Relevant bibliographies by topics / Insect pests. Malaria Plasmodium

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Academic literature on the topic 'Insect pests. Malaria Plasmodium'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Insect pests. Malaria Plasmodium"

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Mala, M., M. Imam, and K. Hassan. "Interaction between parasite and vector for Malaria disease transmission-a review on Malaria." Progressive Agriculture 27, no. 2 (August 17, 2016): 168–74. http://dx.doi.org/10.3329/pa.v27i2.29327.

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The parasite, Plasmodium needs an insect vector (mosquito) and a vertebrate host (human) to successful malaria transmission. The parasite use the vertebrate hosts for their asexual reproduction and insect host for sexual multiplication. In order to know the mechanism of disease transmission, knowledge about the possible interactions causes by the three components, vector, parasite and host is important. The mosquito feeding behaviour greatly contributes in the rate of malaria transmission. To assist the rate of transmission of malaria, the parasite, Plasmodium completes a complex developmental stage in the mosquito. In the mosquito the parasite, passes complex developmental stages and ensuing changes into three important forms of their life cycle: ookinete, oocyst and sporozoites. This review study concludes that, the interactions among vector, parasite and host in terms of reproductive behaviour and blood-feeding behaviour helps in transmitting malaria to the vertebrate hosts mainly, human being.Progressive Agriculture 27 (2): 168-174, 2016
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Tiwari, Savitri, Nivedita Sharma, Guru Prasad Sharma, and Neelima Mishra. "Redox interactome in malaria parasite Plasmodium falciparum." Parasitology Research 120, no. 2 (January 18, 2021): 423–34. http://dx.doi.org/10.1007/s00436-021-07051-9.

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Delić, Denis, Mohamed Dkhil, Saleh Al-Quraishy, and Frank Wunderlich. "Hepatic miRNA expression reprogrammed by Plasmodium chabaudi malaria." Parasitology Research 108, no. 5 (November 18, 2010): 1111–21. http://dx.doi.org/10.1007/s00436-010-2152-z.

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Lindenthal, Christoph, Peter G. Kremsner, and Mo-Quen Klinkert. "Commonly recognised Plasmodium falciparum parasites cause cerebral malaria." Parasitology Research 91, no. 5 (November 1, 2003): 363–68. http://dx.doi.org/10.1007/s00436-003-0975-6.

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Muhaimin, Muhaimin, Yusnaidar Yusnaidar, and Hilda Amanda. "Antimalaria Activity of Macaranga Gigantea Leaves Extracts." Journal of The Indonesian Society of Integrated Chemistry 10, no. 2 (April 6, 2019): 23–32. http://dx.doi.org/10.22437/jisic.v10i2.6581.

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The utilization of traditional medicinal plants to medicate malaria caused by Plasmodium is currently more increased along with drug resistance and rising drug prices, and the side effects of using modern medicine. Additionally, Macaranga gigantea plant species have a unique ecological function such as a pioneer plant with good morphological and physiological adaptability to extreme conditions in dealing with natural stress due to pests, temperatures, and UV rays. Therefore, they have a unique biochemical system and a variety of new natural bioactive compounds produced with various activities such as antimicrobial, antioxidant and antiviral in the forest. As the results of previous study, antimalarial activity was shown on the bioactivity of methanol leaves extract of Merkubung (Macaranga gigantea). In short, this study aimed to obtain an active antimalarial fraction of Merkubung leaf (Macaranga gigantea). In this case, fractionation of methanol extract of Merkubung leaf (Macaranga gigantea) was carried out by using different organic solvents followed by an antimalarial bioactivity test using Plasmodium berghei. The results indicated that ethanol fraction of Merkubung left (Macaranga gigantea) had better antimalarial activity than others as a new candidate and supplemental source of antimalarial drugs. Keyword: Macaranga gigantea, malaria, Plasmodium berghei, fraction
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Guimarães, Lilian de Oliveira, Roseli França Simões, Carolina Romeiro Fernandes Chagas, Regiane Maria Tironi de Menezes, Fabiana Santos Silva, Eliana Ferreira Monteiro, Marcia Moreira Holcman, et al. "Assessing Diversity, Plasmodium Infection and Blood Meal Sources in Mosquitoes (Diptera: Culicidae) from a Brazilian Zoological Park with Avian Malaria Transmission." Insects 12, no. 3 (March 3, 2021): 215. http://dx.doi.org/10.3390/insects12030215.

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Avian malaria parasites are widespread parasites transmitted by Culicidae insects belonging to different genera. Even though several studies have been conducted recently, there is still a lack of information about potential vectors of Plasmodium parasites, especially in Neotropical regions. Former studies with free-living and captive animals in São Paulo Zoo showed the presence of several Plasmodium and Haemoproteus species. In 2015, a pilot study was conducted at the zoo to collect mosquitoes in order to find out (i) which species of Culicidae are present in the study area, (ii) what are their blood meal sources, and (iii) to which Plasmodium species might they be potential vectors. Mosquitoes were morphologically and molecularly identified. Blood meal source and haemosporidian DNA were identified using molecular protocols. A total of 25 Culicidae species were identified, and 6 of them were positive for Plasmodium/Haemoproteus DNA. Ten mosquito species had their source of blood meal identified, which were mainly birds, including some species that were positive for haemosporidian parasites in the former study mentioned. This study allowed us to expand the list of potential vectors of avian malaria parasites and to improve our knowledge of the evolutionary and ecological relationships between the highly diverse communities of birds, parasites, and vectors present at São Paulo Zoo.
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Collins, William E. "Plasmodium knowlesi: A Malaria Parasite of Monkeys and Humans." Annual Review of Entomology 57, no. 1 (January 7, 2012): 107–21. http://dx.doi.org/10.1146/annurev-ento-121510-133540.

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Artemov, Gleb, Vladimir Stegniy, Maria Sharakhova, and Igor Sharakhov. "The Development of Cytogenetic Maps for Malaria Mosquitoes." Insects 9, no. 3 (September 17, 2018): 121. http://dx.doi.org/10.3390/insects9030121.

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Anopheline mosquitoes are important vectors of human malaria. Next-generation sequencing opens new opportunities for studies of mosquito genomes to uncover the genetic basis of a Plasmodium transmission. Physical mapping of genome sequences to polytene chromosomes significantly improves reference assemblies. High-resolution cytogenetic maps are essential for anchoring genome sequences to chromosomes as well as for studying breakpoints of chromosome rearrangements and chromatin protein localization. Here we describe a detailed pipeline for the development of high-resolution cytogenetic maps using polytene chromosomes of malaria mosquitoes. We apply this workflow to the refinement of the cytogenetic map developed for Anopheles beklemishevi.
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Kute, Vivek B., Aruna V. Vanikar, Pankaj R. Shah, Jigar D. Shrimali, Manoj R. Gumber, Himanshu V. Patel, Pranjal R. Modi, and Hargovind L. Trivedi. "Postrenal transplant Plasmodium vivax malaria: neglected and not benign." Parasitology Research 112, no. 4 (December 13, 2012): 1791–93. http://dx.doi.org/10.1007/s00436-012-3225-y.

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Taherian Fard, Atefeh, Amna Salman, Bahram Kazemi, and Habib Bokhari. "In silico comparative genome analysis of malaria parasite Plasmodium falciparum and Plasmodium vivax chromosome 4." Parasitology Research 104, no. 6 (January 29, 2009): 1361–64. http://dx.doi.org/10.1007/s00436-009-1338-8.

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Dissertations / Theses on the topic "Insect pests. Malaria Plasmodium"

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Grech, Katrina. "The ecology and evolution of malaria : laboratory studies of Plasmodium chabaudi and its rodent and insect hosts." Thesis, Open University, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.434280.

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In this thesis I investigated selective pressures that could shape the ecology and evolution of the malaria parasite and its mosquito host. I used the rodent malaria parasite Plasmodium chabaudi, laboratory mice and the lab adapted malaria vector Anopheles stephensi. Key theory underpinning much of this work is the virulence trade-off hypothesis, where virulence (harm to host) is thought to be an unavoidable consequence of the parasites effort to optimise fitness. Implicit in this theory is that virulence is an intrinsic parasite trait, so that a virulent parasite would be relatively virulent in all host types. Using four genetically distinct P. chabaudi clones and four distinct host strains I found for the most part, this assumption to be true; the virulence phenotype of each of the four parasites was the same across the four host strains tested. I further used these genetically distinct P. chabaudi clones along with candidate malaria vaccine AMA-l to study 1) vaccine efficacy against single and mixed clone infections and 2) the potential selective effects of vaccination. I found that vaccination reduced parasite density in a strain specific manner and that diversity at loci other than the target antigen influenced vaccine efficacy. In both narve and vaccinated hosts, selection for increased virulence was observed in mixed genotype infections, but in no case did vaccination enhance this selective pressure. As Plasmodium evolution is intricately linked to vector population dynamics, I explored environmental conditions that may influence Anopheles fitness. Specifically, I investigated whether the amount of food Anopheles parents experienced influenced the success of their offspring. I found that the food level of both parents and offspring were important in Anopheles fitness. In particular, daughters from parents reared in low food conditions produced more eggs that daughters from parents reared in high food conditions. In sum, my results indicate that the within-host environment of the malaria parasite can act as a selective agent. In addition, the environmental conditions experienced by Anopheles larvae could impact vector population dynamics. Altering the within-host environment of the parasite or the environment of its vector could dramatically alter the ecology and evolution of the malaria parasite.
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Santos-Ciminera, Patricia Dantas Ciminera Patricia Dantas Santos Santos Patricia. "Molecular epidemiology of epidemic severe malaria caused by Plasmodium vivax in the state of Amazonas, Brazil /." Download the dissertation in PDF, 2005. http://www.lrc.usuhs.mil/dissertations/pdf/Santos2005.pdf.

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Bisi, Dawn C. "Engineering the mosquito symbiont Pantoea agglomerans to secrete Plasmodium inhibitory proteins." 2009. http://digital.library.duq.edu/u?/etd,109427.

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Books on the topic "Insect pests. Malaria Plasmodium"

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Integrated vector management: Controlling vectors of malaria and other insect vector borne diseases. Chichester, West Sussex, UK: Wiley-Blackwell, 2011.

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Matthews, Graham. Integrated Vector Management: Controlling Vectors of Malaria and Other Insect Vector Borne Diseases. Wiley & Sons, Limited, John, 2011.

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Matthews, Graham. Integrated Vector Management: Controlling Vectors of Malaria and Other Insect Vector Borne Diseases. Wiley & Sons, Limited, John, 2011.

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Matthews, Graham. Integrated Vector Management: Controlling Vectors of Malaria and Other Insect Vector Borne Diseases. Wiley & Sons, Limited, John, 2011.

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Ddt In Indoor Residual Spraying Human Health Aspects. World Health Organization, 2012.

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Book chapters on the topic "Insect pests. Malaria Plasmodium"

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Malcolm, C. A., D. A. Welsby, and B. B. El Sayed. "SIT for the Malaria Vector Anopheles arabiensis in Northern State, Sudan: an Historical Review of the Field Site." In Area-Wide Control of Insect Pests, 361–72. Dordrecht: Springer Netherlands, 2007. http://dx.doi.org/10.1007/978-1-4020-6059-5_34.

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Cliff, A. D., M. R. Smallman-Raynor, P. Haggett, D. F. Stroup, and S. B. Thacker. "Disease Emergence and Re-emergence Prior to 1850." In Infectious Diseases: A Geographical Analysis. Oxford University Press, 2009. http://dx.doi.org/10.1093/oso/9780199244737.003.0011.

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Infectious diseases have been evolving since the dawn of humankind. In Section 1.3, we noted some of the palaeopathological studies that have extended our knowledge of the occurrence of human infections back into pre-history, while recent genetic studies have indicated that the agents of diseases such as malaria (Plasmodium spp.) and leprosy (Mycobacterium leprae) first emerged in the human species many thousands of years ago (Carter and Mendis 2002; Monot et al. 2005). For the most part, however, our knowledge of the long history of disease emergence is based on the written record of earlier ages. In the present chapter, in so far as the historical evidence allows, we provide a brief and necessarily highly selective overview of disease emergence and cyclical re-emergence from the beginning of the written record to the mid-nineteenth century. McMichael (2004) identifies four great historical transitions in the relationship of humans and microbes that, since the initial advent of agriculture and livestock herding, have promoted the emergence and re-emergence diseases. These four transitions, each associated with a progressive increase in the geographical scale of operation (local → continental → intercontinental → global), are: (i) First historic transition (5,000–10,000 years ago). A local transition when early agrarian-based settlements brought humans into contact with sylvatic enzootic pathogens. As described under the ‘domestic-origins hypothesis’ in Section 1.3.2, close and prolonged exposure to domesticated animals and urban pests (for example, rodents and flies) resulted in the cross-species transmission of the ancestral agents of many modern-day human infectious diseases, including influenza, measles, smallpox, tuberculosis, and typhoid. (ii) Second historic transition (1,500–3,000 years ago). A continental-level transition fuelled by the military and trade contacts of early Eurasian civilizations which resulted in the cross-civilization transmission of infectious agents. In the wake of this historical transition, a trans- European ‘equilibration’ of infectious agents occurred and the diseases became endemic to the population. (iii) Third historic transition (200–500 years ago). An intercontinental transition associated with European expansion, resulting in the transoceanic spread of infectious agents.
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