Relevant bibliographies by topics / Inositol derive

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  2. Dissertations / Theses
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Academic literature on the topic 'Inositol derive'

Author: Grafiati
Published: 4 June 2021
Last updated: 12 February 2022

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Journal articles on the topic "Inositol derive"

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López-Gambero, Antonio J., Carlos Sanjuan, Pedro Jesús Serrano-Castro, Juan Suárez, and Fernando Rodríguez de Fonseca. "The Biomedical Uses of Inositols: A Nutraceutical Approach to Metabolic Dysfunction in Aging and Neurodegenerative Diseases." Biomedicines 8, no. 9 (2020): 295. http://dx.doi.org/10.3390/biomedicines8090295.

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Inositols are sugar-like compounds that are widely distributed in nature and are a part of membrane molecules, participating as second messengers in several cell-signaling processes. Isolation and characterization of inositol phosphoglycans containing myo- or d-chiro-inositol have been milestones for understanding the physiological regulation of insulin signaling. Other functions of inositols have been derived from the existence of multiple stereoisomers, which may confer antioxidant properties. In the brain, fluctuation of inositols in extracellular and intracellular compartments regulates ne
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Chattopadhyay, Ansuman, та Graham Carpenter. "PLC-γ1 is required for IGF-I protection from cell death induced by loss of extracellular matrix adhesion". Journal of Cell Science 115, № 10 (2002): 2233–39. http://dx.doi.org/10.1242/jcs.115.10.2233.

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Phospholipase C-γ1, a tyrosine kinase substrate, hydrolyses phosphatidylinositol 4,5-bisphosphate to produce inositol 1,4,5-trisphosphate and diacylglycerol, which act as second messenger moleculesto mobilize intracellular calcium and activate protein kinase C, respectively. We have investigated the role of phospholipase C-γ1 in anoikis, or cell death,induced by the loss of extracellular matrix adhesion. Spontaneously immortalized mouse embryonic fibroblasts nullizygous at the Plcg1locus (Plcg1-/-), referred to as Null cells, were derived from targeted gene disruption experiments. Subsequently
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Li, Xingyao, Chunfang Gu, Sarah Hostachy, et al. "Control of XPR1-dependent cellular phosphate efflux by InsP8 is an exemplar for functionally-exclusive inositol pyrophosphate signaling." Proceedings of the National Academy of Sciences 117, no. 7 (2020): 3568–74. http://dx.doi.org/10.1073/pnas.1908830117.

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Homeostasis of cellular fluxes of inorganic phosphate (Pi) supervises its structural roles in bones and teeth, its pervasive regulation of cellular metabolism, and its functionalization of numerous organic compounds. Cellular Pi efflux is heavily reliant on Xenotropic and Polytropic Retrovirus Receptor 1 (XPR1), regulation of which is largely unknown. We demonstrate specificity of XPR1 regulation by a comparatively uncharacterized member of the inositol pyrophosphate (PP-InsP) signaling family: 1,5-bis-diphosphoinositol 2,3,4,6-tetrakisphosphate (InsP8). XPR1-mediated Pi efflux was inhibited b
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Christophe, J. "Pancreatic tumoral cell line AR42J: an amphicrine model." American Journal of Physiology-Gastrointestinal and Liver Physiology 266, no. 6 (1994): G963—G971. http://dx.doi.org/10.1152/ajpgi.1994.266.6.g963.

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AR42J cells derive from azaserine-induced malignant nodules from the rat pancreas. They differ from normal acinar cells for at least three reasons: 1) they proliferate rapidly; 2) they synthesize, store, and secrete digestive enzymes but the regulation of their exocrine function is abnormal, from the emergence of atypical receptors (e.g., cholecystokinin octapeptide type B and pituitary adenylate cyclase-activating polypeptide type I receptors) to unusual inositol phosphate metabolism and cytoskeleton disorganization; and 3) they possess an added neuroendocrine-regulated pathway characterized
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Lin, Xiaobo, Lina Ma, Chaya Gopalan, and Richard E. Ostlund. "d-chiro-Inositol is absorbed but not synthesised in rodents." British Journal of Nutrition 102, no. 10 (2009): 1426–34. http://dx.doi.org/10.1017/s0007114509990456.

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d-chiro-inositol (DCI) and pinitol (1d-3-O-methyl-chiro-inositol) are distinctive inositols reported to possess insulin-mimetic properties. DCI-containing compounds are abundant in common laboratory animal feed. By GC–MS of 6 m-HCl hydrolysates, Purina Laboratory Rodent Diet 5001 (diet 5001) contained 0·23 % total DCI by weight with most found in the lucerne and soya meal components. In contrast, only traces of l-chiro-inositol were observed. The DCI moiety was present in a water-soluble non-ionic form of which most was shown to be pinitol. To measure the absorption of dietary inositols, rats
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Turner, Benjamin L., Michael J. Papházy, Philip M. Haygarth, and Ian D. Mckelvie. "Inositol phosphates in the environment." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 357, no. 1420 (2002): 449–69. http://dx.doi.org/10.1098/rstb.2001.0837.

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The inositol phosphates are a group of organic phosphorus compounds found widely in the natural environment, but that represent the greatest gap in our understanding of the global phosphorus cycle. They exist as inositols in various states of phosphorylation (bound to between one and six phosphate groups) and isomeric forms (e.g. myo , D– chiro , scyllo , neo ), although myo –inositol hexakisphosphate is by far the most prevalent form in nature. In terrestrial environments, inositol phosphates are principally derived from plants and accumulate in soils to become the dominant class of organic p
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Loss, Omar, Chun Ting Wu, Antonella Riccio, and Adolfo Saiardi. "Modulation of inositol polyphosphate levels regulates neuronal differentiation." Molecular Biology of the Cell 24, no. 18 (2013): 2981–89. http://dx.doi.org/10.1091/mbc.e13-04-0198.

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The binding of neurotrophins to tropomyosin receptor kinase receptors initiates several signaling pathways, including the activation of phospholipase C-γ, which promotes the release of diacylglycerol and inositol 1,4,5-trisphosphate (IP3). In addition to recycling back to inositol, IP3 serves as a precursor for the synthesis of higher phosphorylated inositols, such as inositol 1,3,4,5,6-pentakisphosphate (IP5) and inositol hexakisphosphate (IP6). Previous studies on the effect of neurotrophins on inositol signaling were limited to the analysis of IP3 and its dephosphorylation products. Here we
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Guo, Dachuan, Alex Fong, Andy Lail, et al. "Simplifying Complex Microarray Data To Derive Gene Expression Profiles Which Identify Childhood Acute Lymphoblastic Leukaemia Patients at Risk of Relapse." Blood 106, no. 11 (2005): 4506. http://dx.doi.org/10.1182/blood.v106.11.4506.4506.

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Abstract The optimal treatment of patients with childhood acute lymphoblastic leukaemia (ALL) depends on establishing accurate diagnosis. Our investigations seek to strategically develop the application of microarray gene expression profiling to identify ALL patients with clinically homogenous presentations but which may respond differently to established treatment regimens. We have determined the gene expression profiles of ALL bone marrow (BM) samples taken from patients at diagnosis. Data analysis has focussed on the use of a novel and innovative statistical technology, Gene-RaVE. This seri
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Stephens, L., P. T. Hawkins, N. Carter, et al. "l-myo-inositol 1,4,5,6-tetrakisphosphate is present in both mammalian and avian cells." Biochemical Journal 249, no. 1 (1988): 271–82. http://dx.doi.org/10.1042/bj2490271.

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When myo-[3H]inositol-prelabelled primary-cultured murine bone-marrow-derived macrophages were challenged with platelet-activating factor (PAF; 200 ng/ml), there was a rapid (2.5-fold at 10 s) rise in the intracellular concentration of D-myo-[3H]inositol 1,4,5-trisphosphate, followed by a rise in myo-[3H]inositol tetrakisphosphate. myo-[3H]Inositol tetrakisphosphate fractions were isolated by high-performance anion-exchange chromatography from myo-[3H]inositol-prelabelled chick erythrocytes and primary-cultured macrophages. In both cases [3H]iditol and [3H]inositol were the only significant pr
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Balla, T., S. S. Sim, A. J. Baukal, S. G. Rhee, and K. J. Catt. "Inositol polyphosphates are not increased by overexpression of Ins(1,4,5)P3 3-kinase but show cell-cycle dependent changes in growth factor-stimulated fibroblasts." Molecular Biology of the Cell 5, no. 1 (1994): 17–27. http://dx.doi.org/10.1091/mbc.5.1.17.

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NIH 3T3 fibroblasts were stably transfected with rat brain inositol 1,4,5-trisphosphate (Ins(1,4,5)P3) 3-kinase to explore the relationship between increased production of Ins(1,3,4,5)P4 and the formation of InsP5 and InsP6. Mass measurements of InsP5 and InsP6 revealed no significant difference between kinase- and vector-transfected fibroblasts. However, such 3-kinase-transfected cells, when labeled with [3H]inositol for 48-72 h, showed lower levels of [3H]InsP5 and [3H]InsP6, as well as [3H]Ins(1,3,4,6)P4 and D/L[3H]Ins(1,4,5,6)P4, than their vector-transfected counterparts. Because Ins(1,4,
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Dissertations / Theses on the topic "Inositol derive"

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Duthu, Brigitte. "Phosphoranylation de polyols : nouvelle voie d'acces aux myo-inositol phosphates." Toulouse 3, 1988. http://www.theses.fr/1988TOU30129.

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Un procede original de syntheses de myo-inositol phosphates a ete mis au point permettant d'obtenir en une seule preparation plusieurs myo-inositol phosphates a la fois. On fait reagir le myo-inositol avec un dioxa-2,8 aza-5 phospha-1 bicyclo(3. 3. 0) octane. Quand la phosphoranylation est totale on obtient les myo-inositol mono, bis, tris, tetrakis phosphates
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Chapon, David. "Coordination des éléments 4f et 5f par des ligands cyclohexaniques polyfonctionnels : Complexes homo- et hétérotrinucléaires en solution." Université Joseph Fourier (Grenoble ; 1971-2015), 2001. http://www.theses.fr/2001GRE10133.

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L'objectif de ce travail est d'etudier les phenomenes mis en jeu dans la complexation des cations lanthanides (iii) et americium (iii) par le taci (1,3,5-triamino-1,3,5-tridesoxy-cis-inositol) ainsi que par ses derives n-fonctionnalises. La synthese de chacun des ligands, puis l'etude de la complexation des cations f trivalents en solution est presentee. Les experiences de potentiometrie montrent la formation exclusive d'un complexe m 3l 2 de type sandwich. Les constantes thermodynamiques de formation sont tres differentes avec une augmentation importante de la stabilite des complexes ln-taci
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Huppert, Jochen. "Synthese und Komplexbildung lipophiler Derivate von 1,3,5-Triamino-1,3,5-tridesoxy-cis-inosit." [S.l.] : [s.n.], 2006. http://deposit.ddb.de/cgi-bin/dokserv?idn=981968147.

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Husson, Christian. "Complexation de lanthanides trivalents par des ligands azotés et oxygénés à base cyclohexanique." Université Joseph Fourier (Grenoble), 1998. http://www.theses.fr/1998GRE10163.

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Ce travail concerne l'elaboration et l'etude de ligands (azotes et oxygenes) a base cyclohexanique, susceptibles d'effectuer la separation selective des lanthanides et des actinides trivalents. Les ligands ont ete obtenus soit par hydrogenation catalytique d'aromatiques polyfonctionnels, soit par substitution nucleophile sur des derives cyclohexaniques polysulfones. Des complexes ml et ml#2 ont ete caracterises en phase solide (par diffraction des rayons x) avec les ligands polyhydroxyles l1 (1,3,5-cyclohexane-triol) et l2 (1,2,3-cyclohexane-triol). Les complexes se forment dans des solvants o
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Welti, Gregor. "1,3,5-Triamino-1,3,5-tridesoxy-cis-inosit-Derivate für die Radioimmunotherapie und zur Behandlung der Thalassaemie /." [S.l.] : [s.n.], 1998. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=12613.

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Weber, Michael. "Alkylierte Derivate von 1,2,5-Triamino-1,3,5-trideoxy-cis-inosit und Templatsynthesen makrocyclischer Kobalt(III)-Komplexe /." Zürich, 1997. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=12071.

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Kutzky, Barbara [Verfasser], and Kaspar [Akademischer Betreuer] Hegetschweiler. "Synthese und Untersuchung von Koordinationsverbindungen des cis-Inosit und alkylierter Derivate / Barbara Kutzky. Betreuer: Kaspar Hegetschweiler." Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2009. http://d-nb.info/1050358686/34.

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MULLER, ANDRE PIERRE. "Analyse des voies de signalisation dependantes de l'ampc et des inositols phosphates dans les cellules cath. A derivees du locus coeruleus : un modele de modulation pre et postsynaptique de tolerance aux opiaces." Université Louis Pasteur (Strasbourg) (1971-2008), 1998. http://www.theses.fr/1998STR13195.

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Les cellules cath. A ont ete utilisees comme modele pour analyser les perturbations d'origine intrinseque et extrinseques auxquelles sont soumises les neurones du locus coeruleus lors de la tolerance et de la dependance aux opiaces. Nous avons montre que ces neurones possedent des recepteurs aux pacap couples positivement a l'adenylate cyclase, sollicitee par les pacap et le vip, et a la phospholipase c, sollicitee exclusivement par les pacap. Ils possedent aussi des recepteurs a l'msh, couples positivement a l'adenylate cyclase et des recepteurs aux opioides, couples negativement a l'adenylat
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Huppert, Jochen [Verfasser]. "Synthese und Komplexbildung lipophiler Derivate von 1,3,5-Triamino-1,3,5-tridesoxy-cis-inosit / von Jochen Huppert." 2006. http://d-nb.info/981968147/34.

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Bartholomä, Mark [Verfasser]. "Synthese und Komplexbildung neuartiger, multidentater Derivate von 1,3,5-Triamino-1,3,5-tridesoxy-cis-inosit / von Mark Bartholomä." 2007. http://d-nb.info/985654090/34.

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Books on the topic "Inositol derive"

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Freeman, Stanley L. Synthesis, structural, and biological activity of oligomers derived from muco-inositol and conduritol-F. 2003.

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Book chapters on the topic "Inositol derive"

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Brosnan, James M., and Dale Sanders. "Inositol Phospholipid-Derived Signals in Plant Cells." In Transport and Receptor Proteins of Plant Membranes. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3442-6_12.

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Loeb, Alex L., Nicholas J. Izzo, Randolph M. Johnson, James C. Garrison, and Michael J. Peach. "Intracellular Calcium Transients Associated with Endothelium-Derived Relaxing Factor Release May Be Mediated by Inositol-1,4,5-Trisphosphate." In Cell Calcium Metabolism. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5598-4_22.

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Conference papers on the topic "Inositol derive"

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Wickham, N. W. R., G. M. Vercellotti, H. Q. Yin, H. S. Jacob, and C. F. Moldow. "ENDOTHELIAL CELLS PRODUCE PLATELET ACTIVATING FACTOR WHICH PRIMES NEUTROPHILS TO RELEASE OXIDANT PRODUCTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642861.

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Thrombin (THR) is generated during ARDS, sepsis and DIC. We wondered whether it might augment PMN/endothelial cell (EC) interaction and hence amplify EC damage by inducing platelet activating factor (PAF) (JCI 76:2235-2246;1985). To examine further this premise and the mechanisms involved, we measured intracel1ular calcium (Ca1) in human EC (grown on glass cover slips), in a scanning spectrof1uorometer at 37°C after loading with FURA 2 (4μM). Resting Ca1 was 148±22 nM (Mean±SEM) which increased following THR 0.5u/ml to 458±160 nM at 30s, peaking after 1 min at 559±176 nM, and returning to 273±
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McNicol, A., and D. E. MacIntyre. "COMPARISON OF PLATELET PHOSPHOINOSITIDE HYDROLYSIS BY THROMBIN AND COMBINATIONS OF OTHER AGONISTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644520.

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The activation of human platelets is mediated by the generation of two distinct second messengers, inositol 1,4,5 trisphosphate (IP3) and 1,2 Diacylglycerol (DG). Both may be derived from agonist-induced phosphoinositide (PI) hydrolysis. We have examined the effects of combinations of agonists on PI hydrolysis (monitored as {32P}-phosphatidate (PA) production). Supramaximal concentrations of PAF (180 nM), 5HT (10 μM) and the thromboxane analogue EP171 (100 nM), but not ADP (1 μM), elicited a 2-6 fold increase in (32P}-PA levels. In combination the above agonists stimulated formation of {32P}-P
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