myc. Pel que fa al primer projecte, hem validat farmacològicament la inhibició dels mastòcits com a estratègia terapèutica contra l’adenocarcinoma ductal de pàncrees (PDAC). Per a fer-ho, hem utilitzat ibrutinib, un inhibidor de la tirosina quinasa de Bruton (BTK) en models de ratolí de PDAC, el tumor de
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9

Cheng, Huiwen. "Effects of N-acetyl-L-cysteine and Nitric Oxide-releasing Nonsteroidal Anti-Inflammatory Drugs on Breast Cancer and Melanoma Cell Adhesion." Ohio University / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1384948906.

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10

Ahmed, Khairiya O. "Relationship between altered myoepithelial phenotype and the inflammatory cell infiltrate in progression of DCIS." Thesis, Queen Mary, University of London, 2015. http://qmro.qmul.ac.uk/xmlui/handle/123456789/7867.

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Changes in the microenvironment have been implicated in the transition of pre-invasive ductal carcinoma in-situ (DCIS) to invasive breast cancer. Normal myoepithelial cells have a tumour suppressor phenotype but they are altered in DCIS and ultimately lost with transition to invasive cancer. A consistent change in DCIS is up-regulation of the integrin αvβ6 in myoepithelial cells. Preliminary observations identified a correlation between myopeithelial αvβ6 and an increased peri-ductal inflammatory infiltrate. The hypothesis of this study is that the altered myoepithelial phenotype influences th
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11

Sistigu, Antonella. "Inflammatory and immune reactions in response to chemotherapy-induced cell death. Viral mimicry chemotherapy : ds RNA sensors and IFNAR signalling indispensable for immunogenic tumor cell death." Thesis, Paris 11, 2013. http://www.theses.fr/2013PA11T052.

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Certains motifs moléculaires associés à la mort cellulaire semblent identifier les cancers prompts à répondre à une thérapie cytotoxique. Ceci en élaborant une réponse anti-tumorale basées sur une réponse T protectrice. Mon travail de thèse montre que le traitement par chimiothérapie immunogène active des voies moléculaires mimant une infection virale. Ceci conduit au niveau des cellules tumorales à une signalisation autocrine via l’IFNαβ / IFNAR1/2, initiée par la reconnaissance d’ARN double brin (dsRNA) endogène par les Récepteurs endosomaux de Reconnaissance des Motifs (PRRs). De façon plus
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12

Villafuerte, Kelly Rocio Vargas. "Avaliação das condições periodontais em mulheres portadoras de câncer de mama sob tratamento quimioterápico." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/58/58132/tde-17062014-100807/.

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Objetivo: Este trabalho avaliou as condições periodontais em mulheres com câncer de mama, sob tratamento quimioterápico, e monitorou o efeito do tratamento periodontal nas condições clínicas e sistêmicas dessas pacientes. Materiais e Métodos As pacientes foram divididas em quatro grupos: pacientes controles saudáveis (SAU) (n=16); pacientes com câncer e sem doença periodontal (CAN) (n=15); pacientes que não tinham câncer porém que tinham doença periodontal (DP) (n=18); e pacientes com câncer e com doença periodontal (CAN-DP) (n=10). Os parâmetros clínicos avaliados foram: Índice de Placa (IP)
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13

Manai, Marwa. "Caractérisation des altérations moléculaires dans le cancer du sein inflammatoire." Thesis, Aix-Marseille, 2016. http://www.theses.fr/2016AIXM5067.

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Le cancer du sein inflammatoire (CSI) est l’une des formes la plus agressive en raison de son potentiel métastatique élevé. Le diagnostic est basé sur des signes cliniques avec une émergence rapide, mais en réalité ces signes sont subjectifs et non spécifiques. Malgré le traitement multidisciplinaire, le pronostic reste mauvais avec une survie à 5 ans inférieure à 50% vs 90% dans le cancer du sein non inflammatoire (CSNI). Les CSI sont rares en Europe et aux Etats-Unis, représentant moins de 2% du CS. En Afrique du Nord et en particulier en Tunisie, ils représentent plus de 5% des CS. Notre éq
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14

Climente, González Héctor. "Études d'association génome entier guidées par des réseaux." Thesis, Université Paris sciences et lettres, 2020. http://www.theses.fr/2020UPSLM001.

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Cette thèse s'intéresse à un ensemble de méthodes utilisées pour identifier les causes génétiques de maladies complexes. Les méthodes d'association génome entier (GWAS), sont généralement utilisées pour étudier des associations univariées, tandis que les méthodes d'association d'interactions génome entier (GWAIS) prennent en considération des interactions entre facteurs génétiques (ou épistasie). Cependant, ces deux approches présentent plusieurs défis, parmi lesquels leur faible puissance statistique, la difficulté de leur interprétation, ainsi que les choix arbitraires qui doivent être faits
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15

Evans, Myron K. "Structural and Functional Analysis of the Caspase –dependent and –independent Domains of the X-linked Inhibitor of Apoptosis Protein in Inflammatory Breast Cancer Tumor Biology." Diss., 2016. http://hdl.handle.net/10161/12113.

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<p>Inflammatory breast cancer (IBC) is an extremely rare but highly aggressive form of breast cancer characterized by the rapid development of therapeutic resistance leading to particularly poor survival. Our previous work focused on the elucidation of factors that mediate therapeutic resistance in IBC and identified increased expression of the anti-apoptotic protein, X-linked inhibitor of apoptosis protein (XIAP), to correlate with the development of resistance to chemotherapeutics. Although XIAP is classically thought of as an inhibitor of caspase activation, multiple studies have revealed t
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16

Hashim, Che Gon. "Identifying predictors of postoperative persistent pain in women with breast cancer: assessments of investigative tools." Master's thesis, 2018. http://hdl.handle.net/1885/162744.

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Persistent pain after surgery in breast cancer has a significant impact on the patient’s survival. The value of escalating research on breast cancer in Malaysia cannot be underestimated. However, it is not known how many of these women experience persistent pain after surgery. This study surveyed previously unknown figures on prevalence, and explored the predictive factors of persistent pain women with breast cancer in Malaysia. There were three objectives. First, to assess the reliability of the already established investigative tools, namely, the Brief
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17

FRALICK, JOHN. "The Incidence, Management, and Outcome of Inflammatory Breast Cancer." Thesis, 2009. http://hdl.handle.net/1974/5210.

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Background: Inflammatory breast cancer (IBC) is a rare form of breast cancer associated with a poor prognosis. This study describes the incidence, survival, and management of IBC in the province of Ontario. Methods: We conducted a retrospective, population-based, cohort study using data systems held at the Division of Cancer Care and Epidemiology at Queen’s University in Kingston, Ontario. Using the Ontario Cancer Registry (OCR), we identified all primary, pathologically confirmed cases of breast cancer. IBC cases were identified using the unique histology code ‘85303’. OCR records were
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18

Putcha, Preeti. "HDAC6 as a novel candidate in the treatment of Inflammatory Breast Cancers." Thesis, 2015. https://doi.org/10.7916/D81835KX.

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Inflammatory Breast Cancer (IBC) is a rare, lethal, and understudied form of breast cancer. Although affecting 1-2% of the population, the remission rate is half that of the spectrum of other breast cancers, and most cases present in the advanced stages due to rapid undetectable development. Of the diagnosed cases, systemic chemotherapeutics are relatively ineffective in comparison to non-IBC breast cancer cases, indicating other unique mechanisms driving IBC progression. Historically, the specific sensitivities of a particular tumor type or subtype have been linked to genetic alterations that
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19

Allensworth, Jennifer. "Identification and Targeting of Therapeutic Resistance Mechanisms in Inflammatory Breast Cancer." Diss., 2013. http://hdl.handle.net/10161/8087.

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<p>Inflammatory breast cancer (IBC) is a rare and highly aggressive form of breast cancer that is characterized by survival signaling through overexpression and/or activation of the epidermal growth factor receptors EGFR/ErbB1 and Her2/ErbB2 and defects in the apoptotic program. The development of therapeutic resistance is a significant barrier to successful treatment in IBC, and thus, strategies targeting the mechanisms that drive drug resistance could prevent or reverse therapeutic resistance, significantly improving patient prognosis. Based on analysis of previously developed models of ther
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20

White, Randie Elizabeth. "Risk factors for developing inflammatory breast cancer: an epidemiological study of a single patient population." Thesis, 2014. https://hdl.handle.net/2144/14382.

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Inflammatory breast cancer (IBC) is a rare and aggressive form of breast cancer, with a particularly poor prognosis. Identification of epidemiologic risk factors for IBC might shed light on causes of the disease, and guide screening and perhaps treatment. Previous studies have suggested that race, geographic location, body mass index (BMI), menopausal status, age at menarche, parity, duration of lactation, and exposure to mouse mammary tumor virus may be key risk factors in the development of IBC. This retrospective epidemiologic study examines the risk factors for IBC in predominantly Caucasi
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21

West, Nathaniel R. "Novel roles of the inflammatory cytokine Oncostatin-M in breast cancer pathogenesis." Thesis, 2012. http://hdl.handle.net/1828/4000.

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Despite ongoing advancement in detection and treatment, breast cancer remains a major clinical challenge worldwide. Cancer has traditionally been conceptualized as a ‘disease of the genes’ by virtue of the mutagenic events necessary for its inception. It is now clear, however, that complex interactions take place between cancer cells and the array of non-cancerous cells and molecules in their immediate surroundings, known generally as the tumour microenvironment. Cancer-microenvironment interactions are increasingly recognized as processes that critically influence the outcome of disease. Ce
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22

Ximenes, Frota Máximo Ilane. "The impact of nonsteroidal anti-inflammatory drugs on endocrine therapy outcomes in breast cancer patients." 2013. http://hdl.handle.net/2152/22501.

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Obesity is a known risk factor for postmenopausal breast cancer, and is associated with worse disease prognosis in pre- and postmenopausal women. Adjuvant hormonal therapies improve disease prognosis in obese women, but many still recur. Given that obesity induces inflammation and increases levels of cyclooxygenase-2 (COX-2) enzyme, resulting in tumor proliferation, this retrospective study investigated if women on anti-inflammatory drugs would have improved disease outcomes by reduced production of prostaglandins by COX-2 pathway. Four hundred and forty women treated for invasive breast cance
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23

Liao, Chu-Chun, and 廖竹鈞. "Research on genetic polymorphisms of obesity-related inflammatory cytokines and breast cancer risk." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/30077669844422610939.

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碩士<br>國防醫學院<br>公共衛生學研究所<br>98<br>Background: Obesity induces different chronic disease through adipose tissue secrete various adipokines and cytokines. TNF-α、IL-6 and IL-1, kinds of pro-inflammatory cytokines, are derived by adipose tissue. These cytokines which induce chronic inflammatory environment and other oncogenic pathway could represent a major role link between obesity and breast cancer. Therefore, either single nucleotide polymorphism (SNP) of TNF-α and IL-6 or variable number of tandem repeat (VNTR) of IL-1RN may increase breast cancer risk by effect circular concentration of cytoki
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24

BRAVATA', Valentina. "Cell, molecular and inflammatory breast cancer response to IntraOperative Electron Radiation Therapy (IOERT)." Doctoral thesis, 2016. http://hdl.handle.net/10447/160618.

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25

Arbizu, Berrocal Shirley Heidi. "Anti-inflammatory and Cytotoxic Activities of Mango (Mangifera indica L. var Keitt) Polyphenols in Cancer and Non-cancer Breast Fibroblasts in Vitro." Thesis, 2013. http://hdl.handle.net/1969.1/151359.

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Breast cancer is the leading cause of cancer death among women worldwide and polyphenols are under investigation as an alternative to conventional treatment approaches of breast cancer. The anti-inflammatory and anti-proliferative activities of polyphenols have been demonstrated in many studies, yet cellular targets and the underlying cellular mechanisms remain unclear. The overall goal of this study was to investigate the anti-inflammatory and cytotoxic properties of polyphenol compounds extracted from the mango variety Keitt in MCF-12A breast non-cancer and MDA-MB231 breast cancer cells by
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26

Aird, Katherine Marie. "Role of X-Linked Inhibitor of Apoptosis Protein in Therapeutic Resistance of Inflammatory Breast Cancer Cells." Diss., 2010. http://hdl.handle.net/10161/2991.

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<p>Apoptotic dysregulation is a hallmark of cancer cells. The inability of cancer cells to undergo apoptosis may lead to therapeutic resistance. Inflammatory breast cancer (IBC) is a highly aggressive subtype of breast cancer that is often characterized by ErbB2 overexpression and ErbB2 activation. ErbB-targeting is clinically relevant using trastuzumab (anti-ErbB2 antibody) and lapatinib (small molecule ErbB1/2 inhibitor). However, acquired resistance is a common outcome even in IBC patients who show an initial clinical response, which limits the efficacy of these agents. Little is known a
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27

Tsai, Wen-Chen, and 蔡汶真. "NAD(P)+-Dependent-Malic Enzyme Suppresses Senescence and Senescence-Inflammatory Response in Human Breast Cancer Cells." Thesis, 2014. http://ndltd.ncl.edu.tw/handle/57062774735410743132.

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碩士<br>國立中興大學<br>生命科學系所<br>102<br>Malic enzyme (Malic Enzyme；ME) has three isoform, which mitochondrial malic enzyme 2 (Mitochondrial NAD(P)+-dependent Malic Enzyme；ME2) affected on the rapid proliferation of tissue, intestinal mucosa, spleen, thymus and tumor cells. ME2 catalytic substrates malate generate pyrevate, accompanied produce NADPH. NADPH in the mitochondrial membrane electron transport chain via oxidative phosphorylation to produce ATP provides direct role in cellular energy. In addition, ME2 also via glutamine metabolic pathway, the extra produce NADPH. The p53 is a tumor suppresso
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28

Wang, Chih-Husan, and 王之璇. "The effect of aspirin inhibits obesity-related inflammatory mediators promoting the growth of breast cancer cell." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/9fx958.

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29

Lin, Ting-Chun. "Influences of Antroquinonol and 4-Acetylantroquinonol B on Inflammatory Tumorigenesis in the MCF-7 Breast Cancer Cell Line with or without TNF-α Stimulation". 2018. https://scholarworks.umass.edu/masters_theses_2/721.

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Breast cancer (BC) is one of the most common cancers among women worldwide that ~25% of new cancer cases diagnosed every year would be BC; moreover, ~15% of cancer deaths per year caused by BC makes it the leading cause of cancer death among women worldwide. To date, though the cause of a large proportion of BC are still unclear, recent studies have revealed that a supportive breast tissue microenvironment is critical for the development and progression of BC, especially the communication with immune cells within breast tissue. Therefore, breast inflammatory microenvironment is currently recei
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30

Roubert, Agathe A. "Obesity-Induced Inflammation in Human Mammary Tissue: A Potential Microenvironment Favorable to the Development of Postmenopausal Breast Cancer Via the Wnt Signaling Pathway." 2015. https://scholarworks.umass.edu/masters_theses_2/296.

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In the United States, over one third of adult women are obese, and one in eight women will be diagnosed with breast cancer in their lifetime. Obesity has been shown to be a risk factor for postmenopausal breast cancer and is associated with increased aggressiveness and poor prognosis regardless of menopausal status. However, the mechanisms involved in the relationship between obesity and breast cancer are still not fully understood. Wnt signaling is often elevated in breast tumors (~60%) and is suspected to play a key role in cancer development. It has been shown that inflammatory cytokines, s
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31

Marques, Oriana Alexandra de Paixão Praças. "Iron and related proteins in the breast cancer microenvironment: expression patterns in epithelial and stromal inflammatory cells and association with clinicopathological markers of behavior and progression." Doctoral thesis, 2016. https://repositorio-aberto.up.pt/handle/10216/83665.

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32

Marques, Oriana Alexandra de Paixão Praças. "Iron and related proteins in the breast cancer microenvironment: expression patterns in epithelial and stromal inflammatory cells and association with clinicopathological markers of behavior and progression." Tese, 2016. https://repositorio-aberto.up.pt/handle/10216/83665.

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