Relevant bibliographies by topics / Interferons (IFNs)

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers
  5. Reports

Academic literature on the topic 'Interferons (IFNs)'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 September 2023

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Journal articles on the topic "Interferons (IFNs)"

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Bruening, Janina, Bettina Weigel, and Gisa Gerold. "The Role of Type III Interferons in Hepatitis C Virus Infection and Therapy." Journal of Immunology Research 2017 (2017): 1–12. http://dx.doi.org/10.1155/2017/7232361.

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The human interferon (IFN) response is a key innate immune mechanism to fight virus infection. IFNs are host-encoded secreted proteins, which induce IFN-stimulated genes (ISGs) with antiviral properties. Among the three classes of IFNs, type III IFNs, also called IFN lambdas (IFNLs), are an essential component of the innate immune response to hepatitis C virus (HCV). In particular, human polymorphisms in IFNL gene loci correlate with hepatitis C disease progression and with treatment response. To date, the underlying mechanisms remain mostly elusive; however it seems clear that viral infection
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Abdolvahab, Mohadeseh Haji, Behrad Darvishi, Mohammad Zarei, Keivan Majidzadeh-A, and Leila Farahmand. "Interferons: role in cancer therapy." Immunotherapy 12, no. 11 (2020): 833–55. http://dx.doi.org/10.2217/imt-2019-0217.

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Interferons (IFNs) are a group of signaling cytokines, secreted by host cells to induce protection against various disorders. IFNs can directly impact on tumor cells or indirectly induce the immune system to protect host cells. The expression levels of IFNs and its functions of are excellently modulated in a way to protect host cells from probable toxicities caused by extreme responses. The efficacy of anticancer therapies is correlated to IFNs signaling. Although IFN signaling is involved in induction of antitumor responses, chronic stimulation of the IFN signaling pathway can induce resistan
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Tilg, Herbert, and Arthur Kaser. "Interferons and Their Role in Inflammation." Current Pharmaceutical Design 5, no. 10 (1999): 771–85. http://dx.doi.org/10.2174/1381612805666230111210939.

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Cytokines are pleiotropic molecules showing a wide variety of biologic functions on various cells and tissues, and several different cytokines exert similar and overlapping functions on certain cells. Interferons (IFNs), among the first cytokines identified, play a crucial role in human disease. The IFN cytokine family consists of type I IFNs (IFN-a and IFN- ) and type II IFN (IFN-y). In the first decades of IFN research, type I IFNs were considered primarily as viral inhibitors, whereas type II IFN, also termed "immune IFN", was generally considered to be uniquely involved in immune reactions
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Manry, Jérémy, Guillaume Laval, Etienne Patin, et al. "Evolutionary genetic dissection of human interferons." Journal of Experimental Medicine 208, no. 13 (2011): 2747–59. http://dx.doi.org/10.1084/jem.20111680.

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Interferons (IFNs) are cytokines that play a key role in innate and adaptive immune responses. Despite the large number of immunological studies of these molecules, the relative contributions of the numerous IFNs to human survival remain largely unknown. Here, we evaluated the extent to which natural selection has targeted the human IFNs and their receptors, to provide insight into the mechanisms that govern host defense in the natural setting. We found that some IFN-α subtypes, such as IFN-α6, IFN-α8, IFN-α13, and IFN-α14, as well as the type II IFN-γ, have evolved under strong purifying sele
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Samuel, Charles E. "Antiviral Actions of Interferons." Clinical Microbiology Reviews 14, no. 4 (2001): 778–809. http://dx.doi.org/10.1128/cmr.14.4.778-809.2001.

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SUMMARY Tremendous progress has been made in understanding the molecular basis of the antiviral actions of interferons (IFNs), as well as strategies evolved by viruses to antagonize the actions of IFNs. Furthermore, advances made while elucidating the IFN system have contributed significantly to our understanding in multiple areas of virology and molecular cell biology, ranging from pathways of signal transduction to the biochemical mechanisms of transcriptional and translational control to the molecular basis of viral pathogenesis. IFNs are approved therapeutics and have moved from the basic
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Kimura, M., J. A. Majde, L. A. Toth, M. R. Opp, and J. M. Krueger. "Somnogenic effects of rabbit and recombinant human interferons in rabbits." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 267, no. 1 (1994): R53—R61. http://dx.doi.org/10.1152/ajpregu.1994.267.1.r53.

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Interferons (IFNs) are antiviral cytokines that possess several central nervous system activities. IFN therapy is associated with sleepiness, and the IFNs expressed during viral infection may be involved in the excess sleep associated with these infections. Most viruses stimulate the production of both IFN-alpha and IFN-beta. Although large doses of human IFN-alpha 2 are somnogenic in rabbits, the effects of species-specific IFNs on sleep in the rabbit have not been documented. We compared the somnogenic and antiviral effects of IFNs derived from rabbits to the effects of recombinant human (rh
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Asselin-Paturel, Carine, and Giorgio Trinchieri. "Production of type I interferons." Journal of Experimental Medicine 202, no. 4 (2005): 461–65. http://dx.doi.org/10.1084/jem.20051395.

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Plasmacytoid dendritic cells (pDCs) are specialized producers of type I interferons (IFNs) that respond to most viruses. Because of their antiviral activity and regulatory functions in innate and adaptive immunity, type I IFNs are important not only for antiviral resistance but also in other types of infections and in immune pathology. Here we discuss recent data that begin to reveal the unique molecular mechanisms underlying the remarkably rapid and efficient type I IFN production by pDCs.
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Sinkovics, Joseph G. "Interferons: Antiangiogenesis Agents (A Reasonable Theory)." Canadian Journal of Infectious Diseases 3, suppl b (1992): 128–32. http://dx.doi.org/10.1155/1992/754560.

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Antiviral and immunomodulatory effects of interferons (IFNs) are well-known. This communication lists arguments for explicit antiangiogenetic effects exerted by interferons. There is strong likelihood that IFNs α, β and γ inhibit the int family of genes whose gene product proteins are the acidic and basic fibroblast growth factors. Some of these growth factors promote the growth of vascular endothelial cells. IFN-α inhibits basic, and IFN-γ inhibits acidic fibroblast growth factors. Inhibition of Kaposi sarcoma cell growth is not due to immunological reconstitution of the host. As IFN-α induce
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Mundra, Akaash, Aram Yegiazaryan, Haig Karsian, et al. "Pathogenicity of Type I Interferons in Mycobacterium tuberculosis." International Journal of Molecular Sciences 24, no. 4 (2023): 3919. http://dx.doi.org/10.3390/ijms24043919.

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Tuberculosis (TB) is a leading cause of mortality due to infectious disease and rates have increased during the emergence of COVID-19, but many of the factors determining disease severity and progression remain unclear. Type I Interferons (IFNs) have diverse effector functions that regulate innate and adaptive immunity during infection with microorganisms. There is well-documented literature on type I IFNs providing host defense against viruses; however, in this review, we explore the growing body of work that indicates high levels of type I IFNs can have detrimental effects to a host fighting
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Plata-Salaman, C. R. "Interferons and central regulation of feeding." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 263, no. 6 (1992): R1222—R1227. http://dx.doi.org/10.1152/ajpregu.1992.263.6.r1222.

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Interferons (IFNs) are immunomodulators with neuromodulatory activities. To study the effects of IFNs on the central regulation of feeding, rats were subjected to various applications. The results show the following. 1) Intracerebroventricular microinfusion of rat IFN (15-225 IU/rat) decreased short-term (2-h) food intake in rats. Computerized analysis of behavioral patterns demonstrated a reduction of meal size and meal duration, whereas meal frequency slightly increased. Nighttime and total daily food intakes were not significantly affected. 2) Short-term food intake suppression by intracere
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Dissertations / Theses on the topic "Interferons (IFNs)"

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Liu, Qinfang. "Interaction of type I interferons and mTOR signaling underlying PRRSV infection." Thesis, Kansas State University, 2016. http://hdl.handle.net/2097/32860.

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Master of Science in Biomedical Sciences<br>Department of Anatomy and Physiology<br>Yongming Sang<br>Animal metabolic and immune systems integrate and inter-regulate to exert effective immune responses to distinct pathogens. The signaling pathway mediated by mechanistic target of rapamycin (mTOR) is critical in cellular metabolism and implicated in host antiviral responses. Recent studies highlight the significance of the mTOR signaling pathway in the interferon (IFN) response. Type I IFNs mediate host defense, particularly, against viral infections, and have myriad roles in antiviral innate a
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Sales, Paula Cristiane Motta. "Regulação da expressão da proteína cinase PKR na ausência de interferons (IFNs) e seu papel na resposta celular mediada por agonistas dos receptores TLR2 e TLR4." Universidade Federal de Minas Gerais, 2012. http://hdl.handle.net/1843/BUOS-9L7NEM.

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The role of protein kinase R (PKR) in the antiviral cell state induced by interferons (IFNs) is well known. The increase in its expression in response to stimulation by IFNs results in its autophosphorylation and phosphorylation of its substrate eIF2-alpha. This molecular event interferes with translation initiation of mRNA, which in turn results in inhibition of protein synthesis. Recent studies suggest the involvement of PKR in bacterial infections. However, how PKR is activated and what is its biological role in these infections is still largely unexplored. Thus, the central objective of ou
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Chen, Shu. "Studies on interferon (IFN) induction and isolation of IFN-inducing mutant viruses." Thesis, University of St Andrews, 2011. http://hdl.handle.net/10023/1678.

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The interferon (IFN) system is a powerful antiviral defense system. Host cell pattern recognition receptors (PRRs) recognise pathogen-associated molecule patterns (PAMPs) which when activated, lead to the transcription of the IFN-β gene. As a consequence IFN is secreted from the cell and activates the JAK-STAT pathway to up-regulate the transcription of IFN-stimulated genes (ISGs). The products of many ISGs inhibit viral replication and cell proliferation. Viruses encode IFN antagonists that dampen down the IFN response, making it less effective. However, within a virus population, there are a
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Walker, Angela Marie Roberts R. M. "The type I IFN of Bos taurus." Diss., Columbia, Mo. : University of Missouri-Columbia, 2008. http://hdl.handle.net/10355/6864.

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The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from PDF of title page (University of Missouri--Columbia, viewed on April 1, 2010). Vita. Thesis advisor: R. Michaels Roberts. "May 2008" Includes bibliographical references
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Esteve-Solé, Ana. "Primary and secondary immunodeficiencies of the IL-12/IFN-γ axis". Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/663924.

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IL-12/IFN-γ axis is a principal pathway for intramacrophagic pathogens immunity such as leishmania or mycobacteria. Alterations in this axis, being both congenic (causing the primary immunodeficiency Mendelian Susceptibility to Mycobacterial Disease, MSMD) or acquired (treatment with anti-TNF-α drugs) cause susceptibility to this type of microbes. MSMD causes susceptibility mainly to non-pathogenic mycobacteria, salmonella and candida; besides, MSMD- causing mutations have been detected in Mycobacterium tuberculosis and Leishmania patients. On the other hand, the death of an anti-TNF-α in-uter
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Killip, Marian J. "RNA virus modulation of IFN, PI3K and apoptosis." Thesis, St Andrews, 2009. http://hdl.handle.net/10023/777.

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Silva, Cláudia Maria de Melo. "Polimorfismos genéticos e associação com a produção de Interferon gama (IFN-y) em pacientes com Tuberculose pulmonar." Universidade Federal do Amazonas, 2014. http://tede.ufam.edu.br/handle/tede/2580.

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Made available in DSpace on 2015-04-11T13:54:31Z (GMT). No. of bitstreams: 1 Claudia Maria de Melo Silva.pdf: 2215806 bytes, checksum: c9afeecd5c357af061e1b38a8a31df56 (MD5) Previous issue date: 2014-04-28<br>FAPEAM - Fundação de Amparo à Pesquisa do Estado do Amazonas<br>Tuberculosis (TB) is a chronic infection caused by Mycobacterium tuberculosis complex and remains a major worldwide public health problem, leading to almost 1.45 million deaths annually. The state of Amazonas has a high rate incidence of TB, about 68.3/100,000 inhabitants in 2012. Only 5 to 10% of infected individuals dev
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Zurney, Jennifer Michelle. "Cardiac Cell Type-Specific Differences in the Interferon (IFN) Response, and Reovirus Repression of IFN." NCSU, 2008. http://www.lib.ncsu.edu/theses/available/etd-06302008-141954/.

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Viral myocarditis is an important human disease associated with a wide variety of viruses. The cardiac damage and inflammation associated with viral myocarditis can be immune mediated and/or due to direct cytopathic effect. Reovirus-induced myocarditis reflects direct virus-mediated apoptosis of cardiac cells, providing an excellent model to study direct cytopathic effect in the heart. Previous work has found interferon-beta (IFN) to be an important determinant for protection against viral myocarditis. Importantly, IFN signals through the Jak-STAT pathway to induce the expression of interferon
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SANTOS, Joelma Carvalho. "Análise de polimorfismos de único nucleotíldeo (SNP) e expressão dos genes das citocinas IFN-a1, IFN-y e do receptor IFN-a r1 em relação à quantificação da carga viral em pacientes com hepatite B." Universidade Federal de Pernambuco, 2014. https://repositorio.ufpe.br/handle/123456789/11489.

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Submitted by Marcelo Andrade Silva (marcelo.andradesilva@ufpe.br) on 2015-03-09T14:28:37Z No. of bitstreams: 2 DISSERTAÇÃO Joelma Carvalho Santos.pdf: 2064030 bytes, checksum: 9d9c233bec668a94294f6d19471cb844 (MD5) license_rdf: 1232 bytes, checksum: 66e71c371cc565284e70f40736c94386 (MD5)<br>Made available in DSpace on 2015-03-09T14:28:37Z (GMT). No. of bitstreams: 2 DISSERTAÇÃO Joelma Carvalho Santos.pdf: 2064030 bytes, checksum: 9d9c233bec668a94294f6d19471cb844 (MD5) license_rdf: 1232 bytes, checksum: 66e71c371cc565284e70f40736c94386 (MD5) Previous issue date: 2014<br>A presença de po
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Wang, Ling, and Shunbin Ning. ""Toll-Free" Pathways for Production of Type I Interferons." Digital Commons @ East Tennessee State University, 2017. https://dc.etsu.edu/etsu-works/6540.

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Pathogen-associated molecular patterns (PAMPs) and damage-associated molecular patterns (DAMPs) are recognized by different cellular pathogen recognition receptors (PRRs), which are expressed on cell membrane or in the cytoplasm of cells of the innate immune system. Nucleic acids derived from pathogens or from certain cellular conditions represent a large category of PAMPs/DAMPs that trigger production of type I interferons (IFN-I) in addition to pro-inflammatory cytokines, by specifically binding to intracellular Toll-like receptors or cytosolic receptors. These cytosolic receptors, which are
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Book chapters on the topic "Interferons (IFNs)"

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Nagarajan, Uma M. "Induction and Function of Type I IFNs During Chlamydial Infection." In Bacterial Activation of Type I Interferons. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09498-4_9.

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Galani, Ioanna E., Ourania Koltsida, and Evangelos Andreakos. "Type III interferons (IFNs): Emerging Master Regulators of Immunity." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15774-0_1.

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Stamatiadis-Smidt, Hilke, and Harald zur Hausen. "Interferone (IFN)." In Thema Krebs. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-10418-7_41.

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Obert, Hans-Joachim, and Dieter Pöhlau. "Dosierungsschemata für IFN-β." In Beta-Interferon. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-59764-0_4.

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Pfeffer, Lawrence M., David B. Donner, Tsutomu Arakawa, Nowell Stebbing, and Igor Tamm. "Early Events in the Interaction of IFN-α with IFN-Sensitive and IFN-Resistant Daudi Cells." In The Biology of the Interferon System 1986. Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3543-3_15.

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Parker, Dane. "Activation of Type I IFN Signaling by Staphylococcus aureus." In Bacterial Activation of Type I Interferons. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09498-4_5.

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Naujoks, Jan, and Bastian Opitz. "Innate Immune and Type I IFN Responses During Legionella pneumophila Infection." In Bacterial Activation of Type I Interferons. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09498-4_3.

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Eshleman, Emily M., and Laurel L. Lenz. "Induction and Consequences of the Type I IFN Response to Listeria monocytogenes." In Bacterial Activation of Type I Interferons. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09498-4_2.

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Schlee, M., W. Barchet, V. Hornung, and G. Hartmann. "Beyond Double-Stranded RNA-Type I IFN Induction by 3pRNA and Other Viral Nucleic Acids." In Interferon: The 50th Anniversary. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71329-6_11.

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Knight, E., D. Fahey, R. Kutny, and D. C. Blomstrom. "Characterization of Two New IFN-Induced Proteins." In The Biology of the Interferon System 1986. Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3543-3_14.

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Conference papers on the topic "Interferons (IFNs)"

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Oke, V., I. Gunnarsson, J. Dorschner, A. Zickert, TB Niewold, and E. Svenungsson. "S5A:4 Circulating type i, ii and iii interferons (ifns) associate with ifn-scores, but define distinct subsets of active sle." In 11th European Lupus Meeting, Düsseldorf, Germany, 21–24 March 2018, Abstract presentations. Lupus Foundation of America, 2018. http://dx.doi.org/10.1136/lupus-2018-abstract.27.

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Gamba, G., L. Dezza, N. Montani, G. Gangale, G. Gangale, and E. Ascari. "EFFECT OF INTERFERON GAMMA ON PROCOAGULANT ACTIVITY FROM HUMAN PROMYELOCYTIC CELL LINE (HL 60)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643662.

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Interferon gamma (IFN), a lymphokine acting as biological response modifier, can induce morphological and phenotypic differentiation of some leukemic cell lines, specially along the monocytic pathway.Furthermore, myeloid leukemic cells and normal monocytes have been demonstrated to possess procoagulant activity (PCA).The aim of this study was to investigate the modulation of PCA induced by treatment with IFN on human promyelocytic cells from HL 60 cell line.Cells were cultured in suspension for 5days in RPMI 1640 medium supplemented with 10% fetal calf serum in the absence or in the presence o
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"Interferon Gamma Levels in Relation to COVID-19 Vaccine." In 4th International Conference on Biological & Health Sciences (CIC-BIOHS’2022). Cihan University, 2022. http://dx.doi.org/10.24086/biohs2022/paper.691.

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One-hundred and twenty cases studied in this study for whom they take vaccine dose or not, for infected and non-infected with coronavirus. They are classified to four groups (Covid-19 – Non-Vaccinated = CN); (Covid-19 – Vaccinated = CV); (Non Covid-19 – Non-Vaccinated = NN); (Non-Covid-19 – Vaccinated = NV) each group involved 30 cases. After withdrawing the blood from each person, the blood poured into two tube (5 ml to Gel tube and 2 ml to EDTA tube) and immunological parameters directly performed by hematological analyzer, while the serum, after centrifugation of blood and storing them in s
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Gugliotta, L., S. Macchi, L. Catani, M. Mattioli Belmonte, L. Gaggioli та S. Tura. "EVALUATION OF THROMBOPOIESIS IN ESSENTIAL THROMBOCYTHAEMIA BEFORE AND AFTER α-INTERFERON TREATMENT". У XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644579.

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The α-interferon (α-IFN) has been shown efficacious in controlling thrombocytosis in chronic myeloproliferative disorders. In order to better understand the mechanisms by which this effect is produced, the main parameters of thrombopoiesis have been evaluated in 8 patients with Essential Thrombocythaemia (ET)just before and at the end of induction therapy with α-IFN. The patients, 2 males and 6 females, 17-54 years old, at diagnosis or at least 3 months off cytotoxic drugs, received α-IFN (Roferon A-Roche) s.c. at a daily dose of 3 × 106 IU for 6-13 weeks. The baseline platelet count of 993±26
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Wang, Wei, Hamada A. Aboubakr, James Vang, Victor Brenk, Sagar M. Goyal, and James Collins. "Nanomagnetic Biosensor for the Detection of Porcine Interferon Gamma." In 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3375.

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Due to the anatomical and physiological similarities to humans that include similar heart size, flow rate, skin, liver enzymes and bone healing, porcine models as a powerful investigational platform have been widely used in research areas such as diabetes, obesity and islet transplantation [1]. The advantages of relative low cost, ease in handling and comparatively short period of breeding time may make swine provide a promising solution to the shortage of human donors and difficulty in isolating purified islets from adult human in future. Porcine cytokines play a significant role in innate im
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Sharma, Shashank, Fawzi Ali, Saikia Sujoy, and Karaj Jola. "Interferon gamma (IFN?) pathway deficiency and severe NTM disease." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa3086.

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Vale, Paula Melo, Juliana Gonzaga Araújo Clark, Natália Campos Ramos, Rachel Calazans De Oliveira Costa Costa, and Sofia Assis Alvarenga. "O USO DE INTERFERON-BETA NO TRATAMENTO DE ESCLEROSE MÚLTIPLA." In I Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/981.

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Introdução: A Esclerose Múltipla (EM) é uma doença autoimune que acomete o sistema nervoso central (SNC), promovendo a destruição da bainha de mielina. É considerada rara, cuja epidemiologia é de 30 para 100.000 pessoas no mundo, sendo mais comum em mulheres. Essa doença apresenta como um dos sintomas mais recorrentes a fadiga, caracterizando-se pela dificuldade em manter a contração muscular. Também são relatados sintomas como astenia, neurite óptica, paresia ou parestesia de membros, disfunção da coordenação e equilíbrio, mielites, disfunções esfincterianas e disfunções cognitivo comportamen
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Cakebread, JA, Y. Xu, D. Sammut, P. Howarth, ST Holgate та DE Davies. "Antiviral Effects of Interferon beta (IFNβ) in Asthmatic Bronchial Epithelial Cell Cultures." У American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5171.

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Ригер, Николай Александрович, Элеонора Николаевна Трушина, and Андрей Николаевич Тимонин. "INFLUENCE OF HIGH PHYSICAL LOADS ON THE IMMUNOREGULATORY STATUS OF ATHLETES." In Сборник избранных статей по материалам научных конференций ГНИИ "Нацразвитие" (Санкт-Петербург, Август 2022). Crossref, 2022. http://dx.doi.org/10.37539/aug304.2022.84.56.002.

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В статье приводятся данные изучения цитокинового профиля (определение уровней содержания интерлейкинов: IL-4, IL-10, IL-18 и γ-интерферона (γ-IFN) методом твердофазного иммуноферментного анализа у17 спортсменов - биатлонистов высшей спортивной квалификации и 17 здоровых добровольцев со средней физической активностью. Полученные результаты подтверждают значительную вариабельность содержания иммунорегуляторных цитокинов в сыворотке крови здоровых людей. Повышение уровней провоспалительных цитокинов у спортсменов свидетельствует о напряженности иммунного ответа при интенсивных физических нагрузка
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Yamauchi, K., Y. Shibata, M. Sato та ін. "Reduced IL-12p40 Signaling with Azithromycin (AZM) in Lipopolysacchalide (LPS) and Interferon (IFN)-γ Stimulated Macrophages." У American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a1938.

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Reports on the topic "Interferons (IFNs)"

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Chen, Cheng-Che, and Hao-En Lin. Survival Benefits and Bleeding Risk of Anti-VEGF Agents for Renal Cell Carcinoma (RCC): A Updated Systematic Review and Meta-Analysis of Phase 2 and 3 Randomized Clinical Trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.3.0007.

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Abstract:
Review question / Objective: To investigate the survival benefits (PFS, DFS, OS) and bleeding risk of the anti-VEGF agents compared with placebo or interferon alpha (IFNa) in patients with RCC. Condition being studied: Part 1. The hazard ratio (HR) of the progression-free survival (PFS) and overall survival (OS) of anti-VEGF agents vs. non/placebo for patients with unresectable, advanced, metastatic, renal cell carcinoma (RCC). Part 2. The HR of the disease-free survival (PFS) and OS of anti-VEGF agents vs. non/placebo for patients with post-nephrectomy RCC (adjuvant use). Part 3. The HR of th
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