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Journal articles on the topic 'Intravascular activity'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Schmedes, Clare M., Steven P. Grover, Yohei M. Hisada, et al. "Circulating Extracellular Vesicle Tissue Factor Activity During Orthohantavirus Infection Is Associated With Intravascular Coagulation." Journal of Infectious Diseases 222, no. 8 (2019): 1392–99. http://dx.doi.org/10.1093/infdis/jiz597.

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Abstract Background Puumala orthohantavirus (PUUV) causes hemorrhagic fever with renal syndrome (HFRS). Patients with HFRS have an activated coagulation system with increased risk of disseminated intravascular coagulation (DIC) and venous thromboembolism (VTE). The aim of the study was to determine whether circulating extracellular vesicle tissue factor (EVTF) activity levels associates with DIC and VTE (grouped as intravascular coagulation) in HFRS patients. Methods Longitudinal samples were collected from 88 HFRS patients. Patients were stratified into groups of those with intravascular coag

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McDonald, Braedon, Rachelle Davis, and Craig N. Jenne. "Neutrophil extracellular traps (NETs) promote disseminated intravascular coagulation in sepsis." Journal of Immunology 196, no. 1_Supplement (2016): 60.8. http://dx.doi.org/10.4049/jimmunol.196.supp.60.8.

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Abstract Neutrophil extracellular traps (NETs; webs of DNA coated in anti-microbial proteins) are released into the vasculature during sepsis and contribute to organ damage. Various components of NETs have been shown to modulate the coagulation cascade in vitro. We hypothesized that NETs released into the vasculature during sepsis activate intravascular coagulation, leading to microvascular hypoperfusion and end-organ damage. Multi-color confocal intravital microscopy was used in mouse models of sepsis (endotoxemia and E. coli peritonitis) to visualize and quantify neutrophil and platelet traf

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Yusupova, A. N., and I. A. Andrushko. "Blood 5´-nucleotidase activity in gestosis complicated pregnancy." Kazan medical journal 68, no. 3 (1987): 202–5. http://dx.doi.org/10.17816/kazmj96060.

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The data on the role of disseminated intravascular coagulation in the occurrence of ischemic lesions in severe forms of this pathology are attracting increasing attention in the study of the pathogenesis of pregnant gestosis. V. P. Skipetrov suggests that coagulopathy developing in pregnant gestosis is the result of increased thromboplastic activity of the blood in response to damaging agents. Direct evidence has been obtained that severe forms of pregnancy gestosis are accompanied by a chronic form of disseminated intravascular coagulation syndrome (DIC). Thromboplastinemia acts as one of the

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4

TURNER, T. T., K. J. BROWN, and C. L. SPANN. "Testicular Intravascular Volume and Microvessel Mitotic Activity: Effect of Experimental Varicocele." Journal of Andrology 14, no. 3 (1993): 180–86. http://dx.doi.org/10.1002/j.1939-4640.1993.tb00377.x.

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ABSTRACT: Testicular blood flow increases bilaterally in animals with experimental left varicocele (ELV). The present experiments were undertaken to determine if increases in testicular intravascular volume and capillary growth were potential contributors to this increase in flow. Testicular intravascular volume (μl/g) was estimated by determining testicular vascular 125I‐albumin space. Microvessel mitotic activity was estimated by examining 3 H‐thymidine incorporation into endothelial nuclei, as assayed by silver grain localization over microvessel endothelial nuclei after exposure of testes

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5

Andrushko, I. A. "Thromboplastinemia — an initiator of continuous hemocoagulation in organism and disseminated interavascular coagulation syndrome." Kazan medical journal 75, no. 3 (1994): 185–87. http://dx.doi.org/10.17816/kazmj89831.

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The concept of thromboplastinemia as an initial factor of phisiologic and pathologic intravascular activation of hemostasis system is fully validated. It is stated that tissue thromboplastin comprises the fragments of cell membranes where 5'-nucleotidase is the marker enzyme. The increase of this enzyme activity is indirect evidence of the presence of tissue thromboplastin in vascular bed, that is bears witness to thromboplastinemia. Experimentally and in clinic the direct correlation is established between the activity increase of 5'-nucleotidase and the extent of hypercoagu- lemia, proving t

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6

Cécilia, Roustit, Vallé Baptiste, Clouzeau Benjamin, et al. "Acute Hemolysis in the Emergency Department: Think aboutClostridium perfringens!" Case Reports in Emergency Medicine 2013 (2013): 1–2. http://dx.doi.org/10.1155/2013/948071.

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Clostridium perfringens(CP) gives several clinical settings, from an asymptomatic to a massive intravascular hemolysis. We report a case of fatal intravascular hemolysis due to CP septicemia having a hepatic supposed starting point in the emergency department. Like in many cases, the diagnosis was made when patient had already gone into shock and died. The CP septicemia often complicated the course of the digestive or genital pathologies. The alpha toxin can damage the structural integrity of the red cell membrane by means of a phospholipase activity. Nevertheless, a massive intravascular hemo

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7

Wenner, Megan M., William C. Rose, Erin P. Delaney, Michael E. Stillabower, and William B. Farquhar. "Influence of plasma osmolality on baroreflex control of sympathetic activity." American Journal of Physiology-Heart and Circulatory Physiology 293, no. 4 (2007): H2313—H2319. http://dx.doi.org/10.1152/ajpheart.01383.2006.

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The purpose of this study was to determine if plasma osmolality alters baroreflex control of sympathetic activity when controlling for a change in intravascular volume; we hypothesized that baroreflex control of sympathetic activity would be greater during a hyperosmotic stimulus compared with an isoosmotic stimulus when intravascular volume expansion was matched. Seven healthy subjects (25 ± 2 yr) completed two intravenous infusions: a hypertonic saline infusion (HSI; 3% NaCl) and, on a separate occasion, an isotonic saline infusion (ISO; 0.9% NaCl), both at a rate of 0.15 ml·kg−1·min−1. To i

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8

Stief, Thomas W. "Coagulation Activation by Lipopolysaccharides." Clinical and Applied Thrombosis/Hemostasis 15, no. 2 (2007): 209–19. http://dx.doi.org/10.1177/1076029607309256.

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Lipopolysaccharides at approximate plasma reactivities >3 ng/mL or β-glucans at >0.5-1 μg/mL are toxic for human blood; lipopolysaccharide interacts with membrane components of susceptible cells (eg, monocytes) activating phospholipase A2 that destroys the cell membrane. Cell fragments (microparticles or DNA) possess polynegative niches that activate intrinsic hemostasis. Pathologic disseminated intravascular coagulation arises. Blood vessels are obstructed by disseminated thrombi, and vital organ areas become ischemic. Multiorgan failure threatens life of the patient. Diagnosis and ther

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9

Tseng, Kuo-Yao, Yu-Tsung Huang, Yu-Ting Huang, et al. "Regulation of candidalysin underlies Candida albicans persistence in intravascular catheters by modulating NETosis." PLOS Pathogens 20, no. 6 (2024): e1012319. http://dx.doi.org/10.1371/journal.ppat.1012319.

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Candida albicans is a leading cause of intravascular catheter-related infections. The capacity for biofilm formation has been proposed to contribute to the persistence of this fungal pathogen on catheter surfaces. While efforts have been devoted to identifying microbial factors that modulate C. albicans biofilm formation in vitro, our understanding of the host factors that may shape C. albicans persistence in intravascular catheters is lacking. Here, we used multiphoton microscopy to characterize biofilms in intravascular catheters removed from candidiasis patients. We demonstrated that, NETos

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Lasson, Å., and K. Ohlsson. "Disseminated Intravascular Coagulation and Antiprotease Activity in Acute Human Pancreatitis." Scandinavian Journal of Gastroenterology 21, sup126 (1986): 35–39. http://dx.doi.org/10.3109/00365528609091890.

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11

Song, Jae Woo, Jong Rak Choi, Kyung Soon Song, and Ji-Hyuk Rhee. "Plasma Factor XIII Activity in Patients with Disseminated Intravascular Coagulation." Yonsei Medical Journal 47, no. 2 (2006): 196. http://dx.doi.org/10.3349/ymj.2006.47.2.196.

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12

DİKMEN, Kürşat, Asiye UĞRAŞ DİKMEN, Hasan BOSTANCI, et al. "The effect of pneumoperitoneum on intravascular fibrinolytic activity in rats." TURKISH JOURNAL OF MEDICAL SCIENCES 46 (2016): 1528–33. http://dx.doi.org/10.3906/sag-1501-53.

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13

Karpanen, T. J., A. L. Casey, B. R. Conway, P. A. Lambert, and T. S. J. Elliott. "Antimicrobial activity of a chlorhexidine intravascular catheter site gel dressing." Journal of Antimicrobial Chemotherapy 66, no. 8 (2011): 1777–84. http://dx.doi.org/10.1093/jac/dkr191.

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14

Kario, Kazuomi, Takefumi Matsuo, Kazuya Kodama, Miyako Matsuo, Katsuichirou Yamamoto, and Hiroko Kobayashi. "Imbalance between Thrombin and Plasmin Activity in Disseminated Intravascular Coagulation." Pathophysiology of Haemostasis and Thrombosis 22, no. 4 (1992): 179–86. http://dx.doi.org/10.1159/000216317.

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15

Dover, Anna R., Patrick W. F. Hadoke, Linsay J. Macdonald, Eileen Miller, David E. Newby, and Brian R. Walker. "Intravascular Glucocorticoid Metabolism during Inflammation and Injury in Mice." Endocrinology 148, no. 1 (2007): 166–72. http://dx.doi.org/10.1210/en.2006-0996.

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11β-Hydroxysteroid dehydrogenases (11βHSDs) catalyze interconversion of 11-hydroxy-glucocorticoids with inactive 11-keto metabolites. In blood vessel walls, loss of 11βHSD1 is thought to reduce local glucocorticoid concentrations, reducing the progression of atheroma and enhancing angiogenesis. Conversely, on the basis that 11βHSD1 is up-regulated approximately 5-fold by inflammatory cytokines in cultured human vascular smooth muscle cells, it has been proposed that increased 11βHSD1 during vascular inflammation provides negative feedback suppression of inflammation. We aimed to determine whet

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Oyekan, A. O., and J. H. Botting. "The Influence of Adrenaline on Gender Difference in Adenosine Diphosphate-Induced Aggregation of Platelets in the Rat." Thrombosis and Haemostasis 60, no. 03 (1988): 481–85. http://dx.doi.org/10.1055/s-0038-1646995.

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SummaryThe role of adrenaline on the inhibitory effects of physiological levels of oestradiol on ADP-induced intravascular aggregation has been studied. Platelets from pro-oestrous female rats aggregated less than those from dioestrous and male rats. Following adrenalectomy, there was no longer any difference(s) in the aggregability of the platelets to ADP in any of the rats. Adrenaline infusion (20 mg kg−1 hr−1) restored platelet aggregation to preadrenalectomy levels in pro-oestrous rate. Measurement of spontaneous fibrinolytic activity of the plasma showed highest value in pro-oestrous rats

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17

Nečasová, Andrea, Lucie Urbanová, Laura Staňková, et al. "Protein C activity in dogs with gastric dilatation and volvulus." Acta Veterinaria Brno 91, no. 1 (2022): 35–42. http://dx.doi.org/10.2754/avb202291010035.

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Protein C activity, a novel prognostic marker, was evaluated in patients with gastric dilatation and volvulus in relation to mortality and severity of the disease. Data on the mortality, duration of clinical signs, degree of gastric torsion, gastric necrosis, splenectomy and occurrence of disseminated intravascular coagulopathy were recorded. Blood samples were obtained at selected time periods: T0 = no longer than 30 min before surgery; T1 = first day after surgery; T2 = second day after surgery. In addition to protein C activity assessment, haematological and haemostaseological analysis was

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18

Hill, M. A., M. J. Davis, and G. A. Meininger. "Cyclooxygenase inhibition potentiates myogenic activity in skeletal muscle arterioles." American Journal of Physiology-Heart and Circulatory Physiology 258, no. 1 (1990): H127—H133. http://dx.doi.org/10.1152/ajpheart.1990.258.1.h127.

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The proposition that arteriolar constriction to increased intravascular pressure is mediated through either the increased local production of an eicosanoid constrictor factor or decreased production of a dilating factor was examined. The myogenic response of arterioles was studied by enclosing anesthetized rats in an airtight Plexiglas box with the cremaster muscle exteriorized into a tissue bath containing Krebs solution. Microvascular responses were observed by video microscopy. The arteriolar response to a 20-mmHg increase in intravascular pressure was examined in the absence or presence of

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Taylor, F. B., S. E. He, A. C. K. Chang, et al. "Infusion of Phospholipid Vesicles Amplifies the Local Thrombotic Response to TNF and Anti-Protein C into a Consumptive Response." Thrombosis and Haemostasis 75, no. 04 (1996): 578–84. http://dx.doi.org/10.1055/s-0038-1650325.

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SummaryInflammation often is considered a contributing factor to both thrombosis and disseminated intravascular coagulation. The molecular mechanisms that dictate which of these clinical manifestations will result from the inflammatory stimulus remain obscure. Bacterial infection and certain tumors are common initiators of the disseminated intravascular coagulant response. Complement activation resulting from bacterial infection shares with selected tumors the capacity to generate or release membrane particles that lack functional adhesion receptors and hence could circulate to amplify a disse

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Kiperman, Ya V., V. V. Zaitsev, and O. N. Makurina. "THE PHYSIOLOGICAL IMPORTANCE OF EVALUATING INTRAVASCULAR PLATELET ACTIVITY IN BIOLOGICAL OBJECTS." On-line Scientific & Educational Bulletin “Health & education millennium” 19, no. 2 (2017): 9–13. http://dx.doi.org/10.26787/nydha-2226-7417-2017-19-2-9-13.

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21

Tu, Juan, Joo Ha Hwang, Thomas J. Matula, Andrew A. Brayman, and Lawrence A. Crum. "Intravascular inertial cavitation activity detection and quantification in vivo with Optison." Ultrasound in Medicine & Biology 32, no. 10 (2006): 1601–9. http://dx.doi.org/10.1016/j.ultrasmedbio.2006.07.015.

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22

KOBAYASHI, Kosuke, Kenji BABA, Masaya IGASE, Takako Shimokawa MIYAMA, Satoshi KAMBAYASHI, and Masaru OKUDA. "Microparticle-associated tissue factor activity in dogs with disseminated intravascular coagulation." Journal of Veterinary Medical Science 82, no. 1 (2020): 56–60. http://dx.doi.org/10.1292/jvms.19-0553.

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23

Chinen, Yoshiaki, Junya Kuroda, Muneo Ohshiro, et al. "Low ADAMTS-13 activity during hemorrhagic events with disseminated intravascular coagulation." International Journal of Hematology 97, no. 4 (2013): 511–19. http://dx.doi.org/10.1007/s12185-013-1308-x.

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24

Parvez, Z., and H. Vik. "Intravascular Contrast Media and Thrombin Generation." Acta Radiologica 35, no. 2 (1994): 172–75. http://dx.doi.org/10.1177/028418519403500215.

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This study focuses on the effect of contrast media (CM) on thrombin generation. In vitro studies consisted of incubating nonanticoagulated whole blood with ionic CM (sodium meglumine diatrizoate, ioxaglate), nonionic CM (iohexol, iopamidol) or glucose in plastic tubes. Thrombin generation was assessed by measuring F1 + 2, ATM and FpA levels in plasma using ELISA assay kits. In a separate protocol, the procoagulant activity of 3 nonionic CM (iohexol, iopamidol, and iopromide) was investigated by one-stage plasma recalcification time method. Rabbit brain tissue thromboplastin and physiologic sal

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Dasgupta, Trisha, Mary E. Fabry, and Dhananjay K. Kaul. "Antisickling property of fetal hemoglobin enhances nitric oxide bioavailability and ameliorates organ oxidative stress in transgenic-knockout sickle mice." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 298, no. 2 (2010): R394—R402. http://dx.doi.org/10.1152/ajpregu.00611.2009.

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In sickle cell disease (SCD), the events originating from hemoglobin S polymerization and intravascular sickling lead to reperfusion injury, hemolysis, decreased nitric oxide (NO) bioavailability, and oxidative stress. Oxidative stress is implicated as a contributing factor to multiple organ damage in SCD. We hypothesize that inhibition of sickling by genetic manipulation to enhance antisickling fetal hemoglobin (HbF) expression will have an ameliorating effect on oxidative stress by decreasing intravascular sickling and hemolysis and enhancing NO bioavailability. We tested this hypothesis in

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West, Lindsey, Ramin Fathi, Rajiv Nijhawan, and Divya Srivastava. "Metastatic basal cell carcinoma with evidence of intravascular invasion: A case report." SAGE Open Medical Case Reports 8 (January 2020): 2050313X1984778. http://dx.doi.org/10.1177/2050313x19847782.

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Although basal cell carcinoma is the most common malignancy in humans, intravascular basal cell carcinoma is rarely reported in the literature. A 78-year-old male presented for Mohs Micrographic surgery of a 1.5 by 1.5 cm2 nodular and focally infiltrative basal cell carcinoma on the left parietal scalp. In the fifth stage, intravascular tumor cells were noted, and the patient was still positive in five out of six specimens with a defect measuring 6.5 × 7.0 cm2. Mohs surgery was stopped due to concern of widespread disease. A positron emission tomography/computed tomography scan was ordered tha

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Barrowcliffe, M. P., C. Otto, and J. G. Jones. "Pulmonary clearance of 99mTc-DTPA: influence of background activity." Journal of Applied Physiology 64, no. 3 (1988): 1045–49. http://dx.doi.org/10.1152/jappl.1988.64.3.1045.

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We examined the effect of intravascular and tissue accumulation of tracer when measuring pulmonary clearance of sodium pertechnetate-labeled diethylenetriamine pentaacetate (99mTc-DTPA). Pigs were intubated with endobronchial tubes, permitting deposition of an aerosol of 99mTc-DTPA only into the left lung. Scintillation detectors recorded radioactivity separately from one thigh and from the lung and chest wall on the left and right side. 99mTc-DTPA was given intravenously after 30 min, so that the chest counts from the left lung could be corrected for background activity in either the right lu

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I., A. Skoryatina, and Yu Zavalishina S. "Impact of Experimental Development of Arterial Hypertension and Dyslipidemia on Intravascular Activity of Rats' Platelets." Annual Research & Review in Biology 14, no. 5 (2017): 1–9. https://doi.org/10.9734/ARRB/2017/33758.

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Great interest is shown by researchers to functional and rheological features of basic regular blood elements. Platelets are among them and take the most active part in hemostasis at rather wide-spread nowadays cardio-vascular and metabolic diseases. The aim is to analyze dynamics’ strengthening of platelets' intravascular activity of rats in conditions of experimental consequent development of arterial hypertension and dyslipidemia. The study used 68 male-rats of Vistar line at the age of 2.5-3 months. Control group was composed of 33 animals. Experimental animals (35 rats) were developed at

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Dennis, Michael, Colin Downey, Nicole Brufatto, Michael Nesheim, Ken Stevenson, and Cheng Toh. "Prothrombinase enhancement through quantitative and qualitative changes affecting very low density lipoprotein in complex with C-reactive protein." Thrombosis and Haemostasis 91, no. 03 (2004): 522–30. http://dx.doi.org/10.1160/th03-08-0548.

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SummaryThe biphasic waveform that can predict for disseminated intravascular coagulation (DIC) is due to the formation of a calcium-dependent complex between C reactive protein (CRP) and very low density lipoprotein (VLDL). As thrombin generation is pivotal to DIC, this aspect has been specifically investigated and the VLDL component has been found to increase prothrombinase activity via both quantitative and qualitative changes. The specific prothrombinase activity of VLDL from patients manifesting the biphasic waveform was 2.5 times that of normal individuals or critically ill patients witho

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Shah, Chirag B., Marc W. Mittelman, J. W. Costerton, et al. "Antimicrobial Activity of a Novel Catheter Lock Solution." Antimicrobial Agents and Chemotherapy 46, no. 6 (2002): 1674–79. http://dx.doi.org/10.1128/aac.46.6.1674-1679.2002.

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ABSTRACT Intravascular catheter-associated bloodstream infections significantly increase rates of morbidity and hospital costs. Microbial colonization and development of biofilms, which are known to be recalcitrant to antibiotic therapy, often lead to the loss of otherwise patent vascular access systems. We evaluated a new taurolidine- and citrate-based catheter lock solution (Neutrolin; Biolink Corporation, Norwell, Mass.) for its activity against planktonic microbes, antimicrobial activity in a catheter model, and biofilm eradication activity. In studies of planktonic microbes, after 24 h of

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Oliievska, S. K., O. V. Kuzminska, and T. I. Trunina. "The changes in the blood agregative system under the influence of lead chloride on the example of albino rats." Likarska sprava, no. 5-6 (March 13, 2019): 122–26. http://dx.doi.org/10.31640/jvd.5-6.2018(21).

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Experimentally established, that lead chloride affects all link systems of hemostasis, causing latent passing of disseminated intravascular coagulation. Observed significant depression of enzymatic, fibrinolytic activity of the cortical substance of the kidney, which indicatesthe prevalence ofcoagulopathic and nephrotoxic effects of small doses of plumbum chloride.

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Kindt, G. C., J. E. Gadek, and J. E. Weiland. "Initial recruitment of neutrophils to alveolar structures in acute lung injury." Journal of Applied Physiology 70, no. 4 (1991): 1575–85. http://dx.doi.org/10.1152/jappl.1991.70.4.1575.

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The adult respiratory distress syndrome and bacterial pneumonia are both characterized by an influx of neutrophils into the lung. The neutrophil has been implicated as having a “pathological” role in adult respiratory distress syndrome, in contrast to its role in bacterial pneumonia. We hypothesized that processes resulting in neutrophil recruitment to the lung are distinct, depending on whether the inflammatory stimulus arises in the intravascular or the alveolar compartment of the lung. Anesthetized sheep with lung lymph fistulas were utilized to access the three compartments of the lung rel

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Vinazzer, H. A. "Antithrombin III in shock and disseminated intravascular coagulation." Kazan medical journal 79, no. 1 (1998): 44–48. http://dx.doi.org/10.17816/kazmj63495.

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Antithrombin III (AT III) is the physiological inhibitor of a series of activated clotting factors. Its diminution increases the tendency of thromboembolism. In cases of acute DIC, AT III binds to thrombin and to other activated factors in circulation. Thereby both partners of the reaction become inactive. When by this mechanism the available biologic activity of AT III diminishes below about 50% of normal any further inactivation of active clotting enzymes is greatly inhibited. In such cases, the mechanism of DIC decompensates and the full symptoms of consumption coagulopathy develop. It was,

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Aras, Omer, Arun Shet, Ronald R. Bach, et al. "Induction of microparticle- and cell-associated intravascular tissue factor in human endotoxemia." Blood 103, no. 12 (2004): 4545–53. http://dx.doi.org/10.1182/blood-2003-03-0713.

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Abstract The precise role of intravascular tissue factor (TF) remains poorly defined, due to the limited availability of assays capable of measuring circulating TF procoagulant activity (PCA). As a model of inflammation-associated intravascular thrombin generation, we studied 18 volunteers receiving an infusion of endotoxin. A novel assay that measures microparticle (MP)-associated TF PCA from a number of cellular sources (but not platelets) demonstrated an 8-fold increase in activity at 3 to 4 hours after endotoxin administration (P &lt; .001), with a return to baseline by 8 hours. TF ant

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Guarini, Anna, Luigi Gugliotta, Claudia Timoncini, et al. "Procoagulant cellular activity and disseminated intravascular coagulation in acute non-lymphoid leukaemia." Scandinavian Journal of Haematology 34, no. 2 (2009): 152–56. http://dx.doi.org/10.1111/j.1600-0609.1985.tb02248.x.

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Bernstain U. Chitkara, H. H., H. Plosker, M. Gettes, and R. L. Berkowitz. "Use of Atracurium Besylate to Arrest Fetal Activity During Intrauterine Intravascular Transfusion." Obstetric Anesthesia Digest 9, no. 2 (1989): 108. http://dx.doi.org/10.1097/00132582-198907000-00059.

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Haynes, Gary, John Lazarchick, Yuko Palesch, Susan Harvey, and Prabhakar Baliga. "ANTITHROMBIN-III ACTIVITY ASSOCIATED WITH DISSEMINATED INTRAVASCULAR COAGULATION IN ORTHOTOPIC LIVER TRANSPLANTATION." Anesthesiology 89, Supplement (1998): 412A. http://dx.doi.org/10.1097/00000542-199809080-00036.

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Andoh, Kiyoshi, Tatsumi Kubota, Masashi Takada, Hiroshi Tanaka, Norio Kobavashi, and Tadashi Maekawa. "Tissue factor activity in leukemia cells. Special reference to disseminated intravascular coagulation." Cancer 59, no. 4 (1987): 748–54. http://dx.doi.org/10.1002/1097-0142(19870215)59:4<748::aid-cncr2820590414>3.0.co;2-e.

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39

Bobyleva, T. A., and S. B. Тkachenko. "Intravascular platelet activity in patients with atrial fibrillation while taking direct anticoagulants." Clinical Medicine (Russian Journal) 102, no. 11-12 (2025): 862–67. https://doi.org/10.30629/0023-2149-2024-102-11-12-862-867.

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The article presents an analysis of changes in the platelet component of the hemostatic system in patients with paroxysmal and persistent forms of atrial ﬁbrillation while taking direct oral anticoagulants. This group of medications is used for the prevention of ischemic stroke and other systemic thromboembolic events. The number of platelets involved in aggregates was within normal limits; however, there is a tendency toward an increase in active forms of platelets in these patients.

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Uzlova, Svetlana G., Konstantin G. Guria, and Georgy Th Guria. "Acoustic determination of early stages of intravascular blood coagulation." Philosophical Transactions of the Royal Society A: Mathematical, Physical and Engineering Sciences 366, no. 1880 (2008): 3649–61. http://dx.doi.org/10.1098/rsta.2008.0109.

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The blood coagulation system (BCS) is a complex biological system playing a principal role in the maintenance of haemostasis. Insufficient activity of the BCS may lead to bleeding and blood loss (e.g. in the case of haemophilia). On the other hand, excessive activity may cause intravascular blood coagulation, thromboses and embolization. Most of the methods currently used for BCS monitoring suffer from the major disadvantage of being invasive. The purpose of the present work is to demonstrate the feasibility of using ultrasonic methods for non-invasive registration of the early stages of blood

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Friedl, Josef, Markus Puhlmann, David L. Bartlett, et al. "Induction of permeability across endothelial cell monolayers by tumor necrosis factor (TNF) occurs via a tissue factor–dependent mechanism: relationship between the procoagulant and permeability effects of TNF." Blood 100, no. 4 (2002): 1334–39. http://dx.doi.org/10.1182/blood.v100.4.1334.h81602001334_1334_1339.

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Tumor necrosis factor (TNF) has marked effects on permeability and procoagulant activity on tumor-associated neovasculature when used in isolation perfusion, the latter effect primarily mediated via induction of cell surface expression of tissue factor (TF) on endothelial tissue. However, the cellular events that result in rapid alterations in endothelial cell (EC) permeability after intravascular TNF administration in isolation perfusion are not well characterized. We demonstrate that short exposure intervals to TNF induces TF expression on ECs but has no effect on permeability as assessed by

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Hisada, Yohei, and Nigel Mackman. "Tissue Factor and Extracellular Vesicles: Activation of Coagulation and Impact on Survival in Cancer." Cancers 13, no. 15 (2021): 3839. http://dx.doi.org/10.3390/cancers13153839.

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Tissue factor (TF) is a transmembrane glycoprotein that functions as a receptor for FVII/FVIIa and initiates the extrinsic coagulation pathway. Tumors and cancer cells express TF that can be released in the form of TF positive (TF+) extracellular vesicles (EVs). In this review, we summarize the studies of tumor TF and TF + EVs, and their association with activation of coagulation and survival in cancer patients. We also summarize the role of tumor-derived TF + EVs in venous thrombosis in mouse models. Levels of tumor TF and TF + EVs are associated with venous thromboembolism in pancreatic canc

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Galeeva, N. V., and I. Kh Valeeva. "Activity an enzyme 5`-nucleotidase in patients with chronic hepatitis C." Terapevticheskii arkhiv 91, no. 11 (2019): 45–48. http://dx.doi.org/10.26442/00403660.2019.11.000244.

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Aim of the study. To determine the activity of enzyme 5`-nucleotidase depending on the of disseminated intravascular coagulation syndrome phases and duration of infection period in patients with chronic hepatitis C. Materials and methods. 166 patients (92 men, 74 women) with chronic hepatitis C were under observation. Conditionally, all patients were devided into 3 groups, based on the number of Tr, according to the phases of hemostasis: I group - hypercoagulation with number Tr>350×109/l; II - transient phase - Tr from 350 to 200×109/l and III - hypocoagulation - Tr

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Barinov, Sergey Vladimirovich, Vladimir Terent'evich Dolgikh, and Irina Vladimirovna Medyannikova. "Integrated assessment haemostasis pregnant women with gestosis." Journal of obstetrics and women's diseases 62, no. 4 (2013): 15–21. http://dx.doi.org/10.17816/jowd62415-21.

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The parameters of the kaolinaktivirovannoy thrombelastographic at physiological gestation, moderate, and severe gestosis. The significant differences between groups for the intensification of intravascular coagulation and fibrinolysis activity. Option and the severity of the changes thrombelastographic determine the need for correction hemocoagulation violations during gestation thrombohemorrhagic to prevent complications in pregnancy.

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Barinov, Sergey Vladimirovich, Vladimir Timofeevich Dolgikh, and Irina Vladimirovna Medyannikova. "Integrated assessment haemostasis pregnant women with gestosis." Journal of obstetrics and women's diseases 62, no. 6 (2013): 5–11. http://dx.doi.org/10.17816/jowd6265-11.

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The parameters of the kaolinaktivirovannoy thrombelastographic at physiological gestation, moderate, and severe gestosis. The significant differences between groups for the intensification of intravascular coagulation and fibrinolysis activity. Option and the severity of the changes thrombelastographic determine the need for correction hemocoagulation violations during gestation thrombohemorrhagic to prevent complications in pregnancy.

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46

Kumar, Arun, Subrata Kar, and William P. Fay. "Thrombosis, physical activity, and acute coronary syndromes." Journal of Applied Physiology 111, no. 2 (2011): 599–605. http://dx.doi.org/10.1152/japplphysiol.00017.2011.

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Acute coronary syndromes (ACS) are common, life-threatening cardiac disorders that typically are triggered by rupture or erosion of an atherosclerotic plaque. Platelet deposition and activation of the blood coagulation cascade in response to plaque disruption lead to the formation of a platelet-fibrin thrombus, which can grow rapidly, obstruct coronary blood flow, and cause myocardial ischemia and/or infarction. Several clinical studies have examined the relationship between physical activity and ACS, and numerous preclinical and clinical studies have examined specific effects of sustained phy

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Elzoheiry, Manal, Akram Da'dara, Armelle deLaforcade, Samar El-Beshbishi, and Patrick Skelly. "The Essential Ectoenzyme SmNPP5 from the Human Intravascular Parasite Schistosoma mansoni is an ADPase and a Potent Inhibitor of Platelet Aggregation." Thrombosis and Haemostasis 118, no. 06 (2018): 979–89. http://dx.doi.org/10.1055/s-0038-1641715.

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AbstractSchistosomes are intravascular parasitic platyhelminthes infecting > 200 million people globally and causing a debilitating disease, schistosomiasis. Despite the relatively large size of the adult worms and their disruption of blood flow, surprisingly, they do not appear to provoke thrombus formation around them in vivo. We hypothesize that proteins expressed at the host–parasite interface are key to this ability. Here, we functionally express an ectonucleotide pyrophosphatase/phosphodiesterase homologue, SmNPP5, that is expressed at the tegumental surface of intravascular Schistoso

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Chen, Ming-Feng, Chung-Chieng Wu, Shiang-Bin Jong, and Chun-Ching Lin. "Differences in Acupuncture Point SP-10 and Non-acupuncture Point Following Subcutaneous Injection of Tc-99m Pertechnetate." American Journal of Chinese Medicine 21, no. 03n04 (1993): 221–29. http://dx.doi.org/10.1142/s0192415x9300025x.

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A new method of radionuclide venography, namely SC-RNV, by subcutaneous injection of Tc-99m pertechnetate at acupuncture points was recently developed in our laboratory .The time-activity curve of SC-RNV can be divided into 3 phases as: the phase of diffusion of radioisotopes through the subcutaneous space, the phase of entrance of radioisotopes into intravascular space to reach a peak concentration and the phase of equilibrium state of radioisotopes in the intravascular space. In this study, we used the 3 phases of SC-RNV to evaluate the absorption of Tc-99m pertechnetate injected subcutaneou

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Chandler, Wayne Leslie. "Tissue Factor Activity and Antigen Levels during Cardiopulmonary Bypass and Disseminated Intravascular Coagulation." Blood 108, no. 11 (2006): 3998. http://dx.doi.org/10.1182/blood.v108.11.3998.3998.

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Abstract Background: in healthy subjects small amounts of tissue factor antigen but little if any active tissue factor can be detected circulating in blood. An increased level of circulating active tissue factor has been proposed as a possible mechanism of systemic coagulation activation. Methods: tissue factor antigen and activity levels were measured in healthy controls (n=20) and in two patient groups associated with systemic coagulation activation: open heart surgery utilizing cardiopulmonary bypass (CPB, n=4) and disseminated intravascular coagulation (DIC, n=15). In heart surgery patient

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Tollefsen, DM, and CA Pestka. "Heparin cofactor II activity in patients with disseminated intravascular coagulation and hepatic failure." Blood 66, no. 4 (1985): 769–74. http://dx.doi.org/10.1182/blood.v66.4.769.bloodjournal664769.

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Heparin cofactor II (HCII) is a glycoprotein in human plasma which inactivates thrombin rapidly in the presence of heparin or dermatan sulfate. We have developed a functional assay for HCII in which inhibition of thrombin by plasma is determined in the presence of dermatan sulfate. The assay is specific for HCII by the following criteria: (a) under the conditions of the assay, 125I-thrombin forms complexes in plasma which comigrate with the thrombin-HCII complex during sodium dodecyl sulfate polyacrylamide gel electrophoresis (SDS- PAGE); (b) activity detected by the assay is decreased in plas

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