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Journal articles on the topic 'Interactions between tissues'

Author: Grafiati

Published: 25 May 2024

Last updated: 31 July 2025

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1

Daniel, Władyslawa Anna, and Jacek Wöjcikowski. "Interactions between Promazine and Antidepressants at the Level of Cellular Distribution." Pharmacology & Toxicology 81, no. 6 (1997): 259–64. http://dx.doi.org/10.1111/j.1600-0773.1997.tb00003.x.

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AbstractThe pharmacokinetic interactions in clinical combinations of a phenolhiazine neuroleptic and antidepressants at the level of cellular distribution were investigated. Uptake experiments were performed on slices of various rat tissues as a system with intact lysosomes. Promazine and antidepressants (imipramine, amitriptyline, sertraline, fluoxetine) were incubated separately or jointly with tissue slices for 1 hr. Initial concentration of each drug was 5 uM. The interaction studies were carried out in the absence and presence of ammonium chloride (20 mM), a lysosomotropic compound which

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2

Kawao, Naoyuki, and Hiroshi Kaji. "Interactions Between Muscle Tissues and Bone Metabolism." Journal of Cellular Biochemistry 116, no. 5 (2015): 687–95. http://dx.doi.org/10.1002/jcb.25040.

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Tomas, Eva, Meghan Kelly, Xiaoqin Xiang, et al. "Metabolic and hormonal interactions between muscle and adipose tissue." Proceedings of the Nutrition Society 63, no. 2 (2004): 381–85. http://dx.doi.org/10.1079/pns2004356.

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From the perspective of a muscle physiologist, adipose tissue has long been perceived predominantly as a fuel reservoir that provides muscle and other tissues with NEFA when exogenous nutrients are insufficient for their energy needs. Recently, studies have established that adipose tissue is also an endocrine organ. Among the hormones it releases are adiponectin and leptin, both of which can activate AMP-activated protein kinase and increase fatty acid oxidation in skeletal muscle and probably other tissues. Deficiencies of leptin or leptin receptor, adiponectin and IL-6 are associated with ob

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Tsang, Anthony H., Johanna L. Barclay, and Henrik Oster. "Interactions between endocrine and circadian systems." Journal of Molecular Endocrinology 52, no. 1 (2013): R1—R16. http://dx.doi.org/10.1530/jme-13-0118.

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In most species, endogenous circadian clocks regulate 24-h rhythms of behavior and physiology. Clock disruption has been associated with decreased cognitive performance and increased propensity to develop obesity, diabetes, and cancer. Many hormonal factors show robust diurnal secretion rhythms, some of which are involved in mediating clock output from the brain to peripheral tissues. In this review, we describe the mechanisms of clock–hormone interaction in mammals, the contribution of different tissue oscillators to hormonal regulation, and how changes in circadian timing impinge on endocrin

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Nammour, S., H. S. Loh, R. De Moor, and C. P. Eduardo. "Interactions between Oral Tissues and External Light and Matters." International Journal of Dentistry 2012 (2012): 1. http://dx.doi.org/10.1155/2012/726259.

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Wang, Congcong, Zihan Yi, Ye Jiao, Zhong Shen, Fei Yang, and Shankuan Zhu. "Gut Microbiota and Adipose Tissue Microenvironment Interactions in Obesity." Metabolites 13, no. 7 (2023): 821. http://dx.doi.org/10.3390/metabo13070821.

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Obesity is an increasingly serious global health problem. Some studies have revealed that the gut microbiota and its metabolites make important contributions to the onset of obesity. The gut microbiota is a dynamic ecosystem composed of diverse microbial communities with key regulatory functions in host metabolism and energy balance. Disruption of the gut microbiota can result in obesity, a chronic metabolic condition characterized by the excessive accumulation of adipose tissue. Host tissues (e.g., adipose, intestinal epithelial, and muscle tissues) can modulate the gut microbiota via microen

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Rainbow, Roshni, Weiping Ren, and Li Zeng. "Inflammation and Joint Tissue Interactions in OA: Implications for Potential Therapeutic Approaches." Arthritis 2012 (June 18, 2012): 1–8. http://dx.doi.org/10.1155/2012/741582.

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It is increasingly recognized that the pathogenesis of cartilage degradation in osteoarthritis (OA) is multifactorial and involves the interactions between cartilage and its surrounding tissues. These interactions regulate proinflammatory cytokine-mediated cartilage destruction, contributing to OA progression as well as cartilage repair. This review explores the pathogenesis of OA in the context of the multiple tissue types in the joint and discusses the implications of such complex tissue interaction in the development of anti-inflammatory therapeutics for the treatment of OA.

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Nagase, T., T. Ito, M. Yanai, J. G. Martin, and M. S. Ludwig. "Responsiveness of and interactions between airways and tissue in guinea pigs during induced constriction." Journal of Applied Physiology 74, no. 6 (1993): 2848–54. http://dx.doi.org/10.1152/jappl.1993.74.6.2848.

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Mechanical interdependence between airways and tissues can modify the magnitude of induced bronchoconstriction. We questioned whether the guinea pig, an animal with abundant airway smooth muscle, would differ from other species in the relative responsiveness of and interactions between airways and tissues. Therefore we induced constriction with aerosolized methacholine (MCh) and partitioned responses into airway and tissue components. We measured tracheal and alveolar pressures using alveolar capsules in open-chest guinea pigs (n = 9) during mechanical ventilation [frequency = 1 Hz, tidal volu

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Elofsson, Hampus, Doroteya Raykova, and Agata Zieba Wicher. "Abstract 6772: Powerful background reduction in fluorescent tissue stains with an improved proximity-based technology for detection of protein-protein interactions." Cancer Research 83, no. 7_Supplement (2023): 6772. http://dx.doi.org/10.1158/1538-7445.am2023-6772.

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Abstract Immunofluorescent staining of tissues via in situ proximity ligation assay (isPLA) is now a well-established tool for highly sensitive detection of protein-protein interactions and their localization. It finds versatile applications in basic research, understanding of cellular processes, visualization of tissue architecture, and identification of biomarkers, among others. However, standard IF and isPLA techniques alike may suffer from high background. Tissues are particularly affected by background problems caused by autofluorescence and tissue infiltration of various cell types (e.g.

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Amponsah, N. T., E. E. Jones, H. J. Ridgway, and M. V. Jaspers. "Microscopy of some interactions between Botryosphaeriaceae species and grapevine tissues." Australasian Plant Pathology 41, no. 6 (2012): 665–73. http://dx.doi.org/10.1007/s13313-012-0159-x.

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11

Herrmann, Marietta, Klaus Engelke, Regina Ebert, et al. "Interactions between Muscle and Bone—Where Physics Meets Biology." Biomolecules 10, no. 3 (2020): 432. http://dx.doi.org/10.3390/biom10030432.

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Muscle and bone interact via physical forces and secreted osteokines and myokines. Physical forces are generated through gravity, locomotion, exercise, and external devices. Cells sense mechanical strain via adhesion molecules and translate it into biochemical responses, modulating the basic mechanisms of cellular biology such as lineage commitment, tissue formation, and maturation. This may result in the initiation of bone formation, muscle hypertrophy, and the enhanced production of extracellular matrix constituents, adhesion molecules, and cytoskeletal elements. Bone and muscle mass, resist

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Borrmann, Helene, and Filipa Rijo-Ferreira. "Crosstalk between circadian clocks and pathogen niche." PLOS Pathogens 20, no. 5 (2024): e1012157. http://dx.doi.org/10.1371/journal.ppat.1012157.

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Circadian rhythms are intrinsic 24-hour oscillations found in nearly all life forms. They orchestrate key physiological and behavioral processes, allowing anticipation and response to daily environmental changes. These rhythms manifest across entire organisms, in various organs, and through intricate molecular feedback loops that govern cellular oscillations. Recent studies describe circadian regulation of pathogens, including parasites, bacteria, viruses, and fungi, some of which have their own circadian rhythms while others are influenced by the rhythmic environment of hosts. Pathogens targe

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Mijanović, Olja, Timofey Pylaev, Angelina Nikitkina, et al. "Tissue Engineering Meets Nanotechnology: Molecular Mechanism Modulations in Cornea Regeneration." Micromachines 12, no. 11 (2021): 1336. http://dx.doi.org/10.3390/mi12111336.

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Nowadays, tissue engineering is one of the most promising approaches for the regeneration of various tissues and organs, including the cornea. However, the inability of biomaterial scaffolds to successfully integrate into the environment of surrounding tissues is one of the main challenges that sufficiently limits the restoration of damaged corneal tissues. Thus, the modulation of molecular and cellular mechanisms is important and necessary for successful graft integration and long-term survival. The dynamics of molecular interactions affecting the site of injury will determine the corneal tra

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Kim-Hellmuth, Sarah, François Aguet, Meritxell Oliva, et al. "Cell type–specific genetic regulation of gene expression across human tissues." Science 369, no. 6509 (2020): eaaz8528. http://dx.doi.org/10.1126/science.aaz8528.

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The Genotype-Tissue Expression (GTEx) project has identified expression and splicing quantitative trait loci in cis (QTLs) for the majority of genes across a wide range of human tissues. However, the functional characterization of these QTLs has been limited by the heterogeneous cellular composition of GTEx tissue samples. We mapped interactions between computational estimates of cell type abundance and genotype to identify cell type–interaction QTLs for seven cell types and show that cell type–interaction expression QTLs (eQTLs) provide finer resolution to tissue specificity than bulk tissue

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Pond, C. M. "Adipose tissue, the immune system and exercise fatigue: how activated lymphocytes compete for lipids." Biochemical Society Transactions 30, no. 2 (2002): 270–75. http://dx.doi.org/10.1042/bst0300270.

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Adipose depots that contain lymph nodes, and probably intermuscular fat in skeletal and cardiac muscle, are specialized to provision adjacent tissue in a paracrine mode. Perinodal adipocytes respond selectively to various cytokines and incorporate proportionately more polyunsaturated fatty acids. Lipolysis in the adipocytes of node-containing depots can be stimulated via inflammation of the enclosed lymph nodes. Repeated immune stimulation elicits properties characteristic of perinodal adipocytes in those elsewhere in the same depot, and hours later in other node-containing depots, but not in

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Davey, Marie L., and Randolph S. Currah. "Interactions between mosses (Bryophyta) and fungi." Canadian Journal of Botany 84, no. 10 (2006): 1509–19. http://dx.doi.org/10.1139/b06-120.

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A taxonomically diverse suite of fungi interacts with bryophytes as pathogens, parasites, saprobes, and commensals. Necrotrophic pathogens such as Tephrocybe palustris (Peck) Donk and Nectria mnii Döbbeler form patches of moribund gametophytes in otherwise healthy mats of mosses. These pathogens exhibit different methods of host cell disruption; N. mnii appears to displace the host cell protoplast with intracellular hyphae, while T. palustris causes host protoplast degeneration. Host responses to infection by bryopathogens are also variable. Host–pathogen relationships can be highly evolved, a

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Mahmood, R., K. C. Flanders, and G. M. Morriss-Kay. "Interactions between retinoids and TGF beta s in mouse morphogenesis." Development 115, no. 1 (1992): 67–74. http://dx.doi.org/10.1242/dev.115.1.67.

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Using immunocytochemical methods we describe the distribution of different TGF beta isoforms and the effects of excess retinoic acid on their expression during early mouse embryogenesis (8 1/2 - 10 1/2 days of development). In normal embryos at 9 days, intracellular TGF beta 1 is expressed most intensely in neuroepithelium and cardiac myocardium whereas extracellular TGF beta 1 is expressed in mesenchymal cells and in the endocardium of the heart. At later stages, intracellular TGF beta 1 becomes very restricted to the myocardium and to a limited number of head mesenchymal cells; extracellular

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Edén, Desirée, Jonas Vennberg, Tanja Büsgen, Doroteya Raykova, and Agata Zieba-Wicher. "Abstract 72: A spatial interactomics approach reveals interplay between the tumor microenvironment and T cell activation." Cancer Research 84, no. 6_Supplement (2024): 72. http://dx.doi.org/10.1158/1538-7445.am2024-72.

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Abstract Background: T cells play a fundamental role in immune response to tumors. However, the tumor microenvironment (TME) - a complex network of cells, signaling molecules, and extracellular matrix components - employs various strategies to evade immune surveillance and suppress T cell activity. Among the many molecular interactions taking place in the TME, the role of CD8/MHC I, LAG3/MHC II and PD1/PDL1 is pivotal in modulating anti-cancer response and the escape routes that cancer in turn uses to withstand immune defense. LAG3/MHC II and PD1/PDL1 interactions are inhibitory, causing T cel

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Noden, Drew M. "Interactions and fates of avian craniofacial mesenchyme." Development 103, Supplement (1988): 121–40. http://dx.doi.org/10.1242/dev.103.supplement.121.

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Craniofacial mesenchyme is composed of three mesodermal populations – prechordal plate, lateral mesoderm and paraxial mesoderm, which includes the segmented occipital somites and the incompletely segmented somitomeres – and the neural crest. This paper outlines the fates of each of these, as determined using quail–chick chimaeras, and presents similarities and differences between these cephalic populations and their counterparts in the trunk. Prechordal and paraxial mesodermal populations are the sources of all voluntary muscles of the head. The latter also provides most of the connective prec

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20

Tsuneda, Akihiko, and Greg Thorn. "Interactions between Lentinula edodes and pseudomonads." Canadian Journal of Microbiology 40, no. 11 (1994): 937–43. http://dx.doi.org/10.1139/m94-150.

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Interactions between four dikaryotic strains of Lentinula edodes and seven strains of Pseudomonas species were investigated in vitro. Two strains of Pseudomonas tolaasii were strongly pathogenic to fruiting bodies of L. edodes, causing browning and lysis of the inoculated tissues. The mode and outcome of interactions on agar varied with the combination between strains of L. edodes and bacteria. Pseudomonas cepacia and two strains of Pseudomonas tolaasii inhibited hyphal growth of L. edodes strains, whereas Pseudomonas fluorescens and one strain of Pseudomonas tolaasii were generally susceptibl

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Nnoromele, Patrick O., McKaily Adams, Annabelle Pan, Ying V. Liu, Joyce Wang, and Mandeep S. Singh. "Cell–cell interactions between transplanted retinal organoid cells and recipient tissues." Current Opinion in Genetics & Development 89 (December 2024): 102277. http://dx.doi.org/10.1016/j.gde.2024.102277.

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22

Rey, P., N. Benhamou, E. Wulff, and Y. Tirilly. "Interactions between tomato (Lycopersicon esculentum) root tissues and the mycoparasitePythium oligandrum." Physiological and Molecular Plant Pathology 53, no. 2 (1998): 105–22. http://dx.doi.org/10.1006/pmpp.1998.0159.

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Sorrell, J. Michael, and Arnold I. Caplan. "Heparan Sulfate: A Regulator of White Adipocyte Differentiation and of Vascular/Adipocyte Interactions." Biomedicines 10, no. 9 (2022): 2115. http://dx.doi.org/10.3390/biomedicines10092115.

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White adipose tissues are major endocrine organs that release factors, termed adipokines, which affect other major organ systems. The development and functions of adipose tissues depend largely upon the glycosaminoglycan heparan sulfate. Heparan sulfate proteoglycans (HSPGs) surround both adipocytes and vascular structures and facilitate the communication between these two components. This communication mediates the continued export of adipokines from adipose tissues. Heparan sulfates regulate cellular physiology and communication through a sulfation code that ionically interacts with heparan-

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Bonnet, Muriel, Nicolas Kaspric, Kimberly Vonnahme, Didier Viala, Christophe Chambon, and Brigitte Picard. "Prediction of the Secretome and the Surfaceome: A Strategy to Decipher the Crosstalk between Adipose Tissue and Muscle during Fetal Growth." International Journal of Molecular Sciences 21, no. 12 (2020): 4375. http://dx.doi.org/10.3390/ijms21124375.

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Crosstalk between adipose and muscular tissues is hypothesized to regulate the number of muscular and adipose cells during fetal growth, with post-natal consequences on lean and fat masses. Such crosstalk largely remains, however, to be described. We hypothesized that a characterization of the proteomes of adipose and muscular tissues from bovine fetuses may enhance the understanding of the crosstalk between these tissues through the prediction of their secretomes and surfaceomes. Proteomic experiments have identified 751 and 514 proteins in fetal adipose tissue and muscle. These are mainly in

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Razek, Adel. "Potential biological effects on living tissues of biodiversity subsequent to exposure to high-frequency electromagnetic fields." Thermal Science and Engineering 8, no. 2 (2025): 11750. https://doi.org/10.24294/tse11750.

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Exposure to high-frequency (HF) electromagnetic fields (EMF) has various effects on living tissues involved in biodiversity. Interactions between fields and exposed tissues are correlated with the characteristics of the exposure, tissue behavior, and field intensity and frequency. These interactions can produce mainly adverse thermal and possibly non-thermal effects. In fact, the most expected type of outcome is a thermal biological effect (BE), where tissues are materially heated by the dissipated electromagnetic energy due to HF-EMF exposure. In case of exposure at a disproportionate intensi

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ten Cate, J. M., and J. D. B. Featherstone. "Mechanistic Aspects of the Interactions Between Fluoride and Dental Enamel." Critical Reviews in Oral Biology & Medicine 2, no. 3 (1991): 283–96. http://dx.doi.org/10.1177/10454411910020030101.

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For many years after the discovery of its caries preventive effect, fluoride was thought to be primarily active by lowering the solubility of the apatitic mineral phase of the dental hard tissues. Recent findings have shed new light on the mechanisms by which fluoride inhibits or delays dental caries. Fluoride present in the oral fluids alters the rate of the naturally occurring dissolution and reprecipitation processes at the tooth-oral fluid interface. Demineralization of enamel is inhibited by concentrations of fluoride in the sub-ppm range. Likewise, remineralization of incipient caries le

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Eun, C. S., E. H. Oh, A. R. Lee, C. H. Park, and D. S. Han. "P913 Interactions between Intestinal Microbiota and micro-RNAs in Inflammatory Bowel Disease." Journal of Crohn's and Colitis 17, Supplement_1 (2023): i1025. http://dx.doi.org/10.1093/ecco-jcc/jjac190.1043.

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Abstract Background Both gut microbiota and micro-RNAs (miRNAs) have been implicated in the pathogenesis of inflammatory bowel disease (IBD). We aimed to investigate the interactions between gut microbiota and miRNAs which are altered in murine colitis models and patients with IBD using multi-omics analysis. Methods Gut microbiota and miRNAs were analyzed in a mouse experiment (7 dextran sodium sulfate [DSS]-treated mice, 7 azoxymethane [AOM] and DSS-treated mice, and 6 controls) and a human experiment (10 patients with active Crohn’s disease, 10 patients with active ulcerative colitis [UC], a

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Barz, Cristian, Marek Petters, Adam Dorsz, and Przemysław Syrek. "Possible Interactions Between Stent and Electromagnetic Field." Science, Technology and Innovation 3, no. 2 (2018): 48–51. http://dx.doi.org/10.5604/01.3001.0012.8156.

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Over the past years, an increase in the amount of the electromagnetic sources could be observed. Model presented in this article is limited to the impact of low frequency fields generated by the e.g. electrical power lines or magnetic coils in a bone fractures therapy. Particularly, the effect of the magnetic component of the electromagnetic field on stents will be evaluated. The conductivity of human tissues will be investigated. Yielded results will be used to simplify complicated, three-dimensional problem of the current distribution in stent branches, to one-dimensional one. The merits of

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Eisenberg, S. R., and A. J. Grodzinsky. "The Kinetics of Chemically Induced Nonequilibrium Swelling of Articular Cartilage and Corneal Stroma." Journal of Biomechanical Engineering 109, no. 1 (1987): 79–89. http://dx.doi.org/10.1115/1.3138647.

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An electromechanical model for charged, hydrated tissues is developed to predict the kinetics of changes in swelling and isometric compressive stress induced by changes in bath salt concentration. The model focuses on ionic transport as the rate limiting step in chemically modulating electrical interactions between the charged macromolecules of the extracellular matrix. The swelling response to such changes in local interaction forces is determined by the relative rates of chemical diffusion and fluid redistribution in the tissue sample. We have tested the model by comparing the experimentally

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Bose, Subhaya, Kinjal Dasbiswas, and Arvind Gopinath. "Matrix Stiffness Modulates Mechanical Interactions and Promotes Contact between Motile Cells." Biomedicines 9, no. 4 (2021): 428. http://dx.doi.org/10.3390/biomedicines9040428.

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The mechanical micro-environment of cells and tissues influences key aspects of cell structure and function, including cell motility. For proper tissue development, cells need to migrate, interact, and form contacts. Cells are known to exert contractile forces on underlying soft substrates and sense deformations in them. Here, we propose and analyze a minimal biophysical model for cell migration and long-range cell–cell interactions through mutual mechanical deformations of the substrate. We compute key metrics of cell motile behavior, such as the number of cell-cell contacts over a given time

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Feng, Tzu-Yu, Francesca N. Azar, Mitchell T. McGinty, and Melanie R. Rutkowski. "Reciprocal interactions between the gut microbiome and mammary tissue microenvironment promote distant regulation of breast tumor metastasis." Journal of Immunology 206, no. 1_Supplement (2021): 56.12. http://dx.doi.org/10.4049/jimmunol.206.supp.56.12.

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Abstract Establishing commensal dysbiosis, defined as an inflammatory gut microbiome with low biodiversity, prior to breast tumor initiation enhances early dissemination of hormone-receptor positive (HR+) mammary tumor cells into the circulation, distal lymph nodes, and lungs. Here, we sought to define mammary tissue mediators of dysbiosis-induced tumor dissemination. Commensal dysbiosis increased mast cell numbers in the pre-malignant mammary tissue, with numbers remaining elevated in the presence of a tumor. During early tumor progression, increased mast cell numbers correlated with increase

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Rhodes, Judith C. "Aspergillusproteinases and their interactions with host tissues." Canadian Journal of Botany 73, S1 (1995): 1126–31. http://dx.doi.org/10.1139/b95-368.

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Invasive aspergillosis is a life-threatening infection that is caused primarily by the species Aspergillus fumigatus and A. flavus, both of which are highly angioinvasive. From this observation, interest has focused on proteinases produced by these organisms and their possible roles in the pathogenesis of infection. Both species produce alkaline serine proteinases (ALP) and metalloproteinases during the course of infection based on immunohistochemistry of experimental lesions and serologic response of patients. These enzymes can be shown to degrade numerous biologically relevant targets, inclu

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Onita (Mladin), Bianca, Paul Albu, Hildegard Herman, et al. "Correlation between Heavy Metal-Induced Histopathological Changes and Trophic Interactions between Different Fish Species." Applied Sciences 11, no. 9 (2021): 3760. http://dx.doi.org/10.3390/app11093760.

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This study assessed the distribution of heavy metals in the gills, kidney, and liver, correlated with the severity of histopathological changes, of three fish species with different feeding habitats (Barbus barbus, Squalius cephalus, and Chondrostoma nasus) from the Crișul Negru river, Romania. The levels of copper (Cu), chromium (Cr), cadmium (Cd), lead (Pb), and zinc (Zn) in fish tissues were measured by atomic absorption spectrophotometry. Histopathology and the expressions of TNF-α and proliferation cell nuclear antigen (PCNA) were investigated by immunohistochemistry and Western blot. Our

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Muzzopappa, Mariana, Lada Murcia, and Marco Milán. "Feedback amplification loop drives malignant growth in epithelial tissues." Proceedings of the National Academy of Sciences 114, no. 35 (2017): E7291—E7300. http://dx.doi.org/10.1073/pnas.1701791114.

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Interactions between cells bearing oncogenic mutations and the surrounding microenvironment, and cooperation between clonally distinct cell populations, can contribute to the growth and malignancy of epithelial tumors. The genetic techniques available in Drosophila have contributed to identify important roles of the TNF-α ligand Eiger and mitogenic molecules in mediating these interactions during the early steps of tumor formation. Here we unravel the existence of a tumor-intrinsic—and microenvironment-independent—self-reinforcement mechanism that drives tumor initiation and growth in an Eiger

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Vanpouille, Christophe, Angélique Biancotto, Andrea Lisco, and Beda Brichacek. "Interactions between Human Immunodeficiency Virus Type 1 and Vaccinia Virus in Human Lymphoid Tissue Ex Vivo." Journal of Virology 81, no. 22 (2007): 12458–64. http://dx.doi.org/10.1128/jvi.00326-07.

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ABSTRACT Vaccinia virus (VACV) has been attracting attention recently not only as a vector for various vaccines but also as an immunization tool against smallpox because of its potential use as a bioterrorism agent. It has become evident that in spite of a long history of studies of VACV, its tissue pathogenesis remains to be fully understood. Here, we investigated the pathogenesis of VACV and its interactions with human immunodeficiency virus type 1 (HIV-1) in the context of human lymphoid tissues. We found that ex vivo-cultured tonsillar tissue supports productive infection by the New York C

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Havran, Wendy L., Julie M. Jameson, and Deborah A. Witherden. "Epithelial Cells and Their Neighbors. III. Interactions between intraepithelial lymphocytes and neighboring epithelial cells." American Journal of Physiology-Gastrointestinal and Liver Physiology 289, no. 4 (2005): G627—G630. http://dx.doi.org/10.1152/ajpgi.00224.2005.

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Intraepithelial γδ-T cells are present in all epithelial tissues, where they reside in close contact with neighboring epithelial cells. Our data support the idea that the role of these cells is to monitor neighboring cells for signs of damage or disease. Once a problem is detected, the intraepithelial γδ-T cells can lyse damaged or malignant epithelial cells, directly participate in tissue repair through production of epithelial growth factors, and play a unique role in the recruitment of inflammatory cells to the site of damage. Intraepithelial γδ-T cells play unique roles in homeostasis and

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Van Ryk, Donald, Sinmanus Vimonpatranon, Joe Hiatt та ін. "The V2 domain of HIV gp120 mimics an interaction between CD4 and integrin ⍺4β7". PLOS Pathogens 19, № 12 (2023): e1011860. http://dx.doi.org/10.1371/journal.ppat.1011860.

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The CD4 receptor, by stabilizing TCR-MHC II interactions, plays a central role in adaptive immunity. It also serves as the HIV docking receptor. The HIV gp120 envelope protein binds directly to CD4. This interaction is a prerequisite for viral entry. gp120 also binds to ⍺4β7, an integrin that is expressed on a subset of memory CD4+ T cells. HIV tropisms for CD4+ T cells and gut tissues are central features of HIV pathogenesis. We report that CD4 binds directly to ⍺4β7 in a dynamic way, consistent with a cis regulatory interaction. The molecular details of this interaction are related to the wa

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Clouthier, D. E., K. Hosoda, J. A. Richardson, et al. "Cranial and cardiac neural crest defects in endothelin-A receptor-deficient mice." Development 125, no. 5 (1998): 813–24. http://dx.doi.org/10.1242/dev.125.5.813.

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Neural crest cells arise in the dorsal aspect of the neural tube and migrate extensively to differentiate into a variety of neural and non-neural tissues. While interactions between neural crest cells and their local environments are required for the proper development of these tissues, little information is available about the molecular nature of the cell-cell interactions in cephalic neural crest development. Here we demonstrate that mice deficient for one type of endothelin receptor, ETA, mimic the human conditions collectively termed CATCH 22 or velocardiofacial syndrome, which include sev

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Xu, Xiaoyuan, Aimin Yang, Yan Han, Siran Li, Guimin Hao, and Na Cui. "Pancancer analysis of the interactions between CTNNB1 and infiltrating immune cell populations." Medicine 103, no. 44 (2024): e40186. http://dx.doi.org/10.1097/md.0000000000040186.

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Recently, evidence has indicated that CTNNB1 is important in a variety of malignancies. However, how CTNNB1 interacts with immune cell infiltration remains to be further investigated. In this study, we focused on the correlations between CTNNB1 and tumorigenesis, tumor progression, mutation, phosphorylation, and prognosis via gene expression profiling interaction analysis; TIMER 2.0, cBioPortal, GTEx, CPTAC, and GEPIA2 database analyses; and R software. CTNNB1 mutations are most found in uterine endometrioid carcinoma and hepatocellular carcinoma. However, no CTNNB1 mutations were found to be

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Zhou, Jiong, Takashi Suzuki, Agnes Kovacic, et al. "Interactions between Prostaglandin E2, Liver Receptor Homologue-1, and Aromatase in Breast Cancer." Cancer Research 65, no. 2 (2005): 657–63. http://dx.doi.org/10.1158/0008-5472.657.65.2.

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Abstract Local synthesis of estrogens within breast adipose tissue by cytochrome P450 aromatase contributes to the growth of postmenopausal breast cancers. One of the major stimulators of aromatase expression in breast is prostaglandin E2 (PGE2) derived from tumorous epithelium and/or infiltrating macrophages. Recently, the orphan nuclear receptor, liver receptor homologue-1 (LRH-1), has also been shown to regulate aromatase expression in breast adipose tissue. We therefore examined the expression of, and correlations between, aromatase and LRH-1 mRNA in a panel of breast carcinoma tissues and

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Goto, Shunsuke, Hisashi Anbutsu, and Takema Fukatsu. "Asymmetrical Interactions between Wolbachia and Spiroplasma Endosymbionts Coexisting in the Same Insect Host." Applied and Environmental Microbiology 72, no. 7 (2006): 4805–10. http://dx.doi.org/10.1128/aem.00416-06.

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ABSTRACT We investigated the interactions between the endosymbionts Wolbachia pipientis strain wMel and Spiroplasma sp. strain NSRO coinfecting the host insect Drosophila melanogaster. By making use of antibiotic therapy, temperature stress, and hemolymph microinjection, we established the following strains in the same host genetic background: the SW strain, infected with both Spiroplasma and Wolbachia; the S strain, infected with Spiroplasma only; and the W strain, infected with Wolbachia only. The infection dynamics of the symbionts in these strains were monitored by quantitative PCR during

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Zhang, Chen-xing, Hui-yu Wang, and Tong-xin Chen. "Interactions between Intestinal Microflora/Probiotics and the Immune System." BioMed Research International 2019 (November 20, 2019): 1–8. http://dx.doi.org/10.1155/2019/6764919.

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The digestive tract is home to millions of microorganisms and is the main and most important part of bacterial colonization. On one hand, the abundant bacterial community in intestinal tissues may pose potential health challenges such as inflammation and sepsis in cases of opportunistic invasion. Thus, the immune system has evolved and adapted to maintain the symbiotic relationship between host and microbiota. On the other hand, the intestinal microflora also exerts an immunoregulatory function to maintain host immune homeostasis, which cannot be neglected. In addition, the interaction of eith

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Criddle, Richard S., Lee D. Hansen, Brian F. Woodfield, and H. Dennis Tolley. "Modeling transthyretin (TTR) amyloid diseases, from monomer to amyloid fibrils." PLOS ONE 19, no. 6 (2024): e0304891. http://dx.doi.org/10.1371/journal.pone.0304891.

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ATTR amyloidosis is caused by deposition of large, insoluble aggregates (amyloid fibrils) of cross-β-sheet TTR protein molecules on the intercellular surfaces of tissues. The process of amyloid formation from monomeric TTR protein molecules to amyloid deposits has not been fully characterized and is therefore modeled in this paper. Two models are considered: 1) TTR monomers in the blood spontaneously fold into a β-sheet conformation, aggregate into short proto-fibrils that then circulate in the blood until they find a complementary tissue where the proto-fibrils accumulate to form the large, i

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Veenstra, R. D., and R. L. DeHaan. "Electrotonic interactions between aggregates of chick embryo cardiac pacemaker cells." American Journal of Physiology-Heart and Circulatory Physiology 250, no. 3 (1986): H453—H463. http://dx.doi.org/10.1152/ajpheart.1986.250.3.h453.

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Synchronization of spontaneously active heart cell aggregates occurs shortly after they are brought into contact. The synchronous rate is determined by pacemaker phase resetting and passive subthreshold electrotonic interactions. To further study the effects of passive electrical interactions, we have used 150-microns diameter aggregates prepared from cells of 4d (4-day ventricle + 1 day in vitro), 7d, and 14d embryonic chick ventricle as models of primary, latent, and nonpacemaker tissues, respectively. Coupling of 4d and 7d aggregates (4d/7d pairs) leads to intermediate synchronous rates. We

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Stern, Claudio D., Sanjay M. Sisodiya, and Roger J. Keynes. "Interactions between neurites and somite cells: inhibition and stimulation of nerve growth in the chick embryo." Development 91, no. 1 (1986): 209–26. http://dx.doi.org/10.1242/dev.91.1.209.

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After neural processes emerge from the neural tube in the chick embryo, their growth is restricted to the cranial halves of the neighbouring somites. In this study we have developed an in vitro system to model the interactions between these tissue types. Pioneer neurites display a hierarchy of preferences in terms of the substrates they can grow on. As expected, tissue culture plastic does not support neural outgrowth, but this can be overcome by coating the plastic substrate with either collagen or poly-L-lysine. Neural crest, cranial half somite, and a number of other tissues support growth

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Retana-Lobo DDS, MSD, Cristina. "Dental Pulp Regeneration: Insights from Biological Processes." Odovtos - International Journal of Dental Sciences 20, no. 1 (2017): 10–16. http://dx.doi.org/10.15517/ijds.v0i0.31269.

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One of the major approaches on dentistry research in this century is the development of biological strategies (tissue engineering) to regenerate/ biomineralize lost dental tissues. During dentin-pulp regeneration, the interaction between stem cells, signaling molecules, biomaterials and the microenvironment in the periapical area drives the process for dental pulp tissue engineering. Understanding the signaling mechanisms and interactions involved with the biological process for the formation of a new tissue, is essential. The knowledge of the micro-environment is the key for the application o

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Retana-Lobo DDS, MSD, Cristina. "Dental Pulp Regeneration: Insights from Biological Processes." Odovtos - International Journal of Dental Sciences 20, no. 1 (2017): 10–16. http://dx.doi.org/10.15517/ijds.v20i1.31269.

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One of the major approaches on dentistry research in this century is the development of biological strategies (tissue engineering) to regenerate/ biomineralize lost dental tissues. During dentin-pulp regeneration, the interaction between stem cells, signaling molecules, biomaterials and the microenvironment in the periapical area drives the process for dental pulp tissue engineering. Understanding the signaling mechanisms and interactions involved with the biological process for the formation of a new tissue, is essential. The knowledge of the micro-environment is the key for the application o

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Wang, Maochun, Guihua Tan, Huiming Jiang, et al. "Molecular crosstalk between articular cartilage, meniscus, synovium, and subchondral bone in osteoarthritis." Bone & Joint Research 11, no. 12 (2022): 862–72. http://dx.doi.org/10.1302/2046-3758.1112.bjr-2022-0215.r1.

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Aims Osteoarthritis (OA) is a common degenerative joint disease worldwide, which is characterized by articular cartilage lesions. With more understanding of the disease, OA is considered to be a disorder of the whole joint. However, molecular communication within and between tissues during the disease process is still unclear. In this study, we used transcriptome data to reveal crosstalk between different tissues in OA. Methods We used four groups of transcription profiles acquired from the Gene Expression Omnibus database, including articular cartilage, meniscus, synovium, and subchondral bon

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Lampiasi, Nadia. "Interactions between Macrophages and Mast Cells in the Female Reproductive System." International Journal of Molecular Sciences 23, no. 10 (2022): 5414. http://dx.doi.org/10.3390/ijms23105414.

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Mast cells (MCs) and macrophages (Mϕs) are innate immune cells that differentiate from early common myeloid precursors and reside in all body tissues. MCs have a unique capacity to neutralize/degrade toxic proteins, and they are hypothesized as being able to adopt two alternative polarization profiles, similar to Mϕs, with distinct or even opposite roles. Mϕs are very plastic phagocytic cells that are devoted to the elimination of senescent/anomalous endogenous entities (to maintain tissue homeostasis), and to the recognition and elimination of exogenous threats. They can adopt several functio

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Vianello, Elena, Marta Kalousová, Elena Dozio, Lorenza Tacchini, Tomáš Zima, and Massimiliano Marco Corsi Romanelli. "Osteopontin: The Molecular Bridge between Fat and Cardiac–Renal Disorders." International Journal of Molecular Sciences 21, no. 15 (2020): 5568. http://dx.doi.org/10.3390/ijms21155568.

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Osteopontin (OPN) is a multifaceted matricellular protein, with well-recognized roles in both the physiological and pathological processes in the body. OPN is expressed in the main organs and cell types, in which it induces different biological actions. During physiological conditioning, OPN acts as both an intracellular protein and soluble excreted cytokine, regulating tissue remodeling and immune-infiltrate in adipose tissue the heart and the kidney. In contrast, the increased expression of OPN has been correlated with the severity of the cardiovascular and renal outcomes associated with obe

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