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Journal articles on the topic 'Johnson Syndrome'

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Published: 3 June 2025

Last updated: 19 July 2025

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1

Fitriany, Julia, and Fajri Alratisda. "STEVENS JOHNSON SYNDROME." AVERROUS: Jurnal Kedokteran dan Kesehatan Malikussaleh 5, no. 1 (2019): 94. http://dx.doi.org/10.29103/averrous.v5i1.1632.

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Stevens Johnson Syndrome (SJS) merupakan suatu sindroma atau kumpulan gejala yang mengenai kulit, selaput lendir, dan mata dengan keadaan umum yang bervariasi dari ringan sampai berat. Penyakit ini bersifat akut dan pada bentuk yang berat dapat menyebabkan kematian, oleh karena itu penyakit ini merupakan salah satu kegawatdaruratan penyakit kulit. Sindroma ini merupakan salah satu contoh immune-complex-mediated hypersensitivity, atau yang juga disebut reaksi hipersensitivitas tipe III, di mana kejadiaannya dapat diinduksi oleh paparan obat, infeksi, imunisasi, maupun akibat paparan fisik lain kepada pasien. Stevens Johnson Syndrome berisiko menimbulkan kematian, perawatan dan pengobatan pasien SJS sangat membutuhkan penanganan yang tepat dan cepat. Adapun terapi yang bisa diberikan antara lain perawatan terhadap kulit dan penggantian cairan tubuh, perawatan terhadap luka, serta perawatan terhadap mata. Kelangsungan hidup pasien Stevens Johnson Syndrome bergantung pada tingkat pengelupasan kulit, di mana apabila pengelupasan kulit semakin meluas, maka prognosisnya dapat menjadi semakin buruk. Selain itu, variabel lain seperti dengan usia penderita, keganasan penyakit tersebut, denyut jantung, kadar glukosa, kadar BUN dan tingkat bikarbonat juga dapat mempengaruhi kelangsungan hidup pasien.

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2

Nurtdinova, G. M., E. S. Galimova, O. I. Kucher, and V. K. Muslimova. "Stevens — Johnson syndrome." Russian Medical Review 4, no. 1 (2020): 52–57. http://dx.doi.org/10.32364/2587-6821-2020-4-1-52-57.

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3

Fein, Jordan D., and Kendal L. Hamann. "Stevens–Johnson Syndrome." New England Journal of Medicine 352, no. 16 (2005): 1696. http://dx.doi.org/10.1056/nejmicm031127.

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4

&NA;. "STEVENS-JOHNSON SYNDROME." Nursing 17, no. 6 (1987): 96–101. http://dx.doi.org/10.1097/00152193-198706000-00027.

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5

Bryant, Bobby G., and B. Lynn Mathews. "Stevens-Johnson Syndrome." Drug Intelligence & Clinical Pharmacy 20, no. 6 (1986): 489–93. http://dx.doi.org/10.1177/106002808602000612.

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Stevens-Johnson syndrome (SJS) is an acute inflammatory eruption of the skin and mucous membranes. Presented here is a case of an 18-month-old child admitted to the hospital with raised erythematous rash with some vesicular formation. The rash and associated symptomatology developed in a manner consistent with SJS. The child was treated for 27 days and was discharged in a much improved condition. This syndrome is reviewed in regard to incidence, etiology, clinical features, and management. Of particular emphasis are the drugs that may precipitate this syndrome.

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6

Starcher, Laura C., and Anne E. Braun. "Stevens-Johnson Syndrome." Dimensions Of Critical Care Nursing 4, no. 6 (1985): 330–34. http://dx.doi.org/10.1097/00003465-198511000-00003.

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7

Edwards, Robbie, and Marie Ridder. "Stevens-Johnson Syndrome." Dimensions Of Critical Care Nursing 4, no. 6 (1985): 335–48. http://dx.doi.org/10.1097/00003465-198511000-00004.

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8

Basak, Arpan Kumar, and Joya Debnath. "Stevens-Johnson Syndrome." KYAMC Journal 8, no. 2 (2018): 31–35. http://dx.doi.org/10.3329/kyamcj.v8i2.35702.

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Stevens-Johnson syndrome (SJS) is an immune complex mediated hypersensitivity complex that typically involves the skin and mucous membranes. While minor presentations may occur, significant involvement of oral, nasal, eye, vaginal, urethral, gastrointestinal, and lower respiratory mucous membranes may develop in the course of the illness. GI and respiratory involvement may progress to necrosis. Stevens-Johnson syndrome is a serious systemic disorder with the potential for severe morbidity and even death. The syndrome was first described in 1922, when the American pediatricians Albert Mason Stevens and Frank Chambliss Johnson reported the cases of 2 boys aged 7 and 8 years with "an extraordinary, generalized eruption with continued fever, inflamed buccal mucosa, and severe purulent conjunctivitis". Both cases had been misdiagnosed by primary care physicians as hemorrhagic measles. Erythema multiforme (EM), originally described by Von Hebra in 1866, was part of the differential diagnosis in both cases but was excluded because of the 'character of skin lesions, the lack of subjective symptoms, the prolonged high fever, and the terminal heavy crusting". Despite the presence of leucopenia in both cases, Stevens and Johnson in their initial report suspected an infectious disease of unknown etiology as the cause. In 1950, Thomas divided EM into 2 catagories: erythema multiforme minor (Von Hebra) and Erythema multiforme major (EMM). Since 1983, erythema multiforme major and Stevens-Johnson syndrome had been considered synonymous. In the 1990s, however, Bastuji and Roujeau each proposed that Erythema multiforme major and Stevens-Johnson syndrome are 2 distinct disorders. Several investigators propose that Stevens-Johnson syndrome and Toxic epidermal necrolysis (TEN) represent the same disease at different levels of severity. Although several classification schemes have been reported, the simplest breaks the disease down as follows: * Stevens-Johnson syndrome-A "minor form of TEN", with less than 10% body surface area (BSA) detachment. * Overlapping Stevens-Johnson syndrome/Toxic epidermal necrolysis (SJS/TEN)-Detachment of 10-30% BSA. * Toxic epidermal necrolysis-Detachment of more than 30%BSA.KYAMC Journal Vol. 8, No.-2, Jan 2018, Page 31-35

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9

Davis, Wesley D., and Phillip A. Schafer. "Stevens–Johnson Syndrome." Advanced Emergency Nursing Journal 40, no. 3 (2018): 176–82. http://dx.doi.org/10.1097/tme.0000000000000197.

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10

Dhannisa Ika Savitri and Mohammad Mimbar Topik. "Stevens-Johnson Syndrome." Detector: Jurnal Inovasi Riset Ilmu Kesehatan 2, no. 1 (2024): 217–26. http://dx.doi.org/10.55606/detector.v2i1.3335.

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Stevens-Johnson Syndrome (SJS) is an antibody-mediated disease characterized by prodromal symptoms followed by severe mucocutaneous manifestations. The more severe form of SJS is Toxic Epidermal Necrolysis (TEN), which causes an inflammatory response resulting in keratinocyte necrosis and perivascular lymphocyte infiltration. The incidence of SJS is said to be 1 - 6 per one million people per year, while the incidence of TEN is only 0.4 - 1.2 per one million people per year. So far, similar studies on SJS/TEN cases in Indonesia are minimal as this disease is one of the rare cases. SJS/TEN is an emergency with high mortality and disability. Moderate to severe cases have a mortality rate of 5-15%. In this case, the patient was a 62-year-old male. Based on history and physical examination, the patient was diagnosed with Stevens-Johnson Syndrome (SJS). In the dermatologic status of the manus region dextra and sinistra, sagging bulla was found, and in the anterior thorax region, erosive lesions due to ruptured bulla were found. In the medialis femoris region dextra, multiple erythematous, annular macular lesions were found. There were erosions and crusts on the labial and genital regions.

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11

Ting, H. C., and B. A. Adam. "Stevens-Johnson Syndrome." International Journal of Dermatology 24, no. 1 (1985): 587–91. http://dx.doi.org/10.1111/j.1365-4362.1985.tb05580.x.

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12

Morii, Kazuhiko, and Takeharu Yamamoto. "Dubin–Johnson Syndrome." New England Journal of Medicine 375, no. 1 (2016): e1. http://dx.doi.org/10.1056/nejmicm1509529.

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13

Ting, H. C., and B. A. Adam. "Stevens-Johnson Syndrome." International Journal of Dermatology 24, no. 9 (1985): 587–91. http://dx.doi.org/10.1111/j.1365-4362.1985.tb05857.x.

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14

Noe del Cueto-Aguilera, Angel, Diego García-Compean, Susanna Isabel Scharrer Cabello, and José Alberto González-González. "Dubin-Johnson Syndrome." American Journal of Gastroenterology 115, no. 9 (2020): 1386. http://dx.doi.org/10.14309/ajg.0000000000000519.

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15

Lesh, Diane. "Stevens-Johnson Syndrome." Journal of the American Academy of Nurse Practitioners 7, no. 11 (1995): 549–51. http://dx.doi.org/10.1111/j.1745-7599.1995.tb01245.x.

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16

Olisova, O. Yu, O. V. Grabovskaya, N. P. Teplyuk, A. A. Lepekhova, D. V. Ignatiev, and Kseniya V. Ignatieva. "Stevens-Johnson syndrome." Russian Journal of Skin and Venereal Diseases 19, no. 4 (2016): 216–20. http://dx.doi.org/10.18821/1560-9588-2016-19-4-216-220.

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The literature review of the epidemiology, etiology and pathogenesis, clinical presentation and diagnosis of the syndrome of Stevens-Johnson severe life-threatening disease characterized by extensive lesions of the skin and mucous membranes, fever and bone and muscle aches, often induced by medication is presented. A review on the methods of treatment and prevention of this disease is presented. The clinical case of patient with the syndrome of Stevens-Johnson is described. The successful treatment of Syndrome Stevens-Johnson with high-dose systemic glucocorticoids was performed.

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17

Bakshi, Satvinder Singh. "Stevens–Johnson syndrome." Internal and Emergency Medicine 14, no. 2 (2018): 323–24. http://dx.doi.org/10.1007/s11739-018-1981-0.

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18

de Ru, M. H., and R. N. Sukhai. "Stevens-Johnson syndrome." European Journal of Pediatrics 166, no. 12 (2007): 1303–4. http://dx.doi.org/10.1007/s00431-006-0402-y.

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19

Bauer, Renee, Jan Weust, and Emily Cannon. "Stevens-Johnson syndrome." American Nurse Journal 19, no. 4 (2024): 50. http://dx.doi.org/10.51256/anj042450.

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20

McGouran, DCR, T. Petterson, JM McLaren, and MP Wolbinski. "Mucositis, Conjunctivitis but no rash – The “Atypical Stevens –Johnson syndrome”." Acute Medicine Journal 10, no. 2 (2011): 81–82. http://dx.doi.org/10.52964/amja.0473.

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The Stevens-Johnson syndrome (SJS) classically involves a rash, conjunctivitis and mucositis. We describe the case of a young adult male with isolated mucositis and conjunctivitis . Previous rare reports of severe SJS like syndromes without a rash are confined to children, usually with mycoplasma pnemoniae infection.1 Terminology for this syndrome includes – “Stevens-Johnson Syndrome without skin lesions”, or “Atypical Stevens – Johnson Syndrome”.2 This case highlights the importance of maintaining an open mind when a “full house” of clinical features is absent. It also illustrates the use of a rapid electronic literature review as a clinical tool. The importance of updating records when a drug has been cleared of causing harm is highlighted.

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21

Oliveira, Alex Ferreira de, Ingrith do Socorro Neves da Silva, Lídia Pinheiro de Brito, et al. "Stevens-Johnson Syndrome, fisiopatológics aspects: A literature review." Revista Científica Multidisciplinar Núcleo do Conhecimento 06, no. 08 (2016): 40–51. http://dx.doi.org/10.32749/nucleodoconhecimento.com.br/health/syndrome-stevens-johnson.

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22

James, Apollo, Ammu A, Jasmin Elizabeth Thomas, Merin Joseph, T. R. Ashok kumar, and T. Sivakumar. "Ofloxacin induced steven johnson syndrome- case report." Asian Pacific Journal of Health Sciences 2, no. 2 (2015): 10–11. http://dx.doi.org/10.21276/apjhs.2015.2.2.3.

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23

Chernysh, V. F., N. N. Haritonova, A. N. Kulikov, P. A. Kacherovich, and A. A. Kol'bin. "Current surgical treatment options for ocular complications of Stevens-Johnson and Lyell's syndromes." Russian Journal of Clinical Ophthalmology 23, no. 2 (2023): 99–106. http://dx.doi.org/10.32364/2311-7729-2023-23-2-99-106.

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Epidermolytic drug reactions (EDR) are severe acute drug-induced allergic disorders characterized by extensive lesions of the skin and mucous membranes. Drug-induced Stevens-Johnson and Lyell's syndromes manifested with acute skin and mucous membrane disorders and often leading to lethal outcome may affect eyelids and ocular surface with different levels of severity and thus can cause serious functional ocular complications. The authors present two clinical reports of patients with ocular EDR manifestations in acute and long-term time periods and review the currently available effective treatment options for patients with such ocular complications and their consequences. The article describes surgical treatments which enabled to achieve satisfactory anatomical and functional results in patients with the above disorders. Timely treatment of Stevens-Johnson and Lyell's syndromes comprising systemic and topical conservative therapy in combination with surgical procedures during the acute stage plays an important role for the prediction of severe long-term ocular complications. The elimination of ocular complications caused by these syndromes is associated with multistage reconstructive surgery. The authors emphasize that long-term follow-up by ophthalmologist will be required for patients who survived Stevens-Johnson and Lyell's syndromes. Keywords: epidermolytic drug reactions, Stevens–Johnson syndrome, Lyell's syndrome, amniotic membrane transplantation, dry eye syndrome, pseudomembranous blepharoconjunctivitis, multistage reconstructive surgery. For citation: Chernysh V.F., Haritonova N.N., Kulikov A.N. et al. Current surgical treatment options for ocular complications of Stevens- Johnson and Lyell's syndromes. Russian Journal of Clinical Ophthalmology. 2023;23(2):99–106 (in Russ.). DOI: 10.32364/2311-7729- 2023-23-2-99-106.

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24

Tsai, Wen-Hua, Ru-Jeng Teng, Jan-Show Chu, Mei-Hwei Chang, and Mei-Mei Ho. "Neonatal Dubin-Johnson Syndrome." Journal of Pediatric Gastroenterology and Nutrition 18, no. 2 (1994): 253. http://dx.doi.org/10.1097/00005176-199402000-00023.

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25

Rosenthal, Philip. "Neonatal Dubin-Johnson Syndrome." Journal of Pediatric Gastroenterology and Nutrition 19, no. 2 (1994): 255. http://dx.doi.org/10.1097/00005176-199408000-00021.

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26

Tsai, Wen-Hua. "Neonatal Dubin-Johnson Syndrome." Journal of Pediatric Gastroenterology and Nutrition 19, no. 2 (1994): 255. http://dx.doi.org/10.1097/00005176-199408000-00022.

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27

Daubeney, P. E. F., and J. W. Scopes. "Recurrent Stevens-Johnson Syndrome." Journal of the Royal Society of Medicine 84, no. 3 (1991): 168. http://dx.doi.org/10.1177/014107689108400319.

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28

Schalock, P. C., and J. G. H. Dinulos. ""Atypical" Stevens-Johnson Syndrome?" PEDIATRICS 120, no. 2 (2007): 451–52. http://dx.doi.org/10.1542/peds.2007-1072.

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29

Rosenthal, Philip. "Neonatal Dubin‐Johnson Syndrome." Journal of Pediatric Gastroenterology and Nutrition 19, no. 2 (1994): 255. http://dx.doi.org/10.1002/j.1536-4801.1994.tb11287.x.

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30

Tsai, Wen‐Hua. "Neonatal Dubin‐Johnson Syndrome." Journal of Pediatric Gastroenterology and Nutrition 19, no. 2 (1994): 255. http://dx.doi.org/10.1002/j.1536-4801.1994.tb11288.x.

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31

Tsai, Wen‐Hua, Ru‐Jeng Teng, Jan‐Show Chu, Mei‐Hwei Chang, and Mei‐Mei Ho. "Neonatal Dubin‐Johnson Syndrome." Journal of Pediatric Gastroenterology and Nutrition 18, no. 2 (1994): 253. http://dx.doi.org/10.1002/j.1536-4801.1994.tb11163.x.

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32

Daniela, Guerrero Carrillo. "Stevens Johnson Syndrome Review." International Journal of Medical Science and Clinical Research Studies 04, no. 06 (2024): 1065–68. https://doi.org/10.5281/zenodo.11563174.

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Stevens-Johnson Syndrome (SJS) and Toxic Epidermal Necrolysis (TEN) are dermatologic emergencies that are distinguished by extensive epidermal necrolysis and sloughing. The incidence rates are location-dependent. Mortality rates vary from 4.8-9% to 14.8-48%, with females being more frequently affected than males.   Supportive care, infection control, wound care, and fluid and electrolyte management comprise nonpharmacologic treatment for SJS/TEN patients. The identification and cessation of the causative substance are the most critical components. The management of fluid, electrolytes, and nutrition is essential for SJS/TEN patients, as they have lower fluid requirements than burn patients.   Prophylactic antibiotics do not improve outcomes; however, infection prevention necessitates appropriate wound care and antiseptic handling. The decision to pursue surgical debridement is contingent upon the location of care, and the function of this treatment option has been a subject of controversy. Anti-shear therapy, which entails the preservation of denuded epidermis and the aspiration of blister fluid, has been demonstrated to be effective in decreasing mortality rates.   The disease's rarity has resulted in a scarcity of prospective studies on pharmacologic treatment for SJS/TEN. There have been reports of a variety of treatment regimens that involve corticosteroids, IVIg, cyclosporine, and TNF-alpha inhibitors. However, it is difficult to ascertain whether the disease's remission was caused by a specific treatment or the natural course of the disease.   Studies have demonstrated that supportive care and cyclosporine have the potential to serve as alternative treatments for SJS/TEN. A meta-analysis of 10 studies revealed that patients who received cyclosporine had a survival advantage, while a meta-analysis of 67 studies revealed that the combination of corticosteroids and IVIg resulted in statistically significant improvements in outcomes. The severity of illness scores in patients with SJS/TEN overlap or TEN was reduced by plasmapheresis, which entails plasmapheresis and corticosteroids. In 86.8% of patients, TNF-alpha inhibitors have also demonstrated positive outcomes, despite their immunosuppressive properties. Nevertheless, the most effective pharmacological treatment for SJS/TEN must be determined by taking into account practical factors, such as cost. There is a lack of consensus regarding the most effective treatment, and additional research is required to ascertain the most effective treatment.

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33

Ramayanti, Sri. "MANIFESTASI ORAL DAN PENATALAKSANAAN PADA PENDERITA SINDROM STEVENS-JOHNSON." Majalah Kedokteran Andalas 35, no. 2 (2011): 91. http://dx.doi.org/10.22338/mka.v35.i2.p91-97.2011.

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AbstrakSindrom Stevens-Johnson adalah bentuk penyakit mukokutan dengan tanda dan gejala sistemik yang parah berupa lesi target dengan bentuk yang tidak teratur, disertai macula, vesikel, bula, dan purpura yang tersebar luas terutama pada rangka tubuh. Sindrom Stevens-Johnson mempunyai tiga gelaja yang khas yaitu kelainan pada mata berupa konjungtivitis, kelainan pada genital berupa balanitis dan vulvovaginitis, serta kelainan oral berupa stomatitis. Diagnosis sindrom Stevens-Johnson terutama berdasarkan atas anamnesis, pemeriksaan klinis dan pemeriksaan penunjang perawatan pada penderita sindrom Stevens-Johnson lebih ditekankan pada perawatan simtomatik dan suportif karena etiologinya belum diketahui secara pasti.Kata Kunci : Sindrom Stevens-Johnson, manifestasi oral, manajemen perawatanAbstractStevens-Johnson Syndrome is a mucocutaneous disease with severe signs and symptoms of systemic form of the target lesion with an irregular shape, with the macula, vesicles, bullae, and widespread purpura, especially in the framework of the body. Stevens-Johnson syndrome have a typical three gelaja disorders of the eye such as conjunctivitis, genital abnormalities in the form of balanitis and vulvovaginitis, as well as oral abnormalities such as stomatitis. Stevens-Johnson syndrome diagnosis is mainly based on history, clinical examination and investigations treatment in patients with Stevens-Johnson syndrome were focused on symptomatic and supportive care because its etiology is not known with certainty.Key word : Stevens-Johnson syndrome, oral manifestation, treatment managementTINJAUAN PUSTAKA

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34

Lusiana, Ika, and Felix Nathan Trisnadi. "Sindroma Steven-Johnson pada Anak Akibat Penggunaan Carbamazepin: Sebuah Laporan Kasus." Jurnal Surya Medika 9, no. 2 (2023): 81–84. http://dx.doi.org/10.33084/jsm.v9i2.5668.

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Steven-Johnson syndrome is rare occurance. In children, the incidence of Steven Johnson Syndrome (SJS) is lower than adults. Steven Johnson Syndrome is often caused by a hypersensitivity reaction to drugs, including carbamazepine. Correct and appropriate therapy management will support the improvement of conditions and reduce mortality

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35

Dr., Prarthana Kishore, Raghavendra Kini Dr., Meghana HC Dr., et al. "Steven Johnsons Syndrome Induced by Carbamezapene." International Journal of Innovative Science and Research Technology 8, no. 4 (2023): 2007–10. https://doi.org/10.5281/zenodo.8020743.

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Stevens – Johnson syndrome (SJS) is characterized by rapidly spreading blisters which occur in the form of macules or target lesions which has clinical features which may be potentially fatal. It is usually associated with drugs such as anticonvulsants including carbamazepine, lamotarigine, phenobarbotal, phenytoin and valproic acid. This is a rare case report in which we describe a case of Steven Johnsons Syndrome induced by Carbamezapene that was prescribed to treat trigeminal neuralgia affecting the oro-facial region.

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36

Ghosh, Somdatta, and Supreeti Biswas Mondal. "Carbamazepine Induced Stevens Johnson Syndrome - A Case Report." International Journal of Science and Research (IJSR) 14, no. 2 (2025): 416–18. https://doi.org/10.21275/sr25206094227.

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37

Rozenbajgier, Martyna, Justyna Wójcik-Grudzień, Paulina Pawłowska, and Alicja Ozga-Stachurska. "Stevens–Johnson Syndrome and Toxic Epidermal Necrolysis, A Review of Pathogenesis, Clinical Features, Diagnosis and Treatment." Journal of Education, Health and Sport 12, no. 9 (2022): 512–18. http://dx.doi.org/10.12775/jehs.2022.12.09.060.

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Stevens Johnson syndrome (SJS) and toxic epidermal necrolysis (TEN) are rare, acute conditions, potentially life-threatening, immune-mediated and often unpredictable. Characteristic for SJS and TEN is acute necrosis of the epidermis and mucous membranes, caused by the extensive death of keratinocytes. These syndromes are considered hypersensitivity reactions. They are most often caused by drugs. There have also been reports of SJS / TEN being caused by infection, SJS/TEN disease is very rare and due to its rarity there is no specific pharmaceutical algorythm. Supportive care and treatment of symptoms are very important. The most crucial part of non-pharmacologic treatment of SJS/TEN is the detection and cessation of the pharmaceutical that caused the disease. The aim of this literature review was to summarize current knowledge about the pathogenesis, clinical features, diagnosis and treatment of Stevens- Johnson Syndrom and Toxic Epidermal Necrolysis. Standard criteria were used to review the literature data. The search of articles in the PubMed and Google Scholar database was carried out using the following keywords: Stevens-Johnson Syndrome, Toxic Epidermal Necrolysis, cutaneous adverse drug reactions

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38

Roka, Yam Bahadur, Sabrina Shrestha, Narayani Roka, and Mohan Karki. "Stevens-Johnson Syndrome in a Child on Phenytoin, Exacerbated with Cefixime." Journal of Nepal Paediatric Society 39, no. 3 (2019): 193–96. http://dx.doi.org/10.3126/jnps.v39i3.24998.

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Steven Johnson syndrome and toxic epidermal necrolysis are rare but potentially life threatening muco-cutaneous disorders. Their incidence ranges from 1.2 to six per million patient-years for Steven Johnson syndrome and 0.4 to 1.2 per million patient-years for toxic epidermal necrolysis. Drugs are the primary cause for these syndromes in majority cases. They might also be due to infections with Mycoplasma Pneumoniae or Herpes Simplex. The mortality ranges from five to 40% in these cases. We report a 10-year old girl who presented with history of multiple skin eruptions involving whole body and oral ulceration for five days. She was a known case of seizure disorder on phenytoin and had been prescribed Cefexime for fever. She was managed with intravenous fluids, corticosteroids, opiates, antacids and topical antibiotics. We want to highlight the possibility of Steven Johnson syndrome following the combination of these two drugs.

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39

Stewart, Michael G., Newton O. Duncan, Daniel J. Franklin, Ellen M. Friedman, and Marcelle Sulek. "Head and Neck Manifestations of Erythema Multiforme in Children." Otolaryngology–Head and Neck Surgery 111, no. 3P1 (1994): 236–42. http://dx.doi.org/10.1177/01945998941113p112.

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Erythema multiforme, Stevens-Johnson syndrome, and toxic epidermal necrolysis are related disorders of skin and mucous membranes, which are typically associated with antecedent medication use or infection. We review 108 cases of erythema multiforme, Stevens-Johnson syndrome, and toxic epidermal necrolysis from Texas Children's Hospital, Houston, Texas, from 1981 to 1991, and illustrate the characteristic skin and mucosal lesions. In addition, we describe in detail two unusual cases requiring intensive airway management. Head and neck manifestations were present in 4 of 79 patients (5%) with erythema multiforme and 26 of 28 patients (93%) with Stevens-Johnson syndrome. In Stevens-Johnson syndrome, mucosal involvement of the lip (93%), conjunctiva (82%), oral cavity (79%), and nose (36%) were most common. Antecedent medication use was identified in 59% of erythema multiforme patients and 68% of Stevens-Johnson syndrome patients. We note a striking increase in the number of cases in our series caused by cephalosporins. Fifty percent of Stevens-Johnson syndrome patients required supplemental hydration or alimentation because of the severity of the oral cavity involvement. The head and neck mucosal manifestations largely respond to local care, and the routine use of prophylactic antibiotics or systemic steroids is not recommended.

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40

Laurencin, Cato T., Richard F. Horan, Patrick B. Senatus, Clara B. Wheeler, and Stephen J. Lipson. "Stevens-Johnson-Type Reaction with Vancomycin Treatment." Annals of Pharmacotherapy 26, no. 12 (1992): 1520–21. http://dx.doi.org/10.1177/106002809202601206.

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OBJECTIVE: To report a case of Stevens-Johnson syndrome caused by vancomycin. CASE SUMMARY: Stevens-Johnson syndrome is an acute mucocutaneous process characterized by epidermal and mucosal desquamation. Its pathogenesis is poorly understood. Mortality rates have ranged from 30 to 100 percent. We describe a case of Stevens-Johnson syndrome related to the use of vancomycin in a 71-year-old woman with rheumatoid arthritis receiving treatment for an infected cervical fusion site. Classic “target” lesions distributed throughout the trunk and extremities along with erosive lesions involving the oral and vaginal mucosae were observed in this patient. DISCUSSION: A number of agents have been implicated in the etiology of Stevens-Johnson syndrome. Serious cutaneous reactions to vancomycin, however, have been uncommon. Cessation of vancomycin treatment in our patient led to eventual resolution of her symptoms. CONCLUSIONS: Vancomycin is a potential causative agent of Stevens-Johnson syndrome.

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Cooper, Karen L. "Drug Reaction, Skin Care, Skin Loss." Critical Care Nurse 32, no. 4 (2012): 52–59. http://dx.doi.org/10.4037/ccn2012340.

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Stevens-Johnson syndrome is a rare, potentially fatal drug reaction that causes necrosis of epidermal cells. Early recognition of the syndrome is essential to prevent complications. This article discusses identification, complications, and treatment of Stevens-Johnson syndrome.

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Muci, Manisa, and Numila Kuneshka. "Stevens-Johnson syndrome: case report." Paediatria Croatica 57, no. 3 (2013): 268–70. http://dx.doi.org/10.13112/pc.676.

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Stevens-Johnson syndrome is a rare and life-threatening acute allergic reaction aff ecting the skin and mucous membranes. We discuss the clinical presentation, evaluation, and treatment of a 9-year-old girl with Stevens-Johnson syndrome, which occurred due to the antiepileptic drug carbamazepine (Tegretol™).

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Demissie, Zekewos. "The First Ethiopian Case Report of Post-Covid-19 Vaccine Associated Guillain-Barre Syndrome." Neurons and Neurological Disorders 1, no. 1 (2022): 01–04. http://dx.doi.org/10.58489/2836-8851/002.

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We report the first Ethiopian case of a rare neurologic complication, Guillain-Barre Syndrome (GBS), following Ad26.COV2. S (Janssen/Johnson & Johnson) COVID-19 vaccination. Although a temporal relationship is identified, a causal relationship between the Janssen/Johnson & Johnson COVID-19 vaccine and GBS is not established. Clinician awareness of this association is essential to early GBS diagnosis and treatment. Further, global data collection is important to ascertain the true magnitude and possible causality of post-COVID-19 vaccine associated GBS.

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Athul Gopan, Anukrishna V P, Devina Janeendran, Athulya Subhash, Jacob Thomas, and Remya Reghu. "Allopurinol Induced Stevens-Johnson Syndrome." International Journal of Research in Pharmaceutical Sciences 12, no. 1 (2021): 829–31. http://dx.doi.org/10.26452/ijrps.v12i1.4189.

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Stevens-Johnson Syndrome (SJS) is an acute, self-limited, rare but life-threatening disease that manifests as severe mucocutaneous blistering and erosions. Here we report a rare case of allopurinol-induced SJS. A 25-year-old male patient with no other comorbidities was admitted to the hospital with complaints of fever, redness of eyes, swelling of lips with discharge and crusting, extensive erosions in the oral mucosa for the last 4 days, following consumption of allopurinol for a duration of 1 month. Investigations were within normal limits. The offending drug was withdrawn and he was treated with corticosteroids, antimicrobials, and other supportive measures. Allopurinol, a Xanthine oxidase inhibitor is mostly used for the treatment of primary and secondary hyperuricemia, Health care professionals must be aware of the spectrum of adverse effects of this drug and must take urgent measures once the diagnosis is suspected especially to save the patient from such severe or fatal reactions like SJS/Toxic epidermal necrolysis (TEN).

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Batouk, Entesar, Abdulmonem Almutawa, Abdullah AlSonbul, and Sulaiman AlMayouf. "Chronic Recalcitrant Stevens-Johnson Syndrome." Annals of Paediatric Rheumatology 1, no. 1 (2012): 77. http://dx.doi.org/10.5455/apr.111520112116.

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Kendirli, Mustafa Tansel, Halit Yasar, and Mehmet Guney Senol. "Carbamazepine Related Stevens-Johnson Syndrome." Journal of the Turkish Epilepsi Society 19, no. 1 (2013): 38. http://dx.doi.org/10.5505/epilepsi.2013.74936.

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Gungam, Praveena, Pruthvi Desireddy, Balakrishna Namala, and Souris Kondaveti. "ACYCLOVIR INDUCED STEVEN JOHNSON SYNDROME." Journal of Evidence Based Medicine and Healthcare 2, no. 15 (2015): 2363–66. http://dx.doi.org/10.18410/jebmh/2015/336.

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Kim, Jae-Wang. "Lamotrigine-induced Stevens-Johnson syndrome." Journal of Medicine and Life Science 7, no. 1 (2010): 157–60. http://dx.doi.org/10.22730/jmls.2010.7.1.157.

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Lamotrigine is a new antiepileptic drug which is effective for partial or generalized seizure. However, administration of lamotrigine alone or in combination with valproic acid may lead to fatal Stevens-Johnson Syndrome(SJS) or toxic epidermal necrolysis(TEN). Although there are many published case reports of lamotrigine-induced SJS or TEN in Western countries, there have been few cases in the Korean Iiterature until now. Herein, we present a case of SJS associated with LTG.

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Nedelcuta, Ramona Mihaela, Vlad Dumitru Baleanu, Dragos Virgil Davitoiu, Tiberiu Stefanita Tenea Cojan, Cosmin Alexandru Ciora, and Gigi Calin. "Steven-Johnson Syndrome in Child." Revista de Chimie 70, no. 9 (2019): 3213–15. http://dx.doi.org/10.37358/rc.19.9.7519.

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Steven-Johnson Syndrome and NET (toxic epidermal necrolysis), different expressions of the same condition were named according to the percentage of affected skin: [10% in Steven-Johnson Syndrome and]30% in NET. Small ages may be a target for these rare diseases, in direct relation to drug abuse (antibiotics, NSAIDs, antiepileptics, etc.). Survival depends on the rapid response of the causative agent, the early investigation of the therapy to block Fas-FasL interaction and to reestablish skin integrity (Fas-receptor on the surface of cells involved in apoptosis). Massive skin damage is associated with a severe prognosis, as is the age of the baby.It is necessary to use wise the medication or automedication for a complete and effective prevention of Steven-Johnson Syndrome.

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Prajapati, Himani, Neetu Bala, and Dinesh Kansal. "Nevirapine induced Stevens Johnson syndrome." Journal of Pharmacovigilance and Drug Research 1, no. 2 (2020): 35–38. http://dx.doi.org/10.53411/jpadr.2020.1.2.6.

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Introduction: Severe and life-threatening SJS is more common with nevirapine than with other NNRTIs Case presentation: A 56-year-old male patient presented with a complaint of lesions all over the body with a burning sensation for 4 days. He was on an ART regimen, containing zidovudine, lamivudine, and efavirenz combination for 4 years. But patient accidentally started nevirapine and after 10 days he developed maculopapular lesions which were diagnosed as SJS syndrome. There was a history of rash with nevirapine when ART was started initially in 2012. This incident of an adverse event could be assigned a term "probable" according to the WHO-UMC scale for causality assessment as the re-challenge was found positive. Conclusion: Physicians and patients must be aware of this adverse effect on early diagnosis and treatment.

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