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Journal articles on the topic 'Influenza pandemic (H1N1) in 2009'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 February 2022

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1

OSHITANI, Hitoshi. "Influenza Pandemic (H1N1) 2009." Uirusu 59, no. 2 (2009): 139–44. http://dx.doi.org/10.2222/jsv.59.139.

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Osztovits, János, Csaba Balázs, and János Fehér. "H1N1 influenza – pandemic, 2009." Orvosi Hetilap 150, no. 50 (2009): 2265–73. http://dx.doi.org/10.1556/oh.2009.28766.

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2009 márciusában egy új, addig csak sertésekben kimutatott influenza A-vírus H1N1-szubtípusa okozott emberi megbetegedéseket Mexikóban, majd három hónap alatt a föld minden régiójában elterjedt. Bár a mortalitás aránya alapján az új H1N1-fertőzések nem súlyosabbak a szezonális influenzajárványoknál, a gyors és globális terjedés miatt az Egészségügyi Világszervezet (WHO) 2009. július 11-én világméretű járványnak (pandémiának) nyilvánította a H1N1-fertőzést. 2009. október elejéig közel 400 000 igazolt H1N1-vírus-fertőzést és 5000 halálesetet ismerünk a világ minden tájáról. A fertőzés terjedésén

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3

Woo, Teri Moser. "2009 H1N1 Influenza Pandemic." Journal of Pediatric Health Care 24, no. 4 (2010): 258–66. http://dx.doi.org/10.1016/j.pedhc.2010.05.001.

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4

Woo, Teri Moser. "2009 H1N1 Influenza Pandemic." Journal of Pediatric Health Care 24, no. 4 (2010): 267–69. http://dx.doi.org/10.1016/j.pedhc.2010.05.004.

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5

Patel, M., A. Dennis, C. Flutter, and Z. Khan. "Pandemic (H1N1) 2009 influenza." British Journal of Anaesthesia 104, no. 2 (2010): 128–42. http://dx.doi.org/10.1093/bja/aep375.

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6

Shieh, Wun-Ju, Dianna M. Blau, Amy M. Denison, et al. "2009 Pandemic Influenza A (H1N1)." American Journal of Pathology 177, no. 1 (2010): 166–75. http://dx.doi.org/10.2353/ajpath.2010.100115.

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7

Jones, Robert S., and Samantha Cunningham. "2009 H1N1 influenza: a pandemic." Osteopathic Family Physician 2, no. 3 (2010): 60–65. http://dx.doi.org/10.1016/j.osfp.2010.02.001.

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8

Lee, Bruce Y., and Ann E. Wiringa. "The 2009 H1N1 influenza pandemic." Human Vaccines 7, no. 1 (2011): 115–19. http://dx.doi.org/10.4161/hv.7.1.13740.

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9

Shulman, Stanford T. "Pandemic: 2009 Influenza a (H1N1)." Pediatric Annals 38, no. 12 (2009): 635–36. http://dx.doi.org/10.3928/00904481-20091117-09.

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10

WANG, X., X. W. CHENG, H. W. MA, et al. "Influenza surveillance in Shenzhen, the largest migratory metropolitan city of China, 2006–2009." Epidemiology and Infection 139, no. 10 (2010): 1551–59. http://dx.doi.org/10.1017/s0950268810002694.

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SUMMARYShenzhen is one of the largest migratory metropolitan cities in China. A standardized influenza surveillance system has been operating in Shenzhen for several years. The objectives of the present study were to describe the epidemiology of influenza in Shenzhen and to assess the impact of pandemic H1N1 on influenza activity. An average rate of 71 cases of influenza-like illness (ILI)/1000 consultations was reported, which was greater than the rate in the preceding 3 years. Laboratory surveillance showed that the annual proportion of specimens positive for influenza was 25·4% in 2009, rep

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11

Faridi, Omar S., Tushar M. Ranchod, Lawrence Y. Ho, and Alan J. Ruby. "Pandemic 2009 influenza A H1N1 retinopathy." Canadian Journal of Ophthalmology 45, no. 3 (2010): 286–87. http://dx.doi.org/10.3129/i10-030.

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Meeyai, Aronrag, Ben Cooper, Richard Coker, Wirichada Pan-ngum, Pasakorn Akarasewi, and Sopon Iamsirithaworn. "Pandemic influenza H1N1 2009 in Thailand." WHO South-East Asia Journal of Public Health 1, no. 1 (2012): 59. http://dx.doi.org/10.4103/2224-3151.206915.

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13

Ryan, Kenneth. "2009 H1N1 Pandemic Influenza: An Overview." Seminars in Cardiothoracic and Vascular Anesthesia 14, no. 3 (2010): 162–64. http://dx.doi.org/10.1177/1089253210377928.

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14

van der Sande, M. A. B., A. Jacobi, A. Meijer, J. Wallinga, W. van der Hoek, and M. van der Lubben. "The 2009 influenza A (H1N1) pandemic." Bundesgesundheitsblatt - Gesundheitsforschung - Gesundheitsschutz 56, no. 1 (2012): 67–75. http://dx.doi.org/10.1007/s00103-012-1582-4.

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15

Shiley, Kevin T., Gregory Nadolski, Timothy Mickus, Neil O. Fishman, and Ebbing Lautenbach. "Differences in the Epidemiological Characteristics and Clinical Outcomes of Pandemic (H1N1) 2009 Influenza, Compared with Seasonal Influenza." Infection Control & Hospital Epidemiology 31, no. 7 (2010): 676–82. http://dx.doi.org/10.1086/653204.

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Background.There are limited data comparing the clinical presentations, comorbidities, and outcomes of patients with infections due to seasonal influenza with patients with infections due to pandemic (H1N1) 2009 influenza.Objective.To compare the epidemiological characteristics and outcomes of pandemic (H1N1) 2009 influenza with those of seasonal influenza.Methods.A cross-sectional study was conducted among patients who received diagnoses during emergency department and inpatient encounters at 2 affiliated academic medical centers in Philadelphia, Pennsylvania. Cases of seasonal influenza duri

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16

Ambrozaitis, Arvydas, Daiva Radzišauskienė, Kęstutis Žagminas, Nerija Kuprevičienė, Stefan Gravenstein, and Ligita Jančorienė. "Influenza A(H1N1)pdm09 and postpandemic influenza in Lithuania." Open Medicine 11, no. 1 (2016): 341–53. http://dx.doi.org/10.1515/med-2016-0064.

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AbstractThe objective of this study is to describe the clinical and epidemiological characteristics of patients hospitalized in Lithuania who are infected with influenza A(H1N1)pdm09 and to compare pandemic A(H1N1) pdm09 infection with postpandemic.In total, 146 subjects hospitalized with influenza A(H1N1) pdm09 were identified from 2009–2011. There were 53 during the initial pandemic wave in the summer of 2009, 69 during the peak pandemic period, and 24 during the “postpandemic” period that we included in this study. There were 22 subjects who died after laboratory confirmation of influenza A

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17

Poon, Leo L. M., Polly W. Y. Mak, Olive T. W. Li, et al. "Rapid Detection of Reassortment of Pandemic H1N1/2009 Influenza Virus." Clinical Chemistry 56, no. 8 (2010): 1340–44. http://dx.doi.org/10.1373/clinchem.2010.149179.

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BACKGROUND Influenza viruses can generate novel reassortants in coinfected cells. The global circulation and occasional introductions of pandemic H1N1/2009 virus in humans and in pigs, respectively, may allow this virus to reassort with other influenza viruses. These possible reassortment events might alter virulence and/or transmissibility of the new reassortants. Investigations to detect such possible reassortants should be included as a part of pandemic influenza surveillance plans. METHODS We established a real-time reverse-transcription (RT)-PCR–based strategy for the detection of reassor

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18

Roselinda, Eka Pratiwi, Agustiningsih, and Vivi Setiawaty. "Lesson Learned from the Emergence of Influenza Pandemic H1N1 in 2009 in Indonesia: The Importance of Influenza-Like Illness (ILI) Surveillance." ISRN Infectious Diseases 2013 (December 29, 2013): 1–6. http://dx.doi.org/10.5402/2013/920806.

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Background. In 2009 there were outbreaks of influenza pandemic H1N1 in Indonesia that were caused by different virus from the previous circulated H1N1. Further, the influenza-like illness (ILI) surveillance plays an important role in the early detection of influenza outbreaks in outpatients. To understand the disease burden of ILI in the community at the time of H1N1 pandemic 2009, a sentinel-based survey was performed. Methods. The nasal and throat swabs were obtained from 20 primary health centers of ILI sentinel in Indonesia in 2009. Identification of virus influenza pandemic H1N1 was carri

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19

Blake, Nancy, Kathleen Stevenson, and Dawn England. "H1N1 Pandemic." AACN Advanced Critical Care 20, no. 4 (2009): 334–41. http://dx.doi.org/10.4037/15597768-2009-4006.

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In March 2009, a child in Mexico was infected with novel influenza A (H1N1), otherwise known as swine flu. Otherwise healthy children in that small town came down with it shortly after, as well as others from other countries who had visited Mexico or been visited by someone from Mexico, as was the case in the United States. The Centers for Disease Control and Prevention confirmed the first 2 cases in April 2009 and has been working together with local health departments to do syndromic surveillance. In June 2009, the World Health Organization raised the pandemic alert to level 6. Pandemic H1N1

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20

Kim, Jae Yeol. "The 2009 H1N1 Pandemic Influenza in Korea." Tuberculosis and Respiratory Diseases 79, no. 2 (2016): 70. http://dx.doi.org/10.4046/trd.2016.79.2.70.

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21

Kim, Woo Joo. "Pandemic Influenza (H1N1 2009): Experience and Lessons." Infection and Chemotherapy 42, no. 2 (2009): 61. http://dx.doi.org/10.3947/ic.2010.42.2.61.

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22

Khanna, Madhu, Neha Gupta, Ankit Gupta, and V. K. Vijayan. "Influenza A (H1N1) 2009: a pandemic alarm." Journal of Biosciences 34, no. 3 (2009): 481–89. http://dx.doi.org/10.1007/s12038-009-0053-z.

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23

The Lancet. "Hajj and 2009 pandemic influenza A H1N1." Lancet 374, no. 9703 (2009): 1724. http://dx.doi.org/10.1016/s0140-6736(09)61971-1.

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24

Markey, Peter, Jiunn-Yih Su, Andre Wattiaux, James Trauer, and Vicki Krause. "H1N1 2009 pandemic influenza in Indigenous Australians." Microbiology Australia 32, no. 1 (2011): 36. http://dx.doi.org/10.1071/ma11036.

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Given the known prevalence of chronic disease in the Australian Indigenous population, and the known risk factors for severe disease from influenza infection, it is not surprising that Indigenous Australians carried a higher burden of disease during the influenza pandemic of 2009. However, other determinants apart from comorbidities might also have affected influenza morbidity in Indigenous Australia. Factors such as overcrowding, sanitation infrastructure, remoteness, access to health care and availability of the specific hardware of the pandemic (such as personal protective equipment ? PPE?

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25

Joob, B., and V. Wiwanitkit. "Hospitalised children with pandemic (H1N1) 2009 influenza." Singapore Medical Journal 57, no. 10 (2016): 586. http://dx.doi.org/10.11622/smedj.2016168.

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26

Silva, Matthew A., and Linda M. Spooner. "2009 H1N1 Pandemic Influenza: Let's Get Involved!" Journal of Pharmacy Practice and Research 39, no. 4 (2009): 259–61. http://dx.doi.org/10.1002/j.2055-2335.2009.tb00468.x.

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27

Kebabci, Nesrin, Halis Akalin, Gulcin Boluk, et al. "Experience of Pandemic Influenza A (H1N1) 2009." Klimik Dergisi/Klimik Journal 25, no. 3 (2014): 117–21. http://dx.doi.org/10.5152/kd.2012.32.

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28

Earnshaw, Valerie A., and Diane M. Quinn. "Influenza stigma during the 2009 H1N1 pandemic." Journal of Applied Social Psychology 43 (May 3, 2013): E109—E114. http://dx.doi.org/10.1111/jasp.12049.

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29

Rockman, Steven, Karen Laurie, and Ian Barr. "Pandemic Influenza Vaccines: What did We Learn from the 2009 Pandemic and are We Better Prepared Now?" Vaccines 8, no. 2 (2020): 211. http://dx.doi.org/10.3390/vaccines8020211.

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In 2009, a novel A(H1N1) influenza virus emerged with rapid human-to-human spread and caused the first pandemic of the 21st century. Although this pandemic was considered mild compared to the previous pandemics of the 20th century, there was still extensive disease and death. This virus replaced the previous A(H1N1) and continues to circulate today as a seasonal virus. It is well established that vaccines are the most effective method to alleviate the mortality and morbidity associated with influenza virus infections, but the 2009 A(H1N1) influenza pandemic, like all significant infectious dis

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30

Starick, Elke, Elke Lange, Sasan Fereidouni, et al. "Reassorted pandemic (H1N1) 2009 influenza A virus discovered from pigs in Germany." Journal of General Virology 92, no. 5 (2011): 1184–88. http://dx.doi.org/10.1099/vir.0.028662-0.

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A natural reassortant influenza A virus consisting of seven genome segments from pandemic (H1N1) 2009 virus and a neuraminidase segment from a Eurasian porcine H1N1 influenza A virus was detected in a pig herd in Germany. The obvious reassortment compatibility between the pandemic (H1N1) 2009 and H1N1 viruses of porcine origin raises concern as to whether swine may become a reservoir for further reassortants of pandemic (H1N1) 2009 viruses with unknown implications for human health and swine production.

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31

Chan, Sandra S., Linda C. W. Lam, and Helen F. K. Chiu. "The emergence of the novel H1N1 virus: implications for global mental health." International Psychogeriatrics 21, no. 6 (2009): 987–89. http://dx.doi.org/10.1017/s1041610209990925.

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The emergence of the novel swine-origin influenza A (H1N1) virus in humans has aroused great concern among medical professionals about the possible evolution of a full-blown influenza pandemic, one on the scale of the “Spanish” influenza pandemic of 1918–19 (Belshe, 2009). It has been speculated that the return of a pandemic virus equivalent in pathogenicity to the virus of 1918 would likely kill more than 100 million people worldwide, including a large number of economically active young people (Taubenberger and Morens, 2006). Health administrations worldwide have stepped up reporting and sur

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Onischenko, G. G., A. P. Agafonov, O. K. Demina, et al. "Properties of Pandemic Influenza Virus Strains Isolated in the Territory of Russia." Problems of Particularly Dangerous Infections, no. 3(101) (June 20, 2009): 5–9. http://dx.doi.org/10.21055/0370-1069-2009-3(101)-5-9.

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The first cases of the disease caused by pandemic (H1N1) 2009 influenza virus in the territory of the Russian Federation were registered at the end of May, 2009. 3 strains of pandemic (H1N1)2009 influenza virus were isolated from patients. Properties of the strains isolated in the territory of Russia were studied in comparison with those of two reference strains A/California/04/2009(H1N1) and A/California/07/2009(H1N1)v. Analysis of primary gene sequence and examination of biological properties of the strains isolated in the territory of Russia suggested their close relationship with A/Califor

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33

Ostovar, Gholamabbas Amin, Nina Kohn, Karl O. A. Yu, Susan Nullet, and Lorry G. Rubin. "Nosocomial Influenza in a Pediatric Hospital: Comparison of Rates of Seasonal and Pandemic 2009 Influenza A/H1N1 Infection." Infection Control & Hospital Epidemiology 33, no. 03 (2012): 292–94. http://dx.doi.org/10.1017/s0195941700030861.

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The rates of nosocomial seasonal (January 2008 to March 2009) and 2009 A/H1N1 (April 2009 to December 2010) influenza infections in a children's hospital were compared. Droplet precautions were used. The rates were similar during both periods, suggesting that use of droplet precautions did not result in a higher rate of influenza A/H1N1 infection.Infect Control Hosp Epidemiol2012;33(3):292-294

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Ostovar, Gholamabbas Amin, Nina Kohn, Karl O. A. Yu, Susan Nullet, and Lorry G. Rubin. "Nosocomial Influenza in a Pediatric Hospital: Comparison of Rates of Seasonal and Pandemic 2009 Influenza A/H1N1 Infection." Infection Control & Hospital Epidemiology 33, no. 3 (2012): 292–94. http://dx.doi.org/10.1086/664046.

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The rates of nosocomial seasonal (January 2008 to March 2009) and 2009 A/H1N1 (April 2009 to December 2010) influenza infections in a children's hospital were compared. Droplet precautions were used. The rates were similar during both periods, suggesting that use of droplet precautions did not result in a higher rate of influenza A/H1N1 infection.Infect Control Hosp Epidemiol 2012;33(3):292-294

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35

Otte, A., M. Sauter, M. A. Daxer, A. C. McHardy, K. Klingel, and G. Gabriel. "Adaptive Mutations That Occurred during Circulation in Humans of H1N1 Influenza Virus in the 2009 Pandemic Enhance Virulence in Mice." Journal of Virology 89, no. 14 (2015): 7329–37. http://dx.doi.org/10.1128/jvi.00665-15.

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ABSTRACTDuring the 2009 H1N1 influenza pandemic, infection attack rates were particularly high among young individuals who suffered from pneumonia with occasional death. Moreover, previously reported determinants of mammalian adaptation and pathogenicity were not present in 2009 pandemic H1N1 influenza A viruses. Thus, it was proposed that unknown viral factors might have contributed to disease severity in humans. In this study, we performed a comparative analysis of two clinical 2009 pandemic H1N1 strains that belong to the very early and later phases of the pandemic. We identified mutations

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36

Chudasama, Rajesh K., Umed V. Patel, and Pramod B. Verma. "Hospitalizations associated with 2009 influenza A (H1N1) and seasonal influenza in Saurashtra region, India." Journal of Infection in Developing Countries 4, no. 12 (2010): 834–41. http://dx.doi.org/10.3855/jidc.1049.

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Introduction: This study investigated the clinico-epidemiological characteristics of patients who were hospitalized with 2009 pandemic H1N1 influenza virus infection and seasonal influenza in the Saurashtra region of India. Methodology: From September 2009 to February 2010, a total of 773 patients with influenza virus attending different hospitals in Rajkot city were studied. Real-time reverse-transcriptase-polymerase-chain-reaction (RT-PCR) testing was used to confirm infection; the clinico-epidemiological features of the disease were closely monitored. Results: Of the 733 patients, 35.4% (27

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37

Fanella, Sergio T., Michelle A. Pinto, Natalie A. Bridger, et al. "Pandemic (H1N1) 2009 Influenza in Hospitalized Children in Manitoba: Nosocomial Transmission and Lessons Learned from the First Wave." Infection Control & Hospital Epidemiology 32, no. 5 (2011): 435–43. http://dx.doi.org/10.1086/659401.

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Objective.To review the experiences at Winnipeg Children's Hospital (WCH) during the 2009 influenza season, with an emphasis on nosocomial transmission and infection prevention and control responses.Design.A case series of patients admitted to WCH who had laboratory-confirmed cases of influenza between January 1 and July 31, 2009, with a comparison of patients with seasonal influenza and those with pandemic (H1N1) 2009 influenza; a review of the impact of infection prevention and control modifications on nosocomial transmission.Patients and Setting.A total of 104 inpatients with influenza, 81

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38

Saxena, Shailendra K., Niraj Mishra, Rakhi Saxena, et al. "Structural and antigenic variance between novel influenza A/H1N1/2009 and influenza A/H1N1/2008 viruses." Journal of Infection in Developing Countries 4, no. 01 (2009): 001–6. http://dx.doi.org/10.3855/jidc.546.

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Background: The emergence of influenza A/H1N1/2009 is alarming. The severity of previous epidemics suggests that the susceptibility of the human population to H1N1 is directly proportional to the degree of changes in hemagglutinin/HA and neuraminidase/NA; therefore, H1N1/2009 and H1N1/2008 were analyzed for their sequence as well as structural divergence. Methodology: The structural and sequence divergence of H1N1/2009 and H1N1/2008 strains were analyzed by aligning HA and NA amino acid sequences by using ClustalW and ESyPred3D software. To determine the variations in sites of viral attachment

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39

Dierig, Alex, Gulam Khandaker, and Robert Booy. "Clinical impact of influenza – lessons learnt from the pandemic influenza A (H1N1) 2009." Microbiology Australia 32, no. 1 (2011): 29. http://dx.doi.org/10.1071/ma11029.

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Influenza is generally an acute, self-limiting, febrile illness without further complications in the majority of people. However, it can be associated with severe morbidity and mortality and the burden of the disease on society is likely to be underestimated. In 2009 an outbreak of H1N1 influenza A virus infection was detected in Mexico with further cases soon observed worldwide. Subsequently, in June 2009, the first influenza pandemic of the 21st century due to influenza A (H1N1) was declared by the World Health Organization (WHO). There were many uncertainties regarding the virulence, clinic

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40

Song, Joon Young, Hee Jin Cheong, Yu Bin Seo, et al. "Long-Term Immunogenicity of the Pandemic Influenza A/H1N1 2009 Vaccine among Health Care Workers: Influence of Prior Seasonal Influenza Vaccination." Clinical and Vaccine Immunology 20, no. 4 (2013): 513–16. http://dx.doi.org/10.1128/cvi.00725-12.

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ABSTRACTHealth care workers (HCWs) are at great risk of influenza infection and transmission. Vaccination for seasonal influenza is routinely recommended, but this strategy should be reconsidered in a pandemic situation. Between October 2009 and September 2010, a multicenter study was conducted to assess the long-term immunogenicity of the A/H1N1 2009 monovalent influenza vaccine among HCWs compared to non-health care workers (NHCWs). The influence of prior seasonal influenza vaccination was also assessed with respect to the immunogenicity of pandemic H1N1 influenza vaccine. Serum hemagglutini

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41

Belser, Jessica A., Debra A. Wadford, Claudia Pappas, et al. "Pathogenesis of Pandemic Influenza A (H1N1) and Triple-Reassortant Swine Influenza A (H1) Viruses in Mice." Journal of Virology 84, no. 9 (2010): 4194–203. http://dx.doi.org/10.1128/jvi.02742-09.

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ABSTRACT The pandemic H1N1 virus of 2009 (2009 H1N1) continues to cause illness worldwide, primarily in younger age groups. To better understand the pathogenesis of these viruses in mammals, we used a mouse model to evaluate the relative virulence of selected 2009 H1N1 viruses and compared them to a representative human triple-reassortant swine influenza virus that has circulated in pigs in the United States for over a decade preceding the current pandemic. Additional comparisons were made with the reconstructed 1918 virus, a 1976 H1N1 swine influenza virus, and a highly pathogenic H5N1 virus.

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42

Duque, Vítor, João Vaz, Vanda Mota, Célia Morais, Saraiva Da Cunha, and António Meliço-Silvestre. "Clinical manifestations of pandemic (H1N1) 2009 in the ambulatory setting." Journal of Infection in Developing Countries 5, no. 09 (2011): 658–63. http://dx.doi.org/10.3855/jidc.2024.

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Introduction: In June 2009, the World Health Organization declared an influenza pandemic associated with the pandemic (H1N1) 2009 strain. It was summer in the northern hemisphere, and therefore travelling and vacation time, which also provided an increased opportunity for the dissemination of respiratory diseases. Methodology: We reviewed the paper case report forms from all the patients with influenza-like illnesses with nasopharyngeal samples submitted for laboratory diagnosis of pandemic (H1N1) 2009 infection during the first wave of pandemic influenza that occurred between June and August

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43

Peacey, Matthew, Richard J. Hall, Stephanie Sonnberg, et al. "Pandemic (H1N1) 2009 and Seasonal Influenza A (H1N1) Co-infection, New Zealand, 2009." Emerging Infectious Diseases 16, no. 10 (2010): 1618–20. http://dx.doi.org/10.3201/eid1610.100116.

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44

Shulman, L. P. "2009 H1N1 vaccination by pregnant women during the 2009-10 H1N1 influenza pandemic." Yearbook of Obstetrics, Gynecology and Women's Health 2012 (January 2012): 77–78. http://dx.doi.org/10.1016/j.yobg.2012.05.023.

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45

Dlugacz, Yosef, Adiel Fleischer, Maria Torroella Carney, et al. "2009 H1N1 Vaccination by Pregnant Women During the 2009–10 H1N1 Influenza Pandemic." Obstetrical & Gynecological Survey 67, no. 8 (2012): 466–67. http://dx.doi.org/10.1097/01.ogx.0000419561.63610.2e.

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46

Dlugacz, Yosef, Adiel Fleischer, Maria Torroella Carney, et al. "2009 H1N1 vaccination by pregnant women during the 2009-10 H1N1 influenza pandemic." American Journal of Obstetrics and Gynecology 206, no. 4 (2012): 339.e1–339.e8. http://dx.doi.org/10.1016/j.ajog.2011.12.027.

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47

Shikov, A. N., E. I. Sergeeva, O. K. Demina, et al. "Development of DNA-Biochip for Identification of Influenza A Virus Subtypes." Problems of Particularly Dangerous Infections, no. 2(112) (April 20, 2012): 89–93. http://dx.doi.org/10.21055/0370-1069-2012-2(112)-89-93.

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Developed was the DNA-biochip to identify subtypes of influenza A virus, pathogenic for humans. Microchip was capable of detecting H1, H3, H5-subtypes of hemagglutinin (including H1-subtype of pandemic A/H1N1(2009) influenza virus ) and neuraminidase subtypes N1,N2 of influenza virus. This microchip was successfully tested on the strains of A/H5N1 highly pathogenic avian influenza virus, A/H1N1(2009) pandemic influenza virus, A/H1N1 and A/H3N2 seasonal influenza viruses.

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Prowse, Stephen J., and John S. MacKenzie. "2009 human H1N1 influenza (swine flu)." Microbiology Australia 30, no. 4 (2009): 127. http://dx.doi.org/10.1071/ma09127.

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The 2009 H1N1 influenza, initially known as swine flu, originated in North America in early 2009. This new strain of influenza A virus (H1N1) came to the attention of the international public health community when several foci of influenza-like illness were identified in Mexico, which had more than 850 cases of pneumonia, of whom 59 had died. Mild cases of influenza-like illness were also reported from Texas and California. Virus isolates were obtained from the cases in California and from samples of cases sent from Mexico to the Canadian National Public Health Laboratory in Winnipeg. Molecula

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Tsvetkov, V. V., E. G. Deeva, D. M. Danilenko, T. V. Sologub, and E. P. Tikhonova. "Molecular genetic factors of pathogenicity of influenza A virus (H1N1) pdm09." Epidemiology and Infectious Diseases 19, no. 4 (2014): 4–11. http://dx.doi.org/10.17816/eid40804.

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Unlike influenza epidemics which affect the population almost yearly, pandemics occur much less frequently, but have more severe medical and social consequences. The investigation of the nature of the course of all modern epidemics and pandemics are acquiring the particular rationale. Pandemic influenza A (H1N1) 2009 was caused by the virus of the mixed (triple) origin. In Russia, the first three cases of disease have been identified in Moscow from 21 to 10 June 2009. In the Far East - 2-2,5 months later compared to the European part of Russia. However, the epidemic of influenza in Russia caus

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Choi, Yoon Seok, Yun Hee Baek, Wonseok Kang, et al. "Reduced Antibody Responses to the Pandemic (H1N1) 2009 Vaccine after Recent Seasonal Influenza Vaccination." Clinical and Vaccine Immunology 18, no. 9 (2011): 1519–23. http://dx.doi.org/10.1128/cvi.05053-11.

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ABSTRACTThe vaccination program against the 2009 pandemic H1N1 influenza virus (2009 H1N1) provided a unique opportunity to determine if immune responses to the 2009 H1N1 vaccine were affected by a recent, prior vaccination against seasonal influenza virus. In the present study, we studied the immune responses to the 2009 H1N1 vaccine in subjects who either received the seasonal influenza virus vaccination within the prior 3 months or did not. Following 2009 H1N1 vaccination, subjects previously given a seasonal influenza virus vaccination exhibited significantly lower antibody responses, as d

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