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Books on the topic 'Ischemia – Complications'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

Napoli, Mario Di. Neuroinflammation: An important role in the pathogenic pathways of cerebral ischemia. Nova Science Publishers, 2008.

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2

Shioshvili, Tamaz J. Kidney transplantation: Denervation, reinnervation, ischemia, hypothermia. Nova Science, 2009.

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3

Smith, T. C. G. 1939-, ed. Ischaemia in head injury: 10th European Congress of Neurosurgery, Berlin 1995 ; proceedings of a special symposium. Springer, 1996.

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4

Johannes, Janse Michiel, ed. The ventricular arrhythmias of ischemia and infarction: Electrophysiological mechanisms. Futura Pub. Co., 1993.

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5

1938-, Rosen Michael R., and Palti Yoram, eds. Lethal arrhythmias resulting from myocardial ischemia and infarction: [proceedings of the Second Rappaport Symposium]. Kluwer Academic Publishers, 1989.

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6

Steinau, Hans-Ulrich. Major limb replantation and postischemia syndrome: Investigation of acute ischemia-induced myopathy and reperfusion injury. Springer-Verlag, 1987.

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7

Hayreh, Sohan Singh. Ischemic optic neuropathies. Springer, 2011.

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8

A, Baethmann, Go K. G, and Unterberg A, eds. Mechanisms of secondary brain damage. Plenum Press, 1986.

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9

Zoppo, Gregory J. Del. Innate inflammation and stroke. Edited by New York Academy of Sciences. Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2010.

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10

D, Silver Malcolm, ed. Sudden death in ischemic heart disease: An alternative view on the significance of morphologic findings. Springer-Verlag, 1995.

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11

International, Symposium on Mechanisms of Secondary Brain Damage (7th 2003? Mules Italy). Mechanisms of secondary brain damage from trauma and ischemia: Recent advances of our understanding : [proceedings of 7th International Symposium on Mechanisms of Secondary Brain Damage, Mauls, Italy]. Springer, 2004.

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12

A, Baethmann, ed. Mechanisms of secondary brain damage from trauma and ischemia: Recent advances of our understanding : [proceedings of 7th International Symposium on Mechanisms of Secondary Brain Damage, Mauls, Italy]. Springer, 2004.

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13

Exercise prescription for the high-risk cardiac patient. Human Kinetics, 1998.

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14

1943-, Hartmann A., Kuschinsky Wolfgang 1944-, and Hoyer S. 1933-, eds. Cerebral ischemia and dementia. Springer-Verlag, 1991.

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15

Khorgami, Zhamak, and Ariel Ortiz Lagardere. Complications of Gastric Plication. Edited by Tomasz Rogula, Philip Schauer, and Tammy Fouse. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190608347.003.0037.

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Laparoscopic gastric plication is infolding of the greater curvature of the stomach to reduce gastric volume. This chapter covers the complications of the procedure, their causes, and their management. The most common complication is gastric obstruction, and it is the main reason for reoperation. Most mild obstructions can be managed conservatively. Patients with signs of ischemia and those with poor response to medical treatment need reoperation. Mild bleeding is not infrequent after gastric plication but severe bleeding is rare. Gastric perforation can occur, and treatment options include conservative management of a minimal contained leak, or surgical exploration with plication reversal and repair. Loss of restriction and weight regain are a result of insufficient reduction of the stomach capacity or plication breakdown. Other complications include persistent heartburn, gastric wall herniation (which can lead to ischemia and perforation), gastric intussusception, and gastric ulcers.
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16

J, Hearse David, Manning Allan S, and Janse Michiel Johannes, eds. Life-threatening arrhythmias during ischemia and infarction. Raven Press, 1987.

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17

Caplan, Louis R. Brain Ischemia: Basic Concepts and Clinical Relevance. Springer-Verlag, 2011.

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18

R, Caplan Louis, ed. Brain ischemia: From basic science to treatment. Springer-Verlag, 1994.

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19

S, Dhalla Naranjan, ed. Myocardial ischemia and preconditioning. Kluwer Academic, 2003.

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20

Takeda, Nobuakira, Naranjan S. Dhalla, Manjeet Singh, and Anton Lukas. Myocardial Ischemia and Preconditioning. Springer, 2012.

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21

Pharm, Boucher Michel B., and Canadian Coordinating Office for Health Technology Assessment., eds. The role of clopidogrel in the secondary prevention of recurrent ischemic vascular events after acute myocardial ischemia: A critical appraisal of the CURE Trial. Canadian Coordinating Office for Health Technology Assessment, 2002.

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22

Pluta, Ryszard. Ischemia-Reperfusion Pathways in Alzheimer's Disease. Nova Science Pub Inc, 2007.

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23

H, Opie Lionel, ed. Stunning, hibernation, and calcium in myocardial ischemia and reperfusion. Kluwer Academic, 1992.

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24

(Editor), Naranjan S. Dhalla, Nobuakira Takeda (Editor), Manjeet Singh (Editor), and Anton Lukas (Editor), eds. Myocardial Ischemia and Preconditioning (Progress in Experimental Cardiology). Springer, 2002.

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25

1928-, Takeshita Hiroshi, Siesjö Bo K, Miller J. Douglas 1937-, Yamaguchi Daigaku, Japan Monbushō, and International Symposium on Brain Resuscitation (1988 : Ube-shi, Japan), eds. Advances in brain resuscitation. Springer-Verlag, 1991.

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26

Lethal Arrhythmias Resulting from Myocardial Ischemia and Infarction (Developments in Cardiovascular Medicine). Springer, 1988.

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27

(Editor), A. Baethmann, N. Plesnila (Editor), F. Ringel (Editor), and J. Eriskat (Editor), eds. Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia: Proceedings of the 6th International Symposium: Mechanisms of ... 1998 (Acta Neurochirurgica Supplementum). Springer, 1999.

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28

D, Heuser, ed. Indikation und Praxis cerebroprotektiver Massnahmen in der Neurochirurgie: Bericht über eine Gesprächsrunde am 8. Juni 1985 in Frankfurt. Springer, 1985.

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29

B, Goldstein Larry, ed. Restorative neurology: Advances in pharmacotherapy for recovery after stroke. Futura, 1998.

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30

Goldstein, Larry B. Restorative Neurology: Advances in Pharmacotherapy for Recovery After Stroke. Blackwell Publishing Limited, 1998.

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31

Baethmann, A., and K. G. Go. Mechanisms of Secondary Brain Damage (Nato a S I Series Series a, Life Sciences). Springer, 1986.

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32

Parikh, Roshni A., and David M. Williams. Obtaining True Lumen Access in Aortic Dissections with Iliac Extension. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0012.

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Aortic dissection resulting in lower extremity ischemia is an emergent condition requiring urgent endovascular treatment of the aorta and iliofemoral arteries to restore blood flow to the lower extremities. This chapter describes the management, applications, challenges, and potential complications when obtaining bilateral true lumen access during the urgent endovascular treatment of lower extremity ischemia after an aortic dissection. Most interventions require retrograde access to the true lumen; however, if one of the common femoral arteries is dissected, then retrograde access can be a challenge. Utilizing the contralateral true lumen, retrograde access can be confidently obtained through the true lumen of a dissected artery. This chapter illustrates the steps involved in successfully obtaining bilateral retrograde access to the common femoral arteries when these challenging cases arise.
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33

(Editor), A. Baethmann, J. Eriskat (Editor), J. Lehmberg (Editor), and N. Plesnila (Editor), eds. Mechanisms of Secondary Brain Damage from Trauma and Ischemia: Recent Advances of Our Understanding (Acta Neurochirurgica Supplementum). Springer, 2004.

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34

Yang, Hui. Carotid Endarterectomy/Stenting. Edited by David E. Traul and Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0007.

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Carotid endarterectomy and carotid artery stenting have been performed worldwide to reduce neurologic symptoms and prevent stroke. Preoperative cardiovascular and neurologic evaluations are essential to optimize a patient for surgery and guide the intra- and postoperative management. The primary goals of anesthetic management are to maintain cerebral perfusion and prevent perioperative myocardial ischemia. A shunt may be placed to provide antegrade cerebral blood flow if signs of cerebral ischemia develop during carotid cross-clamping. An awake patient is the gold standard for monitoring the adequacy of cerebral perfusion, although neurologic monitoring may be used if a patient is under general anesthesia. Rapid emergence allows immediate assessment of neurologic function. Any delayed emergence necessitates prompt management and communication to surgeon. All patients need to be closely monitored postoperatively for any neurologic, cardiovascular, or respiratory complications.
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35

J, Reulen H., Philippon Jacques, and European Association of Neurosurgical Societies. Meeting, eds. Prevention and treatment of delayed ischaemic dysfunction in patients with subarachnoid haemorrhage: An update. Springer-Verlag, 1988.

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36

Sokolov, E. I. Obesity And Diabetes Mellitus. CORONET BOOKS, 1997.

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37

Fraser, Britt. Acute Fluid Resuscitation for Intussusception. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0004.

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Intussusception occurs when a proximal section of bowel invaginates into more distal bowel and is then advanced by peristalsis. It is the most common cause of intestinal obstruction in infants, and untreated can lead to bowel ischemia and perforation. Early recognition and treatment can prevent the need for surgical intervention and complications. Intussusception can also result in significant dehydration due to vomiting and diarrhea. An essential aspect of the perioperative management is to identify and treat dehydration.
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38

Turner, Joshua A., and Todd M. Brown. Treadmill Exercise Testing. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0011.

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Treadmill exercise testing is an important tool in the field of cardiology and is very commonly used because it is readily available, inexpensive, noninvasive, and provides pertinent diagnostic and prognostic information in assessing for the presence and severity of coronary artery disease (CAD). For decades, its primary use was to provoke and diagnose myocardial ischemia, but its clinical indications have become more numerous with time. In this chapter, we will review role of treadmill exercise testing in patients with known or suspected CAD, as well as the contraindications, complications, performance, interpretation, and its prognostics utility.
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39

1946-, Feuerstein Giora Z., ed. Inflammation and stroke. Birkhäuser, 2001.

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40

Patel, Mikin V., and Steven Zangan. Optimizing Carbon Dioxide Peripheral Arteriography. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0103.

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Angiography relies on the use of contrast medium for visualization of the vessel. Iodinated contrast can be contraindicated in patients with renal impairment or iodinated contrast allergy, so carbon dioxide (CO2) gas can be a useful alternative. A number of technical and postural parameters can optimize CO2 angiography, and vasodilators can be used to improve imaging of peripheral vessels. Although CO2 has distinct advantages, the limitations of CO2 angiography must be well understood. Operators should be aware that CO2 angiography can lead to overestimation of vessel size and can lead to complications such as transient ischemia of tissues, alterations of blood chemistry, neurotoxicity, and “vapor lock.”
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41

Waberski, Andrew T., and Nina Deutsch. Transposition of the Great Arteries. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0010.

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Transposition of the great arteries is a congenital cardiac abnormality that presents in the neonatal period, most commonly as cyanosis. While variations in anatomic features exist, dextro-transposition of the great arteries, the most common form, results in 2 separate circulatory systems in parallel, such that the right ventricle pumps deoxygenated blood to the systemic circulation, and the left ventricle sends oxygenated blood back to the pulmonary circulation. To ensure survival, early diagnosis and intervention to allow for adequate mixing of blood is necessary. The arterial switch operation is the definitive treatment, usually undertaken in the first few days of life. Known complications of surgery include ischemia, bleeding, hemodynamic compromise, and arrhythmias. Anesthetic management must take these factors into account.
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42

Chong, Ji Y., and Michael P. Lerario. Fevers in a Patient with Valve Replacement. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0020.

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Infective endocarditis can cause neurological complications, including stroke, hemorrhage, and mycotic aneurysms. Patients with endocarditis and neurological complications need vascular imaging to evaluate for aneurysm formation. Patients with acute ischemic stroke from septic emboli may not respond to conventional intravenous thrombolysis and have higher rates of hemorrhagic complications.
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43

Steinberg, Alexis, and Bradley J. Molyneaux. Acute Stroke (DRAFT). Edited by Raghavan Murugan and Joseph M. Darby. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190612474.003.0019.

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The development of a stroke is an acute neurologic emergency that requires rapid evaluation as any delay in treatment worsens outcome. There are two main types of strokes, hemorrhagic and ischemic, each requiring specific rapid assessment and interventions. If an acute ischemic stroke is suspected, then a decision regarding thrombolytic therapy and endovascular thrombectomy has to be made quickly. A hemorrhagic stroke demands rapid medical management of blood pressure, reversal of coagulopathy, and early neurosurgical consult for possible external ventricular drain (EVD) placement and hemorrhage evacuation. This chapter expands on the indicated work-up in a suspected stroke patient in the setting of the rapid response team (RRT) calls, different imaging modalities, management options in the acute and subacute periods, and post-stroke complications.
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44

Chong, Ji Y., and Michael P. Lerario. Treatment of Acute Right-Sided Weakness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0001.

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Intravenous tissue plasminogen activator (IV tPA) is the mainstay of stroke therapy and is US Food and Drug Administration-approved for the treatment of acute ischemic stroke. Its benefit on functional outcome has been established in multiple randomized trials when administered within 3 hours. Select patients may be treated off-label up to 4.5 hours from symptom onset. Eligibility criteria need to be reviewed carefully to optimize benefit and to minimize complications, namely reperfusion hemorrhage.
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45

Chong, Ji Y., and Michael P. Lerario. Puff of Smoke. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0023.

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Moyamoya is a rare cause of stroke caused by progressive distal carotid occlusion. Children and adults may be affected, but it is more common in Asian populations, particularly in patients presenting with intracranial hemorrhage. Angiographic findings of distal internal carotid artery stenosis with lenticulostriate or choroidal neocollaterals are important in the diagnosis of moyamoya disease. A similar pathophysiological state to moyamoya disease is associated with several clinical conditions, some of which are acquired and some of which have a genetic etiology. Treatment is with surgical revascularization, which can reduce both ischemic and hemorrhagic complications.
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46

Dunn, Lauren E., Joshua Z. Willey, and Ronald M. Lazar. Neuroprotection for Mechanical Circulatory Support. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0012.

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This chapter examines the adverse neurological events associated with mechanical circulatory support (MCS), which includes left ventricular assist devices (LVAD) and percutaneous devices for cardiac disease. The most frequently encountered neurological events are ischemic and hemorrhagic stroke, as well as intracerebral hemorrhage (ICH), heart failure and cardiovascular disease. The management of acute cerebrovascular conditions in this population poses unique challenges, given concomitant anticoagulation usage, hemodynamically unstable patients, and lack of randomized controlled trials investigating these clinical scenarios. Other acute neurological events include cerebral hyperperfusion and cerebral air embolism. This chapter describes these complications and their risk factors, and the available evidence-based and institutional management strategies.
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47

Traul, David E. Postoperative Visual Loss in Spine Surgery. Edited by David E. Traul and Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0026.

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Postoperative visual loss (POVL) is a rare but devastating condition associated with many types of nonocular surgery. In spine surgery, the most common causes of POVL are ischemic optic neuropathy (ION), central retinal artery occlusion (CRAO), and cortical blindness. Although the association of POVL with spine surgery has long been recognized, the low incidence of this complication hinders the identification of patient and perioperative risk factors and limits our understanding of the causes of POVL. In adult spine surgery, POVL is most frequently attributed to ION whereas CRAO is more commonly seen in cardiac procedures. POVL due to cortical blindness has the highest incidence in pediatric spine surgery. While several risk factors for POVL have been identified in spine surgery, there are currently no standardized practice guidelines to eliminate the risk for POVL. Currently, there are no effective treatments for POVL, and recovery from ION and CRAO is limited.
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48

AlJaroudi, Wael. Risk Assessment Before Noncardiac Surgery. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0014.

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Perioperative risk assessment is essential in screening patients before noncardiac surgery. Cardiovascular complications such as fatal and non-fatal myocardial infarction (MI), ventricular arrhythmia, pulmonary edema, and stroke are important in-hospital causes of morbidity and mortality intra and post-operatively. The optimal approach is to identify patients at increased risk so that appropriate testing and therapeutic interventions are undertaken a priori to minimize such risk. The initial preoperative evaluation includes identification of surgery-specific risk, patient exercise functional capacity and clinical risk profile. Patients with major predictors of events such as acute coronary syndromes, recent MI, unstable arrhythmia, and severe valvular disease warrant further management and optimization that often lead to delaying surgery. Those with three or more predictors (history of ischemic heart disease, compensated heart failure, diabetes, renal insufficiency, or history of cerebrovascular disease) undergoing high- risk surgery often require stress testing. Although data from randomized prospective trials are lacking, numerous studies have demonstrated the utility of myocardial perfusion imaging (MPI) for determination of perioperative cardiac risk. The goal of this chapter is to review the use of MPI for preoperative risk assessment and the recommendations from the current guidelines. The focus will be on short-term and long-term prognosis including special groups such as after coronary stenting and before vascular surgery, liver and renal transplantation.
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49

L, Sutton Amy, ed. Stroke sourcebook: Basic consumer health information about stroke, including ischemic, hemorrhagic, and mini strokes, as well as risk factors, prevention guidelines, diagnostic tests, medications and surgical treatments, and complications of stroke ; along with rehabilitation techniques and innovations, tips on staying healthy and maintaining independence after stroke, a glossary of related terms, and a directory of resources for stroke survivors and their families. 2nd ed. Omnigraphics, 2008.

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