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Dissertations / Theses on the topic 'Interferon alpha-beta'

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Published: 7 June 2025
Last updated: 2 August 2025

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1

Dandoy, Françoise. "Localisation des gènes d'interféron alpha et beta murins." Paris 6, 1987. http://www.theses.fr/1987PA066326.

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2

Hidmark, Åsa. "Induction of type I interferons and viral immunity /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-227-9/.

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3

Gray, Reginald Courtney. "Regulation of Interferon Alpha Beta Induction and Dendritic Cell Function by CpG Oligodeoxynucleotides." Case Western Reserve University School of Graduate Studies / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=case1186014244.

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4

Alper, Deborah. "Characterization of distinct mechanisms regulating induction of human interferon alpha and beta gene transcription." Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=59825.

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The virus-inducible Type I interferon (IFN) genes serve as relevant models to examine the nature of cis-acting sequences and trans-acting protein factors involved in transcription control. The alpha and beta interferon genes are activated differentially depending on the nature of the inducer and the specific cellular background. A transient expression system was developed to analyze the activation of the interferon promoters in a homologous human cellular background. In epithelial cells, the IFN-$ beta$ but not the IFN-$ alpha$1 promoter was virus responsive with induction kinetics similar to
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5

Su, Leon L. "Mechanisms of STAT activation via the interferon-[alpha]/[beta] and B cell antigen receptor and immunomodulatory role of interferons on lymphocyte development /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2000. http://wwwlib.umi.com/cr/ucsd/fullcit?p9988315.

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6

Moraga, Gonzalez Ignacio. "Etude des voies de signalisation activées par l'interféron alpha 2 et l'interféron beta conduisant à des activités différentielles." Paris 6, 2009. http://www.theses.fr/2009PA066597.

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Le génome humain encode 3 familles d’interféron : les interférons de type I (IFN/), de type II (IFN), de type III (). Chez l’homme, les interférons de type I sont divisés en 16 sous-types très conservés, nommé IFN/. Ces cytokines se lient au récepteur à l’interféron de type I constitué de deux sous-unités IFNAR1 et IFNAR2 induisant la même voie de signalisation Jak/Stat. Cependant le mécanisme moléculaire par lequel différents interférons agissent avec des activités biologiques différentes reste inconnu. La première partie de mon travail est axée sur l’impact de la densité des récepteurs
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7

Liljeqvist, Maria. "Molecular characterization of the interaction between Tick-borne encephalitis virus NS5 protein and the Interferon alpha/beta and gamma receptors." Thesis, Södertörn University College, School of Life Sciences, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:sh:diva-1274.

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<p>Flaviviruses, family flaviviridae, are often associated with severe diseases and many of the viruses have been shown to affect the immune response. Interferons confer important ways of defending the host against viral infections because they provide a link between the innate and the adaptive immunity. Langat virus (LGTV), belonging to the Tick-Borne Encephalitis (TBE) complex of viruses, has recently been shown to provide interactions with interferon receptors and inhibit the interferon-mediated response. Similarly, it has been demonstrated that the TBE virus NS5 protein affects type 1 inte
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8

Perroud, Ana Paula de Almeida Salles. "Estudo da expressão de citocinas e componentes de apoptose no tumor de Walker 256, comparação entre a linhagem A e variant Ar." [s.n.], 2005. http://repositorio.unicamp.br/jspui/handle/REPOSIP/311776.

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Orientador: Ricardo de Lima Zollner<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas<br>Made available in DSpace on 2018-08-09T02:29:32Z (GMT). No. of bitstreams: 1 Perroud_AnaPauladeAlmeidaSalles_M.pdf: 4222371 bytes, checksum: 5ff171cf192f89e5602e731098f85f66 (MD5) Previous issue date: 2005<br>Resumo: Duas variantes do Walker 256 foram descritas como Walker 256 A e AR. A variante A é mais agressiva e pode induzir os efeitos sistêmicos como anorexia, retenção de sódio, líguido de perda de peso e morte. Os mecanismos envolvidos na regressão e progr
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9

Gartenschläger, Sören. "Untersuchung der Interleukin-5-Rezeptor-Expression und der Aktivierung humaner eosinophiler Granulozyten Einfluss von Interferon [gamma], transforming growth factor [beta]1 sowie der hämatopoetischen Zytokine IL-3, IL-5 und GM-CSF auf den Aktivierungsmarker CD69 sowie auf die Interleukin-5-Rezeptor-[alpha]-Expression humaner eosinophiler Granulozyten /." [S.l.] : [s.n.], 1999. http://deposit.ddb.de/cgi-bin/dokserv?idn=963698958.

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10

VELAZQUEZ, MEDINA LAURA E. "Identification et etude fonctionnelle de tyk2, une tyrosine kinase impliquee dans la reponse cellulaire aux interferons alpha/beta." Paris 7, 1994. http://www.theses.fr/1994PA077312.

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Une approche genetique a permis l'isolement de mutants de reponse a l'interferon (ifn) alpha/beta. Le mutant 11,1 est resistant aux ifns alpha tout en conservant une faible reponse a l'ifn beta. Il presente egalement une activite de liaison a l'ifn alpha2 fortement reduite. A l'aide de techniques d'adn recombinant et d'un systeme de contre-selection puissant des revertants, il a ete possible de cloner le gene et l'adnc humain corrigeant le phenotype des cellules mutantes. Ce gene code pour la proteine tyk2 appartenant a la sous-famille de ptk non recepteur nommee jak, possedant deux domaines c
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11

Vodjdani, Guilane. "Etude des interferons alpha et beta murins : caracterisation et analyse des genes, induction et regulation de leur expression dans des cellules eucaryotes." Paris 6, 1988. http://www.theses.fr/1988PA066595.

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Caracterisation et expression des genes alpha 7 et alpha 8 d'interferon et du gene de l'interferon beta de souris etudie dans les cellules c243-2 apres induction par un virus, realisation de genes hybride en vue d'en etudier la regulation
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12

Vodjdani, Guilane. "Etude des interférons alpha et beta murins caractérisation et analyse des gènes, induction et régulation de leur expression dans des cellules euracyotes /." Grenoble 2 : ANRT, 1988. http://catalogue.bnf.fr/ark:/12148/cb376191737.

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13

LEISSNER, PHILIPPE. "Therapie genique du sida par transfert retroviral des genes codant pour les interferons alpha, beta et gamma humains dans les cellules cibles du vih-1." Université Louis Pasteur (Strasbourg) (1971-2008), 1995. http://www.theses.fr/1995STR13205.

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Dans le cas du sida, la therapie genique somatique consiste a introduire dans les cellules cibles du vih-1 ou dans leurs cellules souches, un gene therapeutique conferant a celles-ci une resistance contre l'infection et/ou la propagation virale. Sur la base de ce principe, nous avons evalue le role des interferons alpha, beta et gamma humains places sous le controle des propres sequences de regulation du virus, dans une therapie genique anti-vih. La transduction retrovirale des genes codant pour les interferons alpha, beta et gamma humains sous le controle des sequences regulatrices du vih-1,
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14

LOCATELLI, LUIGI. "Expression of aVB6 integrin by Pkhd1-defective cholangiocytes links enhanced ductal secretion of Macrophage chemokines to progressive portal fibrosis in Congenital Hepatic Fibrosis." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2013. http://hdl.handle.net/10281/41733.

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BACKGROUND AND AIMS: Congenital Hepatic Fibrosis (CHF) is caused by mutations in PKHD1, a gene encoding for fibrocystin, a protein of unknown function, expressed in cholangiocyte cilia and centromers. In CHF, biliary dysgenesis is accompanied by severe progressive portal fibrosis and portal hypertension. The mechanisms responsible for portal fibrosis in CHF are unclear. The αvβ6 integrin mediates local activation of TGFβ1 and is expressed by reactive cholangiocytes during cholestasis. To understand the mechanisms of fibrosis in CHF we studied the expression of αvβ6 integrin and its regulation
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15

Jia, Danlin. "Influenza Virus H5N1 Non-structural Protein 1 Alters Interferon-alpha/beta Signaling." Thesis, 2009. http://hdl.handle.net/1807/18772.

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Type I interferons (IFNs) function as the first line of defense against viral infections by modulating numerous biological processes to establish an antiviral state and influencing the activation of various immune cells. During influenza A infection, the NS1 encoded by the virus genome disrupts many cellular processes to block type I IFN responses. We show that expression of H5N1 NS1 in HeLa cells reduces IFN-inducible activation of STAT proteins and its subsequent binding to DNA complexes. Subsequent analysis suggests NS1 blocks IFN signaling by inhibiting expression of type I IFN receptor su
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