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Books on the topic 'Instability genetic'

Author: Grafiati

Published: 4 June 2021

Last updated: 8 February 2022

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1

Kastan, Michael B., ed. Genetic Instability and Tumorigenesis. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60505-5.

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2

Gonzalez, Madelena. Generic instability and identity in the contemporary novel. Cambridge Scholars, 2010.

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3

International Conference on Developmental Instability, Its Origins and Evolutionary Implications (1993 Tempe, Ariz.). Developmental instability, its origins and evolutionary implications: Proceedings of the International Conference on Developmental Instabilbity, It's Origins and Evolutionary Implications, Tempe, Arizona, 14-15 June 1993. Kluwer Academic, 1994.

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4

The ideology of genre: A comparative study of generic instability. Pennsylvania State University Press, 1994.

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5

Genome instability and transgenerational effects. Nova Science Publishers, 2010.

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6

Quickfall, Jenny Claire. Scindebant magni vincula parva pedes: The implications of generic instability in Ovid's 'Fasti'. University of Birmingham, 2000.

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7

European Environmental Mutagen Society. Meeting. Workshop on chromosome instability and cell cycle control: Istituto Superiore di Sanità, Rome, September 3-7, 1996 : abstract book. L'Istituto, 1996.

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8

Yosef, Shiloh, and SpringerLink (Online service), eds. The DNA Damage Response: Implications on Cancer Formation and Treatment. Springer Netherlands, 2009.

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9

1938-, Lindahl T., and Imperial Cancer Research Fund (Great Britain)., eds. Genetic instability in cancer. Cold Spring Harbor Laboratory Press, 1996.

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10

DNA Repair and Genetic Instability. HAYLE MEDICAL, 2018.

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11

DNA REPAIR, GENETIC INSTABILITY, AND CANCER. World Scientific Publishing, 2007.

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12

Wei, Qingyi, Lei Li, and David J. Chen. DNA Repair, Genetic Instability, and Cancer. WORLD SCIENTIFIC, 2007. http://dx.doi.org/10.1142/6228.

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13

DNA repair, genetic instability, and cancer. World Scientific, 2006.

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14

Qingyi, Wei, Li Lei Dr, and Chen David Dr, eds. DNA repair, genetic instability, and cancer. World Scientific, 2007.

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15

New Research on Genomic Instability. Nova Biomedical Books, 2007.

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16

1962-, Hisama Fuki M., Weissman Sherman M, and Martin George M. 1927-, eds. Chromosomal instability and aging: Basic science and clinical implications. Marcel Dekker, 2003.

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17

1946-, Oostra Ben A., ed. Trinucleotide diseases and instability. Springer, 1998.

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18

Nigg, Erich A. Genome Instability in Cancer Development. Springer, 2008.

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19

Genome Instability in Cancer Development. Springer, 2006.

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20

Weissman, Sherman M., Fuki Hisama, and George M. Martin. Chromosomal Instability and Aging: Basic Science and Clinical Implications. Informa Healthcare, 2003.

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21

Jean-Nicolas, Volff, ed. Genome and disease. Karger, 2006.

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22

Kastan, M. B. Genetic Instability and Tumorigenesis (Current Topics in Microbiology and Immunology). Springer-Verlag Telos, 1997.

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23

Walsh, Richard A. Siblings with Instability. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190607555.003.0015.

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Over the past 5 years, there has been a shift in the approach to searching for a genetic diagnosis in familial ataxic syndromes. Whereas in the past, a limited but expensive search through a selection of commercially available genes using Sanger sequencing was performed, there is now widespread availability of gene panels utilizing next-generation sequencing techniques. This is an efficient and powerful approach that may achieve a diagnosis in more than 30% of patients with a familial ataxia that remain undiagnosed. However, accurate phenotyping remains critical to allow interpretation of sequence variants of uncertain significance, to identify biomarkers that may be useful to monitor future therapies, and to assist with the identification of underlying pathophysiology.

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24

1947-, Bignold Leon P., ed. Cancer: Cell structures, carcinogens and genomic instability. Birhäuser, 2005.

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25

C, Poon Randy Y., ed. Polyploidization and cancer. Springer Science+Business Media, 2010.

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26

Mackenzie, Sally Ann. Genetic and molecular analysis of fertility restoration and instability in cytoplasmic male sterile Phaseolus vulgaris L. 1986.

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27

Genome Stability And Human Diseases. Springer, 2009.

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28

Igor, Kovalchuk, and Kovalchuk Olga MD, eds. Transgenerational genome instability. Nova Science Publishers, 2009.

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29

Michal, Polak, ed. Developmental instability: Causes and consequences. Oxford University Press, 2003.

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30

Enrico, Mihich, Hartwell Leland, and Pezcoller Symposium on Genomic Instability and Immortality in Cancer (1997 : Trento, Italy), eds. Genomic instability and immortality in cancer. Plenum Press, 1997.

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31

Markow, T. A. Developmental Instability: Its Origins and Evolutionary Implications (Contemporary Issues in Genetics and Evolution). 8th ed. Springer, 2007.

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32

Powell, Stephen David. Textual and generic instability in the Middle English Roberd of Cisyle: A study with critical edition. 1995.

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33

Soileau, Michael J., and Kelvin L. Chou. Parkinson Disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0002.

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Parkinson disease is a neurodegenerative disorder characterized clinically by tremor, rigidity, bradykinesia, and postural instability and pathologically by loss of nigrostriatal neurons and deposition of alpha-synuclein in neuronal cell bodies and neuritis. Non-motor symptoms such as psychiatric disorders, cognitive abnormalities, sleep dysfunction, autonomic dysfunction, and sensory manifestations are also common. This chapter gives a broad overview of this disorder. Sections cover pathophysiology, genetics, clinical manifestations, and disease course. The chapter also briefly discusses how to make the diagnosis, and alternative conditions that should be considered.

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34

Soleta, Amy, and Joelle Karlik. Goldenhar Syndrome. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0058.

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Goldenhar syndrome (also known as oculo-auriculo-vertebral spectrum, facio-auriculo-vertebral syndrome, and Goldenhar-Gorlin syndrome) is caused by fetal growth disturbances of the first two brachial clefts. Diagnostic criteria include eye, ear, mandibular, and/or vertebral anomalies. These patients may also have cardiac and renal malformations with varying degrees of severity. Airway management for Goldenhar patients may include difficult ventilation and intubation, which may become increasingly difficult with age. Vertebral anomalies including fused cervical vertebrae and/or cervical instability further complicate airway management. Pulmonary complications occur due to congenital malformations and scoliosis, which can lead to thoracic insufficiency syndrome. This chapter discusses genetics, presentation, and management of Goldenhar syndrome.

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35

“Reading Nara’s Diary or the Deluding Strategies of the Implied Author in The Rift by V.Y. Mudimbe” in “Generic Instability and Identity in the Contemporary Novel” . Cambridge Scholars, 2010.

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36

“Reading Nara’s Diary or the Deluding Strategies of the Implied Author in The Rift by V.Y. Mudimbe” in "Generic Instability and Identity in the Contemporary Novel". Cambridge Scholars, 2010.

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37

Middleton, Roger. The Great Depression in Europe. Edited by Nicholas Doumanis. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199695669.013.11.

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The Great Depression was a global phenomenon in origin and impact, but one typically viewed through an American lens. This chapter provides a European perspective, detailing and contextualizing European developments within that global context. The focus is not individual country experiences, which were significantly varied, but instead generic forces and factors set within an economist’s framework of the twin problem of attaining simultaneous internal and external balance in an age of marked instability. Three questions are addressed: the scale and scope of the depression and the unevenness of countries’ recoveries by the eve of the Second World War; the general causes, domestic and international, of the depression of the European economies; and the reasons why individual countries fared so differently as they sought to recover from the depression.

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38

Stone, Ken. Animating the Bible’s Animals. Edited by Danna Nolan Fewell. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199967728.013.38.

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This chapter discusses the potential relevance of interdisciplinary animal studies for biblical interpretation. The story of Jacob and his family in Genesis 25–32 is examined from the perspective of a “critical animal hermeneutics.” Three features of such a hermeneutics, characteristic of contemporary animal studies, are emphasized: (1) the constitutive importance of “companion species,” emphasized by Donna Haraway, including in Israel’s case goats and sheep; (2) the instability of the human/animal binary, emphasized by Jacques Derrida and other thinkers; and (3) ubiquitous associations between species difference and differences among humans, particularly, in the case of biblical literature, gender and ethnic differences. Each of these features is used to read the story of Jacob and several related biblical texts.

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39

Hakim, Alan J., and Rodney Grahame. Hypermobility syndromes. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0159.

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Hypermobility-related syndromes constitute a family of heritable disorders of connective tissue (HDCT) that derive from abnormalities affecting genes that encode for the connective tissue matrix proteins such as collagen, fibrillin, and tenascin. They range from such commonplace though poorly recognized conditions such as the joint hypermobility syndrome (JHS) to the better-known, if more rare, eponymous syndromes such as Marfan's syndrome (MFS) and the different types of the Ehlers-Danlos syndrome (EDS). The more common presentations are with skin pathology (bruising, scaring), joint or spinal and/or muscle pain and instability with vulnerability to injury and chronic widespread pain, cardiac valve pathologies, and in MFS and vascular EDS, arterial dilatation with the risk of dissection and rupture. JHS (widely considered synonymous with the EDS hypermobility type) is further complicated by cardiovascular autonomic dysfunction such as orthostatic intolerance, palpitations, and syncope, and the recently described and commonly encountered pangastrointestinal dysmotility. The latter can manifest as gastro-oesophageal reflux, gastroparesis, slow-transit constipation, or rectal evacuatory dysfunction with rectal intussusception. In addition, HDCT are associated with bladder and uterine problems as a consequence of pelvic floor weakness. Such multisystemic conditions need to be managed by a multidisciplinary team able to draw on medical, surgical, physical, and psychological interventions by appropriately experienced specialists and therapists. This chapter introduces the reader to the epidemiology, genetics, classification, and clinical presentation of JHS, EDS, and MFS. It also describes the key investigations required to support a diagnosis and assess complications of an HDCT, as well as the multidisciplinary approach to management.

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