Relevant bibliographies by topics / Integrin modulation

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Integrin modulation'

Author: Grafiati
Published: 2 November 2024
Last updated: 31 July 2025

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Journal articles on the topic "Integrin modulation"

1

Hughes, Paul E., and Martin Pfaff. "Integrin affinity modulation." Trends in Cell Biology 8, no. 9 (1998): 359–64. http://dx.doi.org/10.1016/s0962-8924(98)01339-7.

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Martin-Bermudo, Maria D., Olga M. Dunin-Borkowski, and Nicholas H. Brown. "Modulation of Integrin Activity is Vital for Morphogenesis." Journal of Cell Biology 141, no. 4 (1998): 1073–81. http://dx.doi.org/10.1083/jcb.141.4.1073.

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Cells can vary their adhesive properties by modulating the affinity of integrin receptors. The activation and inactivation of integrins by inside-out mechanisms acting on the cytoplasmic domains of the integrin subunits has been demonstrated in platelets, lymphocytes, and keratinocytes. We show that in the embryo, normal morphogenesis requires the α subunit cytoplasmic domain to control integrin adhesion at the right times and places. PS2 integrin (αPS2βPS) adhesion is normally restricted to the muscle termini, where it is required for attaching the muscles to the ends of other muscles and to
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Tozer, Eileen Collins, Paul E. Hughes, and Joseph C. Loftus. "Ligand binding and affinity modulation of integrins." Biochemistry and Cell Biology 74, no. 6 (1996): 785–98. http://dx.doi.org/10.1139/o96-085.

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Integrins are cell adhesion receptors that mediate cell–cell and cell–extracellular matrix interactions. The extracellular domains of these receptors possess binding sites for a diverse range of protein ligands. Ligand binding is divalent cation dependent and involves well-defined motifs in the ligand. Integrins can dynamically regulate their affinity for ligands (inside-out signaling). This ability to rapidly modulate their affinity state is key to their involvement in such processes as cell migration and platelet aggregation. This review will focus on two aspects of integrin function: first,
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Li, Yifan, Shantong Peng, Jiatong Xu, Wenjie Liu, and Qi Luo. "Integrin signaling in tumor biology: mechanisms of intercellular crosstalk and emerging targeted therapies." PeerJ 13 (May 7, 2025): e19328. https://doi.org/10.7717/peerj.19328.

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Integrins, a family of transmembrane cell adhesion receptors, mediate intercellular and cell–extracellular matrix crosstalk via outside-in and inside-out signaling pathways. Integrins, categorized into 24 distinct combinations of α and β subunits, exhibit tissue-specific expression and perform unique or overlapping roles in physiological and pathophysiological processes. These roles encompass embryonic angiogenesis, tissue repair, and the modulation of tumor cell angiogenesis, progression, invasion, and metastasis. Notably, integrins are significant contributors to tumor development, offering
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Ashe, Hilary L. "Modulation of BMP signalling by integrins." Biochemical Society Transactions 44, no. 5 (2016): 1465–73. http://dx.doi.org/10.1042/bst20160111.

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The bone morphogenetic protein (BMP) pathway is a major conserved signalling pathway with diverse roles in development and homeostasis. Given that cells exist in three-dimensional environments, one important area is to understand how the BMP pathway operates within such complex cellular environments. The extracellular matrix contains information regarding tissue architecture and its mechanical properties that is transmitted to the cell via integrin receptors. In this review, I describe various examples of modulation of the BMP pathway by integrins. In the case of the Drosophila embryo and some
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Huang, Jing, Lance C. Bridges, and Judith M. White. "Selective Modulation of Integrin-mediated Cell Migration by Distinct ADAM Family Members." Molecular Biology of the Cell 16, no. 10 (2005): 4982–91. http://dx.doi.org/10.1091/mbc.e05-03-0258.

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A disintegrin and a metalloprotease (ADAM) family members have been implicated in many biological processes. Although it is recognized that recombinant ADAM disintegrin domains can interact with integrins, little is known about ADAM-integrin interactions in cellular context. Here, we tested whether ADAMs can selectively regulate integrin-mediated cell migration. ADAMs were expressed in Chinese hamster ovary cells that express defined integrins (α4β1, α5β1, or both), and cell migration on full-length fibronectin or on its α4β1 or α5β1 binding fragments was studied. We found that ADAMs inhibit i
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Sethi, Tariq, Mark H. Ginsberg, Julian Downward, and Paul E. Hughes. "The Small GTP-binding Protein R-Ras Can Influence Integrin Activation by Antagonizing a Ras/Raf-initiated Integrin Suppression Pathway." Molecular Biology of the Cell 10, no. 6 (1999): 1799–809. http://dx.doi.org/10.1091/mbc.10.6.1799.

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The rapid modulation of ligand-binding affinity (“activation”) is a central property of the integrin family of cell adhesion receptors. The small GTP-binding protein Ras and its downstream effector kinase Raf-1 suppress integrin activation. In this study we explored the relationship between Ras and the closely related small GTP-binding protein R-Ras in modulating the integrin affinity state. We found that R-Ras does not seem to be a direct activator of integrins in Chinese hamster ovary cells. However, we observed that GTP-bound R-Ras strongly antagonizes the Ras/Raf-initiated integrin suppres
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Mahabeleshwar, Ganapati H., Juhua Chen, Weiyi Feng, Payaningal R. Somanath, Olga V. Razorenova, and Tatiana V. Byzova. "Integrin affinity modulation in angiogenesis." Cell Cycle 7, no. 3 (2008): 335–47. http://dx.doi.org/10.4161/cc.7.3.5234.

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Woods, Anne, and John R. Couchman. "Integrin Modulation by Lateral Association." Journal of Biological Chemistry 275, no. 32 (2000): 24233–36. http://dx.doi.org/10.1074/jbc.r000001200.

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Yu, Tao, Xing Wu, Kiran B. Gupta та Dennis F. Kucik. "Affinity, lateral mobility, and clustering contribute independently to β2-integrin-mediated adhesion". American Journal of Physiology-Cell Physiology 299, № 2 (2010): C399—C410. http://dx.doi.org/10.1152/ajpcell.00039.2009.

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Affinity changes and avidity modulation both contribute to activation of β2-integrin-mediated adhesion, an essential, early step in inflammation. Avidity modulation, defined as an increase in adhesiveness independent of integrin conformational changes, might be due to integrin clustering, motion, or both. Increased integrin diffusion upon leukocyte activation has been demonstrated, but whether it is proadhesive in itself, or just constitutes a mechanism for integrin clustering, remains unclear. To understand the proadhesive effects of integrin affinity changes, clustering, and motion, an exper
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Dissertations / Theses on the topic "Integrin modulation"

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Buttery, Robert Christians. "Integrin affinity modulation and lung cancer." Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/29025.

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Recent work has shown that the transmembrane protein CD98 is able to influence the affinity with which β1 integrins bind to extracellular ligands. The first part of this thesis presents confocal microscopy and co-immunoprecipitation experiments that confirm the physical juxtaposition of the two proteins within the cell membrane, suggesting a direct functional link between the two. It also demonstrated that cross-linking CD98 stimulates both phosphoinositide 3-kinase intracellular signalling and increased β1 integrin-dependent cellular adhesion. Because of the role of CD98 in integrin affinity
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Coppolino, Marc Gabriel. "Modulation of integrin function by calreticulin." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ35131.pdf.

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Elliott, Paul Anthony. "Integrin affinity modulation and survival signalling." Thesis, University of Edinburgh, 2008. http://hdl.handle.net/1842/4393.

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Integrins are heterodimeric transmembrane proteins that provide a bi-directional link between the cell’s internal biological mechanisms and the extracellular environment. During inside-out signalling, intracellular messages converge on the integrin cytoplasmic domain to induce a conformational change. This is transmitted to the extracellular domain where it results in an alteration in affinity for integrin ligands such as fibronectin and laminin. In this way the cell has developed the ability to modulate the critical functions of adhesion and cell movement. In outside-in signalling, the integr
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Lad, Yatishkumar. "Integrin affinity modulation by Ras signalling molecules." Thesis, University of Edinburgh, 2002. http://hdl.handle.net/1842/24800.

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In this thesis I have sought to understand the mechanism by which H-Ras and its effectors modulate integrin affinity. H-Ras is a member of the Ras superfamily of small GTP binding proteins. Expression of the constitutively active variant of H-Ras (Ras G12V) within an integrin affinity reporter system (αβ-py cells) reduced integrin affinity (suppressed integrin). Ras effector mutants revealed that integrin suppression is mediated by Raf-dependent and Raf-independent signalling pathways. Raf-independent signalling pathways activated by Ral-GEFs and PI3-kinase were not recognisable for integrin s
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El-Aouni, Chiraz. "In Vivo modulation of integrin linked kinase using transgenic mice." Diss., lmu, 2006. http://nbn-resolving.de/urn:nbn:de:bvb:19-61756.

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van, Wieringen Tijs. "Intra- and Extracellular Modulation of Integrin-directed Connective Tissue Cell Contraction." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-102349.

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All blood vessels in the microvasculature are embedded in loose connective tissue, which regulates the transport of fluid to and from tissues. The intersti-tial fluid pressure (IFP) is one of the forces that control this transport. A lowering of IFP in vivo results in an increased transport of fluid from the circulation into the underhydrated connective tissues, resulting in edema formation. During homeostasis, contractile connective tissue cells exert a tension on the connective tissue fibrous network by binding with β1 in-tegrins, thereby actively controlling IFP. During inflammation, the IF
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Walsh, Erin. "Crossreactivity of alpha9beta1 integrin with p75NTR in modulation of proinvasive activities of glioma cells." Diss., Temple University Libraries, 2011. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/143048.

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Biology<br>Ph.D.<br>Gliomas are the most common and difficult to treat tumors of the central nervous system. Current treatments often fail to slow progression of disease due to the high invasive nature of glioma leading to a high percentage of recurrence. Our previous studies have demonstrated that the levels of alpha; 9 beta; 1 integrin found on high grade glioma were significantly increased in comparison to normal brain tissue where the levels were negligible. We also found that interaction between alpha; 9 beta; 1 integrin and nerve growth factor (NGF) plays a major role in progression of e
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Coyer, Sean R. "Modulation of cell adhesion strengthening by nanoscale geometries at the adhesive interface." Diss., Georgia Institute of Technology, 2010. http://hdl.handle.net/1853/34763.

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Cell adhesion to extracellular matrices (ECM) is critical to many cellular processes including differentiation, proliferation, migration, and apoptosis. Alterations in adhesive mechanisms are central to the behavior of cells in pathological conditions including cancer, atherosclerosis, and defects in wound healing. Although significant progress has been made in identifying molecules involved in adhesion, the mechanisms that dictate the generation of strong adhesive forces remain poorly understood. Specifically, the role of nanoscale geometry of the adhesive interface in integrin recruitment an
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Lygoe, Kate Alexandra. "Modulation of the myofibroblast phenotype is via the interaction of extracellular matrix proteins with integrin receptors." Thesis, University College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.411053.

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Hönig, Ellena [Verfasser], та Ralf [Akademischer Betreuer] Jacob. "Modulation der β1-Integrin-Lokalisation an der apikalen Plasmamembran durch Galektin-3 / Ellena Hönig ; Betreuer: Ralf Jacob". Marburg : Philipps-Universität Marburg, 2017. http://d-nb.info/1129358348/34.

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Books on the topic "Integrin modulation"

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Schöning, Michael. Beeinflussung des metastatischen Potentials von Kolon-Karzinomzellen durch Modulation von Integrin-Rezeptoren. [s.n.], 1998.

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Chander, Ashok Coil. Integrin-Linked Kinase, ECM Composition, and Substrate Rigidity Regulate Focal Adhesion - Actin Coupling, Modulating Survival, Proliferation and Migration: Towards a Biophysical Cancer Biomarker. [publisher not identified], 2012.

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Coppolino, Marc Gabriel. Modulation of integrin function by calreticulin. 1998.

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Carmeliet, G. Modulation of B1 Integrin Function During Neurite Outgrowth. Leuven University Press, 1994.

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Leone, Dino. Tamoxifen-inducible glia-specific cre mice for somatic mutagenesis in oligodendrocytes and schwann cells and [beta]1-integrin regulates neural progenitor maintenance through modulation of growth factor signaling. 2003.

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Kintzel, Claudia. Expression, Modulation und funktionelle Bedeutung von Integrinen auf Kolonkarzinomzellinien. 1993.

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Yust, Jason. Tonal Structure. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190696481.003.0003.

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The concept of tonal structure is intimately associated with the person of Heinrich Schenker, but ultimately in order to enter dialogue with Schenker the theory of tonal structure must take the place of “Schenkerian analysis” in our discourse. A number of useful principles of tonal structure may be derived from Schenker’s theory: Schenkerian notation agrees with the network representation for temporal structure, and linear progressions are a good starting point for a tonal structure discovery procedure. The theory of the Ursatz, however, cannot be understood as an empirical claim but rather as
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Book chapters on the topic "Integrin modulation"

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Leitinger, Birgit. "DDRs: Binding Properties, Cell Adhesion and Modulation of Integrin Function." In Discoidin Domain Receptors in Health and Disease. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-6383-6_1.

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Wright, Samuel D. "Integrin Modulating Factor and the Regulation of Leukocyte Integrins." In Cellular Adhesion. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2466-3_2.

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Mousa, Shaker A., and Paul J. Davis. "Integrin Antagonists and Angiogenesis." In Angiogenesis Modulations in Health and Disease. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6467-5_11.

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Eckert, J. "Structure Modulation and Nanocrystallization of Metallic Glasses: How to Tune Mechanical Properties." In Structural Integrity. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91989-8_81.

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Iacob, Nicusor, Ioan-Alexandru Ivan, Gabriel Schinteie, Claudiu Locovei, Mihai Burdusel, and Victor Kuncser. "Modulation of Water Jets by Detonation of Shaped Charges, for Penetration of Metallic Structures." In Structural Integrity. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-49723-0_37.

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Mohan, Lalit. "Multilinear Fourier Integral Operators on Modulation Spaces." In Trends in Mathematics. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-71989-9_9.

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Defilippi, Paola, Chiarella Bozzo, Massimo Geuna, Paola Rossino, Lorenzo Silengo, and Guido Tarone. "Modulation of extracellular matrix receptors (integrins) on human endothelial cells by cytokines." In Experientia Supplementum. Birkhäuser Basel, 1992. http://dx.doi.org/10.1007/978-3-0348-7001-6_29.

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Bazan, Nicolas G., and Anasheh Halabi. "Docosahexaenoic Acid Signalolipidomics in the Homeostatic Modulation of Photoreceptor/Retinal Pigment Epithelial Cell Integrity During Oxidative Stress." In Studies on Retinal and Choroidal Disorders. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-606-7_7.

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Whittle, Brendan J. R. "Relationship between Sensory Neuropeptides and other Local Vasoactive Mediators in Modulating Gastric Mucosal Integrity." In Advances in Experimental Medicine and Biology. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4899-0744-8_13.

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Ito, Makoto, Motohiro Tani, and Yukihiro Yoshimura. "Neutral Ceramidase as an Integral Modulator for the Generation of S1P and S1P-Mediated Signaling." In Sphingolipid Biology. Springer Japan, 2006. http://dx.doi.org/10.1007/4-431-34200-1_13.

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Conference papers on the topic "Integrin modulation"

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Kireev, Arseny Y., Nikolai S. Laskavyi, and Anton A. Zhuravlev. "Experimental Study of an Integral-Optical Modulator Based on a Mach-Zehnder Interferometer with Increased Modulation Depth." In 2024 IEEE 25th International Conference of Young Professionals in Electron Devices and Materials (EDM). IEEE, 2024. http://dx.doi.org/10.1109/edm61683.2024.10615119.

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Isichenko, Andrei, Andrew Kortyna, Nitesh Chauhan, et al. "Tunable 778 nm Integrated Brillouin Laser Probe for a Rubidium Two-Photon Optical Atomic Clock." In CLEO: Science and Innovations. Optica Publishing Group, 2024. http://dx.doi.org/10.1364/cleo_si.2024.sm1r.7.

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We demonstrate a frequency-controllable 778 nm integrated Brillouin laser with 40 Hz fundamental and 5 kHz integral linewidths maintained over &gt;10 kHz modulation. Stabilization to a rubidium two-photon transition results in stability 2e-13 at 100s.
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Lee, Hee Doo, Michael Y. Park, Ok‐Hee Jeon, and Doo‐Sik Kim. "Abstract B42: Modulation of integrin‐mediated cell adhesion by ADAM15 disintegrin‐like domain." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Dec 6–9, 2009; Houston, TX. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1940-6207.prev-09-b42.

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Geraghty, Terese D., Anugraha Rajagopalan, Rabail Aslam, and Vineet Gupta. "Abstract PR4: Modulation of integrin CD11b as a novel therapeutic strategy against lung cancer." In Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; November 17-20, 2019; Boston, MA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/2326-6074.tumimm19-pr4.

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Dagnino, Lina, and Ernest Ho. "Abstract 1249: Modulation of epidermal growth factor induction of migration by integrin-linked kinase." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1249.

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Ek-Rylander, Barbro, and Goran Andersson. "MODULATION OF OSTEOCLAST ADHESION AND MIGRATION ON PHOSPHORYLATED OSTEOPONTIN (OPN) BY TARTRATE-RESISTANT ACID PHOSPHATASE (TRAP)." In 3rd International Conference on Osteopontin and SIBLING (Small Integrin-Binding Ligand, N-linked Glycoprotein) Proteins, 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.234.

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Barlow, LaMont, Rebecca Meyer, Ethan Shelkey, Bishoy Faltas, and Mark Rubin. "Abstract 5941: Integrin signaling modulation demonstrates potential therapeutic strategy in bladder cancer using three-dimensional organoid culture." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-5941.

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Ashkar, Samy, Sandra Pavao, and Georg F. Weber. "MODULATION OF CELL ATTACHMENT AND MIGRATION BY OSTEOPONTIN THROUGH DIFFERENTIAL ACTIVATION OF PKC ALPHA, BETA1, AND EPSILON." In 3rd International Conference on Osteopontin and SIBLING (Small Integrin-Binding Ligand, N-linked Glycoprotein) Proteins, 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.333.

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El-Kurdi, Mohammed S., J. Scott Van Epps, Robert J. Toth, et al. "Regulation of Cell Adhesion and De-Adhesion Proteins in Veins Perfused Under Arterial Conditions Ex-Vivo." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-61531.

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Failure of veins employed as arterial bypass grafts via intimal hyperplasia (IH) often occurs within 5 years after implantation, requiring re-operation in 60% of all cases1. IH is characterized by de-adhesion, followed by migration of medial and adventitial smooth muscle cells (SMCs) and myofibroblasts into the intima, where they demonstrate uncontrolled proliferation. It is thought that this process may be induced by the abrupt exposure of the veins to the dynamic mechanical environment of the arterial circulation2. Veins are much thinner walled and more distensible than arteries. Therefore,
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DeBeer, D., E. Usadi, and S. R. Hartmann. "Ultrafast modulations in time-delayed four-wave mixing experiments." In OSA Annual Meeting. Optica Publishing Group, 1988. http://dx.doi.org/10.1364/oam.1988.me5.

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Two transient time-delayed four-wave mixing experiments have been performed on the Na D doublet and modulations of 1.9 ps and 980 as have been observed. These periods correspond to the difference and sum frequencies of the two Na D lines. Both experiments were performed using 7-ns pulses of light with frequency components at each of the D-line transitions. The effects of superimposition-state modulations are observed in the integrated TDFWM signal as a function of the time delay. As the time delay is varied, the lowest diffraction order mixing signal is modulated. Higher diffraction order mixi
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Reports on the topic "Integrin modulation"

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Philosoph-Hadas, Sonia, Peter B. Kaufman, Shimon Meir, and Abraham H. Halevy. Inhibition of the Gravitropic Shoot Bending in Stored Cut Flowers Through Control of Their Graviperception: Involvement of the Cytoskeleton and Cytosolic Calcium. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7586533.bard.

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Original objectives: The basic goal of the present project was to study the mechanism involved in shoot graviperception and early transduction, in order to determine the sequence of events operating in this process. This will enable to control the entire process of gravity-induced differential growth without affecting vertical growth processes essential for development. Thus, several new postulated interactions, operating at the perception and early transduction stages of the signaling cascade leading to auxin-mediated bending, were proposed to be examined in snapdragon spikes and oat shoot pu
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