Academic literature on the topic 'Intestinal mucosal immune system'

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Intestinal mucosal immune system.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Intestinal mucosal immune system"

1

Igietseme, Joseph U., John L. Portis, and Linda L. Perry. "Inflammation and Clearance of Chlamydia trachomatis in Enteric and Nonenteric Mucosae." Infection and Immunity 69, no. 3 (2001): 1832–40. http://dx.doi.org/10.1128/iai.69.3.1832-1840.2001.

Full text
Abstract:
ABSTRACT Immunization(s) fostering the induction of genital mucosa-targeted immune effectors is the goal of vaccines against sexually transmitted diseases. However, it is uncertain whether vaccine administration should be based on the current assumptions about the common mucosal immune system. We investigated the relationship between mucosal sites of infection, infection-induced inflammation, and immune-mediated bacterial clearance in mice using the epitheliotropic pathogenChlamydia trachomatis. Chlamydial infection of the conjunctival, pulmonary, or genital mucosae stimulated significant chan
APA, Harvard, Vancouver, ISO, and other styles
2

Wang, Li, Limeng Zhu, and Song Qin. "Gut Microbiota Modulation on Intestinal Mucosal Adaptive Immunity." Journal of Immunology Research 2019 (October 3, 2019): 1–10. http://dx.doi.org/10.1155/2019/4735040.

Full text
Abstract:
The mammalian intestine harbors a remarkable number of microbes and their components and metabolites, which are fundamental for the instigation and development of the host immune system. The intestinal innate and adaptive immunity coordinate and interact with the symbionts contributing to the intestinal homeostasis through establishment of a mutually beneficial relationship by tolerating to symbiotic microbiota and retaining the ability to exert proinflammatory response towards invasive pathogens. Imbalance between the intestinal immune system and commensal organisms disrupts the intestinal mi
APA, Harvard, Vancouver, ISO, and other styles
3

Li, Tianming, Mei Liu, Siyu Sun, Xuying Liu, and Dongyan Liu. "Epithelial Cells Orchestrate the Functions of Dendritic Cells in Intestinal Homeostasis." Journal of Biomedical Research & Environmental Sciences 1, no. 7 (2020): 343–52. http://dx.doi.org/10.37871/jbres1165.

Full text
Abstract:
The gastrointestinal tract represents the largest mucosal membrane surface and is the one of the most complex human organs. The intestinal barrier dysfunction contributes to systemic immune activation. The mucosal immune system has extremely arduous tasks to resist invaders and promote tolerance of food antigens and the microbiota. The intestinal mucosal immune system fulfills these tasks through complex interactions between immune cells and the local microenvironment in intestine. Intestinal Epithelial Cells (IECs) play important roles in these complex interactions. IECs not only constitute t
APA, Harvard, Vancouver, ISO, and other styles
4

Schuppler, Markus, and Martin J. Loessner. "The Opportunistic PathogenListeria monocytogenes: Pathogenicity and Interaction with the Mucosal Immune System." International Journal of Inflammation 2010 (2010): 1–12. http://dx.doi.org/10.4061/2010/704321.

Full text
Abstract:
Listeria monocytogenesis an opportunistic foodborne pathogen causing listeriosis, an often fatal infection leading to meningitis, sepsis, or infection of the fetus and abortion in susceptible individuals. It was recently found that the bacterium can also cause acute, self-limiting febrile gastroenteritis in healthy individuals. In the intestinal tract,L. monocytogenespenetrates the mucosa directly via enterocytes, or indirectly via invasion of Peyer’s patches. Animal models forL. monocytogenesinfection have provided many insights into the mechanisms of pathogenesis, and the development of new
APA, Harvard, Vancouver, ISO, and other styles
5

Astafieva, N. G., I. V. Gamova, E. N. Udovitchenko, I. A. Perfilova, D. Y. Kobzev, and І. Ae Michailova. "Mucosal immune system: the regulatory action of probiotics." Russian Journal of Allergy 12, no. 5 (2015): 17–30. http://dx.doi.org/10.36691/rja423.

Full text
Abstract:
The evidence of the beneficial effects of dairy products on the intestinal microflora was given for the first time in 1908 by I.I. Mechnikov in the famous article «A few words about the sour milk». Since that time probiotics - the living microorganisms for regulation of intestinal microbiota are the case of interest. Interactions between the probiotics and macroorganism are very complex and include a network of genes receptors, signaling molecules and a variety of other factors that determine the natural course of the disease.
APA, Harvard, Vancouver, ISO, and other styles
6

Yoo, Ji, Maureen Groer, Samia Dutra, Anujit Sarkar, and Daniel McSkimming. "Gut Microbiota and Immune System Interactions." Microorganisms 8, no. 10 (2020): 1587. http://dx.doi.org/10.3390/microorganisms8101587.

Full text
Abstract:
Dynamic interactions between gut microbiota and a host’s innate and adaptive immune systems are essential in maintaining intestinal homeostasis and inhibiting inflammation. Gut microbiota metabolizes proteins and complex carbohydrates, synthesizes vitamins, and produces an enormous number of metabolic products that can mediate cross-talk between gut epithelium and immune cells. As a defense mechanism, gut epithelial cells produce a mucosal barrier to segregate microbiota from host immune cells and reduce intestinal permeability. An impaired interaction between gut bacteria and the mucosal immu
APA, Harvard, Vancouver, ISO, and other styles
7

Fiocchi, Claudio. "Intestinal inflammation: a complex interplay of immune and nonimmune cell interactions." American Journal of Physiology-Gastrointestinal and Liver Physiology 273, no. 4 (1997): G769—G775. http://dx.doi.org/10.1152/ajpgi.1997.273.4.g769.

Full text
Abstract:
Intestinal inflammation has traditionally been viewed as a process in which effector immune cells cause the destruction of other mucosal cells that behave as passive bystander targets. Progress in understanding the process of intestinal inflammation has led to a much broader and more integrated picture of the various mucosal components, a picture in which cytokines, growth factors, adhesion molecules, and the process of apoptosis act as functional mediators. Essentially all cellular and acellular components can exert immunelike activities, modifying the classical concept of selected immune cel
APA, Harvard, Vancouver, ISO, and other styles
8

Sun, Ruicong, Chunjin Xu, Baisui Feng, Xiang Gao, and Zhanju Liu. "Critical roles of bile acids in regulating intestinal mucosal immune responses." Therapeutic Advances in Gastroenterology 14 (January 2021): 175628482110180. http://dx.doi.org/10.1177/17562848211018098.

Full text
Abstract:
Bile acids are a class of cholesterol derivatives that have been known for a long time for their critical roles in facilitating the digestion and absorption of lipid from the daily diet. The transformation of primary bile acids produced by the liver to secondary bile acids appears under the action of microbiota in the intestine, greatly expanding the molecular diversity of the intestinal environment. With the discovery of several new receptors of bile acids and signaling pathways, bile acids are considered as a family of important metabolites that play pleiotropic roles in regulating many aspe
APA, Harvard, Vancouver, ISO, and other styles
9

Heyman, M. "How dietary antigens access the mucosal immune system." Proceedings of the Nutrition Society 60, no. 4 (2001): 417–26. http://dx.doi.org/10.1079/pns2001117.

Full text
Abstract:
The intestinal epithelium is a selective barrier where incompletely-digested food antigens are transmitted to the immune system. Food antigens are often the starting point of intestinal diseases such as food allergy or coeliac disease. The intestinal epithelial cells (IEC) take up and process food antigens mainly by fluid-phase transcytosis involving two functional pathways, one minor direct pathway without degradation and another major lysosomal degradative pathway. Among the peptidic metabolites generated during transepithelial transport of luminal antigens, some have a molecular mass compat
APA, Harvard, Vancouver, ISO, and other styles
10

Yue, Bei, Xiaoping Luo, Zhilun Yu, Sridhar Mani, Zhengtao Wang, and Wei Dou. "Inflammatory Bowel Disease: A Potential Result from the Collusion between Gut Microbiota and Mucosal Immune System." Microorganisms 7, no. 10 (2019): 440. http://dx.doi.org/10.3390/microorganisms7100440.

Full text
Abstract:
Host health depends on the intestinal homeostasis between the innate/adaptive immune system and the microbiome. Numerous studies suggest that gut microbiota are constantly monitored by the host mucosal immune system, and any slight disturbance in the microbial communities may contribute to intestinal immune disruption and increased susceptibility to inflammatory bowel disease (IBD), a chronic relapsing inflammatory condition of the gastrointestinal tract. Therefore, maintaining intestinal immune homeostasis between microbiota composition and the mucosal immune system is an effective approach t
APA, Harvard, Vancouver, ISO, and other styles
More sources

Dissertations / Theses on the topic "Intestinal mucosal immune system"

1

Thompson, Fiona Marie. "Activation of the mucosal immune system and growth of the small intestine at weaning /." Title page, abstract and contents only, 1994. http://web4.library.adelaide.edu.au/theses/09PH/09pht4677.pdf.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Stange, Jörg. "Studies on host-pathogen interactions at mucosal barrier surfaces using the murine intestinal parasite Eimeria falciformis." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2013. http://dx.doi.org/10.18452/16716.

Full text
Abstract:
Wir nutzten in dieser Studie den apikomplexen Parasiten Eimeria falciformis als Modell. Unsere Ergebnisse zeigen, dass das in infizierten Wildtypmäusen dominierende Zytokin IFN-γ für Immunschutz und für die Entwicklung der Darmpathologie entbehrlich war. E. falciformis-infizierte IFN-γR-/- and IFN-γ-/- Mäuse zeigten extremen Körpergewichtsverlust und starke Pathologie im Darm. Die Entwicklung des Parasiten in diesen Mäusen war überraschenderweise reduziert. Diese Beobachtungen gingen mit einer drastisch erhöhten Produktion von parasiten-spezifischem IL-17A und IL-22 durch CD4+ T Zellen einher.
APA, Harvard, Vancouver, ISO, and other styles
3

Tyrer, Peter Charles, and n/a. "Targeting M-cells for oral vaccine delivery." University of Canberra. Health Sciences, 2004. http://erl.canberra.edu.au./public/adt-AUC20060427.122012.

Full text
Abstract:
An in vitro model of the follicle-associated epithelia that overlie the Peyer's patches of the small intestine was developed and validated to examine the mechanisms of mucosal antigen sampling. This model displays many phenotypic and physiological characteristics of M cells including apical expression of [alpha]5[beta]l integrin and enhanced energy dependent participate transport. CD4+ T-cells were shown to be an important influence on the development of Mlike cells. The model was used to examine the M cell mediated uptake of several putative whole-cell killed bacterial vaccines. Greater numbe
APA, Harvard, Vancouver, ISO, and other styles
4

Munro, Grant Hamilton. "Murine giardiasis : intestinal mucosal immune responses." Thesis, University of Edinburgh, 1985. http://hdl.handle.net/1842/20043.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Hu, Hong-Zhen. "Purinergic neurogenic intestinal mucosal secretion." Connect to this title online, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1100028634.

Full text
Abstract:
Thesis (Ph. D.)--Ohio State University, 2004.<br>Document formatted into pages; contains 171 p. Includes bibliographical references. Abstract available online via OhioLINK's ETD Center; full text release delayed at author's request until 2005 Nov. 10.
APA, Harvard, Vancouver, ISO, and other styles
6

Santos, Liliane Martins dos. "Breakdown of intestinal homeostasis by mucosal infections triggers adaptive immune responses against antigens from commensal bacteria." Universidade Federal de Minas Gerais, 2011. http://hdl.handle.net/1843/BUOS-8XUR5V.

Full text
Abstract:
The gastrointestinal tract of mammals is inhabited by thousands of distinct species of commensal microorganisms that exist in a mutualistic relationship with the host. It has previously been shown that these gut microbes play an important role in modulating host immune responses. On the other hand, commensals can also contribute to pathology in the context of acute infection. For instance, oral infections with Toxoplasma gondii in certain inbred strains of mice lead to an exacerbated intestinal inflammation that is accompanied by a loss of diversity within the gut flora. Furthermore, the micro
APA, Harvard, Vancouver, ISO, and other styles
7

Morris, Bruce C. "Intestinal Mucosal Mast Cell Immune Response and Pathogenesis of Two Eimeria Acervulina Isolates in Broiler Chickens." Thesis, Virginia Tech, 2002. http://hdl.handle.net/10919/36228.

Full text
Abstract:
Five experiments were conducted comparing differential intestinal immune responses to two isolates of Eimeria acervulina (EA), EA1 and EA2. In three experiments, broiler chicks were divided into control (non-challenged), EA1, or EA2 challenged (14 days of age) groups. On day 6 post-challenge (PC), changes in body weight were determined, intestinal lesions were scored, and duodenal tissue was evaluated for morphometric alterations and mucosal mast cell responses. EA1 produced duodenal lesions and reduced villus height to crypt depth ratios when compared to controls; however, no differences we
APA, Harvard, Vancouver, ISO, and other styles
8

Etling, Michele R. "THE AGING MUCOSAL IMMUNE SYSTEM IN THE INTERLEUKIN-10-DEFICIENT MOUSE." Case Western Reserve University School of Graduate Studies / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=case1184295867.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Blazek, Alisa D. "A Simulated Altitude Device can Improve Endurance Performance without Mucosal Immune System Compromise." The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1267567607.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Pardo, Camacho Cristina. "Intestinal mucosal humoral response and neuro-immune interaction as contributors to the pathophysiology of diarrhea-predominant Irritable Bowel Syndrome." Doctoral thesis, Universitat Autònoma de Barcelona, 2020. http://hdl.handle.net/10803/670719.

Full text
Abstract:
La Síndrome de l’Intestí Irritable (SII) és un trastorn gastrointestinal crònic i prevalent que cursa amb alteracions a la motilitat intestinal i amb dolor abdominal. La SII constitueix un trastorn recurrent i potencialment incapacitant sense un marcador de diagnòstic específic i únicament hi ha disponibles tractaments pal·liatius. L’absència d’una patofisiologia ben establerta destaca la necessitat d’identificar les causes orgàniques subjacents a les alteracions intestinal i la generació de símptomes. A la mucosa intestinals d’aquests pacients s’ha identificat un cert grau d’inflamació així
APA, Harvard, Vancouver, ISO, and other styles
More sources

Books on the topic "Intestinal mucosal immune system"

1

Å, Hanson Lars, and Svanborg-Edén Catharina, eds. Mucosal immunobiology: Cellular-molecular interactions in the mucosal immune system. Karger, 1988.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

Whitehead, Richard. Mucosal biopsy of the gastrointestinal tract. 3rd ed. Saunders, 1985.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

Jihad, Hayek, and Federman Micheline, eds. Gastrointestinal mucosal biopsy. Churchill Livingstone, 1996.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

Mucosal biopsy of the gastrointestinal tract. 3rd ed. Saunders, 1985.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

A, Livolsi Virginia, ed. Mucosal biopsy of the gastrointestinal tract. 4th ed. Saunders, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
6

Mucosal biopsy of the gastrointestinal tract. 5th ed. Saunders, 1997.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

1951-, MacDonald Thomas T., ed. Ontogeny of the immune system of the gut. CRC Press, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
8

(Editor), Richard Blumberg, and Markus F. Neurath (Editor), eds. Immune Mechanisms in Inflammatory Bowel Disease (Advances in Experimental Medicine and Biology). Springer, 2006.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
9

Blaser, Annika Reintam, and Adam M. Deane. Normal physiology of the gastrointestinal system. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0172.

Full text
Abstract:
The gastrointestinal (GI) system is responsible for digestion and absorption, but also has important endocrine, immune and barrier functions. Additionally, the GI system plays a major role in fluid, electrolyte and acid-base balance. The GI system is regulated by complex myogenic, neural and humoral mechanisms, and, in health, these are affected by the presence of luminal nutrient, thereby modulating function of the GI system. Accordingly, GI function varies depending on whether a person is fasted or in the postprandial state. Adequate fasting and postprandial perfusion, motility and exocrine
APA, Harvard, Vancouver, ISO, and other styles
10

Frenkel, Joost, and Hans R. Waterham. Mevalonate Kinase Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0039.

Full text
Abstract:
Mevalonate kinase deficiency (MKD) is an autosomal recessive inborn error of isoprenoid biosynthesis, a pathway yielding sterols and nonsterol isoprenoids.In patients, the enzyme activity of mevalonate kinase is severely reduced due to mutations in the encoding gene, MVK. The substrate, mevalonate, accumulates and is elevated in blood and urine. Shortage of certain downstream products of the pathway, nonsterol isoprenoids, leads to dysregulation of the innate immune system, activation of inflammasomes, and interleukin (IL)-1 mediated inflammation.Symptoms start in early childhood with recurren
APA, Harvard, Vancouver, ISO, and other styles
More sources

Book chapters on the topic "Intestinal mucosal immune system"

1

James, S. P., W. C. Kwan, and M. C. Sneller. "T cell lymphokine mRNA expression, lymphokine utilization, and regulatory function in the intestinal mucosal immune system." In Advances in Mucosal Immunology. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_218.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Barton, J. R., S. O’Mahoney, and A. Ferguson. "Regulation of antibodies to food proteins within the common mucosal immune system: lack of correlation between antibody titres in saliva and intestinal fluid." In Advances in Mucosal Immunology. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_147.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Georgiev, Vassil St. "Mucosal Immune System." In National Institute of Allergy and Infectious Diseases, NIH. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-297-1_42.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Brandtzaeg, Per, and Finn-Eirik Johansen. "IgA and Intestinal Homeostasis." In Mucosal Immune Defense: Immunoglobulin A. Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-72232-0_10.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Agarwal, Shradha, and Lloyd Mayer. "The Mucosal Immune System." In Food Allergy. John Wiley & Sons Ltd, 2014. http://dx.doi.org/10.1002/9781118744185.ch1.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Klimov, Vladimir V. "Skin and Mucosal Immune System." In From Basic to Clinical Immunology. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-03323-1_2.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Roux, M. E., N. H. Slobodianik, P. Gauffin Cano, and G. Perdigón. "Mucosal Immune System and Malnutrition." In Gut Flora, Nutrition, Immunity and Health. Blackwell Publishing Ltd, 2008. http://dx.doi.org/10.1002/9780470774595.ch7.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Thrane, P. S., T. O. Rognum, and P. Brandtzaeg. "Ontogenesis of the human secretory immune system." In Advances in Mucosal Immunology. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_130.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Johansen, Finn-Eirik, Ranveig Braathen, Else Munthe, Hilde Schjerven, and Per Brandtzaeg. "Regulation of the Mucosal IgA System." In Mucosal Immune Defense: Immunoglobulin A. Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-72232-0_5.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Mestecky, Jiri. "Homeostasis of the Mucosal Immune System." In Advances in Experimental Medicine and Biology. Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1371-1_26.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Intestinal mucosal immune system"

1

Polosukhin, Vasiliy V., Pierre P. Massion, Jae W. Lee, Scott H. Randell, and Timothy S. Blackwell. "TOBACCO SMOKE IMPAIRS THE BRONCHIAL MUCOSAL SECRETORY IgA IMMUNE SYSTEM THROUGH ALTERATION OF BRONCHIAL EPITHELIAL CELL DIFFERENTIATION." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1424.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Orlenkovich, Lilija. "CORRELATIONS ANALYSIS OF IMMUNE SYSTEM AND GUT MICROBIOTA INDICES OF RATS IN THE CHRONIC EXPOSITION TO BIOINSECTICIDE ENTOMOPHTHORIN." In XIV International Scientific Conference "System Analysis in Medicine". Far Eastern Scientific Center of Physiology and Pathology of Respiration, 2020. http://dx.doi.org/10.12737/conferencearticle_5fd728a1ea3837.21988844.

Full text
Abstract:
The analysis of the variation in the number, intensity and direction of correlations between the immune system and the gut microbiota of rats revealed that the T-, B-system and humoral immunity changes as well as cellular and humoral factors of an organism nonspecific defense are accompanied by changes of the Intestinal microbiota of intact and experimental rats
APA, Harvard, Vancouver, ISO, and other styles
3

Slawinski, Piotr R., Weston M. Lewis, and Benjamin S. Terry. "Performance Assessment of a Noninvasive Swallowable Biosensor Deployment System in Microgravity." In ASME 2016 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2016. http://dx.doi.org/10.1115/imece2016-65039.

Full text
Abstract:
Ingestible capsule endoscope technology has been a topic of research since the middle of the 20th century and has become a prominent area of study since the commercialization of capsule endoscopy in 2000. Ingestible telemetry capsules have been investigated by NASA in the last 20 years as a means for monitoring human body temperature during periods of physical exhaustion, but are limited in sensing time due to passage through the digestive system. In this work, we present a feasibility study on a sensor that attaches to the intestinal mucosa after being delivered to the bowel via ingestible ca
APA, Harvard, Vancouver, ISO, and other styles
4

Smith, Alana, Breia Reed, Joseph F. Pierre, Beverly Lyn-Cook, and Athena Starlard-Davenport. "Abstract B064: Investigation of the breast microbiome and mucosal immune system in African American and non-Hispanic White women with and without breast cancer: A pilot study." In Abstracts: Eleventh AACR Conference on The Science of Cancer Health Disparities in Racial/Ethnic Minorities and the Medically Underserved; November 2-5, 2018; New Orleans, LA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7755.disp18-b064.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Krishnakumar, D., and K. S. Jaganathan. "Development of nasal HPV vaccine formulations." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685403.

Full text
Abstract:
Cervical cancer is the second most cancer in women worldwide with over 500000 new cases and 275000 deaths being registered every year. With nearly 73000 women dying every year, India now tops the world in cervical cancer deaths. India represents 26.4% of all women dying of cervical cancer globally. Cervical cancer estimated to be responsible for about 5% of human cancers worldwide. Currently available vaccines may not provide complete protection against all HPV types as the protection is primarily type specific. Furthermore, the available vaccines are delivered via intramuscular route and requ
APA, Harvard, Vancouver, ISO, and other styles
6

Faulkner, M. F., and J. Brandon Dixon. "Engineered Model of the Intestine Suggests Active Transport of Lipid by Lymphatics." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53903.

Full text
Abstract:
The lymphatic system has long been thought of as little more than a series of passive ducts as they serve to return fluid and proteins from interstitial spaces back to the blood, provide a route for immune cell trafficking, and transport dietary lipid from the intestine to the blood. Recent evidence has revealed that the lymphatics play an active role in lipid trafficking, and alterations in this function have been correlated with the presence of lymphatic diseases (Dixon, 2010). Here we describe the use of a two-cell, tissue engineered model to explore mechanisms of lipid transport across lym
APA, Harvard, Vancouver, ISO, and other styles
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!