Relevant bibliographies by topics / Intestinal obstruction

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Intestinal obstruction'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 July 2024

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Journal articles on the topic "Intestinal obstruction"

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Sipahi, Mesut, Kasim Caglayan, Ergin Arslan, Mustafa Fatih Erkoc, and Faruk Onder Aytekin. "Intestinal Malrotation: A Rare Cause of Small Intestinal Obstruction." Case Reports in Surgery 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/453128.

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Background. The diagnosis of intestinal malrotation is established by the age of 1 year in most cases, and the condition is seldom seen in adults. In this paper, a patient with small intestinal malrotation-type intraperitoneal hernia who underwent surgery at an older age because of intestinal obstruction is presented.Case. A 73-year-old patient who presented with acute intestinal obstruction underwent surgery as treatment. Distended jejunum and ileum loops surrounded by a peritoneal sac and located between the stomach and transverse colon were determined. The terminal ileum had entered into the transverse mesocolon from the right lower part, resulting in kinking and subsequent segmentary obstruction. The obstruction was relieved, and the small intestines were placed into their normal position in the abdominal cavity.Conclusion. Small intestinal malrotations are rare causes of intestinal obstructions in adults. The appropriate treatment in these patients is placement of the intestines in their normal positions.
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Kirchmann, Hiltrud M. A., and Steffen W. Bender. "Intestinal Obstruction in Crohn's Disease in Childhood." Journal of Pediatric Gastroenterology and Nutrition 6, no. 1 (January 1987): 79–83. http://dx.doi.org/10.1002/j.1536-4801.1987.tb09248.x.

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SummaryOut of 540 children and adolescents from the Multicenter Pediatric Crohn's Disease Study Group, 42 patients presented with decompensated intestinal obstruction. In 26 patients only one intestinal obstruction occurred, and in 16 children up to five intestinal obstructions occurred. Conservative measures were successful in 37 of 72 episodes of intestinal obstruction (51.4%), while 19 of 42 patients underwent emergency surgery (45.2%) and 16 of 42 (38.1%) were operated on following conservative treatment. From analysis of clinical, intraoperative, and histological findings in surgically treated patients, criteria for emergency surgery and elective surgery of intestinal obstruction in Crohn's disease are derived. One patient with adenocarcinoma of the large bowel, presenting with recurrent obstruction, signals caution in delaying proper diagnosis.
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Babu, Ganesh, Sudarsan ., and Vinay . "Rare Presentation of Intestinal Obstruction." New Indian Journal of Surgery 8, no. 1 (2017): 101–3. http://dx.doi.org/10.21088/nijs.0976.4747.8117.20.

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Dong, Zhen-Yu, Rui-Xian Shi, Xiao-Biao Song, Ming-Yue Du, and Ji-Jun Wang. "Postoperative abdominal herpes zoster complicated by intestinal obstruction: A case report." World Journal of Clinical Cases 12, no. 6 (February 26, 2024): 1138–43. http://dx.doi.org/10.12998/wjcc.v12.i6.1138.

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BACKGROUND Intestinal obstruction is a common occurrence in clinical practice. However, the occurrence of herpes zoster complicated by intestinal obstruction after abdominal surgery is exceedingly rare. In the diagnostic and treatment process, clinicians consider it crucial to identify the primary causes of its occurrence to ensure effective treatment and avoiding misdiagnosis. CASE SUMMARY Herein, we present the case of a 40-year-old female patient with intestinal obstruction who underwent laparoscopic appendectomy and developed herpes zoster after surgery. Combining the patient's clinical manifestations and relevant laboratory tests, it was suggested that the varicella zoster virus reactivated during the latent period after abdominal surgery, causing herpes zoster. Subsequently, the herpes virus invaded the visceral nerve fibers, causing gastrointestinal dysfunction and loss of intestinal peristalsis, which eventually led to intestinal obstruction. The patient was successfully treated through conservative treatment and antiviral therapy and subsequently discharged from the hospital. CONCLUSION Pseudo-intestinal obstruction secondary to herpes zoster infection is difficult to distinguish from mechanical intestinal obstruction owing to various causes. In cases of inexplicable intestinal obstructions, considering the possibility of a viral infection is essential to minimize misdiagnosis and missed diagnoses.
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Holder, Walter D. "Intestinal Obstruction." Gastroenterology Clinics of North America 17, no. 2 (June 1988): 317–40. http://dx.doi.org/10.1016/s0889-8553(21)00365-4.

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Hargreaves, Cathy. "Intestinal obstruction." Nursing Standard 20, no. 27 (March 15, 2006): 67–68. http://dx.doi.org/10.7748/ns.20.27.67.s59.

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MAHMOOD, KHALID, SHAHBAZ AHMED, SAJID HAMEED, and Liaquat Ali. "INTESTINAL OBSTRUCTION." Professional Medical Journal 14, no. 02 (September 6, 2007): 355–59. http://dx.doi.org/10.29309/tpmj/2007.14.02.4905.

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Objective: To identify the various causes of intestinal obstruction ininfants. Design: Retrospective study. Period: July 1995 to March 2007. Setting: Department of Paediatric Surgery,Allied Hospital, Faisalabad. Patients and Methods: The record of all infants who presented with intestinal obstructionduring the study period was reviewed. Results: Two hundred infants presented with intestinal obstruction. It was thecommonest cause for admission (57%) from the emergency department and constituted 8.2% of the infants operatedin the unit. They were predominantly males. Average duration of symptoms was 3 days. Common causes wereintussusception (46.5%), adhesions (16%), bands (13.5%) and incarcerated inguinal hernia (8%). Wound infection(12%) and wound dehiscence (6%) were the common complications. Mortality rate was 5%. Conclusion: Intestinalobstruction is seen frequently in infants and is associated with significant morbidity. Early recognition and prompttreatment are needed.
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Byrne, John J. "Intestinal obstruction." Postgraduate Medicine 87, no. 6 (May 1990): 217–20. http://dx.doi.org/10.1080/00325481.1990.11716345.

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Shelton, Brenda K. "Intestinal Obstruction." AACN Clinical Issues: Advanced Practice in Acute and Critical Care 10, no. 4 (November 1999): 478–91. http://dx.doi.org/10.1097/00044067-199911000-00007.

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Brown, Steven R., and IJ Adam. "Intestinal Obstruction." Surgery (Oxford) 20, no. 7 (July 2002): 157–64. http://dx.doi.org/10.1383/surg.20.7.157.14394.

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Dissertations / Theses on the topic "Intestinal obstruction"

1

Smith, Brian Patrick. "Surgery Improves Survival Among Patients With Intestinal Obstruction." Master's thesis, Temple University Libraries, 2010. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/84371.

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Clinical Research and Translational Medicine
M.S.
Introduction: Intestinal obstruction is a common cause of hospital admissions and carries a mortality rate around 5%. We hypothesized that surgical intervention reduces mortality among these patients. Methods: We conducted a retrospective cohort study using the 2006 Nationwide Inpatient Sample (NIS) to analyze patients with a diagnosis of intestinal obstruction without hernia. We used multiple variable logistic regression to calculate the odds ratio for surgery as a predictor of death after adjusting for illness severity. Results: Among 38,931 patients, 17,544 (45.1%) underwent operative intervention for intestinal obstructions. Surgical patients were slightly younger than non-surgical patients (65 vs. 68 years), and had more severe illness, as measured by the disease staging: mortality scale (115.45 vs. 97.95, p<0.001). After adjusting for illness severity, surgery was protective from mortality (adjusted odds ratio 0.617, 95% CI 0.535-0.710, p<0.001). This finding was validated with 2 other methods of severity adjustment. Among surgery patients, there were fewer days to surgery among survivors (1 day) than non-survivors (2 days), p<0.001. The risk of bowel necrosis increased as time from admission to surgery increased. A greater percentage of surgical patients (77.5%) were discharged home compared to non-surgical patients (76.3%), p=0.007. Conclusion: Surgery is associated with a reduced odds of in-hospital mortality among patients urgently or emergently admitted with intestinal obstruction without hernia. Delaying operative intervention is associated with an increased odds of bowel necrosis and death in these patients.
Temple University--Theses
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Vitko, Megan Sue. "Intestinal Dysfunction in Cystic Fibrosis." Case Western Reserve University School of Graduate Studies / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=case1459248266.

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Пак, Василь Якович, Василий Яковлевич Пак, and Vasyl Yakovych Pak. "Оптимізація рентгенологічної діагностики спайкової кишкової непрохідності." Thesis, Вид-во СумДУ, 2006. http://essuir.sumdu.edu.ua/handle/123456789/7711.

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Семенчук, О. В. "Рання післяопераційна спайкова кишкова непрохідність, як причина релапаротомії." Thesis, Сумський державний університет, 2017. http://essuir.sumdu.edu.ua/handle/123456789/58135.

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Crispín-Trebejo, Brenda, María Cristina Robles-Cuadros, Edwin Orendo-Velásquez, and Felipe P. Andrade. "Internal abdominal hernia: Intestinal obstruction due to trans-mesenteric hernia containing transverse colon." Elsevier B.V, 2014. http://hdl.handle.net/10757/320534.

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INTRODUCTION Internal abdominal hernias are infrequent but an increasing cause of bowel obstruction still often underdiagnosed. Among adults its usual causes are congenital anomalies of intestinal rotation, postsurgical iatrogenic, trauma or infection diseases. PRESENTATION OF CASE We report the case of a 63-year-old woman with history of chronic constipation. The patient was hospitalized for two days with acute abdominal pain, abdominal distension and inability to eliminate flatus. The X-ray and abdominal computerized tomography scan (CT scan) showed signs of intestinal obstruction. Exploratory laparotomy performed revealed a trans-mesenteric hernia containing part of the transverse colon. The intestine was viable and resection was not necessary. Only the hernia was repaired. DISCUSSION Internal trans-mesenteric hernia constitutes a rare type of internal abdominal hernia, corresponding from 0.2 to 0.9% of bowel obstructions. This type carries a high risk of strangulation and even small hernias can be fatal. This complication is specially related to trans-mesenteric hernias as it tends to volvulize. Unfortunately, the clinical diagnosis is rather difficult. CONCLUSION Trans-mesenteric internal abdominal hernia may be asymptomatic for many years because of its nonspecific symptoms. The role of imaging test is relevant but still does not avoid the necessity of exploratory surgery when clinical features are uncertain.
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Lopes, Maristela de Cassia Seudo [UNESP]. "Efeitos do tramadol no modelo de dor induzida por obstrução intestinal em eqüinos." Universidade Estadual Paulista (UNESP), 2010. http://hdl.handle.net/11449/86644.

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Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
Avaliaram-se os efeitos clínico e comportamental da injeção intravenosa do tramadol no controle da dor induzida experimentalmente, por obstrução intestinal extraluminal, com dreno de Pen Rose. Foram utilizados 24 cavalos distribuídos em quatro grupos: controle (GC, n=6); obstrução duodenal (GD, n=6); obstrução de íleo (GI, n=6) e obstrução de flexura pélvica (GFP, n=6). Após medicação pré-anestésica com a associação de acepromazina (0,025 mg.kg-1 IV), xilazina (0,5 mg.kg-1 IV) e meperidina (4 mg.kg-1 IM), o tramadol foi administrado nas doses de 1,0 mg.kg-1 e 1,5 mg.kg-1, por via intravenosa (IV), imediatamente após a obstrução intestinal, em três cavalos de cada grupo. Avaliaram-se as freqüências cardíaca (FC) e respiratória (f), temperatura retal (TºC), tempo de preenchimento capilar (TPC), motilidade intestinal, comportamento relacionado à dor (olhar para o flanco, cavar, deitar e rolar) hemograma e hemogasometria venosa, nos intervalos: M0 (basal) a cada 0,5 hora de M1 a M6 , na fase de obstrução, e até três horas após a reversão do processo obstrutivo (M7 a M12). Os resultados demonstraram que não houve diferença significativa entres as doses utilizadas dentro de cada grupo, assim como entre os grupos. Houve aumento da FC em M11 no GD e em M12 no GFP. Os sinais de dor abdominal e atonia intestinal iniciaram-se em M5 no GFP e em M6 no GI. Nos animais do GD, os sinais de desconforto não progrediram. No leucograma foi observado um quadro característico de estresse e na hemogasometria os animais do GD tendenciaram à alcalose metabólica com compensação respiratória. Clinicamente, observou-se que a dose de 1,5 mg.kg-1 de tramadol proporcionou melhor conforto para os animais, porem sem significado estatístico, quando comparado coma dose de 1,0 mg.kg-1...
The clinical and behavioral effects of the intravenous injection of tramadol were evaluated during the control of pain induced experimentally due to intestinal extraluminal obstruction using “Pen Rose” drain. A total of 24 horses were used and distributed in four groups: control (GC, n=6); duodenal obstruction (GD, n=6); ileum obstruction (GI, n=6) and pelvic flexure obstruction (GFP, n=6). After administration of pre-anesthetic medications using association of acepromazine (0.025 mg.kg-1 IV), xylazine (0.5 mg.kg-1 IV) and meperidine (4 mg.kg-1 IM), tramadol was administered at doses of 1.0 mg.kg-1 and 1.5 mg.kg-1 intravenously (IV), immediately after the intestinal obstruction in three horses of each group. Evaluations were performed, including heart rate (HR), respiratory rate (RR), rectal temperature (RT), capillary refill time (CRT), gut motility, pain-related behaviour (look for the sidewall, dig down and roll) and blood gases from venous blood at the time: M0 (baseline) and every 0.5 hours from M1 to M6, during obstruction process and also until three hours after the obstructive process be reverted (M7 to M12). The results showed no significant difference among the doses used in the same group as among groups. There was an increase in HR in the GD M11 and M12 of GFP. Signs of abdominal pain and intestinal atony began at M5 in GFP and at M6 in GI. In animals from GD, the discomfort signs did not showed progress. On the leucogram was observed a typical stress and on the blood gas analysis the animals from GD showed a tendency to metabolic alkalosis with respiratory compensation. Clinically, was observed that the dose of 1.5 mg.kg-1 of tramadol provided better comfort to the animals, but there was not statistical significance, compared with the dose 1.0 mg.kg-1... (Complete abstract click electronic access below)
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White, J. S. "Models of intestinal barrier function and their application in the study of biliary obstruction." Thesis, Queen's University Belfast, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368529.

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Rouillon, Jean-Michel. "Etude du tonus intestinal lors des réflexes viscéraux chez l'homme sain et chez trois sujets atteints de pseudo-obstruction intestinale chronique." Montpellier 1, 1989. http://www.theses.fr/1989MON11098.

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Lopes, Maristela de Cassia Seúdo. "Efeitos do tramadol no modelo de dor induzida por obstrução intestinal em eqüinos /." Jaboticabal : [s.n.], 2010. http://hdl.handle.net/11449/86644.

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Orientador: Carlos Augusto Araújo Valadão
Banca: Juan Carlos Duque Moreno
Banca: Paulo Sérgio Patto dos Santos
Resumo: Avaliaram-se os efeitos clínico e comportamental da injeção intravenosa do tramadol no controle da dor induzida experimentalmente, por obstrução intestinal extraluminal, com dreno de Pen Rose. Foram utilizados 24 cavalos distribuídos em quatro grupos: controle (GC, n=6); obstrução duodenal (GD, n=6); obstrução de íleo (GI, n=6) e obstrução de flexura pélvica (GFP, n=6). Após medicação pré-anestésica com a associação de acepromazina (0,025 mg.kg-1 IV), xilazina (0,5 mg.kg-1 IV) e meperidina (4 mg.kg-1 IM), o tramadol foi administrado nas doses de 1,0 mg.kg-1 e 1,5 mg.kg-1, por via intravenosa (IV), imediatamente após a obstrução intestinal, em três cavalos de cada grupo. Avaliaram-se as freqüências cardíaca (FC) e respiratória (f), temperatura retal (TºC), tempo de preenchimento capilar (TPC), motilidade intestinal, comportamento relacionado à dor (olhar para o flanco, cavar, deitar e rolar) hemograma e hemogasometria venosa, nos intervalos: M0 (basal) a cada 0,5 hora de M1 a M6 , na fase de obstrução, e até três horas após a reversão do processo obstrutivo (M7 a M12). Os resultados demonstraram que não houve diferença significativa entres as doses utilizadas dentro de cada grupo, assim como entre os grupos. Houve aumento da FC em M11 no GD e em M12 no GFP. Os sinais de dor abdominal e atonia intestinal iniciaram-se em M5 no GFP e em M6 no GI. Nos animais do GD, os sinais de desconforto não progrediram. No leucograma foi observado um quadro característico de estresse e na hemogasometria os animais do GD tendenciaram à alcalose metabólica com compensação respiratória. Clinicamente, observou-se que a dose de 1,5 mg.kg-1 de tramadol proporcionou melhor conforto para os animais, porem sem significado estatístico, quando comparado coma dose de 1,0 mg.kg-1... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: The clinical and behavioral effects of the intravenous injection of tramadol were evaluated during the control of pain induced experimentally due to intestinal extraluminal obstruction using "Pen Rose" drain. A total of 24 horses were used and distributed in four groups: control (GC, n=6); duodenal obstruction (GD, n=6); ileum obstruction (GI, n=6) and pelvic flexure obstruction (GFP, n=6). After administration of pre-anesthetic medications using association of acepromazine (0.025 mg.kg-1 IV), xylazine (0.5 mg.kg-1 IV) and meperidine (4 mg.kg-1 IM), tramadol was administered at doses of 1.0 mg.kg-1 and 1.5 mg.kg-1 intravenously (IV), immediately after the intestinal obstruction in three horses of each group. Evaluations were performed, including heart rate (HR), respiratory rate (RR), rectal temperature (RT), capillary refill time (CRT), gut motility, pain-related behaviour (look for the sidewall, dig down and roll) and blood gases from venous blood at the time: M0 (baseline) and every 0.5 hours from M1 to M6, during obstruction process and also until three hours after the obstructive process be reverted (M7 to M12). The results showed no significant difference among the doses used in the same group as among groups. There was an increase in HR in the GD M11 and M12 of GFP. Signs of abdominal pain and intestinal atony began at M5 in GFP and at M6 in GI. In animals from GD, the discomfort signs did not showed progress. On the leucogram was observed a typical stress and on the blood gas analysis the animals from GD showed a tendency to metabolic alkalosis with respiratory compensation. Clinically, was observed that the dose of 1.5 mg.kg-1 of tramadol provided better comfort to the animals, but there was not statistical significance, compared with the dose 1.0 mg.kg-1... (Complete abstract click electronic access below)
Mestre
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Uribe, Diaz Andrea del Pilar [UNESP]. "Aspectos clínicos-laboratoriais do uso do azul de metileno na obstrução experimental do jejuno em equinos expostos ao lipopolissacarídeo (LPS)." Universidade Estadual Paulista (UNESP), 2009. http://hdl.handle.net/11449/101103.

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Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)
Trabalhos recentes relatam a eficácia do azul de metileno na prevenção dos danos impostos por espécies reativas de oxigênio aos tecidos de vários órgãos, em vários modelos de isquemia/reperfusão. Este estudo foi concebido com o fito de avaliar o efeito do azul de metileno sobre as respostas clínico-laboratoriais, na obstrução experimental do jejuno em equinos, associada à exposição de lipopolissacarídeo. Dois grupos de animais foram submetidos à indução da endotoxemia e à obstrução experimental do jejuno em período anterior. Posteriormente administrou-se em um deles, infusão de azul de metileno (3 mg/kg I.V), 15 minutos antes da obstrução experimental, e no outro, a mesma infusão 15 minutos antes da desobstrução do jejuno. Foi realizada avaliação clínica, hematológica e bioquímico-sérica, e perfil bioquímico e citológico do líquido peritoneal a partir de aferições em oito tempos durante 12 horas, também foram caracterizadas as lesões intestinais. Após 3 horas de isquemia, verificou-se hemorragia, edema, infiltração de neutrófilos e desprendimento da mucosa. Essas lesões manifestaram-se predominantemente após a reperfusão, e de forma concomitante com o aumento dos componentes celulares e moleculares da inflamação, tanto no sangue quanto no líquido peritoneal. Contudo, todos os achados foram discretamente menos evidentes nos animais que receberam o azul de metileno antes da fase de reperfusão. Não é possível afirmar efeito benéfico do azul de metileno sobre a resposta dos equinos na obstrução experimental do jejuno.
Intestinal ischemia is one of the most serious intra-abdominal alterations and reflects extremely elevated morbility and mortality. Reoxygenation on the ischemic tissue produces deleterious inflammatory events with consequences even more severe than the ischemia itself. Methylene blue, due to this action as an inhibitor of free-radical formation. The objective of this test was to study the effects of methylene blue on the clinical and laboratory response before and after the experimental obstruction of the jejunum, associated to the exposition of lypopolysacharide. Two groups of animals were submitted to endotoxemia and experimental obstruction of the jejunum. After, in one group was administered, intravenously, a solution of methylene blue (3 mg/kg), immediately before the experimental obstruction, in the other group, the solution was administered immediately before interrupt the obstructive process in the intestinal segment. The horses were submitted to the evaluation of clinical signs and laboratory response during the 12 hours of study. Based on the clinical and laboratory findings we concluded that, the administration of methylene blue was not able to avoid the clinical and laboratory responses in the experimental model proposed for this study.
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Books on the topic "Intestinal obstruction"

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P, Fielding L., and Welch John P. 1942-, eds. Intestinal obstruction. Edinburgh: Churchill Livingstone, 1987.

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Petrov, V. P. Kishechnaia neprokhodimost'. Moskva: Meditsina, 1989.

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Parker, James N., and Philip M. Parker. The official patient's sourcebook on intestinal pseudo-obstruction. Edited by Icon Group International Inc and NetLibrary Inc. San Diego, Calif: Icon Health Publications, 2002.

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Fisher, Stephen J. Colon cancer & the polyps connection. Tuscon, Ariz: Fisher Books, 1995.

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G, Hampton Beverly, Bryant Ruth A, and International Association for Enterostomal Therapy., eds. Ostomies and continent diversions: Nursing management. St. Louis: Mosby-Year Book, 1992.

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Keith, Lierre. The vegetarian myth: Food, justice and sustainability. Crescent City, Ca: Flashpoint Press, 2009.

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Gant, Samuel Goodwin. Constipation and Intestinal Obstruction. Franklin Classics Trade Press, 2018.

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Constipation and Intestinal Obstruction. Franklin Classics, 2018.

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Intestinal obstruction from gall-stone. [S.l: s.n., 1985.

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Gant, Samuel Goodwin. Constipation And Intestinal Obstruction, Obstipation. Kessinger Publishing, LLC, 2007.

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Book chapters on the topic "Intestinal obstruction"

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Stephen, Andrew H., Charles A. Adams, and William G. Cioffi. "Intestinal Obstruction." In Geriatric Trauma and Critical Care, 139–44. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8501-8_14.

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McCallion, Kevin. "Intestinal obstruction." In Gastrointestinal emergencies, 220–24. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118662915.ch30.

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Augustin, Goran. "Intestinal Obstruction." In Acute Abdomen During Pregnancy, 221–77. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-05422-3_7.

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McKenzie, Travis J., and D. Dean Potter. "Intestinal Obstruction." In Guide to Pediatric Urology and Surgery in Clinical Practice, 231–37. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84996-366-4_25.

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Stephen, Andrew H., Charles A. Adams, and William G. Cioffi. "Intestinal Obstruction." In Geriatric Trauma and Critical Care, 161–68. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-48687-1_16.

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Sotiropoulou, Maria. "Intestinal Obstruction." In Encyclopedia of Pathology, 409–12. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-40560-5_1499.

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Gold, Michael S., and Stephen E. Goldstone. "Intestinal Obstruction." In Handbook of Hematologic and Oncologic Emergencies, 205–13. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4899-0476-8_18.

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Augustin, Goran. "Intestinal Obstruction." In Acute Abdomen During Pregnancy, 269–343. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-72995-4_7.

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O'Connell, P. Ronan, Andrew W. McCaskie, and Robert D. Sayers. "Intestinal obstruction." In Bailey & Love's Short Practice of Surgery, 1375–92. 28th ed. Boca Raton: CRC Press, 2022. http://dx.doi.org/10.1201/9781003106852-89.

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Lim, Jae Hoon. "Intestinal Obstruction." In Ultrasound of the Gastrointestinal Tract, 45–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/174_2013_798.

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Conference papers on the topic "Intestinal obstruction"

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"Clinical Study on the Treatment of Metastatic Malignant Bowel Obstruction with Transgastric Intestinal Obstruction Catheter Arrangement Small Intestinal Enterostomy." In 2018 International Conference on Medicine, Biology, Materials and Manufacturing. Francis Academic Press, 2018. http://dx.doi.org/10.25236/icmbmm.2018.48.

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"The Study of Complications of Intestinal Obstruction Catheter." In 2018 International Conference on Medicine, Biology, Materials and Manufacturing. Francis Academic Press, 2018. http://dx.doi.org/10.25236/icmbmm.2018.49.

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Zaghloul, M., M. Emara, H. Albatee, I. Amer, A. Mahrous, and M. Ahmed. "CROHN´S DISEASE PRESENTING WITH SUBACUTE INTESTINAL OBSTRUCTION." In ESGE Days. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1704670.

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Khan, Muhammad Umar, Sumair Aziz, Maira Sohail, Syed Zohaib Hassan Naqvi, Sana Samer, and Zurria Sajid. "Detection of Subacute Intestinal Obstruction from Surface Electromyography Signatures." In 2020 International Conference on Emerging Trends in Smart Technologies (ICETST). IEEE, 2020. http://dx.doi.org/10.1109/icetst49965.2020.9080710.

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Rijo Poueriet, A. A., E. Hernandez Caro, S. Sood, L. A. Parton, A. Ramirez, H. Christie, A. J. Dozor, and S. S. Krishnan. "Fecal Calprotectin and Intestinal Microbiota in Children With Cystic Fibrosis and Distal Intestinal Obstruction Syndrome." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a4136.

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Zhu, Yunhao, Linyu Ni, Laura A. Johnson, Jie Yuan, Xueding Wang, Peter D. R. Higgins, and Guan Xu. "Characterizing intestinal obstruction using a photoacoustic-ultrasound catheter (Conference Presentation)." In Photons Plus Ultrasound: Imaging and Sensing 2020, edited by Alexander A. Oraevsky and Lihong V. Wang. SPIE, 2020. http://dx.doi.org/10.1117/12.2544305.

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Sautin, A., Y. Kaminskaya, and K. Marakhouski. "Endoscopy Of Congenital Partial High Intestinal Obstruction: Improving Diagnostic Accuracy." In ESGE Days 2021. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1724947.

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Rhoads, S., M. F. Blundin, and D. Banerjee. "Distal Intestinal Obstruction Syndrome in a Pregnant Woman with Cystic Fibrosis." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a2823.

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Kolesnikov, Ye B., and M. I. Znaievskyi. "Artificial intelligence in the diagnosis and treatment of acute intestinal obstruction." In INNOVATIONS IN MEDICINE: ACHIEVEMENTS OF DOMESTIC AND FOREIGN REPRESENTATIVES. Baltija Publishing, 2024. http://dx.doi.org/10.30525/978-9934-26-439-9-6.

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Jansen, J., E. Van Nieuwenhuysen, P. Neven, T. Van Gorp, I. Vergote, and SN Han. "EP877 Intestinal (sub)obstruction in ovarian cancer patients: management, complications and survival." In ESGO Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-esgo.925.

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Reports on the topic "Intestinal obstruction"

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Huang, Yishen, Zhongxin Li, and Yitao Jia. Risk factors for postoperative intestinal obstruction in colorectal cancer. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2021. http://dx.doi.org/10.37766/inplasy2021.4.0125.

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Garcias, Lucas. Obstruction of the Small Intestine in the Abdomen without Surgery: Presentation of 5 Cases. Science Repository, December 2022. http://dx.doi.org/10.31487/j.jscr.2022.02.04.

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Abstract:
Introduction: The majority of SBOs develop secondary to postoperative adhesions, however nonsurgical etiologies must also be considered. Patients with no surgical history can develop SBO secondary to hernias, radiation, and other miscellaneous causes. Materials and Methods: Five cases of intestinal occlusion in a patient without previous abdominal surgery are presented. Discussion: Small bowel obstruction has been recognized as a life-threatening disease process. Stable patients should undergo conservative treatment and progress to surgical intervention only after failure of conservative treatment. Conclusion: Intestinal occlusion in patients without previous surgeries is a challenge for the Surgeon.
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Newman-Toker, David E., Susan M. Peterson, Shervin Badihian, Ahmed Hassoon, Najlla Nassery, Donna Parizadeh, Lisa M. Wilson, et al. Diagnostic Errors in the Emergency Department: A Systematic Review. Agency for Healthcare Research and Quality (AHRQ), December 2022. http://dx.doi.org/10.23970/ahrqepccer258.

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Abstract:
Objectives. Diagnostic errors are a known patient safety concern across all clinical settings, including the emergency department (ED). We conducted a systematic review to determine the most frequent diseases and clinical presentations associated with diagnostic errors (and resulting harms) in the ED, measure error and harm frequency, as well as assess causal factors. Methods. We searched PubMed®, Cumulative Index to Nursing and Allied Health Literature (CINAHL®), and Embase® from January 2000 through September 2021. We included research studies and targeted grey literature reporting diagnostic errors or misdiagnosis-related harms in EDs in the United States or other developed countries with ED care deemed comparable by a technical expert panel. We applied standard definitions for diagnostic errors, misdiagnosis-related harms (adverse events), and serious harms (permanent disability or death). Preventability was determined by original study authors or differences in harms across groups. Two reviewers independently screened search results for eligibility; serially extracted data regarding common diseases, error/harm rates, and causes/risk factors; and independently assessed risk of bias of included studies. We synthesized results for each question and extrapolated U.S. estimates. We present 95 percent confidence intervals (CIs) or plausible range (PR) bounds, as appropriate. Results. We identified 19,127 citations and included 279 studies. The top 15 clinical conditions associated with serious misdiagnosis-related harms (accounting for 68% [95% CI 66 to 71] of serious harms) were (1) stroke, (2) myocardial infarction, (3) aortic aneurysm and dissection, (4) spinal cord compression and injury, (5) venous thromboembolism, (6/7 – tie) meningitis and encephalitis, (6/7 – tie) sepsis, (8) lung cancer, (9) traumatic brain injury and traumatic intracranial hemorrhage, (10) arterial thromboembolism, (11) spinal and intracranial abscess, (12) cardiac arrhythmia, (13) pneumonia, (14) gastrointestinal perforation and rupture, and (15) intestinal obstruction. Average disease-specific error rates ranged from 1.5 percent (myocardial infarction) to 56 percent (spinal abscess), with additional variation by clinical presentation (e.g., missed stroke average 17%, but 4% for weakness and 40% for dizziness/vertigo). There was also wide, superimposed variation by hospital (e.g., missed myocardial infarction 0% to 29% across hospitals within a single study). An estimated 5.7 percent (95% CI 4.4 to 7.1) of all ED visits had at least one diagnostic error. Estimated preventable adverse event rates were as follows: any harm severity (2.0%, 95% CI 1.0 to 3.6), any serious harms (0.3%, PR 0.1 to 0.7), and deaths (0.2%, PR 0.1 to 0.4). While most disease-specific error rates derived from mainly U.S.-based studies, overall error and harm rates were derived from three prospective studies conducted outside the United States (in Canada, Spain, and Switzerland, with combined n=1,758). If overall rates are generalizable to all U.S. ED visits (130 million, 95% CI 116 to 144), this would translate to 7.4 million (PR 5.1 to 10.2) ED diagnostic errors annually; 2.6 million (PR 1.1 to 5.2) diagnostic adverse events with preventable harms; and 371,000 (PR 142,000 to 909,000) serious misdiagnosis-related harms, including more than 100,000 permanent, high-severity disabilities and 250,000 deaths. Although errors were often multifactorial, 89 percent (95% CI 88 to 90) of diagnostic error malpractice claims involved failures of clinical decision-making or judgment, regardless of the underlying disease present. Key process failures were errors in diagnostic assessment, test ordering, and test interpretation. Most often these were attributed to inadequate knowledge, skills, or reasoning, particularly in “atypical” or otherwise subtle case presentations. Limitations included use of malpractice claims and incident reports for distribution of diseases leading to serious harms, reliance on a small number of non-U.S. studies for overall (disease-agnostic) diagnostic error and harm rates, and methodologic variability across studies in measuring disease-specific rates, determining preventability, and assessing causal factors. Conclusions. Although estimated ED error rates are low (and comparable to those found in other clinical settings), the number of patients potentially impacted is large. Not all diagnostic errors or harms are preventable, but wide variability in diagnostic error rates across diseases, symptoms, and hospitals suggests improvement is possible. With 130 million U.S. ED visits, estimated rates for diagnostic error (5.7%), misdiagnosis-related harms (2.0%), and serious misdiagnosis-related harms (0.3%) could translate to more than 7 million errors, 2.5 million harms, and 350,000 patients suffering potentially preventable permanent disability or death. Over two-thirds of serious harms are attributable to just 15 diseases and linked to cognitive errors, particularly in cases with “atypical” manifestations. Scalable solutions to enhance bedside diagnostic processes are needed, and these should target the most commonly misdiagnosed clinical presentations of key diseases causing serious harms. New studies should confirm overall rates are representative of current U.S.-based ED practice and focus on identified evidence gaps (errors among common diseases with lower-severity harms, pediatric ED errors and harms, dynamic systems factors such as overcrowding, and false positives). Policy changes to consider based on this review include: (1) standardizing measurement and research results reporting to maximize comparability of measures of diagnostic error and misdiagnosis-related harms; (2) creating a National Diagnostic Performance Dashboard to track performance; and (3) using multiple policy levers (e.g., research funding, public accountability, payment reforms) to facilitate the rapid development and deployment of solutions to address this critically important patient safety concern.
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