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Dissertations / Theses on the topic 'Kidney disease'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Herrera, Añazco Percy, Holguín Edward Mezones, and Adrian V. Hernández. "Global kidney disease." Elsevier B.V, 2014. http://hdl.handle.net/10757/322401.

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We read with interest the Lancet Series on Global Kidney Disease. Valerie Luyckx and colleagues describe the economics and medical management of chronic kidney disease in sub-Saharan Africa.1 We note clear similarities with patients in Peru. Indeed, in Peru, the Ministry of Health (MINSA)—which covers 70% of the population—does not have a comprehensive programme for the management of patients with chronic kidney disease, including renal replacement therapies. However, the Social Security System (Essalud)—which covers 20% of the population—has a chronic kidney disease programme.<br>Revisión por

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2

Wei, Jin. "Acute Kidney Injury and Chronic Kidney Disease." Scholar Commons, 2017. http://scholarcommons.usf.edu/etd/6780.

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Ischemia and reperfusion are natural steps during kidney transplantation, and IRI is considered one of the most important nonspecific factors affecting allograft dysfunction. Transplanted organs experience several episodes of ischemia, in which cold ischemia occurs during allograft storage in preservation solutions. Even though cold ischemia has been studied extensively, all of the studies have been carried out in vitro and ex vivo models. There is no in vivo model available to examine renal IRI induced solely by cold ischemia. In the present study, we developed an in vivo mouse model to study

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3

Philips, L. G. "Disease management in chronic kidney disease /." abstract and full text PDF (free order & download UNR users only), 2005. http://0-wwwlib.umi.com.innopac.library.unr.edu/dissertations/fullcit/1430446.

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Thesis (M.B.A.)--University of Nevada, Reno, 2005.<br>"May, 2005." Includes bibliographical references (leaves 92-97). Online version available on the World Wide Web. Library also has microfilm. Ann Arbor, Mich. : ProQuest Information and Learning Company, [2005]. 1 microfilm reel ; 35 mm.

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4

Papadopoulos, Theofilos. "MiRNAs in kidney disease." Thesis, Toulouse 3, 2016. http://www.theses.fr/2016TOU30194/document.

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Les microARNs sont reconnus comme des régulateurs essentiels de l'expression des protéines. Des anomalies dans leur fonction sont associées au développement de nombreuses pathologies.tiel des microARNs en tant que biomarqueurs ou cibles thérapeutiques dans une grande variété de pathologies. Dans le cadre de cette thèse, nous avons étudié :1) L'association des microARNs urinaires avec l'évolution de la maladie rénale chronique (MRC) chez l'adulte. La prévalence de la MRC est actuellement estimée à 5-10% de la population et est en constante augmentation. La détection précoce et l'identification

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5

Ramzan, Naveen, Shimin Zheng, Hemang Panchal, Edward Leinaar, Christian Nwabueze, and Timir K. Paul. "Investigating The Association Between Chronic Kidney Disease and Clinical Outcomes." Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/asrf/2019/schedule/21.

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Background Chronic Kidney Disease (CKD) can be described as the loss of the kidney function over time. Symptoms usually develop slowly, and it may not appear in early stages. Lab tests can confirm a CKD diagnosis. The approximate number of incidents per year is more than 200,000 cases, and approximately 30 million people are living with CKD today in the United States. This long-standing disease ultimately leads to renal failure at the end. At this present time, there are no known cures for CKD, and the only treatment available is dialysis. Objectives The purpose of this study is to determine

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6

Gale, D. "Genetic investigation of kidney disease." Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/763753/.

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Kidney disease is an important contributor to the burden of ill-health worldwide. Genetic factors have an important role in determining which people are affected by kidney disease, and this study aimed to identify the genes responsible for disease in families in which unusual kidney diseases were transmitted in a pattern suggesting autosomal dominant inheritance. I performed genome-wide single nucleotide polymorphism-based linkage studies and identified two new human disease genes. Hypoxia Inducible Factor-2α (HIF2α) is a widely expressed transcription factor which is rapidly broken down in th

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7

Wong, Germaine. "Cancer and chronic kidney disease." Thesis, The University of Sydney, 2008. https://hdl.handle.net/2123/28229.

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Introduction Chronic kidney disease (CKD) is a common and important public health problem, with significant impact on the person’s quality of life and chances of survival. Cardiovascular disease is major cause of morbidity and mortality among people with CKD. Cancer is also a well-recognised complication in people on dialysis and with kidney transplants, but has not been adequately assessed in people with mild to moderately reduced kidney function. It is uncertain whether the basis of such increased risk in the end-stage kidney and transplant populations is solely related to their immunocompr

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8

Brunmark, Charlott. "Type IV collagen and renal disease." Lund : Dept. of Nephrology, University of Lund, 1994. http://books.google.com/books?id=owdrAAAAMAAJ.

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9

Antoniv, A. A. "Kidneys functional status in patients with chronic kidney disease and nonalcoholic steatohepatitis." Thesis, БДМУ, 2020. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18082.

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10

Patenaude, Anne-Marie. "Wnt signaling in kidney development and implication in polycystic kidney disease." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=84066.

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Autosomal dominant polycystic kidney disease (PKD) is caused by mutations of the PKD1 or PKD2 genes. Cyst cells exhibit sustained expression of fetal genes such as PAX2. Normally, PAX2 is involved in kidney development and is rapidly downregulated after birth. Overactivity of the canonical beta-catenin signaling pathway has also been linked to the formation of renal cysts. To determine whether beta-catenin activity is linked to the level of PAX2 expression in vivo, we created a transgenic mouse overexpressing PAX2 in mature proximal tubules of the kidney. Here we report that the canonic

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11

Miller, Michelle. "WNT signalling in kidney development and autosomal dominant polycystic kidney disease." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=103736.

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During kidney development, there is a switch from predominantly canonical to non-canonical WNT signalling. This switch transitions the developing kidney from a state of active proliferation to that of terminal differentiation. The current belief is that a defect in this switch is an underlying mechanism in the pathogenesis of autosomal dominant polycystic kidney disease. We first hypothesized that a failure to suppress canonical WNT signalling would lead to cyst formation and following this line of reasoning, crossed a β-catenin transcriptional activity reporter mouse to mice with mutations

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12

Yengkopiong, Jada. "Characterization of polycystic kidney disease in the Lewis Polycystic Kidney rat." Thesis, Yengkopiong, Jada (2010) Characterization of polycystic kidney disease in the Lewis Polycystic Kidney rat. PhD thesis, Murdoch University, 2010. https://researchrepository.murdoch.edu.au/id/eprint/4067/.

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13

Tomlinson, Laurie. "Arterial Stiffness and Chronic Kidney Disease." Thesis, University of Brighton, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.518323.

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Introduction: Chronic kidney disease (CKD) common, particularly in the elderly, and is linked to an increased risk of cardiovascular disease (CVD). This is partly explained by joint risk factors such as hypertension and diabetes but novel risk factors such as arterial stiffness, arterial calcification and endothelial dysfunction may play a role. Our aims were 1) to prospectively investigate whether aortic stiffness was linked with rate of decline of renal dysfunction, 2) to investigate the associations of arterial stiffness in patients with moderate renal dysfunction, and 3) to investigate whe

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14

Li, Zhaoli Amy, and 李昭立. "Aquaporins in kidney development and disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B29505987.

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15

Connor, T. M. F. "A study of inherited kidney disease." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1398923/.

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Kidney disease is a common, expensive, and growing worldwide health problem. Genetic factors play an important role in the aetiology of kidney disease. Current research suggests that these genetic factors are predominantly rare variants with large phenotypic effects. In this thesis I have used a range of genetic techniques to identify rare variants in different families with kidney disease, and to study how they might cause disease. The Turkish-Cypriot population of Northern Cyprus has a high incidence of renal disease, much of which is undiagnosed and may be inherited. I collected DNA from th

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16

Navaneethan, Sankar. "METABOLIC SYNDROME AND CHRONIC KIDNEY DISEASE." Case Western Reserve University School of Graduate Studies / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=case1401967446.

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17

Xie, Jeffrey Xinshuo. "Molecular Insights into Chronic Kidney Disease." University of Toledo Health Science Campus / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=mco1500201100900825.

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18

White, Joanna D. "Investigations into feline chronic kidney disease." Thesis, The University of Sydney, 2010. https://hdl.handle.net/2123/28931.

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Chronic kidney disease (CKD) is arguably the most common disease of older cats. A disproportionate number of younger, male cats with CKD was identified among a cohort of cats with CKD and was hypothesised to be due to membranous glomerulopathy or an FIV associated nephropathy. Examination of epidemiologic, histologic, immunohistologic and survival data revealed an association between the presence of CKD and FIV infection among young cats and an adverse effect of FIV infection on survival among cats with CKD, but no specific histological changes were seen among FIV positive cats. Glomer

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19

So, Beng Hock. "Chronic kidney disease : determining chronicity, prevalence, variation and survival in a community chronic kidney disease (CKD) cohort." Thesis, University of Glasgow, 2018. http://theses.gla.ac.uk/30671/.

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Chronic kidney disease (CKD) is insidious and most cases are diagnosed through opportunistic serum creatinine (SCr) testing before symptoms develop. However, efforts to accurately assess prevalence have been hampered by the lack of a universally agreed definition of SCr thresholds for the diagnosis of CKD. At the turn of the millennium, two crucial developments occurred. The first was the description of the Modification of Diet in Renal Disease estimated Glomerular Filtration Rate (eGFR) which closely correlated to cumbersome measured GFR and could be used instead in daily clinical practice. T

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20

Clark, Laura Elizabeth. "The epidemiology of chronic kidney disease in Grampian." Thesis, Available from the University of Aberdeen Library and Historic Collections Digital Resources, 2009. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=33407.

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21

O’Lone, Emma. "Cardiovascular disease: priorities and outcomes in end stage kidney disease." Thesis, The University of Sydney, 2020. https://hdl.handle.net/2123/22326.

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Introduction End stage kidney disease (ESKD) accounts for 5-10 million deaths annually worldwide. The current treatment modalities for ESKD include dialysis, transplant and supportive care. The leading cause of death for people with ESKD is cardiovascular disease (CVD). CVD is a collective term for disease affecting the heart and blood vessels including coronary, cerebral and peripheral blood vessels. CVD causes significant morbidity and has a substantial impact on quality of life for people with ESKD. Improving cardiovascular outcomes for people living with ESKD is a priority. The esca

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22

Warner, Joshua Dale. "Kidney segmentat ion and image analysis in autosomal dominant polycystic kidney disease." Thesis, College of Medicine - Mayo Clinic, 2016. http://pqdtopen.proquest.com/#viewpdf?dispub=10111486.

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<p> Autosomal Dominant Polycystic Kidney Disease (ADPKD) is among the most prevalent life-threatening genetic conditions. Despite this, no approved medical therapies exist to treat the disease. Until the recent past, no methods could reliably measure the course of the disease far in advance of end stage renal disease (ESRD). As normal tissue is progressively destroyed or blocked by enlarging cysts, remaining nephrons compensate in a process called hyperfiltration. This beneficial physiological response confounds tests of renal function. Thus, potential interventions could not be tested against

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23

Müller, Tilman [Verfasser]. "Apelinergic system in the kidney: implications for diabetic kidney disease / Tilman Müller." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2020. http://d-nb.info/1218075961/34.

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24

Antoniv, A. A. "The kidneys functional state in chronic kidney disease in patients with nonalcoholic steatohepatitis." Thesis, БДМУ, 2021. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18584.

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25

Hermans, Marcus Matheus Hendrik. "Arterial wall abnormalities in chronic kidney disease." Maastricht : Maastricht : Universitaire Pers Maastricht ; University Library, Universiteit Maastricht [host], 2007. http://arno.unimaas.nl/show.cgi?fid=9384.

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26

Parham, Rhian. "Caregiver burden in paediatric chronic kidney disease." Thesis, Canterbury Christ Church University, 2011. http://create.canterbury.ac.uk/10347/.

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Section A provides an overview of the role of family caregivers of individuals with chronic illness, and describes key conceptualisations and theories posited in the caregiver literature. This is followed by an overview of research conducted with caregivers of children with chronic kidney disease (CKD), a summary of the limitations of this research, and suggestions for future research. Section B Despite a recognised need to monitor caregiver burden in caregivers of children with CKD, there is no measurement tool currently available to meet this aim. The present research documents the developme

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27

Huang, J. L. "Polycystic kidney disease and the renal circulation." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1416827/.

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Polycystic kidney disease (PKD) is the most common inherited cause of kidney failure affecting 1 in 1000 adults and children worldwide. Many studies focus on the disrupted cellular functions within the cyst epithelia but promising therapeutic strategies have not yet translated to clinically approved treatments for human PKD. This thesis takes a different approach and hypothesised that cyst growth requires the support of the underlying renal microvasculature and that targeting vessels could be an alternative therapeutic strategy. The renal vasculature in cystic and wild-type kidneys was first c

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28

Zhou, Yu Simona. "Podocyte repair and recovery in kidney disease." Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/5959.

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Introduction Podocytes are terminally differentiated, highly specialized glomerular cells that form the final barrier to protein loss. Podocyte injury is characterised by proteinuria. Proteinuria is an important prognostic marker in kidney diseases, and lowering proteinuria has become a principal clinical goal. Compelling evidence supports the notion that continuing loss of podocytes plays a major role in the initiation and progression of glomerular diseases. It is my hypothesis that interventions that reduce the disruption by rescuing susceptible podocytes next to injured ones are potential t

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29

Ferreira, Luciana Carolina Lopes. "Autosomal dominant polycystic kidney disease - genetic diagnosis." Master's thesis, Universidade de Aveiro, 2012. http://hdl.handle.net/10773/10731.

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Mestrado em Biotecnologia<br>A doença renal poliquística autossómica dominante (ADPKD) é uma doença hereditária e monogénica comum que resulta no desenvolvimento de quistos renais que aumentam em tamanho e em número com o avanço da idade, muitas vezes conduzindo ao aparecimento da doença renal terminal. Cerca de 85% dos casos identificados são causados por mutações no gene PKD1, um gene complexo de elevadas dimensões (~ 47kb). Atualmente, a sequenciação completa do gene PKD1 permite, com elevada sensibilidade, detetar variantes alélicas e pode ser utilizada em determinadas situações clínicas

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30

Verdeguer, Francisco. "Role of HNF1β in Polycystic Kidney Disease". Paris 6, 2010. http://www.theses.fr/2010PA066543.

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Le facteur de transcription Hepocyte Nuclear Factor 1β est nécessaire pour l’expression de plusieurs gènes kystiques et sa délétion prénatale induit une polykystose rénale. J’ai montré que l’inactivation de HNF1β chez la souris à partir de 10 jours après la naissance, après la fin de la prolifération morphogénique, ne provoque pas de dilatations tubulaires kystiques. La résistance kystogénique est intrinsèquement liée à la quiescence cellulaire. En effet, lorsque les cellules déficientes pour HNF1β sont forcées à proliférer avec une ischémie/reperfusion, elles donnent lieu à la formation de ky

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31

Manfredi, Eugene Trent. "Immunodiagnostic methods for the detection of bacterial kidney disease in salmonid fishes /." Thesis, Connect to this title online; UW restricted, 1986. http://hdl.handle.net/1773/5282.

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32

Gallagher, Sean. "Reducing acute kidney injury in patients with chronic kidney disease undergoing cardiac surgery." Thesis, Queen Mary, University of London, 2014. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8669.

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Patients with chronic kidney disease (CKD) are a group with a markedly increased risk of adverse events following cardiac surgery. A particular problem for these patients is the development of post-operative acute kidney injury (AKI), which is associated with a significant increase in morbidity and mortality. Currently, there are no effective therapies proven to modify AKI in patients undergoing cardiac surgery. This thesis has three parts. The first describes an analysis of the Barts Health NHS Trust cardiac surgical dataset. Specifically, outcomes of patients with CKD and AKI were examined.

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33

Scholes-Robertson, Nicole. "Improving access to kidney replacement therapy for rural Australians with chronic kidney disease." Thesis, The University of Sydney, 2022. https://hdl.handle.net/2123/28686.

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People with chronic kidney disease (CKD) living in rural communities have a higher risk of mortality, morbidity, hospitalisation, and decreased quality of life. They are less likely to access nephrology services and receive the recommended testing and education about CKD. Many barriers to accessing healthcare exist for rural patients with CKD, including geography and travel, particularly in large countries like Australia. This thesis aims to summarise existing evidence, generate new and comprehensive knowledge about the experiences, perspectives and needs of rural patients in accessing kidney

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34

Brizi, Valerio. "Engineering complex kidney structures for disease modelling, drug testing, and studying kidney development." Thesis, Open University, 2018. http://oro.open.ac.uk/53445/.

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Although existing kidney tissue engineering systems and cell-based strategies favoured significant advances in the field, they cannot reproduce the organ’s complex architecture. This prevented the use of these tissues in studying kidney development realistically, modelling diseases, and establishing therapeutic approaches. To fill these gaps, we devised a 3D engineering system for rapid generation of custom-made geometrically predefined kidney units that more faithfully resemble their counterparts <i>in vivo</i>. Combining 3D printing and PDMS prototyping, we fabricated differently sized and s

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35

Yau, Chung-fai Forrest. "DNA microsatellites co-segregation of polycystic kidney disease genes (PKD1 & PKD2) in autosomal dominant polycystic kidney disease (ADPKD) families & cell culture models for ADPKD /." Hong Kong : University of Hong Kong, 1999. http://sunzi.lib.hku.hk/hkuto/record.jsp?B21904030.

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36

Uniacke, Mark. "The natural history of acute kidney injury and its relationship to chronic kidney disease." Thesis, University of Southampton, 2012. https://eprints.soton.ac.uk/361330/.

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37

Chitalia, Nihil A. "Vitamin D, inflammation and cardiovascular disease in patients with chronic kidney disease." Thesis, St George's, University of London, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.616978.

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Cardiovascular (CV) disease is the commonest cause of mortality in CKD. CV mortality is not entirely explained by traditional CV risk factors and therefore systemic inflammation and vitamin D deficiency are thought to play a major role in CKD. Vitamin D [25 (hydroxy vitamin D; 25(OH)D] modulates adaptive immune responses and 25(OH)D deficiency is associated with CV mortality. Vascular endothelial dysfunction is a surrogate marker of atherosclerotic CV disease and related to systemic inflammation in CKD. However, the role of vitamin D on inflammation and endothelial function in CKD is largely u

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38

Kim, Siah. "Chronic kidney disease and cardiovascular disease in Aboriginal children and young adults." Thesis, The University of Sydney, 2016. http://hdl.handle.net/2123/15945.

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Introduction Addressing the gap in health between Aboriginal and Torres Strait Islander and non-Indigenous Australians is a national health priority. The life expectancy of Aboriginal and Torres Strait Islander Australians is currently 70 years for males and 74 years for females, which is 10 years lower than that of non-Aboriginal Australians. Around 80% of the mortality gap is attributable to chronic disease across all ages, with cardiovascular diseases specifically accounting for around 24% of the mortality gap. Aboriginal and Torres Strait Islander Australians have a higher prevalence of ca

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39

Palanca, Ana. "Subclinical atherosclerosis in chronic kidney disease and diabetes." Doctoral thesis, Universitat Autònoma de Barcelona, 2020. http://hdl.handle.net/10803/670707.

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La malaltia cardiovascular és la primera causa de morbiditat i mortalitat mundial. Els individus amb diabetis i malaltia renal crònica (MRC) presenten un major risc d’esdeveniments cardiovasculars (ECV) respecte a la població general. En la diabetis, l’increment de el risc cardiovascular és heterogeni i s’ha relacionat amb el grau d’afectació renal. D’altra banda, els algoritmes tradicionals per calcular el risc cardiovascular no traslladen amb suficient precisió el risc futur de ECV. L’avaluació de l’aterosclerosi subclínica (AS) mitjançant ecografia multiterritorial representa una eina vàlid

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40

Webster, Angela C. "Immunosuppression and malignancy in end stage kidney disease." Connect to full text, 2006. http://hdl.handle.net/2123/1186.

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Thesis (Ph. D.)--University of Sydney, 2006.<br>Title from title screen (viewed 21 May 2007). Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the School of Public Health, Faculty of Medicine. Includes bibliographical references. Also available in print form.

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Owen, Paul. "Body composition and function in chronic kidney disease." Thesis, University of Nottingham, 2013. http://eprints.nottingham.ac.uk/14576/.

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Chronic kidney disease (CKD) is a significant public health issue. The uraemic milieu is associated with profound alterations in body composition and function. Therapeutic interventions to preserve renal function and to provide adequate homeostasis to improve outcomes in all stages of chronic kidney disease may promote other unwanted functional adversities which with careful attention to individualised treatment may be modifiable. The aim of this thesis is to clearly document these disorders of body composition and function and investigate whether commonly practiced interventions can indeed ha

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42

Chakrabarti, Shubro. "Mechanisms of fibrosis in feline chronic kidney disease." Thesis, Royal Veterinary College (University of London), 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572451.

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Darisipudi, Venkata Surya Narayana Murty. "Chemokines and cysteine proteases in diabetic kidney disease." Diss., Ludwig-Maximilians-Universität München, 2013. http://nbn-resolving.de/urn:nbn:de:bvb:19-173379.

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44

Montoya, Vicki. "Improving Chronic Kidney Disease Care with Group Visits." Doctoral diss., University of Central Florida, 2013. http://digital.library.ucf.edu/cdm/ref/collection/ETD/id/5676.

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First year death rates remain unacceptable high for the end-stage renal disease (ESRD) population. New effective methods are vital to improve first year morbidity and mortality outcomes for the population transitioning from Stage 4 chronic kidney disease (CKD) to ESRD)/Stage 5 CKD. Based on current methods, evidence-based recommendations made by nephrology providers are frequently not heeded by patients in Stage 4 CKD. Low levels of patient knowledge, self-efficacy, and a poor ability to self-manage CKD negatively influence a patient's ability to follow provider recommendations. The group vis

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45

Afzal, Ali Reza. "Molecular studies of autosomal dominant polycystic kidney disease." Thesis, St George's, University of London, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392478.

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46

Long, David Andrew. "Angiopoietin growth factors in models of kidney disease." Thesis, University College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.401031.

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47

Brzoska, H. L. "Planar cell polarity in kidney development and disease." Thesis, University College London (University of London), 2017. http://discovery.ucl.ac.uk/1558749/.

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Planar cell polarity (PCP) is a signalling pathway regulating epithelial cell alignment and coordinated organisation, processes that are essential for the development and maintenance of healthy organs and tissues. In this thesis, I hypothesised that the core PCP protein, Celsr1 is essential for normal kidney development. Furthermore, I predicted that PCP proteins would be disrupted in a model of renal disease induced by folic acid. Kidneys of mice homozygous for a mutation in Celsr1 (Celsr1Crsh/Crsh) were significantly smaller compared with wild-type littermates and contained a reduced number

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48

Simms, Roslyn Jane. "Zebrafish models of cystic kidney disease related ciliopathies." Thesis, University of Newcastle upon Tyne, 2013. http://hdl.handle.net/10443/2473.

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Cystic kidney diseases are a fascinating cluster of discrete conditions and an important, common cause of established renal failure. Both isolated and syndromic inherited cystic kidney diseases are known to be linked by their pathogenesis involving ciliary dysfunction. Interestingly to date, all mutated genes which have been related to cystic kidney disease, encode proteins which are located on cilia, the basal body or centrosomes and are required for ciliary function. To date, over 50 causal genes have been identified and are capable of causing additional disease phenotypes, such as neurologi

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49

Khalaf, Fatimah. "Regulation of Renal Inflammation in Chronic Kidney Disease." University of Toledo Health Science Campus / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1588943852414778.

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50

Berezova, M. S., and S. A. Akentiev. "The effects of obesity on chronic kidney disease." Thesis, БДМУ, 2017. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/17110.

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