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Journal articles on the topic 'Insulin; Diabetes; Hypertension'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 February 2022

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1

Axelrod, L. "Insulin, prostaglandins, and the pathogenesis of hypertension." Diabetes 40, no. 10 (1991): 1223–27. http://dx.doi.org/10.2337/diabetes.40.10.1223.

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2

Nosadini, R., P. Fioretto, R. Trevisan, and G. Crepaldi. "Insulin-Dependent Diabetes Mellitus and Hypertension." Diabetes Care 14, no. 3 (1991): 210–19. http://dx.doi.org/10.2337/diacare.14.3.210.

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3

Yudkin, J. S. "Hypertension and non-insulin dependent diabetes." BMJ 303, no. 6805 (1991): 730–32. http://dx.doi.org/10.1136/bmj.303.6805.730.

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4

Walden, R. "Hypertension and non-insulin dependent diabetes." BMJ 303, no. 6809 (1991): 1063. http://dx.doi.org/10.1136/bmj.303.6809.1063-b.

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5

Donnelly, R., and J. M. Connell. "Hypertension and non-insulin dependent diabetes." BMJ 303, no. 6810 (1991): 1134. http://dx.doi.org/10.1136/bmj.303.6810.1134-a.

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6

Crepaldi, G., A. Carraro, E. Brocco, et al. "Hypertension and non-insulin-dependent diabetes." Acta Diabetologica 32, no. 3 (1995): 203–8. http://dx.doi.org/10.1007/bf00838494.

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7

Sowers, James R. "Insulin resistance and hypertension." American Journal of Physiology-Heart and Circulatory Physiology 286, no. 5 (2004): H1597—H1602. http://dx.doi.org/10.1152/ajpheart.00026.2004.

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Diminished insulin (Ins) sensitivity is a characteristic feature of various pathological conditions such as the cardiometabolic syndrome, Type 2 diabetes, and hypertension. Persons with essential hypertension are more prone than normotensive persons to develop diabetes, and this propensity may reflect decreased ability of Ins to promote relaxation and glucose transport in vascular and skeletal muscle tissue, respectively. There are increasing data suggesting that ANG II acting through its ANG type 1 receptor inhibits the actions of Ins in vascular and skeletal muscle tissue, in part, by interf

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8

OʼHare, James A. "Insulin, insulin resistance, and hypertension." Current Opinion in Endocrinology and Diabetes 1, no. 1 (1994): 147–52. http://dx.doi.org/10.1097/00060793-199400010-00027.

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9

HSUEH, W. "Complicated hypertension patients-diabetes and insulin resistance." American Journal of Hypertension 11, no. 4 (1998): 247A. http://dx.doi.org/10.1016/s0895-7061(97)91623-x.

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10

Haffner, S. "Insulin resistance, hypertension and type 2 diabetes." American Journal of Hypertension 13, no. 6 (2000): S319. http://dx.doi.org/10.1016/s0895-7061(00)00802-5.

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11

Bell, David S. H. "Hypertension, diabetes, insulin resistance, and postprandial hyperglycemia." Drug Development Research 67, no. 7 (2006): 595–96. http://dx.doi.org/10.1002/ddr.20130.

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12

LAAKSO, M., H. SARLUND, and L. MYKKÄNEN. "Essential hypertension and insulin resistance in non-insulin-dependent diabetes." European Journal of Clinical Investigation 19, no. 6 (1989): 518–26. http://dx.doi.org/10.1111/j.1365-2362.1989.tb00269.x.

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13

Jarrett, R. J. "Hypertension in Diabetic Patients and Differences Between Insulin-Dependent Diabetes Mellitus and Non-Insulin-Dependent Diabetes Mellitus." American Journal of Kidney Diseases 13, no. 1 (1989): 14–16. http://dx.doi.org/10.1016/s0272-6386(89)80107-6.

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14

Monson, J. P., G. Koios, G. C. Toms, et al. "Relationship between Retinopathy and Glycaemic Control in Insulin-Dependent and Non-Insulin-Dependent Diabetes." Journal of the Royal Society of Medicine 79, no. 5 (1986): 274–76. http://dx.doi.org/10.1177/014107688607900506.

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The possible relationship between antecedent diabetic control, as determined by serial glycosylated haemoglobin (HbA1) measurements, and diabetic retinopathy was examined in 40 insulin-dependent and 41 non-insulin-dependent diabetics selected consecutively from our clinic population. Multiple logistic regression analysis demonstrated a significant association between mean HbA1 and prevalence of retinopathy in both groups of patients. This association was independent of duration of diabetes which was also significantly associated with retinopathy prevalence. Hypertension and smoking were not ob

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15

Kawamori, Ryuzo. "Insulin Resistance Seen in Non-Insulin Dependent Diabetes Mellitus and Hypertension." Hypertension Research 19, SupplementI (1996): S61—S64. http://dx.doi.org/10.1291/hypres.19.supplementi_s61.

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16

Barri, Yousri M., Biff F. Palmer, and Venkata S. Ram Venkata S. Ram. "Microalbuminuria, insulin resistance, diabetes, hypertension, and kidney function." Postgraduate Medicine 122, no. 6 (2010): 34–45. http://dx.doi.org/10.3810/pgm.2005.12.suppl43.230.

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17

Anderson, Julie, and Albert P. Rocchini. "Hypertension in Individuals with Insulin-Dependent Diabetes Mellitus." Pediatric Clinics of North America 40, no. 1 (1993): 93–104. http://dx.doi.org/10.1016/s0031-3955(16)38483-8.

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18

Klein, R. "The incidence of hypertension in insulin-dependent diabetes." Archives of Internal Medicine 156, no. 6 (1996): 622–27. http://dx.doi.org/10.1001/archinte.156.6.622.

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19

Klein, Ronald. "The Incidence of Hypertension in Insulin-Dependent Diabetes." Archives of Internal Medicine 156, no. 6 (1996): 622. http://dx.doi.org/10.1001/archinte.1996.00440060042005.

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20

Ko, Seung-Hyun, Wenhong Cao, and Zhenqi Liu. "Hypertension Management and Microvascular Insulin Resistance in Diabetes." Current Hypertension Reports 12, no. 4 (2010): 243–51. http://dx.doi.org/10.1007/s11906-010-0114-6.

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21

Aviv, A. "The roles of cell Ca2+, protein kinase C and the Na(+)-H+ antiport in the development of hypertension and insulin resistance." Journal of the American Society of Nephrology 3, no. 5 (1992): 1049–63. http://dx.doi.org/10.1681/asn.v351049.

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There is evidence that the cytosolic free Ca2+, protein kinase C, and the Na(+)-H+ antiport cross-communicate with one another through positive and negative feedback mechanisms, thereby maintaining cellular Ca2+ and pH homeostasis. This triumvirate may play a role in the development of insulin resistance--a common characteristic of both essential hypertension and non-insulin-dependent diabetes mellitus. Circulating cells from patients with essential hypertension and non-insulin-dependent diabetes mellitus demonstrate elevated cytosolic free Ca2+, increased protein kinase C activity, or both, a

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22

Viberti, G. C., and K. Earle. "Predisposition to essential hypertension and the development of diabetic nephropathy." Journal of the American Society of Nephrology 3, no. 4 (1992): S27. http://dx.doi.org/10.1681/asn.v34s27.

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Only a subset of insulin-dependent diabetic patients are at risk of developing nephropathy. Prospective studies of uncomplicated insulin-dependent diabetic cohorts have shown that a rise in systemic arterial pressure is a concomitant feature of the progression to early nephropathy. The development of hypertension is an integral feature of established nephropathy in diabetes, and its amelioration retards the progression of disease and may improve overall mortality. Family studies have suggested that nondiabetic parents of insulin-dependent diabetic patients with nephropathy have a greater preva

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23

Rasoulzadegan, Mohammad Hossein, Hamid Reza Soltani, Masoud Rahmanian, and Nakisa Amid. "Insulin Resistance in Patients with Essential Hypertension Using Homeostatic Model Assessment." Internal Medicine and Medical Investigation Journal 3, no. 3 (2018): 104. http://dx.doi.org/10.24200/imminv.v2i4.131.

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Introduction: Insulin resistance has a strong relationship with the incidence of type II diabetes. It also has a direct relationship with other risk factors of diabetes, which together are known as metabolic syndrome. The aim of this study was to investigate the relationship between insulin resistance and hypertension.Materials and Methods: In this historical cohort study, 90 patients were divided into three different groups: those without hypertension, those with controlled hypertension, and those with uncontrolled hypertension. Systolic and diastolic blood pressure, body mass index, and labo

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24

Grinnan, Daniel, Grant Farr, Adam Fox, and Lori Sweeney. "The Role of Hyperglycemia and Insulin Resistance in the Development and Progression of Pulmonary Arterial Hypertension." Journal of Diabetes Research 2016 (2016): 1–7. http://dx.doi.org/10.1155/2016/2481659.

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Pulmonary hypertension is a progressive disorder which often leads to right ventricular failure and death. While the existing classification system for pulmonary hypertension does not account for the impact of diabetes mellitus, evidence is emerging that suggests that diabetes is associated with pulmonary hypertension and that diabetes modifies the course of pulmonary hypertension. There is also growing radiographic, hemodynamic, biochemical, and pathologic data supporting an association between diabetes and pulmonary hypertension. More robust epidemiologic studies are needed to confirm an ass

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25

Saad, M. F., W. C. Knowler, D. J. Pettitt, R. G. Nelson, D. M. Mott, and P. H. Bennett. "Insulin and hypertension. Relationship to obesity and glucose intolerance in Pima Indians." Diabetes 39, no. 11 (1990): 1430–35. http://dx.doi.org/10.2337/diabetes.39.11.1430.

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26

Brands, Michael W., and M. Marlina Manhiani. "Sodium-retaining effect of insulin in diabetes." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 303, no. 11 (2012): R1101—R1109. http://dx.doi.org/10.1152/ajpregu.00390.2012.

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Insulin has long been hypothesized to cause sodium retention, potentially of enough magnitude to contribute to hypertension in obesity, metabolic syndrome, and Type II diabetes. There is an abundance of supportive evidence from correlational analyses in humans, acute insulin infusion studies in humans and animals, and chronic insulin infusion studies in rats. However, the absence of hypertension in human insulinoma patients, and negative results for sodium-retaining or blood pressure effects of chronic insulin infusion in a whole series of dog studies, strongly refute the insulin hypothesis. W

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27

Mohan, Gurinder, Ranjeet Kaur, Gursimran Singh Nayyar, and Parminder Singh. "To study the prevalence of insulin resistance in non-diabetes hypertensive subjects." International Journal of Advances in Medicine 4, no. 1 (2017): 92. http://dx.doi.org/10.18203/2349-3933.ijam20170035.

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Background: Hypertension is an important medical and public health problem in both developed and developing countries. It is an important risk factor for morbidity and mortality from coronary heart disease, stroke and renal disease. It is well recognized that hypertension coexists in varying degrees with conditions of obesity, insulin resistance/hyperinsulinemia and dyslipidemia, the interrelated metabolic disorders characteristic of metabolic syndrome.Methods: The study was carried out at Sri Guru Ram Das hospital, Amritsar, Punjab, India. A total of 150 patients were taken, out of which 75 w

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28

Landsberg, Lewis. "HYPERTENSION AND INSULIN RESISTANCE SYNDROME." Endocrine Practice 9, Supplement 2 (2003): 63–66. http://dx.doi.org/10.4158/ep.9.s2.63.

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29

Chen, S., C. Yuan, J. F. Schooley, F. J. Haddy, and M. B. Pamnani. "A Consistent Model of Insulin-Dependent Diabetes Mellitus Hypertension." American Journal of Hypertension 5, no. 10 (1992): 671–80. http://dx.doi.org/10.1093/ajh/5.10.671.

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30

Hachmi, L. Ben Salem, R. Bouguerra, O. Maatki, et al. "Blood hypertension and insulin therapy in type 2 diabetes." Journal of Hypertension 25, no. 12 (2007): A10. http://dx.doi.org/10.1097/01.hjh.0000298993.17516.56.

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31

Trevisan, M., O. Vaccaro, M. Laurenzi, et al. "Hypertension, non-insulin-dependent diabetes, and intracellular sodium metabolism." Hypertension 11, no. 3 (1988): 264–68. http://dx.doi.org/10.1161/01.hyp.11.3.264.

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32

CAZZO, Everton, Martinho Antonio GESTIC, Murillo Pimentel UTRINI, Ricardo Rossetto MACHADO, José Carlos PAREJA, and Elinton Adami CHAIM. "CONTROL OF HYPERTENSION AFTER ROUX-EN-Y GASTRIC BYPASS AMONG OBESE DIABETIC PATIENTS." Arquivos de Gastroenterologia 51, no. 1 (2014): 21–24. http://dx.doi.org/10.1590/s0004-28032014000100005.

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Context Hypertension is a common disorder in general practice and has a widely known association with type 2 diabetes mellitus. Low adhesion to clinical treatment may lead to poor results. Obesity surgery can bring early and relevant resolution rates of both morbidities. Objective To describe hypertension evolution after Roux-en-Y gastric bypass in patients with type 2 diabetes mellitus. Method Descriptive observational study designed as a historical cohort of 90 subjects with hypertension and diabetes who underwent Roux-en-Y gastric bypass and were evaluated before and after surgery. Results

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33

Passa, Philippe. "Hyperinsulinemia, Insulin Resistance and Essential Hypertension." Hormone Research 38, no. 1-2 (1992): 33–38. http://dx.doi.org/10.1159/000182478.

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34

KHURSHID, MUHAMMAD USMAN, and MANSOOR-UL-HASSAN ALV I. "INSULIN DEPENDENT DIABETES MELLITUS." Professional Medical Journal 16, no. 02 (2009): 178–86. http://dx.doi.org/10.29309/tpmj/2009.16.02.2892.

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A i m s & O b j e c t i v e s : To test the hypothesis that an increased plasma concentration of sialic acid, a marker of the acutephaseresponse, is related to the presence of diabetic retinopathy in type 1 diabetes mellitus or Insulin Dependant Diabetes Mellitus (IDDM).R e s e a r c h D e s i g n a n d M e t h o d s : We investigated the relationship between plasma sialic acid concentration and diabetic retinopathy in across-sectional survey of 1,369 people with type 1 diabetes. Subjects were participants in the IDDM Complications Study, which involveddiabetic centers of four different ho

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35

Barreto, Maria Nelly Sobreira de Carvalho, Eduarda Ângela Pessoa Cesse, Rodrigo Fonseca Lima, et al. "Analysis of access to hypertensive and diabetic drugs in the Family Health Strategy, State of Pernambuco, Brazil." Revista Brasileira de Epidemiologia 18, no. 2 (2015): 413–24. http://dx.doi.org/10.1590/1980-5497201500020010.

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OBJECTIVE: To evaluate the access to drugs for hypertension and diabetes and the direct cost of buying them among users of the Family Health Strategy (FHS) in the state of Pernambuco, Brazil. METHODS: Population-based, cross-sectional study of a systematic random sample of 785 patients with hypertension and 823 patients with diabetes mellitus who were registered in 208 randomly selected FHS teams in 35 municipalities of the state of Pernambuco. The selected municipalities were classified into three levels with probability proportional to municipality size (LS, large-sized; MS, medium-sized; SS

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36

Sharma, A. M., and V. T. Chetty. "Obesity, hypertension and insulin resistance." Acta Diabetologica 42, S1 (2005): s3—s8. http://dx.doi.org/10.1007/s00592-005-0175-1.

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37

Bloomgarden, Z. T. "Obesity, Hypertension, and Insulin Resistance." Diabetes Care 25, no. 11 (2002): 2088–97. http://dx.doi.org/10.2337/diacare.25.11.2088.

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38

Ferri, C., C. Bellini, G. Desideri, et al. "Relationship between insulin resistance and nonmodulating hypertension: linkage of metabolic abnormalities and cardiovascular risk." Diabetes 48, no. 8 (1999): 1623–30. http://dx.doi.org/10.2337/diabetes.48.8.1623.

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39

Meeks, Karlijn A. C., Amy R. Bentley, Adebowale A. Adeyemo, and Charles N. Rotimi. "Evolutionary forces in diabetes and hypertension pathogenesis in Africans." Human Molecular Genetics 30, R1 (2021): R110—R118. http://dx.doi.org/10.1093/hmg/ddaa238.

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Abstract Rates of type 2 diabetes (T2D) and hypertension are increasing rapidly in urbanizing sub-Saharan Africa (SSA). While lifestyle factors drive the increases in T2D and hypertension prevalence, evidence across populations shows that genetic variation, which is driven by evolutionary forces including a natural selection that shaped the human genome, also plays a role. Here we report the evidence for the effect of selection in African genomes on mechanisms underlying T2D and hypertension, including energy metabolism, adipose tissue biology, insulin action and salt retention. Selection effe

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40

Elliott, W. J. "Insulin Therapy, Hyperglycemia, and Hypertension in Type 1 Diabetes Mellitus." Yearbook of Cardiology 2009 (January 2009): 111–13. http://dx.doi.org/10.1016/s0145-4145(08)79404-8.

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41

Frontoni, Simona, Daniela Bracaglia, and Fabrizio Gigli. "Relationship between autonomic dysfunction, insulin resistance and hypertension, in diabetes." Nutrition, Metabolism and Cardiovascular Diseases 15, no. 6 (2005): 441–49. http://dx.doi.org/10.1016/j.numecd.2005.06.010.

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42

Perry, I. J., G. Wannamethee, M. Walker, A. G. Thompson, P. H. Whincup, and A. G. Shaper. "Hypertension and non-insulin-dependent diabetes mellitus: a prospective study." Journal of Hypertension 11, no. 10 (1993): 1153–54. http://dx.doi.org/10.1097/00004872-199310000-00043.

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43

Madacsy, L., A. Yasar, T. Tulassay, et al. "Relative nocturnal hypertension in children with insulin-dependent diabetes mellitus." Acta Paediatrica 83, no. 4 (1994): 414–17. http://dx.doi.org/10.1111/j.1651-2227.1994.tb18132.x.

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44

Doria, Alessandro, Tomio Onuma, Gary Gearin, M. Beatriz S. Freire, James H. Warram, and Andrzej S. Krolewski. "Angiotensinogen Polymorphism M235T, Hypertension, and Nephropathy in Insulin-Dependent Diabetes." Hypertension 27, no. 5 (1996): 1134–39. http://dx.doi.org/10.1161/01.hyp.27.5.1134.

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45

de Boer, Ian H. "Insulin Therapy, Hyperglycemia, and Hypertension in Type 1 Diabetes Mellitus." Archives of Internal Medicine 168, no. 17 (2008): 1867. http://dx.doi.org/10.1001/archinternmed.2008.2.

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46

Ismail, Nuhad, Bryan Becker, Piotr Strzelczyk, and Eberhard Ritz. "Renal disease and hypertension in non–insulin-dependent diabetes mellitus." Kidney International 55, no. 1 (1999): 1–28. http://dx.doi.org/10.1046/j.1523-1755.1999.00232.x.

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47

Yakhontov, D., E. Khilchenko, and D. Tsyrendarzhiev. "CYTOKINE ACTIVITY IN HYPERTENSION ASSOCIATED WITH INSULIN INDEPENDENT DIABETES MELLITUS." Journal of Hypertension 22, Suppl. 2 (2004): S369. http://dx.doi.org/10.1097/00004872-200406002-01294.

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48

Feher, M. D. "Hypertension in non-insulin dependent diabetes mellitus and its management." Postgraduate Medical Journal 67, no. 792 (1991): 938–46. http://dx.doi.org/10.1136/pgmj.67.792.938.

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49

Madácsy, L., A. Yasar, T. Tulassay, et al. "Relative nocturnal hypertension in children with insulin-dependent diabetes mellitus." Pediatric Nephrology 9, no. 3 (1995): 391. http://dx.doi.org/10.1007/bf02254225.

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50

Viberti, G. C. "Prevalence of hypertension in Type 1 (insulin-dependent) diabetes mellitus." Diabetologia 34, no. 1 (1991): 63–64. http://dx.doi.org/10.1007/bf00404033.

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