Relevant bibliographies by topics / Intermittent systemic hypoxia

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Intermittent systemic hypoxia'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Intermittent systemic hypoxia"

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Artūrs, Paparde, Plakane Līga, and Circenis Kristaps. "Effect of Short Systemic Intermittent Hypoxia on Systemic Hemodynamics Blunted in Cutaneous Microcirculation." Cardiology and Angiology: An International Journal 6, no. 2 (2017): 1–11. https://doi.org/10.9734/CA/2017/33514.

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<strong>Background</strong><strong>:</strong> Hypoxic stimulus induces a homeostatic disruption to enhance physiological adaptation. Blood flow in the microcirculation plays an important role in maintaining healthy tissues by delivering oxygen. The cutaneous microcirculations responses to short systemic hypoxia and especially its duration are poorly understood; however the mechanisms of this phenomenon are at the microcirculatory level. The aim of our study was to determine the short systemic intermittent hypoxia's influence on blood flow in skin, local regulatory mechanism fluctuations and ch
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Torres, Marta, Mauricio Rojas, Noelia Campillo, et al. "Parabiotic model for differentiating local and systemic effects of continuous and intermittent hypoxia." Journal of Applied Physiology 118, no. 1 (2015): 42–47. http://dx.doi.org/10.1152/japplphysiol.00858.2014.

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Hypoxia can be damaging either because cells are directly sensitive to low oxygen pressure in their local microenvironment and/or because they are exposed to circulating factors systemically secreted in response to hypoxia. The conventional hypoxia model, breathing hypoxic air, does not allow one to distinguish between these local and systemic effects. Here we propose and validate a model for differentially applying local and systemic hypoxic challenges in an animal. We used parabiosis, two mice sharing circulation by surgical union through the skin, and tested the hypothesis that when one of
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Hunyor, Imre, and Kristina M. Cook. "Models of intermittent hypoxia and obstructive sleep apnea: molecular pathways and their contribution to cancer." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 315, no. 4 (2018): R669—R687. http://dx.doi.org/10.1152/ajpregu.00036.2018.

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Obstructive sleep apnea (OSA) is common and linked to a variety of poor health outcomes. A key modulator of this disease is nocturnal intermittent hypoxia. There is striking epidemiological evidence that patients with OSA have higher rates of cancer and cancer mortality. Small-animal models demonstrate an important role for systemic intermittent hypoxia in tumor growth and metastasis, yet the underlying mechanisms are poorly understood. Emerging data indicate that intermittent hypoxia activates the hypoxic response and inflammatory pathways in a manner distinct from chronic hypoxia. However, t
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Fletcher, Eugene C. "Invited Review: Physiological consequences of intermittent hypoxia: systemic blood pressure." Journal of Applied Physiology 90, no. 4 (2001): 1600–1605. http://dx.doi.org/10.1152/jappl.2001.90.4.1600.

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One of the major manifestations of obstructive sleep apnea is profound and repeated hypoxia during sleep. Acute hypoxia leads to stimulation of the peripheral chemoreceptors, which in turn increases sympathetic outflow, acutely increasing blood pressure. The chronic effect of these repeated episodic or intermittent periods of hypoxia in humans is difficult to study because chronic cardiovascular changes may take many years to manifest. Rodents have been a tremendous source of information in short- and long-term studies of hypertension and other cardiovascular diseases. Recurrent short cycles o
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McNulty, P. H., C. Ng, W. X. Liu, et al. "Autoregulation of myocardial glycogen concentration during intermittent hypoxia." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 271, no. 2 (1996): R311—R319. http://dx.doi.org/10.1152/ajpregu.1996.271.2.r311.

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During hypoxia, the heart consumes glycogen to generate ATP. Tolerance of repetitive hypoxia logically requires prompt replenishment of glycogen, a process whose regulation is not fully understood. To examine this, we imposed a defined hypoxic stimulus on the rat heart while varying its workload. In intact rats, hypoxia reduced myocardial glycogen approximately 30% and increased both the fraction of glycogen synthase in its physiologically active (GS I) form (from 0.24 +/- 0.06 to 0.82 +/- 0.07; P &lt; 0.005) and glycogen synthesis (from 0.087 +/- 0.011 to 0.375 +/- 0.046 mumol.g-1.min-1; P &l
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Zhang, Jing, Xu Guo, Yanwei Shi, Jing Ma, and Guangfa Wang. "Intermittent hypoxia with or without hypercapnia is associated with tumorigenesis by decreasing the expression of brain derived neurotrophic factor and miR-34a in rats." Chinese Medical Journal 127, no. 1 (2014): 43–47. http://dx.doi.org/10.3760/cma.j.issn.0366-6999.20131683.

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Background Very recent studies revealed that obstructive sleep apnoea (OSA) is a contributor of the increased incidence and mortality of cancer in humans, but mechanisms of how OSA promotes tumorigenesis remains largely unknown. We investigated whether intermittent hypoxia with and without hypercapnia plays a role in tumorigenesis. Methods First, Sprague-Dawley (SD) male rats (12 weeks old) were subjected to different hypoxia exposures: intermittent hypoxia and intermittent hypoxia with hypercapnia; continuous hypoxia and normal air. The systemic application of chronic fast rate hypoxia with o
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Reinke, Christian, Shannon Bevans-Fonti, Luciano F. Drager, Mi-Kyung Shin, and Vsevolod Y. Polotsky. "Effects of different acute hypoxic regimens on tissue oxygen profiles and metabolic outcomes." Journal of Applied Physiology 111, no. 3 (2011): 881–90. http://dx.doi.org/10.1152/japplphysiol.00492.2011.

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Obstructive sleep apnea (OSA) causes intermittent hypoxia (IH) during sleep. Both obesity and OSA are associated with insulin resistance and systemic inflammation, which may be attributable to tissue hypoxia. We hypothesized that a pattern of hypoxic exposure determines both oxygen profiles in peripheral tissues and systemic metabolic outcomes, and that obesity has a modifying effect. Lean and obese C57BL6 mice were exposed to 12 h of intermittent hypoxia 60 times/h (IH60) [inspired O2 fraction (FiO2) 21–5%, 60/h], IH 12 times/h (FiO2 5% for 15 s, 12/h), sustained hypoxia (SH; FiO2 10%), or no
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Peng, Ying-Jie, and Nanduri R. Prabhakar. "Effect of two paradigms of chronic intermittent hypoxia on carotid body sensory activity." Journal of Applied Physiology 96, no. 3 (2004): 1236–42. http://dx.doi.org/10.1152/japplphysiol.00820.2003.

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Reflexes arising from the carotid bodies may play an important role in cardiorespiratory changes evoked by chronic intermittent hypoxia (CIH). In the present study, we examined whether CIH affects the hypoxic sensing ability of the carotid bodies and, if so, by what mechanisms. Experiments were performed on adult male rats (Sprague-Dawley, 250–300 g) exposed to two paradigms of CIH for 10 days: 1) multiple exposures to short durations of intermittent hypoxia per day (SDIH; 15sof5%O2 + 5 min of 21% O2, 9 episodes/h, 8 h/day) and 2) single exposure to longer durations of intermittent hypoxia per
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Iturriaga, Rodrigo, Rodrigo Del Rio, and Julio Alcayaga. "Carotid Body Inflammation: Role in Hypoxia and in the Anti-inflammatory Reflex." Physiology 37, no. 3 (2022): 128–40. http://dx.doi.org/10.1152/physiol.00031.2021.

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Emergent evidence indicates that the carotid body (CB) chemoreceptor may sense systemic inflammatory molecules and is an afferent arm of the anti-inflammatory reflex. Moreover, a proinflammatory milieu within the CB is involved in the enhanced CB chemosensory responsiveness to oxygen following sustained and intermittent hypoxia. In this review, we focus on the physiopathological participation of CBs in inflammatory diseases, such as sepsis and intermittent hypoxia.
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Uzun, Andreea-Bianca, Mădălina Gabriela Iliescu, Liliana-Elena Stanciu, et al. "Effectiveness of Intermittent Hypoxia–Hyperoxia Therapy in Different Pathologies with Possible Metabolic Implications." Metabolites 13, no. 2 (2023): 181. http://dx.doi.org/10.3390/metabo13020181.

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Intermittent oxygen therapy (IHT), initially used in the hypoxic administration variant, has been shown to be effective in various pathologies studied, from cardiopulmonary to vascular and metabolic pathologies and more. IHT used to prevent and treat various diseases has thus gained more and more attention as the years have passed. The mechanisms underlying the beneficial effects have been investigated at multiple biological levels, from systemic physiological reactions to genomic regulation. In the last decade, a new method of intermittent oxygen therapy has been developed that combines hypox
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Dissertations / Theses on the topic "Intermittent systemic hypoxia"

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Détrait, Maximin. "Impact de l'activité sympathique dans l'aggravation de la cardiopathie ischémique causée par l'hypoxie intermittente Intermittent Hypoxia Triggers Early Cardiac Remodeling and Contractile Dysfunction in the Time‐Course of Ischemic Cardiomyopathy in Rats Short‐term Intermittent Hypoxia Induces Simultaneous Systemic Insulin Resistance and Higher Cardiac Contractility in Lean Mice." Thesis, Université Grenoble Alpes, 2020. https://thares.univ-grenoble-alpes.fr/2020GRALV035.pdf.

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L’association d’un trouble respiratoire au cours du sommeil (TRS) chez des patients présentant une cardiopathie ischémique est un facteur de mauvais pronostic. Au cours des TRS, les patients souffrent d’épisodes de désaturation-resaturation aussi appelés hypoxie intermittente (HI) et décrite comme l’une des caractéristiques des TRS la plus délétère sur le système cardiovasculaire. Les mécanismes induits par l’HI et responsables des complications cardiovasculaires ne sont pas totalement explicités et nécessitent d’être affinés afin de proposer de nouvelles stratégies thérapeutiques. Aujourd’hui
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Coelho, Nuno Filipe Ramos. "Aryl hydrocarbon receptor (ahr): a therapeutic target for systemic hypertension associated with chronic intermittent hypoxia." Doctoral thesis, 2020. http://hdl.handle.net/10362/102012.

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RESUMO: A síndrome da apneia obstrutiva do sono (SAOS) é uma perturbação respiratória relacionada com o sono, que tem vindo a ser cada vez mais prevalente. Esta condição clínica é responsável por uma multiplicidade de comorbilidades, com destaque para a hipertensão arterial sistémica (HTA). A etiopatogenia da SAOS inclui quatro principais características clínicas, nomeadamente a hipóxia crónica intermitente (HCI), a hipercapnia, o aumento da pressão intratorácica e a fragmentação do sono. Destas, a HCI assume-se como um factor chave no desenvolvimento de HTA. A SAOS é uma das causas pri
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Books on the topic "Intermittent systemic hypoxia"

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Jacquemyn, Yves, and Anneke Kwee. Antenatal and intrapartum fetal evaluation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0006.

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Antenatal and intrapartum fetal monitoring aim to identify the beginning of the process of fetal hypoxia before irreversible fetal damage has taken place. Fetal movement counting by the mother has not been reported to be of any benefit. The biophysical profile score, incorporating ultrasound and fetal heart rate monitoring, has not been proven to reduce perinatal mortality in randomized trials. Doppler ultrasound allows the exploration of the perfusion of different fetal organ systems and provides data on possible hypoxia and fetal anaemia. Maternal uterine artery Doppler can be used to select
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Book chapters on the topic "Intermittent systemic hypoxia"

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Peng, Yingjie, Guoxiang Yuan, Jeffrey L. Overholt, Ganesh K. Kumar, and Nanduri R. Prabhakar. "Systemic and Cellular Responses to Intermittent Hypoxia: Evidence for Oxidative Stress and Mitochondrial Dysfunction." In Advances in Experimental Medicine and Biology. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4419-9280-2_71.

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Yan, Qiaozhen, and Liping Dong. "Chronic Intermittent Hypoxia Training on Rat Myocardial Mitochondria Atpase Impact Study." In Advances in Intelligent Systems and Computing. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-2568-1_183.

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Dugral, Esra. "Obstructive Sleep Apnea Syndrome (OSAS) and Menopause." In Insomnia From Childhood to Elderly [Working Title]. IntechOpen, 2024. http://dx.doi.org/10.5772/intechopen.1004412.

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Obstructive Sleep Apnea Syndrome (OSAS) is a sleep disorder in which intermittent hypoxia and systemic inflammation mechanisms are prominent, leading to many serious health problems. Cardiovascular and metabolic diseases are among the leading clinical problems caused by OSAS. When left untreated, its reflection on society is increased morbidity and mortality rates. For this reason, many clinical studies have focused on the reasons that increase the risk of OSAS. Menopause has taken its place in research as one of these reasons. The fluctuation in the secretion of female reproductive hormones m
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Čekerevac, Ivan, and Bojan Djokić. "OSA in Patients with Severe Asthma-Alternative Overlap Syndrome." In Severe Asthma Forum - Monitoring and Treatable Traits in Severe Asthma. Založba Univerze na Primorskem, 2013. http://dx.doi.org/10.26493/978-961-293-297-8.47-51.

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Bronchial asthma and obstructive sleep apnea (OSA) are common chronic diseases of the respiratory system. During the last decade, there has been a growing interest in the connection between these two disorders. Studies show that asthma patients are at increased risk for OSA, and the prevalence is on average around 70% in severe asthma patients. Rhinitis, gastroesophageal reflux disease and obesity are common comorbidities for both entities. OSA is an independent factor in the exacerbation of asthma and each condition in itself can contribute to the exacerbation of the other. Asthma, by its mec
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F. Rodriguez, Ramón, Robert J. Aughey, and François Billaut. "The Respiratory System during Intermittent-Sprint Work: Respiratory Muscle Work and the Critical Distribution of Oxygen." In Respiratory Physiology. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.91207.

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In healthy individuals at rest and while performing moderate-intensity exercise, systemic blood flow is distributed to tissues relative to their metabolic oxygen demands. During sustained high-intensity exercise, competition for oxygen delivery arises between locomotor and respiratory muscles, and the heightened metabolic work of breathing, therefore, contributes to limited skeletal muscle oxygenation and contractility. Intriguingly, this does not appear to be the case for intermittent-sprint work. This chapter presents new evidence, based on inspiratory muscle mechanical loading and hypoxic g
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Bakovic Darija, Palada Ivan, and Dujic Zeljko. "Apnea Diving." In Biomedical and Health Research. IOS Press, 2010. https://doi.org/10.3233/978-1-60750-497-9-431.

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Men and women have been diving for thousands of years to gather a variety of products, including food, sponges, and pearls, to conduct salvage and military operations, and to explore. In 1911, one of the first freediving competitions was held when a Greek fisherman, Yorgos Haggi Statti, was offered a few dollars to dive and to rescue the anchor of an Italian ship which had become stuck in the Aegean Sea. The Italian ship was set free and Yorgos became known as the &amp;ldquo;father of freediving&amp;rdquo;. After World War II, breath hold diving became an international sport where athletes tes
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Conference papers on the topic "Intermittent systemic hypoxia"

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Acosta Gutierrez, Carmen Maria, Pedro Landete Rodriguez, Beatriz Aldave Orzaiz, et al. "Role of intermittent or continuous hypoxia in systemic inflammation measured by echography." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2505.

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Cao, Weijun, Patty J. Lee, and Vahid Mohsenin. "HO-1 Prevents Intermittent Hypoxia Induced Systemic And Pulmonary Hypertension By Ameliorating Oxidants And Endoglin Expression." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6632.

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King, A., C. Arnaud, G. Vial, et al. "The effect of the GLP-1 analogue Liraglutide on intermittent hypoxia-induced systemic insulin resistance in vivo." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.2182.

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Klyuchnikova, E. A., and M. V. Balykin. "Intermittent normobaric hypoxia as a method of correction and prevention of functional changes in old age." In VIII Vserossijskaja konferencija s mezhdunarodnym uchastiem «Mediko-fiziologicheskie problemy jekologii cheloveka». Publishing center of Ulyanovsk State University, 2021. http://dx.doi.org/10.34014/mpphe.2021-118-121.

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The study involved men aged 60-65 years. The course of intermittent normobaric hypoxia (APG) was carried out for 3 weeks with breathing hypoxic gas mixture with 18-15-13-10% O2. Before and after the course, the reactivity of the cardiovascular system and blood system, physical performance were evaluated. It has been established that the course of APG increases oxidative metabolism, leads to a decrease in the level of sugar, total lipids and low-density lipids in the blood, an increase in the number of red blood cells, helps to reduce and stabilize blood pressure, increase physical performance
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Harki, O., Q. Boete, J. L. Pépin, et al. "Impact of Intermittent Hypoxia on Vascular Structure and Function in Animal Models: A Systematic Review and Metaanalysis." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a4685.

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