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Journal articles on the topic 'Keshan disease'

Author: Grafiati
Published: 4 June 2021
Last updated: 22 February 2023

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1

Chen, X. "SELENIUM AND CARDIOMYOPATHY (KESHAN DISEASE)." Acta Pharmacologica et Toxicologica 59 (March 13, 2009): 325–30. http://dx.doi.org/10.1111/j.1600-0773.1986.tb02772.x.

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2

Yang, Fuyu. "Keshan disease and mitochondrial cardiomyopathy." Science in China Series C: Life Sciences 49, no. 6 (December 2006): 513–18. http://dx.doi.org/10.1007/s11427-006-2041-y.

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3

Yu, Pei-Liang. "Keshan disease: An entity or not." Human Pathology 19, no. 7 (July 1988): 874. http://dx.doi.org/10.1016/s0046-8177(88)80275-2.

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4

Jiang, S., F. L. Li, Q. Dong, H. W. Liu, C. F. Fang, C. Shu, H. Cheng, et al. "H558R polymorphism in SCN5A is associated with Keshan disease and QRS prolongation in Keshan disease patients." Genetics and Molecular Research 13, no. 3 (2014): 6569–76. http://dx.doi.org/10.4238/2014.august.28.1.

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5

Loscalzo, Joseph. "Keshan Disease, Selenium Deficiency, and the Selenoproteome." New England Journal of Medicine 370, no. 18 (May 2014): 1756–60. http://dx.doi.org/10.1056/nejmcibr1402199.

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6

Barbaro, Giuseppe. "HIV-Associated Cardiomyopathy is not Keshan Disease." Journal of the Royal Society of Medicine 95, no. 6 (June 2002): 324. http://dx.doi.org/10.1177/014107680209500627.

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7

Li, Guangsheng, Fan Wang, Deren Kang, and Cai Li. "Keshan disease: An endemic cardiomyopathy in China." Human Pathology 16, no. 6 (June 1985): 602–9. http://dx.doi.org/10.1016/s0046-8177(85)80110-6.

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8

Barbaro, G. "HIV-associated cardiomyopathy is not Keshan disease." JRSM 95, no. 6 (June 1, 2002): 324. http://dx.doi.org/10.1258/jrsm.95.6.324.

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9

Li, Qi, Mingfa Liu, Jie Hou, Changxing Jiang, Shaochen Li, and Tong Wang. "The prevalence of Keshan disease in China." International Journal of Cardiology 168, no. 2 (September 2013): 1121–26. http://dx.doi.org/10.1016/j.ijcard.2012.11.046.

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10

YANG, T. "Myocardial metabolic derangement of keshan disease (KD)." Journal of Molecular and Cellular Cardiology 24 (May 1992): S42. http://dx.doi.org/10.1016/0022-2828(92)90959-4.

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11

Wei-Han, Yu, Meng Xian-Zhong, Zheng Shao-Juan, Li Zhe-Hong, and Yang Gao-Yang. "Progress in the study of Keshan disease." Heart and Vessels 1, S1 (March 1985): 286–87. http://dx.doi.org/10.1007/bf02072411.

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12

Zhu, Yanhe, Bingqi Lai, Xiaolin Niu, Jin Wei, Wuhong Tan, and Xinfeng Wang. "Long-term Prognostic Value of Major and Minor ECG Abnormalities in Latent Keshan Disease With Suspect Chronic Keshan Disease." Journal of Epidemiology 24, no. 5 (2014): 385–91. http://dx.doi.org/10.2188/jea.je20130180.

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13

Ren, Li-Qun. "Coxsackievirus B3infection and its mutation in Keshan disease." World Journal of Gastroenterology 10, no. 22 (2004): 3299. http://dx.doi.org/10.3748/wjg.v10.i22.3299.

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14

Levander, Orville A., and Melinda A. Beck. "Interacting nutritional and infectious etiologies of Keshan disease." Biological Trace Element Research 56, no. 1 (January 1997): 5–21. http://dx.doi.org/10.1007/bf02778980.

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15

Ye, Yuanming, Jingwang Qu, Yao Pu, Shen Rao, Feng Xu, and Chu Wu. "Selenium Biofortification of Crop Food by Beneficial Microorganisms." Journal of Fungi 6, no. 2 (May 3, 2020): 59. http://dx.doi.org/10.3390/jof6020059.

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Selenium (Se) is essential for human health, however, Se is deficient in soil in many places all around the world, resulting in human diseases, such as notorious Keshan disease and Keshin–Beck disease. Therefore, Se biofortification is a popular approach to improve Se uptake and maintain human health. Beneficial microorganisms, including mycorrhizal and root endophytic fungi, dark septate fungi, and plant growth-promoting rhizobacteria (PGPRs), show multiple functions, especially increased plant nutrition uptake, growth and yield, and resistance to abiotic stresses. Such functions can be used for Se biofortification and increased growth and yield under drought and salt stress. The present review summarizes the use of mycorrhizal fungi and PGPRs in Se biofortification, aiming to improving their practical use.
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16

YANG, T., P. ZHU, G. DAI, S. BEI, and Y. ZHAO. "Characteristics of metabolism in Keshan disease (an endemic cardiomyopathy)." Journal of Molecular and Cellular Cardiology 19 (1987): S36. http://dx.doi.org/10.1016/s0022-2828(87)80461-3.

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17

Lei, Cong, Xiaolin Niu, Xiangke Ma, and Jin Wei. "Is selenium deficiency really the cause of Keshan disease?" Environmental Geochemistry and Health 33, no. 2 (June 24, 2010): 183–88. http://dx.doi.org/10.1007/s10653-010-9331-9.

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18

Ouyang Yibing and Yang Tungshu. "Free radical metabolism in autopsy myocardium of Keshan disease." Journal of Molecular and Cellular Cardiology 24 (May 1992): 171. http://dx.doi.org/10.1016/0022-2828(92)90537-a.

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19

Hou Lizhong, Yang Tongshu, and Ouyang Yibing. "Cellular enzymatic spectrum in myocardium of Keshan disease patients." Journal of Molecular and Cellular Cardiology 24 (May 1992): 215. http://dx.doi.org/10.1016/0022-2828(92)90670-u.

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20

Hou Lizhong, Yang Tongshu, and Zhou Zijie. "Serum biochemical assay in population in Keshan disease area." Journal of Molecular and Cellular Cardiology 24 (May 1992): 221. http://dx.doi.org/10.1016/0022-2828(92)90687-u.

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21

Ruszel, Kinga, Piotr Pokorski, and Barbara Nieradko-Iwanicka. "Controversies about selenium supplementation." Polish Journal of Public Health 131, no. 1 (January 1, 2021): 20–26. http://dx.doi.org/10.2478/pjph-2021-0005.

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Abstract Introduction. Selenium (Se) is a trace element found mainly in meat, seafood, nuts and grains. Se is found in selenoproteins such as selenocystein or selenomethionin. A well balanced diet provides enough Se. Many regulatory and metabolic enzymes contain Se as their component, which is why Se supplementation is used in the treatment as well as prevention of multiple disorders. Se may, however, be toxic if overdosed. Aim. The aim of this review is to summarize the data about functions of Se in human body and to discuss its use in treatment and prevention of diseases. Materials and methods. The search was conducted using the PubMed and Google Scholar databases in March and April 2020. The key words used were: ‘selenium’, ‘cardiovascular disease’, ‘selenium supplementation’, ‘Keshan disease’, ‘source of selenium’. A total of 68 articles were analysed. Results. The first cases of chronic Se deficiency cases were documented 85 years ago in China. The patients with cardiomyopathy, extensive fibrosis and degenerative changes in the heart were diagnosed with Keshan disease. Human selenoproteonome consists of at least 25 selenoproteins. Se plays a role in immunity and metabolism via its role in functioning of numerous enzymes: glutathione peroxidase, thioredoxine and methionine sulfoxide reductase, methionine-sulfoxide reductase B1. Se plays a role in glucose homeostasis, Alzheimer’s disease, thyroid disorders, infectious, inflammatory diseases, vascular diseases and fertility. Conclusion. Se deficiency increases the risk of Keshan disease, but there is not enough evidence to recommend its supplementation for prevention of cardiovascular disease. However, Se status is important part of health assessment. Se supplementation should not exceed the dose of 55μg/day.
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22

Burke, Michael Philip, and Kenneth Opeskin. "Fulminant Heart Failure Due to Selenium Deficiency Cardiomyopathy (Keshan Disease)." Medicine, Science and the Law 42, no. 1 (January 2002): 10–13. http://dx.doi.org/10.1177/002580240204200103.

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23

Zhang, Xiao, Tong Wang, Shie Li, Chao Ye, Jie Hou, Qi Li, Hong Liang, et al. "A spatial ecological study of selenoprotein P and Keshan disease." Journal of Trace Elements in Medicine and Biology 51 (January 2019): 150–58. http://dx.doi.org/10.1016/j.jtemb.2018.10.011.

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24

Zhang, Xiao, Tong Wang, Shie Li, Chao Ye, Jie Hou, Qi Li, Hong Liang, et al. "A Spatial Ecology Study of Keshan Disease and Hair Selenium." Biological Trace Element Research 189, no. 2 (August 31, 2018): 370–78. http://dx.doi.org/10.1007/s12011-018-1495-7.

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25

Ouyang Yibing and Yang Tongshu. "Free radical metabolism in model animal myocardium with Keshan disease." Journal of Molecular and Cellular Cardiology 24 (May 1992): 170. http://dx.doi.org/10.1016/0022-2828(92)90536-9.

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26

LI, Fang-Sheng, Jin-Yang GUAN, Li-Ming ZOU, You-Jin DUAN, Yu-Hua ZHAO, Li LI, and Qun JIN. "Metabolic Disorder of Phospholipid and Membrane Damage in Keshan Disease Model." Journal of Clinical Biochemistry and Nutrition 1, no. 3 (1986): 209–17. http://dx.doi.org/10.3164/jcbn.1.209.

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27

LIU, Yunbao, Momoko CHIBA, Yutaka INABA, and Masao KONDO. "Keshan Disease-a Review from the Aspect of History and Etiology." Nippon Eiseigaku Zasshi (Japanese Journal of Hygiene) 56, no. 4 (2002): 641–48. http://dx.doi.org/10.1265/jjh.56.641.

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28

Schrauzer, G. N. "Chinese Keshan Disease Research Groups recipients ofKlaus Schwarz medals for 1984." Biological Trace Element Research 7, no. 1 (February 1985): 63–66. http://dx.doi.org/10.1007/bf02916548.

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29

LIU, YUNBAO, WEIXING ZHANG, and LINGWANG ZHOU. "Studies on viral infection in Keshan disease patients in Heilongjiang Province." Juntendo Medical Journal 47, no. 2 (2001): 221–28. http://dx.doi.org/10.14789/pjmj.47.221.

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30

Yang Jie, Mai Yinqiao, and Yang Tongshu. "Functions of myocardial plasma membrane of model animal with Keshan disease." Journal of Molecular and Cellular Cardiology 24 (May 1992): 125. http://dx.doi.org/10.1016/0022-2828(92)90400-t.

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31

Yang Jie and Yang Tongshu. "Correlation of myocardium and serum enzymes in Keshan disease model animals." Journal of Molecular and Cellular Cardiology 24 (May 1992): 221. http://dx.doi.org/10.1016/0022-2828(92)90688-v.

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32

Zhao Jiangang and Yang Tongshu. "Superoxide dismutase activities in erythrocytes from population in Keshan disease area." Journal of Molecular and Cellular Cardiology 24 (May 1992): 221. http://dx.doi.org/10.1016/0022-2828(92)90689-w.

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33

Li Shaowei and Yang Tongshu. "Analysis of myosin light chains in myocardium from Keshan disease patients." Journal of Molecular and Cellular Cardiology 24 (May 1992): 222. http://dx.doi.org/10.1016/0022-2828(92)90690-2.

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34

Li, Shie, Tong Wang, Chao Ye, Qi Li, Zidan Guo, Huan Wu, Hong Liu, et al. "An approach to assessment of Keshan disease elimination at the township level." International Health 8, no. 6 (November 2016): 398–404. http://dx.doi.org/10.1093/inthealth/ihw045.

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35

Zhao Jiangang and Yang Tongshu. "The study of cytochrome oxidase (CCO) and its role in Keshan disease." Journal of Molecular and Cellular Cardiology 24 (May 1992): 222. http://dx.doi.org/10.1016/0022-2828(92)90692-s.

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36

LI, Fang-Sheng, Shiu-Fang ZHANG, Jin-Yang GUAN, Li-Ming ZOU, You-Jin DUAN, Xu-Dong QUAN, Ping MA, et al. "Oxidative Damage in Myocardial Mitochondria of Keshan Disease. An Endemic Cardiomyopathy in China." Journal of Clinical Biochemistry and Nutrition 11, no. 2 (1991): 139–51. http://dx.doi.org/10.3164/jcbn.11.139.

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37

Hou, Jie, Tong Wang, Mingfa Liu, Shaochen Li, Jie Chen, Chunxu Liu, Hui Zhang, et al. "Suboptimal Selenium Supply—A Continuing Problem in Keshan Disease Areas in Heilongjiang Province." Biological Trace Element Research 143, no. 3 (January 21, 2011): 1255–63. http://dx.doi.org/10.1007/s12011-011-8961-9.

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38

Zhou, Huihui, Tong Wang, Qi Li, and Dandan Li. "Prevention of Keshan Disease by Selenium Supplementation: a Systematic Review and Meta-analysis." Biological Trace Element Research 186, no. 1 (April 7, 2018): 98–105. http://dx.doi.org/10.1007/s12011-018-1302-5.

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39

YANG, J. "Cardiac enzymatic alterations in keshan disease model and cardiomyopathic hamster J-2-N." Journal of Molecular and Cellular Cardiology 24 (May 1992): S57. http://dx.doi.org/10.1016/0022-2828(92)91003-n.

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40

Wang, Yanan, Yuanjie Zou, Tong Wang, Shan Han, Xu Liu, Yiyi Zhang, Shengqi Su, Huihui Zhou, Xiao Zhang, and Hong Liang. "A spatial study on serum selenoprotein P and Keshan disease in Heilongjiang Province, China." Journal of Trace Elements in Medicine and Biology 65 (May 2021): 126728. http://dx.doi.org/10.1016/j.jtemb.2021.126728.

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41

Wei, H. L., J. R. Pei, C. X. Jiang, L. W. Zhou, T. Lan, M. Liu, and T. Wang. "Analysis of glutathione peroxidase 1 gene polymorphism and Keshan disease in Heilongjiang Province, China." Genetics and Molecular Research 10, no. 4 (2011): 2996–3001. http://dx.doi.org/10.4238/2011.december.2.1.

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42

Sun, Yuxiao, Chuanyu Gao, Xianqing Wang, and Yuhao Liu. "Preliminary quantitative proteomics analysis in chronic and latent Keshan disease by iTRAQ labeling approach." Oncotarget 8, no. 62 (November 11, 2017): 105761–74. http://dx.doi.org/10.18632/oncotarget.22397.

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43

LIU, NIAN-QING, LING-NA YAN, SHUN-YING ZHAO, MIN-QIN REN, SONG-LIN FENG, QIN-FANG QIAN, LIAN-ZHEN ZHU, JIAN-HUA PIAO, and YUN-YU CHEN. "STUDY ON RUBIDIUM CONCENTRATION OF DIFFERENT SAMPLES FROM KESHAN DISEASE AREA OF SICHUAN PROVINCE." International Journal of PIXE 02, no. 04 (January 1992): 603–7. http://dx.doi.org/10.1142/s0129083592000646.

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Rubidium (Rb) concentration in different samples collected from Keshan disease (KD) area of Sichuan Province has been determined with PIXE and NAA (Neutron Activity Analysis), and compared with that in three non-KD areas. About 200 erythrocyte samples of healthy children aged from 6 to 11 were collected from four different areas with various selenium status. 20 rice samples and 6 soil samples were taken from two of above four areas. The results showed that Rb content of erythrocyte of children in KD area (Dechang county) was three times that in the other three non-KD areas. The Rb contents of rice and soil in KD area were higher than that in non-KD areas, too. The abnormal high Rb concentration in erythrocyte of children depended on the Rb intake and the Rb level of the living environment.
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44

ZHAO, Y., and T. YANG. "Effect of the grain from Keshan disease areas on myocardial ATP contents inexperimental rats." Journal of Molecular and Cellular Cardiology 19 (1987): S29. http://dx.doi.org/10.1016/s0022-2828(87)80439-x.

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45

DAI, G., and T. YANG. "The changes of myocardial respiratory function in rats kept on Keshan disease area grain." Journal of Molecular and Cellular Cardiology 19 (1987): S29. http://dx.doi.org/10.1016/s0022-2828(87)80440-6.

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46

Cermelli, Claudio, Marco Vinceti, Elisa Scaltriti, Erika Bazzani, Francesca Beretti, Gianfranco Vivoli, and Marinella Portolani. "Selenite inhibition of Coxsackie virus B5 replication: implications on the etiology of Keshan disease." Journal of Trace Elements in Medicine and Biology 16, no. 1 (January 2002): 41–46. http://dx.doi.org/10.1016/s0946-672x(02)80007-4.

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47

Wang, Yanan, Xiao Zhang, Tong Wang, Jie Hou, Zhongying Guo, Xiaomin Han, Huihui Zhou, Hong Liang, and Zhifeng Xing. "A spatial study on Keshan disease prevalence and selenoprotein P in the Heilongjiang Province, China." International Journal of Occupational Medicine and Environmental Health 34, no. 5 (September 3, 2021): 659–66. http://dx.doi.org/10.13075/ijomeh.1896.01749.

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48

Wang, Sen, Zheng Fan, Bing Zhou, Yingting Wang, Peiru Du, Wuhong Tan, Mikko Lammi, and Xiong Guo. "Roles of Glycoproteins in the Diagnosis and Differential Diagnosis of Chronic and Latent Keshan Disease." Molecules 22, no. 5 (May 8, 2017): 746. http://dx.doi.org/10.3390/molecules22050746.

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49

SUN, Yuxiao, Xin LIU, Dengfeng GAO, Xin DONG, Canzhan ZHU, Zhenhua HAN, Yiqiang YUAN, Yanhe ZHU, and Xiaolin NIU. "Serum level of galectin-3 and its clinical significance in patients with chronic Keshan disease." Journal of Medical Colleges of PLA 28, no. 3 (June 2013): 152–59. http://dx.doi.org/10.1016/s1000-1948(13)60029-5.

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50

Xiang, YouZhang, Qun Xu, WuHong Tan, ShuLan He, XiaoWei Shi, WenMing Zhang, Jing Wang, XiuHong Wang, and WeiJuan Ma. "Serum Biomarkers of Keshan Disease Assessed Using a Protein Profiling Approach Based on ClinProt Technique." Protein Journal 33, no. 4 (May 20, 2014): 344–53. http://dx.doi.org/10.1007/s10930-014-9567-9.

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