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Relevant bibliographies by topics / Intrinsic Sleep Disorders

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Academic literature on the topic 'Intrinsic Sleep Disorders'

Author: Grafiati

Published: 4 June 2021

Last updated: 24 January 2023

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Journal articles on the topic "Intrinsic Sleep Disorders"

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Venn, P. J. H. "Intrinsic sleep disorders and anaesthesia." Current Anaesthesia & Critical Care 13, no. 1 (February 2002): 6–15. http://dx.doi.org/10.1054/cacc.2002.0376.

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Monk, T. "Intrinsic circadian rhythm sleep disorders." Sleep Medicine Reviews 3, no. 3 (September 1999): 177–78. http://dx.doi.org/10.1016/s1087-0792(99)90000-x.

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Richert, Allen C., and Alp Sinan Baran. "A Review of Common Sleep Disorders." CNS Spectrums 8, no. 2 (February 2003): 102–9. http://dx.doi.org/10.1017/s1092852900018320.

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AbstractWhat should clinicians know about sleep disorders? This article briefly introduces the reader to sleep medicine and succinctly reviews common sleep disorders. First, the authors describe the diagnostic tools unique to sleep medicine: the over-night polysomnogram and the multiple sleep latency test. Next, the authors review essential features of a subset of the sleep, described in the International Classification of Sleep Disorders-Revised, that sleep disorder specialists commonly evaluate, diagnose, and treat. The disorders reviewed include the intrinsic and circadian rhythm subsets of the dyssomnias group and the parasomnia group of sleep disorders. The authors identify the core signs and symptoms, polysomnogram findings, multiple sleep latency test findings, and treatment of these disorders.

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Mainieri, Greta, Giuseppe Loddo, and Federica Provini. "Disorders of Arousal: A Chronobiological Perspective." Clocks & Sleep 3, no. 1 (January 21, 2021): 53–65. http://dx.doi.org/10.3390/clockssleep3010004.

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Non-rapid eye movement (NREM) sleep parasomnias are characterized by motor and emotional behaviors emerging from incomplete arousals from NREM sleep and they are currently referred to as disorders of arousal (DoA). Three main clinical entities are recognized, namely confusional arousal, sleep terror and sleepwalking. DoA are largely present in pediatric populations, an age in which they are considered as transitory, unhabitual physiological events. The literature background in the last twenty years has extensively shown that DoA can persist in adulthood in predisposed individuals or even appear de novo in some cases. Even though some episodes may arise from stage 2 of sleep, most DoA occur during slow wave sleep (SWS), and particularly during the first two sleep cycles. The reasons for this timing are linked to the intrinsic structure of SWS and with the possible influence on this sleep phase of predisposing, priming and precipitating factors for DoA episodes. The objective of this paper is to review the intrinsic sleep-related features and chronobiological aspects affecting SWS, responsible for the occurrence of the majority of DoA episodes during the first part of the night.

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Campbell, S. "Etiology and treatment of intrinsic circadian rhythm sleep disorders." Sleep Medicine Reviews 3, no. 3 (September 1999): 179–200. http://dx.doi.org/10.1016/s1087-0792(99)90001-1.

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Kaplan, Robert. "Obstructive Sleep Apnoea and Depression — Diagnostic and Treatment Implications." Australian & New Zealand Journal of Psychiatry 26, no. 4 (December 1992): 586–91. http://dx.doi.org/10.3109/00048679209072093.

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Sleep apnoea (OSA), a common sleep disorder, is well recognised as a cause of morbidity including psychiatric disorders. There is increasing recognition of the link between OSA and depression. Sleep changes are intrinsic to depressive disorders, most notably disturbances of REM sleep; OSA causes predominantly REM sleep disturbances. The neuro-vegetative features of depression are similar or identical to the symptoms of OSA — an issue which has not achieved wide clinical recognition. A growing number of studies confirm the statistical link between the two conditions. The implications are twofold: OSA needs to be excluded in cases of chronic or resistant depression and treatment of OSA will make it easier to treat the primary depressive disorder. A new method of treatment for OSA, the Sullivan continuous positive airway pump (CPAP), raises the theoretical possibility of treating depression by this means as well.

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Wijnen, Herman, Catharine Boothroyd, Michael W. Young, and Adam Claridge-Chang. "Molecular genetics of timing in intrinsic circadian rhythm sleep disorders." Annals of Medicine 34, no. 5 (January 2002): 386–93. http://dx.doi.org/10.1080/078538902320772133.

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Blum, Nathan J., and William B. Carey. "Sleep Problems Among Infants and Young Children." Pediatrics In Review 17, no. 3 (March 1, 1996): 87–92. http://dx.doi.org/10.1542/pir.17.3.87.

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Parental concerns about their young children's sleep are among the most frequent behavior problems discussed with pediatricians. In view of this high prevalence, it is important to review the origins of these problems and emerging information about how best to manage them. Definition Sleep disorders among children can be classified as dyssomnias, parasomnias, and disruptions secondary to other conditions. The dyssomnias are disturbances in the amount or timing of the sleep. They include intrinsic sleep disorders such as narcolepsy and sleep apnea, interactional sleep disorders such as excessive night waking, and sleep phase disorders, when the time the parents assign for sleep and the child's period of needing it are not synchronous. Parasomnias, on the other hand, are abnormal behaviors that occur during sleep, such as night terrors, nightmares, sleep walking, and sleep talking. Sleep also can be disturbed as the result of various mental and physical conditions, including asthma, epilepsy, and anxiety disorders. The dyssomnias (sleep refusal and night waking) and parasomnias (night terrors and nightmares) of infancy and early childhood are the most common complaints of parents to pediatricians. Sometimes, however, parents may not seek help for these problems; at other times, they may be concerned by what actually is normal sleep behavior.

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Bruni, O., P. Fabrizi, S. Ottaviano, F. Cortesi, F. Giannotti, and V. Guidetti. "Prevalence of Sleep Disorders in Childhood and Adolescence with Headache." Cephalalgia 17, no. 4 (June 1997): 492–98. http://dx.doi.org/10.1046/j.1468-2982.1997.1704492.x.

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Although a relationship between headache and sleep disturbances has been reported in adults, only few data have been available in children. Accordingly, we performed a survey to determine the prevalence of sleep disturbances in children with migraine and tension-type headache. A questionnaire of history and clinical data and of sleep disturbances was given to parents of 283 headache subjects (164 with migraine and 119 with tension-type headache). Results were compared to a normative group comparable for age and sex of 893 normal healthy subjects. Migraine subjects showed a higher prevalence of sleep disturbances during infancy as well as 3-month colic. In both headache groups, more parents had sleep disturbances and there was a higher occurrence of co-sleeping and napping. A high frequency of sleep disturbances involving sleep quality, night awakening, nocturnal symptoms and daytime sleepiness was reported in headache children. No statistical differences were found in the prevalence of sleep disturbances between migraine and tension-type headache. However, the migraine group tended to have “disturbed sleep” more often with increased prevalence of nocturnal symptoms such as sleep breathing disorders and parasomnias. Our results give further support to an association between sleep and migraine that may have a common intrinsic origin.

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Emanuel, Hina, Amee Revana, Tue Te, and Kevin Kaplan. "830 Combined Phototherapy and Melatonin for treatment of Circadian Rhythm Disorder in a Patient with Cornelia de Lange Syndrome." Sleep 44, Supplement_2 (May 1, 2021): A323—A324. http://dx.doi.org/10.1093/sleep/zsab072.827.

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Abstract Introduction Cornelia de Lange syndrome (CdLS) is a rare genetic disorder characterized by variable physical, cognitive, and behavioral characteristics. Sleep disturbances have been frequently reported in CdLS including insomnia, sleep-disordered breathing, intrinsic sleep disorders, and circadian rhythm disorders (CRDs). The characterization and prevalence of CRDs in CdLS remain ill- defined. We report a case of a 13-year-old female with CdLS presenting with advanced sleep wake phase disorder (ASWPD). Report of case(s) A 13-year-old female with a past medical history of CdLS, developmental delay, bilateral cleft palate status post repair presents with inability to fall asleep at night and excessive daytime sleepiness.(EDS) Her sleep history consists of going to bed at 4 pm with no delayed sleep onset. She wakes at 2:30 am which has occurred since infancy. Mother reports the patient will remain awake from 2:30 am until she goes to school at 7:30 am. History is consistent with EDS and sleeping during the day while at school. Total sleep time of approximately 11–12 hours was reported in 24-hour period. History of obstructive sleep apnea, parasomnias, insomnia, restless leg syndrome, and psychotropic medications were not reported. Patient was treated with timed low dose melatonin therapy 0.5 mg at 4 pm and bright light therapy using 10,000 lux for 30 minutes at 7 am and 4 pm. Dim lights starting at 7:30 pm with structured scheduled sleep hygiene ensuring consistent bedtime at 9:30 pm. A consistent wake time at 7 am and no naps during the day was recommended. Follow up visits report successful response to therapy with attainment of desired sleep wake rhythm (bedtime at 9:30 pm and wake time at 7 am) and resolution of sleepiness during the day. Our patient was able to be weaned off of melatonin and light therapy and her circadian rhythm remained entrained. Conclusion Patients with disorder such as CdLS are at risk for circadian rhythm disorders. Our patient responded well to treatment with combined timed phototherapy and low dose melatonin therapy. Better knowledge and characterization of typology of CRDs in CdLS patients could permit a more specific therapeutic approach to sleep disorders in this population. Support (if any) None

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Books on the topic "Intrinsic Sleep Disorders"

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Horvath, Tamas, Susan Redline, and Priyattam Shiromani. Sleep Loss and Obesity: Intersecting Epidemics. Springer, 2012.

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Horvath, Tamas, Susan Redline, Priyattam Shiromani, and Eve Van Cauter. Sleep Loss and Obesity: Intersecting Epidemics. Springer, 2012.

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Horvath, Tamas, Susan Redline, Priyattam Shiromani, and Eve Van Cauter. Sleep Loss and Obesity: Intersecting Epidemics. Springer London, Limited, 2012.

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Horvath, Tamas, Susan Redline, Priyattam Shiromani, and Eve Van Cauter. Sleep Loss and Obesity: Intersecting Epidemics. Springer, 2016.

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Jones, Christopher R. Neurobiology of Circadian Rhythms Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0175.

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Temporal organization of nervous system function includes daily rhythms driven by a molecular-genetic hypothalamic “clock” with an intrinsic period length of approximately (circa) one day (diem). The resulting circadian rhythm influences all aspects of brain function and internally synchronizes the circadian oscillations inherent in all other body tissues. Idiosyncratic circadian characteristics interact with perceived environmental stimuli to determine each individual’s entrainment pattern of external synchronization with the day-night cycle. Idiosyncratic entrainment patterns that may come to medical attention include delayed, free-running, advanced, or absent sleep rhythms. Prolonged jet travel and shift work are difficult entrainment challenges for most people.

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Book chapters on the topic "Intrinsic Sleep Disorders"

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Sampat, Ajay, and Armand Ryden. "Circadian Rhythm Disorders." In Sleep Medicine and the Evolution of Contemporary Sleep Pharmacotherapy [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99816.

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Circadian rhythm disorders are a group of sleep conditions that involve a misalignment of an individual’s internal timekeeping system with that of one’s desired sleep-wake time. This desynchrony can compromise sleep health as well as the functioning of other organ system, and significantly diminish one’s quality of life. There are six well-defined circadian rhythm disorders that can be classified as either intrinsic or extrinsic, based on the underlying factors that contribute to the condition. Intrinsic circadian disorders include the following: 1) advanced sleep-wake phase disorder, 2) delayed sleep-wake phase disorder, 3) irregular sleep-wake rhythm disorder, and 4) non-24-hour sleep-wake rhythm disorder. The two circadian disorders caused by external factors include 1) shift work disorder, and 2) jet lag disorder, both of which are due to behaviorally mediated misalignments of circadian system. This chapter serves to summarize these disorders, guide clinicians towards screening and evaluation of these conditions, and introduce basic treatment strategies that can be applied by non-sleep medicine clinicians.

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Rahman, Shadab A., and Elizabeth B. Klerman. "Circadian Sleep Disorders." In Integrative Sleep Medicine, edited by Valerie Cacho and Esther Lum, 529–48. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190885403.003.0032.

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This chapter discusses circadian rhythm sleep wake disorders (CRSWDs) as defined by the International Classification of Sleep Disorders (3rd ed.), including the etiology, clinical characteristics, diagnostic methods, and treatment options for both “intrinsic” (delayed and advanced sleep–wake phase disorders, non–24-hour sleep–wake rhythm disorder, and irregular sleep–wake rhythm disorder) and “extrinsic” (shift work and jet lag disorders) types of CRSWDs. Cognitive and psychiatric comorbidities commonly associated with these disorders are also highlighted. The chapter reviews the current gaps in our understanding of the pathophysiology and treatment options of these disorders and discusses the need for considering an integrative approach, one combining complementary and conventional medicine, to treat these disorders. Recommendations are also presented for future research that may improve diagnosis and treatment.

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Brown, Gregory M., Seithikurippu R. Pandi-Perumal, and Daniel P. Cardinali. "Circadian Rhythm Sleep Disorders." In Management of Sleep Disorders in Psychiatry, edited by Amit Chopra, Piyush Das, and Karl Doghramji, 172–92. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190929671.003.0013.

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Circadian rhythm sleep disorders (CRSDs) cause disturbances in sleep and wakefulness due to a misalignment between the timing of the body’s intrinsic circadian clock and environmental light and social activity cycles. This chapter reviews these disorders with an emphasis on their neural pathways, genetic mechanisms, and regulatory factors. The authors discuss the relationship of CSRDs to physical and mental health, the treatment of CRSDs with circadian rhythm adjustment techniques, and the relationship of CSRDs to psychiatric disorders, along with potential chronobiologic treatments of psychiatric disorders. The chapter specifically addresses delayed sleep phase disorder, advanced sleep phase disorder, non-24-hour sleep–wake rhythm disorder, irregular sleep–wake disorder, shift work disorder, and chronobiology and psychiatric disorders. Melatonin and bright light therapy are covered.

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Verbraecken, J. "Impact of Intrinsic Sleep Disorders on Sleep Disordered Breathing." In Reference Module in Biomedical Sciences. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-08-102723-3.00133-5.

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Kyle, Simon D., Alasdair L. Henry, and Colin A. Espie. "Management of insomnia and circadian rhythm sleep–wake disorders." In New Oxford Textbook of Psychiatry, edited by John R. Geddes, Nancy C. Andreasen, and Guy M. Goodwin, 1167–78. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198713005.003.0114.

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Insomnia disorder and circadian rhythm sleep–wake disorders (CRSWDs) are prevalent and impairing sleep disorders and often co-present with psychiatric disorder. Insomnia is characterized by difficulty with initiation and/or maintenance of sleep, driven primarily by cognitive behavioural processes. CRSWDs manifest because of alterations to the endogenous circadian clock (intrinsic) or as a consequence of environmental circumstances (extrinsic). This chapter reviews evidence-based treatments for the management of insomnia and intrinsic CRSWDs (delayed sleep–wake phase disorder, advanced sleep–wake phase disorder, non-24-hour sleep–wake disorder, irregular sleep–wake rhythm disorder). The chapter covers cognitive behavioural therapies, sleep-promoting hypnotics, phototherapy, and exogenous melatonin administration. The chapter also highlight gaps in the existing clinical science and reflects on emergent therapeutic approaches.

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Aloia, Mark S. "Part II: BSM Protocols for Adherence and Treatment of Intrinsic Sleep Disorders: Introduction." In Behavioral Treatments for Sleep Disorders, 167–68. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-381522-4.00048-1.

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Eriksson, Sofia. "Sleep walking and other NREM parasomnias." In Oxford Handbook of Sleep Medicine, 161–70. Oxford University Press, 2022. http://dx.doi.org/10.1093/med/9780192848253.003.0017.

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Circadian Rhythm Sleep-Wake Disorders (CRSWDs) are an important group of sleep disorders, in which problems arise from aberrant timing of the sleep-wake cycle. When standard societal timetables are superimposed upon this, major problems, such as insomnia, chronic sleep restriction, and excessive daytime sleepiness may arise. These enduring problems may often have profound impacts on performance, as well as mental and physical health. Numerous CRSWDs exist. Of these, a very small number are truly ‘intrinsic’ – that is due to inherited molecular dysfunction of the pacemaker. Ageing, behaviour, illness and the environment, may influence many of the disorders, and are likely to have a cumulative effect in any one individual. However, some disorders are more ‘extrinsic’ than others, such as shift work and jet lag disorder. The various types of CRSWDs are discussed within this chapter, as are the behavioural and pharmacological interventions that can be used in their management.

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Conference papers on the topic "Intrinsic Sleep Disorders"

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Watenpaugh, Donald E., Dzu H. Dao, and John R. Burk. "Do Spouses With Intrinsic Sleep Disorders Disturb Each Others’ Sleep?" In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6525.

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