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Relevant bibliographies by topics / Insulin – Mechanism of action – Research

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Academic literature on the topic 'Insulin – Mechanism of action – Research'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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Journal articles on the topic "Insulin – Mechanism of action – Research"

1

Petersen, Max C., and Gerald I. Shulman. "Mechanisms of Insulin Action and Insulin Resistance." Physiological Reviews 98, no. 4 (2018): 2133–223. http://dx.doi.org/10.1152/physrev.00063.2017.

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The 1921 discovery of insulin was a Big Bang from which a vast and expanding universe of research into insulin action and resistance has issued. In the intervening century, some discoveries have matured, coalescing into solid and fertile ground for clinical application; others remain incompletely investigated and scientifically controversial. Here, we attempt to synthesize this work to guide further mechanistic investigation and to inform the development of novel therapies for type 2 diabetes (T2D). The rational development of such therapies necessitates detailed knowledge of one of the key pa

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2

Yu, Qiu-Jun, Rui Si, Ning Zhou та ін. "Insulin inhibits β-adrenergic action in ischemic/reperfused heart: a novel mechanism of insulin in cardioprotection". Apoptosis 13, № 2 (2007): 305–17. http://dx.doi.org/10.1007/s10495-007-0169-2.

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3

Laekeman, Gert. "Spathodea Campanulata: Een Antidiabetisch Geneesmiddel?" Afrika Focus 8, no. 2 (1992): 131–44. http://dx.doi.org/10.1163/2031356x-00802004.

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Decoctions of the stem back of Spathodea campanulata (SC) are currently used in Rwanda to treat insulin- and non insulin-dependent diabetics. In order to confirm and farther examine hypoglycemic activity of this plant we set up a research project with as final objective a possible standardisation of Spathodea in antidiabetic therapy in Rwanda. Up to now we were able to confirm the blood sugar lowering effect of SC in hyperglycemic mice (hyperglycemia provoked by streptozotocin = SZ). Also hyperglycemia in glucose loaded mice (glucose tolerance test = GTT) could be tempered by SC. Despite these

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4

Bonen, Arend, G. Lynis Dohm, and Luc J. C. van Loon. "Lipid metabolism, exercise and insulin action." Essays in Biochemistry 42 (November 27, 2006): 47–59. http://dx.doi.org/10.1042/bse0420047.

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Skeletal muscle constitutes 40% of body mass and takes up 80% of a glucose load. Therefore, impaired glucose removal from the circulation, such as that which occurs in obesity and type 2 diabetes, is attributable in large part to the insulin resistance in muscle. Recent research has shown that fatty acids, derived from adipose tissue, can interfere with insulin signalling in muscle. Hence, insulin-stimulated GLUT4 translocation to the cell surface is impaired, and therefore, the rate of glucose removal from the circulation into muscle is delayed. The mechanisms provoking lipid-mediated insulin

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Vitali, Valerio, Florian Horn, and Francesco Catania. "Insulin-like signaling within and beyond metazoans." Biological Chemistry 399, no. 8 (2018): 851–57. http://dx.doi.org/10.1515/hsz-2018-0135.

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Abstract Insulin signaling is pivotal in controlling animals’ lifespan and responses to environmental changes and, when altered, it may lead to pathogenic states. Despite its importance and relevance for biomedical research, insulin’s mechanism of action and the full range of its pathophysiological effects remain incompletely understood. Likewise, the evolutionary origin of insulin and its associated signaling components are unclear. Notwithstanding the common view that insulin signaling originated within metazoans, experimental evidence from non-metazoans suggest a more widespread distributio

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6

Blyth, Andrew J., Nicholas S. Kirk, and Briony E. Forbes. "Understanding IGF-II Action through Insights into Receptor Binding and Activation." Cells 9, no. 10 (2020): 2276. http://dx.doi.org/10.3390/cells9102276.

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The insulin-like growth factor (IGF) system regulates metabolic and mitogenic signaling through an intricate network of related receptors and hormones. IGF-II is one of several hormones within this system that primarily regulates mitogenic functions and is especially important during fetal growth and development. IGF-II is also found to be overexpressed in several cancer types, promoting growth and survival. It is also unique in the IGF system as it acts through both IGF-1R and insulin receptor isoform A (IR-A). Despite this, IGF-II is the least investigated ligand of the IGF system. This revi

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7

Cardoso, Naiane Silva, Julia Rosental de Souza Cruz, Ramon Alves de Oliveira Paula, Stella Maris da Silveira Duarte, Maria Rita Rodrigues, and Fernanda Borges de Araújo Paula. "Unsaturated fatty acid as functional food for the treatment of Diabetes mellitus type 2." Research, Society and Development 10, no. 9 (2021): e41410917231. http://dx.doi.org/10.33448/rsd-v10i9.17231.

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Several studies have been demonstrating to a relationship between unsaturated fatty acids, improvement of pancreatic function and insulin secretion. In this context, this review presents the most recent findings on the pathophysiology of type 2 diabetes mellitus, the action mechanism of unsaturated fatty acids on pancreatic function, and clinical studies in diabetic patients. We evaluated here articles from MedLine/PubMed and the Science direct database, published between 2014 and 2020. Of the 637 results, 13 were selected. From their analysis, we could observe that mono and polyunsaturated fa

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Plum, L., F. T. Wunderlich, S. Baudler, W. Krone, and J. C. Brüning. "Transgenic and Knockout Mice in Diabetes Research: Novel Insights into Pathophysiology, Limitations, and Perspectives." Physiology 20, no. 3 (2005): 152–61. http://dx.doi.org/10.1152/physiol.00049.2004.

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Insulin resistance and type 2 diabetes are serious public health threats. Although enormous research efforts have been focused on the pathogenesis of these diseases, the underlying mechanisms remain only partly understood. Here we review mouse phenotypes resulting from inactivation of molecules responsible for the control of glucose metabolism that have led to novel insights into insulin action and the development of insulin resistance. In addition, more sophisticated strategies to manipulate genes in mice in the future are presented.

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Dimova, Rumyana, and Tsvetalina Tankova. "The Role of Vaspin in the Development of Metabolic and Glucose Tolerance Disorders and Atherosclerosis." BioMed Research International 2015 (2015): 1–7. http://dx.doi.org/10.1155/2015/823481.

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In recent years, most research efforts have been focused on studying insulin-sensitizing adipokines. One of the most recently discovered adipokines is vaspin, a visceral adipose tissue-derived serine protease inhibitor. Vaspin levels have been found significantly increased in mice with obesity and insulin resistance. It has been assumed that vaspin serves as an insulin sensitizer with anti-inflammatory effects and might act as a compensatory mechanism in response to decreased insulin sensitivity. Most studies in humans have shown a positive correlation between vaspin gene expression and serum

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Gatford, K. L. "059. POOR GROWTH BEFORE BIRTH IMPAIRS INSULIN SECRETION - WHAT WE HAVE LEARNT ABOUT THE MECHANISMS FROM THE PLACENTALLY-RESTRICTED SHEEP." Reproduction, Fertility and Development 21, no. 9 (2009): 14. http://dx.doi.org/10.1071/srb09abs059.

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Diabetes occurs when insulin secretion fails to increase sufficiently to compensate for developing insulin resistance. This implies that the increased risk of diabetes in adults who were small at birth reflects impaired insulin secretion as well as their well-known insulin resistance. More recently, direct evidence has been obtained that adults and children who were growth-restricted before birth secrete less insulin than they should, given their level of insulin resistance. Our research group is using the placentally-restricted (PR) sheep to investigate the mechanisms underlying impaired insu

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Dissertations / Theses on the topic "Insulin – Mechanism of action – Research"

1

Carr, Kelley. "The Mechanism of the Prolonged Action of the Single-Chain Insulin, 70-01." Case Western Reserve University School of Graduate Studies / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1511982660839103.

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2

Hall, David A. 1968. "Insulin-mimetic peroxovanadium compounds : synthesis, reactivity,and mechanism of action." Thesis, McGill University, 1996. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=40355.

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The insulin mimetic bisperoxovanadium (bpV) complexes $ rm K sb2 lbrack VO(O sb2) sb2(OHpic) rbrack (OHpic = 3$-hydroxypicolinato) and $ rm K sb3 lbrack VO(O sb2) sb2(L$-$ rm L sp prime ) rbrack $ have been prepared and characterized, where L-$ rm L sp prime$ is a pyridinedicarboxylate (2,3-pdc, 2,4-pdc, and 2,5-pdc) or 3-acetatoxypicolinato (acetpic). The single crystal X-ray structures of three of the compounds have been determined: bpV(OHpic), bpV(2,4-pdc) and bpV(acetpic). These complexes, with the addition of one other previously reported unsubstituted bpV(pic) complex, comprise two serie

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3

Patel, Payal R. "A Role for CEACAM2 in Insulin Homeostasis and Action." University of Toledo Health Science Campus / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=mco1279320879.

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4

Gray, Alison Mary. "Studies on the efficacy and mechanism of action of traditional plant treatments for diabetes mellitus." Thesis, University of Ulster, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.390120.

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Ghanem, Simona S. "A Role for CEACAM2 protein in Insulin Secretion, Clearance and Action." University of Toledo Health Science Campus / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=mco1491497714470047.

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Sampson, Louise E. "Investigations into the mechanism of action of tumour necrosis factor." Thesis, Open University, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.304660.

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7

Cadagan, David. "The molecular mechanism of insulin action in human theca and adipocyte cells in polycycstic ovarian syndrome." Thesis, University of Nottingham, 2013. http://eprints.nottingham.ac.uk/28112/.

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PCOS is one of the leading causes of infertility worldwide affecting 1 in 10 women of a reproductive age. One of the fundamental abnormalities in women with PCOS can be seen within hormonal irregularities, which may include hyperandrogenemia hyperinsulinemia and hyper secretion of luteinising hormone (LH); and it is hypothesised that a defect in steroid secreting ovarian theca cells is involved due to their contribution in non-PCOS hormonal synthesis. Hyperinsulinemia has been associated with hyper-androgenemia through in vitro studies of cultured PCOS theca, where it has been suggested that i

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Racoma, Ira Oliva. "Targeting Glioblastoma Survival Signaling: A Novel Mechanism of Action of Thymoquinone." The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1369923513.

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9

Wright, David C. "The role of PLC, cPKC, L-type calcium channels and CAMKII in insulin stimulated glucose transport in skeletal muscle." Virtual Press, 2002. http://liblink.bsu.edu/uhtbin/catkey/1233206.

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10

Al-Share, Qusai Y. "Reduction of Hepatic CEACAM1 Levels: an Early Mechanism of Insulin Resistance Induced by High-Fat Diet." University of Toledo Health Science Campus / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=mco1201787222.

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Books on the topic "Insulin – Mechanism of action – Research"

1

International Symposium on Molecular and Cellular Biology of Insulin and IGFs (3rd 1990 Gainesville, Fla.). Molecular biology and physiology of insulin and insulin-like growth factors. Plenum Press, 1991.

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2

Ross Conference on Medical Research (11th 1991 Ponte Vedra Beach, Fla.). Cytokines in critical illness: Report of the 11th Ross Conference on Medical Research. Edited by Roche Alex F. 1921- and Gussler Judith D. Ross Laboratories, 1992.

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3

Sven-Erik, Dahlén, ed. Leukotrienes as mediators of asthma and inflammation: Basic and applied research, second International Symposium on Trends in Eicosanoid Biology, Interlaken, Switzerland. Raven Press, 1994.

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4

R, Spriggs Dale, and Koff Wayne C, eds. Topics in vaccine adjuvant research. CRC Press, 1991.

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5

Mechanism of action of TENS. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199673278.003.0009.

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There has been an abundance of research using animal, healthy human, and patients on the mechanism of action of TENS. Activity in peripheral nerves generated during TENS will influence physiological actions at peripheral, spinal, and supraspinal sites of the nervous system. Some of these physiological actions are mediated by alterations in the concentrations of neurochemicals dependent on the electrical characteristics of TENS. The purpose of this chapter is to discuss research evidence that has contributed to our knowledge about the physiological and pharmacological actions of TENS, including

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6

T, Babcock Gerald, and Naturvetenskapliga forskningsrådet (Sweden), eds. Biochemistry, especially molecular mechanisms: Report to the Research Council. Swedish Natural Science Research Council, 1989.

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7

(Editor), Alfred N. Fonteh, and Robert L. Wykle (Editor), eds. Arachidonate Remodeling and Inflammation (Progress in Inflammation Research). Birkhäuser Basel, 2004.

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8

Attanasio, A. 10th International Symposium on Endocrinology and Development: Growth and Metabolism : Obesity, Insulin Action and Use of Anthropometry (Hormone Research). S Karger Pub, 1993.

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9

Kunz, Barbara, and Kevin Kunz. Medical Applications of Reflexology : Findings in Research about Safety, Efficacy, Mechanism of Action and Cost-Effectivness of Reflexology. Reflexology Research Project, 1999.

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10

(Editor), Derek LeRoith, and Mohan K. Raizada (Editor), eds. Molecular Biology and Physiology of Insulin and Insulin-Like Growth Factors (Advances in Experimental Medicine and Biology). Springer, 1991.

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Book chapters on the topic "Insulin – Mechanism of action – Research"

1

White, Morris F. "Mechanism of Insulin Action." In Textbook of Diabetes. John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118924853.ch8.

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2

Srivastava, Ashok K., and Sanjay K. Pandey. "Potential mechanism (S) involved in the regulation of glycogen synthesis by insulin." In Insulin Action. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5647-3_14.

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3

De Meyts, Pierre, Waseem Sajid, Jane Palsgaard, et al. "Insulin and IGF-I Receptor Structure and Binding Mechanism." In Mechanisms of Insulin Action. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-72204-7_1.

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4

Cohen, Philip, Peter J. Parker, and James R. Woodgett. "The Molecular Mechanism by Which Insulin Activates Glycogen Synthase in Mammalian Skeletal Muscle." In Molecular Basis of Insulin Action. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4874-0_13.

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5

Posner, B. I., C. R. Yang, and A. Shaver. "Mechanism of Insulin Mimetic Action of Peroxovanadium Compounds." In ACS Symposium Series. American Chemical Society, 1998. http://dx.doi.org/10.1021/bk-1998-0711.ch025.

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6

Nolan, John J., and Jerrold M. Olefsky. "Insulin Action and Insulin Resistance in Noninsulin-Dependent Diabetes Mellitus." In Clinical Research in Diabetes and Obesity. Humana Press, 1997. http://dx.doi.org/10.1007/978-1-4757-3906-0_8.

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7

Krentz, Andrew J., Lutz Heinemann, and Marcus Hompesch. "Methods for Quantifying Insulin Sensitivity and Determining Insulin Time-Action Profiles." In Translational Research Methods for Diabetes, Obesity and Cardiometabolic Drug Development. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-4920-0_1.

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8

Krentz, Andrew J., Christian Weyer, and Marcus Hompesch. "Quantification of Insulin Action in Human Subjects." In Translational Research Methods in Diabetes, Obesity, and Nonalcoholic Fatty Liver Disease. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11748-1_1.

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9

Streusand, V. J., and R. J. Eilers. "Mechanism of Action of the Herbicidal Compound LY181977." In Current Research in Photosynthesis. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0511-5_138.

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Vane, J. R. "The Mechanism of Action of Anti-Inflammatory Drugs." In Advances in Eicosanoid Research. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-662-04047-8_1.

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Conference papers on the topic "Insulin – Mechanism of action – Research"

1

Xiaoping, Liu, and Qi Liangqun. "Research on Enterprise's Dynamic Capabilities Action Mechanism." In 2011 International Conference on Information Management, Innovation Management and Industrial Engineering (ICIII). IEEE, 2011. http://dx.doi.org/10.1109/iciii.2011.415.

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2

Jádna Silva Frederico, Marisa, Paola Miranda Sulis, Alessandra Mascarello, et al. "Mechanism of action of derivative sulfonamide in glucose tolerance and in incretin and insulin secretion." In Simpósio de Bioquímica e Biotecnologia. Galoa, 2017. http://dx.doi.org/10.17648/simbbtec-2017-80941.

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Qiang Ma. "The mechanism of action and channelizing research of online sporting news commentary." In 2012 International Conference on Computer Science and Information Processing (CSIP). IEEE, 2012. http://dx.doi.org/10.1109/csip.2012.6309076.

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Chan, Andrew T. "Abstract CN05-02: Exploiting aspirin's mechanism of action for combination chemoprevention." In Abstracts: Thirteenth Annual AACR International Conference on Frontiers in Cancer Prevention Research; September 27 - October 1, 2014; New Orleans, LA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1940-6215.prev-14-cn05-02.

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Zhu, Yuan, and Xiaolong Xue. "Research on the Mechanism of Action of Transactive Memory System in Engineering Project Teams." In 2014 International Conference on Construction and Real Estate Management. American Society of Civil Engineers, 2014. http://dx.doi.org/10.1061/9780784413777.089.

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Ahmed, Sumaya, and Nasser Rizk. "The Expression of Bile Acid Receptor TGR5 in Adipose Tissue in Diet-Induced Obese Mice." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0212.

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Bile acids are significant physiological factors for digestion, solubilization, absorption, toxic metabolites and xenobiotics. In addition, bile acids are responsible of signal transduction as well as metabolic regulation that activate several receptors such as farnesoid X receptor (FXR) and the membrane G-protein receptor 5 (TGR5). Activation of TGR5 by bile acids is associated with prevention of obesity as well as ameliorating the resistance to insulin via increasing energy expenditure. The objective of this research is to investigate TGR5 gene expression level in different fat depots includ

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Xiao, Zhixiong, and Yuanjian Qin. "Research on the Action Mechanism of "Knowledge Catfish" in Team Knowledge Absorption of Software Outsourcing Companies." In 2010 International Conference on Information Management, Innovation Management and Industrial Engineering (ICIII). IEEE, 2010. http://dx.doi.org/10.1109/iciii.2010.281.

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Chen, Qiang, and Xiaodong Jia. "Research on the Factors Influencing Labor Dispute and its Mechanism of Action in an Open Condition." In 2015 International Conference on Economics, Management, Law and Education. Atlantis Press, 2015. http://dx.doi.org/10.2991/emle-15.2015.69.

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Moore, Zachary, Gaurab Chakrabarti, Costas Lyssiotis, Ralph Deberardinis, and Boothman A. David. "Abstract B47: Modulating the NQO1-dependent ‘kiss of death’ mechanism of action of NQO1 bioactivatable drugs." In Abstracts: AACR Special Conference on Pancreatic Cancer: Innovations in Research and Treatment; May 18-21, 2014; New Orleans, LA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.panca2014-b47.

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Tuo, Zhong, Hai-bin Cong, and Chang-chun Gao. "A Research on the Mechanism of Action of the Knowledge Management to the Core Competitiveness in Publishing Enterprises." In 2011 International Conference on Management and Service Science (MASS 2011). IEEE, 2011. http://dx.doi.org/10.1109/icmss.2011.5998944.

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Reports on the topic "Insulin – Mechanism of action – Research"

1

Harris, Gregory, Brooke Hatchell, Davelin Woodard, and Dwayne Accardo. Intraoperative Dexmedetomidine for Reduction of Postoperative Delirium in the Elderly: A Scoping Review. University of Tennessee Health Science Center, 2021. http://dx.doi.org/10.21007/con.dnp.2021.0010.

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Background/Purpose: Post-operative delirium leads to significant morbidity in elderly patients, yet there is no regimen to prevent POD. Opioid use in the elderly surgical population is of the most significant risk factors for developing POD. The purpose of this scoping review is to recognize that Dexmedetomidine mitigates cognitive dysfunction secondary to acute pain and the use of narcotic analgesia by decreasing the amount of norepinephrine (an excitatory neurotransmitter) released during times of stress. This mechanism of action also provides analgesia through decreased perception and modul

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