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Relevant bibliographies by topics / Interleukin-12

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Dissertations / Theses
Books
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Academic literature on the topic 'Interleukin-12'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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Journal articles on the topic "Interleukin-12"

1

Leonard, Patricia, and Sanjiv Sur. "Interleukin-12." BioDrugs 17, no. 1 (2003): 1–7. http://dx.doi.org/10.2165/00063030-200317010-00001.

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&NA;. "Interleukin-12." Inpharma Weekly &NA;, no. 1124 (1998): 7. http://dx.doi.org/10.2165/00128413-199811240-00013.

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Trinchieri, Giorgio. "Interleukin-12." Clinical Immunotherapeutics 3, no. 4 (1995): 262–70. http://dx.doi.org/10.1007/bf03259278.

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Clinton, Steven K., Eduardo Canto, and Michael A. O'Donnell. "INTERLEUKIN-12." Urologic Clinics of North America 27, no. 1 (2000): 147–55. http://dx.doi.org/10.1016/s0094-0143(05)70242-1.

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Weiner, David. "Plasmid Interleukin 12." AIDS 14, no. 12 (2000): 1759–60. http://dx.doi.org/10.1097/00002030-200008180-00010.

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Leng, S. X., and J. A. Elias. "Interleukin-11 inhibits macrophage interleukin-12 production." Journal of Immunology 159, no. 5 (1997): 2161–68. http://dx.doi.org/10.4049/jimmunol.159.5.2161.

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Abstract IL-12 is a heterodimeric cytokine produced by phagocytic and other cells with important physiologic and pathologic properties. Regulated IL-12 production is crucial for the generation of protective Th1 responses to infectious agents. In contrast, IL-12 excess contributes to the pathogenesis of a variety of autoimmune and inflammatory disorders. To further understand the processes regulating IL-12 production, we determined whether IL-11 regulated monocyte/macrophage production of this cytokine moiety. IL-11 did not alter the IL-12 (p70) production of unstimulated THP-1 monocytic cells

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Fox, Robert J., and Abdolmohamad M. Rostami. "Anti???Interleukin-12 Antibody." BioDrugs 13, no. 4 (2000): 233–41. http://dx.doi.org/10.2165/00063030-200013040-00002.

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&NA;. "Interleukin-12 trial halted." Reactions Weekly &NA;, no. 556 (1995): 2. http://dx.doi.org/10.2165/00128415-199505560-00003.

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&NA;. "Interleukin-12 studies continue." Reactions Weekly &NA;, no. 558 (1995): 2. http://dx.doi.org/10.2165/00128415-199505580-00003.

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&NA;. "Interleukin-12 trial halted." Inpharma Weekly &NA;, no. 992 (1995): 21. http://dx.doi.org/10.2165/00128413-199509920-00047.

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Dissertations / Theses on the topic "Interleukin-12"

1

Balu, Sucharitha. "Cloning and characterisation of chicken interleukin-12 and the interleukin-12 receptor." Thesis, University of Bristol, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.419213.

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Werner, Christoph. "Von Interleukin-12 zur p40-Zytokinfamilie Interleukin-12-unabhängige Wirkungen von p40-Zytokinen in der Infekt- und Tumorabwehr /." [S.l.] : [s.n.], 2003. http://dol.uni-leipzig.de/pub/2003-34.

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Werner, Christoph. "Von Interleukin-12 zur p40-Zytokinfamilie: Interleukin-12-unabhängige Wirkungen von p40-Zytokinen in der Infekt- und Tumorabwehr." Doctoral thesis, Universitätsbibliothek Leipzig, 2004. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-37550.

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Interleukin-12 (IL-12) ist ein zentrales Zytokin in der Entwicklung einer protektiven, zellulären Typ 1-Immunantwort. Es ist aus einer p40- und einer p35-Untereinheit aufgebaut. Es stimuliert NK- und T-Zellen zur Ausschüttung großer Mengen IFN-gamma und setzt so eine Typ 1-Immunantwort in Gang. Die p40-Untereinheit des IL-12 kommt auch in anderen biologisch wirksamen Verbindungen wie beispielsweise als Monomer, Homodimer oder als IL-23 (in Verbindung mit einer p19-Untereinheit) vor. Während das Homodimer in der vorliegenden Arbeit als reiner Antagonist zu IL-12 zu wirken scheint, wurden für IL

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Walter, Claudia. "Bestimmung der Zytokine Interleukin-1ra, Interleukin-6, Interleukin-10 und Interleukin-12 im Vaginalsekret bei Frauen mit Bakterieller Vaginose." Diss., lmu, 2004. http://nbn-resolving.de/urn:nbn:de:bvb:19-28461.

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Chakir, Habiba. "Role of interleukin-12 and interleukin-18 in murine immune cell regulation." Thesis, University of Ottawa (Canada), 2003. http://hdl.handle.net/10393/28980.

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Interleukin 12 plays a central role in NK cell activation and CD4 +T cell differentiation. IL-12 acts via a receptor composed of IL-12Rbeta1 and IL-12Rbeta2 subunits. IL-12Rbeta1 plays a primary role in ligand binding whereas IL-12Rbeta2 is responsible for signaling. IL-18 shares some functional activities of IL-12. However, there is a considerable amount of contradictory data in the literature regarding the requirements for IL-12 and IL-18 responsiveness. This work examined the expression of IL-12Rbeta2 on murine NK and T cells and the requirements for acquisition of IL-12/IL-18 responsivenes

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Swinburne, Sarah Jane. "A study of the molecular and biological characteristics of ovine interleukin-12." Title page, contents and summary only, 2000. http://web4.library.adelaide.edu.au/theses/09PH/09phs9777.pdf.

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Bibliography: leaves 172-214. Interleukin-12 (IL-12) is a heterodimeric cytokine composed of two disulphide-linked subunits, p35 and p40, which form biologically active p70. IL-12 is able to induce IFN-y production from T and NK cells, and promote the proliferation of mitogen-activated T cells. It is thought that IL-12 may be an important cytokine in the initiation and progression of allograft destruction. This thesis describes the characterisation of ovine IL-12.

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Rempel, Julia D. "Interleukin-12 and interleukin-10 regulation of antibody responses upon protein antigen immunization." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ41623.pdf.

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Athie, Morales Veronica. "Interleukin 12 signalling pathways in human T lymphocytes." Thesis, University College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.249566.

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Schardt, Victor. "Vergleichende Untersuchungen zur Hefepilzbesiedelung von Mundhöhle und Vagina und Bestimmung von Interleukin-4, Interleukin-10 und Interleukin-12." Diss., lmu, 2013. http://nbn-resolving.de/urn:nbn:de:bvb:19-155152.

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Naseer, Tanveer 1971. "In vivo expression of interleukin-12 and interleukin-13 in the pathogenesis of asthma." Thesis, McGill University, 1996. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=23925.

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IL-12 and IL-13 are recently discovered cytokines which belong to Th1 and Th2 subtypes, respectively. To further support the hypothesis that Th2-type cytokines are involved in asthma, mRNA levels for IL-12 and IL-13 were measured in bronchial biopsies from asthmatics and normal controls as well as in moderately severe asthmatics before and after corticosteroid therapy using the technique of in situ hybridization. The number of IL-13 mRNA positive cells in asthmatics was significantly higher than that found in normal controls. However, there was a significant decrease in the number of IL-12 mRN

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Books on the topic "Interleukin-12"

1

Lasek, Witold, and Radoslaw Zagozdzon. Interleukin 12: Antitumor Activity and Immunotherapeutic Potential in Oncology. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-46906-5.

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Il-12 (Chemical Immunology). S. Karger AG (Switzerland), 1997.

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Interleukin 12: Cellular and molecular immunology of an important regulatory cytokine. New York Academy of Sciences, 1996.

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Lasek, Witold, Radoslaw Zagozdzon, and Marek Jakóbisiak. Interleukin 12: Antitumor Activity and Immunotherapeutic Potential in Oncology. Springer, 2016.

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Interleukin 12: Antitumor Activity and Immunotherapeutic Potential in Oncology. Springer International Publishing AG, 2016.

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(Editor), Michael T. Lotze, Giorgio Trinchiere (Editor), Maurice Gately (Editor), and Stanley Wolf (Editor), eds. Interleukin 12: Cellular and Molecular Immunology of an Important Regulatory Cytokine (Annals of the New York Academy of Sciences). New York Academy of Sciences, 1996.

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New York Academy of Sciences (Corporate Author), Inc. Genetics Institute (Corporate Author), and Michael T. Lotze (Editor), eds. Interleukin 12: Cellular and Molecular Immunology of an Important Regulatory Cytokine (Annals of the New York Academy of Sciences, V. 795). New York Academy of Sciences, 1996.

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8

Laubert-Reh, Dagmar. Untersuchungen zur Bildung von Interleukin-12 in Knochenmarkmakrophagen der Maus. 1996.

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9

Dorfmüller, Andreas. Die Immunantwort Interleukin-12-defizierter Mäuse nach Infektion mit Listeria monocytogenes. 1999.

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Gutenberg-Universität, Johannes, ed. Interleukin-12 als Mediator und Adjuvans in der Induktionsphase des allergischen Kontaktekzems. 1996.

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Book chapters on the topic "Interleukin-12"

1

Kaufman, Howard L., and Neal Dharmadhikari. "Interleukin-12." In Cancer Therapeutic Targets. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4419-0717-2_144.

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Dieckman, Tessa, Antonie Zwiers, Georg Kraal, and Gerd Bouma. "Interleukin 12." In Compendium of Inflammatory Diseases. Springer Basel, 2016. http://dx.doi.org/10.1007/978-3-7643-8550-7_220.

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Bukowski, Ronald M., and Charles Tannenbaum. "Interleukin-12." In Melanoma. Humana Press, 2002. http://dx.doi.org/10.1007/978-1-59259-159-6_8.

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Kaufman, Howard L., and Neal Dharmadhikari. "Interleukin-12." In Cancer Therapeutic Targets. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6613-0_144-1.

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Dieckman, Tessa, Antonie Zwiers, Georg Kraal, and Gerd Bouma. "Interleukin 12." In Encyclopedia of Inflammatory Diseases. Springer Basel, 2014. http://dx.doi.org/10.1007/978-3-0348-0620-6_220-1.

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6

Bryan, Shannon, Michiko Kobayashi, and Sanjiv Sur. "Interleukin-12, Interleukin-18 and Interferon-γ." In New Drugs for Asthma, Allergy and COPD. KARGER, 2001. http://dx.doi.org/10.1159/000062155.

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Pile, Kevin D., Garry G. Graham, and Stephen M. Mahler. "Interleukin-12/23 Inhibitors: Ustekinumab." In Compendium of Inflammatory Diseases. Springer Basel, 2016. http://dx.doi.org/10.1007/978-3-7643-8550-7_232.

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Pile, Kevin D., Garry G. Graham, and Stephen M. Mahler. "Interleukin-12/23 Inhibitors: Ustekinumab." In Encyclopedia of Inflammatory Diseases. Springer Basel, 2015. http://dx.doi.org/10.1007/978-3-0348-0620-6_232-1.

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Aulitzky, W. E., and C. Huber. "Interleukin-12: Biology and Clinical Studies." In Symposium in Immunology VI. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60562-8_10.

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Fernández-Ruiz, Mario. "Interleukin-12 and -23 Targeted Agents." In Infectious Complications in Biologic and Targeted Therapies. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-11363-5_11.

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Conference papers on the topic "Interleukin-12"

1

"03 Targeting interleukin 12/23." In 8th ANNUAL MEETING OF THE LUPUS ACADEMY, Warsaw, Poland, September 6–8, 2019. Lupus Foundation of America, 2019. http://dx.doi.org/10.1136/lupus-2019-la.15.

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Conway, R., L. O’Neill, P. Gallagher, et al. "SAT0542 Interleukin-12 and interleukin-23 are key pathogenic players in giant cell arteritis." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.2850.

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Canataroglu, A., E. Erken, and R. Gunesacar. "FRI0165 Serum levels of interleukin-12 in behcet’s disease." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.225.

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Vo, Jimmy Nhu. "Abstract 1535: Intratumoral chitosan/interleukin-12 immunotherapy reduces breast cancer metastasis." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1535.

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Ursic Valentinuzzi, K., U. Kamensek, S. Kos, S. Kranjc Brezar, M. Cemazar, and G. Sersa. "P08.03 Interleukin-12 gene electrotransfer as an adjuvant immunotherapy to electrochemotherapy." In iTOC9 – 9th Immunotherapy of Cancer Conference, September 22–24, 2022 – Munich, Germany. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-itoc9.49.

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Ishihara, Jun, Aslan Mansurov, and Jeffrey Hubbell. "1227 Eliminating the immunotoxicity of interleukin-12 through protease-sensitive masking." In SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.1227.

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Smith, Sean, Jack Baltz, Bhanu Koppolu, Sruthi Ravindranathan, Khue Nguyen, and David Zaharoff. "Abstract B020: Effector cells in chitosan/interleukin-12 immunotherapy of bladder tumors in mice." In Abstracts: CRI-CIMT-EATI-AACR Inaugural International Cancer Immunotherapy Conference: Translating Science into Survival; September 16-19, 2015; New York, NY. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr15-b020.

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Yang, Xi, Ashlee Tietje, Xianzhong Yu, and Yanzhang Wei. "Abstract 4874: Interleukin-12/FasTI: A novel bi-functional fusion protein for cancer immunotherapy." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-4874.

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Cheema, Tooba A., Hiroaki Wakimoto, Samuel Rabkin, and Robert L. Martuza. "Abstract 5389: Oncolytic herpes simplex virus expressing interleukin-12 for treating glioma stem cells." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-5389.

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Herpels, Melanie, Chanthirika Ragulan, Aslan Mansurov, Jun Ishihara, and Anguraj Sadanandam. "1212 Bioengineered tumour-targeted interleukin-12 selectively cures murine triple-negative breast cancer (TNBC)." In SITC 38th Annual Meeting (SITC 2023) Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/jitc-2023-sitc2023.1212.

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Reports on the topic "Interleukin-12"

1

นุชประยูร, สุรางค์. แผนการวิจัยเรื่อง การศึกษาภูมิคุ้มกันวิทยาเชิงลึกของโรคเท้าช้าง : มุ่งสู่การป้องกันภาวะเท้าช้างและการกำจัดโรคอย่างถาวร : รายงานการวิจัยฉบับสมบูรณ์. จุฬาลงกรณ์มหาวิทยาลัย, 2008. https://doi.org/10.58837/chula.res.2008.25.

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โรคเท้าช้าง (Lymphatic filariasis) เกิดจากพยาธิ 2 ชนิดหลัก คือ Wuchereria bancrofti และ Brugia malayi ทางองค์การอนามัยโลกได้กำหนดให้โรคเท้าช้างเป็นโรคทางปรสิตที่ควรกำจัดให้หมดไปภายในปี พ.ศ. 2563 โดยมีแนวทางหลักในการควบคุมและป้องกันโรคเท้าช้างคือการจัดให้มีโปรแกรมการรักษาแบบหมู่ โดยให้ยา diethylcarbamazine (DEC) ร่วมกับยา albendazole แก่ประชากรในพื้นที่ที่มีความชุกของโรคสูง และการควบคุมพยาธิภาวะ ปัญหาที่สำคัญของการรักษาโรคเท้าช้างคือ การใช้ยา DEC ที่ก่อให้เกิดปฏิกิริยาหลังการรักษา กลไกของการเกิดพยาธิสภาพของโรคและการเกิดปฏิกิริยาหลังการรักษายังไม่เป็นที่ทราบแน่ชัดได้ จึงเป็นอุปสรรคที่สำคัญอย่างย

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Tumwasorn, Somying, and Versalovic, James. Identification of probiotic lactobacillus with anti-inflammatory activity in the gastrointestinal tract. Chulalongkorn University, 2009. https://doi.org/10.58837/chula.res.2009.15.

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In order to identify probiotic Lactobacillus for potential use as immunoprobiotics for the amelioration or treatment of inflammatory diseases in the gastrointestinal tract such as Helicobacter pylori-induced gastro-duodenal diseases and Clostridium difficile-associated disease (CDAD), we chose to test for the ability of human-derived Lactobacillus to modulate interleukin-8 (IL8) production in gastrointestinal epithelial cells. Eighty-seven gastric biopsy-derived Lactobacillus isolates were tested for their ability to modulate IL-8 production in H.pylori-stimulated AGS cells and 37 feces-derive

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นุชประยูร, สุรางค์, จินตนา จิรถาวร, อนุพงค์ สุจริยากุล та อลิสา จันทร์ปี. การศึกษาภูมิคุ้มกันวิทยาเชิงลึกของโรคเท้าช้าง : มุ่งสู่การป้องกันภาวะเท้าช้างและการกำจัดโรคอย่างถาวร : แผนการวิจัย : รายงานการวิจัยฉบับสมบูรณ์. จุฬาลงกรณ์มหาวิทยาลัย, 2009. https://doi.org/10.58837/chula.res.2009.21.

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โรคเท้าช้าง (Lymphatic filariasis) เกิดจากพยาธิ 2 ชนิดหลัก คือ Wuchereria bancrofti และ Brugia malayi ทางองค์การอนามัยโลกได้กำหนดให้โรคเท้าช้างเป็นโรคทางปรสิตที่ควรกำจัดให้หมดไปภายในปี พ.ศ. 2563 โดยมีแนวทางหลักในการควบคุมและป้องกันโรคเท้าช้างคือการจัดให้มีโปรแกรมการรักษาแบบหมู่ โดยให้ยา diethylcarbamazine (DEC) ร่วมกับยา albendazole แก่ประชากรในพื้นที่ที่มีความชุกของโรคสูง และการควบคุมพยาธิภาวะ ปัญหาที่สำคัญของการรักษาโรคเท้าช้าง คือ การใช้ยา DEC ที่ก่อให้เกิดปฏิกิริยาหลังการรักษา กลไกของการเกิดพยาธิสภาพของโรคและการเกิดปฏิกิริยาหลังการรักษายังไม่เป็นที่ทราบแน่ชัดได้ จึงเป็นอุปสรรคที่สำคัญอย่าง

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