Relevant bibliographies by topics / Intrapartum damage

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Academic literature on the topic 'Intrapartum damage'

Author: Grafiati
Published: 28 May 2022
Last updated: 30 May 2022

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Journal articles on the topic "Intrapartum damage"

1

Rei, M., D. Ayres-de-Campos, and J. Bernardes. "Neurological damage arising from intrapartum hypoxia/acidosis." Best Practice & Research Clinical Obstetrics & Gynaecology 30 (January 2016): 79–86. http://dx.doi.org/10.1016/j.bpobgyn.2015.04.011.

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2

Gunaratne, Shehara Amanthi, Siromi Dilhara Panditharatne, and Edwin Chandraharan. "Prediction of Neonatal Acidosis Based on the Type of Fetal Hypoxia Observed on the Cardiotocograph (CTG)." European Journal of Medical and Health Sciences 4, no. 2 (2022): 8–18. http://dx.doi.org/10.24018/ejmed.2022.4.2.1308.

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Cardiotocograph (CTG) was introduced into clinical practice to promptly recognize the features of intrapartum fetal hypoxic stress, so that timely action could be taken to avoid hypoxic-ischaemic encephalopathy (HIE) and perinatal deaths. However, the current systematic evidence suggests that the introduction of CTG into clinical practice over 50 years has not resulted in improvement in the rates of cerebral palsy or perinatal deaths. This is because most fetuses are able to withstand intrapartum hypoxic stresses without sustaining damage, and if the features of fetal compensatory responses are erroneously considered as “pathological”, “Abnormal” or “Category III” CTG tracing, it would lead to an exponential increase in unnecessary operative interventions without any improvement in perinatal outcomes. Neonatal acidosis at birth, determined by the estimation of pH in the umbilical artery has been considered as a surrogate marker of poor perinatal outcome. This is because significant intrapartum fetal hypoxic stress which leads to fetal decompensation, would lead to the onset of anaerobic metabolism and production of lactic acid in fetal tissues and organs. Entry of lactic acid into the fetal systemic circulation may cause damage to fetal central organs resulting in organ damage and death, and this lactate may lower the pH in the umbilical artery. Understanding the different types of fetal hypoxia on the CTG trace may help practicing clinicians to predict the rate of fall in fetal pH, and therefore, predict the umbilical cord pH at birth. It is important to appreciate that non-hypoxic pathways of fetal compromise such as chorioamnionitis may not be associated with low umbilical arterial pH at birth. Fetal pathophysiological approach to CTG interpretation based on deeper understanding of types of intrapartum hypoxia and features of non-hypoxic pathways of injury may help avoid the onset of neonatal metabolic acidosis and improve perinatal outcomes.
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3

Maeda, Kazuo. "Intrapartum Hypoxic Damage is detected by Hypoxia Index to Prevent Cerebral Palsy." Obstetrics and Gynecology Research 01, no. 01 (2018): 9–18. http://dx.doi.org/10.26502/ogr.4560002.

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4

Manapova, R. M. "Assessment of Immune Status Indicators in Children with Consequences of Perinatal CNS Damage and Gastroduodenal Pathology." Effective Pharmacotherapy 16, no. 34 (2020): 6–8. http://dx.doi.org/10.33978/2307-3586-2020-16-34-6-8.

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Intrauterine fetal hypoxia, because of disturbances in the fetoplacental complex, is a pathogenic link in the mechanism of development of cerebral lesions in newborns. The immune system is a universal indicator of all pathological processes occurring in the fetus against the background of chronic placental insufficiency. Therefore, scientific studies devoted to the study of the immune status in children who have undergone intrapartum resuscitation at birth are of great importance
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5

Ramenghi, L. A., M. Fumagalli, L. Bassi, and F. Mosca. "Encefalopatia ipossico-ischemica perinatale." Rivista di Neuroradiologia 16, no. 3 (2003): 339–44. http://dx.doi.org/10.1177/197140090301600303.

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Hypoxic-ischaemic encephalopathy is one of the clinical expressions of hypoxic-is-chaemic damage in which concomitant hypoxia and ischaemia are responsible for the death of the foetus or neonate, or result in organ impairment with possible long-term sequelae. Neurological deficits are among the most common and severe sequelae, leading to hypoxic-ischaemic encephalopathy whose incidence is between 0.6 and 0.9 per thousand live births in Anglosaxon countries. The following four major and two minor criteria must be present to establish the link between intrapartum hypoxic-ischaemic injury and neurological deficit: metabolic acidosis on umbilical cord sampling, persistent Apgar score of < 5 after the fifth minute, neonatal neurological symptoms, multiorgan involvement, abnormal cardiotocographic tracing, passage of meconium. None of the six criteria alone is diagnostic: the more criteria are present, the more likely the presence of asphyxia. Early electroencephalographic recording may yield useful information for the diagnosis of hypoxic-ischaemic injury to determine the neurological outcome and the diagnosis of convulsions. Neuroradiological investigations will identify and define the extent of cerebral lesions, confirming intrapartum hypoxic-ischaemic damage suspected clinically and yielding additional prognostic information on the basis of the anatomical structures involved.
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6

Malla, D. S. "EPISIOTOMY : A CHALLENGING OBSTETRIC INTERVENTION." Journal of Nepal Medical Association 42, no. 145 (2003): 54–58. http://dx.doi.org/10.31729/jnma.791.

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ABSTRACTThe professional literatures on the development of widely practiced procedure, episiotomy through theyears from the first publication in 1742 are reviewed. It reveals the change in number of publication as wellas the contributors to the development of perception about episiotomy. So it consisted expression of opinionof doctors initially then the co-workers like nurses and researchers and clients or consumers themselvestoo. It concludes that episiotomies prevent anterior perineal tear but fails to accomplish other benefitstraditionally ascribed to pelvic floor damage and relaxation including its sequel and also protection ofnewboin from intracranial haemorrhage and intrapartum asphyxia. Episiotomy substantially increasematernal blood loss during delivery and risk of anal sphincter damage with their long term morbidity.There is an urgent need to restrict the use of episiotomy in vaginal delivery.Key Words: Episiotomy, Perineal tear, anal sphincter damage.
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7

Hornbuckle, Janet, and James G. Thornton. "The fetal circulatory response to chronic placental insufficiency and relation to pregnancy outcome." Fetal and Maternal Medicine Review 10, no. 3 (1998): 137–52. http://dx.doi.org/10.1017/s0965539598000321.

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Chronic fetal growth restriction is associated with both increased perinatal mortality and impaired neurodevelopment. Although it is a much more important cause of fetal neurological damage than intrapartum birth asphyxia, it is more difficult to treat, since the main intervention, timed delivery, carries its own risks. Since it is associated with a range of circulatory changes, which may also cause fetal damage, understanding these may improve management. In this article we review these fetal circulatory changes and assess their significance for predicting perinatal and long term neurodevelopmental outcome. We describe the Doppler assessment techniques, their clinical role in prediction of adverse outcome and the pathogenesis of brain injury in the preterm growth restricted fetus.
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8

Vannucci, Robert C. "Current and Potentially New Management Strategies for Perinatal Hypoxic-Ischemic Encephalopathy." Pediatrics 85, no. 6 (1990): 961–68. http://dx.doi.org/10.1542/peds.85.6.961.

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Given the current dilemma in the brain-oriented therapy of newborn infants sustaining cerebral hypoxia-ischemia, it is not surprising that management strategies vary widely among neonatal intensive care units.9 Thus, there is no uniform standard of care, and it remains for future research to uncover new and effective modes of therapy for the neurologically compromised infant. Prevention, or at least optimal management, of prepartum and intrapartum asphyxia remains the best available means of reducing the incidence and severity of peninatal hypoxic-ischemic brain damage.
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9

Kim, Kyung Won, and Sunhee Lee. "Childbirth outcomes and perineal damage in women with natural childbirth : a review of medical records in a natural birth center in Korea." Korean Journal of Women Health Nursing 27, no. 4 (2021): 379–87. http://dx.doi.org/10.4069/kjwhn.2021.08.31.

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Purpose:. This study aimed to determine the actual state of childbirth in women with natural childbirth and the degree of damage to the perineum during childbirth. Methods: This retrospective study analyzed the medical records of mothers who had natural childbirth at a hospital in Seoul, Korea in 2018. Data from 358 women with cephalic births at greater than 37 gestational weeks were analyzed. To determine natural childbirth characteristics and the degree of damage to the perineum, descriptive statistics were done using IBM SPSS Statistics version 28.0 for Windows. The difference in the degree of perineal injury according to obstetric characteristics was analyzed using independent t-test and one-way analysis of variance.Results: The mean age was 33.18±3.68 years, and 49.2% were primiparas, while 39% gave birth with a doula. The degree of perineal damage differed by age (F=9.15, p<.001), parity (t=19.13, p<.001), number of births in multiparity (F=3.68, p=.027), previous vaginal delivery in multiparity (F=3.00, p=.032) and birthing posture (F=7.44, p<.001). Having received therapeutic procedures (t=–4.62, p<.001), specifically fluid administration (t=–2.72, p=.007), oxygen supply (t=-–2.76, p=.006) and epidural anesthesia (t=–2.77, p=.006) were statistically significant for perineal damage. There were no differences, however, by gestational period, doula use, body mass index at delivery, baby head circumference, or birth weight. Conclusion: Study findings suggest that support for older women, primiparas, and those who require therapeutic procedures may help to decrease the possibility of perineal damage. As perineal damage was also associated with birthing posture, this should be considered when providing intrapartum nursing care.
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10

Önalan, Erhan, Şüheda Ataş, and Kübra Oral. "A case of Sheehan Syndrome with chronic diffuse muscle pain and weakness." Medical Science and Discovery 6, no. 11 (2019): 310–12. http://dx.doi.org/10.36472/msd.v6i11.315.

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Objective: Sheehan’s syndrome is pituitary deficiency induced by intrapartum and postpartum hemorrhage and hypovolemia. It is still frequent in underdeveloped and developing countries. Sheehan’s syndrome is one of the reason of empty sella. The symptoms of the syndrome can be seen months to years later depend on the degree of pituitary damage. History of postpartum hemorrhage, failure to lactate and cessation of menses are important clues to the diagnosis. Early diagnosis and appropriate treatment are very important to reduce morbidity and mortality of the patients.
 Case: In this study sheehan’s syndrome which led to auto pan-hypopituitarism and developed gradually in a patient with sheehan‘s syndrome which in this case, delivered a baby at home 27 years ago and had severe postpartum hemorrhage will be presented. And this 63-year-old female patient was diagnosed as Sjogren’s syndrome and sheehan syndrome by clinical and laboratory findings for the purpose of further investigation and treatment because of symptoms of fever and anemia
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