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Journal articles on the topic 'Inflammatory reactions'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

Chung, King Lueh, Cormac Convery, Ifeoma Ejikeme, and Ali M. Ghanem. "A Systematic Review of the Literature of Delayed Inflammatory Reactions After Hyaluronic Acid Filler Injection to Estimate the Incidence of Delayed Type Hypersensitivity Reaction." Aesthetic Surgery Journal 40, no. 5 (2019): NP286—NP300. http://dx.doi.org/10.1093/asj/sjz222.

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Abstract Background Hyaluronic acid (HA) dermal filler injection is believed to be a safe procedure. However, with the increase in the number of performed procedures and indications, the number of product-related complications, especially delayed inflammatory reactions, has also increased. Delayed-type hypersensitivity (DTH) reaction is one of these delayed inflammatory reactions, which is preventable by performing a pretreatment skin test. Objectives The authors sought to find the incidence of delayed inflammatory reactions and DTH reaction after HA injection and to determine whether a pretre

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2

Kambara, Takeshi. "Inflammatory Reactions and Cytokines." Ensho 13, no. 4 (1993): 313–14. http://dx.doi.org/10.2492/jsir1981.13.313.

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3

Scheffer, J., and W. König. "Cephalosporins and Inflammatory Host Reactions." Respiration 60, no. 1 (1993): 25–31. http://dx.doi.org/10.1159/000196246.

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Carter, RyaLynn M., Brooke Halliwell, and Gerald Harkins. "Pelvic Inflammatory Reactions to Floseal." Journal of Minimally Invasive Gynecology 18, no. 3 (2011): 284. http://dx.doi.org/10.1016/j.jmig.2010.03.021.

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Silveira, Camila Maggi Maia, Shelon Cristina Souza Pinto, Rosário de Arruda Moura Zedebski, Fábio André Santos, and Gibson Luiz Pilatti. "Biocompatibility of four root canal sealers: a histopathological evaluation in rat subcutaneous connective tissue." Brazilian Dental Journal 22, no. 1 (2011): 21–27. http://dx.doi.org/10.1590/s0103-64402011000100003.

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The aim of this study was to evaluate the subcutaneous biocompatibility of: Epiphany, AH Plus, Pulp Canal Sealer and Sealapex root canal sealers. Sixty rats were randomly assigned to 4 groups, according to the sealer. Polyethylene tubes containing the tested materials were inserted into the connective tissue. The implants were removed after 7, 15 and 30 days, and the tissue samples were processed, stained and examined by light microscopy. The descriptive analysis considered: thickness of the fibrous capsule, severity of the inflammatory reaction, and presence of giant cells. After 7 days, all

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Ikonnikova, Evgeniya V., Nataliya E. Manturova, and Larisa S. Kruglova. "Delayed inflammatory reaction of hyaluronic acid fillers against the background of flu-like illness." Russian Journal of Skin and Venereal Diseases 24, no. 1 (2021): 93–100. http://dx.doi.org/10.17816/dv61472.

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Traditionally, it is considered that inflammatory reactions with delayed onset that occur due to the use of fillers based on hyaluronic acid (HA) are included in the category of rare complications. But in recent years, due to the increasing popularity of injection procedures, such reactions have become more common, which attracts the attention of both the scientific community and clinicians. This article presents a clinical case of a late inflammatory response, describes the diagnosis and tactics of therapy, and provides an overview of the etiology, pathogenesis, diagnosis, and therapy o

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7

Placha, Daniela, and Josef Jampilek. "Chronic Inflammatory Diseases, Anti-Inflammatory Agents and Their Delivery Nanosystems." Pharmaceutics 13, no. 1 (2021): 64. http://dx.doi.org/10.3390/pharmaceutics13010064.

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Inflammatory diseases, whether caused by excessive stress on certain tissues/parts of the body or arising from infections accompanying autoimmune or secondary diseases, have become a problem, especially in the Western world today. Whether these are inflammations of visceral organs, joints, bones, or the like, they are always a physiological reaction of the body, which always tries to eradicate noxious agents and restore tissue homeostasis. Unfortunately, this often results in damage, often irreversible, to the affected tissues. Nevertheless, these inflammatory reactions of the body are the res

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8

Makarov, I. Yu, and R. A. Barabash. "MORPHOLOGY OF INFLAMMATORY REACTIONS IN THE LUNG TISSUE WITH DRUG-RESISTANT TUBERCULOSIS." Amur Medical Journal, no. 15-16 (2016): 85–86. http://dx.doi.org/10.22448/amj.2016.15-16.85-86.

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9

Dharmaraj, Rajmohan, Tess Pei Lemon, Rasha Elmaoued, Ricardo Orlando Castillo, and Razan Alkhouri. "Infusion Reactions to Infliximab in Pediatric Patients with Inflammatory Bowel Disease." Children 11, no. 11 (2024): 1366. http://dx.doi.org/10.3390/children11111366.

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Infliximab (IFX) is a recombinant DNA-derived chimeric IgG monoclonal antibody protein that inhibits tumor necrosis factor alpha (TNF-α). IFX, like other agents derived from foreign proteins, can cause infusion reactions both during and after the infusion. The incidence of infusion reactions ranges between 0% and 15% in pediatric patients. The potential underlying mechanisms for these reactions may include anaphylaxis and anaphylactoid reactions, cytokine release syndrome, serum sickness-like reactions, and the development of antibodies against IFX. Several precautions can help reduce the risk

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10

Orellana, Karen, Alejandro Carías, Wendy Cruz, Fredy Rodriguez Rivas, David Naira, and Perla Simons Morales. "Reacciones adversas por antibióticos y anti-inflamatorios no esteroideos en pacientes ambulatorios en Honduras." Journal of Pharmacy & Pharmacognosy Research 10, no. 2 (2022): 186–95. http://dx.doi.org/10.56499/jppres21.1152_10.2.186.

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Context: Adverse drug reactions represent one of the leading causes of hospitalization and death. Aims: To characterize adverse reactions due to antibiotics and nonsteroidal anti-inflammatory drugs in outpatients over 18 years of age. Methods: Cross-sectional descriptive observational study. It included outpatients aged 18 years or older who went to public health care centers and who presented a health problem, for which they were treated with antibiotics or nonsteroidal anti-inflammatory drugs (NSAIDs). The information was collected through an adaptation of the adverse drug reaction (ADR) rep

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11

Michael John Dochniak. "Allergic Reactions and Solid Tumors." GSC Biological and Pharmaceutical Sciences 31, no. 3 (2025): 168–70. https://doi.org/10.30574/gscbps.2025.31.3.0225.

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The effect allergies have on cancer is complex. Immune cells associated with the allergy cascade produce anti-inflammatory, pro-inflammatory, or pleiotropic cytokines. Allergies and solid tumors may have a paradoxical relationship based on the cytokine expression and concentration. Rapid release of pro-inflammatory cytokines into the tumor microenvironment (acute allergic reaction) may stimulate the immune system to disrupt a solid tumor. Alternatively, a slow release of pro-inflammatory cytokines (chronic allergic reaction) may suppress the immune system’s ability to disrupt the tumor. This r

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12

Khidirova, L. D., and N. N. Mayanskaya. "INFLAMMATORY REACTIONS IN EXPERIMENTAL MYOCARDIAL DAMAGE." Rational Pharmacotherapy in Cardiology 8, no. 5 (2012): 717–20. http://dx.doi.org/10.20996/1819-6446-2012-8-5-717-720.

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13

Makita, Chantal, Norbert N’Soundhat, Charles Ngabou, and Honoré Ntsiba. "Ocular Reactions during Chronic Inflammatory Rheumatisms." Ophthalmology Research: An International Journal 8, no. 2 (2018): 1–5. http://dx.doi.org/10.9734/or/2018/39234.

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14

Zeng, Wenjing, Meredith Osterman, and Peter J. Stern. "Inflammatory Reactions to Xenogenic Nerve Wraps." JBJS Case Connector 9, no. 3 (2019): e0302-e0302. http://dx.doi.org/10.2106/jbjs.cc.18.00302.

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15

Wood, N., H. S. Pall, A. C. Williams, and C. Dieppe. "Extrapyramidal reactions to anti-inflammatory drugs." Journal of Neurology, Neurosurgery & Psychiatry 51, no. 5 (1988): 731–32. http://dx.doi.org/10.1136/jnnp.51.5.731.

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16

Mather, C. M. P., L. B. Ready, and M. Newman. "Inflammatory Cutaneous Reactions to Epidural Catheters." Anesthesiology 78, no. 1 (1993): 200–203. http://dx.doi.org/10.1097/00000542-199301000-00029.

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17

Oxholm, P., and K. Winther. "Thrombocyte Involvement in Immune Inflammatory Reactions." Allergy 41, no. 1 (1986): 1–10. http://dx.doi.org/10.1111/j.1398-9995.1986.tb00268.x.

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18

Barker, S. M., and R. K. Winkelmann. "Inflammatory lymphadenoid reactions with dermatofibroma/histiocytoma." Journal of Cutaneous Pathology 13, no. 3 (1986): 222–26. http://dx.doi.org/10.1111/j.1600-0560.1986.tb01648.x.

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19

Tirelli, Nicola. "Smart Nano-Systems and Inflammatory Reactions." Advanced Materials Research 745 (August 2013): 167–72. http://dx.doi.org/10.4028/www.scientific.net/amr.745.167.

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Inflammatory pathologies are typically characterized by an oxidizing environment. Our research aims to develop smart (nano) materials that respond to oxidizing conditions in order to establish new anti-inflammatory therapies with less side effects. For example, if drugs can be released in an inflammation-responsive fashion, it is possible to localize their action and reduce their overall amount.In this communication, we specifically focus on how polysulfide-based nanomaterials (nanoparticles, micelles) react to biological oxidants, and specifically on whether their response can depend on the i

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20

Silwal, Prashanta, Kyu Lim, Jun-Young Heo, Jong IL Park, Uk Namgung, and Seung-Kiel Park. "Adenine attenuates lipopolysaccharide-induced inflammatory reactions." Korean Journal of Physiology & Pharmacology 22, no. 4 (2018): 379. http://dx.doi.org/10.4196/kjpp.2018.22.4.379.

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21

Leso, Veruscka, Luca Fontana, and Ivo Iavicoli. "Nanomaterial exposure and sterile inflammatory reactions." Toxicology and Applied Pharmacology 355 (September 2018): 80–92. http://dx.doi.org/10.1016/j.taap.2018.06.021.

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22

Bradley, J. R. "Endothelial Cell Activation in Inflammatory Reactions." Clinical Science 85, s29 (1993): 33P. http://dx.doi.org/10.1042/cs085033pa.

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23

Yashchenko, L. V., L. M. Gakh, and G. M. Pyataeva. "Relations between inflammatory and immune reactions." Bulletin of Experimental Biology and Medicine 103, no. 4 (1987): 530–32. http://dx.doi.org/10.1007/bf00842490.

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24

Ginovart-Panisello, Gerardo José, Ignasi Iriondo, Tesa Panisello Monjo, Silvia Riva, Jordi Casadó Cancer, and Rosa Ma Alsina-Pagès. "Acoustic Detection of Vaccine Reactions in Hens for Assessing Anti-Inflammatory Product Efficacy." Applied Sciences 14, no. 5 (2024): 2156. http://dx.doi.org/10.3390/app14052156.

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Acoustic studies on poultry show that chicken vocalizations can be a real-time indicator of the health conditions of the birds and can improve animal welfare and farm management. In this study, hens vaccinated against infectious laryngotracheitis (ILT) were acoustically recorded for 3 days before vaccine administration (pre-reaction period) and also from vaccination onwards, with the first 5 days being identified as the “reaction period” and the 5 following days as “post reaction”. The raw audio was pre-processed to isolate hen calls and the 13 Mel-frequency cepstral coefficients; then, the sp

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25

Weg, V. B., T. J. Williams, R. R. Lobb, and S. Nourshargh. "A monoclonal antibody recognizing very late activation antigen-4 inhibits eosinophil accumulation in vivo." Journal of Experimental Medicine 177, no. 2 (1993): 561–66. http://dx.doi.org/10.1084/jem.177.2.561.

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Using an in vivo test system, the role of the beta 1 integrin very late activation antigen-4 (VLA-4) in eosinophil accumulation in allergic and nonallergic inflammatory reactions was investigated. Eosinophil infiltration and edema formation were measured as the local accumulation of intravenously injected 111In-labeled eosinophils and 125I-human serum albumin. The inflammatory reactions investigated were a passive cutaneous anaphylaxis (PCA) reaction and responses elicited by intradermal soluble inflammatory mediators (platelet-activating factor, leukotriene B4, C5a des Arg), arachidonic acid,

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26

Boyer, Debra L., B. Ulysses K. Li, John N. Fyda, and Roger A. Friedman. "Sulfasalazine‐Induced Hepatotoxicity in Children with Inflammatory Bowel Disease." Journal of Pediatric Gastroenterology and Nutrition 8, no. 4 (1989): 528–32. http://dx.doi.org/10.1002/j.1536-4801.1989.tb09800.x.

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SummaryA case of severe sulfasalazine‐induced hepatotoxicity is reported in a 14‐year‐old girl with Crohn's disease. Fourteen days after beginning sulfasalazine, she developed a systemic reaction characterized by high fever, maculopapular rash, lymphadenopathy, abdominal pain, and malaise, with tender hepatomegaly and elevated liver functions, leukemoid reaction, with eosinophilia, and immune complexes. She responded promptly to high‐dose intravenous steroids with complete recovery. This case was compared to the three similar pediatrie cases in the literature. The latent period was 11–19 days

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27

Cunha, Suely Aparecida, Fuad Jacob Abi Rached Jr, Edson Alfredo, Jorge Esquiche León, and Danyel Elias da Cruz Perez. "Biocompatibility of sealers used in apical surgery: a histological study in rat subcutaneous tissue." Brazilian Dental Journal 22, no. 4 (2011): 299–305. http://dx.doi.org/10.1590/s0103-64402011000400007.

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The aim of this study was to evaluate the biocompatibility of sealers used in apical surgery in rat subcutaneous tissue. Sterile polyethylene tubes were filled with the following sealers: Sealapex, Sealapex with addition of zinc oxide, Sealer 26, Sealer 26 with thicker consistency (greater powder-to-resin ratio) and White MTA. The tubes were implanted in the dorsum of male rats and after 7, 21 and 42 days, the animals were killed, obtaining 5 specimens for each sealer in each evaluation period. The lateral surface of the tube was used as negative control. The inflammatory reaction to contact w

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28

Wong, Jason, Ashley Sandoval, Tiffany Jean, and Asal Gharib Naderi. "A case of delayed maculopapular eruption to ibuprofen and acute urticaria to acetaminophen." SAGE Open Medical Case Reports 9 (January 2021): 2050313X2110564. http://dx.doi.org/10.1177/2050313x211056409.

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We present the unique case of a 33-year-old male referred to our clinic in search of analgesic options who was found to have a delayed hypersensitivity reaction to ibuprofen manifesting as a maculopapular rash and acute urticaria to acetaminophen. Non-steroidal anti-inflammatory drugs are associated with predictable reactions as well as immunoglobulin E-mediated reactions or T-cell mediated reactions. This case highlights the importance of knowledge of the different types of reactions to non-steroidal anti-inflammatory agents as well as the risk of cross reactivity. Delayed reaction to a singl

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29

Toutouzas, Konstantinos, Antonios Karanasos, and Christodoulos Stefanadis. "Inflammatory Mechanisms of Adverse Reactions to BMS." Current Vascular Pharmacology 11, no. 4 (2013): 379–91. http://dx.doi.org/10.2174/1570161111311040002.

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30

Fan, Jianglin, and Teruo Watanabe. "Inflammatory Reactions in the Pathogenesis of Atherosclerosis." Journal of Atherosclerosis and Thrombosis 10, no. 2 (2003): 63–71. http://dx.doi.org/10.5551/jat.10.63.

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31

Schempp, Harald, Dieter Weiser, and Erich Elstner. "Biochemical Model Reactions Indicative of Inflammatory Processes." Arzneimittelforschung 50, no. 04 (2011): 362–72. http://dx.doi.org/10.1055/s-0031-1300215.

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32

Blanca-López, Natalia, Ester Barrionuevo, Inmaculada Andreu, and María G. Canto. "Hypersensitivity reactions to nonsteroidal anti-inflammatory drugs." Current Opinion in Allergy and Clinical Immunology 14, no. 4 (2014): 271–77. http://dx.doi.org/10.1097/aci.0000000000000077.

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33

YAMAKI, KOJI. "Mechanism of leukocytic infiltration in inflammatory reactions." Kagaku To Seibutsu 37, no. 8 (1999): 534–36. http://dx.doi.org/10.1271/kagakutoseibutsu1962.37.534.

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34

Robinson, Dwight R., Sumio Tateno, Balkrishna Patel, and Aizan Hirai. "Lipid Mediators of Inflammatory and Immune Reactions." Journal of Parenteral and Enteral Nutrition 12, no. 6_suppl (1988): 37S—42S. http://dx.doi.org/10.1177/014860718801200602.

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35

Pfuhlmann, Bruno, Christoph Hiemke, Stephan Unterecker, et al. "Toxic Clozapine Serum Levels During Inflammatory Reactions." Journal of Clinical Psychopharmacology 29, no. 4 (2009): 392–94. http://dx.doi.org/10.1097/jcp.0b013e3181acd20b.

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36

SHALIT, M., D. CAMPBELL, C. VONALLMEN, P. ATKINS, S. DOUGLAS, and B. ZWEIMAN. "Neutrophil activation in human inflammatory skin reactions." Journal of Allergy and Clinical Immunology 80, no. 1 (1987): 87–93. http://dx.doi.org/10.1016/s0091-6749(87)80196-3.

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37

Guina, Tina, Fiorella Biasi, Simone Calfapietra, Mario Nano, and Giuseppe Poli. "Inflammatory and redox reactions in colorectal carcinogenesis." Annals of the New York Academy of Sciences 1340, no. 1 (2015): 95–103. http://dx.doi.org/10.1111/nyas.12734.

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38

Stevenson, Donald D. "ADVERSE REACTIONS TO NONSTEROIDAL ANTI-INFLAMMATORY DRUGS." Immunology and Allergy Clinics of North America 18, no. 4 (1998): 773–98. http://dx.doi.org/10.1016/s0889-8561(05)70038-2.

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39

Darragh, Margot, Joshua W.-H. Chang, Roger J. Booth, and Nathan S. Consedine. "The placebo effect in inflammatory skin reactions." Journal of Psychosomatic Research 78, no. 5 (2015): 489–94. http://dx.doi.org/10.1016/j.jpsychores.2015.01.011.

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40

Bigby, Michael, and Robert Stern. "Cutaneous reactions to nonsteroidal anti-inflammatory drugs." Journal of the American Academy of Dermatology 12, no. 5 (1985): 866–76. http://dx.doi.org/10.1016/s0190-9622(85)70110-7.

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41

Chiba, Yukio, Ryusuke Muraoka, Akio Ihaya, et al. "Postoperative Inflammatory Reactions of Impregnated Dacron Grafts." Surgery Today 29, no. 11 (1999): 1225–28. http://dx.doi.org/10.1007/s005950050573.

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42

Chiba, Yukio, Ryusuke Muraoka, Akio Ihaya, et al. "Postoperative inflammatory reactions of impregnated Dacron grafts." Surgery Today 29, no. 11 (1999): 1225–28. http://dx.doi.org/10.1007/bf02482279.

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43

Torres, Maria Jose, Esther Barrionuevo, Marek Kowalski, and Miguel Blanca. "Hypersensitivity Reactions to Nonsteroidal Anti-Inflammatory Drugs." Immunology and Allergy Clinics of North America 34, no. 3 (2014): 507–24. http://dx.doi.org/10.1016/j.iac.2014.04.001.

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44

Ehrchen, Jan, Lars Steinmüller, Katarzyna Barczyk, et al. "Glucocorticoids induce differentiation of a specifically activated, anti-inflammatory subtype of human monocytes." Blood 109, no. 3 (2006): 1265–74. http://dx.doi.org/10.1182/blood-2006-02-001115.

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AbstractMonocytes and macrophages may either promote or down-regulate inflammatory reactions depending on their state of activation. The effects of glucocorticoids (GCs), the most widely used immunosuppressive drugs, on monocytes are currently not well defined. By analyzing the GC-induced expression pattern in human monocytes by microarray technology, we identified for the first time GC-dependent regulation of 133 genes, including anti-inflammatory molecules such as adenosine A3 receptor, CD1d, and IL-1 receptor II. The results were independently confirmed by real-time polymerase chain reactio

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45

Gupta, Neha, Amol Bansal, and Aarti Bansal. "Erythema Multiforme- Case Report with Review." Dental Journal of Advance Studies 02, no. 03 (2014): 166–68. http://dx.doi.org/10.1055/s-0038-1672005.

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AbstractErythema Multiforme is a rare, acute, inflammatory mucocutaneous condition caused by a hypersensitivity reaction with the appearance of cytotoxic T lymphocytes in the epithelium that induce apoptosis in keratinocytes, which leads to satellite cell necrosis. Reactions to drugs are quite common and are generally mild, hence not reported. However, occasionally life threatening reactions including Erythema multiforme major (Steven Johnson’s syndrome) and Toxic Epidermal Necrolysis may occur. A wide spectrum of drugs can sometimes give rise to Erythema Multiforme. We report a case of Erythe

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46

Wan, Lei, Hui-Ju Lin, and Yu-An Hsu. "Inflammatory responses in myopia progression (HUM7P.317)." Journal of Immunology 192, no. 1_Supplement (2014): 184.26. http://dx.doi.org/10.4049/jimmunol.192.supp.184.26.

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Abstract Myopia is an important and common eye disease as it can induce many other associated eye diseases and even lead to legal blindness. The prevention and treatment of myopia is an important public problem worldwide. We found that the expression levels of several inflammatory molecules are altered in the retina and sclera of FDM hamsters. To affirm the possible correlation between inflammatory reaction and myopia progression, cyclosporine A was used to treat FDM rats. We found that inhibiting inflammatory responses in the eye slowed myopia progression, whereas increasing inflammation by L

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47

Wang, Rui, Ran Ma, Ke Feng, Hongchen Lu, Wei Zhao, and Hongzhen Jin. "Total Synthesis and Anti-Inflammatory Evaluation of Osajin, Scandenone and Analogues." Pharmaceuticals 17, no. 1 (2024): 86. http://dx.doi.org/10.3390/ph17010086.

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In this study, the total synthesis of osajin, scandenone and their analogues have been accomplished. The key synthetic steps include aldol/intramolecular iodoetherification/elimination sequence reactions and a Suzuki coupling reaction to assemble the tricyclic core, chemoselective propargylation and Claisen rearrangement reactions to obtain natural compounds. In addition, we also designed and synthesized twenty-five natural product analogues. All synthetic compounds were screened for anti-inflammatory activity against tumor necrosis factor-α (TNF-α) and interleukin-6 (IL-6) in lipopolysacchari

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48

Grisham, Matthew B., David Jourd’Heuil, and David A. Wink. "I. Physiological chemistry of nitric oxide and its metabolites: implications in inflammation." American Journal of Physiology-Gastrointestinal and Liver Physiology 276, no. 2 (1999): G315—G321. http://dx.doi.org/10.1152/ajpgi.1999.276.2.g315.

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The role of nitric oxide (NO) in inflammation represents one of the most studied yet controversial subjects in physiology. A number of reports have demonstrated that NO possesses potent anti-inflammatory properties, whereas an equally impressive number of studies suggest that NO may promote inflammation-induced cell and tissue dysfunction. The reasons for these apparent paradoxical observations are not entirely clear; however, we propose that understanding the physiological chemistry of NO and its metabolites will provide a blueprint by which one may distinguish the regulatory/anti-inflammator

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49

Antonelli, Elisabetta, Gabrio Bassotti, Marta Tramontana, et al. "Dermatological Manifestations in Inflammatory Bowel Diseases." Journal of Clinical Medicine 10, no. 2 (2021): 364. http://dx.doi.org/10.3390/jcm10020364.

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Inflammatory bowel diseases (IBDs) may be associated with extra-intestinal manifestations. Among these, mucocutaneous manifestations are relatively frequent, often difficult to diagnose and treat, and may complicate the course of the underlying disease. In the present review, a summary of the most relevant literature on the dermatologic manifestations occurring in patients with inflammatory bowel diseases has been reviewed. The following dermatological manifestations associated with IBDs have been identified: (i) specific manifestations with the same histological features of the underlying IBD

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50

Makino, Yusuke, Kaoru Fujikawa, Miwako Matsuki-Fukushima, Satoshi Inoue, and Masanori Nakamura. "Role of Innate Inflammation in the Regulation of Tissue Remodeling during Tooth Eruption." Dentistry Journal 9, no. 1 (2021): 7. http://dx.doi.org/10.3390/dj9010007.

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Tooth eruption is characterized by a coordinated complex cascade of cellular and molecular events that promote tooth movement through the eruptive pathway. During tooth eruption, the stratum intermedium structurally changes to the papillary layer with tooth organ development. We previously reported intercellular adhesion molecule-1 (ICAM-1) expression on the papillary layer, which is the origin of the ICAM-1-positive junctional epithelium. ICAM-1 expression is induced by proinflammatory cytokines, including interleukin-1 and tumor necrosis factor. Inflammatory reactions induce tissue degradati

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