Relevant bibliographies by topics / Iron deficiency diseases

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Iron deficiency diseases'

Author: Grafiati
Published: 4 June 2021
Last updated: 2 February 2022

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Journal articles on the topic "Iron deficiency diseases"

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Bermejo, Fernando, and Santiago García-López. "A guide to diagnosis of iron deficiency and iron deficiency anemia in digestive diseases." World Journal of Gastroenterology 15, no. 37 (2009): 4638. http://dx.doi.org/10.3748/wjg.15.4638.

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Mayer-Pröschel, Margot, Dan Morath, and Mark Noble. "Are Hypothyroidism and Iron Deficiency Precursor Cell Diseases?" Developmental Neuroscience 23, no. 4-5 (2001): 277–86. http://dx.doi.org/10.1159/000048711.

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Boyko, T. "Iron metabolism and diagnostic of iron deficiency in inflammatory bowel diseases." Medicni perspektivi (Medical perspectives) 18, no. 3 (October 2, 2013): 70–77. http://dx.doi.org/10.26641/2307-0404.2013.3.18995.

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Mansoor, Malahat, and Hammad Raza. "IRON DEFICIENCY ANEMIA." Professional Medical Journal 23, no. 06 (June 10, 2016): 673–79. http://dx.doi.org/10.29309/tpmj/2016.23.06.1606.

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Around 65% of pregnant women in South Asia suffer from IDA & in Indiansub-continent alone, the rate of developing IDA during pregnancy is 88%. Moreover anemicpregnant patients are more likely to give birth to low birth weight babies which itself is anotherfactor adding to socio-economic burden on the whole family. The food has not been fortified forIron, Zinc & Vitamin D & hence the prevention of anemia has not yet been achieved Objectives:To study Awareness of women about food fortification & prevention of IDA Vs cost for treatinganemia. Period: August 2015-Dec 2015. Study Design: Observational Study. Settings: At BhattiInternational Hospital, Kasur Results: Among the selected anemic women, almost half (55%)had mild anemia, while rest had moderate to severe anemia indicting that the prevalence is verycommon. The treatment offered was oral &/or IV Iron with blood transfusions. The cost of iv Irontherapy & blood transfusions estimates in thousands with added risks of Transfusion Reactions,allergic reactions& transmission of blood-borne diseases like HCV,HBV,HIV(AIDS)&others. Theawareness level was found to be poor among these women .Only 8/60 i.e 13% had someidea about iron deficiency anemia & almost none knew about food fortifications. The insightabout their own disease of IDA was also 35% (21/60) indicating that most of the women incommunity live with IDA without any understanding of a preventable condition. Conclusion:The cost & time spend to treat IDA can be minimizes creating awareness about food fortification& supplementation.
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Bi, Yaguang, Amir Ajoolabady, Laurie J. Demillard, Wenjun Yu, Michelle L. Hilaire, Yingmei Zhang, and Jun Ren. "Dysregulation of iron metabolism in cardiovascular diseases: From iron deficiency to iron overload." Biochemical Pharmacology 190 (August 2021): 114661. http://dx.doi.org/10.1016/j.bcp.2021.114661.

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Smirnova, N. N., N. B. Kuprienko, V. P. Novikova, and A. I. Khavkin. "Iron metabolism and chronic kidney disease." Voprosy detskoj dietologii 18, no. 6 (2020): 27–34. http://dx.doi.org/10.20953/1727-5784-2020-6-27-34.

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Iron is involved in all kinds of metabolism. Iron deficiency, even in the absence of anaemia, promotes the development of many diseases. But in inflammation-associated diseases iron accumulates in the liver, kidneys and macrophages; resulting in impairment of effective erythropoiesis. The review presents modern evidence of the molecular mechanisms of iron metabolism and metabolic changes in most common diseases of the organs of the urinary system – pyelonephritis, glomerulonephritis, acute kidney injury. In most cases, anaemia has a dual nature – true iron deficiency anaemia and anaemia of chronic disease. Key words: iron, erythropoiesis, anaemia, renal pathology
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Tatarova, Nina A., and Margarita S. Airapetian. "Vitamin D deficiency and iron deficiency anemia in women in menopausal transition period. Clinical case." Gynecology 22, no. 5 (November 23, 2020): 87–90. http://dx.doi.org/10.26442/20795696.2020.5.200446.

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Vitamin D is a regulator of immune response, and plays a role in the development of cardiovascular diseases, insulin resistance and diabetes mellitus, obesity, autoimmune disorders, iron binding, anemia, respiratory viral and other diseases. In addition, vitamin D deficiency contributes to the chronicity of infections and an increased risk for number of oncological pathology. The developing immunosuppression in women with iron deficiency anemia in the menopausal transition period does not allow to fully compensate for iron deficiency with monotherapy if vitamin D deficiency is present. The drug of choice in this situation is micellized (water-soluble) vitamin D3(Aquadetrim).
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de Vizia, Basilio, Vincenzo Poggi, Rodolfo Conenna, Amedeo Fiorillo, and Luigi Scippa. "Iron Absorption and Iron Deficiency in Infants and Children with Gastrointestinal Diseases." Journal of Pediatric Gastroenterology and Nutrition 14, no. 1 (January 1992): 21–26. http://dx.doi.org/10.1097/00005176-199201000-00005.

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Shaw, Julia G., and Jennifer F. Friedman. "Iron Deficiency Anemia: Focus on Infectious Diseases in Lesser Developed Countries." Anemia 2011 (2011): 1–10. http://dx.doi.org/10.1155/2011/260380.

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Iron deficiency anemia is thought to affect the health of more than one billion people worldwide, with the greatest burden of disease experienced in lesser developed countries, particularly women of reproductive age and children. This greater disease burden is due to both nutritional and infectious etiologies. Individuals in lesser developed countries have diets that are much lower in iron, less access to multivitamins for young children and pregnant women, and increased rates of fertility which increase demands for iron through the life course. Infectious diseases, particularly parasitic diseases, also lead to both extracorporeal iron loss and anemia of inflammation, which decreases bioavailability of iron to host tissues. This paper will address the unique etiologies and consequences of both iron deficiency anemia and the alterations in iron absorption and distribution seen in the context of anemia of inflammation. Implications for diagnosis and treatment in this unique context will also be discussed.
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Baizhanova, K. T., and G. Zh Sadirkhanova. "Iron deficiency status associated with diseases of gastrointestinal tract." International Professional Journal "Medicine" 196, no. 10 (October 2018): 29–32. http://dx.doi.org/10.31082/1728-452x-2018-196-10-29-32.

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Dissertations / Theses on the topic "Iron deficiency diseases"

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Oti-Boateng, Peggy. "Effects of dietary calcium on intestinal non-haem iron absorption during weaning." Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09pho881.pdf.

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Corrigenda tipped to title page. Bibliography: leaves 313-353. This study investigated the iron status and dietary intakes in 6-24 month old children in Australia and Ghana and assessed the effects of dietary calcium on intestinal iron absorption. The true prevalence of non-anaemic iron deficiency (NAID) and iron deficiency anaemia (IDA) and dietary intakes in infants and toddlers from a broad socio-economic background were assessed by haematological and biochemical parameters, semi-quantitative diet recall and anthropometric measurements. The high prevalence of iron deficiency and anaemia found in Australian and Ghanaian children can be attributed to the low intake of bioavailable iron in weaning diets which are often ingested with large amounts of calcium. While calcium has been shown to inhibit the absorption of iron, its mechanism of interaction with iron absorption at the intestinal level is not known. The rat was used as an experimental model to investigate the effects of dietary calcium on duodenal iron uptake. The results indicate there is a critical period during weaning when the consumption of high dietary calcium with low iron can retard growth potential. Dietary calcium significantly inhibits non-haem iron absorption at the intracellular level by up-regulating villus enterocyte ferritin concentrations under iron deficiency conditions.
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Oppenheimer, Stephen James. "Iron deficiency and susceptibility to infection : a prospective study of the effects of iron deficiency and iron prophylaxis in infants in Papua New Guinea." Thesis, University of Oxford, 1987. http://ora.ox.ac.uk/objects/uuid:1891d054-1564-47f5-b2e0-b6da5f60e996.

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Investigation of the relationship between iron deficiency, iron supplementation and susceptibility to infection, was suggested by the author's initial observations of an association of anaemia with serious bacterial infections in infancy in Papua New Guinea. The bulk of previous longitudinal clinical intervention studies in infancy showed beneficial effects of iron supplementation. However, defects of control and design and recording in these studies and contradictory anecdotal reports left the question unresolved. A prospective, placebo-controlled, randomised, double-blind trial of iron prophylaxis (3ml intramuscular iron dextran = 150mg Fe) to two month old infants was carried out on the North Coast of Papua New Guinea where there is high transmission of malaria. A literature review, pilot studies, protocol, demography, geography and laboratory methods developed are described. Findings indicate that the placebo control group became relatively iron deficient over the first year of life and that the iron dextran group had adequate, although not excessive iron stores and a higher mean haemoglobin; however, the prevalence and effects of malaria recorded in the field were higher in the iron dextran group. Analysis of field and hospital infectious morbidity in the trial indicated a deleterious association with iron dextran for all causes including respiratory infections (the main single reason for admission). Total duration of hospitalisation was significantly increased in the iron dextran group. Analysis of other factors showed (1) a higher admission rate associated with low weight-for-height recorded at the start of the trial; (2) a significant positive correlation between birth haemoglobin and hospital morbidity rates; (3) increased malaria rates in primiparous mothers of the cohort infants who received iron infusion during pregnancy; (4) lower relative risk of malaria associated with iron prophylaxis in individuals with alpha thalassaemia, which was found to be highly prevalent in this region. In conclusion, it is suggested that policies of iron supplementation, total dose iron injection and routine presumptive iron therapy for anaemia which are widely in practice in malaria endemic areas should be closely reviewed.
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Adish, Abdulaziz A. "Risk factors and an assessment of control strategies for iron deficiency anemia in children in northern Ethiopia." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ36948.pdf.

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Newhouse, Ian Joseph. "The effects of prelatent and latent iron deficiency on physical work capacity." Thesis, University of British Columbia, 1987. http://hdl.handle.net/2429/27470.

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In order to examine the effects of prelatent/latent iron deficiency on physical work capacity and selected muscle enzyme activities, forty female subjects were studied before and after eight weeks of supplementation with either oral iron or a matching placebo. Initially, female volunteers engaged in regular endurance running were screened for iron deficiency by blood analysis (serum ferritin and hemoglobin). Forty non-anemic subjects with deficient iron stores underwent physiological and anthropometric tests to obtain a comprehensive profile. The specific physical work capacity tests were alactic and lactacid power on the Wingate cycle ergometer test, lactacid capacity on the anaerobic speed test, anaerobic (ventilatory) threshold using gas exchange variables, V0₂ max. and the max. treadmill velocity during the V0₂ max. test. Muscle biopsy samples pre-, and post- treatment were assayed for citrate synthase and alpha-glycerophosphate dehydrogenase activity. Treatment was oral iron supplementation (320 mg ferrous sulfate = 100 mg elemental iron taken as SLOW-Fe® twice a day) or a matching placebo. The subjects were randomly assigned to one of the treatment groups and a double-blind method of administration of the supplements was used. It was hypothesized that work capacity would be enhanced following oral iron supplementation, possibly due to the repletion of iron containing oxidative enzymes important in energy production. Results could not strongly support this hypothesis with the difference between the two groups on the work capacity and enzyme activity variables being statistically nonsignificant. Serum ferritin values rose from a mean of 12.4+4.5 to 37.7+19.7 ngml⁻¹ for the experimental group and 12.2±4.3 to 17.2±8.9 for the controls; (p=0.0025). Hemoglobin levels remained fairly constant for both treatment groups; 13.4±0.6 to 13.5±0.5 gdl⁻¹ (experimental), and 13.0±0.6 to 13.1+0.5 (control); (p=0.6). Pre to post values on the work capacity variables, experimental vs control respectively were: Alactic power, 8.8 to 8.4 watts-kg⁻¹ body wt. vs 8.4 to 8.2; lactacid capacity, 6.9 to 6.9 watts-kg⁻¹ body wt. vs 7.0 to 6.0; anaerobic speed test, 41.3 to 45.1 seconds vs 43.7 to 44.8; anaerobic threshold, 7.4 to 7.5 mileshour⁻¹ vs 7.2 to 7.2; V0₂ max, 51.3 to 52.7 ml-kg⁻¹ min⁻¹ vs 50.6 to 50.6; max velocity during V0₂ max, 9.8 to 9.8 mileshour⁻¹ vs 9.6 to 9.5. Except for alactic power, the change in work capacity favored the iron treated group. Noting this trend, further study may be warranted. Prelatent/latent iron deficiency appeared not to depress the activities of the two enzymes measured. Cytoplasmic alpha-glycerophosphate dehydrogenase activity rose from 0.066 to 0.085 units for the experimental group (p=.58) vs .058 to .066 for the control group and citrate synthase activity changed from 0.047 to 0.048 (experimental) vs 0.039 to 0.042 (control). It can be concluded that eight weeks of iron supplementation to prelatent/latent iron deficient, physically active females does not significantly enhance work capacity nor the activity of 2 oxidative muscle enzymes (citrate synthase and cytoplasmic alpha-glycerophosphate dehydrogenase). Within the limitations of this study the presence of a serum ferritin below 20 ng-ml⁻¹ does not pose a significant handicap to anaerobic or aerobic capacity.
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Harding, Scott V. "Evaluation of the iron, antioxidant and dietary status of iron supplemented breastfed healthy infants /." Internet access available to MUN users only, 2003. http://collections.mun.ca/u?/theses,160317.

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Power, Harold Michael. "A study of iron nutrition and immunity in infancy." Master's thesis, University of Cape Town, 1988. http://hdl.handle.net/11427/25837.

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Motivation and study design: Iron deficiency is a common condition in infancy, particularly in lower socio-economic groups. In Cape Town it remains a problem in spite of public health measures taken against it: a recent survey found a prevalence of iron deficiency anaemia of 34% in healthy 1-year old term infants who had ready access to a municipal health clinic where iron fortified milk formula is sold at subsidized prices. The consequences of iron deficiency extend beyond anaemia- to involve all organ systems including the immune system. Since Helen Mackay's report in 1928 of a striking decrease in incidents of infection in infants treated with iron, clinicians have assumed that iron deficiency predisposes to infection. Despite a sound theoretical basis for this belief, the clinical evidence for the assumption is poor as studies to date have displayed methodological deficiencies. On the other hand, iron is also essential for the growth of micro-organisms. As such, supplemental iron may predispose to infection. Indeed, there is much laboratory and clinical evidence to show that excess iron can result in the recrudescence of quiescent infections and increase the virulence of newly acquired infections. Thus, the competition between host and parasite may sometimes hinge on the relative availability of iron and it has been speculated that excess iron in infant milk formula may increase susceptibility to infectious diarrhoeal disease. The problem addressed by this thesis was to determine the utility of increasing the level of iron fortification of infant milk formula. Three questions were posed: Does increasing the level of iron fortification of conventional infant milk formula improve the iron nutrition of normal infants fed on the formula? Does increased iron fortification of infant milk formula alter immunity as reflected by incidence of infection and laboratory tests of immune function? Are there any handful effects of increasing the quantity of iron in conventional infant milk formula? A double blind randomized trial was carried out in 1983 and 1984 to answer these questions. A group of 149 healthy, well-nourished infants from a lower socio-economic community of so called Cape Coloureds were followed from the age of 3 months to 1 year. Half of the infants, the Control group, were given a commercially available infant milk formula (Lactogen Full Protein) which has 8.3 mg Fe/ 100 g formula and 37 mg ascorbic acid/ 100 g. The other half of subjects, the Test group, were given the same milk formula but fortified with iron to a concentration of 40 mg Fe/ 100 g. The children were examined every 3 or 4 weeks and any infection or history of infection was noted. Laboratory tests were done at the start of the trial and again on completion. During the trial, laboratory tests were performed only if clinically indicated. The tests included full blood count and differential analysis, red cell zinc protoporphyrin, plasma ferritin, plasma and hair zinc and lymphocyte subtyping with monoclonal antibodies. Within each group, half of the infants were randomly selected for assay of neutrophil bactericidal activity. The other half were assayed for lymphocyte blastogenic response to stimulation with phytohaemagglutinin. Tests of delayed cutaneous hypersensitivity to Candida antigen and PPD were done and all children and their mothers had antibodies to tetanus and polio determined. Results: 74 infants in the Control group started the trial and 62 completed it. In the Test group, 75 infants began and 70 completed the study. Intake of milk and solid foods was not quantified, but the ages of weaning and of introduction of new foods were determined. The Control and Test groups did not differ significantly on any test item. The mean age of completion of weaning was 3.60 months for the Control group and 4.04 months for the Test group. The Control group was first given meat or fish at a mean age of 5.19 months; the Test. group had meat or fish introduced to their diets at a mean age of 4.36 months. These differences were not statistically significant. The children in the Control group were lighter and shorter than the Test group at the end of the year. Mean standard deviation scores for weight were 0.23 and 0.48 respectively (P = 20%), while for length the SD scores were -0.13 and 0.06 (P = 20%).
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Amarakoon, Amarakoon Rajapakse Wasala Mohotti Mudiyanselage Darshika. "Iron Biofortification Potential of Field Pea (Pisum Sativum L.)." Thesis, North Dakota State University, 2012. https://hdl.handle.net/10365/26518.

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Iron (Fe) deficiency affects more than 3 billion of the global population. The objectives of this study were to (1) determine the genetic and environmental variation of seed Fe concentration and food matrix factors that govern Fe bioavailability in field peas (Pisum sativum L.) grown in North Dakota, USA in 2010 and 2011, and (2) determine the genetic variation of Fe uptake by field pea grown under greenhouse conditions with different Fe treatments. Seed Fe concentration in field pea samples from the field study ranged between 46-53 mg/kg with a mean of 51 mg/kg. Mean concentrations of the food matrix factors in those field peas were as follows: phytic acid=5.1 mg/g, xanthophyll=17.3 mg/100 g, canthaxanthin=86.8 mg/100 g, beta-carotene=516.8 ?g/100 g, kestose=1697 mg/100g, quercetin=54.3 mg/100 g, and ferulic acid=46.9 mg/100 g. DS Admiral and CDC Golden showed high concentrations of Fe promoter compounds and low concentrations of phytic acid. DS Admiral showed high Fe uptake with increasing Fe fertilizer rates in the greenhouse study. Therefore, DS Admiral and CDC Golden could be potential field pea genotypes for future Fe biofortification efforts.
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Sinclair, Lisa M. "Effect of iron supplementation on endurance performance in iron deficient trained males and females /." free to MU campus, to others for purchase, 2004. http://wwwlib.umi.com/cr/mo/fullcit?p1422965.

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Verrall, Tanya Christine. "Preventing iron deficiency anemia : communication strategies to promote iron nutrition for at-risk infants in northern Quebec." Thesis, McGill University, 2004. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=85102.

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A sustainable primary prevention strategy for infant iron deficiency anemia (IDA) was implemented and evaluated in a community with at-risk infants in northern Quebec, Canada. Communication strategies were used to promote iron-rich complementary food rather than iron-fortified formula, which can interfere with breastfeeding practice. This food-based approach has been successfully implemented in developing countries, but has not been applied in an industrialized country setting.
Mass media (i.e., radio dialogues, key messages, print material, point-of-purchase grocery store display) and interpersonal (i.e., homemade baby food cooking activity) communication strategies were developed in collaboration with community members and implemented in partnership with an existing community program. Reach and exposure of the strategies were measured using a questionnaire administered to a post-intervention sample (n = 45). Sales of promoted iron-rich infant food were examined pre- and post-intervention period. A repeat cross-sectional design was used for the impact evaluation. Two groups of mothers with infants, aged 7-10 months at Time 1 (n = 32) and Time 2 (n = 22) were interviewed. Outcome variables were infants' total iron and complementary food iron intakes measured by two 24-hour recalls. Secular trends in infants' hemoglobin values and milk type consumption were examined in the study community and two comparison communities.
Multiple communication channels increased awareness of IDA and influenced self-reported use of iron-rich infant food. Iron-rich infant food sales increased from pre- to post-intervention (p < 0.05). Complementary food intake iron increased between Time 1 (3.2 +/- 0.8 mg) and Time 2 (4.4 +/- 1.1 mg) (p < 0.05). The proportion of infants with anemia (hemoglobin < 110 g/L) significantly decreased from the period before (37.2%) to during (14.3%) the intervention (p < 0.05). No significant difference was found for this variable within the comparison communities. The proportion of infants receiving iron-fortified formula in the study community did not differ between Time 1 and Time 2, but increased from Time 1 (55%) to Time 2 (73%) (p < 0.05) in the comparison communities, indicating an erosion of breastfeeding practice.
These results suggest the effectiveness of communication strategies to improve infant iron nutrition in a community with good access to iron-rich infant food. The potential for this strategy in other communities warrants further investigation.
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Durrett, Timothy P. "Understanding Arabidopsis ion homeostasis in the post-genomic era assigning function to two proteins involved in iron metabolism /." Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/4437.

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Thesis (Ph.D.)--University of Missouri-Columbia, 2006.
The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file (viewed on April 27, 2009) Vita. Includes bibliographical references.
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Books on the topic "Iron deficiency diseases"

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Dincer, Yildiz. Iron deficiency and its complications. Hauppauge, N.Y: Nova Science Publishers, 2011.

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Yehuda, Shlomo. Iron deficiency and overload: From basic biology to clinical medicine. New York, N.Y: Humana Press, 2010.

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Reddy, M. Ramakrishna. Prevalence of iron deficiency anaemia and malnuitrition in India. Bangalore: Institute for Social and Economic Change, 2004.

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Ruel, Marie T. Can food-based strategies help reduce vitamin A and iron deficiencies?: A review of recent evidence. Washington, D.C: International Food Policy Research Institute, 2001.

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Nils-Georg, Asp, Hallberg Leif, Berzelius Symposium, Stiftelsen Svensk näringsforskning, and Svenska läkaresällskapet, eds. Iron Nutrition in Health and Disease: The Swedish Nutrition Foundation 20th International Symposium; the Swedish Society of Medicine Berzelius Symposium XXXI. London: John Libbey, 1996.

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Thom, Ellen. A baseline survey on the prevalence of anaemia among antenatal women attending antenatal clinics in Balaka District. [Lilongwe: s.n., 1999.

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Dow, A. I. Iron chlorosis in Washington orchards and vineyards. Pullman Wash: Cooperative Extension, College of Agriculture & Home Economics, Washington State University, 1985.

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F, Waters William, and Boy Erick, eds. Fortificación en casa con micronutrientes de los alimentos de los niños y niñas de 6 a 59 meses de edad para combatir la anemia por falta de hierro y otras deficiencias: Una estrategia familiar efectiva al nivel local para disminuir la alta prevalencia de anemia nutricional, usando chis paz de salud. Quito, Ecuador: Aliméntate Ecuador, 2008.

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International Symposium on Iron Nutrition and Interactions in Plants (7th 1993 Zaragoza, Spain). Iron nutrition in soils and plants: Proceedings of the Seventh International Symposium on Iron Nutrition and Interactions in Plants, June 27-July 2, 1993, Zaragoza, Spain. Dordrecht: Kluwer Academic Publishers, 1995.

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International Symposium on Iron Nutrition and Interactions in Plants (5th 1989 Jerusalem). Iron nutrition and interactions in plants: Proceedings of the Fifth International Symposium on Iron Nutrition and Interactions in Plants, 11-17 June 1989, Jerusalem, Israel, 1989. Dordrecht: Kluwer Academic, 1991.

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Book chapters on the topic "Iron deficiency diseases"

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Schenck, John F. "MRI of Brain Iron and Neurodegenerative Diseases: A Potential Biomarker." In Iron Deficiency and Overload, 223–40. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-462-9_13.

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Greuter, Thomas, and Stephan R. Vavricka. "Diagnosis and Management of Iron Deficiency in Inflammatory Bowel Disease." In Nutritional Management of Inflammatory Bowel Diseases, 53–64. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26890-3_4.

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Youdim, M. B. H., K. L. Leenders, and D. Ben-Shachar. "Brain Iron Uptake and Transport in Animal Model of Iron Deficiency, Tardive Dyskinesia and Neurodegenerative Diseases." In Blood—Brain Barrier, 147–56. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-0579-2_12.

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Ghoda, Manoj K. "Case 38: A 3-Year-Old Boy with Recurrent Jaundice and Severe Iron Deficiency Anemia." In Neonatal and Pediatric Liver and Metabolic Diseases, 215–16. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-9231-7_39.

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Gartler, Stanley M., R. Scott Hansen, Vinzenz Oji, Heiko Traupe, Julia Horn, Bodo Grimbacher, Srijita Sen-Chowdhry, et al. "Iron Deficiency." In Encyclopedia of Molecular Mechanisms of Disease, 1084–85. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_974.

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Sempos, Christopher T., Anne C. Looker, Daniel L. McGee, and Jürgen Rehm. "Iron and Heart Disease: A Review of the Epidemiologic Data." In Iron Deficiency and Overload, 279–98. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-462-9_16.

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Schipper, Hyman M. "Brain Iron Deposition in Aging and Disease: Role of HO-1." In Iron Deficiency and Overload, 125–39. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-462-9_7.

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Schulze, Hermann, and Axel Dignass. "Optimizing Treatment of Iron Deficiency Anemia." In Clinical Dilemmas in Inflammatory Bowel Disease, 237–40. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781444342574.ch52.

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Johnson, Martha B., Caroline A. Enns, and An-Sheng Zhang. "Iron in mammals: pathophysiological mechanisms of overload and deficiency in relation to disease." In Topics in Current Genetics, 155–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/4735_99.

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Rendle-Short, John, O. P. Gray, and J. A. Dodge. "IRON-DEFICIENCY ANAEMIA." In A Synopsis of Children's Diseases, 400–402. Elsevier, 1985. http://dx.doi.org/10.1016/b978-1-4831-8407-4.50134-6.

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Conference papers on the topic "Iron deficiency diseases"

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Wardani, Endah Kusuma, Nurul Eko Widiyastuti, Lutvia Dwi Rofika, and Wahyu Adri Wirawati. "Factors Affecting Stunting among Children Under Five Years of Age in Banyuwangi, East Java." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.03.80.

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ABSTRACT Background: Stunting, chronic malnutrition, results from the exposure of the fetus and young child to nutritional deficiency and infectious disease. In Indonesia, 30.8% of children were stunted, in which 26.2% was in East Java and 8.1% Banyuwangi Regency. This study aimed to investigate the factors affecting stunting among children under five years of age in Banyuwangi, East Java. Subjects and Method: This was a cross-sectional study conducted at Klatak and Wonosobo Community Health Centers, Central Java. a sample of 60 children under five years of age was selected for this study. The study variables were child’s gender, maternal age at pregnancy, maternal education, maternal work status, iron intake, history of chronic energy deficiency, exclusive breastfeeding, supplementary feeding, and history of infectious disease. The frequency distribution data were reported descriptively. Results: The majority of stunted children under study were male (53.3%). Most of the women were at age 20 to 34 years during pregnancy (58.3%). As many as 73.3% mothers were low educated. Most of the mothers were housewives (85%). 78.3% of women took iron supplement during pregnancy. Most of the children did not have the history of chronic energy deficiency (60%). Most of the children received exclusive breastfeeding (61.7%) and supplementary feeding (65%). Only a few children had the history of infectious disease (6.7%). Conclusion: The characteristics of subjects under study vary with maternal age at pregnancy, maternal education, maternal work status, iron intake, history of chronic energy deficiency, exclusive breastfeeding, supplementary feeding, and history of infectious disease. Keywords: stunting, children under five years of age, factors Correspondence: Endah Kusuma Wardani. Midwifery Program, School of Health Sciences Banyuwangi. Jl. Letkol Istiqlah No. 109, Banyuwangi, East Java, 68422. Email: qsuma89@yahoo.com. Mobile: +6282257193736. DOI: https://doi.org/10.26911/the7thicph.03.80
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Klimov, Leonid, Marina Stoyan, Ekaterina Zavyalova, Irina Zakharova, and Victoria Kuryaninova. "GP187 Anthropometric parameters in children with celiac disease complicated by iron deficiency status." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.248.

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Mostafa, E. "186 Ferrokinetics in anaemiaanemia of chronic disease associated with systemic lupus erythematosus and iron deficiency anaemia." In LUPUS 2017 & ACA 2017, (12th International Congress on SLE &, 7th Asian Congress on Autoimmunity). Lupus Foundation of America, 2017. http://dx.doi.org/10.1136/lupus-2017-000215.186.

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Nassar, A., M. Hunsley, and D. Singh. "ARE WE DOING UNNECESSARY DUODENAL BIOPSIES FOR COELIAC DISEASE IN PATIENTS REFERRED WITH IRON DEFICIENCY ANAEMIA (IDA)?" In ESGE Days 2019. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1681568.

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Dhandayuthapani, Rajkumar, Helen Doble, Fiona Cameron, Manjula Velayudhan Nair, Marcus Auth, Sarang Arun Tamhne, Stephen Allen, Jeng Cheng, and Elizabeth Renji. "P27 Hypophosphatemia after treatment of iron deficiency with ferric carboxylmaltose (FCM) infusion in paediatric inflammatory bowel disease in a tertiary paediatric gastroenterology centre." In Abstracts of the BSPGHAN Virtual Annual Meeting, 27–29 April 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/flgastro-2021-bspghan.37.

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Abdel Noor, R. A., M. H. Abu-Zaid, S. A. Elshweikh, E. S. Rabee, and G. E. Khedr. "FRI0081 The importance of transferrin saturation, serum ferritin, log ferritin and transferrin/log ferritin in differentiating iron deficiency anaemia from anaemia of chronic disease in rheumatoid arthritispatients." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.3969.

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Reports on the topic "Iron deficiency diseases"

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Iron deficiency in people with chronic kidney disease can be managed with either oral or IV therapy. National Institute for Health Research, June 2019. http://dx.doi.org/10.3310/signal-000773.

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