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Journal articles on the topic 'Interleukin 10'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Zakharov, S.V. "Cytokine profile in patients with early latent syphilis." Medicni perspektivi 23, no. 1 (2018): 71–75. https://doi.org/10.26641/2307-0404.2018.1.124933.

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The purpose of this study was to study the change in the content of the most active cytokines (interleukins 6 and 10) during the formation of the immune response in patients with latent early syphilis, as well as to study the possible relationship between the concentrations of these cytokines and the duration of the disease. In 50 patients with early latent syphilis, the concentration of interleukins 6 and 10 in serum was studied. The serum level of interleukins was studied by the enzyme immunoassay. A statistically significant increase in the concentration of interleukin 6 in the blood of pat

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2

&NA;. "Interleukin-10." Drugs in R & D 1, no. 3 (1999): 262–64. http://dx.doi.org/10.2165/00126839-199901030-00017.

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3

Asadullah, K., R. Sabat, M. Friedrich, W. Docke, H. Volk, and W. Sterry. "Interleukin-10." Anti-Inflammatory & Anti-Allergy Agents in Medicinal Chemistry 5, no. 3 (2006): 223–31. http://dx.doi.org/10.2174/187152306778017700.

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4

Parsons, Polly E. "Interleukin-10." Critical Care Medicine 26, no. 5 (1998): 818–19. http://dx.doi.org/10.1097/00003246-199805000-00007.

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5

Goldman, Michel, Thierry Velu, and Marina Pretolani. "Interleukin-10." BioDrugs 7, no. 1 (1997): 6–14. http://dx.doi.org/10.2165/00063030-199707010-00002.

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6

Moore, K. W., A. O'Garra, R. W. Malefyt, P. Vieira, and T. R. Mosmann. "Interleukin-10." Annual Review of Immunology 11, no. 1 (1993): 165–90. http://dx.doi.org/10.1146/annurev.iy.11.040193.001121.

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7

Umetsu, Dale T., and Rosemarie H. DeKruyff. "Interleukin-10." American Journal of Respiratory Cell and Molecular Biology 21, no. 5 (1999): 562–63. http://dx.doi.org/10.1165/ajrcmb.21.5.f171.

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8

Zlotnik, Albert, and Kevin W. Moore. "Interleukin 10." Cytokine 3, no. 5 (1991): 366–71. http://dx.doi.org/10.1016/1043-4666(91)90039-g.

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9

Klava, Andrew. "Interleukin-10." Archives of Surgery 132, no. 4 (1997): 425. http://dx.doi.org/10.1001/archsurg.1997.01430280099016.

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10

DEWAALMALEFYT, R., M. RONCAROLO, H. SPITS, and J. DEVRIES. "Interleukin-10." Current Opinion in Immunology 4, no. 3 (1992): 314–20. http://dx.doi.org/10.1016/0952-7915(92)90082-p.

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11

Reineke, Ulrich, Robert Sabat, Hans-Dieter Volk, and Jens Schneider-Mergener. "Mapping of the interleukin-10/interleukin-10 receptor combining site." Protein Science 7, no. 4 (1998): 951–60. http://dx.doi.org/10.1002/pro.5560070412.

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12

Shah, Neil, Jochen Kammermeier, Mamoun Elawad, and Erik-Oliver Glocker. "Interleukin-10 and Interleukin-10–Receptor Defects in Inflammatory Bowel Disease." Current Allergy and Asthma Reports 12, no. 5 (2012): 373–79. http://dx.doi.org/10.1007/s11882-012-0286-z.

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13

Mittal, Sharad K., Kyung-Jin Cho, Satoshi Ishido, and Paul A. Roche. "Interleukin 10 (IL-10)-mediated Immunosuppression." Journal of Biological Chemistry 290, no. 45 (2015): 27158–67. http://dx.doi.org/10.1074/jbc.m115.682708.

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14

Abasova, Dunya, Sevda Kazimova, Nazrin Macidova, Telli Shirinova, and Shalala Babayeva. "STUDY OF STOKIN STATUS IN PATIENTS WITH TOXOPLASMOSIS." Annali d'Italia 50 (December 26, 2023): 17–19. https://doi.org/10.5281/zenodo.10432472.

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The problem of opportunistic infections is becoming an urgent problem of modern times is becoming, which is a constant increase in the number of people infected with HIV, it is related to the increase in aggressive treatment methods of oncopathologies. This infections Pathogens that can normally cause disease in healthy individuals caused by Up to 20 typical causative agents of opportunistic infections are known, and in modern infectious diseases this list is constantly growing. without symptoms: 60% - cytomegalovirus, 90% - first and second type herpavirus, 30% are toxoplasma c

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15

Oleksandr, Hruzevskyi. "The cytokine system's status in bacterial dysbiosis and bacterial vaginosis." ScienceRise: Medical Science, no. 3(36) (May 31, 2020): 50–56. https://doi.org/10.15587/2519-4798.2020.204094.

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Interrelations of conditionally-pathogenic microflora and vaginal mucosa’s APC are realized by forming of proinflammatory and regulatory cytokines which can provoke bacterial dysbiosis progression and bacterial vaginosis development. <strong>The a</strong><strong>im of the study: </strong>to determine cytokine system’s status in the blood and vaginal fluid in bacterial dysbiosis and bacterial vaginosis. <strong>Material and methods</strong>. There were used data from 298 women, divided into groups according to the index of conditionally pathogenic microflora and normobiota ind

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16

Holland, Gina, and Albert Zlotnik. "Interleukin-10 and Cancer." Cancer Investigation 11, no. 6 (1993): 751–58. http://dx.doi.org/10.3109/07357909309046950.

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17

Kato, Masato. "Interleukin-10 and Surgery." Critical Care Medicine 29, no. 5 (2001): 1093. http://dx.doi.org/10.1097/00003246-200105000-00054.

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18

Cohen, Stacey L., Aideen M. Moore, and Wendy E. Ward. "Interleukin-10 Knockout Mouse:." Inflammatory Bowel Diseases 10, no. 5 (2004): 557–63. http://dx.doi.org/10.1097/00054725-200409000-00009.

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19

Rennick, Donna, Dan Berg, and Gina Holland. "Interleukin 10: An overview." Progress in Growth Factor Research 4, no. 3 (1992): 207–27. http://dx.doi.org/10.1016/0955-2235(92)90020-i.

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20

Elson, C. O. "Interleukin-10 and counting …" Gastroenterology 100, no. 6 (1991): 1778. http://dx.doi.org/10.1016/0016-5085(91)90687-g.

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21

Sabat, Robert, Gerald Grütz, Katarzyna Warszawska, et al. "Biology of interleukin-10." Cytokine & Growth Factor Reviews 21, no. 5 (2010): 331–44. http://dx.doi.org/10.1016/j.cytogfr.2010.09.002.

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22

Brennan, F. "Interleukin 10 and arthritis." Rheumatology 38, no. 4 (1999): 293–97. http://dx.doi.org/10.1093/rheumatology/38.4.293.

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23

Montero, J. G. "Interleukin 10 and Sepsis." Archives of Surgery 135, no. 7 (2000): 875–76. http://dx.doi.org/10.1001/archsurg.135.7.875.

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24

Peng, Hui, Wei Wang, Mo Zhou, Rui Li, Hai-Feng Pan, and Dong-Qing Ye. "Role of interleukin-10 and interleukin-10 receptor in systemic lupus erythematosus." Clinical Rheumatology 32, no. 9 (2013): 1255–66. http://dx.doi.org/10.1007/s10067-013-2294-3.

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25

Peng, Chih-Wen, Hao-Jen Hsu, Chun-Chun Chang, Cheng-Der Liu, Sheng-Feng Pan, and Wei-Han Huang. "Targeting of interleukin-10 receptor by a potential human interleukin-10 peptide efficiently blocks interleukin-10 pathway-dependent cell proliferation." Tzu Chi Medical Journal 32, no. 3 (2020): 245. http://dx.doi.org/10.4103/tcmj.tcmj_237_19.

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26

Wang, P., P. Wu, JC Anthes, MI Siegel, RW Egan, and MM Billah. "Interleukin-10 inhibits interleukin-8 production in human neutrophils." Blood 83, no. 9 (1994): 2678–83. http://dx.doi.org/10.1182/blood.v83.9.2678.2678.

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Abstract In highly purified human polymorphonuclear leukocyte (PMN) preparations containing less than 0.1% contaminating monocytes, significant amounts of interleukin-8 (IL-8) and small amounts of IL-1 alpha, IL-1 beta, and tumor necrosis factor-alpha (TNF-alpha) were produced by lipopolysaccharide (LPS) stimulation. Contrary to published reports, IL- 6 production could not be detected. IL-10 inhibited the production of IL-1 alpha, IL-1 beta, IL-8, and TNF-alpha in LPS-stimulated PMNs, as it did in human blood mononuclear cell (MNC) preparations enriched in monocytes. Subsequent investigation

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27

Wang, P., P. Wu, JC Anthes, MI Siegel, RW Egan, and MM Billah. "Interleukin-10 inhibits interleukin-8 production in human neutrophils." Blood 83, no. 9 (1994): 2678–83. http://dx.doi.org/10.1182/blood.v83.9.2678.bloodjournal8392678.

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In highly purified human polymorphonuclear leukocyte (PMN) preparations containing less than 0.1% contaminating monocytes, significant amounts of interleukin-8 (IL-8) and small amounts of IL-1 alpha, IL-1 beta, and tumor necrosis factor-alpha (TNF-alpha) were produced by lipopolysaccharide (LPS) stimulation. Contrary to published reports, IL- 6 production could not be detected. IL-10 inhibited the production of IL-1 alpha, IL-1 beta, IL-8, and TNF-alpha in LPS-stimulated PMNs, as it did in human blood mononuclear cell (MNC) preparations enriched in monocytes. Subsequent investigation of cytoki

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28

Rentzos, M., C. Nikolaou, E. Andreadou, et al. "Circulating interleukin-10 and interleukin-12 in Parkinson’s disease." Acta Neurologica Scandinavica 119, no. 5 (2009): 332–37. http://dx.doi.org/10.1111/j.1600-0404.2008.01103.x.

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29

Iwasaki, Yasuo, Shigeji Baba, and Ken Ikeda. "Interleukin 4 and Interleukin 10 in Creutzfeldt-Jakob Disease." Archives of Neurology 63, no. 6 (2006): 911. http://dx.doi.org/10.1001/archneur.63.6.911-a.

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30

Ammirati, Enrico, Carlo V. Cannistraci, Nicole A. Cristell, et al. "Identification and Predictive Value of Interleukin-6 + Interleukin-10 + and Interleukin-6 − Interleukin-10 + Cytokine Patterns in ST-Elevation Acute Myocardial Infarction." Circulation Research 111, no. 10 (2012): 1336–48. http://dx.doi.org/10.1161/circresaha.111.262477.

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31

Freer, Giulia. "Interleukin 10 in Antiviral Responses." Current Immunology Reviews 12, no. 1 (2016): 20–26. http://dx.doi.org/10.2174/1573395512666151216210559.

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32

Benjamin, David, Chanun D. Park, and Venkatanarayanan Sharma. "Human B Cell Interleukin 10." Leukemia & Lymphoma 12, no. 3-4 (1994): 205–9. http://dx.doi.org/10.3109/10428199409059591.

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33

Cihakova, Daniela. "Interleukin-10 stiffens the heart." Journal of Experimental Medicine 215, no. 2 (2018): 379–81. http://dx.doi.org/10.1084/jem.20180049.

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Cardiac-resident macrophages are a diverse population of cells that have a critical role in the pathogenesis of heart failure. A new understanding of communication between macrophages and cardiac fibroblasts could lead to novel therapeutic strategies for heart failure with preserved ejection function.

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34

SCHREIBER, STEFAN. "Interleukin-10 in the intestine." Gut 41, no. 2 (1997): 274–75. http://dx.doi.org/10.1136/gut.41.2.274.

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35

Garofalo, Roberto, Sadhana Chheda, Fang Mei, et al. "Interleukin-10 in Human Milk." Pediatric Research 37, no. 4 (1995): 444–49. http://dx.doi.org/10.1203/00006450-199504000-00010.

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36

Bogdan, C., Y. Vodovotz, and C. Nathan. "Macrophage deactivation by interleukin 10." Journal of Experimental Medicine 174, no. 6 (1991): 1549–55. http://dx.doi.org/10.1084/jem.174.6.1549.

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Recombinant mouse interleukin 10 (IL-10) was exceedingly potent at suppressing the ability of mouse peritoneal macrophages (m phi) to release tumor necrosis factor alpha (TNF-alpha). The IC50 of IL-10 for the suppression of TNF-alpha release induced by 0.5 microgram/ml lipopolysaccharide was 0.04 +/- 0.03 U/ml, with as little as 1 U/ml suppressing TNF-alpha production by a factor of 21.4 +/- 2.5. At 10 U/ml, IL-10 markedly suppressed m phi release of reactive oxygen intermediates (ROI) (IC50 3.7 +/- 1.8 U/ml), but only weakly inhibited m phi release of reactive nitrogen intermediates (RNI). Si

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37

Marchant, A., M. Goldman, J. Devière, B. Byl, J. L. Vincent, and D. De Groote. "Interleukin-10 production during septicaemia." Lancet 343, no. 8899 (1994): 707–8. http://dx.doi.org/10.1016/s0140-6736(94)91584-9.

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38

Moore, Kevin W., Rene de Waal Malefyt, Robert L. Coffman, and Anne O'Garra. "INTERLEUKIN-10AND THEINTERLEUKIN-10 RECEPTOR." Annual Review of Immunology 19, no. 1 (2001): 683–765. http://dx.doi.org/10.1146/annurev.immunol.19.1.683.

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39

Howard, Maureen, Anne O'Garra, Hiroshi Ishida, René de Waal Malefyt, and Jan De Vries. "Biological properties of interleukin 10." Journal of Clinical Immunology 12, no. 4 (1992): 239–47. http://dx.doi.org/10.1007/bf00918147.

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40

Howard, Maureen, and Anne O'Garra. "Biological properties of interleukin 10." Immunology Today 13, no. 6 (1992): 198–200. http://dx.doi.org/10.1016/0167-5699(92)90153-x.

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41

Asadullah, Khusru, Wolf-Dietrich Döcke, Robert Sabat, Merle Ebeling, Hans-Dieter Volk, and Wolfram Sterry. "Interleukin-10 in der Dermatologie." Der Hautarzt 50, no. 1 (1999): 12–19. http://dx.doi.org/10.1007/s001050050858.

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42

Lee, G., D. Vollmer, and X. Han. "A Phytocannabinoid Formula Containing Palmitoylethanolamide, Acmella Oleracea Extract, and Bovine Colostrum Filtrate Inhibited Cytokine and Chemokine Production in Human Peripheral Blood Mononuclear Cells Stimulated Ex Vivo." Current Topics in Nutraceutical Research 20, no. 2 (2021): 438–46. http://dx.doi.org/10.37290/ctnr2641-452x.20:438-446.

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A phytocannabinoid formula containing copaiba essential oil, palmitoylethanolamide, Sichuan pepper extract, Acmella oleracea extract, cruciferous vegetable extracts blend, and bovine colostrum filtrate was tested for its ex vivo effect on inhibiting stimulated cytokine release in human peripheral blood mononuclear cells. Effects of the phytocannabinoid formula on the peripheral blood mononuclear cells viability were measured by alamarBlue™ to confirm no obvious cytotoxicity. The phytocannabinoid formula was compared to reference compounds for its ability to inhibit both the phytohemagglutinin-

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43

Wande, I. Nyoman, Endang Retnowati, and Juli Soemarsono. "KADAR INTERLEUKIN 10 (IL-10) MALARIA DAN ANEMIA." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 18, no. 1 (2016): 4. http://dx.doi.org/10.24293/ijcpml.v18i1.767.

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Anaemia is an important complication of malaria, and its pathogenesis is not well understood. High level of the Th2 cytokine (such as IL-10), which counteract the Th1 cytokine, might prevent the development of severe malarial anaemia. The purpose of this study was to know the comparation between the plasma level of IL-10 in malaria patients with anaemia and without anaemia. The plasma level of IL-10 was examined in 16 malaria patients with anaemia and 16 malaria caused by P. falciparum patients without anaemia samplestaken from patients at the primary health centres in West Lombok and Centre L

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44

Furukawa, Yutaka, Gerold Becker, Jennifer L. Stinn, Koichi Shimizu, Peter Libby, and Richard N. Mitchell. "Interleukin-10 (IL-10) Augments Allograft Arterial Disease." American Journal of Pathology 155, no. 6 (1999): 1929–39. http://dx.doi.org/10.1016/s0002-9440(10)65512-5.

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45

Salwen, S. A., T. Sato, K. Masuoka, G. Inoue, M. J. Mastrangelo, and D. Berd. "INTERLEUKIN-10 (IL-10) PRODUCTION BY MELANOMA CELLS." Journal of Immunotherapy 18, no. 2 (1995): 129. http://dx.doi.org/10.1097/00002371-199508000-00014.

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46

BENDTZEN, KLAUS, MORTEN B. HANSEN, MARCUS DIAMANT, CHRISTIAN ROSS та MORTEN SVENSON. "Naturally Occurring Autoantibodies to Interleukin-1α, Interleukin-6, Interleukin-10, and Interferon-α". Journal of Interferon Research 14, № 4 (1994): 157–58. http://dx.doi.org/10.1089/jir.1994.14.157.

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47

Diller, R., D. Palmes, K. H. Dietl, N. Senninger, G. Winde, and H. U. Spiegel. "Interleukin-6, interleukin-8, and interleukin-10 in kidney transplantation: improved risk strategy?" Transplantation Proceedings 35, no. 4 (2003): 1333–37. http://dx.doi.org/10.1016/s0041-1345(03)00529-3.

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48

Digban, Awharentomah Kester, Matthew Eturhobore Adu, and Mary Abiodun Adenuga. "EVALUATION OF SERUM INTERLEUKINS 1 AND 10 IN TYPE 2 DIABETES MELLITUS SUBJECTS IN BENIN CITY." FUDMA JOURNAL OF SCIENCES 7, no. 2 (2023): 146–51. http://dx.doi.org/10.33003/fjs-2023-0702-1203.

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Diabetes mellitus is a metabolic and endocrinological disease that results in hyperglycemia as a result of either insulin absence or non-responsiveness of insulin receptors. Interleukins have been implicated in the pathogenesis of diabetes mellitus. This study sought to evaluate serum interleukin-1 and 10 in diabetes mellitus. A total of three hundred subjects comprising of one hundred and eighty diabetes mellitus and one hundred and twenty apparently healthy subjects were recruited. Of the one hundred and eighty diabetes mellitus is made of 41.67% male and 58.33% females. Venous blood samples

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49

Tilg, Herbert, Michael B. Atkins, Charles A. Dinarello, and James W. Mier. "Induction of circulating interleukin 10 by interleukin 1 and interleukin 2, but not interleukin 6 immunotherapy." Cytokine 7, no. 7 (1995): 734–39. http://dx.doi.org/10.1006/cyto.1995.0087.

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50

Al-Rasheed, A., H. Scheerens, D. M. Rennick, H. M. Fletcher, and D. N. Tatakis. "Accelerated Alveolar Bone Loss in Mice Lacking Interleukin-10." Journal of Dental Research 82, no. 8 (2003): 632–35. http://dx.doi.org/10.1177/154405910308200812.

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Interleukin-10 regulates pro-inflammatory cytokines, including those implicated in alveolar bone resorption. We hypothesized that lack of interleukin-10 leads to increased alveolar bone resorption. Male interleukin-10(−/−) mice, on 129/SvEv and C57BL/6J background, were compared with age-, sex-, and strain-matched interleukin-10(+/+) controls for alveolar bone loss. Immunoblotting was used for analysis of serum reactivity against bacteria associated with colitis and periodontitis. Interleukin-10(−/−) mice had significantly greater alveolar bone loss than interleukin-10(+/+) mice (p = 0.006). T

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