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Dissertations / Theses on the topic 'Kidney epithelial cells'

Author: Grafiati

Published: 22 June 2024

Last updated: 31 July 2025

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1

Tang, Chi-wai Sydney. "The many facets of the renal proximal tubular epithelial cell in human." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31992468.

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2

Tang, Chi-wai Sydney, and 鄧智偉. "The many facets of the renal proximal tubular epithelial cell inhuman." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B31992468.

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3

Measures, H. R. "A study of desmosome formation in kidney epithelial cells." Thesis, University of Southampton, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.234435.

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4

Xie, Jianxun. "Involvement of transcription factors in cadmium-induced apoptosis and cell cycle arrest in rat kidney cells /." View online ; access limited to URI, 2005. http://0-wwwlib.umi.com.helin.uri.edu/dissertations/dlnow/3206258.

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5

Lim, Ai Ing, and 林艾盈. "Shedding of kidney injury molecule-1 by kidney proximal tubular epithelial cells: the role of matrixmetalloproteinase-3." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B49799745.

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Regardless of the original cause and etiology, the progression of kidney disease follows a final common pathway associated with tubulointerstitial injury, in which proximal tubular epithelial cells (PTEC) are instrumental. Kidney injury molecule-1 (KIM-1) is an emerging biomarker of kidney tubular damage. It is markedly expressed and released into urine in various animal models and human kidney diseases. This study aimed to explore the underlying mechanism regulating the release of KIM-1 by PTEC. First, expression and release of KIM-1 by primary cultured human PTEC were examined. In quiesc

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6

Zhou, Li. "The molecular mechanisms of aristolochic acid nephropathy." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B43224349.

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7

Laestadius, Åsa. "Cellular mechanisms of interaction between uropathogenic Escherichia coli and renal epithelial cells /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-187-X.

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8

Sampangi, Sandeep. "Autologous human kidney proximal tubule epithelial cells (PTEC) modulate dendritic cell (DC), T cell and B cell responses." Thesis, Queensland University of Technology, 2015. https://eprints.qut.edu.au/82033/1/Sandeep_Sampangi_Thesis.pdf.

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This is a comprehensive study of human kidney proximal tubular epithelial cells (PTEC) which are known to respond to and mediate the pathological process of a range of kidney diseases. It identifies various molecules expressed by PTEC and how these molecules participate in down-regulating the inflammatory process, thereby highlighting the clinical potential of these molecules to treat various kidney diseases. In the disease state, PTEC gain the ability to regulate the immune cell responses present within the interstitium. This down-regulation is a complex interaction of contact dependent/indep

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9

Broadbelt, Nalini V. "Regulation of iNOS expression : in response to pressure in proximal tubule epithelial cells /." Access full-text from WCMC, 2008. http://proquest.umi.com/pqdweb?did=1619205731&sid=2&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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10

Miskovic, Dragana. "A characterization of BiP gene expression in Xenopus laevis embryos and A6 kidney epithelial cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0021/NQ38257.pdf.

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11

Söderblom, Tomas. "Effects of bacterial toxins on calcium homeostasis in renal inflammation /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-904-8/.

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12

Olteanu, Dragos S. "Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers a model of polycystic kidney disease /." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2007. https://www.mhsl.uab.edu/dt/2009r/olteanu.pdf.

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13

Zhou, Li, and 周莉. "The molecular mechanisms of aristolochic acid nephropathy." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43224349.

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14

Law, Becker M. P. "The functional characterisation of human innate lymphocytes in renal fibrosis and chronic kidney disease." Thesis, Queensland University of Technology, 2019. https://eprints.qut.edu.au/132513/1/Becker%20Meng-Po_Law_Thesis.pdf.

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This thesis by publication is a step forward in understanding the function of discrete immune cell populations in kidneys with chronic inflammation and fibrosis. We have successfully identified various human immune cells of the innate immune system as critical drivers of chronic kidney disease. The findings of this thesis sheds light on novel functions of innate immune cells and opens opportunities for the development of novel kidney therapies.

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15

Olsson, Magnus. "Nuclear pore membrane glycoprotein 210 as a new marker for epithelial cells." Doctoral thesis, Uppsala University, Department of Cell and Molecular Biology, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3265.

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<p>Epithelial cell polarisation is a prerequisite for the branching morphogenesis in several organs. Differential screening techniques were used to identify genes, which are upregulated during induction of epithelium in early kidney development. This investigation revealed two separate genes, Nuclear localising protein 1 (Nulp1), a previously undescribed gene with sequence characteristics of the basic helix-loop-helix transcription factor family, and glycoprotein 210 (gp210, POM210), an integral membrane protein constituent of the nuclear pore complex (NPC). Of these, gp210 was found to be upr

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16

Asselman, Marino. "Hyaluronan biology and regulation in renal tubular epithelial cells and its role in kidney stone disease." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2008. http://hdl.handle.net/1765/13147.

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17

Hovater, Michael. "Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia : implications for polycystic kidney disease." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2006. http://www.mhsl.uab.edu/dt/2007m/hovater.pdf.

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18

Abou, Samra Elias. "Elucidation of the Role of NKR‐P1: CLR Recognition Systems in Intestinal & Renal Epithelial Cell Homeostasis and Immunity." Thesis, Université d'Ottawa / University of Ottawa, 2017. http://hdl.handle.net/10393/35747.

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Natural killer (NK) cells represent a crucial component of the innate immune system and are primarily regulated by the interactions of their activation and inhibitory receptors with ligands available on target cells. The genetically linked Ly49 and NKR-P1 family of receptors constitute two of the major regulatory receptor systems used by NK cells and have been shown to bind different ligands. Whereas the Ly49 receptors survey MHC-I ligands on target cells, the NKR-Pl receptor family members bind to various members of the C-type lectin-related (Clr) family. Interestingly, NKR-P1 and Clr haploty

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19

Daniels, Nigel Allan. "Investigation of the role of novel SGK1 isoforms in regulation of sodium transport in kidney epithelial cells." Thesis, University of Newcastle Upon Tyne, 2010. http://hdl.handle.net/10443/936.

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Serum/glucocorticoid regulated kinase 1 (SGK1) is a key component of the pathway that leads to activation of the epithelial sodium channel (ENaC) in the aldosterone-sensitive distal nephron (ASDN). Regulation of ENaC is a major determinant of renal Na+ absorption and overall body fluid homeostasis and blood pressure. Studies from our laboratory have shown that human skin cells express multiple SGK1 isoforms (A-F) that arise from alternative transcriptional start sites and RNA splicing at the SGK1 locus. The aim of this study was to investigate if SGK1 isoforms are also expressed in the ASDN an

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20

Ohan, Nicholas. "An examination of small heat shock protein gene expression in Xenopus laevis embryos and A6 kidney epithelial cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq21373.pdf.

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21

Hambitzer, Martin. "Induction of interferon beta in human kidney epithelial cells by virulent and non-virulent strains of Escherichia coli." Thesis, Malmö högskola, Fakulteten för hälsa och samhälle (HS), 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-24963.

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Urinvägsinfektioner (UVI) är ett vanligt hälsoproblem som drabbar miljontals människor. Den allvarligaste formen, akut pyelonefrit (APN) kan ge svåra komplikationer. Urinvägspatogena Escherichia coli (UPEC) som orsakar APN uttrycker P fimbrier som specifikt binder till glykosphingolipider på ytan av uroepitelceller. Det sätter igång en toll-like receptor 4 (TLR4) beroende men LPS-oberoende immunreaktion. Den roll som interferon beta (IFN-β) spelar vid bakterieinfektioner är inte helt klarlagd men studier som gjorts på IFN-β knockoutmöss visade på en ökad infektionsbenägenhet och svåra njursymp

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22

Roccio, Federica. "Autophagy in the interplay between shear stress and kidney epithelium homeostasis." Electronic Thesis or Diss., Université Paris Cité, 2021. http://www.theses.fr/2021UNIP5170.

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L'autophagie est un mécanisme de dégradation et de recyclage du matériel cytoplasmique permettant aux cellules de s'adapter à diverses stimuli notamment aux forces de cisaillement générées par les flux de liquides corporels. L'autophagie contribue ainsi au maintien de l'homéostasie cellulaire et tissulaire. L'altération de l'autophagie est observée dans de nombreuses maladies comme les maladies rénales. Au cours de cette thèse, nous avons étudié le rôle de l'autophagie induite par des forces de cisaillement dans les cellules épithéliales rénales qui sont constamment exposées au flux urinaire.

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23

Polk, William Wyatt. "Role of protein kinase C zeta in lipopolysaccharide-mediated nuclear factor kappa B aactivation [i.e. activation] and aactivity [i.e. activity] in kidney epithelial cells /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/8463.

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24

Sheremet, Andriy. "Bioinspired polyethersulfone-based hollow fiber membranes as the scaffolds in renal assist device for protein-bound toxins removal from blood." Master's thesis, Faculdade de Ciências e Tecnologia, 2014. http://hdl.handle.net/10362/13308.

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Dissertation for obtaining the Master degree in Membrane Engineering<br>Erasmus Mundus Master in Membrane Engineering<br>Using bioartificial kidney is the promising approach for removal of non-dializable, proteinbound uremic toxins, which are responsible for high mortality and morbidity in treating kidney failure related conditions. Additionaly, bioartificial kidney device could perform the physiological roles of the kidney such as metabolic replacement, endocrine function and immunomodulation. In the current work two commercial polyethersulfone-based membranes, Gambro HCO 1100 and Membrana

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25

Silva, Crysthiane Saveriano Rubião. "Apoptose precoce, proliferação celular sincrônica tardia e perfil de expressão de proteínas ao complexo esclerose tuberosa e às doenças renais policísticas durante tubulogênese in vitro." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-01082013-145925/.

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O complexo esclerose tuberosa (CET) e as doenças renais policísticas autossômica dominante (DRPAD) e autossômica recessiva (DRPAR) são doenças monogênicas associadas a cistogênese renal. Os produtos dos genes mutados nessas enfermidades, respectivamente tuberina e hamartina para CET, policistina-1 (PC1) e policistina-2 para DRPAD, e poliductina/fibrocistina para DRPAR, modulam proliferação, diferenciação, apoptose, crescimento e/ou migração celular. Neste estudo empregamos um sistema tridimensional de cultura de células IMCD para caracterizar os perfis de expressão dessas proteínas durante a t

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26

Kozlov, Vladimir. "Étude du rôle des gènes SoxC au cours du développement du rein de souris." Electronic Thesis or Diss., Université Côte d'Azur, 2020. http://www.theses.fr/2020COAZ6010.

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Les anomalies congénitales des reins et des voies urinaires (CAKUT) sont un groupe de malformations fréquemment trouvées chez les patients humains qui résultent de défauts dans le programme de développement de ces organes. Sox11 fait partie de la famille des facteurs de transcription SoxC, qui joue un rôle important dans le développement de divers organes chez les vertébrés. Les souris Sox11-/- meurent juste après la naissance et présentent une grande variété de CAKUT. Alors que les reins duplex sont une anomalie courante du développement rénal, le mécanisme moléculaire qui amène les mutants S

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27

Schnatwinkel, Carsten. "Characterisation of Novel Rab5 Effector Proteins in the Endocytic Pathway." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2004. http://nbn-resolving.de/urn:nbn:de:swb:14-1106824900192-45576.

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Endocytosis, a process of plasma membrane invaginations, is a fundamental cellular mechanism, ensuring uptake of nutrients, enhanced communication between cells, protective functions against invasive pathogens and remodelling of the plasma membrane composition. In turn, endocytic mechanisms are exploited by pathogens to enter their host cells. Endocytosis comprises multiple forms of which our molecular understanding has mostly advanced with respect to clathrin-mediated endocytosis and phagocytosis. Studies on the small GTPase Rab5 have provided important insights into the molecular mechanism o

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28

Nicholson, Benjamin. "The regulation of high affinity glutamate transport a bovine renal epithelial cell line." Thesis, University of Bristol, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336919.

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29

Takayama, Akira. "Transport of cyclosporin A in kidney epithelial cell line (LLC-PK[1])." Kyoto University, 1995. http://hdl.handle.net/2433/160728.

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本文データは平成22年度国立国会図書館の学位論文(博士)のデジタル化実施により作成された画像ファイルを基にpdf変換したものである<br>Kyoto University (京都大学)<br>0048<br>新制・論文博士<br>博士(医学)<br>乙第8919号<br>論医博第1514号<br>新制||医||613(附属図書館)<br>UT51-95-P410<br>(主査)教授藤田潤, 教授吉田修, 教授乾賢一<br>学位規則第4条第2項該当

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30

Parry, Robin Geoffrey. "Cytokines in minimal change nephropathy." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341511.

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31

Shukla, D. H. "Manipulation of the VHL/HIF pathway in mouse kidney epithelia and pancreatic β-cells". Thesis, University College London (University of London), 2011. http://discovery.ucl.ac.uk/1306810/.

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Biallelic inactivation of the von Hippel-Lindau tumour suppressor gene (VHL) underlies both, sporadic clear cell renal cell carcinoma (ccRCC) that arises as part of an inherited multi-cancer VHL syndrome. Pancreatic tumours also occur in VHL disease and are thought to be of neuroendocrine origin. The most extensively studied role of VHL is hypoxia-inducible-factor (HIF) regulation. The HIF pathway is known operates in all cell types examined to date and plays a central role in many physiological responses to oxygen concentration. This thesis investigates the role of the Vhlh tumour suppressor

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32

Ryan, Sean P. "Autosomal Recessive Polycystic Kidney Disease Epithelial Cell Model Reveals Multiple Basolateral EGF Receptor Sorting Pathways." Case Western Reserve University School of Graduate Studies / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=case1274887553.

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33

Li, Moying [Verfasser], and Hans-Joachim [Akademischer Betreuer] Anders. "Mdm2 prevents spontaneous tubular epithelial cell death and acute kidney injury / Moying Li ; Betreuer: Hans-Joachim Anders." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2019. http://d-nb.info/1186629444/34.

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34

Huynh, Carl. "The cytoprotective role of Ras signaling in glomerular epithelial cell injury /." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=112639.

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In experimental membranous nephropathy, complement C5b-9-induced glomerular epithelial cell (GEC) injury leads to breakdown of glomerular peimselectivity and proteinuria. This study addresses mechanisms that limit complement-mediated injury, focusing on Ras. Complement-mediated injury was attenuated in cultured GEC expressing a constitutively active form of Ras (V12Ras), compared with Neo (control) GEC. V12Ras GEC showed constitutive activation of phosphatidylinositol 3-kinase and extracellular signal-regulated kinase pathways, but inhibition of these pathways did not reverse the protective ef

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35

Naillat, F. (Florence). "Roles of Wnt4/5a in germ cell differentiation and gonad development & ErbB4 in polarity of kidney epithelium." Doctoral thesis, Oulun yliopisto, 2011. http://urn.fi/urn:isbn:9789514295751.

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Abstract The embryonic urogenital system generates the metanephric kidneys, the gonads and the adrenal glands, and its development is based on sequential and reciprocal cell and tissue interactions. The mechanisms which regulate urogenital ontogeny are still poorly understood. In this thesis, the roles of Wnt-4 and ErbB4 functions in gonad and kidney development were analysed by using in vivo functional genomic technologies. Wnt-4 is crucial in female development since its absence leads to a partial female to male sex reversal. We found that Wnt-4 mediated the interactions between the somatic

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36

Rondeau, Eric. "Les activateurs du plasminogene du rein humain : identification, localisation, facteurs de secretion." Paris 6, 1987. http://www.theses.fr/1987PA066607.

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Les glomerules isoles du cortex renal chez l'homme, contiennent des activateurs du plasminogene. L'activateur tissulaire du plasminogene (t-pa) represente 80% de l'activite profibrinolytique et l'eurokinase 20% de cette activite. Les cellules epitheliales glomerulaires en culture contiennent l'urokinase. L'activite profibrinolytique des surnageant glomerulaires augmente lorsque les glomerules sont incubes en presence du calcium et a ph alcalin. Les acides gras polyinsatures (acide arachidonique, acide eicosopentaenoique ou acide eicosatrienoique) augmentent cette activite alors que les acides

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37

Murugan, S. (Subramanian). "Control of nephrogenesis by Wnt4 signaling:mechanisms of gene regulation and targeting of specific lineage cells by tissue engineering tools." Doctoral thesis, Oulun yliopisto, 2012. http://urn.fi/urn:isbn:9789526200323.

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Abstract Wnt4, a member of the Wnt family of secreted factors, is essential for kidney organogenesis since the kidney fails to develop in its absence. Besides the kidney, Wnt4 signaling is involved in the control of development of several other organs such as the gonads, adrenal glands and pituitary gland. In the context of the embryonic kidney, Wnt4 signaling induces mesenchymal to epithelial transition of the progenitor cells in the metanephric mesenchyme, an early step in nephrogenesis. Wnt4 signaling may also be relevant in the development of a childhood kidney tumor, the Wilms’ tumor, tha

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38

Toutain, Hervé. "Développement et caractérisation de modèles expérimentaux pour l'étude ex-vivo et in-vitro de la cellule tubulaire proximale de rein de lapin." Rouen, 1989. http://www.theses.fr/1989ROUES036.

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Une suspension de cellules tubulaires proximales de rein de lapin est isolée par une nouvelle méthode qui ne fait pas intervenir d'enzymes protéolytiques. Ces cellules, après une caractérisation biochimique, morphologique, et métabolique, sont séparées en deux populations hautement purifiées par une technique d'électrophorèse ou flux libre en veine liquide. Présentation d'un modèle de culture primaire de cellules tubulaires proximales de rein de lapin

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39

Friedlander, Gérard. "Etude des facteurs qui modulent l'effet des hormones sur des cellules epitheliales d'origine renale." Paris 7, 1987. http://www.theses.fr/1987PA077204.

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40

Ruscica, Biagina. "The critical role of YAP and TAZ in tubular homeostasis." Electronic Thesis or Diss., Université Paris Cité, 2024. https://wo.app.u-paris.fr/cgi-bin/WebObjects/TheseWeb.woa/wa/show?t=6623&f=77103.

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Des études épidémiologiques et expérimentales suggèrent que la progression de la maladie rénale chronique (MRC) après une lésion initiale est génétiquement déterminée, mais les réseaux génétiques qui contribuent à cette prédisposition restent inconnus. Parmi les voies moléculaires potentielles impliquées dans la MRC, cette étude s'est concentrée sur la voie Hippo, une cascade de signalisation conservée au cours de l'évolution et cruciale pour la régulation de la taille des organes et de la prolifération cellulaire. Les protéines paralogues YAP et TAZ, deux coactivateurs transcriptionnels de la

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41

Yang, An-Hang. "Reactive oxygen metabolism in cultured kidney epithelial cells." 1987. http://catalog.hathitrust.org/api/volumes/oclc/16310902.html.

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42

Ren, Shuyu. "The regulation of Nephrin expression in kidney epithelial cells and during inflammatory kidney diseases /." 2005. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=014191117&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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43

Dai, Long-jun. "Control of intracelluar Ca²⁺ in epithelial cells of the kidney thick ascending limb." Thesis, 1995. http://hdl.handle.net/2429/7260.

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The cortical thick ascending limb of Henle’s loop (cTAL) plays a fundamental role in salt reabsorption and concentration-dilution processes within the nephron. This study was performed to characterize the role of intracellular free Ca²⁺ ([Ca²⁺]i) in hormone-mediated signal transduction and to describe the function of Na⁺/Ca²⁺ exchange in control of [Ca²⁺]i in isolated cTAL cells from porcine kidney. Parathyroid hormone, arginine vasopressin, and atrial natriuretic peptide transiently increased [Ca²⁺]i, in a dose-dependent manner. The increment in [Ca²⁺]i induced by these hormones was by

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44

Music, Ena. "Stress-induced accumulation of heme oxygenase-1 in Xenopus laevis A6 kidney epithelial cells." Thesis, 2014. http://hdl.handle.net/10012/8403.

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Abstract Previous studies have examined stress-induced heme oxygenase-1 (HO-1) expression primarily in mammalian systems. The present study examines, for the first time in amphibians, the effect of heat shock, sodium arsenite, cadmium chloride, and the proteasomal inhibitor MG132 on HO-1 accumulation in Xenopus laevis A6 kidney epithelial cells. Western blot analysis revealed that exposure of A6 cells to a range of heat shock temperatures (30-35 °C), which induced HSP30 accumulation, did not induce HO-1 accumulation. In contrast, cells treated with sodium arsenite (5-50 μM), cadmium chloride

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45

Payne, Emily Harman. "Polyploidy and Mitotic Cell Death are Two Distinct HIV-1 Vpr-Driven Outcomes in Renal Tubule Epithelial Cells." Diss., 2016. http://hdl.handle.net/10161/12190.

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<p>Given the emerging epidemic of renal disease in HIV+ patients and the fact that HIV DNA and RNA persist in the kidneys of HIV+ patients despite therapy, it is necessary to understand the role of direct HIV-1 infection of the kidney. HIV-associated kidney disease pathogenesis is attributed in large part to viral proteins. Expression of Vpr in renal tubule epithelial cells (RTECs) induces G2 arrest, apoptosis and polyploidy. The ability of a subset of cells to overcome the G2/M block and progress to polyploidy is not well understood. Polyploidy frequently associates with a bypass of cell dea

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46

Lin, Pei-ying, and 林佩瑩. "Effect of zingerone on the anti-oxidation and anti-inflammatory in rat kidney epithelial cells." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/62233197664593538772.

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碩士<br>大仁科技大學<br>食品科技研究所<br>98<br>Ginger is a common spice used in popular cuisine and has been widely applied in research of diseases due to its rich ingredients. The purpose of this research is to probe into the antioxidation activity and anti-inflammatory effect of zingerone (ZO) of NRK 52E cells (Normal rat kidney epithelial cells). The experiment design covers two parts: the first test is “In vitro” in which ZO of different concentrations (i.e. 1, 5, 7.5, 10 mM) are used to analyze DPPH activity, SOD, FIC and reducing power. The results show that ZO demonstrates good effects in removing DP

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47

Woolfson, Jessica Pearl. "Examination of Cadmium-Induced Heat Shock Protein Gene Expression in Xenopus laevis A6 Kidney Epithelial Cells." Thesis, 2008. http://hdl.handle.net/10012/3723.

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Cadmium is a highly toxic chemical and has been classified by the International Agency for Research on Cancer as a human carcinogen. Cadmium is abundant in the environment, at specific work places, and in food and water. Toxicological responses to cadmium exposure include respiratory diseases, neurological disorders and kidney damage. The present study examined the effects of cadmium on heat shock protein (HSP) accumulation in Xenopus laevis A6 kidney epithelial cells. HSPs are molecular chaperones involved in protein folding and translocation. In response to environmental stress these protein

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Khan, Saad. "Examination of curcumin-induced heat shock protein gene expression in Xenopus laevis A6 kidney epithelial cells." Thesis, 2010. http://hdl.handle.net/10012/5338.

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Abstract:

The heat shock response is a cellular homeostatic mechanism that is activated in response to stressful stimuli (e.g. heat shock, heavy metals, disease states etc.), which causes an increase in unfolded protein, which triggers the expression of heat shock protein (hsp) genes. HSPs are molecular chaperones that assist in protein synthesis, folding and degradation and prevent stress-induced protein aggregation. Since stressor-induced tissue damage is associated with different disease states, indirect evidence suggested that HSP inducers may be therapeutically beneficial for certain diseases. Curc

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49

Khan, Saad. "Analysis of heat shock protein 30 gene expression and function in Xenopus laevis A6 kidney epithelial cells." Thesis, 2014. http://hdl.handle.net/10012/8398.

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Heat shock proteins (HSPs) are molecular chaperones that assist in protein synthesis, folding and degradation and prevent stress-induced protein aggregation. The present study examined the pattern of accumulation of HSP30 and HSP70 in cells recovering from heat shock as well as the effect of proteasome inhibition on cytoplasmic/nuclear and endoplasmic reticulum (ER) molecular chaperone accumulation, large multimeric HSP30 complexes, stress granule and aggresome formation in Xenopus laevis A6 kidney epithelial cells. Initial immunoblot analysis revealed the presence of elevated levels of HSP30

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50

Bellin, Gloria. "Heparanase regulates M1 macrophage polarization and the crosstalk between macrophages and tubular epithelial cells after kidney ischemia/reperfusion injury." Doctoral thesis, 2018. http://hdl.handle.net/11562/985324.

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l’HPSE è un elemento chiave coinvolto nei complessi processi biologici attivati dal danno da I/R, regolando la attivazione/polarizzazione dei macrofagi e il crosstalk tra queste cellule infiammatorie e l’epitelio tubulare renale.

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