Relevant bibliographies by topics / Johnson Syndrome

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Johnson Syndrome'

Author: Grafiati
Published: 3 June 2025
Last updated: 19 July 2025

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Journal articles on the topic "Johnson Syndrome"

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Fitriany, Julia, and Fajri Alratisda. "STEVENS JOHNSON SYNDROME." AVERROUS: Jurnal Kedokteran dan Kesehatan Malikussaleh 5, no. 1 (2019): 94. http://dx.doi.org/10.29103/averrous.v5i1.1632.

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Stevens Johnson Syndrome (SJS) merupakan suatu sindroma atau kumpulan gejala yang mengenai kulit, selaput lendir, dan mata dengan keadaan umum yang bervariasi dari ringan sampai berat. Penyakit ini bersifat akut dan pada bentuk yang berat dapat menyebabkan kematian, oleh karena itu penyakit ini merupakan salah satu kegawatdaruratan penyakit kulit. Sindroma ini merupakan salah satu contoh immune-complex-mediated hypersensitivity, atau yang juga disebut reaksi hipersensitivitas tipe III, di mana kejadiaannya dapat diinduksi oleh paparan obat, infeksi, imunisasi, maupun akibat paparan fisik lain kepada pasien. Stevens Johnson Syndrome berisiko menimbulkan kematian, perawatan dan pengobatan pasien SJS sangat membutuhkan penanganan yang tepat dan cepat. Adapun terapi yang bisa diberikan antara lain perawatan terhadap kulit dan penggantian cairan tubuh, perawatan terhadap luka, serta perawatan terhadap mata. Kelangsungan hidup pasien Stevens Johnson Syndrome bergantung pada tingkat pengelupasan kulit, di mana apabila pengelupasan kulit semakin meluas, maka prognosisnya dapat menjadi semakin buruk. Selain itu, variabel lain seperti dengan usia penderita, keganasan penyakit tersebut, denyut jantung, kadar glukosa, kadar BUN dan tingkat bikarbonat juga dapat mempengaruhi kelangsungan hidup pasien.
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Nurtdinova, G. M., E. S. Galimova, O. I. Kucher, and V. K. Muslimova. "Stevens — Johnson syndrome." Russian Medical Review 4, no. 1 (2020): 52–57. http://dx.doi.org/10.32364/2587-6821-2020-4-1-52-57.

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Fein, Jordan D., and Kendal L. Hamann. "Stevens–Johnson Syndrome." New England Journal of Medicine 352, no. 16 (2005): 1696. http://dx.doi.org/10.1056/nejmicm031127.

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&NA;. "STEVENS-JOHNSON SYNDROME." Nursing 17, no. 6 (1987): 96–101. http://dx.doi.org/10.1097/00152193-198706000-00027.

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Bryant, Bobby G., and B. Lynn Mathews. "Stevens-Johnson Syndrome." Drug Intelligence & Clinical Pharmacy 20, no. 6 (1986): 489–93. http://dx.doi.org/10.1177/106002808602000612.

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Stevens-Johnson syndrome (SJS) is an acute inflammatory eruption of the skin and mucous membranes. Presented here is a case of an 18-month-old child admitted to the hospital with raised erythematous rash with some vesicular formation. The rash and associated symptomatology developed in a manner consistent with SJS. The child was treated for 27 days and was discharged in a much improved condition. This syndrome is reviewed in regard to incidence, etiology, clinical features, and management. Of particular emphasis are the drugs that may precipitate this syndrome.
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Starcher, Laura C., and Anne E. Braun. "Stevens-Johnson Syndrome." Dimensions Of Critical Care Nursing 4, no. 6 (1985): 330–34. http://dx.doi.org/10.1097/00003465-198511000-00003.

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Edwards, Robbie, and Marie Ridder. "Stevens-Johnson Syndrome." Dimensions Of Critical Care Nursing 4, no. 6 (1985): 335–48. http://dx.doi.org/10.1097/00003465-198511000-00004.

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Basak, Arpan Kumar, and Joya Debnath. "Stevens-Johnson Syndrome." KYAMC Journal 8, no. 2 (2018): 31–35. http://dx.doi.org/10.3329/kyamcj.v8i2.35702.

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Stevens-Johnson syndrome (SJS) is an immune complex mediated hypersensitivity complex that typically involves the skin and mucous membranes. While minor presentations may occur, significant involvement of oral, nasal, eye, vaginal, urethral, gastrointestinal, and lower respiratory mucous membranes may develop in the course of the illness. GI and respiratory involvement may progress to necrosis. Stevens-Johnson syndrome is a serious systemic disorder with the potential for severe morbidity and even death. The syndrome was first described in 1922, when the American pediatricians Albert Mason Stevens and Frank Chambliss Johnson reported the cases of 2 boys aged 7 and 8 years with "an extraordinary, generalized eruption with continued fever, inflamed buccal mucosa, and severe purulent conjunctivitis". Both cases had been misdiagnosed by primary care physicians as hemorrhagic measles. Erythema multiforme (EM), originally described by Von Hebra in 1866, was part of the differential diagnosis in both cases but was excluded because of the 'character of skin lesions, the lack of subjective symptoms, the prolonged high fever, and the terminal heavy crusting". Despite the presence of leucopenia in both cases, Stevens and Johnson in their initial report suspected an infectious disease of unknown etiology as the cause. In 1950, Thomas divided EM into 2 catagories: erythema multiforme minor (Von Hebra) and Erythema multiforme major (EMM). Since 1983, erythema multiforme major and Stevens-Johnson syndrome had been considered synonymous. In the 1990s, however, Bastuji and Roujeau each proposed that Erythema multiforme major and Stevens-Johnson syndrome are 2 distinct disorders. Several investigators propose that Stevens-Johnson syndrome and Toxic epidermal necrolysis (TEN) represent the same disease at different levels of severity. Although several classification schemes have been reported, the simplest breaks the disease down as follows: * Stevens-Johnson syndrome-A "minor form of TEN", with less than 10% body surface area (BSA) detachment. * Overlapping Stevens-Johnson syndrome/Toxic epidermal necrolysis (SJS/TEN)-Detachment of 10-30% BSA. * Toxic epidermal necrolysis-Detachment of more than 30%BSA.KYAMC Journal Vol. 8, No.-2, Jan 2018, Page 31-35
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Davis, Wesley D., and Phillip A. Schafer. "Stevens–Johnson Syndrome." Advanced Emergency Nursing Journal 40, no. 3 (2018): 176–82. http://dx.doi.org/10.1097/tme.0000000000000197.

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Dhannisa Ika Savitri and Mohammad Mimbar Topik. "Stevens-Johnson Syndrome." Detector: Jurnal Inovasi Riset Ilmu Kesehatan 2, no. 1 (2024): 217–26. http://dx.doi.org/10.55606/detector.v2i1.3335.

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Stevens-Johnson Syndrome (SJS) is an antibody-mediated disease characterized by prodromal symptoms followed by severe mucocutaneous manifestations. The more severe form of SJS is Toxic Epidermal Necrolysis (TEN), which causes an inflammatory response resulting in keratinocyte necrosis and perivascular lymphocyte infiltration. The incidence of SJS is said to be 1 - 6 per one million people per year, while the incidence of TEN is only 0.4 - 1.2 per one million people per year. So far, similar studies on SJS/TEN cases in Indonesia are minimal as this disease is one of the rare cases. SJS/TEN is an emergency with high mortality and disability. Moderate to severe cases have a mortality rate of 5-15%. In this case, the patient was a 62-year-old male. Based on history and physical examination, the patient was diagnosed with Stevens-Johnson Syndrome (SJS). In the dermatologic status of the manus region dextra and sinistra, sagging bulla was found, and in the anterior thorax region, erosive lesions due to ruptured bulla were found. In the medialis femoris region dextra, multiple erythematous, annular macular lesions were found. There were erosions and crusts on the labial and genital regions.
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Dissertations / Theses on the topic "Johnson Syndrome"

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Chartier, Claire Patey Olivier. "Infection à Mycoplasma pneumoniae et syndrome de Stevens-Johnson." Créteil : Université de Paris-Val-de-Marne, 2006. http://doxa.scd.univ-paris12.fr:80/theses/th0236637.pdf.

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Leclercq, Emmanuel. "Syndrome de stevens-johnson associee a un syndrome de detresse respiratoire de type adulte : un cas pediatrique." Lille 2, 1989. http://www.theses.fr/1989LIL2M392.

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Gaultier, Frédérick. "Contribution à l'étude de la nécrolyse épidermique toxique et du syndrome de Stevens-Johnson." Paris 5, 2006. http://www.theses.fr/2006PA05M001.

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La nécrolyse épidermique toxique et le Syndrome de Stevens-Johnson sont des toxidermies médicamenteuses rares d'étiologies inconnues. Au cours de notre étude nous avons cherché à préciser les mécanismes physiopathogéniques impliqués dans la genèse de ces pathologies. L'histologie a montré une atteinte et une désorganisation modérées de la matrice extracellulaire. L'immunohistochimie et les études biochimiques nous ont permis de mettre en évidence un déséquilibre de la balance MMPs/TIMPs en faveur des MMPs avec un schéma d'expression essentiellement épidermique. Enfin, nous proposons un processus étio-pathogénique compatible avec nos résultats et ceux de la littérature qui permet d'envisager une alternative thérapeutique<br>Toxic Epidermal Necrolysis and Stevens-Johnson Syndrome are rare toxic mucocutaneous drugs reactions withunknown etiologies. In our study, we tried to determine the mechanism involved in these diseases. Histological studies points out few disorganisation of the extracellular matrix. Histochemistry and biochemistry studies led us to evidence a break down of the MMPs/TIMPS balance with more MMPs than TIMPs especially localised in the epidermis. At last, we proposed a theory in accordance with scientific literature and our results in order to get an alternative therapy
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Van, Zyl Lourens Marthinus. "Prevalence of chronic ocular complications in Stevens-Johnson Syndrome and toxic epidermal necrolysis." Master's thesis, University of Cape Town, 2013. http://hdl.handle.net/11427/2899.

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Includes abstract.<br>Includes bibliographical references.<br>The main objective of the study is to identify and grade the severity of chronic ocular complications in patients who were treated for SJS and TEN at Groote Schuur Hospital. The secondary objective is to identify patients who need referrals to specialist ophthalmic clinics for treatable ocular complications of SJS and TEN.
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Wolkenstein, Pierre. "Prédisposition métabolique et mécanisme de mort cellulaire au cours du syndrome de Stevens-Johnson et de la nécrolyse épidermique toxique." Paris 12, 1996. http://www.theses.fr/1996PA120079.

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Le but de notre travail etait d'etudier les predispositions metaboliques et la mort keratinocytaire au cours de la necrolyse epidermique toxique. Nous avons montre que: 1) la necrolyse epidermique toxique aux sulfamides et aux anticomitiaux etait liee a un deficit hautement specifique de la detoxication des metabolites reactifs du medicament responsable, constitutionnel et genetiquement transmis ; 2) un genotype d'acetylation lente est un facteur de risque de la necrolyse epidermique toxique induite par les sulfamides ; 3) le cytochrome p450 3a4 a un role majeur dans la formation des metabolites reactifs de la carbamazepine et est present dans l'epiderme humain ; 4) le mecanisme de mort cellulaire au cours la necrolyse epidermique toxique est apoptotique. En conclusion, la necrolyse epidermique toxique est associee a une predisposition metabolique conduisant a une accumulation des metabolites reactifs du medicament responsable ; chez certains sujets, la formation d'un neoantigene (metabolites-p450) declencherait une cytotoxicite a mediation cellulaire qui conduirait a l'apoptose des keratinocytes
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Poletti, Jabbour Jamil, Rospigliosi Andrés Wiegering, Elías Reneé Pereyra, and Barrera Carmen Cecilia Elías. "Carbamazepine and oxcarbazepine: reflections after an oxcarbazepine-induced Stevens-Johnson syndrome/toxic epidermal necrolysis overlap." Springer International Publishing, 2016. http://hdl.handle.net/10757/609483.

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AMRAOUI, ABDERRAHIM. "L'erytheme polymorphe majeur ou syndrome de stevens-johnson associe a une infection a mycoplasma pneumoniae : une observation pediatrique." Lille 2, 1993. http://www.theses.fr/1993LIL2M149.

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Giron, Sabrina Delverdier Maxence. "L'Érythème polymorphe chez le chien et le chat données bibliographiques récentes /." [S.l.] : [s.n.], 2008. http://oatao.univ-toulouse.fr/1147/1/jan_1147.pdf.

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Cooper, Ryan. "Intravenous Immunoglobulin Use in the Treatment of Toxic Epidermal Necrolysis and Stevens-Johnson Syndrome: A 10-year Retrospective Analysis of Patients of a Single Burn Center." Thesis, The University of Arizona, 2014. http://hdl.handle.net/10150/315845.

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A Thesis submitted to The University of Arizona College of Medicine - Phoenix in partial fulfillment of the requirements for the Degree of Doctor of Medicine.<br>Stevens - Johnson syndrome and Toxic Epidermal Necrolysis Syndrome are rare, but serious conditions affecting skin and mucous membranes that are primarily treated with supportive care. Other more specific therapies have limited evidence to support the benefit of their use; one such treatment is intravenous immunoglobulin (IVIG). The use of IVIG in the treatment of these syndromes remain controversial due to mixed results demonstrated in the literature, and at present is not considered a component of the standard of care. This study seeks to provide additional data regarding the efficacy of IVIG treatment on mortality in a small cohort of patients presenting with these syndromes at a regional burn center over a 10-year period; data was retrospectively collected from patient medical records. On analysis of this data, IVIG use showed a potential, but not significant. improvement on mortality in comparison to the non-treatment group. Compared with the non-treatment group, odds ratios for death were 0.81 (95% CI 0.3-2.0) for IVIG. There is ultimately no new evidence that the benefit of IVIG in the treatment of Stevens - Johnson syndrome and Toxic Epidermal Necrolysis Syndrome is anything more than potential. Further investigation should include a rigorous analysis and comparison of different dosing regimens.
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Tanno, Luciana Kase. "Estudo de associação de fatores genéticos em indivíduos com reações de hipersensibilidade tardia induzida por anticonvulsivantes aromáticos." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/5/5146/tde-01122014-113756/.

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Intrdodução: As terapias com anticonvulsivantes de anel aromático (ACA) são freqüentemente associadas a reações adversas. No entanto, reações de hipersensibilidade (RH) não-imediatas (tardias) a estes fármacos são raras, imprevisíveis e geralmente relacionadas à alta morbidade e mortalidade. Foi demonstrado que estas RH aos ACA estão fortemente associadas ao Antígenio de Leucócitos Humanos (HLA)-B*1502 em pacientes chineses e ao HLA-A*3101 em caucasianos. Polimorfismos de genes do metabolismo do Citocromo P450 (CYP)2C9 foram mais associados a estas reações em pacientes orientais. Objetivo: Nosso objetivo é analisar a associação das reações de hipersensibilidade a anticonvulsivantes de anel aromático com os polimorfismos descritos e de interesse, bem como realizar a tipificação de HLA em uma população de São Paulo, Brasil. Métodos: Estudo tipo caso-controle com genotipagem dos polimorfismos de interesse por reação em cadeia da polimerase (PCR) em tempo real e tificação de HLA A, B, C, DRB, DQA, DQB por PCR seguido de deteção utilizando método LuminexR. A avaliação fenotípica se baseou em sistemas de escores padronizados, utilizando um questionário adaptado da ENDA (Rede Européia de Alergia a Medicamentos), em registros médicos e no acompanhamento clínico. O teste de contato com o medicamento suspeito foi realizado de acordo com as recomendações da ENDA, nos pacientes que apresentaram reação. Resultados: Foram estudados 506 pacientes, 65% do gênero feminino e a idade média foi de 43,6 anos. Oitenta por cento era de etnia mista. Polimorfismos de HLA-A*3101, HLA-B*1502, CYP2C9, CYP2C19 e CYP3A5 foram analisados de 55 indivíduos com reações de hipersensibilidade (RH) a antiepilépticos, de 85 tolerantes e de 366 controles sadios. Dos 55 casos foram validados como RH, 32 apresentaram Reação a Drogas com eosinofilia e sintomas sistêmicos (DRESS), 12 Síndrome de Stevens-Johnson (SSJ) e 11 exantema maculo-papular. De todos os 46 testes de contato com medicamento, 29 (63%) foram positivos, tanto em SSJ como em DRESS. Houve associação significativa entre polimorfismo de HLA-A*1502 e casos. Nenhum de nossos grupos de estudo apresentou associação positiva com polimorfismos de HLAA* 3101. Verificamos uma forte associação entre a atividade normal do CYP3A5 e indivíduos tolerantes quando comparado com casos (p = 0,0002, OR = 4,8). A tipificação de HLA demonstrou associação significante de HLA-A*31, HLA-A*74, HLA-B*35 e HLA-B*53 com reações graves aos ACA e de HLA-B*44 e HLA-C*03 com indivíduos tolerantes. Conclusão: Estes resultados sugere fortemente a existência de fatores genéticos de risco e/ou de proteção a RH a ACA em indivíduos brasileiros, mas não devem ser considerados de forma isolada. Assim, a relevância deste estudo extrapola o objetivo de estudo caso-controle e sugere um modelo como forma de prevenção primária às RH aos ACA.<br>Background: Antiepileptics with aromatic ring (AAR) therapies are frequently associated with adverse reactions. Nevertheless non-immediate (late) hypersensitivity reactions (HR) to these drugs are rare, unpredictable and usually related with high morbidity and mortality. A strong pharmacogenetic association has been reported in Chinese patients with these HR and Human Leukocyte Antigen (HLA)-B*1502 and with HLA- A*3101 in caucasians. Polymorphism of genes of P450 Cytocrome (CYP)2C9 has been related to these reactions in patients of oriental origin. Objective: Our aim is to analyze the association between hypersensitivity reactions due to AAR and the described polymorphisms, as well as perform the typification of HLA in a population of São Paulo, Brazil. Methods: Case-control study genotyping the polymorphisms of interest by polymerase chain reaction (PCR) real time and typifying HLA A, B, C, DRB, DQA, DQB by PCR followed by LuminexR .The phenotype evaluation was based on standardized scoring systems using an adapted ENDA (European Network of Drug Allergy) questionnaire, medical records and on the clinical follow-up in our Allergy Clinic. The patch test with the culprit drug was performed in patients who experienced HR according to the ENDA recommendations. Results: We studied 506 subjects, 65% female and mean age was 43,6 years. Eighty percent had mixed ethnicity. Polymorphisms of HLA-B*1502, HLA- A*3101, CYP2C9, CYP2C19 e CYP3A5 were studied in 55 subjects with antiepileptics HR, 85 tolerants, and 366 control subjects. Of 55 cases were validated as AHR, 32 presented Drug Reaction with Eosinophilia and Systemic Symptoms (DRESS), 12 Stevens-Johnson Syndrome (SJS) and 11 maculopapular exanthema. Of all 46 drug patch tests, 29 (63%) were positive, in both SJS and DRESS. A significant association between polymorphism of HLA-A*1502 and cases was found. None of our study groups presented positive association with HLA-A*3101 polymorphisms. We found a strong association between the normal activity of CYP3A5 and tolerants subjects when compared to HR (p=0.0002, OR=4.8). The HLA typification showed a significant association between HLA-A*31, HLA-A*74, HLAB* 35 e HLA-B*53 and severe AAR reactions and HLA-B*44 and HLA-C*03 in tolerants subjects. Conclusion: These results strongly suggests the existence of genetic risk and/or protective factors to the development of HR to AAR AAR in Brazilian subjects, but it should not be considered in a isolated manner. So, the relevance of this study extrapolates the aim of a case-control study and suggests a system of primary prevention to HR due to AAR
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Books on the topic "Johnson Syndrome"

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Jennison, Elizabeth. Johnson Brothers Company, Pittsburgh, Pennsylvania. U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 1995.

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National Institute for Occupational Safety and Health, ed. Johnson Brothers Company, Pittsburgh, Pennsylvania. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 1995.

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Joe, Cocalis, and National Institute for Occupational Safety and Health, eds. Johnson Brothers Company, Pittsburgh, Pennsylvania. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 1995.

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Parker, James N., and Philip M. Parker. Stevens-Johnson Syndrome: A medical dictionary, bibliography, and annotated research guide to internet references. ICON Health Publications, 2004.

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Mehta, Jodhbir, Mayumi Ueta, and Shigeru Kinoshita, eds. Update on Stevens Johnson Syndrome. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-314-8.

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Johnson Controls, Inc., Lexington, Kentucky. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 1994.

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Publications, ICON Health. Stevens-Johnson Syndrome - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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Kānsưksā wičhai ʻubatkān kānkœ̄t læ khwāmsamphan kap kānchai yā khō̜ng kānphǣ thāng phiunang nai klum Stevens-Johnson syndrome. Sūn Tittām ʻĀkān ʻAn Mai Phưng Prasong Čhāk Kānchai Yā, Kō̜ng Wichākān, Samnakngān Khana Kammakān ʻĀhān læ Yā, Krasūang Sāthāranasuk, 1999.

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MELVIN, Mike. Stevens - Johnson Syndrome Active Remedy: Ultimate Guide on How to Completely Defeat Symptoms Effectively, and Get Your Immediate and Restorative Healing. Independently Published, 2022.

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Taghipour, Kathy. Mucosal disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0255.

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This chapter discusses the following mucous membrane disorders: pemphigus vulgaris, lichen planus, and Stevens–Johnson syndrome. Pemphigus vulgaris is an autoimmune disease that affects the skin and the mucosal membranes with blisters and erosions. Lichen planus is a cell-mediated immunological mucocutaneous disease; oral lichen planus may present with erosions, white streaks, or plaques in the oral cavity. Stevens–Johnson syndrome is an emergency dermatological condition in which an immunological hypersensitivity causes erosions and inflammation of mucosal membranes and the skin. As well as providing definitions of these diseases, this chapter discusses their etiology, typical symptoms, uncommon symptoms, demographics, natural history, complications, diagnostic approach, other diagnoses that should be considered, prognosis, and treatment.
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Book chapters on the topic "Johnson Syndrome"

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Siddiqui, Aazim A., and Allen O. Eghrari. "Stevens Johnson Syndrome." In Encyclopedia of Ophthalmology. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-35951-4_594-1.

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Siddiqui, Aazim A., and Allen O. Eghrari. "Stevens Johnson Syndrome." In Encyclopedia of Ophthalmology. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_594.

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Coster, D. J. "Stevens-Johnson Syndrome." In Cicatrising Conjunctivitis. KARGER, 1997. http://dx.doi.org/10.1159/000060696.

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Pollard, Margaret C., Laura M. Le, and Deepinder K. Dhaliwal. "Stevens–Johnson Syndrome." In Corneal Emergencies. Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-5876-1_17.

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Leung, Alexander K. C., Cham Pion Kao, Andrew L. Wong, et al. "Stevens-Johnson Syndrome." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1672.

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Metze, Dieter, Tam Nguyen, Birgit Haack, et al. "Dubin-Johnson Syndrome." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_515.

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Gammanpila, Ranthilaka R., and Eckart Haneke. "Stevens–Johnson syndrome." In Dermatoses in Dark Vs Light Skin. CRC Press, 2025. https://doi.org/10.1201/9781003479512-114.

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Nabavi-Rad, Ali, Hamid Asadzadeh Aghdaei, and Abbas Yadegar. "Dubin-Johnson Syndrome (DJS)." In Genetic Syndromes. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-319-66816-1_1001-1.

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Abdel-Naser, Mohamed Badawy. "Erythema Multiforme and Stevens-Johnson Syndrome." In Pigmented Ethnic Skin and Imported Dermatoses. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-69422-1_27.

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Yoshida, Satoshi. "Stevens–Johnson Syndrome/Toxic Epidermal Necrolysis." In Challenging Cases in Allergic and Immunologic Diseases of the Skin. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-60761-296-4_6.

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Conference papers on the topic "Johnson Syndrome"

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Strukar, Nedim, Verica Mišanović, Adisa Čengić, Aida Karačić, Alma Mujić, and Emina Ribić. "147 Stevens-Johnson syndrome." In 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.147.

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Walsh, B. C. "Chlordiazepoxide Induced Steven-Johnson Syndrome." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6125.

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Ramasli Gursoy, Tugba, Ayse Tana Aslan, Hacer Ilbilge Ertoy Karagol, et al. "A case of Stevens-Johnson Syndrome associated with bronchiolitis obliterans." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3523.

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Mehdi, N. F., and R. Katz. "Bronchiolitis Obliterans Secondary to Steven Johnson Syndrome, Diagnostic and Therapeutic Challenges." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4931.

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Sandhya Rani, D. C. "Foresight Gives Eyesight...Amniotic Membrane Transplantation for Steven Johnson Syndrome-Anaesthetic Management." In ISACON KARNATAKA 2017 33rd Annual Conference of Indian Society of Anaesthesiologists (ISA), Karnataka State Chapter. Indian Society of Anaesthesiologists (ISA), 2017. http://dx.doi.org/10.18311/isacon-karnataka/2017/ep086.

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Gans, A., J. Taylor, and N. N. Goel. "A Case of Drug-Induced Stevens-Johnson Syndrome With Primarily Visceral Involvement." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a5678.

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Sabogal, Carlos, Katarzyna Zarzycki, and Daniel F. Garcia. "Use Of Inflixamab On The Treatment Of Bronchiolitis Obliterans After Stevens-Johnson Syndrome." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a1871.

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Pant, A., A. Thatayatikom, M. Schecter, and D. R. Ezmigna. "A Rare Case of Sjogren’s Syndrome Following Steven Johnson Syndrome and Bronchiolitis Obliterans in a 15-year-old Female." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a1897.

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Chaudhary, Himanshi, Dharmagat Bhattarai, Pandiarajan Vignesh, et al. "159 Pediatric onset lupus with stevens johnson syndrome/toxic epidermal necrolysis: an unusual association." In 13th International Congress on Systemic Lupus Erythematosus (LUPUS 2019), San Francisco, California, USA, April 5–8, 2019, Abstract Presentations. Lupus Foundation of America, 2019. http://dx.doi.org/10.1136/lupus-2019-lsm.159.

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Karingattil, J., and R. Samad. "Rapid Progression of Chronic Obstructive Pulmonary Disease as a Complication After Steven Johnson Syndrome." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a2381.

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Reports on the topic "Johnson Syndrome"

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Stewart, Thomas, Hemali Shah, and John Frew. Infectious complications of Stevens Johnson syndrome and toxic epidermal necrolysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.1.0124.

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Stewart, Thomas, Joshua Farrell, and John Frew. Psychological complications of Stevens Johnson syndrome and toxic epidermal necrolysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.1.0097.

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Chee, Elyssa, Esther Hong, and Thomas Stewart. Stevens Johnson syndrome and toxic epidermal necrolysis due to topical drugs: A systematic review of case reports. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2025. https://doi.org/10.37766/inplasy2025.7.0052.

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Stewart, Thomas, Jeremy Chan, and John Frew. A systematic review and meta-analysis of Non-SCORTEN mortality predictors in Stevens Johnson syndrome and toxic epidermal necrolysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.4.0005.

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Stewart, Thomas, Nicole Seebacher, and John Frew. A systematic review and meta-analysis of case-control studies on cytokines in blister fluid and skin of patients with Stevens Johnson syndrome and toxic epidermal necrolysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.2.0123.

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Severe Cutaneous Adverse Reactions. A consensus by a CIOMS Working Group. CIOMS, 2025. https://doi.org/10.56759/lrty1600.

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Abstract:
In clinical practice, there is mounting concern about the burden of SCAR in relation to novel biologics as well as the increasing cost of diagnosis and management. This consensus report provides unique insights and the latest thinking from renowned experts on this important topic. The skin is among the parts of the body most commonly affected by adverse drug reactions (ADRs). Cutaneous ADRs affect 2% to 3% of all hospitalized patients and have a wide spectrum of clinical manifestations, are caused by various medicinal products, and result from different pathophysiologic mechanisms. Hence, their diagnosis and management are challenging. However, approximately 0.1% to 1% of patients with medicinal product eruptions have serious ADRs which can lead to disabling sequelae and in some cases, fatalities. Although Severe Cutaneous Adverse Reactions (SCAR) are rare, they are a significant health challenge and hinder the safe and effective use of medicines. In short, they pose substantial hurdles to drug developers, medicines regulators and health professionals. SCAR include Stevens-Johnson syndrome (SJS), toxic epidermal necrolysis (TEN), drug reaction with eosinophilia and systemic symptoms (DRESS), acute generalized exanthematous pustulosis (AGEP), and generalized bullous fixed drug eruptions (GBFDE). Premarketing randomized clinical trials have limited power to detect SCAR. There is also a lack of specific diagnostic tests for SCAR, which today, depend on subjective causality assessment methods. These factors highlight the urgent need for guidelines, including how to predict, prevent, detect and diagnose SCAR either during drug development or in the post-marketing phase.
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