Relevant bibliographies by topics / Inhibitor chemokinu

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Inhibitor chemokinu'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Inhibitor chemokinu"

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Calkins, Casey M., Denis D. Bensard, Julie K. Heimbach, et al. "l-Arginine attenuates lipopolysaccharide-induced lung chemokine production." American Journal of Physiology-Lung Cellular and Molecular Physiology 280, no. 3 (2001): L400—L408. http://dx.doi.org/10.1152/ajplung.2001.280.3.l400.

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Chemokines stimulate the influx of leukocytes into tissues. Their production is regulated by nuclear factor-κB (NF-κB), an inducible transcription factor under the control of inhibitory factor κB-α (IκB-α). We have previously demonstrated that l-arginine (l-Arg) attenuates neutrophil accumulation and pulmonary vascular injury after administration of lipopolysaccharide (LPS). We hypothesized thatl-Arg would attenuate the production of lung chemokines by stabilizing IκB-α and preventing NF-κB DNA binding. We examined the effect of l-Arg on chemokine production, IκB-α degradation, and NF-κB DNA b
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Gewirtz, AM, J. Zhang, J. Ratajczak, et al. "Chemokine regulation of human megakaryocytopoiesis." Blood 86, no. 7 (1995): 2559–67. http://dx.doi.org/10.1182/blood.v86.7.2559.2559.

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Abstract We have previously shown that platelet factor 4 (PF4), a platelet-specific CXC chemokine, can directly and specifically inhibit human megakaryocyte colony formation. We therefore hypothesized that PF4 might function as a negative autocrine regulator of megakaryocytopoiesis. Herein we present additional studies characterizing the inhibitory effect of CXC chemokines on human megakaryocyte development. We first corroborated our initial studies by showing that recombinant human (rH) PF4, like the native protein, inhibited megakaryocytopoiesis. We then examined the inhibitory properties of
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Gewirtz, AM, J. Zhang, J. Ratajczak, et al. "Chemokine regulation of human megakaryocytopoiesis." Blood 86, no. 7 (1995): 2559–67. http://dx.doi.org/10.1182/blood.v86.7.2559.bloodjournal8672559.

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We have previously shown that platelet factor 4 (PF4), a platelet-specific CXC chemokine, can directly and specifically inhibit human megakaryocyte colony formation. We therefore hypothesized that PF4 might function as a negative autocrine regulator of megakaryocytopoiesis. Herein we present additional studies characterizing the inhibitory effect of CXC chemokines on human megakaryocyte development. We first corroborated our initial studies by showing that recombinant human (rH) PF4, like the native protein, inhibited megakaryocytopoiesis. We then examined the inhibitory properties of other CX
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Chen, Xin, Lu Yang, Ning Zhang, et al. "Shikonin, a Component of Chinese Herbal Medicine, Inhibits Chemokine Receptor Function and Suppresses Human Immunodeficiency Virus Type 1." Antimicrobial Agents and Chemotherapy 47, no. 9 (2003): 2810–16. http://dx.doi.org/10.1128/aac.47.9.2810-2816.2003.

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ABSTRACT Shikonin is a major component of zicao (purple gromwell, the dried root of Lithospermum erythrorhizon), a Chinese herbal medicine with various biological activities, including inhibition of human immunodeficiency virus (HIV) type 1 (HIV-1). G protein-coupled chemokine receptors are used by HIV-1 as coreceptors to enter the host cells. In this study, we assessed the effects of shikonin on chemokine receptor function and HIV-1 replication. The results showed that, at nanomolar concentrations, shikonin inhibited monocyte chemotaxis and calcium flux in response to a variety of CC chemokin
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Wu, X., G. J. Dolecki, and J. B. Lefkowith. "GRO chemokines: a transduction, integration, and amplification mechanism in acute renal inflammation." American Journal of Physiology-Renal Physiology 269, no. 2 (1995): F248—F256. http://dx.doi.org/10.1152/ajprenal.1995.269.2.f248.

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We recently observed that cytokine-induced neutrophil chemoattractant (CINC), a GRO chemokine, contributes to neutrophil migration into the inflamed glomerulus in rat. Therefore, we sought to clarify how expression of the GRO chemokines, CINC and macrophage inflammatory protein-2 (MIP-2), is regulated in mesangial cells in vitro and the kidney in vivo. Mesangial cells expressed both GRO chemokine mRNAs in response to mediators of acute renal inflammation [interleukin-1 beta (IL-1 beta), tumor necrosis factor-alpha (TNF-alpha), and lipopolysaccharides (LPS)], but not chronic renal inflammation
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RECKLESS, Jill, and David J. GRAINGER. "Identification of oligopeptide sequences which inhibit migration induced by a wide range of chemokines." Biochemical Journal 340, no. 3 (1999): 803–11. http://dx.doi.org/10.1042/bj3400803.

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We have identified an amino acid sequence, termed peptide 3, corresponding to amino acids 51-62 of the mature human monocyte chemoattractant protein-1 (MCP-1), which inhibits human mononuclear-cell and THP-1-cell migration induced by a wide range of chemokines. For example, peptide 3 inhibited MCP-1-induced THP-1 migration in a transwell assay with an ED50 of approx. 8 μM. Peptide 3 binds directly to THP-1 cells with an association constant of approx. 10 μM, and is therefore likely to be a direct receptor antagonist for CC and CXC chemokine receptors. By performing a structure-function analysi
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Kim, Jung-Hoon, Hye-Sun Lim, Hyekyung Ha, Chang-Seob Seo та Hyeun-Kyoo Shin. "Inulae Flos and Its Compounds Inhibit TNF-α- and IFN-γ-Induced Chemokine Production in HaCaT Human Keratinocytes". Evidence-Based Complementary and Alternative Medicine 2012 (2012): 1–11. http://dx.doi.org/10.1155/2012/280351.

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The present study is to investigate which kinds of solvent extracts of Inulae Flos inhibit the chemokine productions in HaCaT cell and whether the inhibitory capacity of Inulae Flos is related with constitutional compounds. The 70% methanol extract showed comparatively higher inhibition of thymus and activation-regulated chemokine (TARC/CCL17) in HaCaT cells, therefore this extract was further partitioned with n-hexane, chloroform, ethyl acetate, butanol, and water. The ethyl acetate fraction inhibited TARC, macrophage-derived chemokine (MDC/CCL22), and regulated on activation of normal T-cell
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Chen, Jing-Yi, You-Syuan Lai, Pei-Yi Chu, Shih-Hsuan Chan, Lu-Hai Wang, and Wen-Chun Hung. "Cancer-Derived VEGF-C Increases Chemokine Production in Lymphatic Endothelial Cells to Promote CXCR2-Dependent Cancer Invasion and MDSC Recruitment." Cancers 11, no. 8 (2019): 1120. http://dx.doi.org/10.3390/cancers11081120.

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Breast cancer-derived vascular endothelial growth factor-C (VEGF-C) has been shown to enhance lymphangiogenesis in lymph nodes to accelerate cancer metastasis. However, the remodeling of lymph node microenvironments by VEGF-C remains elusive. By in vivo selection, we established a subline (named as “LC”) with strong lymphatic tropism and high VEGF-C expression from the human MDA-MB-231 breast cancer cell line. Co-culture with LC cells or treatment with LC-conditioned medium upregulated the expression of CXC chemokines in lymphatic endothelial cells (LECs), which could be inhibited by pre-incub
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Liu, Dongxiang, Navid Madani, Ying Li, et al. "Crystal Structure and Structural Mechanism of a Novel Anti-Human Immunodeficiency Virus and d-Amino Acid-Containing Chemokine." Journal of Virology 81, no. 20 (2007): 11489–98. http://dx.doi.org/10.1128/jvi.02845-06.

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ABSTRACT Chemokines and their receptors play important roles in normal physiological functions and the pathogeneses of a wide range of human diseases, including the entry of human immunodeficiency virus type 1 (HIV-1). However, the use of natural chemokines to probe receptor biology or to develop therapeutic drugs is limited by their lack of selectivity and the poor understanding of mechanisms in ligand-receptor recognition. We addressed these issues by combining chemical and structural biology in research into molecular recognition and inhibitor design. Specifically, the concepts of chemical
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Watanabe, Toshio, Kazuhide Higuchi, Masaki Hamaguchi та ін. "Monocyte chemotactic protein-1 regulates leukocyte recruitment during gastric ulcer recurrence induced by tumor necrosis factor-α". American Journal of Physiology-Gastrointestinal and Liver Physiology 287, № 4 (2004): G919—G928. http://dx.doi.org/10.1152/ajpgi.00372.2003.

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TNF-α has numerous biological activities, including the induction of chemokine expression, and is involved in many gastric injuries. C-C chemokines [monocyte chemotactic protein (MCP)-1 and macrophage inflammatory protein (MIP)-1α] and C-X-C chemokines [MIP-2 and cytokine-induced neutrophil chemoattractant (CINC)-2α] mediate chemotaxis of monocytes and neutrophils, respectively. We examined the roles of TNF-α and dynamics of chemokine expression in gastric ulceration including ulcer recurrence and indomethacin-induced injury. Rats with healed chronic gastric ulcers received intraperitoneal TNF
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Dissertations / Theses on the topic "Inhibitor chemokinu"

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Simmons, Graham. "Human immunodeficiency syndrome virus type 1 cell tropism and inhibition by chemokines and chemokine analogues." Thesis, Institute of Cancer Research (University Of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368041.

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Zhang, Li. "Structural study of the interaction between poxvirus-encoded cc chemokine inhibitor vcci and human mip-1beta." Diss., Texas A&M University, 2008. http://hdl.handle.net/1969.1/85901.

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Chemokines (chemotactic cytokines) comprise a large family of proteins that recruit and activate leukocytes, giving chemokines a major role in both immune response and inflammation-related diseases. Viral CC chemokine inhibitor (vCCI) is a poxvirus encoded protein that has been shown to bind tightly and inhibit the action of many CC chemokines. This function suggests that vCCI could be explored as an antiinflammatory therapeutic, a possibility that has been supported in mouse studies. The structure of vCCI in unbound form was determined by others, but to date no structure has been reported of
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Montero, Rosa Maria. "Chemokines and macrophage migration inhibitory factor in diabetic nephropathy." Thesis, Imperial College London, 2013. http://hdl.handle.net/10044/1/29851.

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Introduction: Diabetic nephropathy (DN) is the leading cause of end-stage renal disease in the Western world. Aim: To determine whether macrophage migration inhibitory factor (MIF), monocyte chemoattractant protein-1 (MCP-1) or CC chemokine ligand 18 (CCL18) have a causative role in the development of renal inflammation and fibrosis in DN and are useful biomarkers of disease progression. Methods: Urine and plasma samples were collected from 115 DM and 116 Non-DM at baseline, previously analysed for MCP-1 and CCL18 ELISA by Dr Qureshi. I measured MIF in these samples and collected 107 DM and 11
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Bono, Johann S. de. "Macrophage inflammatory protein-1#alpha# : an inhibitor of clonogenic epidermal keratinocyte proliferation?" Thesis, University of Glasgow, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298214.

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Kok, Tsz-wai. "Blockade of chemokine (C-X-C motif) receptor 4 for the inhibition of hepatocellular carcinoma metastasis." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B4068765X.

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Brahm, Kevin [Verfasser], Katja [Akademischer Betreuer] Schmitz, and Harald [Akademischer Betreuer] Kolmar. "Untersuchung von Peptidomimetika als Inhibitoren für das Chemokin CXCL8 / Kevin Brahm ; Katja Schmitz, Harald Kolmar." Darmstadt : Universitäts- und Landesbibliothek Darmstadt, 2020. http://d-nb.info/1211726185/34.

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Kok, Tsz-wai, and 郭梓瑋. "Blockade of chemokine (C-X-C motif) receptor 4 for the inhibition of hepatocellular carcinoma metastasis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B4068765X.

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Jöst, Marina [Verfasser], Katja [Akademischer Betreuer] Schmitz, and Harald [Akademischer Betreuer] Kolmar. "Etablierung von zellbasierten Assays zur Identifizierung von Inhibitoren des Chemokins CXCL8 / Marina Jöst. Betreuer: Katja Schmitz ; Harald Kolmar." Darmstadt : Universitäts- und Landesbibliothek Darmstadt, 2016. http://d-nb.info/1112333231/34.

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Eriksson, Catharina. "Immunological mechanisms in systemic autoimmunity : autoantibodies and chemokines in systemic lupus erythematosus and during treatment with TNF inhibitors in rheumatoid arthritis." Doctoral thesis, Umeå universitet, Klinisk immunologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-42954.

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Background. Rheumatoid Arthritis (RA) is an autoimmune inflammatory disease that, without powerful treatment, may lead to irreversible joint damage. During the past decade, anti-cytokine therapy has become available, e.g., infliximab, a chimeric antibody targeting the pro-inflammatory cytokine TNF that has a central role in the inflammatory process in RA patients. Systemic Lupus Erythematosus (SLE) is a systemic autoimmune disease that may affect all organs and is characterized by a massive antibody production. Chemokines, chemokine receptors and lipoprotein receptor-related protein 1(CD91) ar
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Amati, Anca-Laura [Verfasser]. "Chemokines (CCL3, CCL4, CCL5) inhibit ATP-induced release of IL-1beta by monocytic cells / Anca-Laura Amati." Gießen : Universitätsbibliothek, 2019. http://d-nb.info/1198109173/34.

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Books on the topic "Inhibitor chemokinu"

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Mease, Philip. Neurobiology of pain in osteoarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0013.

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Significant advances in our understanding of the neurobiology of pain in osteoarthritis (OA) have occurred in the last decade and are herein summarized. Pain is the predominant symptom of OA and occurs at multiple levels from non-cartilage peripheral tissues to spinal cord, and brain and back. At each level, nerve function is regulated by complex ionic channels, neuropeptide expression, and cytokine and chemokine activity. Previously considered a non-inflammatory condition, it is now recognized that cell proliferation and inflammatory cytokine production occurs in OA synovium, contributing to
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Book chapters on the topic "Inhibitor chemokinu"

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Hippe, Andreas, Bernhard Homey, and Anja Mueller-Homey. "Chemokines." In Angiogenesis Inhibition. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-78281-0_4.

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Graham, Gerard J., and Robert J. B. Nibbs. "Macrophage Inflammatory Protein-1α and Stem Cell Inhibition." In Chemokines and Cancer. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-701-7_16.

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Su, S. B., H. Ueda, O. M. Z. Howard, et al. "Inhibition of the Expression and Function of Chemokine Receptors on Human CD4+ Leukocytes by HIV-1 Envelope Protein gp120." In Chemical Immunology and Allergy. KARGER, 1999. http://dx.doi.org/10.1159/000058731.

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Zhang, W., C. Smith, R. Monette, J. Hutchison, and D. B. Stanimirovic. "Indomethacin and Cyclosporin a Inhibit in Vitro Ischemia-Induced Expression of ICAM-1 and Chemokines in Human Brain Endothelial Cells." In Brain Edema XI. Springer Vienna, 2000. http://dx.doi.org/10.1007/978-3-7091-6346-7_10.

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Trkola, Alexandra, Timothy Wells, and Amanda Proudfoot-Fichard. "Chemokine Receptors." In Cytokine Inhibitors. CRC Press, 2000. http://dx.doi.org/10.1201/9780203904244.ch7.

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Cairns, J. Scott, and M. Patricia D’Souza. "Therapies to Prevent or Inhibit Chemokine Receptor Expression." In Chemokine Receptors and AIDS. CRC Press, 2019. http://dx.doi.org/10.1201/9780429074974-12.

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Lüscher, Thomas F., and Paul M. Ridker. "Anti-inflammatory therapies for cardiovascular disease." In ESC CardioMed. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0272.

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Inflammation involves many blood cells and the molecules released by them such as cytokines, chemokines, and antibodies. In particular, cells derived from monocyte/macrophage lines are involved in atherogenesis, as are numerous chemokines, cytokines, and adhesion molecules expressed in the vasculature or adipose tissue. Different inflammatory pathways have been considered in the prevention and treatment of cardiovascular disease. Specifically, as outlined previously in a review of the authors, several lines of evidence support the concept that inhibition of the central immune pathway linking interleukin 1, tumour necrosis factor alpha, and interleukin 6 might be novel targets.
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Barnes, Debra A., Steven W. Jones, and H. Daniel Perez. "High throughput screening for identification of RANTES chemokine expression inhibitors." In Methods in Enzymology. Elsevier, 1997. http://dx.doi.org/10.1016/s0076-6879(97)87021-8.

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Kuang, Yi-Qun. "The Development and Clinical Progress on Chemokine Receptor-Based HIV Entry Inhibitors." In Frontiers in Clinical Drug Research - HIV. BENTHAM SCIENCE PUBLISHERS, 2019. http://dx.doi.org/10.2174/9781681085265119040006.

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Mukherjee, Sitabja, and Santosh K. Kar. "Curcuminoids: The Novel Molecules of Nature." In Herbs and Spices - New Processing Technologies [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99201.

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Curcuminoids inactivate Nuclear Factor-Kappa B (NF-κB), a key pro-inflammatory transcription factor which is involved in inflammation and immune response in diseases like cancer. NF-κB activation is necessary to determine tumor microenvironment which controls migration and metastatis of cancer cells through chemokines and their receptors and involvement of some cell adhesion molecules. Therefore inhibition of NF-κB by curcuminoids could be a new approach in treatment of cancer by immune modulation. Curcuminoids are not bioavailable and therefore there were problems in efficacy. Now by using bioavailable curcuminoid formulations the problem has been resolved to a great extent. Out of 49 placebo controlled double blind clinical trials using curcuminoids, 17 have been found to be successful. Therefore curcuminoids could be developed as an adjunct therapy for diseases like cancer to save human life.
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Conference papers on the topic "Inhibitor chemokinu"

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"Impact of Heparan Sulphate Binding Domain of Chemokine CCL21 to Migration of Breast Cancer Cells." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0132.

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Lymph node metastasis constitutes a key event in breast cancer progression. Chemokines are small proteins, which can promote metastatic spread by inducing cancer cell migration and invasion. Chemokine function is dependant upon their binding to both cell surface heparan sulphate (HS) molecules and to their specific receptor. Our group has demonstrated a significant increase in chemokine receptor CCR7 expression in cancerous breast epithelia compared to healthy controls. This study is designed to test the hypothesis that a non-HS binding forms of chemokine CCL21 can disrupt the normal response
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Renner, Andreas, Katharina Marth, Helmut Schmutz, Julia Romanova, Boris Ferko, and Wolfgang Pohl. "Phase 1 trial of a novel chemokine inhibitor." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa3712.

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Ju, Donghong, Cecelia Speyer, David Gorski, and Mary A. Kosir. "Abstract 3008: Effects of HSPG on inhibition of chemokine-induced angiogenesis." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3008.

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Seitz, S., C. Stange, T. Dreyer, J. Nikonov, M. Kiechle, and H. Bronger. "Der PARP-Inhibitor Olaparib induziert die tumor-suppressiven Chemokine CXCL9 und CX3CL1 im Ovarialkarzinom." In Kongressabstracts zur Tagung 2020 der Deutschen Gesellschaft für Gynäkologie und Geburtshilfe (DGGG). © 2020. Thieme. All rights reserved., 2020. http://dx.doi.org/10.1055/s-0040-1718124.

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Zhang, Jihui, Jon Richardson, Jie Chen, et al. "A Novel Der P 1 Inhibitor ADZ 51,457 Inhibits Eosinophil Recruitment And Chemokine Release Following House Dust Mite Allergen Challenge In Brown Norway Rats." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2792.

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Mir, Hina, Neeraj Kapur, Rajesh Singh, Guru Sonpavde, James W. Lillard, and Shailesh Singh. "Abstract 5362: Andrographolide inhibits prostate cancer by modulating chemokine and cytokines." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-5362.

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Akakura, Shin, Masashi Muramatsu, Hyun Kyung Ko, and Irwin H. Gelman. "Abstract 3429: AKAP12/SSeCKS inhibits peritoneal metastasis by regulating chemokine production." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-3429.

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Blake, David J. "Inhibition Of Cigarette Smoke-Induced Chemokine Production By The NRF2 Activator Sulforaphane." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2846.

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Ganesan, Shyamala, Sangbrita Chattoraj, Andrea N. Faris, Adam Comstock, and Umadevi Sajjan. "Quercetin Inhibits Rhinovirus Replication And Subsequent Chemokine Response In Airway Epithelial Cells." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6847.

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Burton, Victoria J., Nicholas Duggan, Betty Shamji, et al. "Inhibition Of The Chemokine Receptor CXCR1/2 Attenuates Experimental Severe Pulmonary Arterial Hypertension." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5091.

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