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Journal articles on the topic 'Iron stores'

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Published: 2 June 2025

Last updated: 31 July 2025

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1

Garry, Philip J. "Iron stores related to iron absorption." American Journal of Clinical Nutrition 66, no. 6 (1997): 1483–84. http://dx.doi.org/10.1093/ajcn/66.6.1483a.

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2

Kulkarni, Shruti V., Mahesh H. Karigoudar, Anita P. Javalgi, and Ambica Chalmeti. "Bone Marrow Iron Stores among Various Hematological Disorders." Indian Journal of Pathology: Research and Practice 6, no. 1 (2017): 83–87. http://dx.doi.org/10.21088/ijprp.2278.148x.6117.13.

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3

Barton, James C., Wen-Pin Chen, Mary J. Emond, et al. "GNPAT p.D519G is Independently Associated with Markedly Increased Iron Stores in HFE p.C282Y Homozygotes." Blood 128, no. 22 (2016): 3617. http://dx.doi.org/10.1182/blood.v128.22.3617.3617.

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Abstract GNPAT (chromosome 1q42.2) encodes the peroxisomal enzyme glyceronephosphate O-acyltransferase. In a previous study, DNA of men with hemochromatosis and HFE p.C282Y homozygosity and either markedly increased iron stores or normal or mildly increased iron stores were evaluated with exome sequencing. Positivity for the GNPAT polymorphism p.D519G (rs11558492) was significantly greater in men with markedly increased iron stores (McLaren CE et al., Hepatology 2015;62:429-39). This result suggests that the p.D519G is a candidate modifier of iron phenotypes in p.C282Y homozygotes. To learn mo

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4

Weeks, Bradley R., Joseph E. Smith, and Janis K. Northrop. "Relationship of serum ferritin and iron concentrations and serum total iron-binding capacity to nonheme iron stores in dogs." American Journal of Veterinary Research 50, no. 2 (1989): 198–200. https://doi.org/10.2460/ajvr.1989.50.02.198.

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SUMMARY The relationships of various iron-related analytes were evaluated in 95 dogs. Liver and spleen nonheme iron content was determined coulometrically on acid-digested tissue specimens. Serum iron concentration and total iron-binding capacity also were measured coulometrically, whereas serum ferritin concentration was measured by ELISA. Significant (P < 0.0002) correlation was found between serum ferritin concentration and nonheme iron stores. Significant correlation was not found. between nonheme iron stores and serum iron concentration or total iron-binding capacity. Serum ferritin co

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5

Fernández-Real, José Manuel, Abel López-Bermejo, and Wifredo Ricart. "Iron Stores, Blood Donation, and Insulin Sensitivity and Secretion." Clinical Chemistry 51, no. 7 (2005): 1201–5. http://dx.doi.org/10.1373/clinchem.2004.046847.

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Abstract Background: Epidemiologists have observed that blood donation is associated with decreased risk of type 2 diabetes and cardiovascular disease. Methods: We investigated the relationship between iron stores and insulin sensitivity, after controlling for known confounding factors, and compared insulin sensitivity between blood donors and individuals who had never donated blood (nondonors). In 181 men, insulin sensitivity and insulin secretion were evaluated through frequently sampled intravenous glucose tolerance tests with minimal model analysis. Men who donated blood between 6 months a

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6

Borràs, Miquel. "Hormone dependency of splenic iron stores in the rat: effect of oestrogens on the recuperation of reserves in ferrodeficient subjects." Laboratory Animals 32, no. 3 (1998): 290–97. http://dx.doi.org/10.1258/002367798780559275.

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Splenic iron stores are negligible in prepuberal rats, increasing quickly from the age of 2 months (at which moment sexual differences become apparent) and stabilizing around 3 months, when females show values approximately two-fold greater than males. Castration, adrenalectomy and hormone replacement studies show that the amount of iron stored depends directly on circulating oestrogens and is slightly but not significantly decreased, in our experimental conditions, by testosterone. The role of oestrogens is emphasized by the high correlation obtained, according to a hyperbolic regression mode

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7

Lundvall, Ove, Aleksander Weinfeld, and Per Lundin. "IRON STORES IN ALCOHOL ABUSERS." Acta Medica Scandinavica 185, no. 1-6 (2009): 259–69. http://dx.doi.org/10.1111/j.0954-6820.1969.tb07332.x.

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8

Lundvall, Ove, and Aleksander Weinfeld. "IRON STORES IN ALCOHOL ABUSERS." Acta Medica Scandinavica 185, no. 1-6 (2009): 271–77. http://dx.doi.org/10.1111/j.0954-6820.1969.tb07333.x.

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9

Cervantes, F., J. M. Mart�, A. L�pez-Guillermo, C. Piera, E. Feliu, and C. Rozman. "Iron stores in essential thrombocythaemia." Blut 58, no. 6 (1989): 291–94. http://dx.doi.org/10.1007/bf00320169.

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10

Olivares, Manuel, Eva Hertrampf, and Fernando Pizarro. "Effect of iron stores on heme iron absorption." Nutrition Research 13, no. 6 (1993): 633–38. http://dx.doi.org/10.1016/s0271-5317(05)80555-x.

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11

Naeem, Ayesha, Mazhar Nazir Chattha, and Abdul Matin Qaisar. "IRON DEFICIENT MOTHERS." Professional Medical Journal 25, no. 08 (2018): 1173–76. http://dx.doi.org/10.29309/tpmj/18.4584.

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12

Sweet, Kendra L., Najla H. Al Ali, Rami S. Komrokji, Jeffrey E. Lancet, and Javier Pinilla-Ibarz. "Macrocytosis and Iron Stores in Patients with Chronic Myeloid Leukemia Being Treated with Tyrosine Kinase Inhibitors." Blood 120, no. 21 (2012): 2769. http://dx.doi.org/10.1182/blood.v120.21.2769.2769.

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Abstract Abstract 2769 Background: Since imatinib was approved in 2001, tyrosine kinase inhibitors (TKIs) have become standard of care for first line treatment of Chronic Myeloid Leukemia (CML). Macrocytosis has been observed in some patients being treated with imatinib but the etiology of this phenomenon is unclear. Altered DNA metabolism resulting from the inhibition of c-kit by imatinib may be an explanation but anecdotal correction of macrocytic anemia after iron replacement has prompted us to explore this further. The correlation between bone marrow iron stores, anemia and macrocytosis ha

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13

Bergström, Erik, Olle Kernell, Bo Lönnerdal, and Lars Åke Persson. "Sex Differences in Iron Stores of Adolescents." Journal of Pediatric Gastroenterology and Nutrition 20, no. 2 (1995): 215–24. http://dx.doi.org/10.1002/j.1536-4801.1995.tb11537.x.

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SummaryWe evaluated iron status and its determinants in healthy adolescents. Fasting morning blood samples from a school‐based cross‐sectional study were analyzed for serum ferritin (SF), serum iron, total iron‐binding capacity, and circulating transferrin receptors. Physical development, chronic disease, medication, dietary intake, and physical activity were assessed using clinical examination, questionnaires, and 7‐day records. The risk of having low serum ferritin values was estimated using bivariate and multivariate regression. Subjects were 867 healthy Swedish adolescents, 14− and 17‐year

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14

Sikström, C., L. Beckman, G. Hallmans, and K. Asplund. "Transferrin Types, Iron-Binding Capacity and Body Iron Stores." Human Heredity 43, no. 6 (1993): 337–41. http://dx.doi.org/10.1159/000154156.

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15

Murray-Kolb, Laura E., Ross Welch, Elizabeth C. Theil, and John L. Beard. "Women with low iron stores absorb iron from soybeans." American Journal of Clinical Nutrition 77, no. 1 (2003): 180–84. http://dx.doi.org/10.1093/ajcn/77.1.180.

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16

V, Anupama K., Purnima S. Rao, Sushma Adappa, Prashantha Balanthimogru, and Chakrapani Mahabala. "Correlation between serum ferritin and bone marrow iron stores." Tropical Doctor 47, no. 3 (2016): 217–21. http://dx.doi.org/10.1177/0049475516678478.

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Bone marrow aspirate examination is a gold standard to assess bone marrow iron stores. The correlation between serum ferritin and bone marrow iron has not been established in detail, as the cutoff value for iron stores have not been uniformly established. Ours was a cross-sectional study. Perl’s Prussian blue stain was used to stain bone marrow, assessed by Gale’s grading. Receiver operating characteristic curve analysis and Spearman’s correlation coefficient calculated. Gale’s grading revealed iron store deficiency in 26 and sufficiency in 13. Spearman’s correlation coefficient of 0.90 showed

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17

Kämmerer, Lara, Goran Mohammad, Magda Wolna, Peter A. Robbins, and Samira Lakhal-Littleton. "Fetal liver hepcidin secures iron stores in utero." Blood 136, no. 13 (2020): 1549–57. http://dx.doi.org/10.1182/blood.2019003907.

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Abstract In the adult, the liver-derived hormone hepcidin (HAMP) controls systemic iron levels by blocking the iron-exporting protein ferroportin (FPN) in the gut and spleen, the sites of iron absorption and recycling, respectively. Impaired HAMP expression or FPN responsiveness to HAMP result in iron overload. HAMP is also expressed in the fetal liver but its role in controlling fetal iron stores is not understood. To address this question in a manner that safeguards against the confounding effects of altered maternal iron homeostasis, we generated fetuses harboring a paternally-inherited ubi

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18

Benkhedda, Karima, Mary R. L'Abbé, and Kevin A. Cockell. "Effect of calcium on iron absorption in women with marginal iron status." British Journal of Nutrition 103, no. 5 (2009): 742–48. http://dx.doi.org/10.1017/s0007114509992418.

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We measured non-haem Fe absorption with and without added Ca in a short-term feeding study, in thirteen women with marginal Fe status, by the use of a double stable isotope technique. Supplementing 500 mg Ca as calcium carbonate significantly (P = 0·0009) reduced Fe absorption from a single meal from 10·2 % (range 2·2–40·6) to 4·8 % (range 0·7–18·9). A significant inverse correlation in the absence ( − 0·67,P = 0·010) and presence ( − 0·58,P = 0·037) of Ca, respectively, was found between Fe absorption and Fe stores measured by serum ferritin (SF). Wide variation in Fe absorption was observed

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19

Haymes, Emily M. "Nutrition and the Physically Active Female." Women in Sport and Physical Activity Journal 1, no. 1 (1992): 35–47. http://dx.doi.org/10.1123/wspaj.1.1.35.

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Muscle glycogen is the primary source of energy during high intensity exercise. Increasing the carbohydrate content of the diet allows more glycogen to be stored. Some adolescent female athletes (gymnasts, dancers) do not consume adequate amounts of vitamin B6, folacin, and E. Many women have low dietary intakes of calcium and iron. Low calcium intake and physical inactivity are factors associated with the development of osteoporosis. Low iron intake is associated with the development of iron deficiency and anemia. Low ferritin levels (an index of body iron stores) are commonly observed in fem

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20

Kovesdy, Csaba P., Grace H. Lee, and Kamyar Kalantar-Zadeh. "Serum Ferritin: Deceptively Simple or Simply Deceptive? Lessons Learned From Iron Therapy in Patients With Chronic Kidney Disease." Journal of Pharmacy Practice 21, no. 6 (2008): 411–19. http://dx.doi.org/10.1177/0897190008318915.

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Iron is an essential micronutrient that is indispensable for erythropoesis. Correct assessment of iron stores is needed both for the diagnosis of iron deficiency and to direct iron replacement therapies. Serum ferritin is a commonly employed measure to assess iron stores, yet there are caveats that influence its accuracy as a diagnostic tool. While low ferritin levels are specific for iron deficiency, high levels can be the result of inflammation, liver disease, or malignancies and could be independent of iron stores. Optimal anemia management involves administration of adequate amounts of iro

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21

WATANABE, Kiyotaka, and Koichi ORINO. "High Iron Stores in Bovine Fetuses." Journal of the Japan Veterinary Medical Association 53, no. 11 (2000): 744–46. http://dx.doi.org/10.12935/jvma1951.53.744.

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22

Charache, Samuel, Alan M. Gittlelsohn, Hubert Allen, et al. "Noninvasive Assessment of Tissue Iron Stores." American Journal of Clinical Pathology 88, no. 3 (1987): 333–37. http://dx.doi.org/10.1093/ajcp/88.3.333.

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23

Pilote, Louise, Lawrence Joseph, Patrick Bélisle, Killian Robinson, Frederic Van Lente, and Ira B. Tager. "Iron stores and coronary artery disease." Journal of Clinical Epidemiology 53, no. 8 (2000): 809–16. http://dx.doi.org/10.1016/s0895-4356(99)00234-6.

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24

Auer, Johann, Robert Berent, Thomas Weber, and Bernd Eber. "Coronary atherosclerosis and body iron stores." Journal of the American College of Cardiology 41, no. 10 (2003): 1848–49. http://dx.doi.org/10.1016/s0735-1097(03)00325-5.

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25

Mascitelli, Luca, Francesca Pezzetta, and Mark R. Goldstein. "Altitude, body iron stores, and cancer." Medical Hypotheses 75, no. 6 (2010): 675–76. http://dx.doi.org/10.1016/j.mehy.2010.07.019.

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26

Mascitelli, Luca, Francesca Pezzetta, and Mark R. Goldstein. "Menopause, increased iron stores and cholesterol." Maturitas 65, no. 4 (2010): 403. http://dx.doi.org/10.1016/j.maturitas.2010.01.006.

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27

Singla, Reecha, Suparna Grover, Ashwani Kumar, and Sukhraj Kaur. "Effects of maternal iron deficiency anemia on the iron stores of the new born- an analytical study." International Journal of Research in Medical Sciences 13, no. 2 (2025): 772–77. https://doi.org/10.18203/2320-6012.ijrms20250252.

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Background: The aim of the study was to determine the effect of maternal iron deficiency anemia on neonatal iron stores. Methods: This prospective study was conducted in the department of obstetrics and gynecology, Government Medical College, Amritsar from January 2023 to March 2024. A total of 200 antenatal patients at gestation ≥37 weeks were allotted to four groups: group A (included non-anemic cases), group B (included cases with mild to moderate anemia with hemoglobin 7-10.9 gm/dl), group C included cases with severe anemia with hemoglobin <7 gm/dl) and group D (included treated cases

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28

Ganz, Tomas. "Systemic Iron Homeostasis." Physiological Reviews 93, no. 4 (2013): 1721–41. http://dx.doi.org/10.1152/physrev.00008.2013.

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The iron hormone hepcidin and its receptor and cellular iron exporter ferroportin control the major fluxes of iron into blood plasma: intestinal iron absorption, the delivery of recycled iron from macrophages, and the release of stored iron from hepatocytes. Because iron losses are comparatively very small, iron absorption and its regulation by hepcidin and ferroportin determine total body iron content. Hepcidin is in turn feedback-regulated by plasma iron concentration and iron stores, and negatively regulated by the activity of erythrocyte precursors, the dominant consumers of iron. Hepcidin

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29

Lynch. "Influence of Infection/Inflammation, Thalassemia and Nutritional Status on Iron Absorption." International Journal for Vitamin and Nutrition Research 77, no. 3 (2007): 217–23. http://dx.doi.org/10.1024/0300-9831.77.3.217.

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Iron balance in human beings is maintained by the control of absorption. Recent observations have demonstrated that a peptide hormone, hepcidin, is the principal regulator of iron homeostasis. It is produced in the liver in response to increasing iron stores. It is also induced by interleukin-6 (IL-6) in infectious and inflammatory diseases. Hepcidin restricts both iron absorption and iron release from stores. Disorders that affect the duodenum or stomach directly, particularly gluten enteropathy and H. pylori infections, also impair iron absorption by damaging enterocytes or reducing gastric

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30

Castro, Oswaldo L., Mehdi Nouraie, Lori Luchtman-Jones, et al. "Lower Ferritin Concentrations Are Associated with Decreased Hemolysis in Sickle Cell Disease Children without Iron Overload." Blood 114, no. 22 (2009): 2571. http://dx.doi.org/10.1182/blood.v114.22.2571.2571.

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Abstract Abstract 2571 Poster Board II-548 The role of iron in the pathophysiology of sickle cell disease (SCD) is complex and not fully understood. Iron overload is associated with disease severity primarily because multiple transfusions are linked to a severe SCD clinical course. Additionally, hemolysis, also associated with disease severity, increases iron absorption. Iron deficiency decreases red cell MCHC, which lowers Hb S polymerization and thus may improve the clinical manifestations of SCD. Such a hypothesis is supported by our recent observation of a homozygous SCD adult with iron de

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31

Johnson, David W., Karen A. Herzig, Ruth Gissane, Scott B. Campbell, Carmel M. Hawley, and Nicole M. Isbel. "Oral versus Intravenous Iron Supplementation in Peritoneal Dialysis Patients." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 21, no. 3_suppl (2001): 231–35. http://dx.doi.org/10.1177/089686080102103s41.

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The vast majority of erythropoietin (EPO)–treated peritoneal dialysis (PD) patients require iron supplementation. Most authors and clinical practice guidelines recommend primary oral iron supplementation in PD patients because it is more practical and less expensive. However, numerous studies have clearly demonstrated that oral iron therapy is unable to maintain EPO-treated PD patients in positive iron balance. Once patients become iron-deficient, intravenous iron administration has been found to more effectively augment iron stores and hematologic response than does oral therapy. We recently

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32

Bozzini, Claudia, Domenico Girelli, Elisa Tinazzi, et al. "Biochemical and Genetic Markers of Iron Status and the Risk of Coronary Artery Disease: An Angiography-based Study." Clinical Chemistry 48, no. 4 (2002): 622–28. http://dx.doi.org/10.1093/clinchem/48.4.622.

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Abstract Background: Iron may promote coronary atherosclerotic disease (CAD) by increasing lipid peroxidation. Studies on biochemical or genetic markers of body iron stores as risk factors for CAD have yielded conflicting results. Methods: We studied 849 individuals with a clear-cut definition of the CAD phenotype, i.e., with (CAD; n = 546) or without (CAD-free; n = 303) angiographically documented disease. We determined serum ferritin, as a biochemical estimate of iron stores, and the C282Y mutation in the HFE gene, i.e., the main cause of hemochromatosis in Caucasians. The relationships of f

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33

Bartnikas, Thomas Benedict, Carolina Herrera, and Michael A. Pettiglio. "Transferrin Deficiency Leads To Specific and Partially Reversible Iron Overload." Blood 122, no. 21 (2013): 963. http://dx.doi.org/10.1182/blood.v122.21.963.963.

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Abstract Studies in hpx mice, a model of inherited transferrin deficiency, have demonstrated that transferrin is essential for iron delivery to the erythron and regulation of expression of hepcidin, a hormone that inhibits dietary iron absorption. Here we address two other putative roles for s transferrin. First, we determined if transferrin is essential for metabolism of metals other than iron. Metal content analysis of fractionated mouse sera indicated that iron was the most abundant metal in transferrin-rich fractions. Organ metal content analysis demonstrated severe imbalances for iron and

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34

Adams, Paul C., and James C. Barton. "How I treat hemochromatosis." Blood 116, no. 3 (2010): 317–25. http://dx.doi.org/10.1182/blood-2010-01-261875.

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AbstractHemochromatosis is a common genetic disorder in which iron may progressively accumulate in the liver, heart, and other organs. The primary goal of therapy is iron depletion to normalize body iron stores and to prevent or decrease organ dysfunction. The primary therapy to normalize iron stores is phlebotomy. In this opinion article, we discuss the indications for and monitoring of phlebotomy therapy to achieve iron depletion, maintenance therapy, dietary and pharmacologic maneuvers that could reduce iron absorption, and the role of voluntary blood donation.

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35

Valenti, Luca, Dorine W. Swinkels, Larry Burdick, et al. "Serum Ferritin Levels Are Associated with Vascular Damage in Patients with Nonalcoholic Fatty Liver Disease." Blood 114, no. 22 (2009): 5098. http://dx.doi.org/10.1182/blood.v114.22.5098.5098.

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Abstract Abstract 5098 Background and Aims Increased ferritin and body iron stores are frequently observed in nonalcoholic fatty liver disease (NAFLD), associated with heightened susceptibility to vascular damage. Conflicting data have been reported on the role of iron in atherosclerosis, with recent data suggesting that excess iron induces vascular damage by increasing levels of the hormone hepcidin, which would determine iron trapping into macrophages, oxidative stress, and promotion of transformation into foam cells. Aim of this study was to investigate the relationship between iron status

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36

Rivera, Seth, Miguel Lopez, Dina Farshidi, Victoria Gabayan, and Tomas Ganz. "Ceruloplasmin Is Essential for the Mobilization of Tissue Iron Stores." Blood 106, no. 11 (2005): 3581. http://dx.doi.org/10.1182/blood.v106.11.3581.3581.

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Abstract In extracellular fluid, iron is in the ferric (oxidized form) but the intracellular form is ferrous iron (reduced). The outflow of iron from cells is dependent on oxidase activity that converts ferrous to ferric iron. Iron-absorbing enterocytes possess a unique iron oxidase, hephaestin. It is presumed that the circulating hephaestin paralog ceruloplasmin fulfils this role in hepatocytes and macrophages. The DiSnA mouse lacks ceruloplasmin. We hypothesized that iron homeostasis in this mouse would be unusually dependent on dietary iron because the mouse would not be able to mobilize ir

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37

Horwitz, Lawrence D., and Eli A. Rosenthal. "Iron-mediated cardiovascular injury." Vascular Medicine 4, no. 2 (1999): 93–99. http://dx.doi.org/10.1177/1358836x9900400207.

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Iron is an essential element for normal cellular function and general health. However, iron may play a pathologic role in certain cardiac conditions including reperfusion injury, hemochromatosis, b-thalassemia and coronary atherosclerosis. It also may play a role in injury due to anthracycline cardiotoxicity. Removal of iron via phlebotomy for hemochromatosis and chelation therapy for b-thalassemia are proven treatments. Cell culture, and isolated organ and animal studies suggest that depleting iron stores may prevent reperfusion injury, restenosis and even atherogenesis. This article will rev

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38

Saha, Ananya, Pradip Mukhopadhyay, Indrajit Nath, Arun Kumar, and Utpal Kumar Biswas. "A quantitative assessment of body iron status and its relationship with glycemic control in patients of type 2 diabetes mellitus in a tertiary care hospital of Kolkata." Asian Journal of Medical Sciences 12, no. 5 (2021): 69–74. http://dx.doi.org/10.3126/ajms.v12i5.33344.

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Background: Diabetes is one of the most common disease which is observed in every household of Indian population. The longevity of the diabetic patients is dependent upon the frequency of complication and comorbidity that they encounter. Serum iron and ferritin, both being the aggravators to the oxidative stress accelerating the development of complications, gives us the reason to venture into the territory exploring the possibility of monitoring the body iron stores and taking prevent measures to control such complication. The current study was designed with an aim to knot the relationship be

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39

Moum, Bjørn, and Stefan Lindgren. "Iron Deficiency and Iron Deficiency Anemia in Chronic Disease—Common, Important, and Treatable." Journal of Clinical Medicine 14, no. 13 (2025): 4519. https://doi.org/10.3390/jcm14134519.

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Iron has many important functions related to energy metabolism. However, hemoglobin synthesis is always a priority. Iron deficiency can be caused by increased loss, insufficient intake, or decreased absorption from the intestine and reduced release from depots in systemic inflammation. Anemia appears when stores are depleted or when utilization of iron from the stores is impaired. Treatment with oral iron is the first choice when the intestine is healthy, and the patient is free of inflammation. Intravenous iron is indicated when oral iron is ineffective or not tolerated and if more rapid corr

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40

Whitfield, J. B., G. Zhu, A. C. Heath, L. W. Powell, and N. G. Martin. "Effects of Alcohol Consumption on Indices of Iron Stores and of Iron Stores on Alcohol Intake Markers." Alcoholism: Clinical and Experimental Research 25, no. 7 (2001): 1037–45. http://dx.doi.org/10.1111/j.1530-0277.2001.tb02314.x.

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41

Pateva, Irina, Elisabeth Kerling, Susan Carlson, Manju Reddy, Dan Chen, and Jakica Tancabelic. "Effect Of Maternal Cigarette Smoking On Newborn Iron Stores." Blood 122, no. 21 (2013): 4671. http://dx.doi.org/10.1182/blood.v122.21.4671.4671.

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Objective Previous small-scale studies suggest that maternal smoking lowers neonatal body iron. Our objective was to study and compare the relationship between maternal and infants’ body iron in smokers and non-smokers in a large matched-pair cohort. Method This was a prospective cohort study involving 144 mothers – 72 smokers and 72 non-smokers and their respective infants. Samples were obtained from maternal blood and infants’ cord blood at delivery for serum transferrin receptor (sTfR) and ferritin levels. Serum TfR and ferritin levels were measured by RAMCO ELISA and RIA assays. The total

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42

Gavin, M. W., D. M. McCarthy, and P. J. Garry. "Evidence that iron stores regulate iron absorption— a setpoint theory." American Journal of Clinical Nutrition 59, no. 6 (1994): 1376–80. http://dx.doi.org/10.1093/ajcn/59.6.1376.

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43

Garry, P. J., K. M. Koehler, and T. L. Simon. "Iron stores and iron absorption: effects of repeated blood donations." American Journal of Clinical Nutrition 62, no. 3 (1995): 611–20. http://dx.doi.org/10.1093/ajcn/62.3.611.

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44

Pizarro, Fernando, Raúl Uicich, Manuel Olivares, et al. "Iron absorption of ferric glycinate is controlled by iron stores." Nutrition Research 18, no. 1 (1998): 3–9. http://dx.doi.org/10.1016/s0271-5317(97)00194-2.

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45

Moazzen, Sara, Maike G. Sweegers, Mart Janssen, Boris M. Hogema, Trynke Hoekstra, and Katja Van den Hurk. "Ferritin Trajectories over Repeated Whole Blood Donations: Results from the FIND+ Study." Journal of Clinical Medicine 11, no. 13 (2022): 3581. http://dx.doi.org/10.3390/jcm11133581.

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Background: Depending on post-donation erythropoiesis, available iron stores, and iron absorption rates, optimal donation intervals may differ between donors. This project aims to define subpopulations of donors with different ferritin trajectories over repeated donations. Methods: Ferritin levels of 300 new whole blood donors were measured from stored (lookback) samples from each donation over two years in an observational cohort study. Latent classes of ferritin level trajectories were investigated separately using growth mixture models for male and female donors. General linear mixed models

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46

Bencaiova, Gabriela, and Christian Breymann. "Mild Anemia and Pregnancy Outcome in a Swiss Collective." Journal of Pregnancy 2014 (2014): 1–7. http://dx.doi.org/10.1155/2014/307535.

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Background. Over half of all women in the world experience anemia during their pregnancy. Our aim was to investigate the relation between hemoglobin and iron status examined in second trimester and pregnancy outcome.Methods. In a prospective longitudinal study, 382 pregnant women were included. Blood samples were examined for hematological status and serum ferritin between 16 and 20 weeks and for hemoglobin before delivery. The adverse maternal and perinatal outcomes were determined. Regression analysis was performed to establish if anemia and low serum ferritin are risk factors for pregnancy

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Khan, Hamzullah, Mohammad Basharat Khan, Shahtaj Khan, Saiqa Zahoor, and Anwar Khan Wazir. "An Analytical Study to Explore Iron Stores in a Population of Nowshera Based on Age and Gender Perspective." Journal of Gandhara Medical and Dental Science 9, no. 1 (2022): 33–38. http://dx.doi.org/10.37762/jgmds.9-1.132.

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OBJECTIVES: To analyze the impact of age and gender on iron stores in a population of the Nowshera region. METHODOLOGY: This cross sectional study was conducted in the Department of Pathology Qazi Hussain Ahmed Medical Complex Nowshera from 1st January 2019 to 31st March 2020. All patients were selected by convenience sampling in the Pathology department irrespective of age and gender. Both descriptive and inferential statistics were applied to analyze data by the latest SPSS version 25. RESULTS: Out of the total study population males were 70 (27.1%) and females 188 (77.9%) with median age 30

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Kasprzak, Annika, Sandra Becker, Martina Rudelius, et al. "Increased Bone Marrow Iron at Diagnosis Is Associated with Inferior Prognosis in Patients with Myelodysplastic Syndromes." Blood 138, Supplement 1 (2021): 3700. http://dx.doi.org/10.1182/blood-2021-152402.

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Abstract Introduction: Iron storage in patients (pts) with myelodysplastic syndromes at the time of diagnosis may vary from normal to iron overload. Even before the first blood transfusion, storage iron can be increased due to down-regulation of hepcidin and subsequent increase in duodenal iron uptake. Iron overload is known to worsen the prognosis of MDS patients, partly due to iron-related organ damage after long-term transfusion therapy, and partly due to an increased risk of infections. However, it is unclear whether increased storage iron at the time of diagnosis already has a prognostic

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49

Fillet, G., Y. Beguin, and L. Baldelli. "Model of reticuloendothelial iron metabolism in humans: abnormal behavior in idiopathic hemochromatosis and in inflammation." Blood 74, no. 2 (1989): 844–51. http://dx.doi.org/10.1182/blood.v74.2.844.844.

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Abstract Iron transport in the reticuloendothelial (RE) system plays a central role in iron metabolism, but its regulation has not been characterized physiologically in vivo in humans. In particular, why serum iron is elevated and RE cells are much less iron-loaded than parenchymal cells in idiopathic hemochromatosis is not known. The processing of erythrocyte iron by the RE system was studied after intravenous (IV) injection of 59Fe heat-damaged RBCs (HDRBCs) and 55Fe transferrin in normal subjects and in patients with iron deficiency, idiopathic hemochromatosis, inflammation, marrow aplasia,

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50

Fillet, G., Y. Beguin, and L. Baldelli. "Model of reticuloendothelial iron metabolism in humans: abnormal behavior in idiopathic hemochromatosis and in inflammation." Blood 74, no. 2 (1989): 844–51. http://dx.doi.org/10.1182/blood.v74.2.844.bloodjournal742844.

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Abstract:

Iron transport in the reticuloendothelial (RE) system plays a central role in iron metabolism, but its regulation has not been characterized physiologically in vivo in humans. In particular, why serum iron is elevated and RE cells are much less iron-loaded than parenchymal cells in idiopathic hemochromatosis is not known. The processing of erythrocyte iron by the RE system was studied after intravenous (IV) injection of 59Fe heat-damaged RBCs (HDRBCs) and 55Fe transferrin in normal subjects and in patients with iron deficiency, idiopathic hemochromatosis, inflammation, marrow aplasia, or hyper

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