Relevant bibliographies by topics / Intermediaires du cycle de Krebs

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Intermediaires du cycle de Krebs'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 February 2022

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Journal articles on the topic "Intermediaires du cycle de Krebs"

1

Masson, Veneta. "Krebs Cycle." Annals of Internal Medicine 166, no. 12 (2017): 911. http://dx.doi.org/10.7326/m17-0374.

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Salway, Jack G. "The Krebs Uric Acid Cycle: A Forgotten Krebs Cycle." Trends in Biochemical Sciences 43, no. 11 (2018): 847–49. http://dx.doi.org/10.1016/j.tibs.2018.04.012.

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Helser, Terry L. "Krebs Cycle Wordsearch." Journal of Chemical Education 78, no. 4 (2001): 515. http://dx.doi.org/10.1021/ed078p515.

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Fisher, Miles. "From bicycle to Krebs' cycle." Practical Diabetes 30, no. 7 (2013): 262–63. http://dx.doi.org/10.1002/pdi.1788.

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Baysal, Bora E. "Krebs cycle enzymes as tumor suppressors." Drug Discovery Today: Disease Mechanisms 2, no. 2 (2005): 247–54. http://dx.doi.org/10.1016/j.ddmec.2005.05.012.

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Ryan, Dylan G., and Luke A. J. O'Neill. "Krebs Cycle Reborn in Macrophage Immunometabolism." Annual Review of Immunology 38, no. 1 (2020): 289–313. http://dx.doi.org/10.1146/annurev-immunol-081619-104850.

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A striking change has happened in the field of immunology whereby specific metabolic processes have been shown to be a critical determinant of immune cell activation. Multiple immune receptor types rewire metabolic pathways as a key part of how they promote effector functions. Perhaps surprisingly for immunologists, the Krebs cycle has emerged as the central immunometabolic hub of the macrophage. During proinflammatory macrophage activation, there is an accumulation of the Krebs cycle intermediates succinate and citrate, and the Krebs cycle–derived metabolite itaconate. These metabolites have
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Minis, Adi, and Hermann Steller. "Krebs Cycle Moonlights in Caspase Regulation." Developmental Cell 37, no. 1 (2016): 1–2. http://dx.doi.org/10.1016/j.devcel.2016.03.016.

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O’Neill, Luke A. J. "A Broken Krebs Cycle in Macrophages." Immunity 42, no. 3 (2015): 393–94. http://dx.doi.org/10.1016/j.immuni.2015.02.017.

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Ritson, Dougal J. "A cyanosulfidic origin of the Krebs cycle." Science Advances 7, no. 33 (2021): eabh3981. http://dx.doi.org/10.1126/sciadv.abh3981.

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The centrality of the Krebs cycle in metabolism has long been interpreted as evidence of its antiquity, and consequently, questions regarding its provenance, and whether it initially functioned as a cycle or not, have received much attention. The present report shows that prebiotic oxidation of α-hydroxy carboxylates can be achieved by UV photolysis of a simple geochemical species (HS−), which leads to α-oxo carboxylates that feature in the Krebs cycle and glyoxylate shunt. Further reaction of these products leads to almost all intermediates of the Krebs cycle proper, succinate semialdehyde by
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Radzikh, Igor, Erica Fatica, Jillian Kodger, Rohan Shah, Ryan Pearce, and Yana I. Sandlers. "Metabolic Outcomes of Anaplerotic Dodecanedioic Acid Supplementation in Very Long Chain Acyl-CoA Dehydrogenase (VLCAD) Deficient Fibroblasts." Metabolites 11, no. 8 (2021): 538. http://dx.doi.org/10.3390/metabo11080538.

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Very long-chain acyl-CoA dehydrogenase deficiency (VLCADD, OMIM 609575) is associated with energy deficiency and mitochondrial dysfunction and may lead to rhabdomyolysis and cardiomyopathy. Under physiological conditions, there is a fine balance between the utilization of different carbon nutrients to maintain the Krebs cycle. The maintenance of steady pools of Krebs cycle intermediates is critical formitochondrial energy homeostasis especially in high-energy demanding organs such as muscle and heart. Even-chain dicarboxylic acids are established as alternative energy carbon sources that reple
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Dissertations / Theses on the topic "Intermediaires du cycle de Krebs"

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Romano, Manuela. "Stage-specific changes in the Krebs cycle network regulate human erythroid differentiation." Thesis, Montpellier, 2018. http://www.theses.fr/2018MONTT077.

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Le processus conduisant à la prolifération et différenciation des cellules souches hématopoïétiques (CSH) en cellules de toutes les lignées sanguines s’appelle l’hématopoïèse. Bien que l'engagement des CSH soit régi par les cytokines, les facteurs de transcription, les modificateurs épigénétiques et la niche des CSH, notre groupe a constaté que leur engagement vers la lignée érythroïde dépendait aussi du métabolisme de la glutamine. La glutaminolyse contribue à la biosynthèse des nucléotides de novo ainsi qu’à la production de l'alpha-kétoglutarate (αKG), intermédiaire métabolique du cycle TCA
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Brière, Jean-Jacques. "Le cycle de Krebs dans la pathologie humaine : succinate désydrogénase et tumorigenèse." Paris 5, 2006. http://www.theses.fr/2006PA05P621.

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Les gènes des sous-unités B, C et D de la succinate déshydrogénase(SDH) sont des suppresseurs de tumeurs. Dans ces tumeurs l’activité de la SDH est quasi nulle et le facteur de transcription lié à l’hypoxie HIF1α est stabilisé. Nous avons montré que la perte d’activité de la SDH conduit à une accumulation de succinate. La stabilisation de HIF1α est causée par l’inhibition, la prolyl hydroxylase (PHD), sensible aux superoxydes, qui utilise le fer réduit comme cofacteur, l’α cétoglutarate comme substrat et produit du succinate. Ni la quantité de fer ou son état de réduction, ni la surproduction
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Janin, Maxime. "Phénotype métabolique des tumeurs associées à des anomalies du cycle de Krebs." Thesis, Sorbonne Paris Cité, 2015. http://www.theses.fr/2015PA05T025/document.

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Le cycle de Krebs occupe une place centrale dans le métabolisme cellulaire et est le point de jonction de nombreuses voies essentielles. Depuis le début des années 2000, un lien a été démontré entre l’apparition de certains cancers et des mutations affectant des gènes codant pour des enzymes du cycle de Krebs, i.e., la succinate déshydrogénase, la fumarase ou les iso-enzymes 1 et 2 de l'isocitrate déshydrogénase (IDH). Les mutations des gènes IDH sont présentes dans 15 à 20 % des leucémies myéloïdes aigues (LAM) et jusqu'à 80 % dans certains gliomes. Ces mutations affectent le site actif des e
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BOURGERON, THOMAS. "Exploration enzymologique et moleculaire d'atteintes de la chaine respiratoire et du cycle de krebs." Paris 6, 1994. http://www.theses.fr/1994PA066060.

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La premiere partie de ce travail est la mise au point d'une technique d'isolement des mitochondries de cellules en culture permettant la caracterisation biochimique des mitochondries isolees de lignees lymphoblastoides humaines et l'etude de la synthese proteique mitochondriale. Disposant de cet outil, l'evolution des activites de la chaine respiratoire et de la proportion d'adnmt mute a ete etudiee dans les cellules en culture d'un enfant atteint d'un syndrome de pearson et presentant une deletion de l'adnmt a l'etat heteroplasmique. Une retombee immediate de cette etude a reside dans la poss
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Desbeaux-Salviat, Béatrice. "Un modèle biochimique, le cycle de Krebs : découverte, diffusion, enseignement à l'université et au lycée." Paris 11, 1997. http://www.theses.fr/1997PA112204.

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Cette recherche vise a traduire en actions pédagogiques un questionnement relatif au savoir biochimique. Une réflexion epistemologique et historique sur les contenus de la biochimie montre en quoi les objectifs de cette discipline différent de ceux de la chimie et de ceux de la biologie, et pose les difficultés d'une approche expérimentale des phénomènes complexes du vivant. Le cycle de l'acide citrique, passage oblige dans l'étude du métabolisme, désignant un processus biochimique représente par des modèles, a été choisi comme objet d'étude approfondi permettant réflexion et propositions pour
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Meek, David J. J. "Molecular and genetic characterization of putative TCA cycle operons on Sinorhizobium meliloti." Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=33808.

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Genetic mapping of pDS15 revealed that this cosmid clone carries the Sinorhizobium meliloti TCA cycle genes mdh, sucCDAB, sdhAB and part of lpdA. Three genes (mdh, sucC , and sucA) were completely sequenced and submitted to GenBank. The nucleotide and amino acid sequences of the TCA cycle genes encoded on pDS15 were aligned and found to be highly homologous with other closely related rhizobial species. S. meliloti cells grown in LBmc express the mdh-sucCDAB operon as one transcript, based on RT-PCR results. Alternative sigma factor sigma54 was not found to have a role in mdh-sucCDAB expression
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Wagner, Tristan. "Structural insights into mycobacterial central carbon metabolism : the catalytic mechanisms and regulatory properties of α-ketoglutarate decarboxylase (KGD)". Paris 6, 2011. http://www.theses.fr/2011PA066421.

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Le nœud métabolique au croisement du cycle de Krebs et de l’assimilation de l’azote est régi par l’α-kétoglutarate déshydrogénase (KDH), un complexe constitué des composants E1o, E2o et E3. Chez les actinobactéries le composant E2o n’existe pas, son domaine catalytique est fusionné à E1o (α-kétoglutarate décarboxylase : KGD). La liaison de KGD au complexe de la pyruvate déshydrogénase assure l’activité KDH dont la régulation est cruciale pour coordonner le flux de carbone. Les corynebactéries ont développé un système de contrôle unique par GarA, une protéine à domaine en tête de fourche (ce do
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Cheung, Kwok-ho Alvin, and 張國豪. "The role of human sodium dicarboxylate cotransporter in oxidative stress." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B27769070.

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Laurent, Sophie. "Développement des hétérocystes chez la cyanobactérie Anabaena PCC 7120 : Identification du signal initiateur de la différenciation. Rôle de PII dans le métabolisme des hétérocytes." Aix-Marseille 2, 2005. http://www.theses.fr/2005AIX22059.

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Varma, Sreejith Jayasree. "Mimicking C-C bond forming reactions of core metabolism." Thesis, Strasbourg, 2018. http://www.theses.fr/2018STRAF038/document.

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Toutes les formes de vie assemblent et désassemblent continuellement des composés chimiques via un processus de consommation d'énergie appelé métabolisme. Le métabolisme est généralement modélisé en chimie et biologie par un cycle. Ce modèle dynamique traduit la transformation de composés de base en une cascade de produits appelés métabolites. Celui-ci est comparable à un ouragan à l’échelle moléculaire. De manière analogique et imagée un cyclone est constitué de deux éléments, l'air et l'eau, et transforme l’environnement qui l’entoure par un processus endothermique (consommateur d’énergie).
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Books on the topic "Intermediaires du cycle de Krebs"

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Osada, Shōmatsu. Kuensan kenkōhō de 100-sai made ikiru. Metamoru Shuppan, 2002.

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2

Sir, Krebs Hans Adolf, Kay J, and Weitzman P. D. J, eds. Krebs' citric acid cycle: Half a century and still turning. Biochemical Society, 1987.

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Kay, J. Krebs Citric Acid Cycle: Half a Century and Still Turning (Biochemical Society Symposia Se .: No 54). Biochemical Society, 1987.

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North Atlantic Treaty Organization. Scientific Affairs Division (Corporate Author) and Asterios S. Tsiftsoglou (Editor), eds. Tumor Biology: Regulation of Cell Growth, Differentiation, and Genetics in Cancer (Nato a S I Series Series H, Cell Biology). Springer-Verlag Telos, 1996.

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1948-, Tsiftsoglou Asterios S., North Atlantic Treaty Organization. Scientific Affairs Division., and NATO Science Institute "Regulation of Cell Growth, Differentiation, and Genetics in Cancer" (1995 : Porto Karras, Chalkidikē, Greece), eds. Tumor biology: Regulation of cell growth, differentiation, and genetics in cancer. Springer, 1996.

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Veech, Richard L., and M. Todd King. Alzheimer’s Disease. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0026.

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Deficits in cerebral glucose utilization in Alzheimer’s disease (AD) arise decades before cognitive impairment and accumulation of amyloid plaques and neurofibrillary tangles in brain. Addressing this metabolic deficit has greater potential in treating AD than targeting later disease processes – an approach that has failed consistently in the clinic. Cerebral glucose utilization requires numerous enzymes, many of which have been shown to decline in AD. Perhaps the most important is pyruvate dehydrogenase (PDH), which links glycolysis with the Krebs cycle and aerobic metabolism, and whose activ
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Jancura, Daniel, and Erik Sedlák. Bioenergetika. Univerzita Pavla Jozefa Šafárika, Vydavateľstvo ŠafárikPress, 2021. http://dx.doi.org/10.33542/be2021-0022-6.

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Prekladaný vysokoškolský učebný text „Bioenergetika“ by mal slúžiť ako úvod do problematiky štúdia v oblasti bioenergetiky. Táto vedná oblast je v súčasnosti vysoko aktuálna, pretože výsledky získané bioenergetickým výskumom v uplynulých rokoch zreteľne ukazujú, že bioenergetické procesy prebiehajúce v živých systémoch neslúžia “len” na transformáciu energie, ale ovplyvňujú aj priebeh procesov ako sú apoptóza, starnutie, vznik a rozvoj mnohých ochorení (predovšetkým neurodegeneratívnych). Tieto skutočnosti jednoznačne naznačujú potrebu existencie kvalitných učebných textov, ktoré by prijateľný
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Book chapters on the topic "Intermediaires du cycle de Krebs"

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Gooch, Jan W. "Krebs Cycle." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14080.

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Ochs, Raymond S. "The Krebs Cycle." In Biochemistry, 2nd ed. CRC Press, 2021. http://dx.doi.org/10.1201/9781003029649-10.

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Alemanno, Fernando. "Tricarboxylic Acids Cycle or Krebs Cycle." In Biochemistry for Anesthesiologists and Intensivists. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-26721-6_3.

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Hederstedt, Lars. "The Krebs Citric Acid Cycle." In Bacillus subtilis and Other Gram-Positive Bacteria. ASM Press, 2014. http://dx.doi.org/10.1128/9781555818388.ch13.

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Sonenshein, Abraham L. "The Krebs Citric Acid Cycle." In Bacillus subtilis and Its Closest Relatives. ASM Press, 2014. http://dx.doi.org/10.1128/9781555817992.ch12.

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Morava, Eva, and Rosalba Carrozzo. "Disorders of the Krebs Cycle." In Physician's Guide to the Diagnosis, Treatment, and Follow-Up of Inherited Metabolic Diseases. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-40337-8_20.

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Fuchs, G. "Alternatives to the Calvin Cycle and the Krebs Cycle in Anaerobic Bacteria: Pathways with Carbonylation Chemistry." In The Molecular Basis of Bacterial Metabolism. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75969-7_2.

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De Lonlay, Pascale, Sandrine Dubois, Vassili Valayannopoulos, Eliane Depondt, Chris Ottolenghi, and Daniel Rabier. "Déficits énergétiques (déficits de la chaîne respiratoire, PDH, PC, cycle de Krebs)." In Prise en charge médicale et diététique des maladies héréditaires du métabolisme. Springer Paris, 2013. http://dx.doi.org/10.1007/978-2-8178-0046-2_19.

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Saavedra, Francisco, Ekaterina Boyarchuk, Francisca Alvarez, Geneviève Almouzni, and Alejandra Loyola. "Metabolic Deregulations Affecting Chromatin Architecture: One-Carbon Metabolism and Krebs Cycle Impact Histone Methylation." In RNA Technologies. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-14792-1_23.

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Tcherkez, Guillaume. "Tracking the Orchestration of the Tricarboxylic Acid Pathway in Plants, 80 Years After the Discovery of the Krebs Cycle." In Advances in Photosynthesis and Respiration. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-68703-2_14.

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Conference papers on the topic "Intermediaires du cycle de Krebs"

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Li, Xiaozhong, Long Wang, Ying Liu, and Yong Li. "Modeling the Krebs Cycle based on hybrid Petri net." In The 2015 11th International Conference on Natural Computation. IEEE, 2015. http://dx.doi.org/10.1109/icnc.2015.7378159.

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Shelton, Laura M., Cheryl L. Strelko, Mary F. Roberts, and Thomas N. Seyfried. "Abstract 53: Krebs cycle substrate-level phosphorylation drives metastatic cancer cells." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-53.

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Kho, Eun-Young, Carolina B. Livi, Phillip Buckhaults, et al. "Abstract 53: Epigenetic silencing of krebs cycle metabolism in kidney cancer." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-53.

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Zhao, Liang, Matthew Arwood, Min-Hee Oh, et al. "Abstract 4376: Targeting glutamine metabolism disables Warburg physiology by inhibiting proximal glycolysis and Krebs cycle rewiring." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-4376.

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Zhao, Liang, Matthew Arwood, Min-Hee Oh, et al. "Abstract 4376: Targeting glutamine metabolism disables Warburg physiology by inhibiting proximal glycolysis and Krebs cycle rewiring." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-4376.

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Reut, Dmitry, Michail Baranov, and Sergey Mayorov. "Development of "Krebs Cycle" Concept Conformably to Change Management in Large-Scale Systems and Prospects of This Subject for Viable Systems." In 2019 Twelfth International Conference "Management of large-scale system development" (MLSD). IEEE, 2019. http://dx.doi.org/10.1109/mlsd.2019.8911001.

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