Academic literature on the topic 'Insulin resistance'

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Journal articles on the topic "Insulin resistance"

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Kumar, Sinha Ritesh, and Chandra Satish. "Insulin resistance." Asian Pacific Journal of Health Sciences, Supplimentary 2014 (2014): 71–78. http://dx.doi.org/10.21276/apjhs.2014.1.1s.15.

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Demir, Şevin, and Çelik Banu Arslan. "Ability of Metabolic Score for Insulin Resistance to Detect Insulin Resistance." Chronicles of Precision Medical Researchers 3, no. 3 (2022): 187–92. https://doi.org/10.5281/zenodo.7195874.

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Aim: To evaluate the usability of metabolic score for insulin resistance (METS-IR), a novel insulin resistance index, in our country and to determine the optimal cut-off value of this index for detecting insulin resistance. Material and Method: One thousand five hundred sixty seven individuals who participated in our check-up program between 2020 and 2021 were retrospectively evaluated with the patient files for inclusion in the study. Insulin resistance was accepted when HOMA-IR≥2.7. Subjects were divided into 4 quartiles according to their METS-IR levels. Receiver-operating characteristic
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Sowers, James R., and Edward D. Frohlich. "Insulin and insulin resistance:." Medical Clinics of North America 88, no. 1 (2004): 63–82. http://dx.doi.org/10.1016/s0025-7125(03)00128-7.

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Jeffery, Alison. "Insulin resistance." Nursing Standard 17, no. 32 (2003): 47–53. http://dx.doi.org/10.7748/ns2003.04.17.32.47.c3381.

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Tomono, Syouichi. "Insulin Resistance :." Kitakanto Medical Journal 62, no. 1 (2012): 73–74. http://dx.doi.org/10.2974/kmj.62.73.

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Jeffery, Alison. "Insulin resistance." Nursing Standard 17, no. 32 (2003): 47–55. http://dx.doi.org/10.7748/ns.17.32.47.s62.

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Park, Kyong Soo. "Insulin Resistance." Journal of the Korean Medical Association 44, no. 3 (2001): 302. http://dx.doi.org/10.5124/jkma.2001.44.3.302.

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Bell, David S. H. "Insulin resistance." Postgraduate Medicine 93, no. 7 (1993): 99–107. http://dx.doi.org/10.1080/00325481.1993.11701704.

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Carroll, P. B., and R. C. Eastman. "Insulin Resistance." Endocrinologist 1, no. 2 (1991): 89–97. http://dx.doi.org/10.1097/00019616-199104000-00005.

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DeFronzo, Ralph A. "Insulin resistance." Current Opinion in Cardiology 5, no. 5 (1990): 592–98. http://dx.doi.org/10.1097/00001573-199010000-00003.

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Dissertations / Theses on the topic "Insulin resistance"

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Walker, Adrian Bernard. "The effect of insulin on resistance artery function in insulin-resistant states." Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312450.

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Maffeis, Laura <1981&gt. "Correlation between insulin resistance and treatment-resistant acne." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2013. http://amsdottorato.unibo.it/5654/1/maffeis_laura_tesi.pdf.

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Physiologically during puberty and adolescence, when juvenile acne usually appears, the response to a glucose load is increased if compared to the one observed in adult and at pre-pubertal age, while insulin sensitivity is reduced. Insulin is a hormone that acts at different levels along the axis which controls the sex hormones. It increases the release of LH and FSH by pituitary gland, stimulates the synthesis of androgens in the gonads and stimulates the synthesis of androgenic precursors in adrenal glands. Finally, it acts in the liver by inhibiting the synthesis of Sex Hormone Binding Glob
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Maffeis, Laura <1981&gt. "Correlation between insulin resistance and treatment-resistant acne." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2013. http://amsdottorato.unibo.it/5654/.

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Physiologically during puberty and adolescence, when juvenile acne usually appears, the response to a glucose load is increased if compared to the one observed in adult and at pre-pubertal age, while insulin sensitivity is reduced. Insulin is a hormone that acts at different levels along the axis which controls the sex hormones. It increases the release of LH and FSH by pituitary gland, stimulates the synthesis of androgens in the gonads and stimulates the synthesis of androgenic precursors in adrenal glands. Finally, it acts in the liver by inhibiting the synthesis of Sex Hormone Binding Glob
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Collison, Mary Williamson. "Insulin signalling in insulin resistance and cardiovascular disease syndromes." Thesis, University of Glasgow, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.366184.

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Svanfeldt, Monika. "Perioperative nutrition and insulin resistance /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-637-9/.

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Isaksson, Bengt. "Insulin resistance in pancreatic cancer /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-493-3/.

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Berends, Lindsey Matara. "Developmental programming of insulin resistance." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648433.

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Ali, Salmin. "GLUT 4 and Insulin Resistance." Wright State University / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=wright1409746939.

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Endre, Tomas. "The hypertension-prone man a study on the pathogenesis of hypertension with regard to insulin sensitivity /." Lund : Dept. of Medicine, Lund University, University Hospital MAS, 1997. http://books.google.com/books?id=3UlsAAAAMAAJ.

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Stuart, Charles A., Mary E. A. Howell, Brian M. Cartwright, et al. "Insulin Resistance and Muscle Insulin Receptor Substrate-1 Serine Hyperphosphorylation." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/etsu-works/4117.

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Insulin resistance in metabolic syndrome subjects is profound in spite of muscle insulin receptor and insulin-responsive glucose transporter (GLUT4) expression being nearly normal. Insulin receptor tyrosine kinase phosphorylation of insulin receptor substrate-1 (IRS-1) at Tyr896 is a necessary step in insulin stimulation of translocation of GLUT4 to the cell surface. Serine phosphorylation of IRS-1 by some kinases diminishes insulin action in mice. We evaluated the phosphorylation status of muscle IRS-1 in 33 subjects with the metabolic syndrome and seventeen lean controls. Each underwent eugl
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Books on the topic "Insulin resistance"

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Eleazar, Shafrir, and Hansen Barbara C, eds. Insulin resistance and insulin resistance syndrome. Taylor & Francis, 2002.

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Zeitler, Philip S., and Kristen J. Nadeau, eds. Insulin Resistance. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-25057-7.

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Reaven, Gerald M., and Ami Laws, eds. Insulin Resistance. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-716-1.

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Zeitler, Philip Scott, and Kristen J. Nadeau, eds. Insulin Resistance. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-192-5.

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Kumar, Sudhesh, and Stephen O'Rahilly, eds. Insulin Resistance. John Wiley & Sons, Ltd, 2004. http://dx.doi.org/10.1002/0470011327.

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Krentz, Andrew J., ed. Insulin Resistance. Blackwell Science Ltd, 2002. http://dx.doi.org/10.1002/9780470698921.

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David, Moller, ed. Insulin resistance. Wiley, 1993.

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Sudhesh, Kumar, and O'Rahilly S, eds. Insulin resistance: Insulin action and its disturbances in disease. J. Wiley, 2005.

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B, Yao E., ed. Insulin resistance: New research. Nova Science, 2009.

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Fantus, I. George, ed. Insulin Resistance and Cancer. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-9911-5.

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Book chapters on the topic "Insulin resistance"

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Siddle, Ken. "The Insulin Receptor and Downstream Signalling." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch1.

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Clarke, Daniel K., and Vidya Mohamed-Ali. "Adipokines and Insulin Resistance." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch10.

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Krebs, Jeremy, and Susan Jebb. "Dietary Factors and Insulin Resistance." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch11.

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Wareham, Nicholas J., Søren Brage, Paul W. Franks, and Rebecca A. Abbott. "Physical Activity and Insulin Resistance." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch12.

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Argyropoulos, George, Steven Smith, and Claude Bouchard. "Genetics of the Metabolic Syndrome." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch13.

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Lamarche, Benoît, and Jean-François Mauger. "Insulin Resistance and Dyslipidaemia." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch14.

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Cleland, Stephen J., and John M. C. Connell. "Insulin Resistance, Hypertension and Endothelial Dysfunction." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch15.

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Potau, Neus. "Insulin Resistance and Polycystic Ovary Syndrome." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch16.

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Savage, David, and Stephen O'Rahilly. "Syndromes of Severe Insulin Resistance (SSIRs)." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch17.

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Randeva, Harpal S., Margaret Clarke, and Sudhesh Kumar. "Therapeutic Strategies for Insulin Resistance." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch18.

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Conference papers on the topic "Insulin resistance"

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Qu, Kaige, Fan Yang, Xingyue Wang, Rui Wang, Dayou Li, and Zuobin Wang. "The Role of Exercise Intervention on Insulin Resistance." In 2024 IEEE International Conference on Manipulation, Manufacturing and Measurement on the Nanoscale (3M-NANO). IEEE, 2024. https://doi.org/10.1109/3m-nano61605.2024.10769676.

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Goodwin, P. "Obesity, Insulin Resistance and Insulin." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-ms2-1.

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Магомедова, И. М. "Insulin resistance in thyropathies." In V ВСЕРОССИЙСКАЯ НАУЧНО-ПРАКТИЧЕСКАЯ КОНФЕРЕНЦИЯ "ЦИФРОВОЕ ОБЩЕСТВО: НАУЧНЫЕ ИНИЦИАТИВЫ И НОВЫЕ ВЫЗОВЫ". Crossref, 2024. http://dx.doi.org/10.26118/6504.2024.87.84.004.

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Al-Jaber, Hend Sultan, Layla Jadea Al-Mansoori, and Mohamed Aghar Elrayess. "The Role of GATA3 in Adipogenesis & Insulin Resistance." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0143.

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Background: Impaired adipogenesis plays an important role in the development of obesityassociated insulin resistance and type 2 diabetes. Adipose tissue inflammation is a crucial mediator of this process. In hyperglycemia, immune system is activated partially through upregulation of GATA3, causing exacerbation of the inflammatory state associated with obesity. GATA3 also plays a role as a gatekeeper of terminal adipocyte differentiation. Here we are examining the impact of GATA3 inhibition in adipose tissue on restoring adipogenesis, reversing insulin resistance and potentially lowering the ri
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Altuve, Miguel, Erika Severeyn, and Sara Wong. "Unsupervised subjects classification using insulin and glucose data for insulin resistance assessment." In 2015 20th Symposium on Signal Processing, Images and Computer Vision (STSIVA). IEEE, 2015. http://dx.doi.org/10.1109/stsiva.2015.7330444.

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Christopher, Pretty,. "Corticosteroids and Insulin Resistance in the ICU." In Modeling and Control in Biomedical Systems, edited by Rees, Stephen, chair Andreassen, Steen and Andreassen, Steen. Elsevier, 2009. http://dx.doi.org/10.3182/20090812-3-dk-2006.00005.

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"Genetic Variations in Women with Insulin Resistance." In International Institute of Chemical, Biological & Environmental Engineering. International Institute of Chemical, Biological & Environmental Engineering, 2015. http://dx.doi.org/10.15242/iicbe.c0615079.

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Aldali, Sara Haitham, and Sownd Sankaralingam. "Induction of Glyoxalase 1 to prevent Methylglyoxal-Induced Insulin Resistance in Cardiomyocytes." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0230.

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Background: Type 2 Diabetes mellitus is characterized by hyperglycemia and insulin resistance. Methylglyoxal (MG) a highly reactive dicarbonyl compound is also increased in diabetes. MG is detoxified by glyoxalase 1 (Glo-1) enzyme using reduced glutathione (GSH) as a co-factor. MG has been shown to have deleterious effects on cardiovascular cells and impairs insulin signaling. Insulin resistance is associated with diabetic cardiomyopathy. Trans-resveratrol (tRES) and Hesperetin (HES) combination has been shown to increase Glo-1 and improve insulin signaling in obese patients. Aim(s): The aim o
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Došenović, Marinković, Biljana Delić-Vujanović, Marko Cincović, Siniša Grubač, and Radojica Đoković. "The relationship of glucose, insulin, NEFA and index of insulin resistance with the metabolic adaptation of cows in the period before and after calving." In Zbornik radova 26. medunarodni kongres Mediteranske federacije za zdravlje i produkciju preživara - FeMeSPRum. Poljoprivredni fakultet Novi Sad, 2024. http://dx.doi.org/10.5937/femesprumns24010d.

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Metabolic indicators of poor energy status of cows are: decreased glucose and insulin concentration and increased NEFA concentration with increased insulin resistance. This metabolic profile is characteristic of the peripartum period in cows and is caused by reduced food intake and increased glucose directing towards the mammary gland and the pregnant uterus. The aim of this research is to examine the differences in insulin resistance in cows in dry and early lactation and examine the relationship between indicators of insulin resistance and metabolic profile parameters in early lactation. A s
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Forstner, Thomas, Christiane Dienhart, Ludmilla Kedenko, Gernot Wolkersdörfer, and Bernhard Paulweber. "Models for Predicting the Development of Insulin Resistance." In 6th International Conference on Data Science, Technology and Applications. SCITEPRESS - Science and Technology Publications, 2017. http://dx.doi.org/10.5220/0006344801210125.

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Reports on the topic "Insulin resistance"

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Nadeau, Jenifer. Insulin Resistance in Horses - Does it Matter? UConn Extension, 2025. https://doi.org/10.61899/ucext.v2.101.2025.

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Chen, Jiankun, Yingming Gu, Lihong Yin, et al. Network meta-analysis of curative efficacy of different acupuncture methods on obesity combined with insulin resistance. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.8.0075.

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Review question / Objective: Population:Patients diagnosed as obesity with insulin resistance. Obesity reference: Consensus of experts on the Prevention and treatment of adult obesity in China in 2011 and Consensus of Chinese experts on medical nutrition therapy for overweight/obesity in 2016 were developed by the Obesity Group of Chinese Society of Endocrinology(CSE); BMI≥28. IR reference: According to the Expert opinions on insulin resistance evaluation published by Chinese Diabetes Society, HOMA-IR≥2.68 is regarded as the standard for the diagnosis of IR. Regardless of age, gender and cours
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Malin, Alecia, Wei Zheng, and Herbert Yu. Insulin Resistance, IGFs and Energy Balance on the Risk of Breast Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada406834.

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Olefsky, Jerrold. Role of Inflammation and Insulin Resistance in Mouse Models of Breast Cancer. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada580520.

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Gao, Hui, Dan Chen, and Miao Zang. Association between phthalate exposure and insulin resistance: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.4.0026.

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Cai, Wangyu, and Jian Xu. Insulin resistance in women with recurrent miscarriage: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.11.0055.

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Gao, Hui, Chen Gong, Shi-chun Shen, et al. A systematic review on the associations between prenatal phthalate exposure and childhood glycolipid metabolism and blood pressure: evidence from epidemiological studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.6.0111.

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Review question / Objective: The present systematic review was performed to obtain a summary of epidemiological evidence on the relationships of in utero exposure to phthalates with childhood glycolipid metabolism and blood pressure. Condition being studied: Childhood cardiovascular risk factors including blood pressure, lipid profile (e.g., triglycerides, total cholesterol, HDL−C, LDL−C) and glucose metabolism (e.g., insulin, insulin resistance, insulin sensitivity, glucose) were the interested outcomes. Eligibility criteria: In brief, epidemiological studies including cohort study, case-cont
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Zhao, Junyu, Qianping Zhang, Yupeng Yang, Jinming Yao, Lin Liao, and Jianjun Dong. High Prevalence of Thyroid Carcinoma in Patients with Insulin Resistance: A Meta-analysis of Case-control Studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.8.0043.

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Miao, Chenyun, Qingge Guo, Xiaojie Fang, Yun Chen, Ying Zhao, and Qin Zhang. Effects of Probiotics and Synbiotics Supplementation on Insulin Resistance in Women with Polycystic Ovary Syndrome: A Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.5.0112.

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Podgorski, Izabela. Biochemical and Genetic Markers in Aggressiveness and Recurrence of Prostate Cancer: Race-Specific Links to Inflammation and Insulin Resistance. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada566642.

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