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Books on the topic 'Intestinal obstruction'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 July 2024

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1

P, Fielding L., and Welch John P. 1942-, eds. Intestinal obstruction. Edinburgh: Churchill Livingstone, 1987.

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2

Petrov, V. P. Kishechnaia neprokhodimost'. Moskva: Meditsina, 1989.

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3

Parker, James N., and Philip M. Parker. The official patient's sourcebook on intestinal pseudo-obstruction. Edited by Icon Group International Inc and NetLibrary Inc. San Diego, Calif: Icon Health Publications, 2002.

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4

Fisher, Stephen J. Colon cancer & the polyps connection. Tuscon, Ariz: Fisher Books, 1995.

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5

G, Hampton Beverly, Bryant Ruth A, and International Association for Enterostomal Therapy., eds. Ostomies and continent diversions: Nursing management. St. Louis: Mosby-Year Book, 1992.

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6

Keith, Lierre. The vegetarian myth: Food, justice and sustainability. Crescent City, Ca: Flashpoint Press, 2009.

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7

Gant, Samuel Goodwin. Constipation and Intestinal Obstruction. Franklin Classics Trade Press, 2018.

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8

Constipation and Intestinal Obstruction. Franklin Classics, 2018.

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9

Intestinal obstruction from gall-stone. [S.l: s.n., 1985.

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10

Gant, Samuel Goodwin. Constipation And Intestinal Obstruction, Obstipation. Kessinger Publishing, LLC, 2007.

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11

Intestinal Obstruction Together with Peritonitis. Gryphon/ Classics of Surgery Library, 1986.

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12

Constipation and Intestinal Obstruction (Obstipation). Creative Media Partners, LLC, 2022.

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13

Constipation and Intestinal Obstruction (Obstipation). Creative Media Partners, LLC, 2022.

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14

Gant, Samuel Goodwin. Constipation and Intestinal Obstruction (Obstipation). Creative Media Partners, LLC, 2018.

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15

Gant, Samuel Goodwin. Constipation and Intestinal Obstruction (Obstipation). Creative Media Partners, LLC, 2018.

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16

Intestinal obstruction from gall-stone. [S.l: s.n., 1985.

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17

Scaglione, Mariano, and Roberto Di Mizio. Small-Bowel Obstruction: CT Features with Plain Film and US Correlations. Springer London, Limited, 2007.

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18

(Editor), Roberto Di Mizio, and Mariano Scaglione (Editor), eds. Small-Bowel Obstruction: CT Features with Plain Film and US correlations. Springer, 2007.

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19

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Intestinal failure. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0013.

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Short-bowel syndrome 98Excessive diarrhoea 100Motility disorders 101Mucosal disorders 102The term intestinal failure (IF) refers to a functionally impaired gastrointestinal tract unable to maintain biochemical homeostasis and support normal growth. Short-bowel syndrome (SBS) is a common cause of IF and usually defined as a severe reduction in functional gut mass below the minimal amount necessary for digestion and absorption adequate to satisfy the nutrient and fluid requirements for growth. Other causes of IF include mucosal abnormalities giving rise to protracted diarrhoea, and neuromuscular disorders resulting in chronic idiopathic intestinal pseudo-obstruction syndrome (CIIPS). See ...
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20

Treves, Frederick. Intestinal Obstruction: Its Varieties With Their Pathology, Diagnosis, and Treatment. Arkose Press, 2015.

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21

Treves, Frederick. Intestinal Obstruction, its Varieties With Their Pathology, Diagnosis, and Treatment. Arkose Press, 2015.

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22

Intestinal Obstruction; Its Varieties: With Their Pathology, Diagnosis, and Treatment. Creative Media Partners, LLC, 2022.

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23

Publications, ICON Health. Intestinal Obstruction - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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24

Intestinal obstruction due to bands: The result of a former appendectomy and complicated by a volvulus of the mesentery. [S.l: s.n., 1985.

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25

Stevens, Philip, and Paul Dark. Ileus and obstruction in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0182.

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Obstruction is the commonest cause of acute intestinal failure in critical care. Management is dependent upon whether it is adynamic or mechanical in origin. Paralytic ileus is managed conservatively by correction of electrolyte disturbances, nutritional support, and minimization of enterostatic drug use. Pharmacological agents aimed at reducing sympathetic stimuli may be useful, although widespread application is limited due to anti-muscarinic side effects. Peripherally acting μ‎-opioid receptor antagonists, may have a role, although data in critical illness are lacking. Prokinetic agents have not been shown to reduce ileus in clinical trials. Colonoscopic decompression may be required when conservative management fails. Rarely, surgical decompression becomes necessary if ileus arises in the context of abdominal compartment syndrome. Mechanical obstruction is more likely to require surgery, although adhesional obstruction, responsible for 80% of small bowel obstruction, may settle within 7 days of conservative management. Large bowel obstruction is more commonly due to tumours, diverticular stricture, or volvulus, and more likely to require endoscopic or surgical intervention. The hallmark of obstruction is colic, characterized by an inability to settle, in contrast to the peritonitic patient who lies completely still. Peritonitis in the presence of obstruction indicates possible perforation or necrosis for which urgent operative intervention is required. Clinical features may be absent in sedated patients hence the index of suspicion should remain high in any critically-ill patient intolerant of enteral feeding.
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26

Stanton, Mike. Congenital abnormalities of the gastrointestinal tract. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759928.003.0001.

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This chapter covers the common congenital abnormalities. This includes detailed discussion of the gut development. Conditions covered include gastroschisis, exomphalos, malrotation, duodenal atresia, small bowel obstruction, distal intestinal obstruction, meconium ileus, Hirschsprung disease, and anorectal malformation.
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27

Publications, ICON Health. The Official Patient's Sourcebook on Intestinal Pseudo-Obstruction: A Revised and Updated Directory for the Internet Age. Icon Health Publications, 2002.

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28

Carton, James. Gastrointestinal pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0007.

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This chapter discusses gastrointestinal pathology, including gastrointestinal malformations, oesophagitis, oesophageal polyps and nodules, oesophageal carcinoma, gastritis, gastric polyps, gastric carcinoma, gastrointestinal stromal tumours, peptic duodenitis, coeliac disease, small bowel infarction, intestinal infections, intestinal obstruction, acute appendicitis, Crohn’s disease, ulcerative colitis, colorectal polyps, colorectal carcinoma, diverticular disease, and anal pathology.
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29

Victorovich Garbuzenko, Dmitry, ed. Intestinal Obstructions. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.89823.

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30

Watson, Max, Caroline Lucas, Andrew Hoy, and Jo Wells. Gastrointestinal symptoms. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199234356.003.0013.

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This chapter covers gastrointestinal symptoms, including oral problems and how to manage them, nausea and vomiting, antiemetic drugs, constipation, diarrhoea, intestinal obstruction, hiccup, anorexia/ cachexia/ asthenia, ascites, tenesmus and temesmoid pain, dyspepsia, gastrointestinal bleeding, and bowel stoma care.
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31

Keshav, Satish, and Alexandra Kent. Starvation and malnutrition. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0332.

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Starvation is a state of severe malnutrition due to a reduction in macro- and micronutrient intake. The basis underlying starvation is an imbalance between energy intake and energy expenditure. The commonest cause of starvation is lack of available food, usually due to environmental, social, and economic reasons, although other causes include anorexia nervosa; depression and other psychiatric disorders; coma and disturbance of consciousness; intestinal failure; and mechanical failure of digestion, including poor dentition and intestinal obstruction. Protein energy malnutrition is usually seen in developing countries. This chapter discusses starvation and malnutrition, focusing on their etiology, symptoms, demographics, natural history, complications, diagnosis, treatment, and prognosis.
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32

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Hirschsprung's disease. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0039.

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Hirschprung's disease 280Neuronal intestinal dysplasia 281Intestinal pseudo-obstruction 281Hirschsprung's disease is the absence of ganglion cells in the myenteric plexus of the most distal bowel. Presentation is with constipation. Incidence is 1 in 5000. Long-segment Hirschsprung's disease is familial, with equal sex incidence. The gene is on chromosome 10. It is associated with Down's syndrome and there is a high frequency of other congenital abnormalities....
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33

Fraser, Britt. Acute Fluid Resuscitation for Intussusception. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0004.

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Intussusception occurs when a proximal section of bowel invaginates into more distal bowel and is then advanced by peristalsis. It is the most common cause of intestinal obstruction in infants, and untreated can lead to bowel ischemia and perforation. Early recognition and treatment can prevent the need for surgical intervention and complications. Intussusception can also result in significant dehydration due to vomiting and diarrhea. An essential aspect of the perioperative management is to identify and treat dehydration.
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34

Hain, Richard D. W., and Satbir Singh Jassal. Palliative care emergencies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198745457.003.0015.

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A palliative care emergency describes a symptom situation that is serious and demands immediate and skilled specialist attention, but is also unusual enough for there to be a possibility that the specialist has not often encountered it. In a palliative care emergency, the patient experiences sudden and severe distress that can only be relieved by prompt and confident intervention by the palliative care team. There is little time to prepare for it, and, to the specialist in paediatric palliative medicine, an emergency is defined by the importance of already having a clear strategy for dealing with it. That can be challenging for those working in paediatric palliative care, because most are rare. This chapter covers the six main emergencies encountered in paediatric palliative medicine: cord compression, catastrophic haemorrhage, severe uncontrolled pain, superior vena cava obstruction, intestinal obstruction, and hypercalcaemia.
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35

Wyatt, Karla E. K., and Olutoyin A. Olutoye. Omphalocele/Gastroschisis. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0049.

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Omphalocele and gastroschisis are the most common types of abdominal wall defects encountered in the neonatal population. Both conditions result in the herniation of abdominal viscera through a defect in the upper or lower abdominal wall. These neonates can present with herniated organs, intestinal obstruction, impaired blood supply to visceral organs, and major fluid deficits. The primary goal for a neonate with an abdominal wall defect is surgical closure. Prior to achieving this goal, maintenance of perfusion of the herniated viscera and minimization of evaporative fluid losses secondary to the exposed surface area are paramount. This chapter discusses the distinctly different etiologies of these conditions, comorbidities, and prognoses, as well as the surgical and anesthetic management strategies and commonly encountered perioperative complications.
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36

White, Maddie. Colorectal and lower gastrointestinal surgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642663.003.0019.

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The lower gastrointestinal tract includes most of the small intestine and all of the large intestine. The patient presenting with an acute abdomen may have a bowel obstruction which could lead to ischaemia and perforation. Thorough assessment and close observation are imperative, because, if surgical intervention is indicated, this needs to be performed promptly. This chapter provides an overview of inflammatory bowel disorders, tumours, hernias, obstructions, the acute abdomen, ischaemic bowel, complications of bowel surgery, and stomas.
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37

Frenkel, Joost, and Hans R. Waterham. Mevalonate Kinase Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0039.

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Mevalonate kinase deficiency (MKD) is an autosomal recessive inborn error of isoprenoid biosynthesis, a pathway yielding sterols and nonsterol isoprenoids.In patients, the enzyme activity of mevalonate kinase is severely reduced due to mutations in the encoding gene, MVK. The substrate, mevalonate, accumulates and is elevated in blood and urine. Shortage of certain downstream products of the pathway, nonsterol isoprenoids, leads to dysregulation of the innate immune system, activation of inflammasomes, and interleukin (IL)-1 mediated inflammation.Symptoms start in early childhood with recurrent attacks of fever, vomiting, diarrhea, headache, sore throat, abdominal pain, arthralgias, painful lymphadenopathy, hepatosplenomegaly, skin rash, and mucosal ulcers. Severely affected patients have additional symptoms, such as intellectual impairment, progressive cerebellar ataxia, and tapetoretinal degeneration. Complications include intestinal obstruction, AA-amyloidosis, hemophagocytosis, and severe infection.Management of MKD is directed at controlling inflammation.
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38

Scaglione, Mariano, and Roberto Di Mizio. Small-Bowel Obstruction: CT Features with Plain Film and US correlations. Springer, 2014.

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39

Two cases of volvulus of small intestine. [S.l: s.n., 1986.

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40

Miller, Rosie. Intussusception and Bowel Obstruction: Symptoms, Diagnosis and Treatment Options. Nova Science Publishers, Incorporated, 2015.

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41

Report of a case of strangulated obturator hernia. [S.l: s.n., 1985.

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42

Gant, Samuel Goodwin. Diarrheal, Inflammatory, Obstructive, and Parasitic Diseases of the Gastro-Intestinal Tract. Arkose Press, 2015.

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43

Thomas, Hugh Owen. Past and Present Treatment of Intestinal Obstructions: Reviewed, with an Improved Treatment Indicated. Creative Media Partners, LLC, 2018.

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44

Thomas, Hugh Owen. The Past and Present Treatment of Intestinal Obstructions: Reviewed, with an Improved Treatment Indicated. Franklin Classics Trade Press, 2018.

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45

Thomas, Hugh Owen. The Past And Present Treatment Of Intestinal Obstructions: Reviewed, With An Improved Treatment Indicated. Franklin Classics, 2018.

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46

The Past And Present Treatment Of Intestinal Obstructions: Reviewed, With An Improved Treatment Indicated. Franklin Classics, 2018.

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47

Helling, Kevin D., and Scott A. Shikora. Intestinal Complications of Roux-en-Y Gastric Bypass. Edited by Tomasz Rogula, Philip Schauer, and Tammy Fouse. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190608347.003.0029.

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Roux-en-Y gastric bypass is a commonly performed bariatric operation, but it is a formidable procedure performed in technically challenging, medically high-risk patients. Although it is highly successful for achieving meaningful and durable weight loss, a variety of intestinal complications may occur. These include small bowel obstructions from a number of sources (internal hernias, adhesions, intussusception, incisional hernias, intestinal volvulus), anastomotic strictures, dumping syndrome, portal vein thrombosis, Roux-en-O construction, and small bowel diverticulitis. This chapter reviews several of the more commonly occurring postoperative intestinal complications. Clinicians need to understand the signs and symptoms of these complications and must be able to quickly diagnose the condition and initiate treatment.
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48

Fruhwald, Sonja, and Peter Holzer. Gastrointestinal motility drugs in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0040.

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Gastrointestinal motility disturbances in critically-ill patients often require treatment with prokinetic drugs. The aetiology of motility disturbances is complex, and involves electrolyte imbalances, hypervolaemia, reduced intestinal secretion, adverse effects of drugs (catecholamines, opioids, or sedatives) and disease- or treatment-related changes of microflora. However, the choice of prokinetics is narrow, and the multiplicity of pathophysiological mechanisms often limits their efficacy. Gastroparesis can be managed with gastrokinetics such as domperidone, metoclopramide and erythromycin. Their choice depends not only on efficacy, but also on adverse effect profile. The arrhythmogenic potential of domperidone limits maximum daily dose and treatment duration. Metoclopramide and erythromycin induce tachyphylaxis, which restricts treatment duration. The combination of metoclopramide and erythromycin serves as rescue therapy in severe gastroparesis. Neostigmine and laxatives are used to manage colonic paralysis, and these treatment options may eventually be extended by drug candidates, such as prucalopride, lubiprostone, and linaclotide, whose utility in the ICU awaits to be evaluated. Neostigmine’s prokinetic efficacy in colonic paralysis is limited, but well documented in patients with acute colonic pseudo-obstruction (Ogilvie syndrome). Care is advocated in dosing because higher doses of neostigmine inhibit motility. Alternative options include osmotic and stimulant laxatives, especially for prophylactic use. The opioid receptor antagonist alvimopan is used for the short-term management of post-operative ileus, while methylnaltrexone is indicated in palliative care and chronic pain management. Since its efficacy in critically-ill patients remains to be proven, the use of methylnaltrexone in the ICU is off-label and requires proper documentation.
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49

Sivak, Erica, Marcus Malek, and Denise Hall-Burton. Hirschsprung Disease. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0037.

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Hirschsprung disease is characterized by the absence of ganglion cells in the enteric nervous system. Inability to pass meconium in the neonatal period, enterocolitis, bowel obstruction, or chronic constipation in older infants and children may be the presenting symptoms. Once diagnosed, surgical intervention is always required. Successful resection of all portions of aganglionic intestine may be accomplished through multiple surgical techniques. Depending upon the surgical approach required, regional anesthesia may be indicated to assist with pain control postoperatively. This chapter describes Hirschsprung disease and considers a variety of questions related to its diagnosis and treatment, as well as risks related to surgery, including anaphylaxis, laparoscopic complications, vascular injury, epidural complications, and issues related to neuraxial analgesia.
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50

Symptômes du Cancer Colorectal: Saignement Rectal, Douleur Abdominale, Changements Dans les Selles, Perte de Poids, Fatigue, Ballonnements, anémie, Satiété Précoce, Sentiment de Selles Incomplètes, Obstruction Ou Perforation Intestinale. Independently Published, 2021.

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