Academic literature on the topic 'Inspiratory muscle work'
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Journal articles on the topic "Inspiratory muscle work"
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Wilson, Theodore A., Maurizio Angelillo, Alexandre Legrand, and André de Troyer. "Muscle kinematics for minimal work of breathing." Journal of Applied Physiology 87, no. 2 (August 1, 1999): 554–60. http://dx.doi.org/10.1152/jappl.1999.87.2.554.
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A mathematical model was analyzed to obtain a quantitative and testable representation of the long-standing hypothesis that the respiratory muscles drive the chest wall along the trajectory for which the work of breathing is minimal. The respiratory system was modeled as a linear elastic system that can be expanded either by pressure applied at the airway opening (passive inflation) or by active forces in respiratory muscles (active inflation). The work of active expansion was calculated, and the distribution of muscle forces that produces a given lung expansion with minimal work was computed. The calculated expression for muscle force is complicated, but the corresponding kinematics of muscle shortening is simple: active inspiratory muscles shorten more during active inflation than during passive inflation, and the ratio of active to passive shortening is the same for all active muscles. In addition, the ratio of the minimal work done by respiratory muscles during active inflation to work required for passive inflation is the same as the ratio of active to passive muscle shortening. The minimal-work hypothesis was tested by measurement of the passive and active shortening of the internal intercostal muscles in the parasternal region of two interspaces in five supine anesthetized dogs. Fractional changes in muscle length were measured by sonomicrometry during passive inflation, during quiet breathing, and during forceful inspiratory efforts against a closed airway. Active muscle shortening during quiet breathing was, on average, 70% greater than passive shortening, but it was only weakly correlated with passive shortening. Active shortening inferred from the data for more forceful inspiratory efforts was ∼40% greater than passive shortening and was highly correlated with passive shortening. These data support the hypothesis that, during forceful inspiratory efforts, muscle activation is coordinated so as to expand the chest wall with minimal work.
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Suzuki, S., J. Suzuki, and T. Okubo. "Expiratory muscle fatigue in normal subjects." Journal of Applied Physiology 70, no. 6 (June 1, 1991): 2632–39. http://dx.doi.org/10.1152/jappl.1991.70.6.2632.
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We examined expiratory muscle fatigue during expiratory resistive loading in 11 normal subjects. Subjects breathed against expiratory resistances at their own breathing frequency and tidal volume until exhaustion or for 60 min. Respiratory muscle strength was assessed from both the maximum static expiratory and inspiratory mouth pressures (PEmax and PImax). At the lowest resistance, PEmax and PImax measured after completion of the expiratory loaded breathing were not different from control values. With higher resistance, both PEmax and PImax were decreased (P less than 0.05), and the decrease lasted for greater than or equal to 60 min. The electromyogram high-to-low frequency power ratio for the rectus abdominis muscle decreased progressively during loading (P less than 0.01), but the integrated EMG activity did not change during recovery. Transdiaphragmatic pressure during loading was increased 3.6-fold compared with control (P less than 0.05). These findings suggest that expiratory resistive loaded breathing induces muscle fatigue in both expiratory and inspiratory muscles. Fatigue of the expiratory muscles can be attributed directly to the high work load and that of the inspiratory muscles may be related to increased work due to shortened inspiratory time.
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McConnell, Alison K., and Michelle Lomax. "The influence of inspiratory muscle work history and specific inspiratory muscle training upon human limb muscle fatigue." Journal of Physiology 577, no. 1 (November 8, 2006): 445–57. http://dx.doi.org/10.1113/jphysiol.2006.117614.
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Yan, S., P. Sliwinski, A. P. Gauthier, I. Lichros, S. Zakynthinos, and P. T. Macklem. "Effect of global inspiratory muscle fatigue on ventilatory and respiratory muscle responses to CO2." Journal of Applied Physiology 75, no. 3 (September 1, 1993): 1371–77. http://dx.doi.org/10.1152/jappl.1993.75.3.1371.
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We evaluated the effect of global inspiratory muscle fatigue on ventilation and respiratory muscle control during CO2 rebreathing in normal subjects. Fatigue was induced by breathing against a high inspiratory resistance until exhaustion. CO2 response curves were measured before and after fatigue. During CO2 rebreathing, global fatigue caused a decreased tidal volume (VT) and an increased breathing frequency but did not change minute ventilation, duty cycle, or mean inspiratory flow. Both esophageal and transdiaphragmatic pressure swings were significantly reduced after global fatigue, suggesting decreased contribution of both rib cage muscles and diaphragm to breathing. End-expiratory transpulmonary pressure for a given CO2 was lower after fatigue, indicating an additional decrease in end-expiratory lung volume due to expiratory muscle recruitment, which leads to a greater initial portion of inspiration being passive. This, combined with the reduction in VT, decreased the fraction of VT attributable to inspiratory muscle contribution; therefore the inspiratory muscle elastic work and power per breath were significantly reduced. We conclude that respiratory control mechanisms are plastic and that the respiratory centers alter their output in a manner appropriate to the contractile state of the respiratory muscles to conserve the ventilatory response to CO2.
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Collett, P. W., and L. A. Engel. "Influence of lung volume on oxygen cost of resistive breathing." Journal of Applied Physiology 61, no. 1 (July 1, 1986): 16–24. http://dx.doi.org/10.1152/jappl.1986.61.1.16.
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We examined the relationship between the O2 cost of breathing (VO2 resp) and lung volume at constant load, ventilation, work rate, and pressure-time product in five trained normal subjects breathing through an inspiratory resistance at functional residual capacity (FRC) and when lung volume (VL) was increased to 37 +/- 2% (mean +/- SE) of inspiratory capacity (high VL). High VL was maintained using continuous positive airway pressure of 9 +/- 2 cmH2O and with the subjects coached to relax during expiration to minimize respiratory muscle activity. Six paired runs were performed in each subject at constant tidal volume (0.62 +/- 0.2 liters), frequency (23 +/- 1 breaths/min), inspiratory flow rate (0.45 +/- 0.1 l/s), and inspiratory muscle pressure (45 +/- 2% of maximum static pressure at FRC). VO2 resp increased from 109 +/- 15 ml/min at FRC by 41 +/- 11% at high VL (P less than 0.05). Thus the efficiency of breathing at high VL (3.9 +/- 0.2%) was less than that at FRC (5.2 +/- 0.3%, P less than 0.01). The decrease in inspiratory muscle efficiency at high VL may be due to changes in mechanical coupling, in the pattern of recruitment of the respiratory muscles, or in the intrinsic properties of the inspiratory muscles at shorter length. When the work of breathing at high VL was normalized for the decrease in maximum inspiratory muscle pressure with VL, efficiency at high VL (5.2 +/- 0.3%) did not differ from that at FRC (P less than 0.7), suggesting that the fall in efficiency may have been related to the fall in inspiratory muscle strength. During acute hyperinflation the decreased efficiency contributes to the increased O2 cost of breathing and may contribute to the diminished inspiratory muscle endurance.
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Van Hollebeke, Marine, Diego Poddighe, Tin Gojevic, Beatrix Clerckx, Jan Muller, Greet Hermans, Rik Gosselink, and Daniel Langer. "Measurement validity of an electronic training device to assess breathing characteristics during inspiratory muscle training in patients with weaning difficulties." PLOS ONE 16, no. 8 (August 26, 2021): e0255431. http://dx.doi.org/10.1371/journal.pone.0255431.
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Inspiratory muscle training (IMT) improves respiratory muscle function and might enhance weaning outcomes in patients with weaning difficulties. An electronic inspiratory loading device provides valid, automatically processed information on breathing characteristics during IMT sessions. Adherence to and quality of IMT, as reflected by work of breathing and power generated by inspiratory muscles, are related to improvements in inspiratory muscle function in patients with chronic obstructive pulmonary disease. The aim of this study was to investigate the validity of an electronic training device to assess and provide real-time feedback on breathing characteristics during inspiratory muscle training (IMT) in patient with weaning difficulties. Patients with weaning difficulties performed daily IMT sessions against a tapered flow-resistive load of approximately 30 to 50% of the patient’s maximal inspiratory pressure. Airflow and airway pressure measurements were simultaneously collected with the training device (POWERbreatheKH2, POWERbreathe International Ltd, UK) and a portable spirometer (reference device, Pocket-Spiro USB/BT100, M.E.C, Belgium). Breath by breath analysis of 1002 breaths of 27 training sessions (n = 13) against a mean load of 46±16% of the patient’s maximal inspiratory pressure were performed. Good to excellent agreement (Intraclass correlation coefficients: 0.73–0.97) was observed for all breathing characteristics. When individual differences were plotted against mean values of breaths recorded by both devices, small average biases were observed for all breathing characteristics. To conclude, the training device provides valid assessments of breathing characteristics to quantify inspiratory muscle effort (e.g. work of breathing and peak power) during IMT in patients with weaning difficulties. Availability of valid real-time data of breathing responses provided to both the physical therapist and the patient, can be clinically usefull to optimize the training stimulus. By adapting the external load based on the visual feedback of the training device, respiratory muscle work and power generation during IMT can be maximized during the training.
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Wheatley, S. West, SJ Cala, and LA Engel. "The effect of hyperinflation on respiratory muscle work in acute induced asthma." European Respiratory Journal 3, no. 6 (June 1, 1990): 625–32. http://dx.doi.org/10.1183/09031936.93.03060625.
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To examine the relationship between end-expiratory lung volume and respiratory muscle work during acute bronchoconstriction, we measured the work of breathing in nine asthmatic subjects, in whom bronchoconstriction was induced with histamine aerosol. When the forced expiratory volume in one second (FEV1) fell below 60% of the control value, work was measured at the spontaneously hyperinflated lung volume (VLS), at a volume equivalent to the control functional residual capacity (FRC) and at a volume 30% of vital capacity (VC) above the control FRC. Hyperinflation to VLS caused a 39% decrease in the total positive work per breath from 2.8 +/- 0.4 to 1.7 +/- 0.1 J, entirely due to a decrease in expiratory work per breath from 1.6 +/- 0.4 to 0.10 +/- 0.05 J. Inspiratory work did not change at any lung volume, because the increase in inspiratory elastic work due to hyperinflation was offset by the decrease in flow resistive work. Breathing above VLS did not alter the total positive muscle work, but did increase the negative work of the inspiratory muscles from 0.4 +/- 0.1 to 0.8 +/- 0.1 J.breath. We conclude that during induced asthma spontaneous hyperinflation minimizes the total respiratory muscle work and may constitute a mechanism for minimizing energy expenditure.
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Hsia, C. C., M. Ramanathan, J. L. Pean, and R. L. Johnson. "Respiratory muscle blood flow in exercising dogs after pneumonectomy." Journal of Applied Physiology 73, no. 1 (July 1, 1992): 240–47. http://dx.doi.org/10.1152/jappl.1992.73.1.240.
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In three foxhounds after left pneumonectomy, the relationships of ventilatory work and respiratory muscle (RM) blood flow to ventilation (VE) during steady-state exercise were examined. VE was measured using a specially constructed respiratory mask and a pneumotach; work of breathing was measured by the esophageal balloon technique. Blood flow to RM was measured by the radionuclide-labeled microsphere technique. Lung compliance after pneumonectomy was 55% of that before pneumonectomy; compliance of the thorax was unchanged. O2 uptake (VO2) of RM comprised only 5% of total body VO2 at exercise. At rest, inspiratory muscles received 62% and expiratory muscles 38% of the total O2 delivered to the RM (QO2RM). During exercise, inspiratory muscles received 59% and expiratory muscles 41% of total QO2RM. Blood flow per gram of muscle to the costal diaphragm was significantly higher than that to the crural diaphragm. The diaphragm, parasternals, and posterior cricoarytenoids were the most important inspiratory muscles, and internal intercostals and external obliques were the most important expiratory muscles for exercise. Up to a VE of 120 l/min through one lung, QO2RM constituted only a small fraction of total body VO2 during exercise and maximal vasodilation in the diaphragm was never approached.
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Ramsook, Andrew H., Yannick Molgat-Seon, Michele R. Schaeffer, Sabrina S. Wilkie, Pat G. Camp, W. Darlene Reid, Lee M. Romer, and Jordan A. Guenette. "Effects of inspiratory muscle training on respiratory muscle electromyography and dyspnea during exercise in healthy men." Journal of Applied Physiology 122, no. 5 (May 1, 2017): 1267–75. http://dx.doi.org/10.1152/japplphysiol.00046.2017.
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Inspiratory muscle training (IMT) has consistently been shown to reduce exertional dyspnea in health and disease; however, the physiological mechanisms remain poorly understood. A growing body of literature suggests that dyspnea intensity can be explained largely by an awareness of increased neural respiratory drive, as measured indirectly using diaphragmatic electromyography (EMGdi). Accordingly, we sought to determine whether improvements in dyspnea following IMT can be explained by decreases in inspiratory muscle electromyography (EMG) activity. Twenty-five young, healthy, recreationally active men completed a detailed familiarization visit followed by two maximal incremental cycle exercise tests separated by 5 wk of randomly assigned pressure threshold IMT or sham control (SC) training. The IMT group ( n = 12) performed 30 inspiratory efforts twice daily against a 30-repetition maximum intensity. The SC group ( n = 13) performed a daily bout of 60 inspiratory efforts against 10% maximal inspiratory pressure (MIP), with no weekly adjustments. Dyspnea intensity was measured throughout exercise using the modified 0–10 Borg scale. Sternocleidomastoid and scalene EMG was measured using surface electrodes, whereas EMGdi was measured using a multipair esophageal electrode catheter. IMT significantly improved MIP (pre: −138 ± 45 vs. post: −160 ± 43 cmH2O, P < 0.01), whereas the SC intervention did not. Dyspnea was significantly reduced at the highest equivalent work rate (pre: 7.6 ± 2.5 vs. post: 6.8 ± 2.9 Borg units, P < 0.05), but not in the SC group, with no between-group interaction effects. There were no significant differences in respiratory muscle EMG during exercise in either group. Improvements in dyspnea intensity ratings following IMT in healthy humans cannot be explained by changes in the electrical activity of the inspiratory muscles. NEW & NOTEWORTHY Exertional dyspnea intensity is thought to reflect an increased awareness of neural respiratory drive, which is measured indirectly using diaphragmatic electromyography (EMGdi). We examined the effects of inspiratory muscle training (IMT) on dyspnea, EMGdi, and EMG of accessory inspiratory muscles. IMT significantly reduced submaximal dyspnea intensity ratings but did not change EMG of any inspiratory muscles. Improvements in exertional dyspnea following IMT may be the result of nonphysiological factors or physiological adaptations unrelated to neural respiratory drive.
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Dodd, D. S., S. Kelly, P. W. Collett, and L. A. Engel. "Pressure-time product, work rate, and endurance during resistive breathing in humans." Journal of Applied Physiology 64, no. 4 (April 1, 1988): 1397–404. http://dx.doi.org/10.1152/jappl.1988.64.4.1397.
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We examined the effect of increasing work rate, without a corresponding increase in the pressure-time product, on energy cost and inspiratory muscle endurance (Tlim) in five normal subjects during inspiratory resistive breathing. Tidal volume, mean inspiratory mouth pressure, duty cycle, and hence the pressure-time product were kept constant, whereas work rate was varied by changing the frequency of breathing. There was a linear decrease in Tlim of -2.1 ± 0.5 s.J-1.min-1 (r = 0.87 ± 0.06) with increasing work rate. The data satisfied a model of energy balance during fatiguing runs (Monod and Scherrer. Ergonomics 8: 329-337, 1965) and were consistent with the hypothesis that the rate of energy supply, or respiratory muscle blood flow, is fixed when the pressure-time product is constant. Our results indicate that during inspiratory resistive breathing against fatiguing loads, work rate determines endurance independently of the pressure-time product. On the basis of the model, our results lead to estimates of respiratory muscle blood flow and available energy stores under the conditions of our experiment.
Dissertations / Theses on the topic "Inspiratory muscle work"
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Brown, Peter Ian. "Physiological consequences of the work of breathing and of inspiratory muscle training." Thesis, Nottingham Trent University, 2009. http://irep.ntu.ac.uk/id/eprint/40/.
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A reduced blood lactate concentration ([lac-]B) is commonly observed during whole-body exercise following inspiratory muscle training (IMT). However, whether the inspiratory muscles are, in part, the source of these reductions remains unknown. Accordingly, this thesis investigated: (I) the contribution of the respiratory muscles to the systemic [lac-]B and (II) the effects of IMT upon inspiratory muscle lactate exchange and clearance. In addition, the thesis also evaluated the determinants of inspiratory muscle strength (maximal inspiratory mouth pressure; MIP). All subjects were healthy, active and free of pulmonary and respiratory muscle disease. Under resting conditions, 10 min intense volitional hyperpnoea at 85% of maximal exercise minute ventilation (VE max) increased [lac-]B by 0.96 mmol.L-1. This was attenuated by 25% following 6 wks IMT. 8 min volitional hyperpnoea at 90% VE max imposed upon exercise at the maximal lactate steady state (MLSS) increased [lac-]B by 0.99 mmol.L-1. Following 6 wk IMT, the steady state and hyperpnoea-mediated increase in [lac-]B were lower by 8 and 26%, respectively. Relative to pre-IMT, loading the trained inspiratory muscles using a low-intensity pressure threshold resistance (15 cmH2O) immediately following maximal exercise accelerated both lactate exchange and clearance capacities by ~70%. Collectively these findings support the notion that the respiratory muscles are capable of net lactate production and are the first to suggest that IMT increases their capacity for lactate clearance. This thesis also demonstrates that the respiratory muscles are responsible, in part, for the reductions observed in [lac-]B during whole-body exercise following IMT. Finally, baseline MIP was positively correlated with the strength of the chest wall inspiratory muscles. The IMT-mediated increase in MIP was negatively correlated with the relative increase in chest wall muscle strength. Therefore, these findings are the first to demonstrate that the lower the initial strength of the chest wall inspiratory muscles, the lower the MIP and the greater the improvement in global inspiratory muscle strength following IMT.
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Mills, D. E. "Respiratory muscle work and inspiratory muscle training on cytokines, oxidative stress and diaphragm fatigue in younger and older populations." Thesis, Nottingham Trent University, 2013. http://irep.ntu.ac.uk/id/eprint/286/.
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Increased respiratory muscle work is encountered during strenuous whole-body exercise, and at rest in older adults and those with pulmonary limitations such as chronic obstructive pulmonary disease (COPD). When sufficiently strenuous it can result in diaphragmatic fatigue, increased blood lactate concentrations, and an alteration in respiratory muscle recruitment patterns. Increased respiratory muscle work also elevates cytokines interleukin-6 (IL-6) and interleukin-1β (IL-1β) within the respiratory muscles and systemically. There is mounting evidence that inflammation contributes significantly to the ageing process and age related diseases. Enhanced oxidative stress, glycogen depletion and diaphragmatic fatigue are all potential stimuli for this production. Whole-body exercise training can attenuate systemic inflammation and oxidative stress in younger adults during exercise, and in older adults who experience this at rest. An attenuation of muscle glycogen or increases in antioxidant enzymes may explain such reductions. Inspiratory muscle training (IMT) may also elicit similar adaptations in the inspiratory muscles, and thus also attenuate these markers. Accordingly, this thesis evaluated in younger adults whether: (I) the respiratory muscles contribute to exercise-induced increases in plasma cytokines and/or systemic oxidative stress measured by DNA damage to peripheral blood mononuclear cells (PBMC) during 1 h of steady-state cycling exercise (EX) and volitional hyperpnoea at rest which mimicked the breathing and respiratory muscle recruitment patterns achieved during EX (HYPEX) and heavy exercise (VH); (II) an increase in these inflammatory markers was related to diaphragmatic fatigue; (III) IMT attenuates these markers during EX, HYPEX and EX; and (IV) IMT changes an estimation of the maximum lactate steady-state and respiratory muscle recruitment patterns during the lactate minimum test. This thesis also evaluated in older adults at rest whether: (V) IMT attenuates plasma cytokines and DNA damage to PBMC. It was found in younger adults that: (I) plasma IL-6 concentrations increased during EX, HYPEX and VH and plasma IL-1β increased during VH. Plasma interleukin-1 receptor antagonist concentration and oxidative DNA damage to PBMC remained unchanged during VH; (II) the increase in IL-6 and IL-1β during VH was not related to the induction of diaphragmatic fatigue; (III) following IMT, plasma IL-6 concentrations were reduced by 33% during EX, 24% during VH, but were unchanged during HYPEX; and (V) following IMT, an estimation of the maximum lactate steady-state and respiratory muscle recruitment patterns remained unchanged during the lactate minimum test. It was found in older adults that: (IV) following IMT, nine plasma cytokines and DNA damage to PBMC remained unchanged. This thesis provides novel evidence that the respiratory muscles contribute to exercise-induced increases in plasma IL-6 and IL-1β concentration and that this increase is not related to diaphragmatic fatigue. IMT attenuates plasma IL-6 concentration during exercise, but not in a range of plasma cytokines in older adults at rest. It is attractive to speculate that the respiratory muscles contribute to the systemic inflammation observed in COPD patients and IMT may reduce the dysregulated cytokine response observed during exercise of COPD patients.
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Rodriguez-Anderson, Ramón F. "The Influence of Respiratory Muscle Work on Locomotor and Respiratory Muscle Oxygenation Trends in Repeated-Sprint Exercise." Thesis, 2018. https://vuir.vu.edu.au/37831/.
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This thesis investigated the role respiratory muscle work has on locomotor and respiratory muscle oxygen (O2) utilisation during multiple sprint work. To measure O2 delivery and uptake in real time, near-infrared spectroscopy (NIRS) can be used. However, there are inconsistent methods of smoothing and determining peaks and nadirs from the NIRS signal. Therefore, the aim of study 1 was to examine the effects of different methodologies commonly used in the literature on the determination of peaks and nadirs in the vastus lateralis deoxyhaemoglobin (HHbVL) signal. Means derived from predetermined windows, irrespective of length and data smoothing, underestimated the magnitude of peak and nadir [HHbVL] compared to a rolling mean approach. Based on the results, we suggest using a digital filter to smooth NIRS data, rather than an arithmetic mean, and a rolling approach to determine peaks and nadirs for accurate interpretation of muscle oxygenation trends.
In the second study, the effects of heightened inspiratory muscle work on [HHbVL] and respiratory muscle deoxyhaemoglobin ([HHbRM]) trends were examined. In response to the heightened inspiratory muscle work, HHbRM was elevated across the sprint series. There were no clear differences in HHbVL trends between exercise conditions. The lack of difference in HHbVL between trials implies respiratory muscle O2 uptake does not limit locomotor oxygenation trends.
Study 3 investigated the role of arterial hypoxemia on respiratory muscle oxygenation trends, and its implications on locomotor oxygenation. While exercising in hypoxia (14.5% O2), HHbVL was higher during the sprint and recovery phases of the repeated-sprint protocol compared to normoxia (21% O2). There were no clear differences in respiratory muscle oxygenation trends between conditions. The clear reduction in locomotor muscle O2 delivery (inferred from HHbVL) while respiratory muscle oxygenation was maintained, suggests preferential blood flow distribution to the respiratory muscle to compensate for arterial hypoxemia, which may explain in part compromise locomotor O2 delivery.
The aim of the final study was to examine the role of respiratory muscle strength on locomotor and respiratory muscle oxygenation trends in repeated-sprint exercise. Inspiratory muscle training (IMT) was used to reduce the relative intensity of exercise hyperpnoea by strengthening the respiratory muscles. Repeat-sprint ability was again assessed in normoxia and hypoxia. After 4 weeks of training, there was a 35% increase of inspiratory muscle pressure in the IMT beyond the control group. Despite the substantial change in respiratory muscle strength, oxygenation trends were not affected in either normoxia or hypoxia.
The findings of this thesis do not support the work of breathing as being a limiting factor in locomotor muscle oxygenation in normoxia. The intermittent nature of repeated-sprint activity is likely a key mediating factor for which O2 delivery can be maintained to both the locomotor and respiratory muscles. However, under conditions of arterial hypoxemia, locomotor muscle oxygenation may be compromised by preferential O2 delivery to the respiratory muscles.
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Lin, Kun-Da, and 林昆達. "The Effects of 5 weeks Inspiratory Muscle Training on the Interval Anaerobic Capacity and Foot Work Performance in Collegiate Male Badminton Players." Thesis, 2014. http://ndltd.ncl.edu.tw/handle/28143107093291587910.
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碩士中國文化大學
體育學系運動教練碩博士班
102
Abstract Purpose: The purpose of this study is to confer the intervene of 5-week inspiratory muscle training to see if it can boost the interval anaerobic sport ability and foot work performance. Method: to select 10 collegiate male badminton players, using the breathing mouth pieces (POWERbreathe, UK). They were paired and randomly assigned to experiment group (EPG, N=5) and control group (CG, N=5). The former proceeds 5 weeks training: 7 days a week, twice a day, 30 times once by using the POWERbreathe. After 5 weeks, the latter is to take turns to performance the same training program. Modified Borg Scale of Rating Perceived Exertion (RPE) was implied to estimate effort and exertion of the subjects. Tow way ANOVA was used to compare the differences. Result: 5 weeks of inspiratory muscle training found the 2 groups have no significant difference in interval anaerobic ability. While on the foot work performance, considering the trainee’s different ability level in the beginning, the grade of each stage is referred to a progress percentage. By analyzing datum to show the progress rate of inspiratory muscle training in the foot work performance on each stage has remarkably better than control group in the same period. While on the respiratory RPE, the training effectively make less difficult of the interval anaerobic sport ability; However, the foot work performance has little progress to have no significant difference. Conclusion: The intervene of a 5-week inspiratory muscle training to collegiate male badminton players can effectively enhance the foot work performance, and make less difficult of the interval anaerobic sport ability and foot work on the RPE.
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Chuang, Fu-Yen, and 莊富延. "The effects of inspiratry muscle training on anaerobic work capacity in sprint athletes." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/10589419337439551642.
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碩士國立東華大學
體育學系
98
The primary objective was to assess the effect of inspiratory muscle training to the anaerobic work capacity in sprint athletes. Subjects were 16 males of college sprint athletes . Counter balance order measured in experimental group and placebo group. The experimental group performed 30 inspiratory efforts per day for 5 days a week against a resistance equivalent 80% maximum inspiratory mouth pressure (PImax) by threshold inspiratory muscle training for 6 week. The placebo group equivalent 20% . The 2×30-s Wingate test, rest interval of 4 min active recovery, to determine the anaerobic capacity. The values of the, spirometry, PImax, anaerobic work capacity were analyzed by mixed design two-way ANOVA ; The values of the blood lactate and respiratory-effort sensation (Rating of Perceived Exertion , RPE) were analyzed by repeated measures two-way ANOVA. The results showed: (a) Forced vital capacity [4.91±0.27(l) V.S. 4.41±0.37(l)]、forced expiratory volume in 1 second [4.20±0.34(l/min) V.S. 3.70±0.32(l/min)] and PImax [170.63±10.36(cmH2O) V.S.152.50±7.41(cmH2O)] were significantly higher in experimental group after training. (b) In the second anaerobic work peak power [902.25±45.00(W) V.S. 854.92±69.36(W)] and average power [770.05±58.65(W) V.S. 723.11±55.46 (W)] were significantly higher in experimental group after training. (c) The blood lactate was no significantly difference in any time. (d) The RPE was no significantly difference in any time. Besides inspiratory muscle training can improve spirometry, PImax, and anaerobic work capacity but blood lactate was not, we suggest that sprint athletics can training the inspiratory muscle to increase the energy resynthesis and anaerobic capacity.
Books on the topic "Inspiratory muscle work"
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Kreit, John W. Patient–Ventilator Interactions and Asynchrony. Edited by John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0011.
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Patient–Ventilator Interactions and Asynchrony describes what happens when the patient and the ventilator do not work together in an effective, coordinated manner. Effective mechanical ventilation requires the synchronized function of two pumps: The mechanical ventilator is governed by the settings chosen by the clinician; the patient’s respiratory system is controlled by groups of neurons in the brain stem. Ideally, the ventilator simply augments and amplifies the activity of the respiratory system. Asynchrony between the ventilator and the patient reduces patient comfort, increases work of breathing, predisposes to respiratory muscle fatigue, and may even impair oxygenation and ventilation. The chapter describes the causes and consequences of patient–ventilator asynchrony during ventilator triggering and the inspiratory phase of the respiratory cycle and explains how to adjust ventilator settings to improve patient comfort and reduce the work of breathing.
Book chapters on the topic "Inspiratory muscle work"
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F. Rodriguez, Ramón, Robert J. Aughey, and François Billaut. "The Respiratory System during Intermittent-Sprint Work: Respiratory Muscle Work and the Critical Distribution of Oxygen." In Respiratory Physiology. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.91207.
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In healthy individuals at rest and while performing moderate-intensity exercise, systemic blood flow is distributed to tissues relative to their metabolic oxygen demands. During sustained high-intensity exercise, competition for oxygen delivery arises between locomotor and respiratory muscles, and the heightened metabolic work of breathing, therefore, contributes to limited skeletal muscle oxygenation and contractility. Intriguingly, this does not appear to be the case for intermittent-sprint work. This chapter presents new evidence, based on inspiratory muscle mechanical loading and hypoxic gas breathing, to support that the respiratory system of healthy men is capable of accommodating the oxygen needs of both locomotor and respiratory muscles when work is interspersed with short recovery periods. Only when moderate hypoxemia is induced, substantial oxygen competition arises in favour of the respiratory muscles. These findings extend our understanding of the relationship between mechanical and metabolic limits of varied exercise modes.
Conference papers on the topic "Inspiratory muscle work"
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Neumannova, Katerina, Veronika Kuzilkova, Monika Zurková, Lia Hubackova, Tamara Michalcikova, Petr Jakubec, and Vitezslav Kolek. "Respiratory muscle training improves the work of breathing and decreases inspiratory muscle fatigue in patients after lung transplantation." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2200.
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