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Dissertations / Theses on the topic 'Influenza virus'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Höfer, Chris Tina. "Influenza virus assembly." Doctoral thesis, Humboldt-Universität zu Berlin, Lebenswissenschaftliche Fakultät, 2015. http://dx.doi.org/10.18452/17251.

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Influenza A Viren besitzen ein segmentiertes, einzelsträngiges RNA-Genom, welches in Form viraler Ribonukleoprotein (vRNP)-Komplexe verpackt ist. Während das virale Genom im Zellkern repliziert wird, finden Assemblierung und Knospung reifer Viruspartikel an der apikalen Plasmamembran statt. Für die Virusbildung müssen die einzelnen viralen Komponenten hierher gebracht werden. Während intrinsische apikale Signale der viralen Transmembranproteine bekannt sind, sind der zielgerichtete Transport und der Einbau des viralen Genoms in neuentstehende Virionen noch wenig verstanden. In dieser Arbeit wu

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2

Mittelholzer, Camilla Maria. "Influenza virus - protection and adaptation /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-656-5/.

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3

Green, P. C. "Serological and immunocytochemical studies on influenza virus and influenza virus infected cells." Thesis, University of Manchester, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.356114.

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4

Wallensten, Anders. "Influenza A virus in wild birds." Doctoral thesis, Linköping : Linköping University, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-7643.

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5

Morgan, David John. "Defective interfering influenza virus reverses the immunopathological effects of standard influenza virus in mice." Thesis, University of Bristol, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.332491.

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6

Seekings, Amanda Hanna. "Emergence of H7 highly pathogenic avian influenza virus from low pathogenicity avian influenza virus." Thesis, Imperial College London, 2017. http://hdl.handle.net/10044/1/52910.

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Outbreaks of highly pathogenic avian influenza virus (HPAIV) may result in the infection of millions of poultry, causing devastating disease and up to 100% mortality. Avian influenza outbreaks and laboratory experiments have shown that HPAIV can emerge from low pathogenicity avian influenza virus (LPAIV) precursors. The multi-basic cleavage site (MBCS) in the haemagglutinin protein is described as the main pathogenic determinant of HPAIV infection in poultry. Identifying a precursor LPAIV is important for understanding the molecular changes involved in the emergence of HPAIV. In 2008, H7N7 HPA

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7

Jia, Nan. "Glycobiology studies of influenza virus." Thesis, Imperial College London, 2015. http://hdl.handle.net/10044/1/50787.

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Glycans represent a class of macromolecules that exhibit vital biological roles in living organisms. They are not only essential for maintaining the normal functionalities of a cell, but are also involved in many pathogenic processes. The influenza A virus binds to glycan receptors that are expressed on the surface of respiratory epithelial cells of human airway and thereby initiates infection. Deciphering the structural features of glycans and comprehending their functional implications are thus crucial to expand our understandings of the disease. To validate the alternative models that are u

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8

Read, Eliot Keith Curtis. "Investigating influenza A virus RNA trafficking." Thesis, University of Cambridge, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609127.

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9

Tan, E.-Pien. "Screening for influenza virus resistance genes." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608229.

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10

Kudryavtseva, Katerine. "Genome packaging in influenza A virus." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648592.

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11

Lau, Lee-hang Lincoln, and 劉力恆. "Influenza virus shedding and transmission in households." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hdl.handle.net/10722/196093.

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Background The dynamics of influenza virus transmission are not yet fully understood, hindering the appropriate development and implementation of control and mitigation strategies. One major uncertainty relates to the profile of infectiousness over time in humans infected by influenza virus, and how variation in infectiousness might contribute to the risk of transmission in households. Methods In 2008 and 2009, two large community-based studies were conducted to study the household transmission of influenza viruses in Hong Kong. I analyzed data on viral shedding and disease in participants,

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12

Lau, Siu-ying, and 劉韶瑩. "Characterizations of antigenic and receptor binding properties of avian H5N1 and 2009 pandemic H1N1 viruses." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47165078.

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Avian H5N1 viruses have perpetuated in poultry and caused sporadic human transmission since 1997. Vaccine candidates for the potential pandemic caused by H5N1 viruses have been continuously updated by World Health Organization. Multiple genetic lineages of H5N1 viruses which co-circulate and rapidly evolve in different regions, together with periodic population replacement of newly emerged genetic and antigenic variants in the field, pose great challenge for H5N1 vaccine candidate selection. The complexity of avian H5N1 viruses evolution raises an important issue for studying antigenic proper

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13

Cheng, Ka-yeung, and 鄭家揚. "Diagnosis and surveillance of human influenza virus infection." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B48079819.

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Background: Early and accurate diagnosis of influenza helps start correct treatment and prevention strategies at individual level. Ongoing systematic collection, analysis and dissemination of the surveillance data from aggregated diagnostic results and other early indicators help gather the foremost disease information for all subsequent control and mitigation strategies in the community. Disease information from surveillance results then feed back to medical practitioners for improving diagnosis. By improving this loop of disease information transfer in terms of accuracy and timeliness

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14

Poon, Leo L. M. "The polyadenylation of influenza virus mRNA." Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312548.

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15

Bishop, K. J. "Study of influenza A virus ribonucleoproteins." Thesis, University of Cambridge, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596669.

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Two expression systems were utilised in an attempt to produce suitable quantities of the influenza polymerase proteins for protein purification. Using the methylotrophic yeast <I>Pichia pastoris, </I>both PB1 and PB2 were expressed at around 0.1-0.4 mg/l. However, it did not prove possible to purify the polypeptides which appeared to be aggregated. The P proteins and NP were also expressed in mammalian cells using recombinant Semliki Forest viruses. However, soluble protein did not accumulate at levels sufficient for large scale purification studies. A transient "reverse genetics" assay whereb

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16

Duhaut, Susan. "The assembly of influenza virus ribonucleoproteins." Thesis, University of Reading, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.306344.

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17

Lam, Tsan-yuk Tommy. "Molecular evolution and epidemiology of influenza A virus." Click to view the E-thesis via HKUTO View the Table of Contents & Abstract, 2010. http://sunzi.lib.hku.hk/hkuto/record/B44137084.

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18

Wen, Xi, and 溫茜. "Study of nuclear factor 90 against influenza A virus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/208613.

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Influenza A virus is one of the most common human pathogens which caused considerable disease burdens through annual epidemics and occasional pandemics. The consequences vary from mild to severe or even fatal. What are the host and viral elements which determine the consequence of infection? In the past 15 years, several avian influenza A viruses including H5N1, H9N2, H7N7 and H7N9 subtypes were found to cross host barrier and infect humans. Question about how avian influenza A viruses gained the ability to replicate in human cells remains unanswered. Studies on host factors associated with vi

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19

Guan, Jing, and 管静. "The role of virus-specific human T cells in influenza A virus infection." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hdl.handle.net/10722/208423.

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Influenza A virus infection is a major cause of human morbidity and mortality. T cell immunity is believed to play critical roles for host defenses against influenza A infection. Once intracellular influenza A infection is established, viral clearance is mainly dependent on virus-specific CD8+ T cells. CD4+ T cells are important for adaptive immunity to natural influenza A infection or vaccination by providing help to B cells for antibody production and also providing help to CD8+ T cells for the generation of cytotoxicity. In addition, virusspecific CD4+ and CD8+ T cells are rich source

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20

Ling, Man-to. "The role of mannose binding lectin in influenza virus infection." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B43085295.

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21

Lam, Tsan-yuk Tommy, and 林讚育. "Molecular evolution and epidemiology of influenza A virus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44137084.

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22

Sun, Jian. "Computer-aided drug design for influenza A virus." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B44205156.

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23

Lu, Lu. "Transmission dynamics of Avian Influenza A virus." Thesis, University of Edinburgh, 2015. http://hdl.handle.net/1842/10481.

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Influenza A virus (AIV) has an extremely high rate of mutation. Frequent exchanges of gene segments between different AIV (reassortment) have been responsible for major pandemics in recent human history. The presence of a wild bird reservoir maintains the threat of incursion of AIV into domestic birds, humans and other animals. In this thesis, I addressed unanswered questions of how diverse AIV subtypes (classified according to antigenicity of the two surface proteins, haemagglutinin and neuraminidase) evolve and interact among different bird populations in different parts of the world, using

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24

Ma, Siu-kit. "Genesis and prevalence of H1N2 swine influenza virus in pigs from southern China /." View the Table of Contents & Abstract, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31495072.

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25

Ma, Siu-kit, and 馬少傑. "Genesis and prevalence of H1N2 swine influenza virus in pigs from southern China." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B4501033X.

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26

Leung, Yin-hung Connie, and 梁彥虹. "Ecology, epidemiology and immunology of avian influenza virus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B46329626.

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27

Jagger, Brett William. "The influenza A polymerase in viral pathogenesis." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610897.

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28

Lee, Hung-chiu. "Synthetic RNA interference against influenza A virus." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B35537814.

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29

Lee, Hung-chiu, and 李洪釗. "Synthetic RNA interference against influenza A virus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B35537814.

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30

Wasson, Peter Stewart. "Development of novel virus vectors for influenza vaccination." Thesis, University of Edinburgh, 2012. http://hdl.handle.net/1842/6492.

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The influenza virus, a member of the Orthomyxoviridae family, causes regular, large-scale morbidity and mortality in birds and humans and significant human suffering and economic loss. The primary aim of this study was to develop a novel influenza vaccine. Vaccines are an essential tool for the control of influenza because they increase resistance to infection, prevent illness and death and help to limit virus transmission to other birds and mammals, including humans. By reducing the environmental contamination of influenza virus in global poultry stocks, the risk of a new pandemic virus being

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31

Föglein, Ágnes. "Cell biology of the influenza A virus polymerase." Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609935.

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32

Pascolutti, Mauro. "The Development of Carbohydrate-Based Probes of Influenza Virus Sialidase." Thesis, Griffith University, 2013. http://hdl.handle.net/10072/366410.

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Influenza is a significant human disease, as evidenced by seasonal epidemics that exact a high toll in morbidity and mortality, and worldwide pandemics, for example the 2009 'swine-origin' influenza A pandemic. Two influenza virus-specific drugs, Relenza® and Tamiflu®, target the viral enzyme sialidase, and are effective against all wild type influenza virus strains. Resistance development to the more widely used drug, Tamiflu®, however, and new insights into the sialidase structure, have fuelled a drive to develop new sialidase inhibitors. The research described in this PhD thesis is focused

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33

Pavan, Carmen M. "Influenza B virus : segment 7 gene expression." Thesis, McGill University, 1989. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=55673.

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34

Smith, D. B. "The production of influenza virus spliced mRNAs." Thesis, University of Cambridge, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.355039.

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35

Jung, Tanis E. "Mutational analysis of the influenza virus polymerase." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.426398.

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36

Harvey, Ruth. "Studies of influenza A virus H5 haemagglutinin." Thesis, University of Reading, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.270832.

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37

Tibbles, K. W. "Studies on the influenza virus RNA polymerase." Thesis, University College London (University of London), 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.380686.

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38

Blok, Vivian Carol. "Studies of the influenza virus RNA polymerase." Thesis, University of Cambridge, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.292959.

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39

Doty, Daniel S. M. Massachusetts Institute of Technology. "CD8⁺ T Cell Response to Influenza Virus." Thesis, Massachusetts Institute of Technology, 2005. http://hdl.handle.net/1721.1/86280.

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Thesis: S.M., Massachusetts Institute of Technology, Department of Biology, 2005.<br>Cataloged from PDF version of thesis.<br>Includes bibliographical references (pages 10-12).<br>The flu is an extremely prevalent and potentially devastating disease, especially dangerous to the very young, the elderly, and to people with compromised immune systems. Influenza has a characteristic course of infection, and is often effectively dispatched by the immune system. The cell-mediated lysis of infected cells is a particularly important step in clearing the infection. Antigen specific CD8+ T lymphocytes a

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40

Pappworth, Isabel Yseult. "Studies in influenza A virus induced apoptosis." Thesis, University of Birmingham, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.434078.

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Influenza viruses induce apoptosis in tissue culture cells in vitro. In a study to examine the role of individual genes in apoptosis a twelve plasmid based reverse genetic system was used to produce a series of viruses, these being: rA/Victoria and rA/WSN, recombinant forms of the parent viruses A/Victoria/75 (H3N2) and A/WSN/33 (H1N1), rA/Vic/WSNNP and rA/Vic/WSNM, recombinant A/Victoria based viruses with the A/WSN nucleoprotein (NP) and matrix (M) gene respectively. These viruses were examined for their ability to induce apoptosis (determined morphologically), cell lysis (determined by lact

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41

Sieben, Christian. "Host cell invasion by influenza A virus." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2013. http://dx.doi.org/10.18452/16743.

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Influenzaviren müssen in die Wirtszelle aufgenommen werden, um dort ihr Genom freizusetzen und ihre Replikation mit Hilfe des Reproduktionsapparats der Zelle einzuleiten. Der komplexe Replikationszyklus der Influenza A Viren ist noch nicht vollständig verstanden. Er beginnt mit der Bindung des viralen Hämagglutinins (HA) an Sialinsäure (SA) auf der Zelloberfläche der Wirtszelle. In dieser Arbeit wurde die Virusbindung an Zellen mit unterschiedlicher Rezeptorkomposition verglichen. Dabei konnte gezeigt werden, dass für die zelluläre Spezifität die Präsentation des Rezeptors innerhalb der Plasma

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42

Ling, Man-to, and 凌文韜. "The role of mannose binding lectin in influenza virus infection." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43085295.

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43

Zhang, Naru, and 张娜茹. "Study on influenza virus-like particles and ssDNA aptamers." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/200167.

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Since there is an urgent need for development of vaccines and antiviral agents to combat influenza pandemics, this study aimed to develop influenza virus-like particles (VLPs) and aptamers targeting the virus particles as vaccine and antiviral agent candidates. Influenza VLPs containing three structural proteins of hemagglutinin (HA), neuraminidase (NA) and matrix 1 (M1) derived from influenza A/Hong Kong/01/2009 (H1N1) virus (HK/01) were constructed using a Bac-to-Bac baculovirus expression system. The expressed VLPs were purified by sucrose density gradient ultracentrifugation and characte

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44

Leung, Sze-Yui Horasis, and 梁思睿. "Fibronectin: role in viral cell association, fusion and entry of influenza A virus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B48329708.

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The influenza A viral hemagglutinin (HA) protein binds to sialic acid (SA) groups of cellular surface glycoproteins to achieve viral attachment and entry. The SA binding specificity of HA is one of the major determinants for controlling viral tropism and host specificity. Fibronectin (FN) is a ubiquitinious glycoprotein secreted on cell surface, either circulating in plasma, or as one of the best characterized components of the extra cellular matrix. With its binding properties towards different types of molecules and pathogens, it has been utilized by different bacterial and viral pathoge

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45

Campbell, Gillian Mhairi. "Influenza virus infection in a compromised immune system." Thesis, University of Edinburgh, 2012. http://hdl.handle.net/1842/6521.

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Severe influenza virus infection, including human infection with highly pathogenic H5N1 viruses is characterised by massive pulmonary inflammation, immunopathology and excessive cytokine production, a process in which macrophages may play a vital role. The aim of this project was to investigate the hypothesis that inhibition of inflammatory responses from infected macrophages, using either alternatively activated bone marrow derived macrophages (BMDMf), or IFNg receptor deficient (IFNgR-/-) mice may ameliorate the devastating immunopathology and inflammation routinely observed in highly pathog

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46

Ng, Chi-ko, and 伍智高. "Antigenicity and oseltamivir resistance of influenza A virus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hub.hku.hk/bib/B50434536.

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Although several risk factors for severe influenza infection have been identified in previous studies, many patients having multiple risk factors only developed mild symptoms while many healthy young patients developed severe complications when infected with A(H1N1)pdm09. Thus, there are still undiscovered factors that affect the progression and severity of influenza. The early innate immune response may be critical in determining the disease progression. Non-neutralizing antibodies existed in the early stage of infection may contribute to the outcome of the disease. In this study, the ass

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47

Mahallawi, Waleed. "Natural immunity to influenza virus in humans following 2009 pandemic H1N1 influenza." Thesis, University of Liverpool, 2013. http://livrepository.liverpool.ac.uk/13137/.

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Influenza is a highly contagious and acute respiratory infection caused by influenza virus in the mucosa of the respiratory tract. Both seasonal and pandemic influenza continue to cause substantial morbidity and mortality in humans. The 2009 pandemic H1N1 (pH1N1) influenza and the potential of a highly pathogenic avian H5N1 (aH5N1) pandemic highlighted the need for effective preventative strategies. Understanding the development of natural immunity following the pH1N1 pandemic may provide important information on host protective immunity in humans, which could inform future more effective vacc

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48

Killian, Mary Lea. "Identification and characterization of H2N3 avian influenza virus from backyard poultry and comparison to novel H2N3 swine influenza virus." [Ames, Iowa : Iowa State University], 2009.

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49

Ling, Man-to, and 凌文韜. "The role of mannose binding lectin in pandemic H1N1 influenza virus infection." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B50567214.

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50

Perera, Kumarapatti Vidanalage Harsha Kumara Kithsiri. "Ecology and evolution of swine influenza virus in Sri Lanka." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/196456.

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Influenza A virus infections in pigs is a disease of concern to the swine industry and to the ecology and epidemiology of influenza viruses in humans. Pigs have been proposed as a “mixing vessel” for generation of pandemics via reassortment between avian and mammalian viruses. The H1N1pdm 2009 virus probably emerged from swine into humans though reassortment between the recent North American triple reassortant H1N2 swine viruses and Eurasian avian-like swine viruses. Swine influenza viruses of H1N1, H1N2 and H3N2 subtypes have been regularly detected in pigs in most parts of the world. Nevert

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