Relevant bibliographies by topics / KASUMI

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Academic literature on the topic 'KASUMI'

Author: Grafiati
Published: 4 June 2021
Last updated: 5 February 2022

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Journal articles on the topic "KASUMI"

1

Jiang, Xuewei, Pan Zengkai, Chen Jin, Yu Pengfei, and Li-Gen Liu. "Establishment of TRAIL-Resistance Kasumi-1 Cell Line and the Analysis of It different mRNA Expression Profile with the Original Kasumi-1 Cell Line." Blood 124, no. 21 (December 6, 2014): 5221. http://dx.doi.org/10.1182/blood.v124.21.5221.5221.

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Abstract Introduction Tumor necrosis factor related apoptosis inducing ligand (TRAIL) can induce the apoptosis of many human leukemia cells while sparing of normal cells, but its resistance is also universal. Our previous study on apoptosis of t(8;21) positive acute myeloid leukemia cell line Kasumi-1 induced by rhTRAIL showed that the survival rate no longer decreased significantly when rsTRAIL reached a certain concentration which implied Kasumi-1 cells might have a resistant tendency to TRAIL. Then, we established a TRAIL-resistant Kasumi-1 cell line (Kasumi-1 TR) by intermittently escalating rsTRAIL concentration in culture media, and compared the mRNA expression profile with the original Kasumi-1 cell line by using Affymetrix Human Genome U133 Plus 2.0 Array. Methods Kasumi-1 TR cell line was established by intermittently treated Kasumi-1 cells with progressively escalating rsTRAIL concentration. Proliferation of leukemia cells were measured by CCK-8 assay, and rsTRAIL IC50 of cells and resistance index were calculated according to proliferation of cells treated with rsTRAIL at different concentrations. TRAIL and TRAIL receptors 1-4 on cells surface were detected by flow cytometry. Expression profiles of Kasumi-1 cells and Kasumi-1 TR cells were analyzed by Affymetrix Human Genome U133 Plus 2.0 Array to identify differentially expressed genes, and the search of genes possibly related with TRAIL-resistance were using by GO functional analysis and pathway enrichment analysis. Results 1) Kasumi-1 TR cells proliferation was faster than that of Kasumi-1 cells(Fig 1A); 2) IC50 of 24 hours for Kasumi-1 cells was 756.833ng/ml (logIC50 2.879 ± 0.148), IC50 of 24 hours for Kasumi-1 TR cells was 1634646.005ng/ ml (logIC50 6.213 ± 0.637), the RI of 24h was 2159 (Fig 1B); IC50 of 48 hours for Kasumi-1 cells was 345.390ng/ml (logIC50 2.538 ± 0.153), IC50 of 48 hours for Kasumi-1 TR cells was 33642.641ng/ml (logIC50 is 4.257 ± 0.317), the RI for 48h was 97 (Fig 1C); 3) Cell surface expression of TRAIL and its receptors 1-4 had no difference between two cell lines(Fig 1D). 4) There were 1537 genes up regulated by more than 2 times while 487 genes down regulated by more than 2 times in Kasumi-1 TR cells compared with the original Kasumi-1 cells (Fig 1E). Of which BCL-2 family antiapoptotic gene BCL2 is increased by 3.153 times and BCL2A1 increased by 18.23 times, IFNAR1 involved in JAK/STAT pathway increased by 12.841 times and TRAIL death receptor TNFRSF10A down regulated by 3.256 times(Fig 1F). Conclusions: The Kasumi-1 cell line with rsTRAIL resistance (Kasumi-1 TR) is established, and its resistance may be associated with the up expression of BCL2, BCL2A1, IFNAR1 and down regulated expression of DR4. Acknowledgment This work was supported by grants from NSFC (30672415) and STCSM (054119528). Figure 1 Figure 1. Disclosures No relevant conflicts of interest to declare.
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2

平井一博. "A Study of「kasumi」「kasumu」after『The Tale of Genji』." Journal of Japanese Culture ll, no. 36 (February 2008): 211–23. http://dx.doi.org/10.21481/jbunka..36.200802.211.

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3

Imai, Norikazu, Masato Shikami, Hiroshi Miwa, Akiko Hattori, Akihito Hiramatsu, Masaya Watarai, Atsushi Satoh, et al. "Serum Dependency of t(8;21) AML Cell Line Is Associated with VEGF/VEGFR Pathway and Early Phosphorylation of Akt." Blood 106, no. 11 (November 16, 2005): 4571. http://dx.doi.org/10.1182/blood.v106.11.4571.4571.

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Abstract Most human leukemia cell lines are dependent on serum supplementation (usually fetal calf serum (FCS)), although the extent of serum dependency differs among each cell line. Kasumi-1, a t(8;21) AML cell line is one of the most serum-dependent cell lines. Since growth and survival of many leukemia cell lines are associated with phosphorylation of Akt, we examined the Akt phosphorylation by FCS treatment. In Kasumi-1, Akt was phosphorylated by culture with FCS in a dose-dependent manner, although no such Akt phosphorylation was observed in NB-4, a t(15;17) cell line. By FCS stimulation, Akt (Thr308, Ser473) was phosphorylated from 0.5 hr and the phosphorylation sustained until 48 hours in Kasumi-1. Then, we tested the effect of VEGF/VEGFR signaling in phosphorylation of Akt by FCS. The addition of VEGFR1/Fc and VEGFR2/Fc (which bind external VEGF and abrogate its function) inhibited the Akt phosphorylation from 2 hours until 10 hours, although the growth of Kasumi-1 was not inhibited. The addition of VEGFR2 kinase inhibitor (which inhibits internal VEGF signal) inhibited the Akt phosphorylation from 0.5 hr until 2 hours, and the growth of Kasumi-1 was greatly inhibited. Taken together, it is suggested that serum dependency of Kasumi-1 is at least in part attributed to VEGF/VEGFR pathway. Then, both external and internal VEGF/VEGFR pathways work in Kasumi-1, which in turn phosphorylate Akt. However, blockade of only internal VEGF signal (by VEGFR2 kinase inhibitor) inhibit the early Akt phosphorylation (0.5 hr), which resulted in growth inhibition, indicating the importance of early Akt phosphorylation.
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4

Kobune, Masayoshi, Kazuyuki Murase, Satoshi Iyama, Tsutomu Sato, Yutaka Kawano, Shohei Kikuchi, Koji Miyanishi, Rishu Takimoto, and Junji Kato. "Hedgehog Inhibitors Reduce the Survival and Drug Resistance in CD34+ Leukemic Cells." Blood 112, no. 11 (November 16, 2008): 1611. http://dx.doi.org/10.1182/blood.v112.11.1611.1611.

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Abstract Aberrant reactivation of Hedgehog (Hh) signaling has been described in a wide variety of human cancers and its role in maintenance of self-renewal of cancer stem cells. We previously showed that Indian Hh (Ihh) and its receptor molecules, Patched and Smoothened are expressed in cord blood (CB) CD34+ cells and Ihh regulate proliferation of hematopoietic myeloid- and lymphoid-repopulating cells in vivo. However, the involvement of Hh signaling in proliferation of leukemic cells has remained unclear. In this study, we assessed the possibility that Hh pathway activation contributes to the growth, survival and drug resistance of acute myeloid leukemic (AML) cells. Hh signaling in leukemic cell lines and bone marrow (BM) CD34+ cells were screened by RT-PCR and a Hh signaling reporter assay. We have found that Ihh were expressed in BM CD34+ cells and most of human AML cell lines, HL60, U937, KG1, Kasumi-1, Kasumi3 and TF-1. In contrast, Hh receptor components, Patched and Smoothened, were detected in BM CD34+ cells and cytokine responsible CD34+ AML cell lines such as Kasumi-1, Kasumi-3 and TF-1. Moreover, the downstream transcription factor GLI1 or GLI2 were expressed in BM CD34+ cells and these CD34+ cell lines, indicating that Hh signaling was active in these cells. We further assessed whether Hh signaling transmit to GLI1 using GLI1-responsive luciferase assay. GLI-responsive reporter plasmid (TK-6GBS-Luc) was transduced into these cells in the presence or absence of anti-Hh neutralizing antibody 5E1. TK-6GBS-Luc-transduced Kasumi-1, Kasumi-3 and TF-1 cells showed high luciferase activity. Furthermore, the luciferase activity of these cells was significantly reduced in the presence of 10 μg/ml anti-Hh neutralizing antibody. These results clearly indicate that Ihh signaling transmits into these CD34+ leukemic cell lines in autocrine manner. We next examined the effect of Hh inhibitors on these CD34+ leukemic cell lines. Inhibition of Hh signaling with the naturally derived chemical Smoothened antagonist Cyclopamine, endogenous Hh inhibitor Hedgehog-interacting protein or anti-Hedgehog neutralizing antibody reduces the entry of cell cycle of CD34+ cell lines and eventually induces apoptosis in these cells after 48 hr exposure although these CD34+ cell lines exhibit resistance to cytarabine (Ara-C) exposure. In contrast, cyclopamine did not affect growth and survival of the U937 or HL-60 cell line that lacks expression of Hh receptor components. Furthermore, combination of 10 μM cyclopamine significantly reduced the drug resistance against Ara-C in CD34+ cell lines. These results suggest that Hh pathway activation is a feature of CD34+ myeloid leukemic cells and inhibition of Hh signaling pathway may be a therapeutic strategy in the treatment of AML.
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5

Berg, Tobias, Mahmoud Abdelkarim, Yalin Guo, Manfred Fliegauf, and Michael Lübbert. "AML1/ETO Expresssion in Myeloid Leukemia Cells Is Associated with Enhanced Growth-Inhibitory and P15/INK4b Demethylating Effects of 5-AZA-2′-Deoxycytidine." Blood 104, no. 11 (November 16, 2004): 1165. http://dx.doi.org/10.1182/blood.v104.11.1165.1165.

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Abstract The chromosomal translocation (8;21) results in the expression of the chimeric transcription factor AML1/ETO, the most frequent fusion gene in acute myeloid leukemia. The AML1/ETO fusion protein acts as a transcriptional repressor by mediating epigenetic silencing through recruitment of histone deacetylases. Recently, it was shown that it also mediates gene silencing by associating with DNA methyltransferase (Dnmt). We therefore hypothesized that cells expressing AML1/ETO might be preferentially sensitive to the effects of an inhibitor of Dnmt activity, and might provide a superior model for in vitro demethylation and reactivation of the promoter of the p15/INK4b gene (encoding a negative regulator of the cell cycle) that is frequently methylated and silenced in AML and MDS. The 3 myeloid cell lines Kasumi-1 cells (AML1/ETO-positive), KG-1, and KG-1a (both AML1-ETO-negative) are all bearing a heavily methylated p15/INK4b promoter. They were treated with 50 – 1000 nM 5-aza-2′-deoxycytidine (DAC) for three pulses of 24 hrs each. After 6 days, cell growth and viability were determined and FACS analysis performed after propidium iodide staining. Kasumi-1 showed the highest sensitivity to DAC treatment (growth inhibition at 500 nM DAC: Kasumi-1 74.28 %, KG-1 69.16 %, KG-1a 62.38 %). In addition, DAC treatment (500 nM) led to a stronger increase in the sub-G1 fraction in Kasumi-1 (30.46%) compared to KG-1a (20.84 %). Regional p15/INK4b promoter methylation was assessed quantitatively by bisulfite sequencing of ≤10 individual cloned alleles (containing 21 CpGs residues) for calculation of methylated CpG percentage. The p15/INK4b was highly methylated in all 3 cell lines (methylated CpGs Kasumi-1 95.2 %; KG-1 89.6 %; KG-1a 98.4 % ). In Kasumi-1 cells, treatment with DAC resulted in a striking, dose-dependent regional demethylation of the p15/INK4b promoter (demethylated CpGs at 200 nM of DAC: Kasumi-1 63.8 %, KG-1 48.9 %, KG-1a 9.3 %). No demethylating effect was achieved with equitoxic doses of cytarabine or melphalan. Effective demethylation of the p15/INK4b promoter was associated with p15/INK4b protein induction as determined by Western Blot. Simultaneous treatment with all-trans retinoic acid (ATRA) enhanced the effects of DAC treatment upon growth inhibition, but not upon p15/INK4b induction. U937 cells with ecdysone inducible AML1/ETO expression (Fliegauf et al, Oncogene 2004) were also treated with different doses of DAC. When AML1/ETO was induced, U937 cells showed a higher growth inhibition (U937 + AML1/ETO 38.6 %, U937 - AML1/ETO 18 % at 25 nM) and increase in Sub-G1 (U937 + AML1/ETO 18 %, U937 - AML1/ETO 10.55 % at 100 nM) after treatment with DAC. Our results imply that the growth-inhibitory and proapoptotic effect of DAC on leukemia cells is modulated by AML1/ETO protein (or its target genes). The greater accessibility of the p15/INK4b promoter to the demethylating effect of DAC in AML1/ETO expressing Kasumi-1 cells may also be due to differences in regional chromatin structure. With their differential sensitivity to DAC, the cell lines Kasumi-1 and KG-1a provide a model for the different responses of leukemic blasts to DAC.
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6

KITSOS, P., M. D. GALANIS, and O. KOUFOPAVLOU. "AN FPGA IMPLEMENTATION OF THE GPRS ENCRYPTION ALGORITHM 3 (GEA3)." Journal of Circuits, Systems and Computers 14, no. 02 (April 2005): 217–31. http://dx.doi.org/10.1142/s0218126605002337.

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The General Packet Radio Service (GPRS) uses the GPRS Encryption Algorithm 3 (GEA3) for data encryption. In this paper, alternative hardware implementations of the GEA3 algorithm are described. GEA3 algorithm is based on the KASUMI block cipher. Various KASUMI block cipher hardware implementations have been examined in order to provide information about the required silicon area and throughput. In order to achieve a significant performance improvement, Double Edge Triggered pipeline technique is used. The S-BOXes, which are fundamental elements of the KASUMI cipher, have been implemented by using combinational logic and ROM memories. The proposed GEA3 algorithm hardware implementation achieves throughput up to 837Mbps, which is much faster comparing to the previous designs. The whole system is implemented and evaluated by using Field Programmable Gate Array (FPGA) devices.
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7

O'Connor, Christine M., and Eain A. Murphy. "A Myeloid Progenitor Cell Line Capable of Supporting Human Cytomegalovirus Latency and Reactivation, Resulting in Infectious Progeny." Journal of Virology 86, no. 18 (July 3, 2012): 9854–65. http://dx.doi.org/10.1128/jvi.01278-12.

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Human cytomegalovirus (HCMV) is a herpesvirus that establishes a lifelong, latent infection within a host. At times when the immune system is compromised, the virus undergoes a lytic reactivation producing infectious progeny. The identification and understanding of the biological mechanisms underlying HCMV latency and reactivation are not completely defined. To this end, we have developed a tractablein vitromodel system to investigate these phases of viral infection using a clonal population of myeloid progenitor cells (Kasumi-3 cells). Infection of these cells results in maintenance of the viral genome with restricted viral RNA expression that is reversed with the addition of the phorbol ester 12-O-tetradecanoylphorbol-13-acetate (TPA, also known as PMA). Additionally, a latent viral transcript (LUNA) is expressed at times where viral lytic transcription is suppressed. Infected Kasumi-3 cells initiate production of infectious virus following TPA treatment, which requires cell-to-cell contact for efficient transfer of virus to other cell types. Importantly, lytically infected fibroblast, endothelial, or epithelial cells can transfer virus to Kasumi-3 cells, which fail to initiate lytic replication until stimulated with TPA. Finally, inflammatory cytokines, in addition to the pharmacological agent TPA, are sufficient for transcription of immediate-early (IE) genes following latent infection. Taken together, our findings argue that the Kasumi-3 cell line is a tractablein vitromodel system with which to study HCMV latency and reactivation.
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8

Sun, Jin, Shujun Liu, Jianhua Yu, Min Wei, Charlene Mao, Haiming Ding, Jessica Kearney, et al. "Characterization of HDACI OSU42 as a Novel Histone Deacetylase Inhibitor in AML Cell Lines." Blood 108, no. 11 (November 16, 2006): 1988. http://dx.doi.org/10.1182/blood.v108.11.1988.1988.

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Abstract Histone acetylation plays a key role in the regulation of gene expression. Histone hyperacetylation is associated with chromatin opening and gene transcription, while histone hypoacetylation is associated with chromatin condensation and gene silencing. Abnormal histone hypoacetylation mediated by aberrant activity of histone deacetylases (HDACs) has been found to be associated with silencing of tumor suppressor and growth inhibitory genes in malignant cells. HDAC inhibitors (HDACIs) can relieve HDAC-mediated gene silencing and thereby induce normal patterns of cell cycle, differentiation and apoptosis in malignant cells. HDACI OSU 42 is a novel hydroxamate tethered phenylbutyrate derivative that was designed and synthesized at our institution, and exhibited IC50s at submicromolar level, compared with millimolar level for other members of this classes of HDACIs such as valproic acid (VPA). We characterized the activity of this compound in acute myeloid leukemia (AML) cells. It is known that the fusion proteins AML1/ETO and PML / RAR alpha that characterized t(8;21) and t(15;17) AML silence target genes through recruitment of HDACs to their promoter regions. Therefore we utilized AML1/ETO-positive Kasumi-1 and PML/RARA-positive NB4 cells to test the activity of HDACI OSU 42 and used THP-1 cells, characterized by AF9/MLL fusion gene, as a control. We hypothesized that by virtue of the fusion genes, Kasumi-1 and NB4 are more susceptible to HDACI treatment. IC50s for proliferation inhibition in Kasumi-1 cells treated with HDACI OSU42 were 71.8±14.3nM for 24hr and 31.3± 0.4nM for 48hr, significantly lower than VPA (2.0mM for 24hr, 0.9mM for 48hr). The IC50s for NB4 were 237.7±6.5nM for 24hr and 119±6.4nM for 48hr. As a contrast, IC50 for THP-1 was 507.3±68.3nM for 48hr. HDACI OSU42 inhibited 80% of total HDAC activity at 125nM in both Kasumi-1 and NB4; 30nM HDACI OSU42 induced hyperacetylation of histone H3 and H4. Apoptosis analysis showed that nearly 60% more of Kasumi-1 and NB4 underwent apoptosis after treated with 1μM of HDACI OSU42 for 24hr, compared with their untreated control. On the other hand, the same treatment only induced 15% more of THP-1 undergoing apoptosis. Apoptotic effect of HDACI OSU42 was mediated by activation of caspase 9 and caspase 3. Cell cycle analysis demonstrated that treatment of Kasumi-1 and NB4 with 150nM of HDACI OSU 42 inhibited cell cycle progression and arrested 20% to 30% more cells at S phase or G2/M phase, whereas this treatment had not effect on cell cycle progression of THP-1. This was consistent with the up-regulated expression of p21 at both transcription level and protein level. Q-PCR data suggested that Kasumi-1 and NB4 treated with HDACI OSU42 expressed ~10 folds of p21 higher than untreated cells. Chromatin immunoprecipitation assay revealed 10 to 50 folds increase in acetylation level of histone H3 and H4 associated with p21 promoter. Kasumi-1 and NB4 cells also show differentiation ability (increase in CD14 and CD 13 expression by flow cytometry) when treated with 30nM of HDACI OSU42, whereas THP-1 remained undifferentiated. These results support the activity of HDACI OSU42 as a new potent HDACI in AML.
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Mahmud, Hasan, Frank JG Scherpen, Tiny Meeuwsen-de Boer, Harm-Jan Lourens, and Eveline S. de Bont. "Essential Role for Phospholipase C Gamma 1 (PLC-γ1) in the Survival of t(8;21) Acute Myeloid Leukemia." Blood 128, no. 22 (December 2, 2016): 1699. http://dx.doi.org/10.1182/blood.v128.22.1699.1699.

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Abstract The t(8;21) (q22;q22) chromosomal translocation is one of the most frequent genetic alterations in acute myeloid leukemia (AML) and it remains a significant clinical problem especially for children which indicates the need for improved therapeutic strategies. Recently, we showed that peptide derived phospholipase C gamma 1 (PLC-γ1) was highly phosphorylated in pediatric t(8;21) AML. In this study, we determined PLC-γ1 phosphorylation and mRNA levels showing that PLC-γ1 expression was significantly higher in t(8;21) AML compared to other AML karyotypes and normal bone marrow (NBM) (peptide phosphorylation: p<0.01 compared to NBM, mRNA: p<0.001, compared to other AML karyotypes). This was confirmed by PLC-γ1 protein phosphorylation using primary AML samples and AML cell lines. PLC-γ1 is known to play a role in cancer progression, however, the impact of PLC-γ1 in AML is currently unknown. Therefore, we aimed to study the functional role of PLC-γ1 by investigating the cellular growth, survival and its underlying mechanism in a t(8;21) AML cell line (Kasumi-1) . ShRNA-mediated knockdown of PLC-γ1 in kasumi-1 cells significantly blocked leukemic cell growth at day 8 after transduction (p<0.05). The percentage of apoptosis in PLC-γ1 suppressed kasumi-1 cells at day 4 after transduction was two-fold higher compared to the scrambled control (p<0.01). The inhibition of cell proliferation and the induction of apoptosis upon PLC-γ1 suppression could be explained by cell cycle arrest and by increased activation of apoptotic related and cell cycle regulatory protein expressions (BAX, BCL2, p53 and Chk2). As the multidrug resistance is one of the major cause of relapse and poor prognosis in t(8;21) AML, therefore, we demonstrated, if PLC-γ1 suppression increased the sensitivity of kasumi-1 leukemia cells to cytotoxic chemotherapeutic agents (methotrexate, amsacrine and etoposide). PLC-γ1 knockdown cells at day 4 after transduction were shown to significantly reduced cell viability to the genotoxic agents, methotrexate (p<0.05, p<0.001), amsacrine (p<0.01, p<0.001) and etoposide (p<0.05, p<0.01 and p<0.001) in kasumi-1 in a dose-dependent manner. These results provide a strong rationale for the development of PLC-γ1-based therapeutic strategies for the enhancement of efficacy in t(8;21) AML treatment. Additionally, PLC-γ1 suppressed kasumi-1 cells showed significantly less proliferation upon hypoxic stress. Taken together, these results strongly support an important role for PLC-γ1 in the survival of t(8;21) AML mimicking kasumi-1 cell line and identify PLC-γ1 as a potential target for t(8;21) AML treatment. Disclosures No relevant conflicts of interest to declare.
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Liu, Shujun, Jiuxia Pang, Jianhua Yu, Zhongfa Liu, Lenguyen Huynh, Jessica Kearney, Peter Paschka, et al. "Bortezomib-Induced Down-Regulation of KIT Is Mediated by Inhibition of Sp1 and NF-kB in AML1/ETO-Positive Cells." Blood 108, no. 11 (November 16, 2006): 4211. http://dx.doi.org/10.1182/blood.v108.11.4211.4211.

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Abstract Activating mutations of KIT, encoding a type III receptor tyrosine kinase, are frequently detected in core binding factor AML (i.e., AML1/ETO and CBFB/MYH11 AML), promote cell survival and proliferation of leukemic cells and predict poor outcome. Kinase inhibitors (e.g., imatinib or PKC-412) have been shown to block constitutively activated KIT. However, novel therapeutic approaches that target mutated KIT are necessary, since resistance to these agents can be predicted in a substantial proportion of patients in these subgroups of AML. We observed that expression levels of KIT in AML1/ETO-positive Kasumi-1 cells are more than 25 fold higher than in AML1/ETO-negative AML cell lines (i.e., THP-1, K562, and MV4–11). We also found that in Kasumi-1 cells, bortezomib (Velcade), a proteasome inhibitor already used in the clinic, induces time- (60nM for 0, 1, 3, 6 12 and 24hr) and dose- (0, 1, 6, 20, 60 and 100nM for 24hr) dependent down-regulation (>90%) of total KIT expression. Noticeably, dephosphorylation of KIT occurred 6hr before reduction of the total KIT level after bortezomib exposure. We also found >50% down-regulation of KIT expression in patients’ primary blasts treated with 60nM bortezomib for 24hr and in Kasumi-1 cells treated with the proteasome inhibitor MG132 (300nM for 24hr). Down-regulation of KIT appeared to be associated with inhibition of NF-kB and Sp1, which is necessary for regulation of the KIT promoter activity by the SCL complex. In fact, treatment with bortezomib inactivated NF-kB and decreased transcription of Sp1 in Kasumi-1 cells. Furthermore, exposure to the NF-kB or Sp1 inhibitors parthenolide (30μM for 24hr) and mithramycin (100ng/ml for 24hr), respectively, resulted in a dose-dependent decrease in KIT expression in Kasumi-1 cells. When cells were treated with bortezomib (20nM) in combination with mithramycin (30ng/ml for 24hr), we observed synergy in down-regulation of KIT RNA and KIT protein. This correlated with growth arrest and increased cell death. Interestingly, the magnitude of these effects was higher when Kasumi-1 cells were pretreated with mithramycin for 24hr before being exposed to bortezomib. Taken all together, our data suggest that bortezomib downregulates KIT expression and might also inhibit KIT phosphorylation and should be considered in future therapeutic strategies targeting AML subgroups harboring mutated KIT.
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Dissertations / Theses on the topic "KASUMI"

1

Akleylek, Sedat. "On The Avalanche Properties Of Misty1, Kasumi And Kasumi-r." Master's thesis, METU, 2008. http://etd.lib.metu.edu.tr/upload/12609407/index.pdf.

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The Global System for Mobile (GSM) Communication is the most widely used cellular technology. The privacy has been protected using some version of stream ciphers until the 3rd Generation of GSM. KASUMI, a block cipher, has been chosen as a standard algorithm in order to be used in 3rd Generation. In this thesis, s-boxes of KASUMI, MISTY1 (former version of KASUMI) and RIJNDAEL (the Advanced Encryption Standard) are evaluated according to their linear approximation tables, XOR table distributions and satisfaction of the strict avalanche criterion (SAC). Then, the nonlinear part, FI function, of KASUMI and MISTY1 are investigated for SAC. A new FI function is defined by replacing both s-boxes of KASUMI by RIJNDAEL&rsquo
s s-box. Calling this new version KASUMI-R, it is found to have an FI function significantly better than others. Finally, the randomness characteristics of the overall KASUMI-R for different rounds are compared to those of MISTY1 and KASUMI, in terms of avalanche weight distribution (AWD) and some statistical tests. The overall performance of the three ciphers is found to be same, although there is a significant difference in their FI functions.
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Jirkovská, Šárka. "Současné bezpečnostní trendy v mobilních sítích." Master's thesis, Vysoké učení technické v Brně. Fakulta elektrotechniky a komunikačních technologií, 2010. http://www.nusl.cz/ntk/nusl-218554.

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This master's thesis deals with the issue of the security of GSM and UMTS mobile communication systems. In the thesis the principles of the authentication and encryption of both the mobile systems are described. Further, the constituent algorithms used for identity verification, key generation and encryption are mentioned. The commonly used algorithms are described along with their weaknesses. In the following part of this thesis, well-known attacks on GSM system are mentioned. In the part where UMTS system is dealt with one can find the algorithms used in this system to increase the security of transmitted data and authentication in comparison with GSM system. Therefore the whole process of authentication and encryption is described separately. In the last part the creation of programming models of authentication and encryption in GSM and encryption in UMTS is described. These models are created in the environment of Matlab version 2009b.
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Wagner, Marcel Stefan. "Influência de protocolos de segurança sobre o desempenho de redes UMTS." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/3/3142/tde-27032009-085123/.

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Este trabalho analisa a influência de mecanismos de segurança, incluindo autenticação de assinante, confidencialidade e integridade de dados, sobre o desempenho do sistema celular de terceira geração Universal Mobile Communication System (UMTS). Em função do serviço e do tamanho das chaves utilizadas foram medidos, através de simulação, o nível de potência gasto pelos equipamentos de usuário (UE) e o tempo de resposta em aplicações do tipo Web. Verificou-se que os efeitos negativos produzidos pelos mecanismos de segurança são pequenos em relação à proteção maior que é obtida nos sistemas UMTS Terrestrial Radio Access Network (UTRAN) e Core Network (CN).
This work is aimed at analyzing the influence of security mechanisms, including user authentication, data confidentiality and integrity, related to system cellular performance of third generation named Universal Mobile Telecommunication System (UMTS). By the service and the size of the used keys, there has been measured through simulation, the power level spent by the user equipments and the response time at web applications usage. It is seen that negative results produced by security mechanisms are small related to the greater protection that is get on UMTS Terrestrial Radio Access Network (UTRAN) systems and Core Network (CN).
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Estling, Jan, and Rosita Ståhl. "Kasam : Graden av Kasam hos polisstudenter." Thesis, Linnéuniversitetet, Institutionen för pedagogik, psykologi och idrottsvetenskap, PPI, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-23642.

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Sammanfattning Huvudsyftet med föreliggande kvantitativa studie var att undersöka om polisstudenter på Linnéuniversitetet i Växjö har ett högre genomsnittligt värde av känsla av sammanhang ( Kasam) i jämförelse med en slumpmässig tillfrågad kontrollgrupp av studenter på Linnéuniversitetet i Växjö. Det sekundära syftet var att utröna om det föreligger några könskillnader. Studenterna fick besvara 29 frågor på en Likertskala 1-7, i livsfrågeformuläret Kasam-29. Resultatet visade på att polisstudenter har en högre genomsnittlig grad av Kasam, men att kön inte har någon betydelse. Antalet tillfrågade respondenter var sammanlagt 92, fördelat på 54 polisstudenter varav 37 var män och 17 var kvinnor. Kontrollgruppen bestod av   38 slumpmässigt tillfrågade studenter, 16 män och 22 kvinnor. Det ojämna antalet respondenter mellan grupperna och könsfördelningen hade ingen signifikant påverkan.
Abstract The main purpose of this quantitative survey was to investigate if police students at Linné University of Växjö would have a higher degree of Sense of Coherence (SOC) in comparison to a random control group of other students at Linné University. The secondary purpose was to determine if there are any gender differences. The students answered 29 questions on the Likert scale 1-7 from the Kasam-29 questionnaire. The result showed that police students had an average higher degree of Sense of Coherence, but that gender had no effect. The total of respondents was 92, distributed on 54 police students; 37 men and 17 women. The control group consisted of  38 randomly chosen students, 16 men and 22 women. The uneven quantity of respondents between the groups and genders did not have any significant influence.
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Sudhakar, Solomon. "Kashmir "let peace and justice prevail"." Online full text .pdf document, available to Fuller patrons only, 2003. http://www.tren.com.

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Czepluch, Hartmut. "Kasus im Deutschen und Englischen : ein Beitrag zur Theorie des abstrakten Kasus /." Tübingen : M. Niemeyer, 1996. http://catalogue.bnf.fr/ark:/12148/cb358530277.

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Ljungberg, Marianne. "KASAM och matematiksvårigheter. Hur lärare kan gynna KASAM hos elever i matematiksvårigheter." Thesis, Malmö högskola, Lärarutbildningen (LUT), 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-30350.

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Studiens syfte är att beskriva lärares uppfattning och erfarenheter av faktorer som påverkar möjligheten att stötta och hjälpa elever i matematiksvårigheter, samt ge dem en god KASAM.Studien ger en överblick över tidigare forskning, samt en beskrivning av den salutogena teorin och KASAM. Med hjälp av kvalitativa forskningsintervjuer har jag studerat hur matematik-lärare i skolår 6-9 kan gynna KASAM hos elever i matematiksvårigheter. Studien beskriver lärarnas åsikter i ett övergripande perspektiv, att se eleven i ett helhetsperspektiv, och också arbetssätt och strategier för att göra undervisningen meningsfull, begriplig och hanterbar för eleverna. Studien beskriver även faktorer lärarna anser vara försvårande för att gynna KASAM hos elever i svårigheter.Sammanfattningsvis visar resultaten på en samstämmighet hos lärarna om vikten av att se hela människan. En god kunskap om, och relation till eleven anses vara grundläggande för att gynna KASAM, samt att eleven ges möjlighet att uppleva trygghet och delaktighet, både bland vuxna och kamrater. I matematikundervisningen anses det viktigt att stärka självkänsla och tillit hos eleven, samt att stärka motivationen och knyta undervisningen till elevers intressen och vardag. Att knyta ny kunskap till kunskaper eleven redan har, ge tillräckligt med tid, anpassa miljö och arbetsuppgifter, samt att strukturera och tydliggöra inlärning och svårigheter är andra faktorer lärarna upplever som väsentliga.
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Sigvardsson, Dan. "Pendlingens påverkan på KASAM." Thesis, Högskolan Dalarna, Psykologi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:du-5385.

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Många hushåll och individer har i dagens samhälle tvingats börja pendla till sina arbeten bl.a. på grund av en förändrad arbetsmarknad. Syftet med föreliggande studie var att se om individer som pendlar regelbundet har ett lägre KASAM. Utgångspunkten var att individer med en mer extern kontrollokus ofta har lägre KASAM och att pendling hypotetiskt sett bör påverka individen lokus i en extern riktning. Sammanlagt 27 försökspersoner i åldrarna 22 – 50 år undersöktes med hjälp av KASAM test och I-E test. Deltagarna togs med bekvämlighetsurval från ett nordiskt flygbolag i Sverige, där 13 personer pendlande till arbetet och 14 var bosatta på arbetsorten, vilket visade att KASAM var signifikant lägre på 5 % nivå för de pendlande individerna, samt att kontrollokus var signifikant mer externt, också på 5 % nivå. Vidare forskning bör fokusera på individer från andra branscher för att se om resultaten är allmänna.
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Egorova, Olga. "Adverbiale Kasus des Deutschen." Doctoral thesis, Humboldt-Universität zu Berlin, Philosophische Fakultät II, 2006. http://dx.doi.org/10.18452/15526.

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Adverbiale Kasus sind Substantivgruppen ohne Präposition, die im Satz die Funktion einer temporalen, lokalen oder modalen Adverbialbestimmung erfüllen. Im Satz treten sie in der Regel als Adjunkte auf und sind in ihrer Form syntaktisch unabhängig. Das Ziel der vorliegenden Dissertation ist, adverbiale Kasus des Deutschen anhand zahlreicher Belege sowohl synchron als auch diachron aus verschiedenen Perspektiven zu beschreiben. In den älteren Sprachstufen sind adverbiale Kasus häufig belegt. Bis zum Gegenwartsdeutschen wurden viele davon adverbialisiert, durch Präpositionalphrasen ersetzt oder sind zu Phraseologismen erstarrt. In dieser Arbeit wird auf die semantisch-lexikalischen Restriktionen, Struktur, interne Ausfüllung bzw. Besetzung, Bezugsmöglichkeiten, Bedeutungen und die stilistischen Besonderheiten adverbialer Kasus ausführlich eingegangen.
Adverbial case is a noun phrase without preposition, functioning as the adverbial modifier of time, place or manner in a sentence. Syntactically they usually serve as adjuncts and are independent in their form. The aim of the present investigation was to describe the German adverbial case synchronically and diachronically on the basis of a large number of speech samples from different points of view. Adverbial cases have been very frequent at the earlier stages of German. Many of them have been adverbialised, replaced by prepositional phrases or have turned into phraseologisms in the course of language development up to the modern German. The present doctor thesis provides a detailed study of the lexical-semantic restrictions of the adverbial cases as well as of their structure and inner filling, their relation to other sentence constituents, aspects of their meaning and stylistic peculiarities.
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Björmram, Gabriella, and Sandra Eliasson. "KASAM : om patienten själv får skatta. Hur cancer- och hjärtpatienter skattar sin KASAM." Thesis, Högskolan i Borås, Institutionen för Vårdvetenskap, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:hb:diva-19630.

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Cancer och hjärtsjukdom är två vanliga sjukdomstillstånd som båda innebär förändring av livet. Det kan hända att patienter upplever minskad livskvalité vid dessa sjukdomar. För att mäta livskvalité finns olika skalor. En av dessa är Aaron Antonovsky’s frågeformulär, vilket är en skala som mäter känsla av sammanhang (KASAM). Då sjuksköterskan är medveten om hur patienten upplever sin livssituation samt hur denne skattar sin livskvalité, är det lättare för henne att ge patienten en god och individanpassad vård, samt att hjälpa patienten att uppnå välbefinnande trots sjukdom. Syftet är att kartlägga hur hjärtsjuka och cancerpatienter skattar sin KASAM, då ökad kunskap om detta ger möjlighet till en bättre vård. Metoden som användes var analys av kvantitativ forskning som sammanställdes i tabeller. Resultatet visar hur de olika patientgrupperna skattar sin KASAM och att det finns en skillnad mellan hur cancer- och hjärtpatienter skattar sin KASAM. Vidare har vi funnit att det finns en skillnad i KASAM-värde bland patienterna som beror på andra faktorer, som exempelvis kön och ålder. Signifikant lägre KASAM-värden angavs av kvinnor med cancer jämfört med både friska kvinnor och män med cancer. Signifikant högre KASAM-värde angavs av äldre cancerpatienter jämfört med yngre cancerpatienter.
Program: Sjuksköterskeutbildning
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Books on the topic "KASUMI"

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Hirofumi, Sugimoto, and Ueno Harumi, eds. Kasumi =: [Kasumi]. New York: Del Rey/Ballantine Books, 2008.

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Natsuki, Shizuko. Kenji Kasumi Yūko yofuke no shukuden. Tōkyō: Shinchōsha, 2000.

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Shizuko, Natsuki. Kenji Kasumi Yūko fūkyoku no misaki. Tōkyō: Shinchōsha, 2007.

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Itō, Gorō. Shashinshū Meiji Taishō Shōwa Kashima Itako Kasumi Ushibori. Tōkyō: Kokusho Kankōkai, 1986.

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Karada ga kawaru sutoretchi byūti book: Kasumi style "angel's stretch". Tōkyō: Gijutsu Hyōronsha, 2005.

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Kasuri. Tōkyō: Hōsei Daigaku Shuppankyoku, 2002.

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Horiuchi, Izuho. Kasuri: Ai to shiro no orinasu sekai = Kasuri. Nara-ken Yoshino-gun Shimoichi-chō: Horiuchi Izuho, 1988.

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Hakubutsukan, Kurayoshi. Kasuri: Honma korekushon. Tottori-ken Kurayoshi-shi: Kurayoshi Hakubutsukan, 1989.

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Mugerwa, R. Kigongo. The Kasubi Tombs. [Kampala, Uganda: R.M.K. Associates, 1991.

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Takashi, Gonoi. Petoro Kibe Kasui. [Ōita-shi]: Ōita-ken Kyōiku Iinkai, 1997.

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Book chapters on the topic "KASUMI"

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De Cannière, Christophe. "Kasumi/Misty1." In Encyclopedia of Cryptography and Security, 669–70. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-5906-5_586.

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Jia, Keting, Leibo Li, Christian Rechberger, Jiazhe Chen, and Xiaoyun Wang. "Improved Cryptanalysis of the Block Cipher KASUMI." In Selected Areas in Cryptography, 222–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-35999-6_15.

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Blunden, Mark, and Adrian Escott. "Related Key Attacks on Reduced Round KASUMI." In Fast Software Encryption, 277–85. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/3-540-45473-x_23.

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Iwata, Tetsu, Tohru Yagi, and Kaoru Kurosawa. "On the Pseudorandomness of KASUMI Type Permutations." In Information Security and Privacy, 130–41. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/3-540-45067-x_12.

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Gupta, Devansh, Somanath Tripathy, and Bodhisatwa Mazumdar. "Correlation Power Analysis on KASUMI: Attack and Countermeasure." In Security, Privacy, and Applied Cryptography Engineering, 142–56. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-030-05072-6_9.

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Biham, Eli, Orr Dunkelman, and Nathan Keller. "A Related-Key Rectangle Attack on the Full KASUMI." In Lecture Notes in Computer Science, 443–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/11593447_24.

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Kang, Ju-Sung, Sang-Uk Shin, Dowon Hong, and Okyeon Yi. "Provable Security of KASUMI and 3GPP Encryption Mode f8." In Advances in Cryptology — ASIACRYPT 2001, 255–71. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/3-540-45682-1_16.

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Sugio, Nobuyuki, Yasutaka Igarashi, Toshinobu Kaneko, and Kenichi Higuchi. "New Integral Characteristics of KASUMI Derived by Division Property." In Information Security Applications, 267–79. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56549-1_23.

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Kang, Ju-Sung, Okyeon Yi, Dowon Hong, and Hyunsook Cho. "Pseudorandomness of MISTY-Type Transformations and the Block Cipher KASUMI." In Information Security and Privacy, 60–73. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/3-540-47719-5_7.

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Yamamoto, Dai, Kouichi Itoh, and Jun Yajima. "A Very Compact Hardware Implementation of the KASUMI Block Cipher." In Information Security Theory and Practices. Security and Privacy of Pervasive Systems and Smart Devices, 293–307. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-12368-9_23.

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Conference papers on the topic "KASUMI"

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SUGIO, Nobuyuki, Yasutaka IGARASHI, and Toshinobu KANEKO. "Integral Cryptanalysis of Reduced-round KASUMI." In 2018 International Symposium on Information Theory and Its Applications (ISITA). IEEE, 2018. http://dx.doi.org/10.23919/isita.2018.8664404.

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Yasir, Ning Wu, Muhammad Rehan Yahya, and Qiangjia Bi. "Area-Efficient Architectures of KASUMI Block Cipher." In 2018 21st Saudi Computer Society National Computer Conference (NCC). IEEE, 2018. http://dx.doi.org/10.1109/ncg.2018.8593101.

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Hui Shi, Fengli Ma, Peng Jia, Yu Guan, and Yuanqing Deng. "Analysis of the avalanche property of the KASUMI algorithm." In International Conference on Automatic Control and Artificial Intelligence (ACAI 2012). Institution of Engineering and Technology, 2012. http://dx.doi.org/10.1049/cp.2012.1025.

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Fang, Ren, Yan Ying-jian, and Fu Xiao-bing. "A Small and Efficienct Hardware Implementation of the KASUMI." In 2009 WASE International Conference on Information Engineering (ICIE). IEEE, 2009. http://dx.doi.org/10.1109/icie.2009.187.

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Halagali, Basavaraj P., and Veena V. Desai. "Implementation of Chaos Based Cryptography in Kasumi Block Cipher." In 2018 International Conference on Communication and Signal Processing (ICCSP). IEEE, 2018. http://dx.doi.org/10.1109/iccsp.2018.8524452.

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Bai, Qilin, Naijie Gu, Junjie Su, and Kuai Yu. "A General Method for Accelerating the Kasumi Algorithm on Intel Processors." In 2018 IEEE 18th International Conference on Communication Technology (ICCT). IEEE, 2018. http://dx.doi.org/10.1109/icct.2018.8600093.

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Madani, Mahdi, Salim Chitroub, and Camel Tanougast. "Two KASUMI components for an optimal implementation of the A5/3 algorithm." In 2017 International Conference on Circuits, System and Simulation (ICCSS). IEEE, 2017. http://dx.doi.org/10.1109/cirsyssim.2017.8023195.

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Mahjabin, Syeda Ishrat, Tanvir Ahmed, Sakif Azline, Timittra Hridi, Zuheb Ahmed, and Md Ashraful Hoque. "An Enriched Kasumi Encryption Algorithm by Modifying F-functions for 4G LTE." In 2019 22nd International Conference on Computer and Information Technology (ICCIT). IEEE, 2019. http://dx.doi.org/10.1109/iccit48885.2019.9038258.

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Vinagre, Nathália, Aline Rangel, Raquel Maia, Fausto Ferraris, and Ana Cristina Nogueira. "Kasumi-1 cell line: an alternative to the potency assay of Filgrastim biopharmaceutical." In III Simpósio Internacional de Imunobiológicos. Instituto de Tecnologia em Imunobiológicos, 2016. http://dx.doi.org/10.35259/isi.sact.2016_28230.

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Chin, Diana, Christine Pietsch, Francis McCabe, Susan Chippari, Elizabeth Kaiser, Rebecca Hanson, and Mariusz Lubomirski. "Abstract 90: Development of a systemic Kasumi-3 acute myeloid leukemia model in NSG mice." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-90.

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Reports on the topic "KASUMI"

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Hussain, Shakeel. Jammu and Kashmir. Fort Belvoir, VA: Defense Technical Information Center, April 2002. http://dx.doi.org/10.21236/ada404200.

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Bennur, Shubhapriya, and Laxmidevi Gavai. How did the Regional Traditional Indian Embroidery “Kasuti” Reach the International Markets? Ames: Iowa State University, Digital Repository, 2013. http://dx.doi.org/10.31274/itaa_proceedings-180814-625.

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Hodermarsky, Daniel G. Lessons from India's Counterinsurgency Campaign in Jammu and Kashmir. Fort Belvoir, VA: Defense Technical Information Center, December 2013. http://dx.doi.org/10.21236/ada606326.

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T., Cronin, and Santoso L. Politik REDD+ di Media: studi Kasus dari Indonesia. Center for International Forestry Research (CIFOR), 2011. http://dx.doi.org/10.17528/cifor/003403.

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Toma, Iulia Andreea. Kasai: The forgotten province of DRC – gender assessment. Oxfam, February 2018. http://dx.doi.org/10.21201/2017.1657.

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Rahman, Muhammad. Sekolah Swasta Berbiaya Rendah (Sebuah Studi Kasus di Jakarta). Jakarta, Indonesia: Center for Indonesian Policy Studies, 2016. http://dx.doi.org/10.35497/270474.

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Schaffer, M. T. Finding a Kashmir Settlement: The Burden of Leadership. Strategic Forum. Number 199. Fort Belvoir, VA: Defense Technical Information Center, June 2003. http://dx.doi.org/10.21236/ada421847.

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timan, Su. PENGARUH BIAYA OPERASIONAL TERHADAP PROFITABILITAS (STUDI KASUS PADA PT GEMILANG ABADI). Jurnal Madani: Ilmu Pengetahuan, Teknologi, dan Humaniora, September 2018. http://dx.doi.org/10.33753/madani.v1i2.20.

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Krishnamurti, Indra, Arief Nugraha, and Mercyta Glorya. Mengoptimalkan Penggunaan Tanah Kas Desa: Studi Kasus Lima Desa di Jawa Tengah. Jakarta, Indonesia: Center for Indonesian Policy Studies, 2019. http://dx.doi.org/10.35497/284674.

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ruddin, Syam, and Fhadilah Nur Hakim. PENGARUH EKUITAS MEREK TERHADAP KEPUTUSAN PEMBELIAN HANDPHONE XIAOMI STUDI KASUS KOMUNITAS MI FANS JAKARTA DAN DEPOK. Jurnal Madani: Ilmu Pengetahuan, Teknologi, dan Humaniora, March 2019. http://dx.doi.org/10.33753/madani.v2i1.41.

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