Relevant bibliographies by topics / Intimal layer of large arteries

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Academic literature on the topic 'Intimal layer of large arteries'

Author: Grafiati
Published: 7 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "Intimal layer of large arteries"

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Zhang, Hui, Ryu Watanabe, Gerald J. Berry, et al. "Immunoinhibitory checkpoint deficiency in medium and large vessel vasculitis." Proceedings of the National Academy of Sciences 114, no. 6 (2017): E970—E979. http://dx.doi.org/10.1073/pnas.1616848114.

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Giant cell arteritis (GCA) causes autoimmune inflammation of the aorta and its large branches, resulting in aortic arch syndrome, blindness, and stroke. CD4+ T cells and macrophages form organized granulomatous lesions in the walls of affected arteries, destroy the tunica media, and induce ischemic organ damage through rapid intimal hyperplasia and luminal occlusion. Pathogenic mechanisms remain insufficiently understood; specifically, it is unknown whether the unopposed activation of the immune system is because of deficiency of immunoinhibitory checkpoints. Transcriptome analysis of GCA-affe
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Planas-Rigol, Ester, Nekane Terrades-Garcia, Marc Corbera-Bellalta, et al. "Endothelin-1 promotes vascular smooth muscle cell migration across the artery wall: a mechanism contributing to vascular remodelling and intimal hyperplasia in giant-cell arteritis." Annals of the Rheumatic Diseases 76, no. 9 (2017): 1624–34. http://dx.doi.org/10.1136/annrheumdis-2016-210792.

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BackgroundGiant-cell arteritis (GCA) is an inflammatory disease of large/medium-sized arteries, frequently involving the temporal arteries (TA). Inflammation-induced vascular remodelling leads to vaso-occlusive events. Circulating endothelin-1 (ET-1) is increased in patients with GCA with ischaemic complications suggesting a role for ET-1 in vascular occlusion beyond its vasoactive function.ObjectiveTo investigate whether ET-1 induces a migratory myofibroblastic phenotype in human TA-derived vascular smooth muscle cells (VSMC) leading to intimal hyperplasia and vascular occlusion in GCA.Method
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Castro Júnior, Cyro, Adamastor Humberto Pereira, and Márcio Bastiani Pasa. "Morphometric analysis of the intimal reaction after stent implantation in iliac arteries submitted to angioplasty in pigs." Acta Cirurgica Brasileira 21, no. 3 (2006): 139–43. http://dx.doi.org/10.1590/s0102-86502006000300004.

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PURPOSE: To compare through digital morphometry, the intimal thickening of the common ilíac arteries (CIA) in pigs, submitted to angioplasty and angioplasty followed by stent implantation. METHODS: Angioplasty was performed in 10 pigs in both CIA, followed by a stent implantation in the left CIA. After four weeks, the aorto-iliac segment was dissected. Histologic slices where divided in three groups: left CIA in the area of the proximal (group 1) and distal (group 2) implantation site of the stent and the region of angioplasty in the right CIA (group 3). The histological slices were digitalize
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Mantskava, Maya, and Nana Momtselidze. "Clinical markers of functional condition of resistive arteries in the young men." Russian journal of biomechanics. 23, no. 4 (2019): 500–504. http://dx.doi.org/10.15593/rjbiomech/2019.4.09.

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Research of resistive vessels, assessment of their functional state is one of the key issues of blood circulation from the point of view of physics, angiology, rheology, blood biomechanics and related sciences. The article examined some physical quantities that describe the functional state of resistive arteries. According to the results of our study, such physical components as the thickness of the intimal medial layer, the coefficient of resistance of the resistive arteries and Young's module turned out to be interesting pathophysiological markers that can reflect the change in the vascular
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Akhtar, R., N. Schwarzer, M. J. Sherratt, et al. "Nanoindentation of histological specimens: Mapping the elastic properties of soft tissues." Journal of Materials Research 24, no. 3 (2009): 638–46. http://dx.doi.org/10.1557/jmr.2009.0130.

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Although alterations in the gross mechanical properties of dynamic and compliant tissues have a major impact on human health and morbidity, there are no well-established techniques to characterize the micromechanical properties of tissues such as blood vessels and lungs. We have used nanoindentation to spatially map the micromechanical properties of 5-μm-thick sections of ferret aorta and vena cava and to relate these mechanical properties to the histological distribution of fluorescent elastic fibers. To decouple the effect of the glass substrate on our analysis of the nanoindentation data, w
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Anda, Takeo, Wataru Haraguchi, Hajime Miyazato, et al. "Ruptured distal middle cerebral artery aneurysm filled with tumor cells in a patient with intravascular large B-cell lymphoma." Journal of Neurosurgery 109, no. 3 (2008): 492–96. http://dx.doi.org/10.3171/jns/2008/109/9/0492.

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The authors describe a very rare case of intravascular large B-cell lymphoma in a woman whose ruptured distal middle cerebral artery (MCA) aneurysms were filled with lymphoma cells. A 69-year-old woman who had undergone artificial graft replacement for an aortic aneurysm presented with transient left hemiparesis. Magnetic resonance imaging demonstrated a small fresh cerebral infarction in the right frontal lobe, although major cervical and cerebral arteries were shown to be intact on MR angiography. Antiplatelet and anticoagulation treatments commenced. On the 21st day after onset, the patient
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Watanabe, Ryu, Toshihisa Maeda, Hui Zhang, et al. "MMP (Matrix Metalloprotease)-9–Producing Monocytes Enable T Cells to Invade the Vessel Wall and Cause Vasculitis." Circulation Research 123, no. 6 (2018): 700–715. http://dx.doi.org/10.1161/circresaha.118.313206.

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Rationale: Giant cell arteritis (GCA)—a primary vasculitis of medium and large arteries—is associated with vessel wall damage, elastic membrane fragmentation, and vascular remodeling. Proteinases are believed to contribute to pathogenesis by degrading extracellular matrix and causing tissue injury. Objective: The MMP (matrix metalloproteinase)-9—a type IV collagenase—is produced in the vasculitic lesions of GCA. It is unknown which pathogenic processes are MMP-9 dependent. Methods and Results: The tissue transcriptome of GCA-affected temporal arteries contained high amounts of MMP-9 transcript
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Franco, E., D. Iancu, E. Cavallone, et al. "P391 THE DIAGNOSTIC CHALLENGE BETWEEN CHRONIC PULMONARY EMBOLISM AND PULMONARY ARTERY SARCOMA: A CASE REPORT." European Heart Journal Supplements 25, Supplement_D (2023): D198. http://dx.doi.org/10.1093/eurheartjsupp/suad111.462.

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Abstract We report the case of a 63–year–old obese and hypertensive woman, admitted in hospital with worsening dyspnea for 2 weeks and syncope. A suspected chronic thromboembolism was diagnosed nine months before for a similar clinical picture; hematologic screening excluded thrombophilia and NOACs were prescribed. Two–dimensional transthoracic echocardiography showed enlargement of right chambers, the interventricular septum shifted to the left ventricle, showing “D” sign. Color Doppler flow imaging showed moderate regurgitation in tricuspid valve, with indirect pulmonary pressure estimation
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Sun, Bing, Anthony P. Davenport, and Morris J. Brown. "Quantitative autoradiography reveals higher densities of specific calcitonin-gene-related peptide receptors in small intramyocardial compared with large epicardial coronary arteries." Clinical Science 84, no. 1 (1993): 55–60. http://dx.doi.org/10.1042/cs0840055.

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1. Binding sites for calcitonin-gene-related peptide were localized and characterized in porcine coronary arteries using quantitative autoradiography, and the density of binding sites was compared between large epicardial and small intramyocardial coronary arteries. 2. A single class of binding sites for calcitonin-gene-related peptide with a dissociation constant of 2.1 ± 0.2 nmol/l was detected in both the large and small coronary arteries. The density of specific binding sites was higher (maximum binding site density 231 ± 14 fmol/mg of protein) in the small coronary arteries than in the la
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Greco, Francesco, Laura Quercioli, Angela Pucci, et al. "Mass Spectrometry Imaging as a Tool to Investigate Region Specific Lipid Alterations in Symptomatic Human Carotid Atherosclerotic Plaques." Metabolites 11, no. 4 (2021): 250. http://dx.doi.org/10.3390/metabo11040250.

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Atherosclerosis is characterized by fatty plaques in large and medium sized arteries. Their rupture can causes thrombi, occlusions of downstream vessels and adverse clinical events. The investigation of atherosclerotic plaques is made difficult by their highly heterogeneous nature. Here we propose a spatially resolved approach based on matrix-assisted laser desorption/ionization (MALDI) mass spectrometry imaging to investigate lipids in specific regions of atherosclerotic plaques. The method was applied to a small dataset including symptomatic and asymptomatic human carotid atherosclerosis pla
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Books on the topic "Intimal layer of large arteries"

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Bochaton-Piallat, Marie-Luce, Carlie J. M. de Vries, and Guillaume J. van Eys. Vascular smooth muscle cells. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0007.

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To understand the function of arteries in the regulation of blood supply throughout the body it is essential to realize that the vessel wall is composed predominantly of smooth muscle cells (SMCs) with only one single layer of luminal endothelial cells. SMCs determine the structure of arteries and are decisive in the regulation of blood flow. This review describes the reason for the large variation of SMCs throughout the vascular tree. This depends on embryonic origin and local conditions. SMCs have the unique capacity to react to these conditions by modulating their phenotype. So, in one situ
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Book chapters on the topic "Intimal layer of large arteries"

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post, Mark, and J. Anthony ware. "Angiogenesis in Atherosclerosis and Restenosis." In Angiogenesis And Cardiovascular Disease. Oxford University PressNew York, NY, 1999. http://dx.doi.org/10.1093/oso/9780195112351.003.0007.

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Abstract Small arteries and arterioles that penetrate the adventitia and the media provide the blood supply of the wall of medium and large-size arteries. The intima and the lumenal part of the media are usually avascular; the thickness of this avascular layer is surprisingly constant (approximately 0.5 mm) even when the arterial wall thickens with age and seems to be determined by the diffusion coefficient of oxygen (Wolinsky and Glagov, 1967). Thus, the thickened arterial wall seen during adulthood is supported by new blood vessels that grow into the media.
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Campbell, Gordon R., John A. Bingley, Ian P. Hayward, and Julie H. Campbell. "Smooth muscle cells and the connective tissue matrix of the intima." In Atherosclerosis. Oxford University PressOxford, 2000. http://dx.doi.org/10.1093/oso/9780198506379.003.0010.

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Abstract Diffuse intimal thickenings in large and medium-sized arteries are sites for the development of atherosclerosis. For the first one to two decades in most humans, the thickening is composed of smooth muscle cells completely surrounded by extracellular matrix and covered by an endothelium. Occasionally, isolated macrophages are observed beneath this endothelium layer. The extracellular matrix provides two primary functions for cells: (i) it maintains the spatial relationships between cells; and maintains their differentiated state. In this context, it is intriguing to consider the fact
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Gaziano, J. Michael, and Daniel Steinberg. "Natural Antioxidants." In Prevention of Myocardial Infarction. Oxford University PressNew York, NY, 1996. http://dx.doi.org/10.1093/oso/9780195085822.003.0013.

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Abstract Natural History of the Atherosclerotic Lesion. Atherosclerosis is an extremely com plex, slowly developing chronic disease of the arteries. There is no longer any doubt that hypercholesterolemia is a major causative factor, a conclusion based on a wealth of experimental, clinical epidemiologic, and interventional data (Consensus Conference 1985). Over the past 10 to 15 years there has been a convergence of view with regard to the sequence of events that initiate atherosclerosis and how low density lipoprotein (LDL) plays a role (Ross 1976; Goode 1977; Steinberg 1983; Hansson 1989; Ste
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Choudhury, Robin P., Joshua T. Chai, and Edward A. Fisher. "Biology and pathology of atherosclerosis." In Oxford Textbook of Medicine, edited by Jeremy Dwight. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0364.

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Atherosclerotic plaques are not randomly distributed, but tend to form at the inner curvatures and branch points of arteries, where laminar flow is either disturbed or insufficient to support the normal, quiescent state of the endothelium (the lining of endothelial cells that separates the circulating blood from the arterial wall). This is an inflammatory process that involves the contribution of endothelial cells, lymphocytes, monocytes, and smooth muscle cells in conjunction with the deposition of atherogenic lipoproteins in the intimal layer of the vascular wall. The initial stage involves
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O’Reilly*, M. J. G. "The Numerical Investigation of a Novel Haemodynamic Control Device which Reduces the Development of Occlusive Arterial Intimal Hyperplasia." In Cardiovascular Flow Modelling and Measurement with Application to Clinical Medicine. Oxford University PressOxford, 1999. http://dx.doi.org/10.1093/oso/9780198505204.003.0012.

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Abstract A computational fluid dynamics investigation of the pulsatile, non Newtonian blood flow through a typical, three-dimensional model of a human femorodistal bypass graft has emphasised the complicated temporal and spatial dependence of the flow patterns at the proximal and distal anastomoses. A novel, simply configured Haemodynamic Control Device (HCD), fit ted at the ends of the graft, judiciously modified the anastomotic flow fields. Flow separation and recirculation were diminished. It is believed that the successful optimisation of the HCD would have important benefits for vascular
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Schneider, Elizabeth, Christy Dyer, Alex Dopico, and Anna Bukiya. "BK Channels, Cholesterol, and Cerebral Arteries." In Potassium in Health and Disease [Working Title]. IntechOpen, 2025. https://doi.org/10.5772/intechopen.1008214.

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Potassium homeostasis is essential for a variety of physiological functions, many of which are critically dependent on proper function and expression of potassium channels. Though only a small portion of the large potassium channel family, voltage- and calcium-gated potassium channels of large conductance (MaxiK, BK) are ubiquitously expressed in the human body, and contribute to potassium homeostasis. Of particular interest, BK channels are major regulators of cerebral artery smooth muscle tone and diameter. BK-mediated currents are inhibited by many endogenous ligands including cholesterol,
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Kuret, Tadeja, and Snežna Sodin-Šemrl. "The Role of Fibroblasts in Atherosclerosis Progression." In Fibroblasts - Advances in Cancer, Autoimmunity and Inflammation [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.98546.

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The following chapter addresses vascular fibroblasts in a healthy, quiescent state, as well during vascular inflammation, focusing on atherosclerosis. The development of atherosclerosis, an inflammatory disease of medium- and large-sized arteries, has traditionally been viewed as an “inside-out” mechanism, with prominent roles of the innermost layer of the artery, consisting of endothelial cells. However, emerging evidence suggests a new paradigm of “outside-in” mechanism, including an earlier role for fibroblasts, constituents of the outermost adventitial layer of the artery. Phenotypic and f
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Benson, Matthew D., Elias I. Traboulsi, and Brian P. Brooks. "Congenital Anomalies of the Optic Nerve." In Genetic Diseases of the Eye, 3rd ed. Oxford University PressNew York, 2025. https://doi.org/10.1093/med/9780197659403.003.0012.

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Abstract This chapter traces the normal development of the optic nerve throughout gestation before discussing aplasia of the optic nerve, a rare condition in which the optic nerve fails to develop, and optic nerve hypoplasia, a congenital, nonprogressive developmental abnormality characterized by reduced numbers of retinal ganglion cells and corresponding defects in the nerve fiber layer of the retina. Other conditions covered are duplication of the optic nerve, an extremely rare condition characterized by the presence of two separate nerves exiting from the globe, pseudo-duplication of the op
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Conference papers on the topic "Intimal layer of large arteries"

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Galvin, D. AJ, A. C. Meek, K. German, R. A. Harper, and C. N. McCollum. "CAROTID INTIMAL TRAUMA: A MODEL FOR PLATELET INTERACTIONS WITH DAMAGED ENDOTHELIUM." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643369.

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Radiolabelled platelet deposition on prosthetic arterial grafts may be used as a method to evaluate antithrombotic drugs but requires large animals or patients and involves artificial flow surfaces. A rabbit model has been developed to investigate platelet uptake following carotid intimal damage.Both carotid arteries of 10 NZW rabbits were exposed by midline incision and intimal damage inflicted by a 3-minute unilateral artery compression using the jaws from non-toothed dissecting forceps mounted on a G-clamp with compression screw. Twenty-four hours later, autologous platelets were labelled w
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Asada, Y., T. Hayashi, and A. Sumiyoshi. "ENDOTHELIAL CELL INJURIES AND SMOOTH MUSCLE CELL PROLIFERATION INDUCED BY MATERIALS RELEASED FROM PLATELET-RICH THROMBUS IN VIVO." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644601.

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It is widely held that the disturbance in the integrity of the arterial endothelium may lead to the development of arteriosclerosis and many factors have been postulated to cause the endothelial injury, such as hemodynamic stress, anoxia, platelet-releasing materials, and so on. However, whether any of these is important for endothelial injury is unclear. We studied whether the released products from activated platelets and/or thrombi could cause endothelial damage and proliferation of smooth muscle cells in large vessels in vivo.Polyethylene tubing was inserted into the ascending aorta of rab
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Lim, Yi Chung, and David S. Long. "Aortic Hemodynamics and Endothelial Gene Expression: An Animal Specific Approach." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53312.

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Atherosclerosis is a major cause of morbidity and mortality in the developed world. This disease is identified by endothelial dysfunction, inflammation and the accumulation of lipids and cellular elements within the intima of medium and large-sized arteries. Within these arteries, the distribution of atherosclerotic lesions is non-uniform; the inner wall of curved sections and the outer walls of bifurcations are susceptible sites. Evidence suggests that the focal nature of the disease is mediated in part by local fluid mechanical stresses at the interface between flowing blood and the vessel w
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Martin, John T., and Virginia L. Ferguson. "Regional Similarities in the Mechanical Properties of the Human Umbilical Artery." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206800.

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The human umbilical cord (UC) bridges the blood flow gap between baby and mother, protecting the blood supply in a way that allows the fetus freedom to move within the amniotic sac. Once the blood supply has been oxygenated by the maternal blood pool via the placenta, the umbilical vein (UV) provides a transport pathway to the fetus. Two umbilical arteries (UA) return the blood supply to the pool to eliminate CO2 and other metabolic wastes [1]. The walls of the UA’s and UV are made up of an intima composed a single layer of large, elongated endothelial cells [2], and a media composed of random
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Booth, R. F. G., J. F. Martin, S. Moncada, and A. C. Honey. "IS ATHEROSCLEROSIS A DISEASE OF THE OUTSIDE OF ARTERIES?" In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643083.

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The early stages of atherosclerosis are characterised by: (i) Intimal infiltration by macrophages, (ii) Intimal smooth muscle cell (SMC) proliferation and (iii) Accumulation of cholesterol within the vessel wall. Many studies have suggested that these changes occur subsequent to endothelial cell (EC) damage and platelet deposition. We now describe a new animal model with characteristics of early human atherosclerosis but without observable EC damage or platelet deposition. 12 rabbits were separated into 2 groups; each was fed normal laboratory chow and one group was supplemented with lg/day ch
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Kopacz, Adrian M., Brandon J. Tefft, Shu Q. Liu, and Wing K. Liu. "Modeling of Endothelial Cell Adhesion Dynamics Modulated by Molecular Engineering." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13269.

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Vascular thrombosis, intimal hyperplasia, and atherosclerosis are common disorders affecting a very large portion of the human population. A potential reduction in these disorders will elicit a significant impact. It has been shown that endothelial cells play a critical role in protecting blood vessels against the formation of thrombosis and atherosclerosis. Hence, a successful endothelial cell lining of arterial constructs will prevent intimal hyperplasia in reconstructed arteries. However, in practice endothelial cells often detach from reconstructed arteries due to weak adhesion strength, h
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Zhao, Shijia, Linxia Gu, and Shengmao Lin. "Mechanical Characterization of the Layer-Specific Human Aorta." In ASME 2012 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/imece2012-87083.

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In this work, three layers of human aorta, i.e. intima, media and adventitia, obtained from a 9 mm CryoValve® aortic valve allograft, were tested along both axial and circumferential directions. Preconditioning with a stretch ratio of 1.35 was used to mimic the physiological pulsatile loading conditions of the tissue. Results suggested that the stiffness along circumferential direction is generally larger than axial direction in each of the three layers. In all three layers, the media layer is the stiffest, and the adventitia layer is the softest regardless of testing directions. The anisotrop
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Zahedmanesh, Houman, Paul Gatenholm, and Caitríona Lally. "Bacterial Cellulose: A Potential Vascular Graft and Tissue Engineering Scaffold." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206343.

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Cardiovascular disease is the major cause of morbidity and mortality in the adult population and treatment of many of the patients struggling with such diseases requires surgical interventions involving replacement of diseased arteries. Although currently it is possible to replace large arteries with synthetic grafts made of materials such as polyester and expanded polytetrafluoroethylene (ePTFE) [1], these materials may not be used as small size grafts given their thrombogenicity and propensity for intimal hyperplasia [2]. As a result, development of suitable grafts as a substitute for small
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Teodorescu, Mircea, Moshe Brand, Jacob Rosen, and Homer Rahnejat. "The Influence of Post Angioplasty Stent Implant Profile on Arterial Wall Stress." In ASME 2010 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2010. http://dx.doi.org/10.1115/detc2010-29233.

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One of the main causes for post angioplasty arterial restenosis is the excessive stress induced in the arterial wall during and after the medical intervention. The closed stent is introduced in the artery wrapped around the deflated balloon catheter and is expanded in the final position by inflating the balloon. Unfortunately, this process also stretches the arterial wall. Additionally, for the stent to be successful, its diameter must be slightly larger than the diameter of the inflated artery. The stent is usually a dense mesh of interconnected beams. Therefore, it is often considered that i
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Long, David S., Hui Zhu, and Morton H. Friedman. "Quantifying the Motion of Murine Epicardial Coronary Arteries." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192065.

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Coronary artery atherosclerosis is a leading cause of morbidity and mortality in western societies. Atherosclerosis is a progressive fibroinflammatory disease identified by intimal thickening, the focal accumulation of lipids, fibrous elements, and cellular elements within the walls of large arteries. These lesions preferentially develop at arterial branches, the outer walls of bifurcations, and the inner walls of curved sections; the cause of this focal vasculopathy is not fully understood. It is, however, understood from epidemiological and clinical studies that individual susceptibility to
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