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Journal articles on the topic 'Interstitium and hypoxic respiratory failure'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Yanagisawa, Kei, Akiko Kameyama, Hiroshi Miyama, et al. "Intravascular Large B-Cell Lymphoma in Acute Hypoxic Respiratory Failure." Case Reports in Critical Care 2023 (August 12, 2023): 1–4. http://dx.doi.org/10.1155/2023/9192396.

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Intravascular large B-cell lymphoma, an extranodal large B-cell lymphoma, is a rare hematological malignancy with only a few reports of lung involvement. We report a case of intravascular large B-cell lymphoma with acute hypoxic respiratory failure and interstitial lung disease diagnosed via random skin biopsies. A 54-year-old woman presented with fever, cough, and dyspnea. Computed tomography imaging revealed findings concerning interstitial lung disease. The patient’s respiratory status worsened despite the treatment with antibiotics and steroids. Generalized edema and thrombocytopenia also

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Krishnappa, Manjunath Hunasenahalli, Prasanna Kumar Thimmarayappa, Vivek Nangia, and Rajat Chatterji. "Outcome of ventilation in patients with hypercapnic respiratory failure in IPF patients: A prospective study." Asian Journal of Medical Sciences 12, no. 9 (2021): 111–15. http://dx.doi.org/10.3126/ajms.v12i9.37155.

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Background: Diffuse parenchymal lung disease (DPLD) is the inflammation and fibrosis of lung interstitium, resulting in respiratory failure. The rate of hypoxic respiratory failure is high as the disease progresses. In idiopathic pulmonary fibrosis (IPF) patients, acute deterioration leads to type 2 respiratory failure, etiology and management of which remains to be completely understood. Aims and Objectives: To identify the causes of type 2 respiratory failure and assess the outcome of invasive and non-invasive ventilation in patients with IPF. Materials and Methods: This prospective single c

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3

Price, Susanna, Suveer Singh, Stephane Ledot, et al. "Respiratory management in severe acute respiratory syndrome coronavirus 2 infection." European Heart Journal: Acute Cardiovascular Care 9, no. 3 (2020): 229–38. http://dx.doi.org/10.1177/2048872620924613.

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The severe acute respiratory syndrome coronavirus 2 pandemic is to date affecting more than a million of patients and is challenging healthcare professionals around the world. Coronavirus disease 2019 may present with a wide range of clinical spectrum and severity, including severe interstitial pneumonia with high prevalence of hypoxic respiratory failure requiring intensive care admission. There has been increasing sharing experience regarding the patient’s clinical features over the last weeks which has underlined the need for general guidance on treatment strategies. We summarise the eviden

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Bruminhent, Jackrapong, Shahla Yassir, and James Pippim. "Acute Interstitial Pneumonia (Hamman-Rich Syndrome) as a Cause of Idiopathic Acute Respiratory Distress Syndrome." Case Reports in Medicine 2011 (2011): 1–4. http://dx.doi.org/10.1155/2011/628743.

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Hamman-Rich syndrome, also known as acute interstitial pneumonia, is a rare and fulminant form of idiopathic interstitial lung disease. It should be considered as a cause of idiopathic acute respiratory distress syndrome. Confirmatory diagnosis requires demonstration of diffuse alveolar damage on lung histopathology. The main treatment is supportive care. It is not clear if glucocorticoid therapy is effective in acute interstitial pneumonia. We report the case of a 77-year-old woman without pre-existing lung disease who initially presented with mild upper respiratory tract infection and then p

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Takekoshi, Daisuke, Seiichi Kobayashi, and Masaru Yanai. "Noninvasive Positive Pressure Ventilation (NIPPV) for Acute Hypoxic Respiratory Failure in Patients With Interstitial Pneumonia." Chest 148, no. 4 (2015): 306A. http://dx.doi.org/10.1378/chest.2269540.

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Helmink, Austin, Samir Atiya, and Ernesto Martinez Duarte. "Pulmonary Alveolar Microlithiasis: A Unique Case of Familial PAM Complicated by Transplant Rejection." Case Reports in Pathology 2021 (April 5, 2021): 1–4. http://dx.doi.org/10.1155/2021/6674173.

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Background. Pulmonary alveolar microlithiasis (PAM) is a rare lung disease characterized by the deposition of calcium phosphate microliths or calcospherites, within the alveolar airspace. Typical imaging findings demonstrate a “sandstorm” appearance due to bilateral, interstitial sand-like micronodularities with basal predominance. Methods and Results. We describe an unusual case of a 48-year-old male with severe, familial PAM ultimately treated with a bilateral lung transplant. Conclusions. PAM is a rare lung disease caused by a mutation in the SLC34A2 gene, which encodes for a sodium-phospha

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West, JB, GL Colice, YJ Lee, et al. "Pathogenesis of high-altitude pulmonary oedema: direct evidence of stress failure of pulmonary capillaries." European Respiratory Journal 8, no. 4 (1995): 523–29. http://dx.doi.org/10.1183/09031936.95.08040523.

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The pathogenesis of high-altitude pulmonary oedema (HAPE) is disputed. Recent reports show a strong correlation between the occurrence of HAPE and pulmonary artery pressure, and it is known that the oedema is of the high-permeability type. We have, therefore, proposed that HAPE is caused by ultrastructural damage to pulmonary capillaries as a result of stress failure of their walls. However, no satisfactory electron microscopy studies are available in patients with HAPE, and animal models are difficult to find. Madison strain Sprague-Dawley rats show a brisk pulmonary pressure response to acut

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8

Young, Iven H., and Peter T. P. Bye. "Gas Exchange in Disease: Asthma, Chronic Obstructive Pulmonary Disease, Cystic Fibrosis, and Interstitial Lung Disease." Comprehensive Physiology 1, no. 2 (2011): 663–97. https://doi.org/10.1002/j.2040-4603.2011.tb00332.x.

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AbstractVentilation‐perfusion (a/) inequality is the underlying abnormality determining hypoxemia and hypercapnia in lung diseases. Hypoxemia in asthma is characterized by the presence of low a/ units, which persist despite improvement in airway function after an attack. This hypoxemia is generally attenuated by compensatory redistribution of blood flow mediated by hypoxic vasoconstriction and changes in cardiac output, however, mediator release and bronchodilator therapy may cause deterioration. Patients with chronic obstructive pulmonary disease have more complex patterns of a/ inequality, w

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9

Liu, Yuming, Jing Zhang, Yayue Hu, et al. "BI 1015550 Improves Silica-Induced Silicosis and LPS-Induced Acute Lung Injury in Mice." Molecules 30, no. 6 (2025): 1311. https://doi.org/10.3390/molecules30061311.

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Silicosis is an interstitial lung disease (ILD) caused by prolonged inhalation of silica particles. Acute lung injury (ALI) is a critical clinical syndrome involving bilateral lung infiltration and acute hypoxic respiratory failure. However, there is currently no effective treatment for these two diseases. Previous research has established that cyclic adenosine monophosphate (cAMP) is pivotal in the pathogenesis of silicosis and acute lung injury. Phosphodiesterase 4 (PDE4) is a hydrolase enzyme of cAMP, and BI 1015550, as an inhibitor of PDE4B, is expected to be a candidate drug for treating

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10

Amin, Amee A., Erica Haught, and Youmna Mousattat. "Do Not Huff, Puff, or Vape That Stuff: Interstitial Airspace Disease in a Teenager." Case Reports in Pediatrics 2020 (December 2, 2020): 1–4. http://dx.doi.org/10.1155/2020/8822362.

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A 17-year-old previously healthy male was admitted to the hospital for intractable and persistent vomiting, fever, cough, abdominal pain, and intermittent diarrhea and dehydration. He presented with severe chest pain and O2 desaturations up to 80% on room air. An infectious (including a nasopharyngeal swab), GI, and cardiac workup was completed and was negative except for elevated inflammatory markers with a C-reactive protein (CRP) level of 261 mg/L, erythrocyte sedimentation rate (ESR) of 53 mm/hr, and a D-dimer level of 0.93 mcg/ml. Chest X-ray showed diffuse multifocal infiltrates. The pat

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11

Trail, PA-C, Allison, Jane Rogers, PharmD, BCOP, and Jaffer Ajani, MD. "Can You Establish the Cause of This Patient’s Shortness of Breath?" Journal of the Advanced Practitioner in Oncology 14, no. 5 (2023): 440–43. http://dx.doi.org/10.6004/jadpro.2023.14.5.8.

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Mr. B is a 56-year-old man diagnosed with metastatic HER2-positive gastroesophageal adenocarcinoma. He received front-line leucovorin, 5-fluorouracil, and oxaliplatin (FOLFOX) and trastuzumab for 10 months before restaging imaging revealed progressive disease. He then received second-line trastuzumab deruxtecan. His treatment was complicated by several admissions felt to be unrelated to his cancer therapy. He was discharged after an episode of pneumonia on a steroid taper with prophylactic trimethoprim/sulfamethoxazole. Once he recovered, he was given a fourth dose of chemotherapy. About a wee

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Belaidi, Elise, Charles Khouri, Olfa Harki, et al. "Cardiac consequences of intermittent hypoxia: a matter of dose? A systematic review and meta-analysis in rodents." European Respiratory Review 31, no. 164 (2022): 210269. http://dx.doi.org/10.1183/16000617.0269-2021.

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AimIntermittent hypoxia (IH) is considered to be a major contributor to obstructive sleep apnoea-related cardiovascular consequences. The present meta-analysis aimed to assess the effects of IH on cardiac remodelling, function and infarct size after myocardial ischaemia across different rodent species and IH severities.Methods and resultsRelevant articles from PubMed, Embase and Web of Science were screened. We performed a random effect meta-analysis to assess the effect of IH on myocardium in rodents by using standardised mean difference (SMD). Studies using rodents exposed to IH and outcomes

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13

Taweesedt, Pahnwat Tonya, Charles W. Nordstrom, Jessica Stoeckel, and Igor Dumic. "Pulmonary Manifestations of Drug Reaction with Eosinophilia and Systemic Symptoms (DRESS) Syndrome: A Systematic Review." BioMed Research International 2019 (September 24, 2019): 1–10. http://dx.doi.org/10.1155/2019/7863815.

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Background. The syndrome of drug reaction with eosinophilia and systemic symptoms (DRESS) is a rare, yet potentially fatal hypersensitivity reaction, most commonly associated with anticonvulsants, sulfonamides, and allopurinol. The reaction commonly manifests as a febrile skin eruption with lymphadenopathy and malaise between two and eight weeks following drug exposure. Internal organ involvement occurs in close to 90 percent of patients, and multiple organs may be involved in approximately half of those affected (most commonly the liver, kidney, and lung). Its long latency period and its vari

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14

Lazar, Mihai, Ecaterina Constanta Barbu, Cristina Emilia Chitu, et al. "Mortality Predictors in Severe SARS-CoV-2 Infection." Medicina 58, no. 7 (2022): 945. http://dx.doi.org/10.3390/medicina58070945.

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Background and Objectives: The severe forms of SARS-CoV-2 pneumonia are associated with acute hypoxic respiratory failure and high mortality rates, raising significant challenges for the medical community. The objective of this paper is to present the importance of early quantitative evaluation of radiological changes in SARS-CoV-2 pneumonia, including an alternative way to evaluate lung involvement using normal density clusters. Based on these elements we have developed a more accurate new predictive score which includes quantitative radiological parameters. The current evolution models used

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15

Srinivas, G., E. Muralinath, Digamber D. Vijay, et al. "Etiology, Epidemiology, Patho physiology and Histo pathology of Idiopathic Pulmonary Fibrosis." Journal of Research and Applications in Pharmacy Practices 1, no. 1 (2025): 35–44. https://doi.org/10.5281/zenodo.15486838.

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<em>The progressive lung condition known as idiopathic pulmonary fibrosis (IPF) is characterised by lung scarring, especially from an unidentified source. The long-term prognosis for the disorder is dismal. IPF's classic symptoms include increasing dyspnoea, a dry, ineffective cough, and a slow development of shortness of breath. Restricted lung function impairment, decreased functional vital capacity, and decreased carbon monoxide diffusing capacity are typically linked to pulmonary function testing. Due to the disease's propensity to develop quickly in its later stages, early identification

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16

Aqeel, Masooma, Bjorn Batdorf, Horatiu Olteanu, and Jayshil J. Patel. "An Unusual Suspect Causing Hypoxemic Respiratory Failure." Journal of Investigative Medicine High Impact Case Reports 5, no. 1 (2017): 232470961668758. http://dx.doi.org/10.1177/2324709616687587.

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Introduction: Antisynthetase syndrome (ASS) is characterized by the presence of anti-Jo-1 antibodies in conjunction with clinical findings of fever, polymyositis-dermatomyositis, and interstitial lung disease (ILD). Inflammatory myopathies carry a high risk of malignancy, but this association is less well outlined in ASS. We present the case of a patient with ASS who developed non-Hodgkin’s lymphoma with acute hypoxemic respiratory failure. Case Presentation: A 44-year-old female with ASS presented with acute hypoxemic respiratory failure. She was empirically treated with broad-spectrum antibi

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17

Wiens, Evan J., and Alexey Mylnikov. "Hypoxic Respiratory Failure Complicating Esophagogastroduodenoscopy." American Journal of Gastroenterology 114, no. 5 (2019): 706. http://dx.doi.org/10.14309/ajg.0000000000000123.

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18

Bosteels, C., M. Truyens, Y. Vande Weygaerde, et al. "Respiratory failure in a tofacitinib treated patient with ulcerative colitis." Acta Gastro Enterologica Belgica 87, no. 2 (2024): 336–39. http://dx.doi.org/10.51821/87.2.11812.

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Tofacitinib is an oral Janus Kinase (JAK) inhibitor recently approved for the management of moderate to severe ulcerative colitis (UC). Safety data on tofacitinib has suggested a moderately increased risk for venous thromboembolism (VTE) as well as non-opportunistic infections and non-infectious drug-related lung disease. We faced a diagnostic dilemma between an infectious and non-infectious cause of acute respiratory insufficiency in a patient on recent tofacitinib therapy. A lung CT scan showed bilateral interstitial pneumonic infiltrates and a PCR on bronchoalveolar lavage fluid was positiv

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Dr SHRINATH V, Dr SIDDHARTHA CHAKRABORTY, Dr RAHUL TYAGI, and Dr ASHOK NARAYANAN. "An Interesting Case of Pulmonary Tuberculosis with Hypoxemic Respiratory Failure Mimicking Interstitial Lung Disease." Indian Journal Of Clinical Practice 34, no. 6 (2023): 26–28. http://dx.doi.org/10.59793/ijcp.v34i6.715.

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Hypoxemic respiratory failure is a rare presentation of active pulmonary tuberculosis (TB). Though tuberculous acuterespiratory distress syndrome (ARDS) is well documented in medical literature, non-ARDS respiratory failure is seldomreported in active pulmonary TB. We herein report a case, which posed a diagnostic dilemma as it clinically mimickedinterstitial lung disease. Imaging showed unilateral localized smooth interlobular septal thickening along with consolidationwithout significant mediastinal lymphadenopathy. To the best of our knowledge, this rare radiological finding has not beenrepo

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Dr SHRINATH V, Dr SIDDHARTHA CHAKRABORTY, Dr RAHUL TYAGI, and Dr ASHOK NARAYANAN. "An Interesting Case of Pulmonary Tuberculosis with Hypoxemic Respiratory Failure Mimicking Interstitial Lung Disease." Indian Journal Of Clinical Practice 34, no. 12 (2024): 33–35. http://dx.doi.org/10.59793/ijcp.v34i12.1007.

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Hypoxemic respiratory failure is a rare presentation of active pulmonary tuberculosis (TB). Though tuberculous acuterespiratory distress syndrome (ARDS) is well documented in medical literature, non-ARDS respiratory failure is seldomreported in active pulmonary TB. We herein report a case, which posed a diagnostic dilemma as it clinically mimickedinterstitial lung disease. Imaging showed unilateral localized smooth interlobular septal thickening along with consolidationwithout significant mediastinal lymphadenopathy. To the best of our knowledge, this rare radiological finding has not beenrepo

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21

Geltser, B. I., I. G. Kurpatov, A. A. Dej, and A. G. Kozhanov. "Respiratory muscles dysfunction and respiratory diseases." Terapevticheskii arkhiv 91, no. 3 (2019): 93–100. http://dx.doi.org/10.26442/00403660.2019.03.000108.

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This review presents an analysis of the literature on the topic of respiratory muscle (RM) dysfunction in various forms of respiratory pathology: chronic obstructive pulmonary disease (COPD), asthma, community-acquired pneumonia, idiopathic pulmonary fibrosis (IPF), sarcoidosis and interstitial lung diseases (ILD), associated with systemic connective tissue diseases (polymyositis, dermatomyositis and systemic lupus erythematosus - SLE). Various clinical and pathophysiological aspects of RM dysfunction and general patterns of its pathogenesis were examined. It was proved that the role of RM in

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22

Jum’ah, H., M. Chitsaz, and S. Kundrapu. "Leukostasis aggravated by red blood cell transfusion in a chronic lymphocytic leukemia patient." American Journal of Clinical Pathology 156, Supplement_1 (2021): S162—S163. http://dx.doi.org/10.1093/ajcp/aqab191.347.

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Abstract Introduction/Objective Leukostasis/symptomatic hyperleukocytosis is commonly seen in acute leukemias and is characterized by high blast counts and symptoms of decreased tissue perfusion with a one-week mortality of 20-40%, if left untreated. It is a rare complication in chronic lymphocytic leukemia (CLL) and is seen in CLL patients with white blood cell (WBC) counts &gt; 500x10^9/L. Studies have shown that transfusion of blood products prior to decreasing the WBC count may lead to increased blood viscosity and worsen leukostasis. We report a CLL patient with a lower WBC count pres

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23

Martinez-Romo, Miguel, Shahram Lotfipour, and C. Eric McCoy. "COVID-19 with Hypoxic Respiratory Failure." Clinical Practice and Cases in Emergency Medicine 4, no. 3 (2020): 458–60. http://dx.doi.org/10.5811/cpcem.2020.7.48793.

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Case Presentation: We describe an elderly male presenting to the emergency department with shortness of breath that progressed to hypoxic respiratory failure. Radiography and computed tomography findings were suggestive of coronavirus disease 2019 (COVID-19). Discussion: We review the clinical presentation of COVID-19 and its complications. We also describe the characteristic presentation of COVID-19 on imaging. Our case illustrates the hallmark findings of bilateral and peripheral ground-glass opacities of COVID-19.

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24

Hornik, Christoph, and Jon Meliones. "Pulmonary Edema and Hypoxic Respiratory Failure." Pediatric Critical Care Medicine 17 (August 2016): S178—S181. http://dx.doi.org/10.1097/pcc.0000000000000823.

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25

Ferrer, M. "NIV in acute hypoxic respiratory failure." Breathe 6, no. 1 (2009): 51–56. http://dx.doi.org/10.1183/18106838.0601.051.

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26

&NA;. "Inhaled nitric oxide in hypoxic respiratory failure ???" Inpharma Weekly &NA;, no. 1077 (1997): 12. http://dx.doi.org/10.2165/00128413-199710770-00025.

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27

Prescott, Hallie C., Michael W. Sjoding, Kenneth M. Langa, Theodore J. Iwashyna, and Daniel F. McAuley. "Late mortality after acute hypoxic respiratory failure." Thorax 73, no. 7 (2017): 618–25. http://dx.doi.org/10.1136/thoraxjnl-2017-210109.

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BackgroundAcute hypoxic respiratory failure (AHRF) is associated with significant acute mortality. It is unclear whether later mortality is predominantly driven by pre-existing comorbid disease, the acute inciting event or is the result of AHRF itself.MethodsObservational cohort study of elderly US Health and Retirement Study (HRS) participants in fee-for-service Medicare (1998–2012). Patients hospitalised with AHRF were matched 1:1 to otherwise similar adults who were not currently hospitalised and separately to patients hospitalised with acute inciting events (pneumonia, non-pulmonary infect

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28

Lutz, Tracey, Andrew Berry, Angela McGillivray, and Kathryn Browning-Carmo. "Neonatal Hypoxic Respiratory Failure: Referral for ECMO." Emergency Care and Medicine 1, no. 3 (2024): 304–11. http://dx.doi.org/10.3390/ecm1030031.

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Background: Neonatal hypoxic respiratory failure (HRF) secondary to PPHN (persistent pulmonary hypertension of the newborn) is an uncommon but life-threatening complication. Despite advances in therapeutic interventions, there are neonates who may require ECMO (extracorporeal membrane oxygenation), which improves survival. In establishing the capability of ECMO in transport in New South Wales, significant variation in referral thresholds and management of PPHN in referring hospitals has been noted. Aim: To review cases referred to the Newborn and paediatric Emergency Transport Service (NETS) f

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29

Neubert, Z., P. Hoffmann, and D. Owshalimpur. "Acute kidney injury with hypoxic respiratory failure." Case Reports 2014, sep22 1 (2014): bcr2014206582. http://dx.doi.org/10.1136/bcr-2014-206582.

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30

Elsasser, Serge, Hartmut Sch??chinger, and Werner Strobel. "Adjunctive Drug Treatment in Severe Hypoxic Respiratory Failure." Drugs 58, no. 3 (1999): 429–46. http://dx.doi.org/10.2165/00003495-199958030-00004.

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31

Young, J. D. "Non-ventilatory treatment of acute hypoxic respiratory failure." British Medical Bulletin 55, no. 1 (1999): 165–80. http://dx.doi.org/10.1258/0007142991902196.

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32

Bartlett, Robert H., Alan H. Morris, H. Barry Fairley, Robert Hirsch, Nicholas O'Connor, and Henning Pontoppidan. "A Prospective Study of Acute Hypoxic Respiratory Failure." Chest 89, no. 5 (1986): 684–89. http://dx.doi.org/10.1378/chest.89.5.684.

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33

Barnes, Meredith, and Thura Abd. "Hypoxic Respiratory Failure Secondary to Acute Lupus Pneumonitis." Chest 142, no. 4 (2012): 468A. http://dx.doi.org/10.1378/chest.1382069.

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34

Buell, Kevin G., and Bhakti K. Patel. "Helmet noninvasive ventilation in acute hypoxic respiratory failure." Current Opinion in Critical Care 29, no. 1 (2022): 8–13. http://dx.doi.org/10.1097/mcc.0000000000001008.

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Dudell, Golde G., and Lucky Jain. "Hypoxic Respiratory Failure in the Late Preterm Infant." Clinics in Perinatology 33, no. 4 (2006): 803–30. http://dx.doi.org/10.1016/j.clp.2006.09.006.

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36

OLAYODE, ADEWALE. "CASE OF HYPOXIC/HYPERCARBIC RESPIRATORY FAILURE FROM CAPNOPERITONEUM." CHEST 164, no. 4 (2023): A2122—A2123. http://dx.doi.org/10.1016/j.chest.2023.07.1444.

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37

Burton-Papp, Helmi C., Alexander I. R. Jackson, Ryan Beecham, et al. "Conscious prone positioning during non-invasive ventilation in COVID-19 patients: experience from a single centre." F1000Research 9 (July 31, 2020): 859. http://dx.doi.org/10.12688/f1000research.25384.1.

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Critically ill patients admitted to hospital following SARS-CoV-2 infection often experience hypoxic respiratory failure and a proportion require invasive mechanical ventilation to maintain adequate oxygenation. The combination of prone positioning and non-invasive ventilation in conscious patients may have a role in improving oxygenation. The purpose of this study was to assess the effect of prone positioning in spontaneously ventilating patients receiving non-invasive ventilation admitted to the intensive care. Clinical data of 81 patients admitted with COVID 19 pneumonia and acute hypoxic r

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Vita, Skoryk, Korsunov Volodymyr, Bocharova Tetiana, Nartov Pavel, and Valentina Maslova. "Clinical and morphological comparison of respiratory failure in patients with COVID-19 who died under different options of respiratory support." ScienceRise: Medical Science, no. 4(43) (July 30, 2021): 4–12. https://doi.org/10.15587/2519-4798.2021.238093.

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<strong>The aim.</strong> Based on the study of the effect of invasive mechanical ventilation and NIV in the CPAP mode on the pathomorphosis of lung damage in patients with HRF caused by SARS-nCoV-2 and deaths in intensive care unit (ICU), determine the safest method of respiratory support. <strong>Materials and methods</strong>. The study included morphological material from 20 patients with HRF caused by SARS-nCoV-2 (COVID-19) who died in ICU. Group 1 included patients who received non-invasive lung ventilation in CPAP mode through a face mask (n=10), group 2 - patients who underwent in

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Harsha, PV, Durga Balagopalan, MM Aneesh, Ponneduthamkuzhy Thomas James, Ajit Bhaskar, and S. KrishnavenI. "Profile of COVID-19 Patients with Persistent Respiratory Symptoms." Indian Journal of Respiratory Care 03, no. 01 (2022): 55–58. http://dx.doi.org/10.5005/jp-journals-11010-1010.

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Background and objective: COVID pandemic struck the entire world causing high mortality and morbidity. After-effects of the infection were manifested in various clinical forms for the varying duration. As the pandemic waxes and wanes, COVID has become one of the differential diagnoses for various clinical and radiological pictures and triggers for respiratory diseases. While COVID sequelae are still much to explore, this study aimed to assess the clinicoradiological profile of COVID-19 patients who have persistent symptoms pertaining to the respiratory system. Materials and methods: This prosp

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Sundh, Josefin, and Magnus Ekström. "Risk factors for developing hypoxic respiratory failure in COPD." International Journal of Chronic Obstructive Pulmonary Disease Volume 12 (July 2017): 2095–100. http://dx.doi.org/10.2147/copd.s140299.

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&NA;. "Is iNO worth the effort in hypoxic respiratory failure?" Inpharma Weekly &NA;, no. 1489 (2005): 14. http://dx.doi.org/10.2165/00128413-200514890-00037.

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Rawat, Munmun, Praveen K. Chandrasekharan, Ashley Williams, et al. "Oxygen Saturation Index and Severity of Hypoxic Respiratory Failure." Neonatology 107, no. 3 (2015): 161–66. http://dx.doi.org/10.1159/000369774.

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43

Polos, Peter G. "Hypoxic Respiratory Failure in a 30-Year-Old Spelunker." Chest 113, no. 4 (1998): 1125–26. http://dx.doi.org/10.1378/chest.113.4.1125.

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Gupta, Shikha, Aliya Noor, and Homer Twigg. "Hypoxic Respiratory Failure After Transarterial Embolization of Hepatocellular Carcinoma." Chest 142, no. 4 (2012): 1031A. http://dx.doi.org/10.1378/chest.1370704.

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Baronner, Ashley. "PULMONARY LYMPHOMATOID GRANULOMATOSIS PRESENTING AS SEVERE HYPOXIC RESPIRATORY FAILURE." Chest 156, no. 4 (2019): A1974. http://dx.doi.org/10.1016/j.chest.2019.08.1956.

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Mina, Bushra, Alexander Newton, and Vijay Hadda. "Noninvasive Ventilation in Treatment of Respiratory Failure-Related COVID-19 Infection: Review of the Literature." Canadian Respiratory Journal 2022 (August 31, 2022): 1–8. http://dx.doi.org/10.1155/2022/9914081.

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The recently diagnosed coronavirus disease 2019 (COVID-19), caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), in December 2019 commonly affects the respiratory system. The incidence of acute hypoxic respiratory failure varied among epidemiological studies with high percentage of patients requiring mechanical ventilation with a high mortality. Noninvasive ventilation is an alternative tool for ventilatory support instead of invasive mechanical ventilation, especially with scarce resources and intensive care beds. Initially, there were concerns by the national societies reg

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Gebara, Bassam M., and Michelle Kroupa-Kulik. "Surfactant Administration in Severe Hypoxic Respiratory Failure Due to Respiratory Syncytial Virus." Clinical Pediatrics 38, no. 4 (1999): 253–54. http://dx.doi.org/10.1177/000992289903800417.

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Valentini, Ricardo, Ricardo Delorme, Mariano Mazzei, and Norberto Frega. "NON INVASIVE POSITIVE-PRESSURE VENTILATION (NIPPV) IN HYPOXIC ACUTE RESPIRATORY FAILURE WITHOUT PRIOR CHRONIC RESPIRATORY FAILURE." Critical Care Medicine 27, Supplement (1999): 136A. http://dx.doi.org/10.1097/00003246-199901001-00382.

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Shah, Neeraj M., and Georgios Kaltsakas. "Respiratory complications of obesity: from early changes to respiratory failure." Breathe 19, no. 1 (2023): 220263. http://dx.doi.org/10.1183/20734735.0263-2022.

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Obesity is a significant and increasingly common cause of respiratory compromise. It causes a decrease in static and dynamic pulmonary volumes. The expiratory reserve volume is one of the first to be affected. Obesity is associated with reduced airflow, increased airway hyperresponsiveness, and an increased risk of developing pulmonary hypertension, pulmonary embolism, respiratory tract infections, obstructive sleep apnoea and obesity hypoventilation syndrome. The physiological changes caused by obesity will eventually lead to hypoxic or hypercapnic respiratory failure. The pathophysiology of

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Andreev, V. M. "All-Union Scientific and Practical Conference "Topical Questions of Respiratory Pathology"." Kazan medical journal 69, no. 5 (1988): 396–97. http://dx.doi.org/10.17816/kazmj99585.

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Professors V. S. Shchelkunov, O. V. Alexandrov, V. M. Kassil, H. R. Paleev, M. M. Seredenko, V. N. Abrosimov, A. G. Marachev made reports on diagnosis, treatment of patients with acute and chronic respiratory failure, on aspects of pulmonary hypertension, treatment of hypoxic conditions, morphological basis of respiratory failure.

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