Relevant bibliographies by topics / Inhibitory neurotransmitters

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Inhibitory neurotransmitters'

Author: Grafiati
Published: 4 June 2021
Last updated: 27 July 2025

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Journal articles on the topic "Inhibitory neurotransmitters"

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Kim, Dongshin, and Jang-Sik Lee. "Emulating Excitatory and Inhibitory Functions in Artificial Synaptic Devices." ECS Meeting Abstracts MA2022-02, no. 33 (2022): 2585. http://dx.doi.org/10.1149/ma2022-02332585mtgabs.

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Synaptic signals are controlled by neurotransmitters. The synaptic signals can be excited or inhibited depending on the types of neurotransmitters. The demonstration of the balancing between excitatory and inhibitory signals has important implications for the complex and efficient computing of the nervous system. Emulating the excitatory-inhibitory balancing behaviors of the nervous system is one way to establish neuromorphic computing. In this study, we demonstrate artificial synapses using PEDOT:PSS channel and neurotransmitter solutions to emulate the excitatory-inhibitory balancing behavio
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Huang, Chun-Ping, Yi-Wen Lin, Der-Yen Lee, and Ching-Liang Hsieh. "Electroacupuncture Relieves CCI-Induced Neuropathic Pain Involving Excitatory and Inhibitory Neurotransmitters." Evidence-Based Complementary and Alternative Medicine 2019 (October 20, 2019): 1–9. http://dx.doi.org/10.1155/2019/6784735.

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Neuropathic pain caused by peripheral tissue injuries to the higher brain regions still has no satisfactory therapy. Disruption of the balance of excitatory and inhibitory neurotransmitters is one of the underlying mechanisms that results in chronic neuropathic pain. Targeting neurotransmitters and related receptors may constitute a novel approach for treating neuropathic pain. We investigated the effects of electroacupuncture (EA) on chronic constriction injury- (CCI-) induced neuropathic pain. The mechanical allodynia and thermal hyperalgesia pain behaviors were relieved by 15 Hz EA but not
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Foxx-Orenstein, A. E., and J. R. Grider. "Regulation of colonic propulsion by enteric excitatory and inhibitory neurotransmitters." American Journal of Physiology-Gastrointestinal and Liver Physiology 271, no. 3 (1996): G433—G437. http://dx.doi.org/10.1152/ajpgi.1996.271.3.g433.

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The contribution of excitatory and inhibitory motor neurotransmitters to colonic propulsion was examined in isolated segments of guinea pig colon. Synthetic fecal pellets were inserted at the proximal end of the segment, and the velocity of pellet propulsion across a fixed distance was measured in the presence and absence of selective neurotransmitter antagonists. The control velocity (0.97 +/- 0.02 mm/s) was inhibited in a concentration-dependent fashion by atropine and the neurokinin (NK)-2a antagonist MEN-10,376 [half-maximal inhibitory concentration (IC50), 1 microM; maximal inhibition, 98
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Nilsson, G. E., and P. L. Lutz. "Release of inhibitory neurotransmitters in response to anoxia in turtle brain." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 261, no. 1 (1991): R32—R37. http://dx.doi.org/10.1152/ajpregu.1991.261.1.r32.

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In mammals a massive release of the excitatory neurotransmitter glutamate, occurring within a few minutes of anoxia/ischemia, is thought to be a major mediator of anoxic brain damage. In contrast to the mammalian brain, the turtle brain is exceptionally anoxia tolerant. Using intracerebral microdialysis in turtle brain striatum, we have found a large increase in the extracellular level of the inhibitory neurotransmitter gamma-aminobutyric acid during anoxia, reaching 90 times the normoxic level after 240 min, whereas no substantial release of glutamate occurred. Moreover, the inhibitory neurot
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Korpi, Esa R., Riikka Mäkelä, and Mikko Uusi-Oukari. "Ethanol: Novel Actions on Nerve Cell Physiology Explain Impaired Functions." Physiology 13, no. 4 (1998): 164–70. http://dx.doi.org/10.1152/physiologyonline.1998.13.4.164.

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Molecular biological tools have revealed receptor proteins for excitatory and inhibitory neurotransmitters on cell membranes as targets of ethanol action. Behavioral and pharmacogenetic assays using rodent lines have supported this neurotransmitter theory of ethanol action and given a firm basis for future identification of the relevant genes and the central physiological processes vulnerable to ethanol.
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Lammers, JW, PJ Barnes, and KF Chung. "Nonadrenergic, noncholinergic airway inhibitory nerves." European Respiratory Journal 5, no. 2 (1992): 239–46. http://dx.doi.org/10.1183/09031936.93.05020239.

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Nonadrenergic, noncholinergic (NANC) nerves, which cause relaxation of airway smooth muscle, have been described in several species including man. Stimulation of efferent vagus nerves during cholinergic and adrenergic blockade induces a pronounced bronchodilation in the cat. In more recent studies in man, capsaicin inhalation or mechanical irritation of the larynx, under conditions of cholinergic and adrenergic blockade, have been shown to cause a transient bronchodilator response. There is some evidence that neuropeptides such as vasointestinal peptide (VIP) or peptide histidine methionine (P
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Neuray, Caroline, Reza Maroofian, Marcello Scala, et al. "Early-infantile onset epilepsy and developmental delay caused by bi-allelic GAD1 variants." Brain 143, no. 8 (2020): 2388–97. http://dx.doi.org/10.1093/brain/awaa178.

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Abstract Gamma-aminobutyric acid (GABA) and glutamate are the most abundant amino acid neurotransmitters in the brain. GABA, an inhibitory neurotransmitter, is synthesized by glutamic acid decarboxylase (GAD). Its predominant isoform GAD67, contributes up to ∼90% of base-level GABA in the CNS, and is encoded by the GAD1 gene. Disruption of GAD1 results in an imbalance of inhibitory and excitatory neurotransmitters, and as Gad1−/− mice die neonatally of severe cleft palate, it has not been possible to determine any potential neurological dysfunction. Furthermore, little is known about the conse
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Chandana, Bandaru, G. Krishna Mohan, and M. Sandhya Rani. "Advanced Molecular Mechanisms of Epilepsy." Journal of Pharmaceutical Quality Assurance and Quality Control 5, no. 2 (2023): 22–34. http://dx.doi.org/10.46610/jqaqc.2023.v05i02.004.

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The Central Nervous System (CNS) is a complex network composed of the cerebral cortex and the vertebral column, orchestrating intricate processes through neural networks and chemical regulation. Neurotransmission in the CNS involves neurotransmitters, neuromodulators, neuromediators, and neurotropic factors, playing distinct roles in cellular activity and synaptic plasticity. Various neurotransmitters such as dopamine, glutamate, GABA, glycine, serotonin, and others exert diverse effects on the CNS through specific receptors, influencing synaptic transmission and neuronal excitability. GABA, t
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Hassan, Ahmed H. E., Hyeon Jeong Kim, Keontae Park, et al. "Synthesis and Biological Evaluation of O6-Aminoalkyl-Hispidol Analogs as Multifunctional Monoamine Oxidase-B Inhibitors towards Management of Neurodegenerative Diseases." Antioxidants 12, no. 5 (2023): 1033. http://dx.doi.org/10.3390/antiox12051033.

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Oxidative catabolism of monoamine neurotransmitters by monoamine oxidases (MAOs) produces reactive oxygen species (ROS), which contributes to neuronal cells’ death and also lowers monoamine neurotransmitter levels. In addition, acetylcholinesterase activity and neuroinflammation are involved in neurodegenerative diseases. Herein, we aim to achieve a multifunctional agent that inhibits the oxidative catabolism of monoamine neurotransmitters and, hence, the detrimental production of ROS while enhancing neurotransmitter levels. Such a multifunctional agent might also inhibit acetylcholinesterase
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Łażewska, Dorota, Paula Zaręba, Justyna Godyń, et al. "Biphenylalkoxyamine Derivatives–Histamine H3 Receptor Ligands with Butyrylcholinesterase Inhibitory Activity." Molecules 26, no. 12 (2021): 3580. http://dx.doi.org/10.3390/molecules26123580.

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Neurodegenerative diseases, e.g., Alzheimer’s disease (AD), are a key health problem in the aging population. The lack of effective therapy and diagnostics does not help to improve this situation. It is thought that ligands influencing multiple but interconnected targets can contribute to a desired pharmacological effect in these complex illnesses. Histamine H3 receptors (H3Rs) play an important role in the brain, influencing the release of important neurotransmitters, such as acetylcholine. Compounds blocking their activity can increase the level of these neurotransmitters. Cholinesterases (a
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Dissertations / Theses on the topic "Inhibitory neurotransmitters"

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Jordan, Russell S. (Russell Stall). "Investigation of Inhibitory Influences in Neuronal Monolayer Networks Cultured from Mouse Spinal Cord." Thesis, University of North Texas, 1992. https://digital.library.unt.edu/ark:/67531/metadc500431/.

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The effects of the inhibitory neurotransmitters gammaamino butyric acid (GABA) and glycine were characterized on spontaneous activity recorded from mouse spinal cord cultures. The GABA concentration which completely inhibited burst activity was chosen as a quantifiable measure of culture drug response and was used to 1) assess interculture and intraculture variability, 2) determine the influence of culture age and initial activity on GABA responses, and 3) compare the GABA responses between networks obtained from whole spinal cord and ventral half spinal cord. Results showed that 1) no signifi
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Hudman, Diane. "Physiological and behavioural actions of putative inhibitory neurotransmitters in sea anemones." Thesis, University of Hull, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363274.

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Chung, Seo-Kyung. "Molecular and biophysical characterization of the glycinergic inhibitory system." Thesis, Swansea University, 2009. https://cronfa.swan.ac.uk/Record/cronfa42277.

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Glycinergic neurotransmission is a major inhibitory influence in the CNS and defects are associated with paroxysmal neuromotor disorder, hyperekplexia with mutations in subunits of the inhibitory glycine receptor which facilitates postsynaptic ligand-binding, ion-channels. This study investigates the human glycinergic system by; 1) Mutation analysis of glycinergic candidate genes in hyperekplexia: the DNA sequencing of GLRAl in 88 hyperekplexia patients revealed 30 sequence variants; 21 were inherited in recessive mode or part of compound heterozygosity, indicating that recessive hyperekplexia
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De, Faoite Andrew. "Modulation of spontaneous activity in the rabbit urethra by excitatory and inhibitory neurotransmitters." Thesis, Queen's University Belfast, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.517278.

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Zhao, Wei. "The inhibitory mechanisms of sound processing in the dorsal cochlear nucleus of the guinea pig." Thesis, Keele University, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297207.

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Ellenberger, Claudia. "Inhibitory and excitatory neurotransmitters in the cerebrospinal fluid of dogs with genetic and primary (idiopathic) epilepsy /." [S.l.] : [s.n.], 2002. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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Kurcyus, Katarzyna [Verfasser], Valentin [Akademischer Betreuer] Riedl, Helmuth [Gutachter] Adelsberger, and Markus [Gutachter] Ploner. "Using magnetic resonance spectroscopy to study neural activity: The role of inhibitory and excitatory neurotransmitters for systems-level brain dynamics / Katarzyna Kurcyus ; Gutachter: Helmuth Adelsberger, Markus Ploner ; Betreuer: Valentin Riedl." München : Universitätsbibliothek der TU München, 2017. http://d-nb.info/1172880034/34.

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Bavro, Vassiliy Nikolaevitch. "Structural studies of molecules involved in clustering and trafficking of inhibitory neurotransmitter receptors." Université Joseph Fourier (Grenoble), 2003. http://www.theses.fr/2003GRE10180.

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Li, Libin Reith Maarten E. A. "Regulation of the human dopamine transporter." Normal, Ill. Illinois State University, 2002. http://wwwlib.umi.com/cr/ilstu/fullcit?p3064534.

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Thesis (Ph. D.)--Illinois State University, 2002.<br>Title from title page screen, viewed February 3, 2006. Dissertation Committee: Maarten E.A. Reith (chair), Hou Tak Cheung, Paul A. Garris, Stephen M. Lasley, Brian J. Wilkinson. Includes bibliographical references and abstract. Also available in print.
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Garcia, Adrian Dante. "Neural correlates of visual perceptual learning and inhibitory neurotransmitter in humans using magnetic resonance spectroscopy." Thesis, University of Birmingham, 2017. http://etheses.bham.ac.uk//id/eprint/7770/.

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The role of excitatory processes in human visual learning has been well characterised through the use of technologies such as functional magnetic resonance imaging (fMRI). However, established imaging modalities do not distinguish excitatory processes from the inhibitory ones that are also involved. Here we investigate inhibitory processes using magnetic resonance spectroscopy (MRS) and the MEGA-PRESS pulse sequence. We measure concentrations of the inhibitory neurotransmitter gamma-aminobutyric acid (GABA) in different brain regions as functional markers of inhibitory potential. We then inves
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Books on the topic "Inhibitory neurotransmitters"

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A, Cavalheiro Esper, Lehmann John 1952-, and Turski Lechoslaw, eds. Frontiers in excitatory amino acid research: Proceedings of an International Symposium "Excitatory Amino Acids '88," held in Manaus, Amazonas, Brazil, March 28-April 2, 1988. Liss, 1988.

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1955-, Burch Ronald M., ed. Bradykinin antagonists: Basic and clinical research. Dekker, 1991.

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E, Becker Robert, and Giacobini Ezio, eds. Cholinergic basis for Alzheimer therapy. Birkhäuser, 1991.

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Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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Stafstrom, Carl E. Disorders Caused by Botulinum Toxin and Tetanus Toxin. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0156.

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Anaerobic organisms of the genus Clostridia (C) can cause significant human disease. Exotoxins secreted by C botulinum and C tetani cause botulism and tetanus, respectively (summarized in Table 156.1). Botulinum neurotoxin causes neuromuscular blockade by interfering with vesicular acetylcholine release, leading to cholinergic blockade at the neuromuscular junctions of skeletal muscle, and consequently, symmetric flaccid paralysis. Tetanus toxin prevents release of inhibitory neurotransmitters at central synapses, leading to overactivity of motor neurons and muscle rigidity and spasms. This ch
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Castellanos, Madeleine M. Female Sexual Biochemistry (DRAFT). Edited by Madeleine M. Castellanos. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190225889.003.0001.

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“Female Sexual Biochemistry” reviews the key hormones and neurotransmitters that have a major role in female sexuality. Estrogens—estradiol, estrone, and estriol—as well as major androgens, such as testosterone and dihydrotestosterone (DHT), are presented with a discussion of their role in the support of the reproductive organs and genitals as well as their actions on the central nervous system to affect sexual desire, arousal, and responsiveness. The interaction and regulation of estrogen by progesterone and thyroid hormone is included. A review of the dual-control model of sexual responsiven
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Nutt, David J., and Liam J. Nestor. The GABA system and addiction. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198797746.003.0008.

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Research points to the potential role of gamma-aminobutyric acid (GABA) in substance addiction. GABA is the major inhibitory neurotransmitter in the brain. Disturbances in the GABA system may predate substance abuse and addiction, whereby its efficacy to modulate other neurotransmitter systems (e.g. dopamine) strongly implicated in substance addiction behaviours is impaired. There are a number of addictive substances that boost GABA functioning, however, such as alcohol and benzodiazepines. Medications that boost the availability of GABA or mimic its effects at receptors may possess some clini
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Ziemann, Ulf. Pharmacology of TMS measures. Edited by Charles M. Epstein, Eric M. Wassermann, and Ulf Ziemann. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780198568926.013.0013.

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This article discusses various aspects of the pharmacology of transcranial magnetic stimulator (TMS) measures. TMS measures reflect axonal, or excitatory or inhibitory synaptic excitability in distinct interneuron circuits. TMS measures can be employed to study the effects of a drug with unknown or multiple modes of action, and hence to determine its main mode of action at the systems level of the motor cortex. TMS experiments can also study acute drug effects that may be different from chronic drug effects. TMS or repetitive TMS may induce changes in endogenous neurotransmitter or neuromodula
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Roze, Emmanuel, and Nenad Blau. Biogenic Monoamine Disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0031.

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Biogenic monoamine disorders are a group of inherited diseases characterized by a defect in the synthesis, transport, or degradation of catecholamines and serotonin. The phenotype mostly reflects the pattern and severity of the monoamine deficiency. Movement disorders due to cerebral dopamine deficiency are almost always prominent, mostly in the form of dystonia and/or parkinsonism. These disorders are potentially devastating yet treatable. Early diagnosis and treatment are crucial to prevent ongoing brain dysfunction. Detection of hyperphenylalaninemia in a neonate could be a good clue to the
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Studying the binding of phosphodiesterase-4 inhibitor R-[11C]rolipram in rat brain: Effects of glutamatergic modulation on various neurotransmitter systems and validation binding assays. National Library of Canada, 2003.

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Book chapters on the topic "Inhibitory neurotransmitters"

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McGeer, Patrick L., John C. Eccles, and Edith G. McGeer. "Inhibitory Amino Acid Neurotransmitters." In Molecular Neurobiology of the Mammalian Brain. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4615-7497-2_7.

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Kruk, Zygmunt L., and Christopher J. Pycock. "Inhibitory amino acids: GABA, glycine and taurine." In Neurotransmitters and Drugs. Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3132-2_7.

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Kruk, Zygmunt L., and Christopher J. Pycock. "Inhibitory amino acids: GABA, glycine and taurine." In Neurotransmitters and Drugs. Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3134-6_7.

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Hammond, D. L. "Inhibitory Neurotransmitters and Nociception: Role of GABA and Glycine." In Handbook of Experimental Pharmacology. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60777-6_14.

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Gillespie, Deda C., and Karl Kandler. "GABA, Glycine, and Glutamate Co-Release at Developing Inhibitory Synapses." In Co-Existence and Co-Release of Classical Neurotransmitters. Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-09622-3_5.

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Dieudonné, Stéphane, and Marco Alberto Diana. "Postsynaptic Determinants of Inhibitory Transmission at Mixed GABAergic/Glycinergic Synapses." In Co-Existence and Co-Release of Classical Neurotransmitters. Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-09622-3_7.

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Barnerssoi, Miriam, and Paul J. May. "Postembedding Immunohistochemistry for Inhibitory Neurotransmitters in Conjunction with Neuroanatomical Tracers." In Neuromethods. Springer New York, 2015. http://dx.doi.org/10.1007/7657_2015_79.

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Smith, Paul F., and Cynthia L. Darlington. "The Contributions of Excitatory and Inhibitory Amino Acid Neurotransmitters to Vestibular Plasticity." In Neurochemistry of the Vestibular System. Routledge, 2023. http://dx.doi.org/10.1201/9780203745922-24.

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Cocks, Tom M., and Stavros Selemidis. "Interactions Between Nitric Oxide and Other NANC Inhibitory Neurotransmitters in the Periphery: Clues from the Endothelium." In Nitric Oxide and Free Radicals in Peripheral Neurotransmission. Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1328-4_3.

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Prada, M., R. Kettler, H. H. Keller, et al. "From moclobemide to Ro 19-6327 and Ro 41-1049: the development of a new class of reversible, selective MAO-A and MAO-B inhibitors." In Neurotransmitter Actions and Interactions. Springer Vienna, 1990. http://dx.doi.org/10.1007/978-3-7091-9050-0_27.

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Conference papers on the topic "Inhibitory neurotransmitters"

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Al-Wadei, Hussein A., Mohammed H. Al-Wadei та Hildegard M. Schuller. "Abstract A6: Nicotine stimulates lung and pancreatic cancers xenografts by systemic increase in stress neurotransmitters and suppression of the inhibitory neurotransmitter γ-aminobutyric acid". У Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Nov 7-10, 2010; Philadelphia, PA. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1940-6207.prev-10-a6.

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Yuniati, Anis, and Retno Dwi Astuti. "Neural Network Synchronization of the Morris-Lecar Neuron Model Coupled with Short-Term Plasticity (STP)." In The 6th International Conference on Science and Engineering. Trans Tech Publications Ltd, 2024. http://dx.doi.org/10.4028/p-ymnn4n.

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This study used the Morris-Lecar (ML) neuron model coupled with Short-Term Plasticity (STP) to simulate neuronal connectivity and synaptic patterns. We analyze this neural network synchronization activity, examined the post-synaptic conductance patterns in the modelled neural network, investigated the dynamics of the neural network membrane potentials in the synchronous state, and analyze the Short-Term Plasticity (STP) synaptic transmission patterns by varying the inter-neuron connection probability for both inhibitory (pi) and excitatory (pe). This computational-based study was executed usin
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Yang, Chun-Lin, Nandan Shettigar, and C. Steve Suh. "A Proposition for Describing Real-World Network Dynamics." In ASME 2021 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2021. http://dx.doi.org/10.1115/imece2021-73360.

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Abstract This study presents a proposition for describing the dynamics of real-world networks under the general framework of complex networks. Outward behaviors of complex networks are the manifestation of the coupled dynamics at the macroscopic level and the individual dynamics at the microscopic level. At the macroscopic level a law of coupling governs the interactions of network constituents. At the microscopic level, the dynamics of individual constituent is defined by energy that follows normal distribution. Constituent dynamics are bounded by physical constraints. Consequently, network d
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Milenković, Dejan, Dušan Dimić, Edina Avdović, et al. "Neurotransmitter-coumarin derivatives as potential SARS-CoV-2 main protease inhibitors." In 6th International Electronic Conference on Medicinal Chemistry. MDPI, 2020. http://dx.doi.org/10.3390/ecmc2020-07308.

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Pradyumna, S. G., and S. S. Rathod. "Analysis of CMOS inhibitory synapse with varying neurotransmitter concentration, reuptake time and spread delay." In 2015 19th International Symposium on VLSI Design and Test (VDAT). IEEE, 2015. http://dx.doi.org/10.1109/isvdat.2015.7208112.

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Nascimento, Letícia A., Érica C. M. Nascimento, and João B. L. Martins. "Análise da estrutura eletrônica da tacrina e do neurotransmissor acetilcolina." In VIII Simpósio de Estrutura Eletrônica e Dinâmica Molecular. Universidade de Brasília, 2020. http://dx.doi.org/10.21826/viiiseedmol2020150.

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Alzheimer's disease (AD) is a more common neurodegenerative process in the elderly population, characterized by a progressive loss of cognitive abilities, such as memory, language skills, disorientation, attention and depression. Cholinergic hypothesis therapy is the most successful approach for the symptomatic treatment of AD. The therapy consists in the use of drugs with inhibitory action against acetylcholinesterase (AChE) to avoid the decrease of acetylcholine concentration in synaptic clefts. Thus, this research aims to carry out the electronic and structural study of tacrina drug compare
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Kono, Akiteru, Takashi Sakurai, Toshiaki Hattori, Koichi Okumura, Makoto Ishida, and Kazuaki Sawada. "Real-time monitoring of inhibitory effects on glutamate-induced neurotransmitter release using a potassium ion image sensor." In THE IRAGO CONFERENCE 2014. AIP Publishing LLC, 2015. http://dx.doi.org/10.1063/1.4913544.

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Tsukimata, Márcio Yutaka, Bianca Lumi Inomata da Silva, and Jennison Alves Guimarães. "Açaí: potential anticonvulsant agent." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.064.

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Background: Convulsion is an involuntary contraction of skeletal muscles. When considering vulnerable populations exposed to the mentioned pathophysiological situation, it is recognized that many of them will not have access to the indicated pharmacological treatment. Therefore, the ingestion of açai, Euterpe oleracea (EO) attenuates the problem, acting as an anticonvulsant. Objectives: evaluate the EO as an anticonvulsant agent. Design and setting: It is a bibliographic research and the data collection was done from the PubMed and Scielo databases. Methods: The descriptor used was “Euterpe ol
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Moore, Nathan, Srunphut Pukma, Nathanael Gray, and Rani George. "Abstract 790: A high-throughput chemical screen identifies synergistic activity between neurotransmitter receptor inhibitors and crizotinib in ALK-mutated neuroblastoma." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-790.

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Schiller, Peter H. "ON and OFF channels of the visual system." In OSA Annual Meeting. Optica Publishing Group, 1990. http://dx.doi.org/10.1364/oam.1990.thf2.

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The receptors of the mammalian retina, which all hyperpolarize to light, make signconserving synapses with OFF bipolars and sign-inverting synapses with ON bipolars. Several hypotheses have been generated to explain this arrangement. To test them we applied the neurotransmitter agonist 2-amino-4-phosphonobutyrate (APB) to the retina, which selectively blocks the ON bipolars. Two kinds of experiments were carried out: In one, we examined the visual responses of single cells while reversibly inactivating the ON channel with APB. In the other, we assessed the visual capacities of monkeys before a
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