Academic literature on the topic 'Ischemic nephropathy'

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Journal articles on the topic "Ischemic nephropathy"

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Fergany, Amr F. "Ischemic nephropathy." Urologic Clinics of North America 28, no. 4 (2001): 805–13. http://dx.doi.org/10.1016/s0094-0143(01)80035-5.

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Nobert, Craig F., and John A. Libertino. "Ischemic nephropathy." Current Opinion in Urology 8, no. 2 (1998): 129–34. http://dx.doi.org/10.1097/00042307-199803000-00011.

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Breyer, Julia A., and Harry R. Jacobson. "Ischemic nephropathy." Current Opinion in Nephrology and Hypertension 2, no. 2 (1993): 216–24. http://dx.doi.org/10.1097/00041552-199303000-00007.

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Tuttle, Katherine R. "Ischemic nephropathy." Current Opinion in Nephrology and Hypertension 10, no. 2 (2001): 167–73. http://dx.doi.org/10.1097/00041552-200103000-00003.

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Kaul, Anupama, and Harsh Vardhan. "Ischemic nephropathy." Clinical Queries: Nephrology 1, no. 4 (2012): 268–78. http://dx.doi.org/10.1016/j.cqn.2012.10.001.

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Chaturvedy, Manish, and Kirti Rana. "Ischemic nephropathy." Clinical Queries: Nephrology 2, no. 2 (2013): 84–90. http://dx.doi.org/10.1016/j.cqn.2013.04.002.

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Novick, Andrew C. "ATHEROSCLEROTIC ISCHEMIC NEPHROPATHY." Urologic Clinics of North America 21, no. 2 (1994): 195–200. http://dx.doi.org/10.1016/s0094-0143(21)00937-x.

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Luo, Fengbao, Jian Shi, Qianqian Shi, Xianlin Xu, Ying Xia, and Xiaozhou He. "Mitogen-Activated Protein Kinases and Hypoxic/Ischemic Nephropathy." Cellular Physiology and Biochemistry 39, no. 3 (2016): 1051–67. http://dx.doi.org/10.1159/000447812.

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Tissue hypoxia/ischemia is a pathological feature of many human disorders including stroke, myocardial infarction, hypoxic/ischemic nephropathy, as well as cancer. In the kidney, the combination of limited oxygen supply to the tissues and high oxygen demand is considered the main reason for the susceptibility of the kidney to hypoxic/ischemic injury. In recent years, increasing evidence has indicated that a reduction in renal oxygen tension/blood supply plays an important role in acute kidney injury, chronic kidney disease, and renal tumorigenesis. However, the underlying signaling mechanisms,
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Lerman, Lilach, and Stephen C. Textor. "Pathophysiology of ischemic nephropathy." Urologic Clinics of North America 28, no. 4 (2001): 793–803. http://dx.doi.org/10.1016/s0094-0143(01)80034-3.

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Alcázar, José, and José Rodicio. "Hypertension and ischemic nephropathy." Current Hypertension Reports 2, no. 4 (2000): 343–44. http://dx.doi.org/10.1007/s11906-000-0034-y.

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Dissertations / Theses on the topic "Ischemic nephropathy"

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Лобода, Андрій Миколайович, Андрей Николаевич Лобода, Andrii Mykolaiovych Loboda та ін. "Цистатин як ранній маркер ішемічної нефропатії у новонароджених". Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27442.

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У дітей з ІН, що виникла внаслідок асфіксії, рівень цистатину С достовірно зростає вже на 1-2 добу життя, досягає максимуму до кінця раннього неонатального періоду та зберігається на високому рівні навіть наприкінці 1-го місяця життя, що свідчить про збереження суттєвих змін клубочкової фільтрації, не зважаючи на стабілізацію основних показників життєдіяльності. Тому всіх пацієнтів, що мали прояви ІН в неонатальному періоді, не залежно від важкості асфіксії, що спричинила розвиток ниркової дисфункції, слід розглядати як групу ризику з формування патології нирок в подальшому. При цитуванні доку
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Лобода, Андрій Миколайович, О. І. Кірой, М. Л. Радченко та ін. "Епідеміологія ішемічної нефропатії у доношених новонароджених Сумської області". Thesis, Видавництво СумДУ, 2011. http://essuir.sumdu.edu.ua/handle/123456789/15805.

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Лобода, Андрій Миколайович, Андрей Николаевич Лобода, Andrii Mykolaiovych Loboda та ін. "Вміст макроелементів у сироватці крові доношених новонароджених з ішемічною нефропатією". Thesis, Видавництво СумДУ, 2011. http://essuir.sumdu.edu.ua/handle/123456789/15920.

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Бабій, О. "Діагностичне значення вмісту прозапальних цитокінів у новонароджених з ішемічною нефропатією". Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27470.

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Ouyang, Nengtai. "Effects of TGF-[beta]1 [TGF-beta-1] in ischemia, reperfusion injury and chronic allograft nephropathy." [S.l.] : [s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=972198628.

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Lui, Sing-leung, and 雷聲亮. "Therapeutic potential of rapamycin in renal parenchymal diseases: insights from murine models of lupusnephritis, adriamycin nephropathy and renal ischemia reperfusioninjury." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41291013.

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Lui, Sing-leung. "Therapeutic potential of rapamycin in renal parenchymal diseases insights from murine models of lupus nephritis, adriamycin nephropathy and renal ischemia reperfusion injury /." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B41291013.

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Ouyang, Nengtai [Verfasser]. "Effects of TGF-β1 [TGF-beta-1] in ischemia, reperfusion injury and chronic allograft nephropathy / Nengtai Ouyang". 2004. http://d-nb.info/972198628/34.

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Book chapters on the topic "Ischemic nephropathy"

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Granata, Antonio, Elnaz Rahbari, Dario Galeano, and Pasquale Fatuzzo. "Ischemic Nephropathy." In Atlas of Ultrasonography in Urology, Andrology, and Nephrology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-40782-1_3.

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Gartler, Stanley M., R. Scott Hansen, Vinzenz Oji, et al. "Ischemic Nephropathy." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6713.

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Granata, Antonio, Leonardo Spatola, Salvatore Granata, et al. "Ischemic Nephropathy." In Atlas of Ultrasonography in Urology, Andrology, and Nephrology. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-78135-3_3.

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von Morze, Cornelius, Galen D. Reed, Zhen J. Wang, Michael A. Ohliger, and Christoffer Laustsen. "Hyperpolarized Carbon (13C) MRI of the Kidneys: Basic Concept." In Methods in Molecular Biology. Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-0978-1_16.

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AbstractExisting clinical markers for renal disease are limited. Hyperpolarized (HP) 13C MRI is based on the technology of dissolution dynamic nuclear polarization (DNP) and provides new avenues for imaging kidney structure, function, and most notably, renal metabolism, addressing some of these prior limitations. Changes in kidney structure and function associated with kidney disease can be evaluated using [13C]urea, a metabolically inert tracer. Metabolic changes can be assessed using [1-13C]pyruvate and a range of other rapidly metabolized small molecules, which mainly probe central carbon m
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Textor, Stephen C. "Renovascular Hypertension and Ischemic Nephropathy." In Hypertension: A Companion to Braunwald's Heart Disease. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-42973-3.00013-5.

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Textor, Stephen C. "Renovascular Hypertension and Ischemic Nephropathy." In Brenner and Rector's The Kidney. Elsevier, 2011. http://dx.doi.org/10.1016/b978-1-4160-6193-9.10047-8.

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Textor, Stephen C., and Sandra J. Taler. "Renovascular Hypertension and Ischemic Nephropathy." In Hypertension. Elsevier, 2024. http://dx.doi.org/10.1016/b978-0-323-88369-6.00013-x.

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"Renovascular Hypertension and Ischemic Nephropathy." In Pocket Companion to Brenner and Rector's The Kidney. Elsevier, 2011. http://dx.doi.org/10.1016/b978-1-4160-6640-8.00023-3.

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Fergany, Amr, and Andrew C. Novick. "Renovascular Hypertension and Ischemic Nephropathy." In Campbell-Walsh Urology. Elsevier, 2012. http://dx.doi.org/10.1016/b978-1-4160-6911-9.00039-6.

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Textor, Stephen C. "Renovascular Hypertension and Ischemic Nephropathy." In Brenner and Rector's The Kidney. Elsevier, 2008. https://doi.org/10.1016/b978-1-4160-3105-5.50045-1.

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