Relevant bibliographies by topics / Isradipine and Quality Control

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Academic literature on the topic 'Isradipine and Quality Control'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Isradipine and Quality Control"

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Chrysant, Steven G., and Michael Cohen. "Sustained Blood Pressure Control with Controlled-Release Isradipine (Isradipine-CR)." Journal of Clinical Pharmacology 35, no. 3 (March 1995): 239–43. http://dx.doi.org/10.1002/j.1552-4604.1995.tb04053.x.

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CHRYSANT, S., and M. COHEN. "Sustained blood pressure control with controlled-release isradipine." American Journal of Hypertension 8, no. 1 (January 1995): 87–89. http://dx.doi.org/10.1016/0895-7061(94)00158-8.

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Johnson, Cary E., Pamala A. Jacobson, and Min H. Song. "Isradipine Therapy in Hypertensive Pediatric Patients." Annals of Pharmacotherapy 31, no. 6 (June 1997): 704–7. http://dx.doi.org/10.1177/106002809703100606.

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OBJECTIVE: To evaluate the dosage and effectiveness of isradipine to control acute or chronic hypertension in pediatric patients. DESIGN: Retrospective medical record review. SETTING: University teaching hospital. PARTICIPANTS: Hospitalized pediatric patients aged 1 day to 16 years with hypertension treated with isradipine between January 1994 and March 1996. MEASURES: Patient age, gender, weight, disease states, current medications, isradipine dosage and formulation, pre- and postsystolic, and pre- and postdiastolic blood pressure measurements with each dose of isradipine. RESULTS: Fifty-three patients with a mean age of 5.8 ± 4.0 years were evaluated. A mean change in the blood pressure measurements taken before the first dose of isradipine compared with the values recorded after the last dose or at discharge for all patients was −11.8% ± 12.5% and −17.4% ± 19.6%, respectively, for systolic and diastolic pressure. The mean dosage of isradipine in 46 patients who received regularly scheduled doses was 0.38 ± 0.22 mg/kg/d. Patients who demonstrated a response received a mean dosage of 0.40 ± 0.20 mg/kg/d. The total daily dosage was administered in one dose for 1 patient, two doses for 15 patients, three doses for 27 patients, and four doses for 3 patients. CONCLUSIONS: Isradipine was an effective antihypertensive agent to reduce the systolic and/or diastolic blood pressure 10% or more compared with pretreatment measurements in 43 (81 %) of 53 pediatric patients. The mean dosage was 0.38 ± 0.22 mg/kg/d, most frequently administered in two or three equally divided doses, which is higher than the normal recommended dosage for adults.
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KANTOLA, I. "Subjective sleep quality during two different antihypertensive medications: isradipine vs metoprolol." American Journal of Hypertension 12, no. 4 (April 1999): 117. http://dx.doi.org/10.1016/s0895-7061(99)80408-7.

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Jacques, Danielle, Ghassan Bkaily, Gaétan Jasmin, Pedro D'Orléans-Juste, and Mirna Chahine. "Isradipine prevents the development of spontaneously occurring cardiac necrosis in cardiomyopathic hamster." Canadian Journal of Physiology and Pharmacology 81, no. 2 (February 1, 2003): 120–24. http://dx.doi.org/10.1139/y03-021.

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Recent studies on the heart necrotizing process at the early stages of hamster polymyopathy have led us to believe that this hereditary disease derives from an anomalous transmembrane ion flux due to the presence of slow Na+ channels that contribute to intracellular Na+ accumulation which promote intracellular Ca2+ overload via the Ca2+ influx through the Na+–Ca2+ exchanger. In the present study, we investigated the potential beneficial effect of chronic treatment with a dual L-type Ca2+ and slow Na+ channel blockers isradipine, on the development of necrosis in myopathic hamster hearts. Young cardiomyopathic (CM) hamsters (CMH) were treated with isradipine (0.1 mg·kg–1·day–1) and nifedipine (1 mg·kg–1·day–1) for 4 consecutive weeks. Microscopic assessments were carried out in staged serial paraffin sections of heart ventricles from tissues freshly dissected at autopsy. In comparison with control nontreated hearts, which exhibited numerous necrotic calcific foci, myolytic lesions, and dilated right ventricle, isradipine treatment prevented, in a significant manner, all the above spontaneous pathological changes, while nifedipine had no effect. Our present observations provide evidence for the first time that in vivo treatment with a DHP Ca+ channel blocker, isradipine, is cardioprotective against the development of necrosis in hereditary cardiomyopathy in the hamster. It is possible that the protective effect of isradipine in CMH could be largely due to the indirect blockade of Ca2+ influx through the Na+–Ca2+ exchanger as well as to possible direct blockade of Ca2+ influx through the T-type Ca2+ channel.Key words: isradipine, cardioprotection, hamster cardiomyopathy, slow Na+ channel.
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Pǐtre, Maryse, Nathalie Gaudreault, Marta Santuré, André Nadeau, and Hélène Bachelard. "Isradipine and insulin sensitivity in hypertensive rats." American Journal of Physiology-Endocrinology and Metabolism 276, no. 6 (June 1, 1999): E1038—E1048. http://dx.doi.org/10.1152/ajpendo.1999.276.6.e1038.

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The present study was designed to investigate the effect of a reduction in blood pressure, by using the calcium channel antagonist isradipine, on insulin sensitivity and vascular responses to insulin in conscious spontaneously hypertensive male rats (SHR). The rats were instrumented with intravascular catheters and pulsed Doppler flow probes to measure blood pressure, heart rate, and blood flows. Insulin sensitivity was assessed by the euglycemic-hyperinsulinemic clamp technique. Two groups of rats received isradipine at a dose of 0.05 or 0.15 mg ⋅ kg−1 ⋅ h−1, whereas a third group received a continuous infusion of vehicle (15% DMSO). Both doses of isradipine were found to decrease mean blood pressure (−25 ± 4 mmHg at the dose of 0.05 mg ⋅ kg−1 ⋅ h−1and −20 ± 2 mmHg at the dose of 0.15 mg ⋅ kg−1 ⋅ h−1) and to improve insulin sensitivity. Moreover, in the rats treated with the low dose of isradipine, we observed vasodilations in renal, superior mesenteric, and hindquarter vascular beds. In the untreated group, the euglycemic infusion of insulin (4 mU ⋅ kg−1 ⋅ min−1) was found to cause vasoconstrictions in superior mesenteric and hindquarter vascular beds, but no changes in mean blood pressure, heart rate, or renal vascular conductance were found. In contrast, in the isradipine-treated groups, the same dose of insulin was found to produce vasodilations in the renal vascular bed and to abolish the vasoconstrictor responses previously observed. We concluded that short-term treatment with isradipine in SHR can lower blood pressure and improve insulin sensitivity, mainly through hemodynamic factors, as supported by experiments with hydralazine as a positive vasodilator control.
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&NA;. "Isradipine does not affect platelet aggregation or metabolic control in NIDDM." Inpharma Weekly &NA;, no. 742 (June 1990): 17. http://dx.doi.org/10.2165/00128413-199007420-00043.

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Wegener, Jörg W., Matthias Lee, and Franz Hofmann. "Hypothyroidism does not affect the dihydropyridine sensitivity of precontracted murine uterus." Canadian Journal of Physiology and Pharmacology 81, no. 9 (September 1, 2003): 890–93. http://dx.doi.org/10.1139/y03-090.

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Thyroid hormones are known to influence various processes of cell differentiation. Recently, it was reported that hypothyroidism reduces the sensitivity to Ca2+-channel antagonists in the rat uterus. We examined the sensitivity to dihydropyridines of the uterus from mice that had reduced thyroid hormone levels. Isradipine relaxed with the same potency precontracted uterine muscle strips from control and hypothyroid mice, independently from a pseudo-pregnant state. These results demonstrate that hypothyroidism does not change dihydropyridine sensitivity (i.e., the pattern of Ca2+-channel expression) in the murine uterus.Key words: uterus, smooth muscle, Ca2+ channel, isradipine.
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Darcie, Silvana, Cléa R. Leone, Valdenise M. L. T. Calil, Elizete P. Prescinotti, Soubhi Kahhale, and Marcelo Zugaib. "Glycemia in newborns of hypertensive mothers according to maternal treatment." Revista do Hospital das Clínicas 59, no. 5 (2004): 244–50. http://dx.doi.org/10.1590/s0041-87812004000500004.

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PURPOSE: To evaluate the evolution of glycemic levels in newborns of hypertensive mothers according to maternal treatment. METHODS: Prospective randomized study, including 93 newborns of mothers treated with isradipine (n = 39), atenolol (n = 40), or low sodium diet (control group - n=14). Glycemia was determined at birth (mother and newborn by the oxidase glucose method) and in the 1st, 3rd, 6th, 12th, and 24th hours after birth (newborn by a test strip method). The evolution of glycemia was analyzed in each group (Friedman test). The groups were compared regarding glycemia (Kruskall-Wallis test), and linear regression models were constructed for the analyses (independent variable = maternal glycemia; dependent variables = umbilical cord, 3rd, and 6th hour glycemia). RESULTS: There were no statistically significant differences among the mean blood glucose levels of the 3 groups in any of the assessments. There was a correlation between maternal and umbilical cord blood glucose in the isradipine (r = 0.61; P <.05) and control (r = 0.84; P <.05) groups. Regarding glycemia levels of the mothers and newborns in the third and sixthhours postpartum, this correlation was present only in the control group (maternal x third hour: r = 0.65; P <.05; maternal x sixth hour: r = 0.68; P <.05). There were no correlations in the atenolol group. Hypoglycemia was detected in 51.3% of the isradipine group, 60% of the atenolol group, and 35.7% of the control group, and it was more frequent in the first hour postpartum in all groups. CONCLUSIONS: The results suggest a similar effect of the 3 types of treatment upon newborn glycemia. The correlation analysis suggests that isradipine could have effects upon newborn glycemia only after birth (correlation only in umbilical cord blood), whereas atenolol could act earlier (there was no correlation at any moment). The results also point to the need for glycemic control from the first hour postpartum of newborns of hypertensive mothers whether they have or have not undergone treatment with antihypertensive drugs.
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Viskoper, J. R., A. Laszt, and D. Faraggi. "Twenty-Four-Hour Blood Pressure Control With Isradipine in Mild Essential Hypertension." American Journal of Hypertension 4, no. 2_Pt_2 (February 1991): 161S—162S. http://dx.doi.org/10.1093/ajh/4.2.161s.

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Dissertations / Theses on the topic "Isradipine and Quality Control"

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Aguiar, Fernando Armani. "Aplicações da eletroforese capilar na análise do biomarcadir alfa-1 glicoproteína ácida, no controle de qualidade do biofármaco interferon alfa 2a e na avaliação da estabilidade enantiosseletiva do fármaco isradipina." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/60/60137/tde-22102013-160148/.

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A eletroforese é uma técnica de separação que se baseia na migração diferencial de compostos iônicos em tubo capilar semicondutor, preenchido com solução eletrolítica, sob a influência de campo elétrico. Na introdução desta tese, princípios, métodos e diferentes tipos de técnicas de eletromigração em capilar foram discutidos. No primeiro capítulo são mostrados os resultados de otimização e validação de um método eletroforético para a determinação das glicoformas da ?1-Glicoproteína Ácida, um biomarcador. A otimização das condições eletroforéticas usando eletrólito de corrida constituído por tricina (10 mmol L-1), cloreto de sódio (10 mmol L-1), acetato de sódio (10 mmol L-1), ureia (7 mol L-1) e putrescina (3,9 mmol L-1), pH de 4,5, tensão de 30 kV, e temperatura de análise de 35 °C levou à resolução mínima de aproximadamente 1,5 entre as oito glicoformas encontradas. Todas as análises foram realizadas em um capilar de sílica fundida não revestido internamente, diâmetro interno de 50 µm e comprimento efetivo de 50,0 centímetros. Após a otimização, o método foi validado, em que a linearidade foi obtida no intervalo de 0,125 a 2,5 mg mL-1 (r >= 0,993). O coeficiente de variação (%) e erros relativos (%) obtidos nos estudos de precisão e exatidão, respectivamente, intra e inter-dias foram inferiores a 15 %. Após a validação o método foi aplicado para a análise da ?1-AGP em amostras de plasma de pacientes com sepse, o qual demonstrou uma variabilidade na concentração da glicoformas. No segundo capítulo, um método simples, rápido e econômico por eletroforese capilar, foi desenvolvido e validado para a determinação de Interferon alfa-2a, um biofármaco, em formulação farmacêutica. Após otimização, os melhores resultados foram obtidos utilizando solução tampão tetraborato de sódio 30 mmol L-1, e pH 8,50, com 50 mmol L-1 de dodecil sulfato de sódio. A tensão aplicada foi de 25 kV e a injeção da amostra foi realizada no modo hidrodinâmico. Todas as análises foram realizadas em capilar de sílica fundida não revestido internamente, diâmetro interno de 75 µm e comprimento efetivo de 50,0 centímetros. Sob estas condições, a análise foi realizada em menos de 10 min. A linearidade foi obtida no intervalo de 0,41-1,54 MUI mL-1 (r >= 0,997). O coeficiente de variação (%) e erros relativos (%) obtidos nos estudos de precisão e exatidão, respectivamente, intra e inter-dias foram inferiores a 5 %. Após a validação o método foi aplicado no controle de qualidade de formulações farmacêuticas contendo o Interferon alfa-2a. No terceiro capítulo, um método enantiosseletivo simples por eletroforese capilar usando ciclodrextrina como seletor quiral foi desenvolvido e validado para a determinação dos enantiômeros da isradipina, um bloqueador de canal de cálcio, em formulação farmacêutica. Além disso, foi realizado estudo de estabilidade dos enantiômeros da isradipina submetidos à oxidação, hidrólise (ácida e alcalina) e fotólise. A resolução completa dos enantiômeros da isradipina foi obtida em menos de 7 minutos utilizando solução tampão borato de sódio 15 mmol L-1 e pH 9,3 e sulfobutil éter-?-ciclodextrina (2,5 %, m/v) como seletor quiral. A tensão aplicada foi de 30 kV, e a injeção da amostra foi realizada no modo hidrodinâmico. Todas as análises foram efetuadas em capilar de sílica fundida não revestido internamente e diâmetro interno de 50 µm e comprimento efetivo de 50 centímetros. A linearidade foi obtida no intervalo de 25 - 150 µg mL-1 para ambos enantiômeros (r >= 0,998). O coeficiente de variação (%) e erros relativos (%) obtidos nos estudos de precisão e exatidão, respectivamente, intra e inter-dias foram inferiores a 5 %. Após o método ter sido validado, este foi aplicado na análise de formulações farmacêuticas contendo os enantiômeros da isradipina. Nos estudos de estabilidade foi observada degradação dos enantiômeros em todas as condições avaliadas. Assim, de acordo com os resultados obtidos após o desenvolvimento dos três métodos, pode ser concluido que a eletroforese capilar é uma poderosa técnica de separação com aplicações na investigação, desenvolvimento, controle de qualidade e estudos de estabilidade de produtos farmacêuticos. Além disso, a eletroforese capilar é uma técnica complementar à cromatografia líquida de alta eficiência, que oferece vantagens como simplicidade, rapidez, baixo custo e consumo de solventes e reagentes e diferentes mecanismos de seletividade, podendo ser aplicada em diferentes tipos de amostras.
Electrophoresis is a separation technique that is based on the differential migration of charged compounds in a semi-conductive medium under the influence of an electric field. In the introduction of this thesis, principles, methods, and different types of electromigrations techniques in capillary were discussed. In the first chapter shows the results of optimization and validation of a electrophoretic method for determining the glycoforms of ?1-AGP. The running buffer after optimization consisted of Tricine (10 mmol L-1), sodium chloride (10 mmol L-1), sodium acetate (10 mmol L-1), urea (7 mol L-1) and putrescine (3.9 mmol L-1), pH 4.5, voltage (30 kV), temperature and analysis (35 ° C) led to resolution of at least of 1.5 among the eight glycoforms found. All analyses were carried out in a fused-silica uncoated capillary with an id of 50 ?m and effective length of 50.0 cm. After optimization method was validated in which the linearity was obtained in the range to 0.125 to 2.5 mg mL-1 (r >= 0.993). The coefficient of variation (%) and relative errors (%) obtained in the studies of precision and accuracy, respectively (intra-day and inter-day) were less than 15 %. After method validation, the analysis of ?1-AGP in plasma of septic patients was performed, which showed variability in the concentration of glycoforms. In the second chapter a simple CE based method was developed and validated for the determination of Interferon alpha-2a in a pharmaceutical formulation. After optimization, the best results were obtained using 30 mmol L-1 tetraborate buffer at pH 8.50 with 50 mmol L-1 of sodium dodecyl sulfate. The applied voltage was 25 kV, and the sample injection was performed in the hydrodynamic mode. All analyses were carried out in a fused-silica uncoated capillary with an id of 75 ?m and effective length of 50.0 cm. Under these conditions, the analysis was achieved in less than 10 min. Linearity was obtained in the range 0.41-1.54 MIU mL-1 (r >= 0.997). The RSD (%) and relative errors (%) obtained in precision and accuracy studies (intra-day and inter-day) were lower than 5 %. Therefore, this method was found to be appropriate for controlling pharmaceutical formulations containing Interferon alpha-2a. In the third chapter a simple enantioselective method based on CE using CD as chiral selector was developed and validated for the determination of isradipine (IRD) enantiomers in a pharmaceutical formulation and for the determination of IRD enantiomers in degradation studies. After optimization, the best results were obtained using 15 mmol L-1 borate buffer at pH 9.3 and sulfobutyl ether-?-cyclodextrin (SBE-?-CD) (2.5 %, w/v) as chiral selector. The applied voltage was 30 kV, and the sample injection was performed in the hydrodynamic mode. All analyses were carried out in a fused-silica uncoated capillary with an internal diameter of 50 ?m and effective length of 50 cm. Under these conditions, a complete separation between IRD enantiomers was achieved in less than 7 min. Linearity was obtained in the range 25 - 150 ?g mL-1 for both enantiomers (r >= 0.998). The RSD (%) and relative errors (%) obtained in precision and accuracy studies (intra-day and inter-day) were lower than 5 %. Therefore, this method was found to be appropriate for controlling pharmaceutical formulations containing IRD enantiomers and the assay was considered stability indicating. The drug was subjected to oxidation, hydrolysis and photolysis. In all stress conditions the drug presented considerable degradation. According to such results, the capillary electrophoresis showed is a powerful separation technique to research and development, quality control, and stability studies of pharmaceuticals. CE offers several advantages over high-performance liquid chromatography (HPLC), a technique commonly used in pharmaceutical analysis. These include simplicity, rapid analysis, automation, different mechanisms for selectivity, and low cost.
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Jin, Ye. "Quality control of phytopharmaceuticals : assessment and quality control of traditional Chinese medicine." Thesis, Liverpool John Moores University, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.327675.

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Bush, Helen Meyers. "Nonparametric multivariate quality control." Diss., Georgia Institute of Technology, 1996. http://hdl.handle.net/1853/25571.

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Sepúlveda, Ariel. "The Minimax control chart for multivariate quality control." Diss., Virginia Tech, 1996. http://hdl.handle.net/10919/30230.

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Crossman, S. H. "Quality control in developing epithelia." Thesis, University College London (University of London), 2018. http://discovery.ucl.ac.uk/10044689/.

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In the fruit-fly Drosophila melanogaster, extensive apoptosis is observed throughout the embryonic epidermis upon the mutation of many essential patterning genes. The molecular basis of cell elimination in this context is poorly understood, although previous studies have suggested the existence of a cell-autonomous quality control mechanism, which detects cells unable to adopt an appropriate terminal fate and removes them through apoptosis. This hypothetical system is thought to protect against patterning errors in order to preserve the integrity of the developing epidermis. To identify factors required for apoptosis in mis-patterned cells, I performed a targeted genetic screen, which identified a potential role for the EGFR signalling pathway in this process. Excess EGFR signalling was shown to rescue the cell death phenotype of the archetypal patterning mutant fushi tarazu (ftz), whilst EGFR null alleles triggered extensive epidermal apoptosis. Upon further experimentation, I was able to show that patterning mutant embryos fail to express the major EGFR activating ligands in the correct spatial pattern. This causes local troughs in EGFR signalling, which trigger transcriptional upregulation of the pro-apoptotic gene hid and subsequent cell death. These results argue against a cell-autonomous mechanism of cell elimination in mis-patterned embryos and instead suggest that the tissue-wide landscape of EGFR activity is responsible for coordinating cell fate and cell survival in the embryonic epidermis. Building on these observations, I have been able to show that the EGFR pathway also regulates apoptosis during normal development, where it specifies the maximum dimensions of embryonic segments. Taken together, these findings provide a novel link between early patterning events, cell viability and compartment size in the developing Drosophila embryo.
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Gordon, Kara Leigh. "TorsinA and protein quality control." Diss., University of Iowa, 2011. https://ir.uiowa.edu/etd/2708.

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DYT1 dystonia (DYT1) is a disabling inherited neurological disorder with juvenile onset. The genetic mutation in DYT1 leads to the deletion of a glutamic acid (E) residue in the protein torsinA. The function of torsinA and how the mutation leads to DYT1 is poorly understood. We hypothesize that how efficiently the disease-linked mutant protein is cleared may be critical for DYT1 pathogenesis. Therefore we explored mechanisms of torsinA catabolism, employing biochemical, cellular, and animal-based approaches. We asked if torsinA(wt) and torsinA(DE) are degraded preferentially through different catabolic mechanisms, specifically the ubiquitin proteasome pathway (UPP) and autophagy. We determined that torsinA(wt) is cleared by autophagy while torsinA(DE) is efficiently degraded by the UPP suggesting degradation processes can modulate torsinA(DE) levels. Proteins implicated in recognizing motifs on torsinA(DE) for targeting to the UPP represent candidate proteins that may modify DYT1 pathogenesis. We examined how removal of the hydrophobic domain and mutation of glycosylated asparagine residues on torsinA altered stability and catabolic mechanism. We found the glycosylation sites on torsinA are important for stability modulate its degradation through the UPP. F-box G-domain protein 1 (FBG1) has been implicated in degradation of glycosylated ER proteins. We hypothesized that FBG1 would promote torsinA degradation and demonstrated that FBG1 modulates levels of torsinA in a non-canonical manner through the UPP and autophagy. We examined if lack of FBG1 in a torsinA(DE) mouse model altered motor phenotypes. We saw no effect which suggests FBG1 does not alter DYT1 pathogenesis despite its promotion of torsinA(DE) degradation. In addition, we explored a potential mechanism for the previously described role of torsinA in modulating cytoplasmic protein aggregation. We hypothesized this endoplasmic reticulum (ER) resident protein would indirectly alter cytoplasmic protein aggregation through modulation of ER stress. We employed a poly-glutamine expanded repeat protein and pharmacological ER stressors to determine that torsinA does not alter poly-glutamine protein aggregation nor ER stress in a mammalian system. In summary, this thesis suggests proteins involved in the catabolism of torsinA(DE) may modify DYT1 pathogenesis and that torsinA and its DYT1-linked mutant are model proteins for investigating ER protein degradation by the UPP and autophagy.
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Hughes, Anthony. "Quality control in radionuclide imaging." Thesis, University of Aberdeen, 1990. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU601994.

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Lennard, Nicola S. "Quality control for carotid endarterectomy." Thesis, University of Leicester, 2004. http://hdl.handle.net/2381/29469.

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The aims of this study are to assess whether the introduction of a rigorous quality control method could produce a sustained reduction in the intraoperative stroke rate in this unit and whether it was feasible and practical to implement such a programme. The second part of this study will assess the incidence of sustained embolisation in the early post-operative period and investigate whether the antiplatelet agent Dextran 40 can help stop this embolisation, potentially preventing carotid artery thrombosis.;A prospective audit of all patients undergoing carotid endarterectomy was performed. The ability to monitor intraoperatively with TCD and perform completion angioscopy was assessed, as was the impact that these quality control techniques had on influencing the surgery. Patients were monitored postoperatively with TCD and any patient who developed sustained embolisation was commenced on an infusion of Dextran 40.;91% had continuous intraoperative TCD monitoring and 94% underwent successful completion angioscopy, a technical error was identified in 5% of angioscopic assessments. The intraoperative stroke rate was 0% during this study. Postoperative monitoring revealed that 5% of patients develop significant embolisation following CEA, Dextran 40 appeared to stop this embolisation. The overall 30-day stroke or death rate following CEA has fallen from 6% prior to 1992 to 2.2% in 1998.;It is possible to implement a quality control programme for CEA and this has been associated with a fall in the overall 30-day death and any stroke rate.
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Kenerson, Jonathan E. "Quality Assurance and Quality Control Methods for Resin Infusion." Fogler Library, University of Maine, 2010. http://www.library.umaine.edu/theses/pdf/KenersonJE2010.pdf.

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Cassady, Charles Richard. "Statistical quality control techniques using multilevel discrete product quality measures." Diss., This resource online, 1996. http://scholar.lib.vt.edu/theses/available/etd-06062008-151120/.

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Books on the topic "Isradipine and Quality Control"

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Forbes, Finlay. Quality control. London: ABG Professional Information, 2002.

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Besterfield, Dale H. Quality control. 8th ed. Upper Saddle River, N.J: Pearson/Prentice Hall, 2009.

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Besterfield, Dale H. Quality control. 4th ed. Englewood Cliffs: Prentice-Hall International, 1994.

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Quality control. 4th ed. Englewood Cliffs, N.J: Prentice Hall Career & Technology, 1994.

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Quality control. 3rd ed. Englewood Cliffs, N.J: Prentice Hall, 1990.

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Besterfield, Dale H. Quality control. 3rd ed. Englewood Cliffs, N.J: Prentice-Hall, 1990.

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Besterfield, Dale H. Quality control. 5th ed. London: Prentice-Hall International, 1998.

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Besterfield, Dale H. Quality control. 8th ed. Upper Saddle River, N.J: Pearson/Prentice Hall, 2009.

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Besterfield, Dale H. Quality control. 2nd ed. Englewood Cliffs, N.J: Prentice-Hall, 1986.

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Quality control. 5th ed. Upper Saddle River, N.J: Prentice Hall, 1998.

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Book chapters on the topic "Isradipine and Quality Control"

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Jaeger, Richard F. "Control Quality Control." In A Collection of Papers Presented at the 1978, 1979, and 1980 Meetings of the Materials & Equipment/Whitewares: Ceramic Engineering and Science Proceedings, Volume 1, Issue 9/10, 815–17. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2008. http://dx.doi.org/10.1002/9780470291047.ch24.

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Weik, Martin H. "quality control." In Computer Science and Communications Dictionary, 1384. Boston, MA: Springer US, 2000. http://dx.doi.org/10.1007/1-4020-0613-6_15215.

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Groshong, Richard H. "Quality Control." In 3-D Structural Geology, 285–304. Berlin, Heidelberg: Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/978-3-540-31055-6_10.

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Wagner, Stefan. "Quality Control." In Software Product Quality Control, 111–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-38571-1_4.

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Alt, Francis B., and Scott D. Grimshaw. "Quality Control." In Encyclopedia of Operations Research and Management Science, 1215–27. Boston, MA: Springer US, 2013. http://dx.doi.org/10.1007/978-1-4419-1153-7_840.

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Buxbaum, Engelbert. "Quality Control." In Biophysical Chemistry of Proteins, 427–34. Boston, MA: Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-7251-4_47.

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Khan, Aman. "Quality Control." In Cost and Optimization in Government, 316–47. Second Edition. | New York : Routledge, 2017. | Series: Public Administration and Public Policy | Previous edition: 2000.: Routledge, 2017. http://dx.doi.org/10.4324/9781315207674-11.

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Aubert, B. "Quality control." In MRI of the Body, 37–43. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-87556-4_3.

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van Meer, S. C. H., and R. A. M. Coenen. "Quality control." In Fibre Metal Laminates, 369–80. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-010-0995-9_24.

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Riepe, W., and M. R. Lahaniatis. "Quality Control." In Analyses of Hazardous Substances in Air, 27–41. Weinheim, FRG: Wiley-VCH Verlag GmbH, 2003. http://dx.doi.org/10.1002/352760023x.ch2.

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Conference papers on the topic "Isradipine and Quality Control"

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Forlizzi, Jodi, Carl DiSalvo, Jeffrey Bardzell, Ilpo Koskinen, and Stephan Wensveen. "Quality control." In the 2011 annual conference extended abstracts. New York, New York, USA: ACM Press, 2011. http://dx.doi.org/10.1145/1979742.1979497.

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Darker, A. "TOTAL QUALITY CONTROL." In 4th International Pacific Conference on Automotive Engineering. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 1987. http://dx.doi.org/10.4271/871263.

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Bonati, Marco A., Peter Moore, and Diego Gomez. "Detector quality control." In Software and Cyberinfrastructure for Astronomy VI, edited by Juan C. Guzman and Jorge Ibsen. SPIE, 2020. http://dx.doi.org/10.1117/12.2556497.

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Nguyen, Xuan Hung. "Networked control systems quality of service aware quality of control adaptation." In Factory Automation (ETFA 2011). IEEE, 2011. http://dx.doi.org/10.1109/etfa.2011.6059154.

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Al-Ameer, Hassan, Hijji Al-Alawi, and Abdulrahman Al-Balawi. "Air Quality Control Initiatives." In International Petroleum Technology Conference. International Petroleum Technology Conference, 2013. http://dx.doi.org/10.2523/iptc-16751-ms.

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Al-Ameer, Hassan, Hijji Al-Alawi, and Abdulrahman Al-Balawi. "Air Quality Control Initiatives." In SPE Middle East Oil and Gas Show and Conference. Society of Petroleum Engineers, 2013. http://dx.doi.org/10.2118/164435-ms.

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Storey, M. C. "Demystifying Log Quality Control." In SPE Asia Pacific Oil & Gas Conference and Exhibition. Society of Petroleum Engineers, 2016. http://dx.doi.org/10.2118/182313-ms.

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Krause, Markus, Farhad Mohammad Afzali, Simon Caton, and Margeret Hall. "Is Quality Control Pointless?" In Hawaii International Conference on System Sciences. Hawaii International Conference on System Sciences, 2019. http://dx.doi.org/10.24251/hicss.2019.636.

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Prabhakaran, Karthik, Harish Madras Sundar, Kathiravan Ekambaram, Dharmarajan Selva Bai, and Karunakaran Sekar. "Embedded quality control system." In 2010 2nd International Conference on Industrial Mechatronics and Automation (ICIMA 2010). IEEE, 2010. http://dx.doi.org/10.1109/icindma.2010.5538237.

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Motte, Adeline, and Samuel Bassetto. "Product driven quality control." In 2012 IEEE International Conference on Industrial Engineering and Engineering Management (IEEM). IEEE, 2012. http://dx.doi.org/10.1109/ieem.2012.6837759.

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Reports on the topic "Isradipine and Quality Control"

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Crosier, Ronald B. Multivariate Quality Control Procedures. Fort Belvoir, VA: Defense Technical Information Center, October 1988. http://dx.doi.org/10.21236/ada203305.

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Zambrano, Carlos, Vincent Drnevich, and Philippe Bourdeau. Advanced Compaction Quality Control. West Lafayette, IN: Purdue University, 2006. http://dx.doi.org/10.5703/1288284313408.

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Landon-Browne, A. R. R., R. D. Knight, B. A. Kjarsgaard, and H. A. J. Russell. Quality control of pXRF spectrometry. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2017. http://dx.doi.org/10.4095/299726.

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Jayachandran, Toke, and Richard Franke. Quality Control of Meteorological Observations. Fort Belvoir, VA: Defense Technical Information Center, September 1991. http://dx.doi.org/10.21236/ada242694.

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Shepherd, S. Jeff, Pei-Jan Paul Lin, John M. Boone, Dianna D. Cody, Jane R. Fisher, G. Donald Frey, Hy Glasser, et al. Quality Control in Diagnostic Radiology. AAPM, 2002. http://dx.doi.org/10.37206/73.

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Kane, J. S. Quality control and reference sample databases. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1993. http://dx.doi.org/10.4095/193261.

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Ong, Ghim Ping (Raymond), Samy Noureldin, and Kumares Sinha. Automated Pavement Condition Data Collection Quality Control, Quality Assurance, and Reliability. West Lafayette, Indiana: Purdue University, 2011. http://dx.doi.org/10.5703/1288284314288.

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Hill, D. G. Geophysical well log calibration and quality control. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1986. http://dx.doi.org/10.4095/123639.

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Chory, Joanne. Signal Transduction Pathways of Chloroplast Quality Control. Office of Scientific and Technical Information (OSTI), January 2018. http://dx.doi.org/10.2172/1416021.

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Kurnik, C., and C. Woodley. NREL Job Task Analysis: Quality Control Inspector. Office of Scientific and Technical Information (OSTI), May 2011. http://dx.doi.org/10.2172/1016434.

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