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Books on the topic 'Iron deposition'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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1

Association for Environmental Archaeology. Conference, ed. Integrating social and environmental archaeologies: Reconsidering deposition. Oxford: Archaeopress, 2010.

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2

Williamson, Robert Duncan. The deposition of iron oxide particles on surfaces from turbulent aqueous suspension. Birmingham: University of Birmingham, 1989.

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3

Ibrahim, Noor Baa'yah. Properties of yttrium iron garnet thin films grown by pulsed laser ablation deposition. [s.l.]: typescript, 1999.

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4

Jaagumagi, R. Development of the Ontario provincial sediment quality guidelines for arsenic, cadmium, chromium, copper, iron, lead, manganese, mercury, nickel, and zinc: Report. [Toronto]: Water Resources Branch, Ontario Ministry of the Environment, 1992.

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5

Lincei, Accademia nazionale dei, ed. Il deposito prelaconico di Borgo Nuovo a Taranto. Roma: Giorgio Bretschneider, 2004.

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6

United States. Congress. House. Select Committee to Investigate Covert Arms Transactions with Iran. Report of the congressional committees investigating the Iran-Contra Affair: Appendix B, depositions. Washington: U.S. House of Representatives Select Committee to Investigate Covert Arms Transactions with Iran, 1988.

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7

Freund, Holger, ed. E&G - Quaternary Science Journal Vol. 61 No 2. Greifswald, Germany: Geozon Science Media, 2012.

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8

Chemical vapor deposition of aluminide coatings on iron, nickel, and superalloys. Mumbai: Bhabha Atomic Research Centre, 2009.

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9

Trocello, Jean-Marc, and France Woimant. Disorders of Copper and Iron Metabolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0044.

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Both copper and iron are essential metals that have a critical function in a series of biochemical pathways. This chapter describes the disorders associated with genetic abnormalities in copper and iron metabolic pathways and their manifestations in adult patients. Mutations in the genes of the copper transporting P-type ATPases, ATP7A and ATP7B are associated with Wilson disease, Menkes disease, occipital horn syndrome and ATP7A-related distal motor neuropathy. Neurodegeneration with brain iron accumulation (NBIA) is a group of disorders characterized by excess iron deposition in globus pallidus, substantia nigra pars reticulata, striata and cerebellar dentate nuclei. Several genes associated with NBIA have been identified.

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10

Harding, Dennis. Death and Burial in Iron Age Britain. Oxford University Press, 2015. http://dx.doi.org/10.1093/oso/9780199687565.001.0001.

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Archaeologists have long acknowledged the absence of a regular and recurrent burial rite in the British Iron Age, and have looked to rites such as cremation and scattering of remains to explain the minimal impact of funerary practices on the archaeological record. Pit-burials or the deposit of disarticulated bones in settlements have been dismissed as casual disposal or the remains of social outcasts. In Death and Burial in Iron Age Britain, Harding examines the deposition of human and animal remains from the period - from whole skeletons to disarticulated fragments - and challenges the assumption that there should have been any regular form of cemetery in prehistory, arguing that the dead were more commonly integrated into settlements of the living than segregated into dedicated cemeteries. Even where cemeteries are known, they may yet represent no more than a minority of the total population, so that other forms of disposal must still have been practised. A further example of this can be found in hillforts which, in addition to domestic and agricultural settlements, evidently played an important role in funerary ritual, as secure community centres where excarnation and display of the dead may have made them a potent symbol of identity. The volume evaluates the evidence for violent death, sacrifice, and cannibalism, as well as age and gender distinctions, and associations with animal burials, and reveals that 'formal' cemetery burial or cremation was for most regions a minority practice in Britain until the eve of the Roman conquest.

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11

Fox, Susan H. Seizures and Shakes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190607555.003.0017.

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Wilson’s disease is an autosomal recessive, treatable heredodegenerative disorder characterized by excessive deposition of copper in the liver, brain, and other tissues including the kidneys, pancreas, and joints. Early recognition of the disorder, which can present with a variety of movement disorders and neuropsychiatric phenomena, is critical to avoid irreversible end organ damage through the initiation of copper chelating agents. Diagnosis relies first on demonstrating evidence of brain iron deposition on magnetic resonance imaging of brain and elevated urinary copper excretion in the appropriate clinical context. Genetic testing for mutations in the ATP7B gene will identify a mutation in up to 90% of cases.

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12

Tuschl, Karin, Peter T. Clayton, and Philippa B. Mills. Disorders of Manganese Metabolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0045.

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Manganese is an essential trace metal for numerous metalloenzymes. Manganese homeostasis requires tight regulation in vivo and disruption of this balance can lead to manganese overload and subsequent accumulation of manganese in brain, liver, and blood. Mutations in SLC30A10, a cell surface-localized manganese efflux transporter, cause an autosomal recessive hypermanganesemia syndrome with two distinct phenotypes: childhood onset dystonia and adult onset Parkinsonism, associated with chronic liver disease, polycythemia and features of iron depletion. MRI brain appearances are characteristic of Mn deposition with hyperintense basal ganglia on T1-weighted images. Chelation therapy with disodium calcium edetate and iron supplementation effectively lower blood manganese levels, halt liver disease progression and improve neurological symptoms.The inherited form of hypermanganesemia can be distinguished from acquired causes of manganese overload including environmental overexposure and acquired hepatocerebral degeneration in cases of end stage liver disease.

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13

Canfield, Donald Eugene. Earth’s Middle Ages: What Came after the GOE. Princeton University Press, 2017. http://dx.doi.org/10.23943/princeton/9780691145020.003.0009.

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This chapter considers the aftermath of the great oxidation event (GOE). It suggests that there was a substantial rise in oxygen defining the GOE, which may, in turn have led to the Lomagundi isotope excursion, which was associated with high rates of organic matter burial and perhaps even higher concentrations of oxygen. This excursion was soon followed by a crash in oxygen to very low levels and a return to banded iron formation deposition. When the massive amounts of organic carbon buried during the excursion were brought into the weathering environment, they would have represented a huge oxygen sink, drawing down levels of atmospheric oxygen. There appeared to be a veritable seesaw in oxygen concentrations, apparently triggered initially by the GOE. The GOE did not produce enough oxygen to oxygenate the oceans. Dissolved iron was removed from the oceans not by reaction with oxygen but rather by reaction with sulfide. Thus, the deep oceans remained anoxic and became rich in sulfide, instead of becoming well oxygenated.

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14

Ellis, David B. Microbiology of the Iron - Depositing Bacteria. Wexford College Press, 2003.

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15

Weekes, Jake. Cemeteries and Funerary Practice. Edited by Martin Millett, Louise Revell, and Alison Moore. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199697731.013.025.

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This chapter applies and attempts to contribute to the funerary process method of investigating late Iron Age and Roman period mortuary ritual in Britain. In this approach, evidence derived from archaeological contexts including tombstones and monuments, possible cemetery surfaces, cemetery boundaries, burials, pyre sites, and other features is reconsidered diachronically in relation to funerary schema. We therefore try to consider objects and actions in their correct funerary contexts, from the selectivity of death itself, through laying-out procedures, modification of the remains and other objects, degrees of spatial separation of the living and the dead, and types of deposition and commemorative acts. The development of tradition and diversity in funerary practice in Roman Britain is considered throughout, and the chapter concludes with a brief reconsideration of the multi-vocality of funerary symbolism.

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16

López-Bertran, Mireia, and Jaime Vives-Ferrándiz. Tiny Bodies for Intimate Worlds. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190614812.003.0007.

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This chapter seeks to understand the material and corporeal significance of human figurines through a comparative examination of two ritual contexts from Iron Age Iberia: the cave sanctuaries of Cástulo (Linares, Jaén) and the urban sanctuary of La Serreta (Alcoi, Alicante). The authors specifically focus on the materiality of the figurines—production technologies, patterns of use, and deposition—to address issues of social significance, body construction, and personhood among the communities that produced, handled, and deposited them. They analyze the web of material and social relations between humans, miniatures of humans, and the materials the miniatures were crafted of—bronze and clay. In the final section, the authors consider the corporeal implications of processes of miniaturization of human bodies and the qualities of the bodies that were sought after by the Iberian communities.

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17

Lei, Peng, Scott Ayton, and Ashley I. Bush. Metal-Protein Attenuating Compounds in Neurodegenerative Diseases. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190233563.003.0015.

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Neurodegenerative disorders including Alzheimer’s (AD), Parkinson’s disease (PD), Huntington’s disease (HD), and amyotrophic lateral sclerosis (ALS) are progressive diseases of the aging population with currently few therapeutic options. While aggregation and deposition of disease-specific proteins link the pathologies of these diseases, targeting these aggregating proteins with therapeutics has not yet been successful in clinical trial. This chapter profiles metals (copper, zinc, and iron) as alternative drug targets for neurodegeneration. Complex changes to metals occur in these neurodegenerative diseases. Accumulating evidences have demonstrated that perturbations to metal homeostasis contribute to the progression of neuronal dysfunction and death. Importantly, several phase II trials have shown that correcting metal dyshomeostasis improves clinical outcomes; the chapter argues that it is now time to explore the therapeutic utility of metal-based drugs in larger, phase III trials.

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18

Bleck, Thomas P. Pathophysiology and causes of seizures. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0231.

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Seizures result from imbalances between excitation and inhibition, and between neuronal synchrony and dyssynchrony. Current models implicate the cerebral cortex in the genesis of seizures, although thalamic mechanisms (particularly the thalamic reticular formation) are involved in the synchronization of cortical neurons. Often, the precipitants of a seizure in the critical care setting are pharmacological. Several mechanisms linked to critical illness can lead to seizures. Failure to remove glutamate and potassium from the extracellular space, functions performed predominantly by astrocytes, occurs in trauma, hypoxia, ischaemia, and hypoglycaemia. Loss of normal inhibition occurs during withdrawal from alcohol and other hypnosedative agents, or in the presence of GABA. Conditions such as cerebral trauma, haemorrhages, abscesses, and neoplasms all produce physical distortions of the adjacent neurons, astrocytes, and the extracellular space. Deposition of iron in the cortex from the breakdown of haemoglobin appears particularly epileptogenic. Although acute metabolic disturbances can commonly produce seizures in critically-ill patients, an underlying and potentially treatable structural lesion must always be considered and excluded.

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19

United States. Congress. Senate. Select Committee on Secret Military Assistance to Iran and the Nicaraguan Opposition., ed. Report of the congressional committees investigating the Iran-Contra Affair: Appendix B, depositions. Washington: U.S. Senate Select Committee on Secret Military Assistance to Iran and the Nicaraguan Opposition, 1988.

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