Log in

Relevant bibliographies by topics / Interferon alpha-beta

Contents

Journal articles
Dissertations / Theses
Books
Book chapters
Conference papers

Academic literature on the topic 'Interferon alpha-beta'

Author: Grafiati

Published: 7 June 2025

Last updated: 2 August 2025

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Interferon alpha-beta.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Interferon alpha-beta"

1

&NA;. "Interferon alpha/interferon beta." Reactions Weekly &NA;, no. 500 (1994): 7. http://dx.doi.org/10.2165/00128415-199405000-00030.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

&NA;. "Interferon alpha 2A/interferon beta." Reactions Weekly &NA;, no. 432 (1992): 11. http://dx.doi.org/10.2165/00128415-199204320-00061.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Hamilton, A. O., L. Jones, L. Morrison, and K. Whaley. "Modulation of monocyte complement synthesis by interferons." Biochemical Journal 242, no. 3 (1987): 809–15. http://dx.doi.org/10.1042/bj2420809.

Full text

Abstract:

Recombinant Escherichia coli-derived gamma-interferon has been shown to stimulate synthesis of the second component of complement (C2), factor B and C1 inhibitor, but to inhibit synthesis of the third component (C3). alpha- and beta-interferons stimulate synthesis of factor B and C3 inhibitor, inhibit C5 synthesis but do not alter synthesis of C2. alpha- and beta-interferons act synergistically with gamma-interferon to enhance both factor B and C1-inhibitor synthesis.

APA, Harvard, Vancouver, ISO, and other styles

4

Pace, J. L., S. W. Russell, P. A. LeBlanc, and D. M. Murasko. "Comparative effects of various classes of mouse interferons on macrophage activation for tumor cell killing." Journal of Immunology 134, no. 2 (1985): 977–81. http://dx.doi.org/10.4049/jimmunol.134.2.977.

Full text

Abstract:

Abstract The effects of mouse interferon-alpha (MuIFN-alpha), -beta (MuIFN-beta), and -gamma (MuIFN-gamma) on macrophage activation for tumor cell killing were determined by using proteose peptone-elicited peritoneal macrophages from C3H/HeN and C3H/HeJ mice under conditions that either included or were free of detectable endotoxin. Alone, under the conditions used, none of the interferons was able to activate macrophages directly for tumor cell killing. However, with a second signal provided to responsive macrophages by contaminating endotoxin, added bacterial lipopolysaccharide (LPS), or hea

APA, Harvard, Vancouver, ISO, and other styles

5

Daly, C., and N. C. Reich. "Double-stranded RNA activates novel factors that bind to the interferon-stimulated response element." Molecular and Cellular Biology 13, no. 6 (1993): 3756–64. http://dx.doi.org/10.1128/mcb.13.6.3756-3764.1993.

Full text

Abstract:

Infection of cells with adenovirus or transfection of cells with double-stranded RNA (dsRNA) activates transcription of the alpha/beta interferon-stimulated genes (ISGs). Induction of ISG expression by adenovirus appears to be mediated through the same DNA target that is responsive to alpha/beta interferons, the interferon-stimulated response element (ISRE). Transcriptional induction by alpha/beta interferons has been shown previously to be mediated by the activation of a latent cytoplasmic transcription factor, ISGF3, that translocates to the nucleus and binds to the ISRE. However, ISG expres

APA, Harvard, Vancouver, ISO, and other styles

6

Daly, C., and N. C. Reich. "Double-stranded RNA activates novel factors that bind to the interferon-stimulated response element." Molecular and Cellular Biology 13, no. 6 (1993): 3756–64. http://dx.doi.org/10.1128/mcb.13.6.3756.

Full text

Abstract:

Infection of cells with adenovirus or transfection of cells with double-stranded RNA (dsRNA) activates transcription of the alpha/beta interferon-stimulated genes (ISGs). Induction of ISG expression by adenovirus appears to be mediated through the same DNA target that is responsive to alpha/beta interferons, the interferon-stimulated response element (ISRE). Transcriptional induction by alpha/beta interferons has been shown previously to be mediated by the activation of a latent cytoplasmic transcription factor, ISGF3, that translocates to the nucleus and binds to the ISRE. However, ISG expres

APA, Harvard, Vancouver, ISO, and other styles

7

Delhaye, Sophie, Vincent van Pesch, and Thomas Michiels. "The Leader Protein of Theiler's Virus Interferes with Nucleocytoplasmic Trafficking of Cellular Proteins." Journal of Virology 78, no. 8 (2004): 4357–62. http://dx.doi.org/10.1128/jvi.78.8.4357-4362.2004.

Full text

Abstract:

ABSTRACT The leader protein of Theiler's virus was previously shown to block the production of alpha/beta interferon by infected cells. Here, we observed that expression of the leader protein in infected cells triggered subcellular redistribution of a nucleus-target green fluorescent protein. It enhanced redistribution of the nuclear polypyrimidine tract-binding protein but had no influence on the localization of the nuclear splicing factor SC-35. The leader protein also interfered with trafficking of the cytoplasmic interferon regulatory factor 3, a factor critical for transcriptional activat

APA, Harvard, Vancouver, ISO, and other styles

8

Shirshev, S. V. "The role of hormonal-cytokine interactions in the formation of humoral immune response." Problems of Endocrinology 41, no. 1 (1995): 32–34. http://dx.doi.org/10.14341/probl11347.

Full text

Abstract:

The functional activity of splenocytes of CBA mice was investigated in a syngeneic transfer system by the level of formation of antibody-producing cells (APC). Splenocytes were preincubated for 1 h in vitro with chorionic gonadotropin and type I recombinant interferons, as well as in hormonal-cytokine combinations. Chorionic gonadotropin in doses 10 and 50 MU/ml depressed APC formation, whereas alpha-interferon (250 MU/ml) stimulated it, and beta-interferon in the same concentration did not influence the level of humoral immune response. Chorionic gonadotropin, if added to splenocyte culture i

APA, Harvard, Vancouver, ISO, and other styles

9

Popescu, L. M., C. Cernescu, I. I. Moraru, et al. "Cell-membrane phospholipase C is involved in inducing the antiviral effect of interferon." Bioscience Reports 9, no. 5 (1989): 531–39. http://dx.doi.org/10.1007/bf01119795.

Full text

Abstract:

A monospecific inhibitory antibody directed to phospholipase C (phosphoinositidase C) blocked the antiviral effect of human interferons alpha and beta when tested on human quiescent fibroblasts challenged with the vesicular stomatitis virus. This action was due to specific inhibition of polyphosphoinositide hydrolysis because (a) the F(ab′)2 fragment of the antibody molecule was also inhibitory; (b) excess antibodies directed to phospholipase A2 and to a phosphatidylcholine-preferring phospholipase C did not have any inhibitory effect, and (c) the combination of 12-O-tetradecanoylphorbol-aceta

APA, Harvard, Vancouver, ISO, and other styles

10

Barbaglia, Matteo Nazzareno, James Michael Harris, Artem Smirnov та ін. "17β-Oestradiol Protects from Hepatitis C Virus Infection through Induction of Type I Interferon". Viruses 14, № 8 (2022): 1806. http://dx.doi.org/10.3390/v14081806.

Full text

Abstract:

Background and Aims: Sex hormones are widely recognised to act as protective factors against several viral infections. Specifically, females infected by the hepatitis C virus display higher clearance rates and reduced disease progression than those found in males. Through modulation of particle release and spread, 17β-oestradiol controls HCV’s life cycle. We investigated the mechanism(s) behind oestrogen’s antiviral effect. Methods: We used cell culture-derived hepatitis C virus in in vitro assays to evaluate the effect of 17β-oestradiol on the innate immune response. Host immune responses wer

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "Interferon alpha-beta"

1

Dandoy, Françoise. "Localisation des gènes d'interféron alpha et beta murins." Paris 6, 1987. http://www.theses.fr/1987PA066326.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Hidmark, Åsa. "Induction of type I interferons and viral immunity /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-227-9/.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Gray, Reginald Courtney. "Regulation of Interferon Alpha Beta Induction and Dendritic Cell Function by CpG Oligodeoxynucleotides." Case Western Reserve University School of Graduate Studies / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=case1186014244.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Alper, Deborah. "Characterization of distinct mechanisms regulating induction of human interferon alpha and beta gene transcription." Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=59825.

Full text

Abstract:

The virus-inducible Type I interferon (IFN) genes serve as relevant models to examine the nature of cis-acting sequences and trans-acting protein factors involved in transcription control. The alpha and beta interferon genes are activated differentially depending on the nature of the inducer and the specific cellular background. A transient expression system was developed to analyze the activation of the interferon promoters in a homologous human cellular background. In epithelial cells, the IFN-$ beta$ but not the IFN-$ alpha$1 promoter was virus responsive with induction kinetics similar to

APA, Harvard, Vancouver, ISO, and other styles

5

Su, Leon L. "Mechanisms of STAT activation via the interferon-[alpha]/[beta] and B cell antigen receptor and immunomodulatory role of interferons on lymphocyte development /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2000. http://wwwlib.umi.com/cr/ucsd/fullcit?p9988315.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Moraga, Gonzalez Ignacio. "Etude des voies de signalisation activées par l'interféron alpha 2 et l'interféron beta conduisant à des activités différentielles." Paris 6, 2009. http://www.theses.fr/2009PA066597.

Full text

Abstract:

Le génome humain encode 3 familles d’interféron : les interférons de type I (IFN/), de type II (IFN), de type III (). Chez l’homme, les interférons de type I sont divisés en 16 sous-types très conservés, nommé IFN/. Ces cytokines se lient au récepteur à l’interféron de type I constitué de deux sous-unités IFNAR1 et IFNAR2 induisant la même voie de signalisation Jak/Stat. Cependant le mécanisme moléculaire par lequel différents interférons agissent avec des activités biologiques différentes reste inconnu. La première partie de mon travail est axée sur l’impact de la densité des récepteurs

APA, Harvard, Vancouver, ISO, and other styles

7

Liljeqvist, Maria. "Molecular characterization of the interaction between Tick-borne encephalitis virus NS5 protein and the Interferon alpha/beta and gamma receptors." Thesis, Södertörn University College, School of Life Sciences, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:sh:diva-1274.

Full text

Abstract:

<p>Flaviviruses, family flaviviridae, are often associated with severe diseases and many of the viruses have been shown to affect the immune response. Interferons confer important ways of defending the host against viral infections because they provide a link between the innate and the adaptive immunity. Langat virus (LGTV), belonging to the Tick-Borne Encephalitis (TBE) complex of viruses, has recently been shown to provide interactions with interferon receptors and inhibit the interferon-mediated response. Similarly, it has been demonstrated that the TBE virus NS5 protein affects type 1 inte

APA, Harvard, Vancouver, ISO, and other styles

8

Perroud, Ana Paula de Almeida Salles. "Estudo da expressão de citocinas e componentes de apoptose no tumor de Walker 256, comparação entre a linhagem A e variant Ar." [s.n.], 2005. http://repositorio.unicamp.br/jspui/handle/REPOSIP/311776.

Full text

Abstract:

Orientador: Ricardo de Lima Zollner<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas<br>Made available in DSpace on 2018-08-09T02:29:32Z (GMT). No. of bitstreams: 1 Perroud_AnaPauladeAlmeidaSalles_M.pdf: 4222371 bytes, checksum: 5ff171cf192f89e5602e731098f85f66 (MD5) Previous issue date: 2005<br>Resumo: Duas variantes do Walker 256 foram descritas como Walker 256 A e AR. A variante A é mais agressiva e pode induzir os efeitos sistêmicos como anorexia, retenção de sódio, líguido de perda de peso e morte. Os mecanismos envolvidos na regressão e progr

APA, Harvard, Vancouver, ISO, and other styles

9

Gartenschläger, Sören. "Untersuchung der Interleukin-5-Rezeptor-Expression und der Aktivierung humaner eosinophiler Granulozyten Einfluss von Interferon [gamma], transforming growth factor [beta]1 sowie der hämatopoetischen Zytokine IL-3, IL-5 und GM-CSF auf den Aktivierungsmarker CD69 sowie auf die Interleukin-5-Rezeptor-[alpha]-Expression humaner eosinophiler Granulozyten /." [S.l.] : [s.n.], 1999. http://deposit.ddb.de/cgi-bin/dokserv?idn=963698958.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

VELAZQUEZ, MEDINA LAURA E. "Identification et etude fonctionnelle de tyk2, une tyrosine kinase impliquee dans la reponse cellulaire aux interferons alpha/beta." Paris 7, 1994. http://www.theses.fr/1994PA077312.

Full text

Abstract:

Une approche genetique a permis l'isolement de mutants de reponse a l'interferon (ifn) alpha/beta. Le mutant 11,1 est resistant aux ifns alpha tout en conservant une faible reponse a l'ifn beta. Il presente egalement une activite de liaison a l'ifn alpha2 fortement reduite. A l'aide de techniques d'adn recombinant et d'un systeme de contre-selection puissant des revertants, il a ete possible de cloner le gene et l'adnc humain corrigeant le phenotype des cellules mutantes. Ce gene code pour la proteine tyk2 appartenant a la sous-famille de ptk non recepteur nommee jak, possedant deux domaines c

APA, Harvard, Vancouver, ISO, and other styles

More sources

Books on the topic "Interferon alpha-beta"

1

Artursson, Karen. Studies on the interferon-alpha/beta system of pigs. Sveriges Lantbruksuniversitet, 1993.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

Sandberg, Kristian. Characterization of the interferon-alpha/beta producing leukocytes and their responses to viral inducers. Sveriges Lantbruksuniversitet, 1991.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Kaisers, Wolfgang. Untersuchung zur Änderung der Expressionstärke der Histokompatibilitätsantigene Beta-2-Mikroglobulin und HLA-DR, sowie des Adhäsionsmoleküls ICAM-1 auf Zellinien des Nierenzellkarzinoms durch die Zytokine Interleukin-2, Interferon-[alpha], Interferon-[gamma] und Tumor-Nekrose-Faktor-[alpha]. [s.n.], 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

Koenigsberg, Adam David. Alpha/Beta and Gamma interferon responses during reovirus-mediated chemoimmunotherapy of L1210 leukemia. 1988.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

5

Lindenthal, Andreas. Bestimmung der Zytokinproduktion in Blutzellkulturen von Patienten während einer Therapie mit 5-Fluoruracil, Interleukin-1-[beta] , Interleukin-2 und einer Kombination mit Interferon-[alpha] und Interleukin-2. 1995.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

6

Knoll, Richard Mathias. Untersuchung zur Regulation der Interleukin - 13 - Rezeptor - alpha1 - Expression auf eosinophilen Granulozyten: Einfluss von Interferon -[gamma], Transforming - Growth Factor - [Beta₁], Tumomekrosefaktor - [Alpha], IL -4, IL -13 sowie der Hämatopoetischen Zytokine IL -3, IL -5 und GM-CSF auf den IL-13R[alpha]1 humaner eosinophiler Granulozyten. 2004.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

7

Nielsen, David A., Dmitri Proudnikov, and Mary Jeanne Kreek. The Genetics of Impulsivity. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0080.

Full text

Abstract:

Impulsivity is a complex trait that varies across healthy individuals, although when excessive, it is generally regarded as dysfunctional. Impulsive behavior may lead to initiation of drug addiction that interferes with inhibitory controls, which may in turn result in facilitation of the individual’s impulsive acts. Although environmental factors play a considerable role in impulsive behavior, a body of evidence collected in twin studies suggests that about 45% of the variance in impulsivity is accounted for by genetic factors. Genetic variants studied in association with impulsivity include t

APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Interferon alpha-beta"

1

Meager, Anthony. "Cell-Based Assays for the Detection of Neutralizing Antibodies to Interferon Beta (IFN-β) and Tumor Necrosis Factor Alpha (TNF-α) Inhibitors." In Detection and Quantification of Antibodies to Biopharmaceuticals. John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118075685.ch8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Uze, G., G. Lutfalla, and K. E. Mogensen. "Receptors for the Alpha and Beta Interferon (IFN) Family." In Guidebook to Cytokines and Their Receptors. Oxford University PressOxford, 1995. http://dx.doi.org/10.1093/oso/9780198599470.003.0033.

Full text

Abstract:

Abstract Receptors are generally found on all human cells whatever their origin, even on cells poorly responsive to interferon. Cell lines showing no binding at all of one or other of the family are relatively rare or deliberate constructs. Binding studies with radiolabelled ligand have been reported for the following human recombinant interferon: alphas, 1(D), 2 (A), 8(8), 4 and for beta interferon. Competitive binding experiments with unlabelled ligand against a given radiolabelled interferon have been reported for a wider range of alpha interferons. Taking account of differences in specific

APA, Harvard, Vancouver, ISO, and other styles

3

lzotova, Lara, and Sidney Pestka. "Receptor binding studies." In Cytokine Molecular Biology. Oxford University PressOxford, 2000. http://dx.doi.org/10.1093/oso/9780199638581.003.0005.

Full text

Abstract:

Abstract The interferons (IFNs) are a family of proteins which elicit a multitude of cellular responses including antiviral, antiproliferative, and immunoregulatory activities. There are four classes of interferons that have been isolated and characterized (1-11). These are the leucocyte or interferon alpha (IFN-a), fibroblast or interferon beta (IFN-13), immune or interferon gamma (IFN-y), and interferon omega (IFN-w). Binding to a specific cell-surface receptor is a necessary, but not sufficient, condition for cellular activation in the case of the IFN-y system (12-16). IFN-a, IFN-13, and IF

APA, Harvard, Vancouver, ISO, and other styles

4

Prochwicz, Anna, and Dorota Krochmalczyk. "Impact of Interferon Alpha/Beta in the Management of Chronic Myeloproliferative Disorders." In Interferon - Immune Metabolism [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.104501.

Full text

Abstract:

It has been noted that interferon can exert an antiproliferative effect by stimulating cells of the immune system. Interferon has been shown to be effective in the treatment of chronic myeloproliferative neoplasms. Over the years, interferon alpha-2a and interferon alpha-2b have been introduced into the treatment of chronic myeloproliferation, followed by their pegylated forms. Studies have been showing the effectiveness of interferon alpha in reducing the number of platelets in essential thrombocythemia, reducing the need for phlebotomies in patients with polycythemia vera and also in reducin

APA, Harvard, Vancouver, ISO, and other styles

5

Meager, Anthony. "Interferons Alpha, Beta, and Omega." In Cytokines. Elsevier, 1998. http://dx.doi.org/10.1016/b978-012498340-3/50026-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Interferon alpha-beta"

1

Maduemem, KE, and A. Dumitrescu. "G56 Novel primary immunodeficiency?: cases of interferon-alpha/beta receptor 2 deficiency." In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 13–15 March 2018, SEC, Glasgow, Children First – Ethics, Morality and Advocacy in Childhood, The Journal of the Royal College of Paediatrics and Child Health. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/archdischild-2018-rcpch.54.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Perez Gandara, B., J. L. Perez Perez, H. A. Goldenberg, et al. "Nicotine in e-Cigarettes Dysregulates Interferon Beta, Tumor Necrosis Factor Alpha, and Matrix Metalloproteinase 12 Expression, Without Effecting Respiratory Syncytial Virus Virulence in Mice." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3104.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!